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1 ly in Arabidopsis thaliana basal immunity to Pseudomonas syringae.
2 tis cinerea, Pectobacterium carotovorum, and Pseudomonas syringae.
3 e resistance after inoculation with virulent Pseudomonas syringae.
4 1D (bzr1-1D) mutants conferred resistance to Pseudomonas syringae.
5 thaliana and its facultative plant pathogen, Pseudomonas syringae.
6 nd pathogenicity) gene regulatory network in Pseudomonas syringae.
7 am immunity to the phytopathogenic bacterium Pseudomonas syringae.
8 tase) was suppressed at high temperatures in Pseudomonas syringae.
9 to increased susceptibility to the bacterium Pseudomonas syringae.
10 r their action, as currently best studied in Pseudomonas syringae.
11 for stomatal immunity against the bacterium Pseudomonas syringae.
12 ose deposition in response to non-pathogenic Pseudomonas syringae.
13 enhanced PTI against the bacterial pathogen Pseudomonas syringae.
14 nerea and susceptibility to the hemibiotroph Pseudomonas syringae.
15 es resistance to both Pythium irregulare and Pseudomonas syringae.
16 confer resistance to the biotrophic pathogen Pseudomonas syringae.
17 HAA production, we discuss its regulation in Pseudomonas syringae.
18 using a novel hetero-regulation module from Pseudomonas syringae.
19 m1 avirulence gene in the bacterial pathogen Pseudomonas syringae.
20 a virulent strain of the bacterial pathogen Pseudomonas syringae.
21 e more susceptible to the bacterial pathogen Pseudomonas syringae.
22 the T3SS gene cluster of the plant pathogen Pseudomonas syringae.
23 basal defense against the bacterial pathogen Pseudomonas syringae.
24 of HopI1, a virulence effector of pathogenic Pseudomonas syringae.
25 plants infected with the bacterial pathogen, Pseudomonas syringae.
26 sis of 3-thiaglutamate in the plant pathogen Pseudomonas syringae.
27 hallenged with the phytopathogenic bacterium Pseudomonas syringae.
28 ired for a complete defence response against Pseudomonas syringae.
29 radation, and susceptibility to the pathogen Pseudomonas syringae.
30 ontaining femtomolar INP concentrations from Pseudomonas syringae.
31 during colonization of Phaseolus vulgaris by Pseudomonas syringae.
32 PR1, Constitutive 1 (SNC1) and Resistance to Pseudomonas syringae 2 (RPS2), for ubiquitination and fu
33 ia solanacearum 1 (RRS1-R) and Resistance to Pseudomonas syringae 4 (RPS4) function together to recog
34 of alginate epimerization, the structure of Pseudomonas syringae AlgG has been determined at 2.1-A r
35 per basal immunity to the bacterial pathogen Pseudomonas syringae Although SARD4 knockout plants show
37 phid (GPA; Myzus persicae) and the pathogens Pseudomonas syringae and Hyaloperonospora arabidopsidis.
38 isplayed compromised resistance to avirulent Pseudomonas syringae and Hyaloperonospora arabidopsidis.
39 cretion was enhanced in plants infected with Pseudomonas syringae and in response to treatment with s
40 ' vector was functional in Escherichia coli, Pseudomonas syringae and Klebsiella pneumoniae, and endo
45 ognize two bacterial effectors, AvrRps4 from Pseudomonas syringae and PopP2 from Ralstonia solanacear
46 nt resistance to the hemibiotrophic pathogen Pseudomonas syringae and the necrotrophic pathogen Botry
47 isease resistance against the hemibiotrophic Pseudomonas syringae and the necrotrophic Pectobacterium
48 responding data for the eubacterial pathogen Pseudomonas syringae and the oomycete pathogen Hyalopero
49 disease resistance to the bacterial pathogen Pseudomonas syringae and the oomycete pathogen Hyalopero
50 rs during infection with the foliar pathogen Pseudomonas syringae and the vascular pathogen Ralstonia
51 ced susceptibility to the bacterial pathogen Pseudomonas syringae and to the fungus Botrytis cinerea
52 mised nonhost resistance to few pathovars of Pseudomonas syringae and Xanthomonas campestris, but als
55 e, Xanthomonas oryzae, Erwinia chrysanthemi, Pseudomonas syringae, and Acidovorax avenae, naringenin
56 ion for environmentally ubiquitous taxa like Pseudomonas syringae, and emphasize that classification
57 important plant pathogens (Botrytis cinerea, Pseudomonas syringae, and Fusarium oxysporum) were used
58 , the hemibiotrophic bacterial phytopathogen Pseudomonas syringae, and herbivorous larvae of the moth
63 minant jaz2Deltajas mutants are resistant to Pseudomonas syringae but retain unaltered resistance aga
64 for the activity of INPs from the bacterium Pseudomonas syringae by combining a high-throughput ice
67 exhibited reduced cell death in response to Pseudomonas syringae carrying avirulent gene avrRpt2, an
70 regulation of stomata under free running and Pseudomonas syringae challenge conditions as well as def
72 a virulent strain of the bacterial pathogen Pseudomonas syringae, coincident with peak disease sympt
73 sed susceptibility to the bacterial pathogen Pseudomonas syringae, consistent with a role in inducibl
74 sed susceptibility to the bacterial pathogen Pseudomonas syringae, consistent with defense-induced li
75 oxaben, displayed enhanced susceptibility to Pseudomonas syringae DC3000 as well as reduced activatio
77 intact in eds1 mutant plants in response to Pseudomonas syringae delivering the effector protein Avr
80 Here, we show that the bacterial pathogen Pseudomonas syringae deploys an effector protein, HopO1-
83 se (HR) typical of ETI is abolished when the Pseudomonas syringae effector AvrRpt2 is bacterially del
84 penetration, in this study we expressed the Pseudomonas syringae effector HopAI known to inactivate
86 UDOMONAS SYRINGAE5 (RPS5), which detects the Pseudomonas syringae effector protein Avirulence protein
89 In Arabidopsis (Arabidopsis thaliana), the Pseudomonas syringae effector proteins AvrB and AvrRpm1
90 from Solanum pimpinellifolium interacts with Pseudomonas syringae effectors AvrPto or AvrPtoB to acti
93 tion of AvrRpt2, one of the first identified Pseudomonas syringae effectors, involves cleavage of the
94 stallographic and biochemical studies on the Pseudomonas syringae ethylene-forming enzyme reveal a br
95 n interactions using purified peptides and a Pseudomonas syringae fliC mutant complemented with diffe
97 ing susceptibility to the bacterial pathogen Pseudomonas syringae Glucose-6-phosphate dehydrogenase (
98 ains of the gram-negative bacterial pathogen Pseudomonas syringae have been used as models for unders
101 scular propagation of the bacterial pathogen Pseudomonas syringae in leaves and, accordingly, some im
103 LR recognizes diverse effector proteins from Pseudomonas syringae, including HopZ1a, and Xanthomonas
104 ted in enhanced susceptibility to pathogenic Pseudomonas syringae, indicating functional redundancy i
105 abidopsis leaves with the bacterial pathogen Pseudomonas syringae induces the expression of genes inv
106 * sfr6 mutants were more susceptible to both Pseudomonas syringae infection and UV-C irradiation.
107 expression and is necessary for tolerance of Pseudomonas syringae infection and UV-C irradiation.
109 iotic and biotic stresses such as drought or Pseudomonas syringae infection induced a similar increas
111 kout mutant displayed enhanced resistance to Pseudomonas syringae infection of immature flowers, but
113 lved in the Arabidopsis thaliana response to Pseudomonas syringae infection: a cytoplasmic localized
116 omato (Solanum lycopersicum) to infection by Pseudomonas syringae involves both detection of pathogen
119 have found that normal infection of the host Pseudomonas syringae is dependent on the action of a hos
122 ic in nature, isolates such as the Antarctic Pseudomonas syringae Lz4W exhibit considerable psychroto
123 ce suggest that the bacterial plant pathogen Pseudomonas syringae manipulates auxin physiology in Ara
124 natine (phytotoxin produced by the bacterium Pseudomonas syringae) or fusicoccin (a fungal toxin prod
125 gainst the hemibiotrophic bacterial pathogen Pseudomonas syringae oxr2 mutant plants are more suscept
127 JMJ27 is induced in response to virulent Pseudomonas syringae pathogens and is required for resis
128 red after primary leaf infection with either Pseudomonas syringae pathovar japonica (Psj) or Xanthomo
130 genes, scd1-1 plants were more resistant to Pseudomonas syringae pathovar tomato (Pst) DC3000 infect
131 type counterparts to the bacterial pathogens Pseudomonas syringae pathovar tomato and Erwinia amylovo
133 ployed T3SS substrates in the plant pathogen Pseudomonas syringae pathovar tomato strain DC3000 posse
134 d that TARK1 CRISPR plants were resistant to Pseudomonas syringae pathovar tomato strain DC3000-induc
136 ith pathogens, such as Soybean mosaic virus, Pseudomonas syringae, Phytophthora sojae, Phakopsora pac
137 fector HopZ1a produced by the plant pathogen Pseudomonas syringae possesses acetyltransferase activit
140 ato, detection by the host Pto kinase of the Pseudomonas syringae proteins AvrPto or AvrPtoB causes l
141 ing proteins (EBPs) HrpR and HrpS (HrpRS) of Pseudomonas syringae (Ps) activate sigma(54)-dependent t
142 rom the Rpg1-b, Rpg3, and Rpg4 loci, against Pseudomonas syringae (Psg) expressing avrB, avrB2 and av
144 rial causal agent of bleeding canker disease Pseudomonas syringae pv aesculi, and the bark-associated
145 bean (Glycine max) RPG1-B (for resistance to Pseudomonas syringae pv glycinea) mediates species-speci
146 ble to virulent bacterial pathogens, such as Pseudomonas syringae pv maculicola (Psm) and P. syringae
148 brassicicola and the bacterial hemibiotroph Pseudomonas syringae pv maculicola ES4326 (Pma ES4326) w
152 protein Q), a type III effector secreted by Pseudomonas syringae pv phaseolicola, is widely conserve
155 ainst several bacterial pathogens, including Pseudomonas syringae pv tomato (Pst) and the insect pest
156 secretion system-deficient bacterial strain Pseudomonas syringae pv tomato (Pst) DC3000 hrcC(-) and
157 induced by the avirulent bacterial pathogen Pseudomonas syringae pv tomato (Pst) DC3000/avrRpt2, and
158 inst a surface-deposited bacterial pathogen, Pseudomonas syringae pv tomato (Pst) DC3000; in contrast
162 are more resistant to an avirulent strain of Pseudomonas syringae pv tomato (Pst-AvrRpm1), which was
165 he Hrp outer protein Q (HopQ1) effector from Pseudomonas syringae pv tomato (Pto) strain DC3000 is co
166 pport increased bacterial growth of virulent Pseudomonas syringae pv tomato DC3000 (Pst) and Pseudomo
170 are confirmed in subsequent experiments with Pseudomonas syringae pv tomato DC3000 and Arabidopsis th
171 erived metabolites that induce T3SS genes in Pseudomonas syringae pv tomato DC3000 and report that ma
172 ry but also when leaves were inoculated with Pseudomonas syringae pv tomato DC3000 and roots with the
173 usceptibility to both the bacterial pathogen Pseudomonas syringae pv tomato DC3000 and the fungal pat
174 expression changes following challenge with Pseudomonas syringae pv tomato DC3000 and the nonpathoge
175 and an increased tolerance to the biotrophic Pseudomonas syringae pv tomato DC3000 bacterium and Beet
176 idopsis (Arabidopsis thaliana) infected with Pseudomonas syringae pv tomato DC3000 expressing AvrRpt2
178 study demonstrated that foliar infection by Pseudomonas syringae pv tomato DC3000 induced malic acid
179 avirulent strains of the bacterial pathogen Pseudomonas syringae pv tomato DC3000 results in a drast
180 e induction and enhancement of resistance to Pseudomonas syringae pv tomato DC3000 were partially red
181 Empoasca spp.), and (3) bacterial pathogens (Pseudomonas syringae pv tomato DC3000), showing that all
182 ea and Alternaria solani, bacterial pathogen Pseudomonas syringae pv tomato DC3000, and larvae of the
188 plants with the avirulent bacterial pathogen Pseudomonas syringae pv tomato DC3000/avrRpt2 induces bi
189 ection with virulent or avirulent strains of Pseudomonas syringae pv tomato generates long-distance S
190 lerated hypersensitive response triggered by Pseudomonas syringae pv tomato in soybean (Glycine max)
191 istance against the hemibiotrophic bacterium Pseudomonas syringae pv tomato, the biotrophic oomycete
200 in limiting growth of the bacterial pathogen Pseudomonas syringae pv. maculicola (Pma) ES4326 and act
201 s important for defense against the pathogen Pseudomonas syringae pv. maculicola ES4326 (Pma ES4326).
202 opsis thaliana) plants locally infected with Pseudomonas syringae pv. maculicola Whole transcriptome
204 hallenged with the cereal bacterial pathogen Pseudomonas syringae pv. oryzae, transgenic EFR wheat li
205 nst Xanthomonas citri subsp. citri (Xcc) and Pseudomonas syringae pv. phaseolicola (Psp) NPS3121.
207 rved this phenomenon with the plant pathogen Pseudomonas syringae pv. phaseolicola where isolates tha
209 study, the role of (p)ppGpp on virulence of Pseudomonas syringae pv. syringae B728a (PssB728a) was i
213 levels of an unknown surfactant produced by Pseudomonas syringae pv. syringae B728a that was not det
216 observed in silenced plants infiltrated with Pseudomonas syringae pv. tabaci expressing AvrPto or Hop
217 mimicking coronatine (COR) toxin produced by Pseudomonas syringae pv. tomato (Pst) DC3000 functions t
218 creases the susceptibility of Arabidopsis to Pseudomonas syringae pv. tomato (Pst) DC3000 independent
219 r disease response to the bacterial pathogen Pseudomonas syringae pv. tomato (Pst) DC3000, including
220 utant of Arabidopsis is hyper-susceptible to Pseudomonas syringae pv. tomato (Pst) DC3000, while Arab
223 induced by the avirulent bacterial pathogen Pseudomonas syringae pv. tomato (Pst) DC3000/avrRpt2, an
232 he ability to resist COR-producing pathogens Pseudomonas syringae pv. tomato and P. syringae pv. macu
234 h to investigate the role of siderophores in Pseudomonas syringae pv. tomato DC3000 (DC3000) virulenc
235 n filament organization after infection with Pseudomonas syringae pv. tomato DC3000 (Pst DC3000), dem
236 ld-type plants, against avirulent strains of Pseudomonas syringae pv. tomato DC3000 (Pst) carrying Av
237 RT2 displayed reduced resistance to virulent Pseudomonas syringae pv. tomato DC3000 (PstDC3000).
238 characterized the molecular function of the Pseudomonas syringae pv. tomato DC3000 (Pto) effector Ho
239 e resistance against the biotrophic bacteria Pseudomonas syringae pv. tomato DC3000 and for susceptib
240 d contributes to resistance to the bacterium Pseudomonas syringae pv. tomato DC3000 and the fungal pa
241 9-mediated PCD, as well as non-host pathogen Pseudomonas syringae pv. tomato DC3000 and the general e
242 sed resistance toward the virulent bacterium Pseudomonas syringae pv. tomato DC3000 and the necrotrop
244 tly increased upon infection with pathogenic Pseudomonas syringae pv. tomato DC3000 lacking hopQ1-1 [
246 merina BMM (PcBMM), but not to the bacterium Pseudomonas syringae pv. tomato DC3000 or to the oomycet
247 activating jasmonate signalling, for example Pseudomonas syringae pv. tomato DC3000 produces coronati
249 ve been investigating how the plant pathogen Pseudomonas syringae pv. tomato DC3000 responds to iron
251 verexpressing this gene were challenged with Pseudomonas syringae pv. tomato DC3000, which is a bacte
254 persicoides confers resistance to strains of Pseudomonas syringae pv. tomato expressing AvrRpt2 and R
256 tomato (Solanum lycopersicum), resistance to Pseudomonas syringae pv. tomato is elicited by the inter
260 double mutant showed enhanced resistance to Pseudomonas syringae pv. tomato, which is consistent wit
262 nse in tomato (Solanum lycopersicum) against Pseudomonas syringae relies on the recognition of E3 lig
265 n of Arabidopsis (Arabidopsis thaliana) with Pseudomonas syringae revealed that LPO is predominantly
269 ot sim-1, was more susceptible to a virulent Pseudomonas syringae strain, and this susceptibility cou
270 nce genes, host range, and aggressiveness of Pseudomonas syringae strains closely related to the toma
271 ease and acts in immunity against pathogenic Pseudomonas syringae strains only when they carry a term
273 ry of ice nucleation-active bacteria such as Pseudomonas syringae supports that they have been part o
276 etyltransferase carried by the phytopathogen Pseudomonas syringae that elicits effector-triggered imm
277 s a toxin produced by the bacterial pathogen Pseudomonas syringae that is known to counteract Arabido
278 or protein from the bacterial plant pathogen Pseudomonas syringae that suppresses plant immunity by i
279 ra arabidopsidis, and the bacterial pathogen Pseudomonas syringae (the latter both in terms of basal
280 creased resistance to the bacterial pathogen Pseudomonas syringae These results suggest that ANT and
281 ent, avirulent and non-pathogenic strains of Pseudomonas syringae, thus limiting the defense function
282 ipase D beta1 (PLDbeta1)-deficient plants by Pseudomonas syringae tomato pv DC3000 (Pst DC30000) resu
283 dmr6 mutants show loss of susceptibility to Pseudomonas syringae, transgenic dmr6 plants expressing
284 hat high humidity can effectively compromise Pseudomonas syringae-triggered stomatal closure in both
285 that SA promotes the interaction between the Pseudomonas syringae type III effector AvrPtoB and NPR1.
291 no increased susceptibility to the pathogen Pseudomonas syringae, unlike gh3.12 mutants, which were
293 coronatine (COR) promotes various aspects of Pseudomonas syringae virulence, including invasion throu
294 plants and the nonpathogenic hrpA mutant of Pseudomonas syringae was able to grow rapidly in the mut
295 It was further revealed the EmhR ortholog in Pseudomonas syringae was also responsible for indole-ind
296 y data of Arabidopsis thaliana infected with Pseudomonas syringae, we analyzed Arabidopsis defense re
297 bacterial bioreporters of the phytopathogen Pseudomonas syringae were constructed that couple a QAC-
298 gainst foliar pathogens Botrytis cinerea and Pseudomonas syringae, which normally result from interac
299 wed increased susceptibility to the pathogen Pseudomonas syringae, with the double mutant showing a s