ライフサイエンスコーパス: Ringer

1 Ringer injections had no effects.

2 Ringer is expressed in sensory neurons before and after

3 Ringer made hypertonic by the addition of 2.5 M sucrose

4 Ringer maintains microtubule stability/dynamics with the

5 Ringer solution and 214 microM VIP(10-28) were each perf

6 Ringer solution containing enzyme was injected into the

7 Ringer solution was infused into the knee joint cavity o

8 Ringer's solution composition changes on the retina-faci

9 ng and 10 older subjects for infusion of (1) Ringer solution (control), (2) 0.5 mm L-tyrosine, (3) 5

10 older (O) human subjects for infusion of (1) Ringer solution (control), (2) 5 mM BH(4), (3) 5 mM BH(4

11 crease if cells were wounded in a low Ca(2+) Ringer's solution that inhibited both membrane resealing

12 en cells were wounded twice in normal Ca(2+) Ringer's solution, decreases in tension at the second wo

13 at, for fibroblasts wounded in normal Ca(2+) Ringer's solution, the membrane tension decreased dramat

14 uld restore membrane resealing in low Ca(2+) Ringer's solution.

15 its replacement by an isotonic Ca(2+)-Mg(2+) Ringer solution and cooling sharply reduced such access.

16 ) was induced by exposure to CO(2)-HCO(3)(-) Ringer's and the opposing outward flux by returning to H

17 Exposure to CO(2)-HCO(3)(-) Ringer's led to a transient decrease in pH(i) (component

18 However, when the CO(2)-HCO(3)(-) Ringer's was removed, the pH(i) increased transiently (c

19 g lumen and bath perfusion with an HCO(3)(-)-Ringer solution.

20 onium (TEA), (3) EMLA + TEA (Combo), and (4) Ringer solution (Control).

21 (L-NAME), (3) Keto + l-NAME (Combo), and (4) Ringer solution (Control).

22 Oxygen tension of a pH 7.5 Ringer's solution is measured in an airtight chamber tha

23 , (4) anti-bFGF antibody, (5) ngIgG, and (6) Ringer's solution.

24 sterior surface is continually bathed with a Ringer's solution in equilibrium with a CO2-gas air mixt

25 lished when cells were bathed in an alkaline Ringer solution.

26 When the cell was bathed in Ca2+-free Ba2+ Ringer solution, the K+ currents were blocked and large

27 h retina superfused with a bicarbonate-based Ringer solution in the subjective day and night; that is

28 se to 40 mM lactate in bicarbonate free (BF) Ringer's that was inhibited by niflumic acid and by MCT

29 ded by replacing bilateral Krebs bicarbonate Ringer (KBR) with Hepes-buffered Ringer solution exhibit

30 the conjunctiva with Na(+)-free bicarbonated Ringer's solution (BRS) were used to estimate contributi

31 to changes in pH(o) in bicarbonate-buffered Ringer solution.

32 bicarbonate Ringer (KBR) with Hepes-buffered Ringer solution exhibited basolateral, but not apical, r

33 vo loop studies HCO3 (-)-Ringer and butyrate-Ringer exhibit similar rates of water absorption in norm

34 Lumen butyrate-Ringer incubation activated NHE3-mediated Na(+) absorpti

35 in DSS-induced inflammation luminal butyrate-Ringer reversed water secretion observed with HCO3 (-)-R

36 intradermal microdialysis sites: control (C, Ringer solution), NO synthase inhibited (NOS-I, 10 mm l-

37 mmHg) men and women to serve as: control (C, Ringer solution), NOS inhibited (NOS-I, 10 mM L-NAME), A

38 ears) human subjects, serving as control (C, Ringer solution), NOS-inhibited (10.0 mM NG-nitro-L-argi

39 xposed to 110 mM hydroxylamine in a low-Ca2+ Ringer solution for a period of 10-50 s beginning 10-17

40 th EGTA or by bathing such cells in low-Ca2+ Ringer's solution.

41 cally paced myocytes (0.5 Hz, 1 mmol/L Ca2+, Ringer's solution [pH 7.4], 22 degrees C).

42 um proteins when incubated in buffered chick Ringer's solution.

43 helial 36Cl fluxes in bicarbonate-containing Ringer's were determined.

44 ger solution in the bath and K(+)-containing Ringer solution in the pipette, both currents were selec

45 With Na(+)-containing Ringer solution in the bath and K(+)-containing Ringer s

46 P < 0.05) and restored the NO contribution (Ringer: 44 +/- 3 % CVCmax vs. AA: 59 +/- 6 % CVCmax; P <

47 , produced cellular acidification in control Ringer.

48 e other 24 degrees C site served as control (Ringer solution + cooling).

49 e HS diet, AA improved the plateau % CVCmax (Ringer: 80 +/- 2 % CVCmax vs. AA: 89 +/- 3 % CVCmax; P <

50 nhibitor) in 50/50 dimethyl sulfoxide (DMSO)/Ringer's solution, 300 KIU aprotinin (a serine protease

51 on of neurones with zero calcium (1 mM EGTA) Ringer solution inhibited depolarization-induced calcium

52 compare the effects of treatment with either Ringer's lactate solution or ethyl pyruvate solution on

53 plantation, kidneys were flushed with either Ringer's solution or CRS at 35-37 degrees C or were not

54 H2O-1 (mean +/- s.e.m.), was 1/39th that for Ringer solution (1.94 +/- 0.01 microliter 2O-1 ).

55 outward flux by returning to HCO(3)(-)-free Ringer's.

56 rslips and then perfused with HCO(3)(-)-free Ringer's.

57 B(OH)(4)(-) (2.5-10 mM) in bicarbonate-free Ringer induced a rapid small acidification (0.01 pH unit

58 ts in normal frog Ringer's solution, Ca-free Ringer's solution, and BAPTA AM-pretreated preparations;

59 he currents observed in divalent cation-free Ringer's solution were due to Cx46 hemichannel opening,

60 Currents were recorded in chloride-free Ringer's solution with low or high concentrations of ext

61 ifts in E(m) in fibres studied in Cl(-)-free Ringer solution consistent with the Goldman-Hodgkin-Katz

62 rfusion bath with a low-HCO(3)(-) Cl(-)-free Ringer's solution (2.85 mM; pH 6.5), in the presence or

63 re perfused with a HCO(3)(-)-rich Cl(-)-free Ringer's solution (28.5 mM; pH 7.5).

64 efflux when cells were bathed in Cl(-)-free Ringer's.

65 d bathing solutions were iso-osmotic Cl-free Ringer's solutions modified using N-methyl-D-glucamine a

66 seen when returning immediately to IBMX-free Ringer solution.

67 studied in Cl(-)-free, normal and Na(+)-free Ringer solutions and in the presence of bumetanide, chlo

68 n isotonic Cl(-)-free, normal and Na(+)-free Ringer solutions showed similar E(m) values consistent w

69 In Na+-free Ringer, apical H2DIDS produced a transient alkalinizatio

70 delivered by pressure ejections of odor-free Ringer solution.

71 Compartments #1, #2 and #5 contained frog Ringer solution, #4 was filled with Vaseline and formed

72 LY evoked similar effects in normal frog Ringer's solution, Ca-free Ringer's solution, and BAPTA

73 Vessels, microperfused with frog Ringer solutions containing 0.1% bovine serum albumin an

74 exposure of muscles to a hypertonic glycerol-Ringer solution, its replacement by an isotonic Ca(2+)-M

75 Fluid challenges at 6 h (Ringer's solution at 80 ml x kg(-1) given over a period

76 In in vivo loop studies HCO3 (-)-Ringer and butyrate-Ringer exhibit similar rates of wate

77 ersed water secretion observed with HCO3 (-)-Ringer to fluid absorption.

78 as incubated for 30 minutes in 25 mM HCO3(-)-Ringer with agents promoting corneal deturgescence or co

79 In the control medium, 25 mM HCO3(-)-Ringer, 86Rb+ was accumulated to yield a T-M ratio of 6.

80 In contrast, fibres exposed to hypertonic Ringer solutions of normal ionic composition showed no s

81 etal muscle fibres by exposure to hypertonic Ringer solutions.

82 In Ringer solution containing both Cl- and HCO3-, the magni

83 In Ringer solution, the Ca2+ in the light-releasable pool c

84 In Ringer's solution, leupeptin delayed globulization witho

85 The proteolytic activity of fiber cells in Ringer's solution containing 10(-)(6) M and 2 x 10(-)(3)

86 ight of much lower intensity if delivered in Ringer solution but not if delivered in 0 Ca(2+), 0 Na(+

87 m or amplitude when rods were pre-exposed in Ringer solution to light which was bright enough to supp

88 llumination, whether the bleach was given in Ringer solution or in 0Ca(2+)/0Na(+) solution.

89 The [Ca2+]i in the globules in Ringer's solution, determined using Calcium Green-2, was

90 fluid had a similar effect to hyaluronan in Ringer solution.

91 i which normally accompanies illumination in Ringer solution.

92 troqinoxaline-2,3-dione (CNQX; 10 microM) in Ringer solution containing physiological concentrations

93 Mortality in Ringer's acetate, stroma-free hemoglobin, and 10% pentas

94 distilled water, in Ringer's solution or in Ringer's solution with added 1 M sucrose.

95 mesial TLE (MTLE) were immediately placed in Ringer's lactate; stearate indicator microelectrodes wer

96 s, which could be observed even from rods in Ringer solution.

97 PLP-null and control white matter soaked in Ringer remained largely compact.

98 th the oscillation period when stimulated in Ringer solution.

99 lined at a rate that was much slower than in Ringer solution and consistent with previous physiologic

100 ed to a third or less of its normal value in Ringer solution.

101 ontrol mice overnight in distilled water, in Ringer's solution or in Ringer's solution with added 1 M

102 The model used was the isolated Ringer perfused sheep choroid plexus.

103 , 6 min, 4 mM), challenge with elevated K(+) Ringer caused a dose-dependent DeltaDC in the range 10-1

104 A brief application (8 seconds) of high K(+) Ringer elicited a robust cytosolic Ca(2+) increase at th

105 ptor agonists had no effect on the high K(+) Ringer-elicited cytosolic Ca(2+) signal at OPL.

106 -40.0 +/- 4.1 mV (n = 14) and in 140 mM K(+) Ringer's it depolarized to -7.4 +/- 1.8 mV.

107 Application of Ba(2+) in 5 mM K(+) Ringer's produced a concentration-dependent depolarizati

108 In 5 mM K(+) Ringer's, the V(m) of cell clusters averaged -40.0 +/- 4

109 Challenge with 100 mM [K(+)] Ringer was used to assess the K(+) permeability of the p

110 C on subsequent challenge with 100 mM [K(+)] Ringer, indicating no effect on perineurial K(+) permeab

111 g sympathetic ganglion neurones in 2.0 mM K+ Ringer solution.

112 um, changing from normal Ringer to high [K+] Ringer (100 mM, KCl replacing NaCl) for 2 min caused neg

113 C) in response to challenge with 100 mM [K+] Ringer was used to assess the K+ permeability of the per

114 The inward current was observed in a KCl Ringer's bath and was almost nonexistent in a NaCl bath.

115 ainage period, animals received either Krebs Ringer Henseleit (the bile-depleted group), or sodium ta

116 t hepatocytes were incubated in anoxic Krebs-Ringer-HEPES buffer at pH 6.2 for 4 hours and reoxygenat

117 In Ca2+-free Krebs-Ringer bicarbonate buffer containing 2.8 mmol/l glucose,

118 e isolated by collagenase digestion in Krebs-Ringer bicarbonate (KRB) buffer at 37 degrees C.

119 Sphincter muscle was incubated in Krebs-Ringer bicarbonate buffer in the absence and presence of

120 lated, cultured, and then perifused in Krebs-Ringer bicarbonate buffer with 2 mmol/l glutamine using

121 without endothelium were suspended in Krebs-Ringer bicarbonate solution for isometric tension record

122 eriphery and immersed in an oxygenated Krebs-Ringer buffer.

123 he perfusate from rat liver exposed to Krebs-Ringer bicarbonate buffer only, 0-1mM [3,4-(13)C(2)]-4-h

124 nitially perfused at 37 degrees C with Krebs-Ringer's (KR) solution (in mmol/L: Ca(2+) 2.5, K(+) 5, M

125 Lactated Ringer's solution and 3-hr cold ischemia time were used

126 intradermal microdialysis with: (1) lactated Ringer solution (Control); (2) 10 mm ascorbate (Ascorbat

127 and each randomly assigned as: (1) lactated Ringer's (control); (2) 20 mm Nomega-nitro-l-arginine me

128 control (90% propylene glycol + 10% lactated Ringer solution); (2) 20 mm capsazepine to inhibit TRPV-

129 eceive either bolus of albumin in a lactated Ringer's solution or lactated Ringer's solution alone du

130 were resuscitated by administering lactated Ringer's solution intravenously to achieve and maintain

131 after each blood withdrawal, after lactated Ringer's resuscitation, and after infusion of shed blood

132 + P), bretylium tosylate (BT), and lactated Ringer solution were infused via intradermal microdialys

133 ), sildenafil (PDE5 inhibitor), and lactated Ringer's solution (control).

134 tive neurons in CA1 vs. Hextend and lactated Ringer's, and CA3 vs. Hextend (p<.05).

135 normal blood pressure with a blood/lactated Ringer's (1:2) mixture.

136 This was followed by lactated Ringer's solution, infused to a target urine output of 1

137 hloride) and balanced crystalloids (lactated Ringer's solution or Plasma-Lyte A).

138 (1, 3, 4, 5 and 7 pmol) with either lactated Ringer solution alone, or with ET(B) R (BQ-788), or nitr

139 o receive a 1-hr infusion of either lactated Ringer's solution (n = 6), 0.9% saline (n = 6), 5% dextr

140 posite effects in outcome, favoring Lactated Ringer.

141 lanced crystalloid solutions (e.g., lactated Ringer's, Plasma-Lyte) are an increasingly used alternat

142 ith its occurrence in animals given lactated Ringer's solution.

143 ally and flushed with cold heparin, lactated Ringer's and procaine (HeLP) solution.

144 ne (5 to 10 microg/kg) or hyperoxic lactated Ringer's solution.

145 induced by infusion of 0.2 M HCl in lactated Ringer solution in the acid group.

146 0.9% saline (n = 6), 5% dextrose in lactated Ringer's solution (D5RL) (n = 6), or 5% dextrose in wate

147 Three doses of EP dissolved in lactated Ringer's solution or lactated Ringer's solution (LR) alo

148 ntrols (n = 6) received intravenous lactated Ringer's solution according this dosing schedule: 1.5 mL

149 All groups received intravenous lactated Ringer's solution at 4 mL.kg-1.%burn(-1).24 hrs-1 for re

150 est the hypothesis that intravenous lactated Ringer's solution, infused at a rate used in resuscitati

151 = 9) solution made up exactly like lactated Ringer's solution except for the substitution of either

152 suggests that balanced fluids like Lactated Ringer's solution may be preferable.

153 8 medical ICU patients receiving no lactated Ringer's solution (RL).

154 t analysis suggested that volume of Lactated Ringer and 0.9% saline infused had opposite effects in o

155 Infusion of lactated Ringer's demonstrated no changes in the measured variabl

156 ention group) or an equal volume of lactated Ringer's solution (acid control group).

157 fusion than after administration of lactated Ringer's solution (p < 0.05).

158 e than 50%, while administration of lactated Ringer's solution provoked an approximately 2.5 times gr

159 volume was returned in the form of lactated Ringer's solution.

160 volume was returned in the form of lactated Ringer's solution.

161 volume was returned in the form of lactated Ringer's solution.

162 ients receiving a rapid infusion of lactated Ringer's solution.

163 nd less efficacious than the use of lactated Ringer.

164 of hemorrhage using 3% NaCl (HS) or lactated Ringer's (LR).

165 line (n = 25), Hextend (n = 25), or lactated Ringer's (n = 10).

166 ed in lactated Ringer's solution or lactated Ringer's solution (LR) alone were given by intravenous i

167 lood + 0.12, 0.24, or 0.36 g/kg) or lactated Ringer's solution (shed blood + 2 x volume of shed blood

168 eperfusion compared with albumin or lactated Ringer's solution administration (p < .001).

169 in a lactated Ringer's solution or lactated Ringer's solution alone during the first 6 hours of flui

170 n, during which either PentaLyte or lactated Ringer's solution-based resuscitation was administered.

171 in, immunosuppressive treatment, or lactated Ringer's therapy.

172 atients who received HES 130/0.4 or lactated Ringer, respectively (P < 0.038).

173 receive either SAAP with oxygenated lactated Ringer's (LR) solution (n = 6) or SAAP with oxygenated H

174 ood (FWB), (2) SAAP with oxygenated lactated Ringer's (LR), 1,600 mL/2 min, or (3) SAAP with oxygenat

175 blood plus albumin than blood plus lactated Ringer's solution (P < 0.01).

176 scitation with red blood cells plus lactated Ringer's solution (RL) is more effective than RL alone i

177 patients were randomized to receive lactated Ringer's solution and 17 to receive HES 130/0.4.

178 eriod; the untreated group received Lactated Ringer (LR) at the same time points.

179 ate-buffered saline, normal saline, lactated Ringer's solution, dextran, hespan, 5% human albumin, 25

180 r Hextend required less volume than lactated Ringer's (both p<.05).

181 e mean arterial blood pressure than lactated Ringer's or Hextend and confer neuroprotection in a mous

182 the PJ34-treated groups than in the Lactated Ringer group at 7 and 24 hours of reperfusion.

183 ded to the storage solution and the lactated Ringer poststorage rinse solution.

184 oup and 83 of 180 (46%) died in the lactated Ringer's group (p = 0.2).

185 oup and 40 of 180 (22%) died in the lactated Ringer's group (p = 0.5).

186 The patients in the lactated Ringer's group, however, received more fluid (P < 0.0001

187 her group (n = 8) received only the lactated Ringer's solution (LRS) vehicle.

188 d to guide the infusion rate of the lactated Ringer's.

189 the PJ34-treated group than in the Lactated Ringer-treated mice at 24 hours of reperfusion.

190 ovolemic shock, HSD (250 mL) versus lactated Ringer's solution (LR) as the initial resuscitation flui

191 ed mean arterial blood pressure vs. lactated Ringer's (p<.05).

192 rodialysis sites were perfused with lactated Ringer solution (Control), 40 pm, 4 nm or 400 nm ET-1; i

193 2 (n = 11) sites were perfused with lactated Ringer solution (Control), 400 nm ET-1, 10 mm N(G) -nitr

194 Mice were treated with lactated Ringer's (vehicle) solution, monophosphoryl lipid A, or

195 o early standard resuscitation with lactated Ringer's in cancer patients with sepsis did not improve

196 CLP-induced sepsis and treated with lactated Ringer's solution (LR, n = 13) survived longer than thos

197 ation than after resuscitation with lactated Ringer's solution (p < 0.05).

198 bsequently either resuscitated with lactated Ringer's solution (three times shed blood volume, n = 18

199 blood was then returned along with lactated Ringer's solution (two times the shed blood volume) to p

200 times the volume of shed blood with lactated Ringer's solution for 60 mins.

201 the volume of maximal bleedout with lactated Ringer's solution over 60 mins.

202 times the volume of shed blood with lactated Ringer's solution over 60 minutes.

203 on, awakened, and resuscitated with lactated Ringer's solution titrated to maintain hematocrit +/- 3%

204 After perfusion with lactated Ringer's solution, extravasated bBSA was detected with i

205 ed rats were then resuscitated with lactated Ringer's solution, four times the maximum shed blood vol

206 n ventilators and resuscitated with lactated Ringer's solution.

207 n ventilators and resuscitated with lactated Ringer's solution.

208 pressure>50 mm Hg for 30 min) with lactated Ringer's, Hextend, or PNPH, and then shed blood was rein

209 polarization produced by a high K(+) (40 mM) Ringer solution that was delivered rapidly and briefly t

210 rs with the endothelium bathed in a modified Ringer's solution and the epithelium bathed with silicon

211 age by aortic tear to receive 250 mL of MP4, Ringer's acetate, 10% pentastarch, or 4 g/dL of stroma-f

212 th different crystalloids (NaCl 0.9% (NaCl), Ringer's acetate (RA)) or colloids (Gelafundin 4% (Gel),

213 al endothelial monolayers perfused with NO3- Ringer's were exposed to I- pulses under isosmotic and,

214 undamaged perineurium, changing from normal Ringer to high [K+] Ringer (100 mM, KCl replacing NaCl)

215 esponses had a longer latency than in normal Ringer solution and were blocked by [D-pGlu1, D-Phe2, D-

216 In normal Ringer's solution, 57% of the rods tested could follow r

217 Acidification of Ringer solution resulted in a reduced antagonism by Zn2+

218 Applications of Ringer solutions with added NH(4)Cl induced rapid reduct

219 crodialysis fibres for the local delivery of Ringer solution and 20 mm ascorbic acid (AA).

220 bleed-out volume was returned in the form of Ringer's lactate (RL).

221 bleed-out volume was returned in the form of Ringer's lactate (RL).

222 r times the shed blood volume in the form of Ringer's lactate during a 60-minute period.

223 imal shed volume was returned in the form of Ringer's lactate solution.

224 r times the shed blood volume in the form of Ringer's lactate).

225 4 times the shed blood volume in the form of Ringer's lactate.

226 hed blood volume was returned in the form of Ringer's lactate.

227 4 times the shed blood volume in the form of Ringer's lactate.

228 tral forearm skin for continuous infusion of Ringer solution or 20 mM l-NAME.

229 ontrol and received a continuous infusion of Ringer solution.

230 oints received intra-articular injections of Ringer vehicle (control) or an activator of classical PK

231 itoneally every 6 hrs for 48 hrs) instead of Ringer's lactate solution starting 2 hrs after the injec

232 elongated fibers, which, in the presence of Ringer's solution (containing 2 mM Ca2+), underwent disi

233 In the presence of Ringer's solution containing 2 x 10(-)(3) M [Ca(2+)](o)

234 l pH was observed at two different values of Ringer solution pH, indicating that the circadian phenom

235 The work of Ringer, Hartmann, and others emphasized the importance o

236 ng VIP(10-28) at the three concentrations or Ringer solution and perfusion was continued for 45-60 mi

237 received an equivalent number of GFP-EPCs or Ringer's saline.

238 (n = 1443; isotonic or hypertonic saline or Ringer lactate solution) for all fluid interventions oth

239 ions, essentially a choice between saline or Ringer's lactate (compound sodium lactate or Hartmann's

240 l survival (at 12 hrs) was 0% with saline or Ringer's vs. 20% with Hextend, p =.05.

241 al time among animals treated with saline or Ringer's was 45% less compared with Hextend-treated anim

242 vely, compared with sucrose-EDTA solution or Ringer's solution containing 10(-)(8) M [Ca(2+)](o).

243 U with either 6% HES 130/0.42 (Tetraspan) or Ringer's acetate at a dose of up to 33 ml per kilogram o

244 testinal sleeve incubated in a physiological Ringer's solution.

245 The contralateral joint received Ringer solution without enzyme as a control.

246 nt therapy, as compared with those receiving Ringer's acetate.

247 alter pHi responses to CO(2)/HCO(3)(-)-rich Ringer, Na(+)-free induced acidification, or the rate of

248 in BCEC in HCO(3)(-)-free or HCO(3)(-)-rich Ringer, with and without niflumic acid (MCT inhibitor),

249 the presence of basolateral bicarbonate-rich Ringer solution.

250 de of the epithelium, was enhanced in simple Ringer's solution over that in tissue culture medium, an

251 After the sucrose/Ringer soak, normal myelin developed foci of decompactio

252 Sydney Ringer would be overwhelmed today by the implications of

253 Experiments using 0.5 nM leptin in the Ringer produced a concentration of leptin in the CSF of

254 ide and replacement of Na+ by choline in the Ringer solution, and irreversibly by both fetal and mate

255 ker, or when the extracellular Ca(2+) in the Ringer was replaced with equal molar EGTA.

256 between Ag/AgCl electrodes connected through Ringer-agar bridges to compartments #3 and #5.

257 s to investigate the effects of ATP added to Ringer's solution perfusing the retinal-facing (apical)

258 d with 172 of 400 patients (43%) assigned to Ringer's acetate (relative risk, 1.17; 95% confidence in

259 therapy versus 65 patients (16%) assigned to Ringer's acetate (relative risk, 1.35; 95% CI, 1.01 to 1

260 also unchanged when the cone was exposed to Ringer solution made up from heavy water, whose solvent

261 compared with that in fiber cells exposed to Ringer's solution alone (1995+/-461 nM, n = 10).

262 ncrease of [Ca2+]i in fiber cells exposed to Ringer's solution was measured, and the effects on the i

263 nutes, the [Ca2+]i of fiber cells exposed to Ringer's solution, containing 2 mM Ni2+ (574.7+/-29 nM;

264 obules generated from fiber cells exposed to Ringer's solution; in addition, no high molecular weight

265 Addition of INS37217 to Ringer's solution bathing the apical membrane transientl

266 fluid drainage rate was reduced relative to Ringer solution (P < 0.001, ANOVA) but increased steeply

267 H (pHi) or Na(+) ([Na(+)](i)) in response to Ringer solutions with/without B(OH)(4)(-) or HCO(3)(-) a

268 When the cilia were returned to Ringer solution after a period in reduced-Na(+) solution

269 Colloids in solutions similar to Ringer's lactate ('balanced solutions') may avoid these

270 of four times the volume of shed blood using Ringer's lactate solution for 60 mins.

271 Three kidneys were perfused using Ringer's lactate to replace excreted urine volume, and 8

272 ly 90 mins, and were then resuscitated using Ringer's lactate.

273 d storage (14% survival with CRS vs. 0% with Ringer's solution).

274 The endothelium was bathed with Ringer's fluid, and the outer surface was covered with s

275 s with non-cerebral trauma should begin with Ringer's lactate solution.

276 sites were then heated to 42 degrees C, with Ringer solution infused in one probe and N-nitro-L-argin

277 on of the isolated rat lens fiber cells with Ringer's solution led to their globulization in 30 +/- 3

278 treated animals (six of seven) compared with Ringer's acetate (two of seven), 10% pentastarch (one of

279 Compared with Ringer's lactate, saline, and saline-based colloids are

280 Compared with Ringer's solution (control), microdialysis delivery of t

281 with kidneys flushed with CRS compared with Ringer's solution or nontreatment.

282 extran 2000 reduced flows in comparison with Ringer solution.

283 were isolated typically after flushing with Ringer's buffer.

284 001), although no correlation was found with Ringer's lactate solution.

285 and perfused at a rate of 5 microL/min with Ringer's solution.

286 t a holding potential of +40 to +60 mV (with Ringer's in the pipette and pseudointracellular solution

287 milar average mass (2000 kDa; n = 7) or with Ringer solution vehicle (n = 2).

288 ion, microdialysis fibres were perfused with Ringer solution (control), a ATP-sensitive potassium cha

289 ays, microdialysis fibres were perfused with Ringer solution (control), a non-specific NO synthase in

290 nto the knees of anaesthetized rabbits, with Ringer solution as control in the contralateral joint.

291 auma sham-shock (T/SS) and resuscitated with Ringer's lactate or hypertonic saline.

292 The animals were resuscitated with Ringer's lactate solution to maintain constant pulmonary

293 ion with that of standard resuscitation with Ringer's lactate.

294 Resuscitation with Ringer's solution resulted in a standard base excess, an

295 magnitude when the cell was superfused with Ringer solution during the 5 s interval between odour ex

296 T:(g) of fiber cells superfused with Ringer's solution containing 2 x 10(-)(3) M, 10(-)(6) M,

297 after CRS was significantly higher than with Ringer's solution or without flushing (80% vs. 25% and 1

298 than in mice with pancreatitis treated with Ringer's lactate solution.

299 Treatment with Ringer's ethyl pyruvate solution ameliorated structural

300 d with four times the shed blood volume with Ringer's lactate solution over 60 minutes.