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1 of SA, involved in response to both UV-C and Pseudomonas infection.
2 bute to the establishment and maintenance of Pseudomonas infection.
3 ion is required for the host defense against Pseudomonas infection.
4 l chemotherapeutic agents designed to combat Pseudomonas infection.
5           Weight loss was greatest 3 d after Pseudomonas infection.
6 tion designed to arrest tissue damage during Pseudomonas infection.
7 ing and local resistance to plant pathogenic Pseudomonas infection.
8 INA3 mRNA abundance decreases in response to Pseudomonas infection.
9 be partly responsible for the persistence of Pseudomonas infections.
10 e MMPs in regulating epithelial responses to Pseudomonas infection and show that a global genomics st
11  from transplant to death was facilitated by pseudomonas infection and single lung transplant.
12 ous substrates of this enzyme in response to Pseudomonas infection and UV treatment.
13 llin-resistant S. aureus, fungal infections, Pseudomonas infections, and C. difficile.
14 lymphocytic bronchiolitis, colonization with pseudomonas, infection, and BAL eosinophilia and neutrop
15                                              Pseudomonas infections are an important cause of morbidi
16 likely that IVIG would be protective against Pseudomonas infections at the dosage being used.
17 tauopathies such as Alzheimer disease, acute Pseudomonas infections cause a pathophysiological sequel
18 se-dependent respiratory burst stimulated by Pseudomonas infection contributes to host defense by mod
19              We utilized data from the Early Pseudomonas Infection Control Clinical Trial (EPIC CT),
20  data on children participating in the Early Pseudomonas Infection Control trial who received standar
21 e of an intact complement system in clearing Pseudomonas infection during phage therapy.
22 ly in macrophages also confers resistance to Pseudomonas infection, highlighting an important role fo
23 elates with reduced FLS2 protein levels upon Pseudomonas infection in a HopU1-dependent manner.
24 developing a lung segmental model of chronic Pseudomonas infection in sheep.
25                    We compared intratracheal Pseudomonas infection in wild type and caveolin-deficien
26 n atypical immune response and resistance to Pseudomonas infection independent of LORE.
27  vancomycin was 0.44 (P = 0.05), whereas for Pseudomonas infection, it was 0.96 (P = 0.95).
28  that, despite systemic signaling actuation, Pseudomonas infection leads only to local CW modificatio
29 tration of long-lived cytotoxic agents after Pseudomonas infection may establish a molecular link to
30 ]; asthma [OR, 1.08; 95% CI, 1.01-1.15]; and Pseudomonas infection [OR, 1.34; 95% CI, 1.03-1.74]), ef
31                                              Pseudomonas infection presents clinical challenges due t
32                         In a murine model of Pseudomonas infection, significantly less pulmonary infl
33 ding that NAC1 mRNA abundance increases upon Pseudomonas infection, the SINA3 mRNA abundance decrease
34          A murine pulmonary model of chronic Pseudomonas infection was used in MIF wild-type mice (mi
35 R and susceptibility to UV-C irradiation and Pseudomonas infection were assessed.
36 severity of vaccine-specific Klebsiella plus Pseudomonas infections were not significantly different
37 e SlNAC1 gene is strongly upregulated during Pseudomonas infection, while repression of the NAC1 orth