ライフサイエンスコーパス: QRS complex

1 g responders (including patients with narrow QRS complexes).

2 outcomes in heart failure patients with wide QRS complex.

3 , characterized by additional notches in the QRS complex.

4 f the first two orthogonal components of the QRS complex.

5 on of different orthogonal components of the QRS complex.

6 d left ventricular (LV) function, and a wide QRS complex.

7 n chronic systolic heart failure with a wide QRS complex.

8 oms, a reduced ejection fraction, and a wide QRS complex.

9 tients with a low ejection fraction and wide QRS complex.

10 the cardiac mortality compared with a narrow QRS complex.

11 ll-cause mortality rate compared with narrow QRS complex.

12 al) from each of nine unknown samples of the QRS complex.

13 ctor of incident CHD events in men with wide QRS complex.

14 ine, having either normal conduction or wide QRS complex.

15 ctuates cyclically back and forth across the QRS complex.

16 n a fairly constant position relative to the QRS complex.

17 n may depolarize tissue after the end of the QRS complex.

18 n negative deflection until greater than the QRS complex.

19 lectrocardiogram in the presence of a narrow QRS complex.

20 nt with (in the opposite direction from) the QRS complex.

21 sk features including wide and low-amplitude QRS complexes.

22 ation pattern was computed from the recorded QRS complexes.

23 mice showed normal baseline heart rates and QRS complexes.

24 Association class 3.4 +/- 0.5) and a widened QRS complex (184 +/- 31 ms) underwent robotic LV lead pl

25 Of the 10 impacts sustained during the QRS complex, 4 resulted in transient complete heart bloc

26 epressed ejection fraction (EF) and a narrow QRS complex, albeit in a small number of patients, and w

27 cles of mutant animals, including diminished QRS complex amplitude consistent with loss of electrical

28 n patients with heart failure (HF) with wide QRS complex and diminished left ventricular (LV) functio

29 were delivered at or before the onset of the QRS complex and never during the complex itself.

30 s, to detect the abnormality in PR interval, QRS complex and QT interval the Coefficient Variation (C

31 ed elevation in the ST-segment and increased QRS complex and QTc duration.

32 ature contractions with an elongation of the QRS complex and the hearts were more susceptible to isop

33 e., the spatial electrical angle between the QRS complex and the T-wave; p = 0.0005), wider QRS compl

34 nds (ms) after the end of the last conducted QRS complex and then scanned decrementally through that

35 G628S mice studied, primarily involving the QRS complex and, more rarely, T-wave morphology.

36 ndex, had elevated heart rate, had prolonged QRS complex, and had lower prevalence of history of prio

37 n a non-LBBB (left bundle branch block) wide QRS complex, and lower left ventricular ejection fractio

38 with a reduced ejection fraction, a widened QRS complex, and NYHA class II or III heart failure, the

39 CG localized features including PR interval, QRS complex, and QT interval from the continuous ECG wav

40 ssification of abnormalities in PR interval, QRS complex, and QT interval.

41 for the entire signal, 0.67 (0.59-0.76) for QRS complexes, and 0.57 (0.35-0.76) for T waves.

42 lethal cardiomyopathy associated with a wide QRS complex arrhythmia.

43 tion functional class I and II and with wide QRS complexes, carvedilol was associated with a 30% redu

44 ctionated late potentials (96+/-47 ms beyond QRS complex) clustering exclusively in the anterior aspe

45 The last QRS complex coincided with the last arterial pulse in 19

46 lure, left ventricular dysfunction, and wide QRS complex compared with an ICD only.

47 The duration of the basal QRS complex does not reliably predict the clinical respo

48 D), high RVP burden >= 20%, and a wide paced QRS complex duration >= 150 ms were randomly assigned to

49 llators (CRT-D) have a very wide (>/=180 ms) QRS complex duration (QRSD).

50 a left ventricular ejection fraction <50% or QRS complex duration 120 ms, or both.

51 The normal physiologic range of QRS complex duration spans between 80 and 125 ms with kn

52 a premature ventricular depolarization or a QRS complex during ventricular tachycardia.

53 hat influenced model predictions (precordial QRS complexes for all outcomes; T waves for LV dysfuncti

54 Fragmented QRS complexes (fQRSs), which include various RSR' patter

55 The sums of the amplitude of QRS complexes from the 12 ECG leads (Sigma QRS) were cor

56 The presence of notching on any QRS complex had 79% sensitivity and 65% specificity of (

57 he electrophysiological underpinnings of the QRS complex has become important not only to predict res

58 Some patients with narrow QRS complexes have echocardiographic evidence of left ve

59 nts for association with ten measures of the QRS complex in 12 leads, using 405,732 electrocardiogram

60 he onset of or within the first 45 ms of the QRS complex in 16 animals.

61 ams revealed significant prolongation of the QRS complex in adult Cx43 -/+ mice (13.4+/-1.8 ms, n = 1

62 te the advantage of in-depth analysis of the QRS complex in conjunction with other cardiovascular phe

63 elatively fixed CI from the preceding normal QRS complex in most patients.

64 ventricular (LV) systolic function and broad QRS complex in the surface electrocardiogram (ECG).

65 cessing method projects all ECG leads of the QRS complex into optimized three perpendicular dimension

66 However, low-voltage QRS complex is not a uniform finding with the infiltrati

67 Whenever a QRS complex is wide and abnormal, such as during ventric

68 sed by delayed ventricular contraction (wide QRS complex), is a common feature of cardiomyopathy and

69 ittal planes consistently represented the P, QRS complex, J point, and T waves.

70 The morphology of single paced QRS complexes may vary, depending on coupling interval,

71 In ICD patients with HF, a wide underlying QRS complex more than doubles the cardiac mortality comp

72 In the next 10 patients, the QRS complex morphology during ventricular overdrive paci

73 All patients with baseline normal QRS complexes (n=30) or left fascicular blocks (4 anteri

74 ct of ADS synchronized to normally conducted QRS complexes (NQRS) and to supraventricular complexes w

75 ction" is the sharp upward deflection in the QRS complex of an electrocardiogram.

76 Features of the QRS complex of the electrocardiogram, reflecting ventric

77 rface ECGs revealed that late notches in the QRS complexes of lateral leads were associated with CCB

78 ed by abnormal amplitude and duration of the QRS complex on the ECG.

79 flected by the amplitude and duration of the QRS complex on the electrocardiogram (ECG).

80 The morphology of the QRS complexes on electrocardiogram is an excellent tool

81 t were significantly delayed with respect to QRS complex onset (3.7+/-0.7 ms in WT [n=6] and 6.5+/-0.

82 QRS complex onset to activation at the right ventricular

83 It is usually coincident with the QRS complex or appears on the ST segment or first half o

84 ed by difficulty in assessing: 1) changes in QRS complexes or P-waves that indicate fusion, and 2) th

85 ifocal ectopic ventricular beats with narrow QRS complexes, originating from various ectopic foci alo

86 ignificant with the duration of the filtered QRS complex (p < 0.001 for QRS duration, p < 0.01 for la

87 S complex and the T-wave; p = 0.0005), wider QRS complex (p = 0.004), longer QTrr (i.e., age- and gen

88 bserved an 80% reduction in amplitude of the QRS complex, profound systolic dysfunction, decreased co

89 is-unrelated bradycardia and PQ interval and QRS complex prolongation.

90 In individuals with wide QRS complex (QRSd > or =120 ms), similar analyses showed

91 on, salutary effects in patients with narrow QRS complexes remain to be demonstrated.

92 First, a novel robust QRS complex segmentation strategy is proposed, which com

93 resulted in a consistent prolongation of the QRS complex, showing up to 40% increase from baseline, e

94 2 groups were detected for the PR interval, QRS complex, ST-segment duration, T-wave duration, QTc,

95 ncordant with (in the same direction as) the QRS complex; ST-segment depression of 1 mm or more in le

96 tive of the atrioventricular block), widened QRS complexes (suggesting intraventricular block), and e

97 evidence of sinus bradycardia and fragmented QRS complex, supporting the critical role of Slc26a6 in

98 The ADFT(50) for QRS complex-synchronized shocks was 183+/-56 V, versus 1

99 The etiology of sustained monomorphic wide QRS complex tachycardia is often uncertain acutely.

100 apeutic agent for patients with regular wide QRS complex tachycardia.

101 gent for patients with undifferentiated wide QRS complex tachycardia.

102 ing in a type of VT with a relatively narrow QRS complex that mimics fascicular VT.

103 ntervals, PAWP was measured gated to the ECG QRS complex to calculate the QRS-gated DPD (diastolic pu

104 the electric delay from the beginning of the QRS complex to the local LV electrogram (QLV), was found

105 t of variation of the time interval from the QRS complex to the onset of expansion and to early diast

106 time" is the interval from the start of the QRS complex to the peak of the R wave.

107 during normal sinus rhythm that reflects the QRS complex vector during prior periods of ventricular p

108 During pace mapping, the stimulus-QRS complex was longer at late potential sites, consiste

109 Low-voltage QRS complex was the sine qua non of infiltrative cardiom

110 In the overall cohort, precordial lead QRS complexes were most salient with high-risk features

111 Very late LAVA (>100 ms after QRS complex) were almost exclusively detected within the

112 chrony also occurs in patients with a narrow QRS complex, which suggests the potential usefulness of

113 ported were transient arrhythmia, reversible QRS-complex widening, transient hypotension and mild non

114 The isoelectric initial preexcited QRS complex with rSR' pattern in lead V1 of the surface

115 Fragmented QRS complex with visible notching on standard 12-lead el

116 VCD did not differ from patients with narrow QRS complexes with regard to occurrence of tachycardias.

117 class II-IV heart failure symptoms, and wide QRS complex, with established chemotherapy-induced cardi