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1 domain that is present in the Gag protein of Rous sarcoma virus.
2 dentified in the gag-specific protein p2b of Rous sarcoma virus.
3 homologue of v-src, the transforming gene of Rous Sarcoma virus.
4 ch controls fibroblast transformation by the Rous sarcoma virus.
5 ive regulator of splicing (NRS) element from Rous sarcoma virus.
6 ng stimulatory element was discovered in the Rous sarcoma virus.
7 nalyses of two in-vitro-assembled capsids of Rous sarcoma virus.
8 that of the naturally nonmyristoylated MA of Rous sarcoma virus.
9 1) and 2, simian immunodeficiency virus, and Rous sarcoma virus.
11 chicken embryo fibroblasts (CEF) infected by Rous sarcoma virus against a confluent background of uni
13 roviruses that undergo type C morphogenesis, Rous sarcoma virus and human immunodeficiency virus, whi
15 s containing TATA and/or initiator elements, Rous sarcoma virus and thymidine kinase promoters in BeW
17 peat (DR) sequences flanking the src gene in Rous sarcoma virus are essential posttranscriptional con
18 e experiments reported here, using the avian Rous sarcoma virus as a model system, further define the
22 that allowed for the first-time formation of Rous sarcoma virus CA into structures resembling authent
25 of charged amino acids on the surface of the Rous sarcoma virus capsid protein in the assembly of app
26 ed sequence similarity, the structure of the Rous sarcoma virus capsid protein is similar to the stru
27 ences that exist in the C-terminal domain of Rous sarcoma virus capsid relative to the other capsid p
28 on of baby hamster kidney fibroblasts by the Rous sarcoma virus causes a significant increase in the
29 totally unrelated late domain sequences from Rous sarcoma virus (contained in its p2b sequence) or eq
33 unodeficiency virus type 1, visna virus, and Rous sarcoma virus exhibited different target site prefe
34 o- deficiency virus type 1, visna virus, and Rous sarcoma virus exhibited distinct preferences for wa
35 here that while deletion of the NC domain of Rous sarcoma virus Gag abolishes formation and budding o
43 purified, slightly truncated version of the Rous sarcoma virus Gag protein, Delta MBD Delta PR, and
44 rescence correlation spectroscopy-to examine Rous sarcoma virus Gag-Gag and -membrane interactions in
45 gion, Mpsi, from the 5' leader region of the Rous sarcoma virus genome that is sufficient to direct t
47 0 amino acid residue capsid protein from the Rous sarcoma virus has been determined by NMR methods.
48 structure of the three-domain integrase from Rous sarcoma virus in complex with viral and target DNAs
49 contrast, CEF cultures heavily infected with Rous sarcoma virus in the same medium underwent pervasiv
50 is tyrosine-phosphorylated when expressed in Rous sarcoma virus-infected chicken embryo fibroblasts (
51 plasmid acceptor, purified bacterium-derived Rous sarcoma virus integrase (IN), and a host cell DNA-b
52 ogous positions in visna virus integrase and Rous sarcoma virus integrase changed the target site pre
53 ous studies revealed that truncated forms of Rous sarcoma virus integrase containing two of the three
55 ct of the retroviral M domain from the avian Rous sarcoma virus is defined and its solution structure
57 quenced a small segment of the 3' end of the Rous sarcoma virus, just inside the poly(A) tail, at the
59 binding chicken Y-box protein that promotes Rous sarcoma virus long terminal repeat (RSV LTR)-driven
60 ilar to the levels observed in vivo with the Rous sarcoma virus long terminal repeat constitutive pro
61 tained human factor IX (hFIX) cDNA driven by Rous sarcoma virus long terminal repeat in the E1 region
62 antibiotic-resistance gene was driven by the Rous sarcoma virus long terminal repeat or the herpes si
65 of four promoters (dihydrofolate reductase, Rous sarcoma virus, long terminal repeat, or cytomegalov
66 1 is shown to transcriptionally activate the Rous sarcoma virus-long terminal repeat promoter, which
67 nducible, constitutively expressed reporter, Rous sarcoma virus-luciferase (RSV-LUC); nor does it blo
69 us, all of the functions associated with the Rous sarcoma virus MA sequence must be contained within
72 the site phosphorylated by gamma-PAK in the Rous sarcoma virus nucleocapsid protein NC in vivo and i
73 ngeable late assembly domains carried by the Rous sarcoma virus p2b protein and human immunodeficienc
75 er of cis-acting sequences within the RNA of Rous sarcoma virus play a role in preserving a large poo
76 is virus integrase and bacterial recombinant Rous sarcoma virus (Prague A strain) integrase (approxim
79 coli lacZ reporter gene under control of the Rous sarcoma virus promoter and mammalian RNA processing
80 cloned into a pMAMneo vector containing the Rous sarcoma virus promoter and the neomycin resistance
81 r carrying the cDNA for C/EBPalpha driven by Rous sarcoma virus promoter elements (AdCEBPalpha) or a
82 which sig-mEndo expression was driven by the Rous sarcoma virus promoter had moderately high serum le
83 ls from the beta-galactosidase gene when the Rous sarcoma virus promoter is used to drive transgene e
84 riptional control of a constitutively active Rous sarcoma virus promoter was regulated identically to
86 as an inducible HIV-1 genome controlled by a Rous sarcoma virus promoter with lac operator sequences.
91 ervative substitutions in this region of the Rous sarcoma virus protein were lethal due to a severe d
93 resolution of a fragment of the integrase of Rous sarcoma virus (residues 49-286) containing both the
101 membrane binding, we fused the MA domains of Rous sarcoma virus (RSV) and HIV-1 to the chemically ind
102 the Gag protein; however, recent studies of Rous sarcoma virus (RSV) and human immunodeficiency viru
107 immunodeficiency virus type 1 (HIV-1) and of Rous sarcoma virus (RSV) are morphologically distinct wh
109 onducted to investigate the initial steps of Rous sarcoma virus (RSV) assembly by examining the assoc
113 d-state NMR (ssNMR) resonance assignments of Rous sarcoma virus (RSV) CA, assembled into hexamer tube
114 sembly incompetent by testing the ability of Rous sarcoma virus (RSV) CA-SP to assemble in vitro into
117 structure of the N-terminal domain (NTD) of Rous sarcoma virus (RSV) capsid protein (CA), with an up
119 an immunodeficiency virus type 1 (HIV-1) and Rous sarcoma virus (RSV) capsid proteins form a beta-hai
121 ence appears to be modular, as the unrelated Rous sarcoma virus (RSV) Env can be made Vpu sensitive b
123 this model, CA proteins from both HIV-1 and Rous sarcoma virus (RSV) form similar hexagonal lattices
124 We have identified an assembly-defective Rous sarcoma virus (RSV) Gag mutant that retains signifi
126 tudy in vitro-assembled, immature virus-like Rous sarcoma virus (RSV) Gag particles and have determin
133 of the NC domain in assembly of VLPs from a Rous sarcoma virus (RSV) Gag protein and have characteri
136 ously reported that nuclear transport of the Rous sarcoma virus (RSV) Gag protein is intrinsic to the
137 med spontaneously in vitro from fragments of Rous sarcoma virus (RSV) Gag protein purified after expr
138 ped three very small, modular regions of the Rous sarcoma virus (RSV) Gag protein that are necessary
140 red that the oncogenic avian alpharetrovirus Rous sarcoma virus (RSV) Gag protein undergoes transient
143 een these two hypotheses, we made use of the Rous sarcoma virus (RSV) Gag protein, the PR of RSV IS i
146 eption was the 11-amino-acid p2b sequence of Rous sarcoma virus (RSV) Gag, which could fully restore
147 essential steps in understanding the chicken Rous sarcoma virus (RSV) genome association with a nonpe
148 elatively short, 82 nucleotide region of the Rous sarcoma virus (RSV) genome, called muPsi, was shown
152 t, the L domains of oncoretroviruses such as Rous sarcoma virus (RSV) have a more N-terminal location
154 Site-directed mutagenesis of recombinant Rous sarcoma virus (RSV) integrase (IN) allowed us to ga
155 nhanced when the serine at amino acid 124 of Rous sarcoma virus (RSV) integrase is replaced by alanin
158 we have examined whether the alpharetrovirus Rous sarcoma virus (RSV) is susceptible to inhibition by
159 proteins that specifically interact with the Rous sarcoma virus (RSV) L domain in vitro and in vivo.
161 taining the ts gene under the control of the Rous sarcoma virus (RSV) long terminal repeat (LTR) and
162 dogenous AAV promoters, p5 and p40, with the Rous sarcoma virus (RSV) long terminal repeat (LTR) and
163 eracts with the Schmidt-Ruppin strain of the Rous sarcoma virus (RSV) long terminal repeat (LTR) betw
166 CC-3' present on the noncoding strand of the Rous sarcoma virus (RSV) long terminal repeat (LTR) in a
167 eceptor (ecoR) cDNA under the control of the Rous sarcoma virus (RSV) long terminal repeat (LTR) prom
168 alovirus (CMV) immediate-early promoter, the Rous sarcoma virus (RSV) long terminal repeat, and the a
169 ts within the avian leukosis virus (ALV) and Rous sarcoma virus (RSV) LTR enhancers in a pattern iden
170 ssociation of HIV-1 Gag, as well as purified Rous sarcoma virus (RSV) MA and Gag, depends strongly on
171 in vitro flotation assay to directly measure Rous sarcoma virus (RSV) MA-membrane interaction in the
172 rted repeat (IR) within the U5 region of the Rous sarcoma virus (RSV) mRNA forms a structure composed
175 135-nucleotide (nt) direct repeats flank the Rous sarcoma virus (RSV) oncogene src and are composed o
176 virus-based vector LNCX, contain an internal Rous sarcoma virus (RSV) or cytomegalovirus (CMV) promot
177 ctivity compared with strong but nonspecific rous sarcoma virus (RSV) or cytomegalovirus promoters.
179 virus (EIAV) is functionally homologous with Rous sarcoma virus (RSV) p2b and human immunodeficiency
180 ent of Gag in in vivo and in vitro assembled Rous sarcoma virus (RSV) particles and to compare these
181 However, in the presence of budding HIV-1 or Rous sarcoma virus (RSV) particles, some glycoproteins,
183 mmunodeficiency virus type 1 (HIV-1) PTAP or Rous sarcoma virus (RSV) PPPY L domain in the p9 protein
184 t the enzymatic and structural properties of Rous sarcoma virus (RSV) PR are exquisitely sensitive to
185 pHyde gene under the control of a truncated Rous sarcoma virus (RSV) promoter (AdRSVpHyde) was gener
186 he cDNA for human iNOS was cloned behind the Rous sarcoma virus (RSV) promoter to create adenovirus (
190 enzyme, resulting in a highly active mutant Rous sarcoma virus (RSV) protease that displays many cha
191 ld be initiated by electroporation of cloned Rous sarcoma virus (RSV) proviral DNA into the developin
192 Expression of the Gag-Pol polyprotein of Rous sarcoma virus (RSV) requires a -1 ribosomal framesh
196 inding site in the 5' untranslated region of Rous sarcoma virus (RSV) RNA play an integral role in mu
197 ing element in the 3' untranslated region of Rous sarcoma virus (RSV) RNA was found to promote Rev-in
199 the single-particle cryo-EM structure of the Rous sarcoma virus (RSV) strand transfer complex (STC) i
200 a mouse monoclonal antibody directed against Rous sarcoma virus (RSV) subgroup A Env that will be use
201 produce milligram quantities of the soluble Rous sarcoma virus (RSV) synaptic complex that is kineti
202 describes new mutations in the CA protein of Rous sarcoma virus (RSV) that were designed to test whet
203 In chick embryo fibroblasts transformed by Rous sarcoma virus (RSV) the tyrosine phosphorylation of
204 V), feline immunodeficiency virus (FIV), and Rous sarcoma virus (RSV) to critically address the role
206 ne transport, the multidomain Gag protein of Rous sarcoma virus (RSV) undergoes importin-mediated nuc
208 discovery from Ray Erikson's group that the Rous sarcoma virus (RSV) v-Src-transforming protein had
209 ol vector containing a constitutively active Rous sarcoma virus (RSV) viral promoter driving the luci
211 y reactions with purified CA proteins of the Rous sarcoma virus (RSV) were used to define factors tha
213 t here that IP6 is also used for assembly of Rous sarcoma virus (RSV), a retrovirus from a different
216 HIV-1, Moloney murine leukemia virus (MLV), Rous sarcoma virus (RSV), and human T-cell lymphotropic
217 human immunodeficiency virus type 1 (HIV-1), Rous sarcoma virus (RSV), and Mason-Pfizer monkey virus
218 articles of the prototypic avian retrovirus, Rous sarcoma virus (RSV), by using scanning transmission
220 ton Rous recovered a virus, now known as the Rous sarcoma virus (RSV), from a chicken sarcoma, which
222 osed based on the existence of MA mutants in Rous sarcoma virus (RSV), murine leukemia virus, human i
227 , a Gammaretrovirus, and the Alpharetrovirus Rous sarcoma virus (RSV), were susceptible to inhibition
228 eplication-competent shuttle vector based on Rous sarcoma virus (RSV), with alternate retroviral PPTs
229 the utility of the system, we developed new Rous sarcoma virus (RSV)-based replication-incompetent v
230 tween the PBS and the CA dinucleotide of the Rous sarcoma virus (RSV)-derived vector RSVP(A)Z to matc
231 the endogenous polypurine tract (PPT) of the Rous sarcoma virus (RSV)-derived vector RSVP(A)Z was rep
234 eplication-defective adenoviral vectors [Adv.Rous sarcoma virus (RSV)-nf] representing three families
235 vector encoding a peptide inhibitor of PKA [Rous sarcoma virus (RSV)-protein kinase A inhibitor (PKI
237 the robustly induced enzymatic activities in Rous sarcoma virus (RSV)-transformed chicken cells.
239 ng a well-established retroviral model-avian Rous sarcoma virus (RSV)-we analyzed changes in an RSV v
250 e 3' splice site in the BPV-1 late pre-mRNA, Rous sarcoma virus src pre-mRNA, human immunodeficiency
251 As also found for other retroviruses, the Rous sarcoma virus structural protein Gag is necessary a
252 he negative regulator of splicing (NRS) from Rous sarcoma virus suppresses viral RNA splicing and is
253 ng (NRS) is a long cis-acting RNA element in Rous sarcoma virus that contributes to unspliced RNA acc
254 a mutant of the viral matrix (MA) protein of Rous sarcoma virus that disrupts viral RNA dimerization.
255 gate Gag protein structure and processing in Rous sarcoma virus, the prototype of the avian sarcoma a
256 e used cryo-electron tomography to visualize Rous sarcoma virus, the prototypic alpharetrovirus.
257 e-bound nonreceptor tyrosine kinase Src from Rous sarcoma virus, these interactions are mediated by a
259 to as antisense, can inhibit replication of Rous sarcoma virus through hybridization to viral RNA.
261 iruses containing the cellular receptors for Rous sarcoma virus (Tva) or ecotropic murine leukemia vi
265 natural or mutated 5' leader sequences from Rous sarcoma virus were expressed in avian cells in the
266 ses, including the prototypic oncoretrovirus Rous sarcoma virus, were synthesized on cytosolic riboso