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1 ROP also predicted the site of branch formation in the a
2 ROP was induced in 21 rats then two concentrations of 2-
3 preplus/plus disease (OR = 2.7, P = 0.003), ROP stage (OR = 4.2 for stage 3 ROP vs. no ROP, P = 0.00
5 cted 452 of 459 infants who developed type 1 ROP (sensitivity, 98.5%; 95% CI, 96.9%-99.3%), reducing
6 n with ROP requiring treatment (i.e., type 1 ROP + advanced ROP) were born at an MGA of 30w4d (95% CI
7 received IVB as primary treatment for type 1 ROP and compare them to findings in patients with ROP th
9 was present at that examination, all type 1 ROP cases would be captured, and the number of examinati
10 ntial role of anti-VEGF treatment for type 1 ROP has become a focus in recent years, but the protract
14 rved more frequently in children with type 1 ROP, appears essential for implementing timely treatment
15 ean outcomes were the sensitivity for type 1 ROP, reductions in infants requiring imaging or examinat
18 AGING (trained reader grading of type 1 or 2 ROP initiates diagnostic examinations), and TARP (CHOP-R
19 osing AP-ROP (94% vs. 78%; P < 0.01), type 2 ROP or worse (92% vs. 84%; P = 0.04), and ROP requiring
20 out ROP, 22.0% had mild ROP, 5.3% had type 2 ROP, 27.3% had type 1 ROP, and 1.5% had advanced disease
22 P = 0.003), ROP stage (OR = 4.2 for stage 3 ROP vs. no ROP, P = 0.006), and blot hemorrhage (OR = 3.
23 es with referral-warranted (RW) ROP (stage 3 ROP, zone I ROP, plus disease) on diagnostic examination
24 separating 56 healthy controls (HC) from 35 ROP patients using rsFC (balanced accuracy of 65.5%, P <
28 ; synonym: INTERACTOR OF CONSTITUTIVE ACTIVE ROP b) directly interacts with RACB in yeast and in plan
31 iring treatment (i.e., type 1 ROP + advanced ROP) were born at an MGA of 30w4d (95% CI, +/- 5d; range
36 2 ROP or worse (92% vs. 84%; P = 0.04), and ROP requiring treatment (89% vs. 79%; P < 0.01) was bett
37 h lobar and global cortical surface area and ROP was significantly negatively correlated with lobar a
38 ide a founding framework revealing auxin and ROP signaling of inner polar nuclear position with some
48 Data extracted from charts included baseline ROP information, visual acuity and other examination fin
52 hiladelphia Retinopathy of Prematurity (CHOP ROP) model uses birth weight (BW), gestational age at bi
56 ic examinations by an ophthalmologist), CHOP-ROP (birth weight and gestational age, with weekly weigh
57 hiladelphia Retinopathy of Prematurity (CHOP-ROP) postnatal weight gain predictive model are 2 approa
58 tes diagnostic examinations), and TARP (CHOP-ROP alarm initiates imaging, and imaging finding of seve
62 r-eye agreements were found when considering ROP features at the first image session, at the last ima
63 Eight studies, 6 including laser-controlled ROP infants and 2 including ROP infants not requiring tr
64 preset subgroups comprising laser-controlled ROP infants and ROP infants not requiring treatment.
67 ociated with an increased risk of developing ROP among an unrestricted cohort but with a reduced risk
69 ng Acute-Phase Retinopathy of Prematurity (e-ROP) Study telemedicine system of remote fundus image gr
70 ng Acute-Phase Retinopathy of Prematurity (e-ROP) study was conducted from May 1, 2011, to October 31
72 ons when the risk cut point is surpassed), e-ROP IMAGING (trained reader grading of type 1 or 2 ROP i
73 was a post hoc analysis of a cohort in the e-ROP Study (a multicenter prospective telemedicine study)
83 sed to analyze the intragrader agreement for ROP diagnosis by the ophthalmologists-in-training during
84 luorescein angiography after bevacizumab for ROP reveals abnormal vascular patterns in all eyes and N
86 mber of 115 infants who met the criteria for ROP screening in three neonatal intensive care units wer
88 an readers graded each eye independently for ROP features in a 5 retinal-image set from each session.
97 tal Growth and Retinopathy of Prematurity (G-ROP) Study (a multicenter retrospective cohort study).
99 owth and Retinopathy of Prematurity Study (G-ROP) 1 study (2006-2012) and the prospective G-ROP 2 stu
101 atus, birth weight, gestational age, gender, ROP treatment method, postmenstrual age at treatment, an
103 y of prematurity (ROP), 38% of eyes (84) had ROP not deemed to require treatment in the neonatal peri
106 rral-warranted (RW) ROP (stage 3 ROP, zone I ROP, plus disease) on diagnostic examination from the Te
107 s' postmenstrual age and followed up only if ROP was present at that examination, all type 1 ROP case
109 d readers showed good inter-eye agreement in ROP characteristics, consistent with the high inter-eye
110 sistent with the high inter-eye agreement in ROP from clinical examinations by ophthalmologists in ot
118 laser-controlled ROP infants and 2 including ROP infants not requiring treatment, were included.
119 a birth weight less than 1251 g and a known ROP outcome enrolled between May 25, 2011, and October 3
120 nts undergoing ROP examinations with a known ROP outcome who were born between January 1, 2006, and D
122 . 29% without opacities; P = 0.003), maximum ROP stage (P = 0.001), preplus or plus disease (24% vs.
124 immature retina without ROP, 22.0% had mild ROP, 5.3% had type 2 ROP, 27.3% had type 1 ROP, and 1.5%
125 l might be used reliably to guide a modified ROP screening schedule and decrease the number of examin
126 ariate analysis adjusted by BW and GA, nasal ROP border distance was a significant predictor of the s
129 intensive care units follow Mexican National ROP guidelines, there have been few reports regarding th
131 nfants had a 4-5 x increased risk of needing ROP treatment (p < 0.001) compared to non-IUGR infants.
134 , ROP stage (OR = 4.2 for stage 3 ROP vs. no ROP, P = 0.006), and blot hemorrhage (OR = 3.1, P = 0.00
136 t improvements (P < 0.01) in the accuracy of ROP diagnosis for plus disease, zone, stage, category, a
137 f plus disease, zone, stage, and category of ROP after completion of the educational intervention.
139 nt risk factors affecting the development of ROP in our study were: low gestational age, low birth we
140 t predictor of the subsequent development of ROP that was treated (odds ratio, 0.86 for every 10-pixe
146 east once was associated with a diagnosis of ROP (62% vs. 29% without opacities; P = 0.003), maximum
149 ders on premature patients with a history of ROP and no treatment during infancy who demonstrated lat
151 r study is aimed at finding the incidence of ROP and its association with some risk factors in Palest
160 that investigated the incidence and onset of ROP in a representative sample of children in Mexico.
162 triction (IUGR) as independent predictors of ROP, we performed a retrospective cohort study of patien
164 required lensectomy owing to progression of ROP and/or presence of lens opacity, then the hazard of
167 restricted cohort but with a reduced risk of ROP among a restricted subcohort of P-VLBW infants.
168 ciation of maternal preeclampsia and risk of ROP among infants in an unrestricted birth cohort and a
169 rences, the association of a reduced risk of ROP among the P-VLBW subcohort also may reflect biases f
174 ty to screen remote areas with a shortage of ROP providers, thereby reducing the burden of disease.
175 re more likely to be older at worst stage of ROP (p < 0.0001) and to develop postnatal growth failure
176 ts were more likely to have a worse stage of ROP and treatment-requiring ROP (both p < 0.0001) compar
177 tational age at birth, birthweight, stage of ROP at presentation, prior treatment (laser or cryothera
178 en paired eyes was 75.3% (0.65) for stage of ROP, 82.3% (0.68) for zone of ROP, 78.7% (0.51) for plus
180 images in 4 neonates with various stages of ROP that were obtained using a prototype handheld device
181 [range, 34-43 weeks]) with various stages of ROP: 3 in the neonatal intensive care unit and 1 in the
182 eria were as follows: comparative studies of ROP patients that (1) included IVB as a treatment arm, (
183 e primary concern regarding IVB treatment of ROP is the potential systemic side effects, especially t
187 ) for stage of ROP, 82.3% (0.68) for zone of ROP, 78.7% (0.51) for plus disease, 84.7% (0.56) for RW-
190 undamental insights into the organocatalytic ROP of these specific six-membered asymmetric cyclic glu
192 l ((*)OH) to treat reverse osmosis permeate (ROP) in potable reuse treatment trains is inefficient, u
195 that MPK1 interacts with and phosphorylates ROP BINDING PROTEIN KINASE 1 (RBK1), a protein kinase th
197 CMI1) as an interactor of the Rho of plants (ROP) effector interactor of constitutively active ROP (I
201 si-zwitterionic ring-opening polymerization (ROP) of an annulated isosorbide derivative (1,4:2,5:3,6-
202 witched between ring-opening polymerization (ROP) of BBL and CHO/CO2 copolymerization by the presence
203 e first aqueous ring-opening polymerization (ROP) of N-carboxyanhydrides (NCAs) using alpha-amino-pol
204 a tin-mediated ring-opening polymerization (ROP), generated their respective polyesters (PE) or poly
205 base-catalyzed ring-opening polymerizations (ROP) of six-membered cyclic d-glucose-based carbonates w
208 with zone I disease or aggressive posterior ROP), the disadvantages are that the ROP recurrence rate
210 plasia (BPD) and retinopathy of prematurity (ROP) are two debilitating disorders that develop in pret
212 dictors of worse retinopathy of prematurity (ROP) but the role of prenatal growth patterns in ROP rem
214 fellow eyes for retinopathy of prematurity (ROP) features (stage, zone and plus disease) and severit
222 ed with neonatal retinopathy of prematurity (ROP), 38% of eyes (84) had ROP not deemed to require tre
223 Cryotherapy for Retinopathy of Prematurity (ROP), 4099 infants weighing less than 1251 g at birth un
224 tients with PDR, retinopathy of prematurity (ROP), and wet age-related macular degeneration (wet AMD)
225 to treat type 1 retinopathy of prematurity (ROP), but there remain concerns about systemic toxicity.
226 (OIR) resembling retinopathy of prematurity (ROP), loss of Casp-8 in ECs was beneficial, as pathologi
228 for treatment of retinopathy of prematurity (ROP), there are few data on their ocular efficacy, the a
229 plasia (BPD) and retinopathy of prematurity (ROP), with cortical maturational changes in VPT infants.
237 ective cohort study of patients who received ROP screening examinations at a level IV neonatal intens
241 covariate adjustment were applied to relate ROP to preeclampsia among the full cohort and in a subco
242 depolymerization of the cytoskeleton removed ROP from the membrane only in recently divided cells, po
243 a worse stage of ROP and treatment-requiring ROP (both p < 0.0001) compared to non-IUGR infants.
247 nalysis of eyes with referral-warranted (RW) ROP (stage 3 ROP, zone I ROP, plus disease) on diagnosti
248 y eyes (153 [80.1%]) interpreted as being RW-ROP positive on imaging evaluation agreed with examinati
249 stational age, 24.8 [1.4] weeks) detected RW-ROP earlier than diagnostic examination (early) in 191 (
250 42.7%) eyes by about 15 days and detected RW-ROP in 123 infants (mean [SD] gestational age, 24.6 [1.5
254 at was lower than previously used for severe ROP, was effective in this study, and could be tested in
257 ter photobleaching studies demonstrated that ROP dynamics do not depend on the cytoskeleton, acute de
258 ble to form gametophores, demonstrating that ROP is essentially for spatial patterning at the cellula
261 he control group, trainees who completed the ROP tele-education system performed better on the postte
263 del was out-of-sample cross-validated in the ROP patients from the CCT trial to assess associations b
264 arily, the attentional gains occurred in the ROP patients who showed impaired SP at baseline only if
268 sterior ROP), the disadvantages are that the ROP recurrence rate is higher, and vigilant and extended
269 es focused on ablative therapy for threshold ROP, earlier treatment for type 1 or pre-threshold disea
271 Black infants appeared less susceptible to ROP, of all severity categories, than nonblack infants.
273 G-ROP Study enrolled all infants undergoing ROP examinations with a known ROP outcome who were born
274 Fundus photographs of all infants undergoing ROP screening examinations between July 1, 2011, and Dec
279 (2) those that developed referral-warranted ROP but did not receive treatment, and (3) those that re
280 ent, 36 (17.1%) developed referral-warranted ROP not requiring treatment, and 28 (13.3%) received tre
281 7 (69.7%) did not develop referral-warranted ROP or receive treatment, 36 (17.1%) developed referral-
282 hose that never developed referral-warranted ROP or received treatment, (2) those that developed refe
283 proliferation in adolescents and adults with ROP can occur rarely with previously regressed ROP.
289 detected in children formerly diagnosed with ROP; a similar sustained choroidal thinning is observed
291 ts were compared among 3 groups of eyes with ROP: (1) those that never developed referral-warranted R
292 cademic medical center among 4 neonates with ROP in the neonatal intensive care unit and in the opera
293 e-field OCT and OCTA images in neonates with ROP using a prototype handheld OCT and OCTA device.
295 r patterns identified by FA in patients with ROP result from the disease process itself rather than a
297 tcomes compared with premature patients with ROP without requirement of treatment (group 1), although
298 Among 138 patients older than 10 years with ROP seen at our tertiary referral center from 2000 throu
299 s follows: 36.4% had immature retina without ROP, 22.0% had mild ROP, 5.3% had type 2 ROP, 27.3% had