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1 licles on the face, neck, chest, and back by Propionibacterium acnes.
2 e immune response to the commensal bacterium Propionibacterium acnes.
3 antimicrobial activity against S. aureus and Propionibacterium acnes.
4 es, is caused by multiple factors, including Propionibacterium acnes.
5 ion of 2.0 micrograms per milliliter against Propionibacterium acnes.
6 against Escherichia coli., and 12 mm against Propionibacterium acnes.
7 46% vs 80%; 3) the most frequent isolate was Propionibacterium acnes (11/26) vs coagulase-negative St
8 iome at the strain level and genome level of Propionibacterium acnes, a dominant skin commensal, betw
9  mechanism by which S. aureus may commandeer Propionibacterium acnes, a key member of the human skin
10 In this issue, Kistowska et al. confirm that Propionibacterium acnes activates inflammasomes leading
11                                              Propionibacterium acnes and coagulase-negative Staphyloc
12 asts, 6 nonfermenters, and 1 isolate each of Propionibacterium acnes and coagulase-negative staphyloc
13 olleagues present strain-based resolution of Propionibacterium acnes and its association with the com
14  the presence of the Gram-positive bacterium Propionibacterium acnes and its potential association wi
15                      In vivo Th1 response to Propionibacterium acnes and lipopolysaccharide in IFN-ga
16 nished in the sera of ICE-/- mice exposed to Propionibacterium acnes and lipopolysaccharide.
17 s associated with formation of resistance in Propionibacterium acnes and other bacteria, with clinica
18 ndependent growth, requiring helpers such as Propionibacterium acnes and Prevotella intermedia for st
19 on in common skin commensal bacteria such as Propionibacterium acnes and Staphylococcus epidermitis.
20              Low virulence organisms such as Propionibacterium acnes are the most common culprit orga
21                     We report a patient with Propionibacterium acnes bacteremia and late prosthetic v
22 ites from the abundant skin-resident microbe Propionibacterium acnes can influence cytokine expressio
23                                              Propionibacterium acnes, causative agent of chronic pros
24 n are compounded by increasing resistance of Propionibacterium acnes clinical isolates.
25 Bradyrhizobium, Anaerococcus, Peptoniphilus, Propionibacterium acnes, Dorea, and Ruminococcus and red
26 rial pathogens including Bacillus anthracis, Propionibacterium acnes, Enterococcus faecalis, and both
27           Among the airborne microorganisms, Propionibacterium acnes, Escherichia coli, Acinetobacter
28 resulting from the immune response targeting Propionibacterium acnes has a significant role in its pa
29                                              Propionibacterium acnes has been identified as a signifi
30                 The microaerophylic organism Propionibacterium acnes has shown consistent association
31 e primed 12 days in advance with heat-killed Propionibacterium acnes, IL-18BP-Fc prevents LPS-induced
32 ase-negative staphylococci in 6.0% (27/448), Propionibacterium acnes in 4.7% (21/448), and Pseudomona
33 ica charantia Linn. var. abbreviata Ser.) on Propionibacterium acnes-induced inflammation and to iden
34 us studies showed that TLR9 and TLR2 mediate Propionibacterium acnes-induced sensitization to lipopol
35                      Agak et al. report that Propionibacterium acnes induces IL-17 expression in peri
36                                              Propionibacterium acnes induction of IL-1 cytokines thro
37                                              Propionibacterium acnes induction of inflammatory respon
38  have been sensitized to endotoxin damage by Propionibacterium acnes infection.
39                                              Propionibacterium acnes is a critical component in the p
40                                              Propionibacterium acnes is a key pathogen involved in th
41                                              Propionibacterium acnes is a key therapeutic target in a
42                                              Propionibacterium acnes is a known cause of postneurosur
43                                              Propionibacterium acnes is a major etiological factor of
44                  The Gram-positive bacterium Propionibacterium acnes is a member of the normal human
45                                              Propionibacterium acnes is a skin commensal bacterium th
46                                              Propionibacterium acnes is a well-known cause of delayed
47                                              Propionibacterium acnes is also associated with inflamma
48             The opportunistic human pathogen Propionibacterium acnes is composed of a number of disti
49                                              Propionibacterium acnes is increasingly recognized as an
50                Cutibacterium acnes (formerly Propionibacterium acnes) is recognized as a pathogen in
51                                              Propionibacterium acnes isolates usually have relatively
52  1), Propionibacterium species (n = 15), and Propionibacterium acnes (n = 19) isolates; all of these
53 stigated the in vitro susceptibilities of 23 Propionibacterium acnes ophthalmic isolates to ertapenem
54     Mycothiol production was not detected in Propionibacterium acnes or in representative species of
55 S (20 microg/mouse) 7 days after heat-killed Propionibacterium acnes (P. acnes) injection into wild-t
56 monstrate that fermentation of glycerol with Propionibacterium acnes (P. acnes), a skin commensal bac
57 ne treatment as a natural antibiotic against Propionibacterium acnes (P. acnes), which promotes folli
58 e investigated the roles of TLR2 and TLR4 in Propionibacterium acnes (P. acnes)-primed, LPS-induced l
59 nd cDC2s during bacterial infection, notably Propionibacterium acnes (P. acnes).
60      In patients with AgP, Actinomyces oris, Propionibacterium acnes, P. aeruginosa, Staphylococcus a
61  relative abundances of propionibacteria and Propionibacterium acnes phage in healthy skin.
62 overed, including Escherichia coli phage T3, Propionibacterium acnes phage PA6, and Streptococcus mit
63 on of early childhood acne, the emergence of Propionibacterium acnes resistance, and the rare but ser
64 teen of the species or phylotypes, including Propionibacterium acnes, Staphylococcus spp., and the op
65  observed the decline of an early-colonizing Propionibacterium acnes strain similar to SK137 and the
66 ny countries reporting that more than 50% of Propionibacterium acnes strains are resistant to topical
67 es of the human skin microbiome suggest that Propionibacterium acnes strains may contribute different
68 ddress the emergence of antibiotic-resistant Propionibacterium acnes strains.
69  Prevotella oris, Staphylococcus aureus, and Propionibacterium acnes strains.
70 and four different Cutibacterium (previously Propionibacterium) acnes strains, and compare outcomes w
71 s-wide comparative analysis of 90 genomes of Propionibacterium acnes that represent the known diversi
72 rect antimicrobial activity in vitro against Propionibacterium acnes, the bacterium linked to the pat
73 nflammation is due in part to the ability of Propionibacterium acnes to activate TLR2.
74                    C57BL/6 mice treated with Propionibacterium acnes to elicit high levels of macroph
75 ntial evidence that links the skin bacterium Propionibacterium acnes to the condition.
76 nas and Peptoniphilus species (type IV), and Propionibacterium acnes (type V).
77       The culture supernatant of a strain of Propionibacterium acnes was investigated for its phospho
78                                              Propionibacterium acnes was the commonest species detect
79 ct to sequence multiple clinical isolates of Propionibacterium acnes, we have produced a draft genome
80 teria such as Staphylococcus epidermidis and Propionibacterium acnes, were identified in bacteriologi
81 ulture showed antimicrobial activity against Propionibacterium acnes, whereas the enhanced antimicrob
82 cular hyperkeratinization, and the action of Propionibacterium acnes within the follicle.
83 t contributes to the pathogenesis of acne is Propionibacterium acnes; yet, the molecular mechanism by