1 n EP mechanism of the periodic transition in QRS axis.

2 nduction disorder, arrhythmias, or change in QRS axis.

3 RS morphology was accompanied by a change in QRS axis.

4      In 22 of 26 episodes, the transition in QRS axis coincided with the transient bifurcation of a p

5 a prolonged QTc interval, a Q wave, abnormal QRS axis deviation, ST segment depression and a patholog

6 he characteristic periodic transition of the QRS axis during TdP VT in the LQTS.

7 th inverted asymmetrical T-wave opposite the QRS axis in lead V5 or V6.

8 inverted asymmetrical T-wave opposite to the QRS axis in leads V5 and/or V6.

9 variate analysis demonstrated differences in QRS axis, limb (I, aVr), and precordial (V1, V2, V6) ECG

10 te precordial transition (TZ) and discordant QRS axis may not be solely explained by anisotropic cond

11 with a left bundle branch block and inferior QRS axis morphology.

12 atients with idiopathic VAs with an inferior QRS-axis morphology.

13 ve electrocardiographic traits (PR interval, QRS axis, QRS duration, and QTc interval) were evaluated

14                                  A change in QRS axis simultaneous with electrocardiographic waveform

15 ion, PR interval, QRS duration, QT interval, QRS axis, Sokolow-Lyon and Cornell voltages, and ST-segm

16  maps could be accurately constructed during QRS-axis transitions in surface ECGs.

17 st polymorphic VT, with an apparent shift in QRS axis, was due to a predominantly single localized ci

18 Z) TZ or inconsistent (IQA) or consistent QA QRS axis were correlated with PMCs.