ライフサイエンスコーパス: Reston

1 Texas, and the Philippines in 1996 (subtype Reston).

2 ines and Reston, Virginia, isolates [subtype Reston]).

3 es, Sudan and Cote d'Ivoire, as well as from Reston, a strain thought to be nonpathogenic in humans,

4 With the exception of Reston and Bombali viruses, the marburgviruses and ebola

5 With the exception of Reston and Lloviu viruses, filoviruses (marburgviruses,

6 athepsin B, while the ebolaviruses Sudan and Reston and Marburg virus are not.

7 inding of the IIDs of two other filoviruses, Reston and Marburg.

8 e differences in human pathogenicity between Reston and the other four ebolavirus species.

9 rus activity against Bundibugyo, Tai Forest, Reston, and Mengla viruses and is the first submicromola

10 IgM capture assay detected anti-EBO (subtype Reston) antibodies in the sera of 5 of 5 experimentally

11 f a human who was infected with EBO (subtype Reston) during a postmortem examination of an infected m

12 mong the filoviruses characterized thus far, Reston Ebola virus (REBOV) is the only Ebola virus that

13 interaction, Zaire, mouse-adapted Zaire, and Reston Ebola virus VP24s displayed similar capacities to

14 The exception is the species Reston Ebola virus, which has not been associated with h

15 roteins from Zaire, mouse-adapted Zaire, and Reston Ebola viruses for interaction with all six member

16 brary against Zaire, Sudan, Ivory Coast, and Reston Ebola viruses.

17 The emergence of Reston ebolavirus (REBOV) in domestic swine in the Phili

18 ells infected with Zaire Ebolavirus (ZEBOV), Reston Ebolavirus (REBOV), and Marburgvirus (MARV), usin

19 e syndrome, have now been discovered to host Reston ebolavirus (REBOV).

20 us (EBOV), Bundibugyo ebolavirus (BDBV), and Reston ebolavirus (RESTV) belong to the same genus but e

21 raced back only within the last 50 years for Reston ebolavirus and Zaire ebolavirus species and sugge

22 re, but more slowly progressive illness; and Reston Ebolavirus caused mild disease that was late in o

23 divided into five distinct species, of which Reston ebolavirus is uniquely nonpathogenic to humans.

24 al structures of the dsRNA-binding domain of Reston ebolavirus VP35.

25 GP from Reston ebolavirus, a nonpathogenic species in humans, sh

26 Marburg marburgvirus, Tai Forest ebolavirus, Reston ebolavirus, Sudan ebolavirus, Zaire ebolavirus, a

27 0(-4) nucleotide substitutions/site/year for Reston ebolavirus.

28 are known: Zaire, Sudan, Cote d'Ivoire, and Reston ebolavirus.

29 la virus (subsequently identified as subtype Reston) infection among cynomolgus monkeys imported from

30 In April 1996, Ebola virus (subtype Reston) infection was identified in another group of cyn

31 equirements, put in place after the original Reston outbreak in 1989-1990, were effective for detecti

32 emented since the first Ebola virus (subtype Reston) outbreak appeared to work well.

33 thogenic in humans, apes, and monkeys, Ebola Reston (REBOV) is pathogenic only in monkeys.

34 Brassica napus (cultivar Reston) seed proteins were analyzed at 2, 3, 4, 5, and 6

35 GP derived from the Reston strain of virus, which causes disease in nonhuman

36 et of recombinant surface glycoproteins from Reston, Tai Forest, Bundibugyo, Zaire, Sudan, and Marbur

37 confocal microscope (Tandem Scanning Corp., Reston, VA).

38 [subtype Zaire] and the 1989 Philippines and Reston, Virginia, isolates [subtype Reston]).

39 ed conference on this topic that occurred in Reston, Virginia.

40 Ebola virus (EBOV) and Reston virus (RESTV) are members of the Ebolavirus genus

41 Both Ebola virus (EBOV) and Reston virus (RESTV) cause disease in nonhuman primates,

42 ns, ranging from asymptomatic infections for Reston virus (RESTV) to severe disease with fatal outcom

43 Reston virus (RESTV), an ebolavirus, causes clinical dis

44 One of these emerging viruses, Reston virus (RESTV), is an enigma among the Orthoebolav

45 bolavirus genus, Bundibugyo virus (BDBV) and Reston virus (RESTV).

46 ndibugyo virus (BDBV), Tai Forest virus, and Reston virus (RESTV).

47 Herein we show that Ebola Reston virus glycoprotein (ResGP) is efficiently incorpo

48 rus, respectively and (iv) antibodies to the Reston virus GP for which no antibodies have been report

49 been proposed to be critical for the lack of Reston virus human pathogenicity because they alter the

50 novel hosts, and only a few SDPs distinguish Reston virus VP24 from VP24 of other Ebolaviruses, human

51 c of an ebolavirus nonpathogenic for humans (Reston virus) and that of an ebolavirus with lower letha

52 n virus, Tai Forest virus, Bundibugyo virus, Reston virus, and Marburg virus and differentiated betwe

53 Since Reston virus, the only non-human pathogenic Ebolavirus s

54 the Bombali virus amino acids match those of Reston virus.

55 stinguish human pathogenic Ebolaviruses from Reston virus.

56 thogenic Ebolaviruses and the non-pathogenic Reston virus.

57 a fascicularis) infected with Ebola (subtype Reston) virus in a US-registered quarantine facility.

58 Reston viruses are the only Ebolaviruses that are not pa

59 This is of concern since Reston viruses circulate in domestic pigs and can infect

60 ll nine Ebolavirus proteins that distinguish Reston viruses from the four human pathogenic Ebolavirus

61 VP24 of other Ebolaviruses, human pathogenic Reston viruses may emerge.

62 ly neutralizes Ebola, Sudan, Bundibugyo, and Reston viruses.

63 t react to the Ebola, Sudan, Bundibugyo, and Reston viruses.

64 ebolavirus species, Ebola (EBOV), Sudan, and Reston viruses.

65 filoviruses: Ebola, Marburg, Tai Forest and Reston viruses.