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1                                              Rasmussen and Svensson correctly point out that there is
2                                              Rasmussen's encephalitis (RE) is a rare disease of the c
3                                              Rasmussen's encephalitis (RE), a childhood disease chara
4                                              Rasmussen's encephalitis is a childhood disease resultin
5                                              Rasmussen's encephalitis is a rare chronic neurological
6                                              Rasmussen's encephalitis is a rare progressive pediatric
7                                              Rasmussen's syndrome is a progressive and intractable fo
8                  Seventy-six subjects with a Rasmussen Disease Score (RDS) of 6 or higher were random
9 gton's diseases, schizophrenia, epilepsy and Rasmussen's encephalitis to stroke.
10  to pathological states such as epilepsy and Rasmussen's encephalitis.
11 s-coalescence evolutionary model (DLCoal) by Rasmussen and Kellis.
12 Our method is based on a method presented by Rasmussen et al. to test whether an archaic genome is fr
13 duced resin cleavage [Pawlas, J.; Andre, C.; Rasmussen, J.
14 covered a 50-cm-thick peat deposit near Cape Rasmussen (65.2 degrees S), in the maritime Antarctic.
15         In this issue of Developmental Cell, Rasmussen et al. investigate the morphogenesis of the Ca
16 utoimmune encephalitis, lupus erythematosus, Rasmussen's encephalitis, and haemophagocytic lymphohist
17 nsult) and progressive in six children (four Rasmussen encephalitis, two Sturge-Weber syndrome).
18          In this issue of the Journal, Gorst-Rasmussen et al. discuss one such approach.
19  hand contralateral to surgery when they had Rasmussen encephalitis (0/61, p = 0.001) or Sturge-Weber
20 uctural alterations were also found in human Rasmussen encephalitis, a CTL-mediated human autoimmune
21 roglia, suggesting a window for treatment if Rasmussen's encephalitis can be diagnosed early.
22 s seen with MRI might be a good biomarker in Rasmussen's encephalitis.
23 onal glutamate receptor subunit 3 (GluR3) in Rasmussen's encephalitis, a severe form of pediatric epi
24                 Astrocyte death may occur in Rasmussen's encephalitis, and destruction of this cell t
25 slow rather than halt disease progression in Rasmussen's encephalitis, without changing the eventual
26 emonstrate that the local immune response in Rasmussen's syndrome includes restricted T cell populati
27                   Vascular sequelae included Rasmussen aneurysms (4%), enlarged bronchial arteries (3
28 nthesis by aqueous SPPS (ASPPS) [Pawlas, J.; Rasmussen, J.
29                In this issue of the Journal, Rasmussen-Torvik et al. have highlighted this problem by
30 s have been implicated in the development of Rasmussen's encephalitis, a rare neurodegenerative disea
31 tors is implicated in the pathophysiology of Rasmussen's encephalitis.
32 in part determine the nature and severity of Rasmussen's encephalitis and other complement-dependent
33 ies support the sequence comparison study of Rasmussen et al., in which there is a catalytic metal-bi
34 al left hemispherectomy for the treatment of Rasmussen's encephalitis.
35 ter left hemidecorticectomy for treatment of Rasmussen's syndrome.
36                      Lindholt JS, Sogaard R, Rasmussen LM, et al.
37       GluR3 autoantibodies are found in some Rasmussen's encephalitis patients, and GluR3 antibodies
38 mmunological studies support the notion that Rasmussen's encephalitis is probably driven by a T-cell
39                        Shibani Pati and Todd Rasmussen summarize progress in preclinical research on
40                  Causes of haemoptysis were: Rasmussen aneurysms (n = 12/24), costocervical trunk pse
41 appearing neocortex obtained from cases with Rasmussen encephalitis, cystic-gliotic encephalopathy, a