ライフサイエンスコーパス: Pseudomonas aeruginosa

1 ia coli) and non-fermenting organisms (e.g., Pseudomonas aeruginosa).

2 airway epithelial cells and cocultures with Pseudomonas aeruginosa.

3 traits in opportunistic pathogens including Pseudomonas aeruginosa.

4 hat often retains activity against resistant Pseudomonas aeruginosa.

5 evelopment of effective strategies to target Pseudomonas aeruginosa.

6 oad array of bacteria, including E. coli and Pseudomonas aeruginosa.

7 Escherichia coli, Citrobacter rodentium, and Pseudomonas aeruginosa.

8 it from three Podoviridae phages that infect Pseudomonas aeruginosa.

9 nas gingivalis, Fusobacterium nucleatum, and Pseudomonas aeruginosa.

10 ffector, Tas1, in the opportunistic pathogen Pseudomonas aeruginosa.

11 nsidering the 'biofilm formation process' in Pseudomonas aeruginosa.

12 S) production and anthranilate metabolism in Pseudomonas aeruginosa.

13 mophilus influenzae, with later emergence of Pseudomonas aeruginosa.

14 mal antimicrobial assays were performed with Pseudomonas aeruginosa.

15 lipid A biosynthesis pathway is essential in Pseudomonas aeruginosa.

16 obacter baumannii, Klebsiella pneumoniae and Pseudomonas aeruginosa.

17 O-Class II genes, and enhanced resistance to Pseudomonas aeruginosa.

18 ce-independent flow sensing in the bacterium Pseudomonas aeruginosa.

19 was restored among multidrug-resistant (MDR) Pseudomonas aeruginosa.

20 Escherichia coli, Citrobacter rodentium and Pseudomonas aeruginosa.

21 nem-resistant acinetobacter species, and MDR Pseudomonas aeruginosa.

22 nge in the treatment of infections caused by Pseudomonas aeruginosa.

23 iocontrol agents to the major human pathogen Pseudomonas aeruginosa.

24 tam (TOL-TAZ) affords broad coverage against Pseudomonas aeruginosa.

25 thogens were Staphylococcus aureus (34%) and Pseudomonas aeruginosa (17%), whereas blood cultures mos

26 ogens were Klebsiella pneumoniae (25.6%) and Pseudomonas aeruginosa (18.9%).

27 59%), followed by Fusarium spp. (4/17; 24%), Pseudomonas aeruginosa (2/17; 12%), and Curvularia spp.

28 The most commonly isolated organism was Pseudomonas aeruginosa (21%).

29 The 2 most common bacterial isolates were Pseudomonas aeruginosa (35.2%) and Staphylococcus specie

30 Qualifying baseline pathogens: Pseudomonas aeruginosa (77%), Klebsiella spp. (16%), oth

31 VRE with greatest reductions for MRSA (97%), Pseudomonas aeruginosa (80%), S. aureus (77%) and Candid

32 athogens in liver transplant recipients, and Pseudomonas aeruginosa (9%) in lung transplant recipient

33 We test our predictions by competing Pseudomonas aeruginosa (a tit-for-tat species) with Vibr

34 PapRecT enables efficient recombineering in Pseudomonas aeruginosa, a concerning human pathogen.

35 Pseudomonas aeruginosa, a Gram-negative bacterium that c

36 sis is the key mechanism for host control of Pseudomonas aeruginosa, a motile Gram-negative, opportun

37 Pseudomonas aeruginosa, a significant opportunistic path

38 Emerging evidence suggests the Pseudomonas aeruginosa accessory genome is enriched with

39 The bacterial pathogen Pseudomonas aeruginosa activates expression of many viru

40 Pathogens such as Pseudomonas aeruginosa advantageously modify animal host

41 development associated with the isolation of Pseudomonas aeruginosa after lung transplantation.

42 reviously characterized in the regulation of Pseudomonas aeruginosa alginate biosynthesis(3,4), as a

43 Furthermore, proteome profiling of a Pseudomonas aeruginosa AlgP transposon mutant provided u

44 Multiple clinical isolates of Pseudomonas aeruginosa, an important human pathogen, hav

45 mprising exotoxin A domain III (PE-III) from Pseudomonas aeruginosa and a cancer-specific antibody fr

46 kingdom QS interactions between a bacterium, Pseudomonas aeruginosa and a yeast, Candida albicans, in

47 n-resistant Staphylococcus aureus (MRSA) and Pseudomonas aeruginosa and accelerates their removal by

48 icant improvement in the inactivation of MDR Pseudomonas aeruginosa and Acinetobacter baumannii (plan

49 undreds of bacteria, including the pathogens Pseudomonas aeruginosa and Acinetobacter baumannii.

50 Pseudomonas aeruginosa and Burkholderia cepacia complex

51 tam antibiotics against carbapenem-resistant Pseudomonas aeruginosa and carbapenem-resistant Enteroba

52 -resistant S. epidermidis, Escherichia coli, Pseudomonas aeruginosa and Enterococcus faecalis.

53 Using microfluidic experiments with Pseudomonas aeruginosa and Escherichia coli, we demonstr

54 dal activity toward both Gram stain-negative Pseudomonas aeruginosa and Gram stain-positive Staphyloc

55 tablished that UQ(9) is the major quinone of Pseudomonas aeruginosa and is required for growth under

56 fically, by using the opportunistic pathogen Pseudomonas aeruginosa and its phage DMS3vir, we show th

57 he basis of crystal structures of TrmDs from Pseudomonas aeruginosa and Mycobacterium tuberculosis, w

58 m progressive and severe pneumonia caused by Pseudomonas aeruginosa and poor wound healing.

59 Pseudomonas aeruginosa and Staphylococcus aureus are opp

60 Adherence of Pseudomonas aeruginosa and Staphylococcus aureus on sPCM

61 xperimentally using both model membranes and Pseudomonas aeruginosa and Staphylococcus aureus, repres

62 own to be coisolated from chronic illnesses, Pseudomonas aeruginosa and Staphylococcus aureus, we obs

63 i, Salmonella enteritidis, Listeria innocua, Pseudomonas aeruginosa and Streptococcus pneumoniae did

64 e is characterized by chronic infection with Pseudomonas aeruginosa and sustained neutrophil-dominant

65 uch as the well-studied relationship between Pseudomonas aeruginosa and the nematode Caenorhabditis e

66 l activity against Staphylococcus aureus and Pseudomonas aeruginosa and with their geographical origi

67 quisition of composite tandem mass spectra ( Pseudomonas aeruginosa), and (3) the overabundance of se

68 encompassing 288 Staphylococcus aureus, 456 Pseudomonas aeruginosa, and 1588 Escherichia coli genome

69 d to interpret results for Enterobacterales, Pseudomonas aeruginosa, and Acinetobacter baumannii comp

70 ens Escherichia coli, Klebsiella pneumoniae, Pseudomonas aeruginosa, and Acinetobacter baumannii with

71 bacteria (Escherichia coli, Klebsiella spp., Pseudomonas aeruginosa, and Acinetobacter baumannii) res

72 emophilus influenzae, Staphylococcus aureus, Pseudomonas aeruginosa, and Aspergillus infections were

73 5 to 2 mg/L for MDR Gram-negative, excluding Pseudomonas aeruginosa, and between 0.03 and 1 mg/L for

74 bsiella pneumoniae, Acinetobacter baumannii, Pseudomonas aeruginosa, and Enterobacter species(ESKAPE)

75 bsiella pneumoniae, Acinetobacter baumannii, Pseudomonas aeruginosa, and Enterobacter species) pathog

76 bsiella pneumoniae, Acinetobacter baumannii, Pseudomonas aeruginosa, and Enterobacter spp. were analy

77 bsiella pneumoniae, Acinetobacter baumannii, Pseudomonas aeruginosa, and Enterobacter spp.) directly

78 tory activity against Staphylococcus aureus, Pseudomonas aeruginosa, and Enterococcus faecalis althou

79 With hosts such as Salmonella enterica, Pseudomonas aeruginosa, and Erwinia amylovora, these pha

80 uation in PDI against Staphylococcus aureus, Pseudomonas aeruginosa, and Escherichia coli A significa

81 oteobacteria motors: Legionella pneumophila, Pseudomonas aeruginosa, and Shewanella oneidensis, provi

82 ms of uropathogenic Escherichia coli (UPEC), Pseudomonas aeruginosa, and Staphylococcus aureus, with

83 ebsiella pneumoniae, Acinetobacter baumanii, Pseudomonas aeruginosa, and Staphylococcus aureus.

84 onella enterica, Mycobacterium tuberculosis, Pseudomonas aeruginosa, and Staphylococcus aureus.

85 e bacteria, such as Acinetobacter baumannii, Pseudomonas aeruginosa, and Stenotrophomonas maltophilia

86 rally required for growth arrest survival of Pseudomonas aeruginosa, and that this requirement is ind

87 acteria, including Staphylococcus aureus and Pseudomonas aeruginosa, and their cell-wall components L

88 luding the human pathogens Escherichia coli, Pseudomonas aeruginosa, and Vibrio cholerae, and the pla

89 Chronic infections by Pseudomonas aeruginosa are characterized by biofilm form

90 Staphylococcus pseudintermedius and Pseudomonas aeruginosa are frequently identified in thes

91 bacter baumannii, Staphylococcus aureus, and Pseudomonas aeruginosa are three difficult-to-treat biof

92 ials (e.g., -0.2 to 1.0 V vs Ag/AgCl), using Pseudomonas aeruginosa as a model organism.

93 e of a SctK protein family member, PscK from Pseudomonas aeruginosa, as well as the structure of its

94 in an untargeted fashion while in contrast, Pseudomonas aeruginosa assembles and fires its T6SS appa

95 taneously visualized from single cultures of Pseudomonas aeruginosa at about 50 mum resolution.

96 3353, Klebsiella pneumoniae ATCC 700603, and Pseudomonas aeruginosa ATCC 27853 were chosen as referen

97 ing arms specific for Staphylococcus aureus, Pseudomonas aeruginosa, B-cells and T-cells, indicating

98 The optimal antibiotic regimen for Pseudomonas aeruginosa bacteremia is controversial.

99 Health care-associated infections such as Pseudomonas aeruginosa bacteremia pose a major clinical

100 hted cohort of 249 adults with uncomplicated Pseudomonas aeruginosa bacteremia, patients receiving sh

101 ding single eukaryotic cells infected by 1-3 Pseudomonas aeruginosa bacteria and paired host-pathogen

102 Pseudomonas aeruginosa belongs to the group of three "cr

103 challenging sample (>90% water) of a mature Pseudomonas aeruginosa biofilm in its native state.

104 As an example, Pseudomonas aeruginosa biofilm is grown from single cell

105 Pseudomonas aeruginosa biofilms are composed of exopolys

106 . fumigatus and Pel polysaccharide-dependent Pseudomonas aeruginosa biofilms at nanomolar concentrati

107 r the range that was previously measured for Pseudomonas aeruginosa biofilms grown from clinical bact

108 of the in vitro electroceutical treatment of Pseudomonas aeruginosa biofilms is demonstrated both at

109 -cost method was proposed for the imaging of Pseudomonas aeruginosa biofilms on metallic surfaces usi

110 contributes to the structure and function of Pseudomonas aeruginosa biofilms.

111 nteractions with extracellular DNA (eDNA) in Pseudomonas aeruginosa biofilms.

112 vaU act coordinately as global repressors in Pseudomonas aeruginosa by binding to AT-rich regions of

113 has been shown to initiate OMV formation in Pseudomonas aeruginosa by interacting with the outer mem

114 its DNA from immunity nucleases of its host, Pseudomonas aeruginosa, by constructing a proteinaceous

115 iofilms of the pathogens Vibrio cholerae and Pseudomonas aeruginosa can induce large deformations of

116 that blocking the peptidoglycan recycling in Pseudomonas aeruginosa causes an important virulence imp

117 Pseudomonas aeruginosa causes severe multidrug-resistant

118 e and induces an iron starvation response in Pseudomonas aeruginosa cells by sequestering Fe(II) at i

119 fy the aggregation levels of cystic fibrosis Pseudomonas aeruginosa (CF-PA) isolates in vitro using 3

120 injury in CF mice after intratracheal LPS or Pseudomonas aeruginosa challenge.

121 atelet activation after intratracheal LPS or Pseudomonas aeruginosa challenge.

122 (GFP) to construct a novel SDS biosensor in Pseudomonas aeruginosa chassis.

123 piric coverage (e.g., Staphylococcus aureus, Pseudomonas aeruginosa, Clostridium difficile, and funga

124 lone and during polymicrobial infection with Pseudomonas aeruginosa Colonization, persistence, and vi

125 Pseudomonas aeruginosa colonizing airways is consistentl

126 ree crystal structures of the TBDT FoxA from Pseudomonas aeruginosa (containing a signalling domain)

127 The cytoplasm of Pseudomonas aeruginosa contains a Cu(+)-sensing transcri

128 ke many bacteria, the opportunistic pathogen Pseudomonas aeruginosa contains two ClpP homologs: ClpP1

129 Here, we reveal how the pathogen Pseudomonas aeruginosa coordinates the motorized activit

130 enemase producers among carbapenem-resistant Pseudomonas aeruginosa (CRPA) isolates warrants an expan

131 e (CRE), Acinetobacter baumannii (CRAB), and Pseudomonas aeruginosa (CRPsA) are a serious cause of he

132 n breakpoints for the Enterobacteriaceae and Pseudomonas aeruginosa, daptomycin breakpoints for Enter

133 ess this question, we purified a five-member Pseudomonas aeruginosa division complex consisting of Ft

134 terobacterales, Acinetobacter baumannii, and Pseudomonas aeruginosa during susceptibility testing.

135 It has been shown that Pseudomonas aeruginosa elastase (LasB) and Clostridium h

136 spond to bacterial challenge with 25,000 CFU Pseudomonas aeruginosa embedded into agarose beads to sl

137 ng members of the order Enterobacterales and Pseudomonas aeruginosa, enriched for drug resistance.

138 fy independent risk factors for mortality in Pseudomonas aeruginosa episodes.

139 d piperacillin-tazobactam on the recovery of Pseudomonas aeruginosa, Escherichia coli, and Klebsiella

140 Pseudomonas aeruginosa exhibits a high requirement for i

141 Next, a hexasaccharide fragment of the Pseudomonas aeruginosa exopolysaccharide Pel was assembl

142 Instead, Pseudomonas aeruginosa exposed to CSE (CSE-PSA) had incr

143 In Pseudomonas aeruginosa, expression of the T3SS is regula

144 We demonstrated that their preexposure to Pseudomonas aeruginosa flagellin modify their inflammato

145 t a change in the rates of ESBL, CRE and MDR Pseudomonas aeruginosa following ASP.

146 istant E. coli (Spain), gentamicin-resistant Pseudomonas aeruginosa (France) and methicillin-resistan

147 nical isolates of the opportunistic pathogen Pseudomonas aeruginosa from patients with cystic fibrosi

148 large-scale comparison of multiple published Pseudomonas aeruginosa gene essentiality datasets, revea

149 The human pathogen Pseudomonas aeruginosa harbors three paralogous zinc pro

150 In particular, electrogenic Pseudomonas aeruginosa has been studied with the utility

151 IGPS from Pseudomonas aeruginosa has the highest turnover number o

152 (PTCR), to investigate large proteoforms of Pseudomonas aeruginosa in a high-throughput fashion.

153 d for treatment of acute lung infection with Pseudomonas aeruginosa in a mouse model.

154 so efficacious at killing the model organism Pseudomonas aeruginosa in biofilms and in a murine chron

155 ects the lung from injury upon intratracheal Pseudomonas aeruginosa in mice.

156 We hypothesized that the isolation of Pseudomonas aeruginosa in respiratory specimens would in

157 rcentage of predicted, SGRQ total score, and Pseudomonas aeruginosa in sputum culture at baseline.

158 strate that upon hitting a host cell, motile Pseudomonas aeruginosa induce a specific gene expression

159 helminth-Heligmosomoides polygyrus infected; Pseudomonas aeruginosa infected; and coinfected.

160 OR, 1.08; P = 3.4 x 10-10), and intermittent Pseudomonas aeruginosa infection (OR, 1.51; P = .004) we

161 Pseudomonas aeruginosa infection elicits the production

162 ng protein (BPI) is strongly associated with Pseudomonas aeruginosa infection in cystic fibrosis (CF)

163 his study to track the IFN-gamma response to Pseudomonas aeruginosa infection in the lung over time i

164 Results for patients with and without Pseudomonas aeruginosa infection were compared.

165 tactic stimuli (a CXCL1 chemokine and a live Pseudomonas aeruginosa infection) were administered 48 h

166 ithelium is seriously damaged upon pulmonary Pseudomonas aeruginosa infection, especially in cystic f

167 three or more exacerbations per year without Pseudomonas aeruginosa infection.

168 Pseudomonas aeruginosa infections are increasingly multi

169 ed by a prophage in P. aeruginosa IMPORTANCE Pseudomonas aeruginosa is a Gram-negative bacterium freq

170 The opportunistic pathogen Pseudomonas aeruginosa is a leading cause of morbidity a

171 The opportunistic bacterial pathogen Pseudomonas aeruginosa is a leading cause of serious inf

172 The opportunistic pathogen Pseudomonas aeruginosa is a major cause of antibiotic-to

173 beta-lactam resistance in pathogens such as Pseudomonas aeruginosa is a major clinical challenge.

174 Pseudomonas aeruginosa is a priority pathogen for the de

175 Pseudomonas aeruginosa is an extracellular opportunistic

176 Pseudomonas aeruginosa is an opportunistic bacterium of

177 Pseudomonas aeruginosa is an opportunistic human pathoge

178 Pseudomonas aeruginosa is an opportunistic multidrug-res

179 Pseudomonas aeruginosa is an opportunistic pathogen in b

180 Pseudomonas aeruginosa is an opportunistic pathogen that

181 Pseudomonas aeruginosa is an opportunistic pathogen that

182 Pseudomonas aeruginosa is an opportunistic pathogen that

183 Pseudomonas aeruginosa is an opportunistic pathogenic ba

184 fibrosis bronchiectasis, lung infection with Pseudomonas aeruginosa is associated with frequent pulmo

185 The prevalence of carbapenem-resistant Pseudomonas aeruginosa is increasing.

186 Searching for new strategies to defeat Pseudomonas aeruginosa is of paramount importance.

187 The opportunistic pathogen Pseudomonas aeruginosa is responsible for much of the mo

188 Live Pseudomonas aeruginosa, isolated from a patient, was giv

189 Also, 46.2% of Pseudomonas aeruginosa isolates were carbapenem-resistan

190 terobacterales, Acinetobacter baumannii, and Pseudomonas aeruginosa isolates were evaluated across th

191 nited States, where 309 Enterobacterales and Pseudomonas aeruginosa isolates were evaluated by NG-Tes

192 We previously identified Pseudomonas aeruginosa isolates with characteristics typ

193 ed DNA- and RNA-sequencing reads of clinical Pseudomonas aeruginosa isolates with clinically relevant

194 rs (126 isolates of the Enterobacterales, 50 Pseudomonas aeruginosa isolates, and 50 Acinetobacter sp

195 isolates (775 Enterobacterales isolates, 119 Pseudomonas aeruginosa isolates, and 83 Acinetobacter ba

196 rtain enteroaggregative Escherichia coli and Pseudomonas aeruginosa isolates, whereas to other bacter

197 of the environmental opportunistic pathogen Pseudomonas aeruginosa, it has been shown that overexpre

198 um infected with clinical isolates of either Pseudomonas aeruginosa, Klebsiella pneumoniae, or Staphy

199 f-5 as a model to elucidate PelX function as Pseudomonas aeruginosa lacks a pelX homologue in its pel

200 Here, we use the Pseudomonas aeruginosa LasR quorum-sensing receptor to e

201 In the pathogen Pseudomonas aeruginosa, LasR is a quorum sensing (QS) ma

202 bioavailable C-glycosidic inhibitors of the Pseudomonas aeruginosa lectin LecB with antibiofilm acti

203 ologous to the historic phage B3 that infect Pseudomonas aeruginosa Like other phage groups, the B3-l

204 Pseudomonas aeruginosa, like many bacilliforms, are not

205 cocrystal structures of inhibitors bound to Pseudomonas aeruginosa LpxC as guides, resulted in the d

206 luc signals could be spectrally unmixed from Pseudomonas aeruginosa lux signals to noninvasively moni

207 All FARs showed bactericidal effect against Pseudomonas aeruginosa, making PA the most susceptible o

208 used whole genome sequencing to characterise Pseudomonas aeruginosa MDR clinical isolates from a hosp

209 The Pseudomonas aeruginosa methyltransferase EftM trimethyla

210 In Pseudomonas aeruginosa, MexAB-OprM plays a central role

211 ug/mL), and also exhibited activity against Pseudomonas aeruginosa (MIC 16 ug/mL) and Staphylococcus

212 1.3 versus TT = 3.3 +/- 0.7 neutrophils/mL; Pseudomonas aeruginosa: mTBI = 9.4 +/- 1.4 versus TT = 5

213 Citrobacter rodentium, Escherichia coli, or Pseudomonas aeruginosa mutant strain DeltapopB Moreover,

214 Acinetobacter baumannii (n = 3687 [26%]) and Pseudomonas aeruginosa (n = 3176 [22%]) were leading cau

215 highest for Enterobacterales and lowest for Pseudomonas aeruginosa Nevertheless, even for Enterobact

216 gation-ready trisaccharide repeating unit of Pseudomonas aeruginosa O11 via a highly stereoselective

217 to define essential genes in nine strains of Pseudomonas aeruginosa on five different media and devel

218 using culture-dependent methods to quantify Pseudomonas aeruginosa, opportunistic pathogens capable

219 epidermidis but did not inhibit biofilms by Pseudomonas aeruginosa or Bacillus subtilis, and inhibit

220 at 48 and 24 hours prior to intraperitoneal Pseudomonas aeruginosa or IV Staphylococcus aureus infec

221 ptococcus pneumoniae, Staphylococcus aureus, Pseudomonas aeruginosa, or Moraxella catarrhalis.

222 During infection of a host, Pseudomonas aeruginosa orchestrates global gene expressi

223 The chronic infections by pathogenic Pseudomonas aeruginosa (P. aeruginosa) remain to be prop

224 o investigate the prevalence, antibiogram of Pseudomonas aeruginosa (P. aeruginosa), and the distribu

225 The opportunistic human pathogen Pseudomonas aeruginosa (Pa) causes several infections ac

226 for filamentous Pf bacteriophage produced by Pseudomonas aeruginosa (Pa) in suppression of immunity a

227 Elimination of pulmonary Pseudomonas aeruginosa (PA) infections is challenging to

228 sensitivity analyses based on lung function, Pseudomonas aeruginosa (PA) status, and follow-up time i

229 A), vancomycin-resistant Enterococcus (VRE), Pseudomonas aeruginosa (PA), and Candida albicans (CA)].

230 system from the opportunistic human pathogen Pseudomonas aeruginosa (Pa).

231 r bacteria, including the common CF pathogen Pseudomonas aeruginosa (Pa).

232 thogens, Escherichia coli (Ec, m/z 1797) and Pseudomonas aeruginosa (Pa, m/z 1446) using on-tissue ac

233 xoelectrogens, Shewanella oneidensis MR1 and Pseudomonas aeruginosa PA01, and many other mutants of t

234 purified small RNAs isolated from pathogenic Pseudomonas aeruginosa (PA14) is sufficient to induce pa

235 ave learned to avoid the pathogenic bacteria Pseudomonas aeruginosa (PA14), they pass this learned be

236 tudy the biochemical properties of FtsZ from Pseudomonas aeruginosa (PaFtsZ) and the effects of its t

237 Pseudomonas aeruginosa PAO1, an opportunistic human path

238 sponses in the lungs from mice infected with Pseudomonas aeruginosa (PAO1) (n = 10 per group) and in

239 e kinases from Escherichia coli (EcPanK) and Pseudomonas aeruginosa (PaPanK), we found ADP to be an e

240 in, a biomarker for early diagnostics of the Pseudomonas aeruginosa pathogen in urine and saliva samp

241 ing enzymes, DsbB and VKOR, are required for Pseudomonas aeruginosa pathogenicity and Mycobacterium t

242 This element, ICEKp2, is similar to the Pseudomonas aeruginosa pathogenicity island PAPI.

243 Previously, we showed that DMBT1 bound Pseudomonas aeruginosa pili, and inhibited twitching mot

244 Pseudomonas aeruginosa pneumonia elicits endothelial cel

245 lly, we developed a humanized mouse model of Pseudomonas aeruginosa pneumonia to determine if pathoge

246 f FABP4 in host defense in a murine model of Pseudomonas aeruginosa pneumonia.

247 ophil recruitment and bacterial clearance in Pseudomonas aeruginosa pneumonia.

248 The bacterial pathogen Pseudomonas aeruginosa produces toxins that disrupt oxid

249 mation by six different bacterial pathogens: Pseudomonas aeruginosa, Proteus mirabilis, Enterococcus

250 Pseudomonas aeruginosa quorum sensing (QS) regulates exp

251 n rat scald burn model, we demonstrated that Pseudomonas aeruginosa readily formed biofilms within fu

252 ne complexes from Salmonella Typhimurium and Pseudomonas aeruginosa, respectively, reveals that Eag c

253 Similarly, infection of corneas with Pseudomonas aeruginosa resulted in secondary infection o

254 al characterization of the CopG protein from Pseudomonas aeruginosa Results from biochemical analyses

255 w that co-culturing Rhizopus microsporus and Pseudomonas aeruginosa results in the inhibition of spor

256 In the opportunistic pathogen Pseudomonas aeruginosa, RsmA is an RNA-binding protein t

257 type III secreted phospholipase effector of Pseudomonas aeruginosa, serves as a prototype to model l

258 tral forms still present in Vibrio cholerae, Pseudomonas aeruginosa, Shewanella oneidensis and Methyl

259 We subjected 2 MC-deficient mouse strains to Pseudomonas aeruginosa skin wound infection and found si

260 main of the lytic transglycosylase RlpA from Pseudomonas aeruginosa (SPOR-RlpA) by mass spectrometry

261 l activity against many pathogens, including Pseudomonas aeruginosa, Staphylococcus aureus, and Esche

262 ted, body mass index (z-score), age at CPET, Pseudomonas aeruginosa status, and CF-related diabetes a

263 f a pathogenic aminoglycoside (AG)-resistant Pseudomonas aeruginosa strain, as well as of a nonresist

264 Using two Pseudomonas aeruginosa strains carrying resistance plasm

265 tized, and tested against colistin-resistant Pseudomonas aeruginosa strains including clinical isolat

266 sing planktonic and biofilm-forming cells of Pseudomonas aeruginosa strains overexpressing the MexAB-

267 Pseudomonas aeruginosa strains with loss-of-function mut

268 terial lawns including Bacillus subtilis and Pseudomonas aeruginosa strongly alter the collective dyn

269 common ocular pathogens, including HSV-1 and Pseudomonas aeruginosa, subsequent to microbial keratiti

270 Here, we show that the ClpXP protease of Pseudomonas aeruginosa suppresses its antimicrobial acti

271 We delve into Pseudomonas aeruginosa swarming, a phenomenon where bill

272 terobacterales, Acinetobacter baumannii, and Pseudomonas aeruginosa tested.

273 ng ventilator-associated pneumonia caused by Pseudomonas aeruginosa that acquired increasing levels o

274 rbapenem susceptible" at breakpoint; and (4) Pseudomonas aeruginosa that merely lack porin OprD?

275 ibit antibiotic-resistant bacteria (MRSA and Pseudomonas aeruginosa), the most common cause of biomat

276 nd effectively inhibits biofilm formation in Pseudomonas aeruginosa, the most widely used model for s

277 er, when we cocultured K279a with strains of Pseudomonas aeruginosa, the T4SS promoted the growth of

278 In Pseudomonas aeruginosa, the transition between planktoni

279 xt of the modal MIC for Enterobacterales and Pseudomonas aeruginosa, the variability of MIC tests, an

280 HE domain of chemoreceptors PctA and TlpQ of Pseudomonas aeruginosa, thus inducing chemotaxis and bio

281 reagents for ticks, we also show that adding Pseudomonas aeruginosa to drinking water quickly leads t

282 C-MS analyses, we monitored GSH oxidation in Pseudomonas aeruginosa to gauge their exposure to HOCl i

283 based model using a bioluminescent strain of Pseudomonas aeruginosa to measure loss of activity and k

284 t pathway used by the opportunistic pathogen Pseudomonas aeruginosa to respond to surface binding on

285 By fusing transcription activation domain to Pseudomonas aeruginosa type I-F Cas proteins, we activat

286 llular protein transfer, and activation of a Pseudomonas aeruginosa type VI secretion system.

287 Pseudomonas aeruginosa uses a type III secretion system

288 d by an extensively drug-resistant strain of Pseudomonas aeruginosa was detected in a hospital in Mad

289 Pseudomonas aeruginosa was the most common CRGNB each ye

290 quent underlying cause of bronchiectasis and Pseudomonas aeruginosa was the most common organism in s

291 produced in planktonic- and biofilm-cultured Pseudomonas aeruginosa We identified a core assembly of

292 Furthermore, using siderophore-deficient Pseudomonas aeruginosa, we discover that the siderophore

293 al activities against Enterobacteriaceae and Pseudomonas aeruginosa were identified.

294 Staphylococcus aureus and Pseudomonas aeruginosa were isolated in 10.5 and 6.4% of

295 as well as bacterial killing curves against Pseudomonas aeruginosa were utilized.

296 nsation of phenazines produced by pathogenic Pseudomonas aeruginosa, which leads to rapid activation

297 strikingly similar to one recently described Pseudomonas aeruginosa, whose tolerance to arsenic also

298 A common Gram-negative pathogenic bacterium Pseudomonas aeruginosa wild-type PAO1 and first-line ant

299 nerated large deletions (7-424 kilobases) in Pseudomonas aeruginosa with near-100% efficiency, while

300 erpretive categories to other organisms like Pseudomonas aeruginosa without supporting evidence.