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1 domain that is present in the Gag protein of Rous sarcoma virus.
2 dentified in the gag-specific protein p2b of Rous sarcoma virus.
3 homologue of v-src, the transforming gene of Rous Sarcoma virus.
4 ch controls fibroblast transformation by the Rous sarcoma virus.
5 ive regulator of splicing (NRS) element from Rous sarcoma virus.
6 ng stimulatory element was discovered in the Rous sarcoma virus.
7 nalyses of two in-vitro-assembled capsids of Rous sarcoma virus.
8 that of the naturally nonmyristoylated MA of Rous sarcoma virus.
9 1) and 2, simian immunodeficiency virus, and Rous sarcoma virus.
10                       One mechanism by which Rous sarcoma virus achieves incomplete splicing involves
11 chicken embryo fibroblasts (CEF) infected by Rous sarcoma virus against a confluent background of uni
12 the cell dramatically reduced the release of Rous sarcoma virus, an avian retrovirus.
13 roviruses that undergo type C morphogenesis, Rous sarcoma virus and human immunodeficiency virus, whi
14 n the SU and TM envelope protein subunits of Rous sarcoma virus and murine leukemia virus.
15 s containing TATA and/or initiator elements, Rous sarcoma virus and thymidine kinase promoters in BeW
16           Tva is the receptor for subgroup A Rous sarcoma virus, and it contains a single LDL-A modul
17 peat (DR) sequences flanking the src gene in Rous sarcoma virus are essential posttranscriptional con
18 e experiments reported here, using the avian Rous sarcoma virus as a model system, further define the
19 erve as mRNA for the envelope protein and in Rous sarcoma virus as src mRNA.
20  polyprotein are intrinsic components of the Rous sarcoma virus assembly pathway.
21      We have constructed and characterized a Rous sarcoma virus-based retroviral vector with the host
22 that allowed for the first-time formation of Rous sarcoma virus CA into structures resembling authent
23                                 Instead, the Rous sarcoma virus capsid protein contains a single cyst
24 olecular disulfide bond during assembly, the Rous sarcoma virus capsid protein does not.
25 of charged amino acids on the surface of the Rous sarcoma virus capsid protein in the assembly of app
26 ed sequence similarity, the structure of the Rous sarcoma virus capsid protein is similar to the stru
27 ences that exist in the C-terminal domain of Rous sarcoma virus capsid relative to the other capsid p
28 on of baby hamster kidney fibroblasts by the Rous sarcoma virus causes a significant increase in the
29 totally unrelated late domain sequences from Rous sarcoma virus (contained in its p2b sequence) or eq
30                                    Using the Rous sarcoma virus-derived vector RCAS, we previously sh
31           Since codon composition values for Rous sarcoma virus distinguished a "foreign" gene from t
32                      We have examined mutant Rous sarcoma virus envelope proteins with truncations or
33 unodeficiency virus type 1, visna virus, and Rous sarcoma virus exhibited different target site prefe
34 o- deficiency virus type 1, visna virus, and Rous sarcoma virus exhibited distinct preferences for wa
35 here that while deletion of the NC domain of Rous sarcoma virus Gag abolishes formation and budding o
36                               Using purified Rous sarcoma virus Gag and Gag truncations, we studied t
37 icles (VLPs), we observed that expression of Rous sarcoma virus Gag failed to produce VLPs.
38                                          The Rous sarcoma virus gag gene contains a cis-acting negati
39 ture of the CA domains in immature assembled Rous sarcoma virus Gag particles.
40                        In the context of the Rous sarcoma virus Gag polyprotein, only the nucleocapsi
41               It was shown previously that a Rous sarcoma virus Gag protein missing only the protease
42                                          The Rous sarcoma virus Gag protein undergoes transient nucle
43  purified, slightly truncated version of the Rous sarcoma virus Gag protein, Delta MBD Delta PR, and
44 rescence correlation spectroscopy-to examine Rous sarcoma virus Gag-Gag and -membrane interactions in
45 gion, Mpsi, from the 5' leader region of the Rous sarcoma virus genome that is sufficient to direct t
46 ckaging signal, MPsi, from the 5' end of the Rous sarcoma virus genome.
47 0 amino acid residue capsid protein from the Rous sarcoma virus has been determined by NMR methods.
48 structure of the three-domain integrase from Rous sarcoma virus in complex with viral and target DNAs
49 contrast, CEF cultures heavily infected with Rous sarcoma virus in the same medium underwent pervasiv
50 is tyrosine-phosphorylated when expressed in Rous sarcoma virus-infected chicken embryo fibroblasts (
51 plasmid acceptor, purified bacterium-derived Rous sarcoma virus integrase (IN), and a host cell DNA-b
52 ogous positions in visna virus integrase and Rous sarcoma virus integrase changed the target site pre
53 ous studies revealed that truncated forms of Rous sarcoma virus integrase containing two of the three
54        A direct comparison demonstrates that Rous sarcoma virus is capable of infecting aphidicolin-a
55 ct of the retroviral M domain from the avian Rous sarcoma virus is defined and its solution structure
56          Examination of the genomes of three Rous sarcoma virus isolates indicated that codon composi
57 quenced a small segment of the 3' end of the Rous sarcoma virus, just inside the poly(A) tail, at the
58         Although the integrity of the entire Rous sarcoma virus leader sequence is important for retr
59  binding chicken Y-box protein that promotes Rous sarcoma virus long terminal repeat (RSV LTR)-driven
60 ilar to the levels observed in vivo with the Rous sarcoma virus long terminal repeat constitutive pro
61 tained human factor IX (hFIX) cDNA driven by Rous sarcoma virus long terminal repeat in the E1 region
62 antibiotic-resistance gene was driven by the Rous sarcoma virus long terminal repeat or the herpes si
63 2(1) collagen promoters and 10-fold from the Rous sarcoma virus long terminal repeat.
64 ng MyoD under transcriptional control of the Rous sarcoma virus long terminal repeat.
65  of four promoters (dihydrofolate reductase, Rous sarcoma virus, long terminal repeat, or cytomegalov
66 1 is shown to transcriptionally activate the Rous sarcoma virus-long terminal repeat promoter, which
67 nducible, constitutively expressed reporter, Rous sarcoma virus-luciferase (RSV-LUC); nor does it blo
68             To further study the role of the Rous sarcoma virus MA sequence in the viral replication
69 us, all of the functions associated with the Rous sarcoma virus MA sequence must be contained within
70 o -90 confers FGF2/FSK responsiveness on the Rous sarcoma virus minimal promoter.
71                         In vivo screening of Rous sarcoma virus mutants was performed with randomly m
72  the site phosphorylated by gamma-PAK in the Rous sarcoma virus nucleocapsid protein NC in vivo and i
73 ngeable late assembly domains carried by the Rous sarcoma virus p2b protein and human immunodeficienc
74 , human immunodeficiency virus type 1 p6, or Rous sarcoma virus p2b.
75 er of cis-acting sequences within the RNA of Rous sarcoma virus play a role in preserving a large poo
76 is virus integrase and bacterial recombinant Rous sarcoma virus (Prague A strain) integrase (approxim
77                                              Rous sarcoma virus pre-mRNA contains an element known as
78                                           In Rous sarcoma virus, previous studies have identified imp
79 coli lacZ reporter gene under control of the Rous sarcoma virus promoter and mammalian RNA processing
80  cloned into a pMAMneo vector containing the Rous sarcoma virus promoter and the neomycin resistance
81 r carrying the cDNA for C/EBPalpha driven by Rous sarcoma virus promoter elements (AdCEBPalpha) or a
82 which sig-mEndo expression was driven by the Rous sarcoma virus promoter had moderately high serum le
83 ls from the beta-galactosidase gene when the Rous sarcoma virus promoter is used to drive transgene e
84 riptional control of a constitutively active Rous sarcoma virus promoter was regulated identically to
85 endogenously expressing SDF-1/CXCL12 under a Rous sarcoma virus promoter were produced.
86 as an inducible HIV-1 genome controlled by a Rous sarcoma virus promoter with lac operator sequences.
87 porter beta-galactosidase gene driven by the Rous Sarcoma Virus promoter.
88  HIT, and CHO cells using cytomegalovirus or Rous sarcoma virus promoters.
89                                          The Rous sarcoma virus protease displays a high degree of sp
90                 Other substrates such as the Rous sarcoma virus protein NC are phosphorylated by gamm
91 ervative substitutions in this region of the Rous sarcoma virus protein were lethal due to a severe d
92 f a weak src 3'ss is necessary for efficient Rous sarcoma virus replication.
93 resolution of a fragment of the integrase of Rous sarcoma virus (residues 49-286) containing both the
94  from human immunodeficiency virus type 1 or Rous sarcoma virus, respectively.
95                       Cell transformation by Rous sarcoma virus results in a dramatic change of adhes
96                                              Rous sarcoma virus RNA contains a negative regulator of
97                    Accumulation of unspliced Rous sarcoma virus RNA is facilitated in part by a negat
98  only 2.6-fold less efficiently than genomic Rous sarcoma virus RNA.
99                 In particular, although both Rous sarcoma virus (RSV) and AMV could replicate in cult
100 ze budding of virus-like particles (VLPs) of Rous sarcoma virus (RSV) and HIV type 1 (HIV-1).
101 membrane binding, we fused the MA domains of Rous sarcoma virus (RSV) and HIV-1 to the chemically ind
102  the Gag protein; however, recent studies of Rous sarcoma virus (RSV) and human immunodeficiency viru
103                          The Gag proteins of Rous sarcoma virus (RSV) and human immunodeficiency viru
104                          The Gag proteins of Rous sarcoma virus (RSV) and human immunodeficiency viru
105                                              Rous sarcoma virus (RSV) and murine leukemia virus (MLV)
106                                              Rous sarcoma virus (RSV) and murine leukemia virus (MLV)
107 immunodeficiency virus type 1 (HIV-1) and of Rous sarcoma virus (RSV) are morphologically distinct wh
108 ssed this question using the alpharetrovirus Rous sarcoma virus (RSV) as a model system.
109 onducted to investigate the initial steps of Rous sarcoma virus (RSV) assembly by examining the assoc
110                                              Rous sarcoma virus (RSV) budding requires an interaction
111                The first few residues of the Rous sarcoma virus (RSV) CA protein comprise a structura
112  of charged residues in the FL region of the Rous sarcoma virus (RSV) CA to particle assembly.
113 d-state NMR (ssNMR) resonance assignments of Rous sarcoma virus (RSV) CA, assembled into hexamer tube
114 sembly incompetent by testing the ability of Rous sarcoma virus (RSV) CA-SP to assemble in vitro into
115 variety of specimens assembled in vitro from Rous sarcoma virus (RSV) CA.
116                           The Gag protein of Rous sarcoma virus (RSV) can direct particle assembly an
117  structure of the N-terminal domain (NTD) of Rous sarcoma virus (RSV) capsid protein (CA), with an up
118                During virion maturation, the Rous sarcoma virus (RSV) capsid protein is cleaved from
119 an immunodeficiency virus type 1 (HIV-1) and Rous sarcoma virus (RSV) capsid proteins form a beta-hai
120         The cellular receptor for subgroup A Rous sarcoma virus (RSV) contains a single LDL-A module
121 ence appears to be modular, as the unrelated Rous sarcoma virus (RSV) Env can be made Vpu sensitive b
122        We have previously described a mutant Rous sarcoma virus (RSV) Env protein, Env-mu26, with an
123  this model, CA proteins from both HIV-1 and Rous sarcoma virus (RSV) form similar hexagonal lattices
124     We have identified an assembly-defective Rous sarcoma virus (RSV) Gag mutant that retains signifi
125                                              Rous sarcoma virus (RSV) Gag nuclear cycling is a unique
126 tudy in vitro-assembled, immature virus-like Rous sarcoma virus (RSV) Gag particles and have determin
127 triphosphate [PI(3,4,5)P(3)] levels impaired Rous sarcoma virus (RSV) Gag PM localization.
128                         The p2 region of the Rous sarcoma virus (RSV) Gag polyprotein contains an ass
129           Nucleocytoplasmic shuttling of the Rous sarcoma virus (RSV) Gag polyprotein is an integral
130                                          The Rous sarcoma virus (RSV) Gag polyprotein undergoes trans
131                                          The Rous sarcoma virus (RSV) Gag precursor polyprotein is th
132                Proteolytic processing of the Rous sarcoma virus (RSV) Gag precursor was altered in vi
133  of the NC domain in assembly of VLPs from a Rous sarcoma virus (RSV) Gag protein and have characteri
134                             We have purified Rous sarcoma virus (RSV) Gag protein and in parallel sev
135                 The first 86 residues of the Rous sarcoma virus (RSV) Gag protein form a membrane-bin
136 ously reported that nuclear transport of the Rous sarcoma virus (RSV) Gag protein is intrinsic to the
137 med spontaneously in vitro from fragments of Rous sarcoma virus (RSV) Gag protein purified after expr
138 ped three very small, modular regions of the Rous sarcoma virus (RSV) Gag protein that are necessary
139                       We discovered that the Rous sarcoma virus (RSV) Gag protein transiently localiz
140 red that the oncogenic avian alpharetrovirus Rous sarcoma virus (RSV) Gag protein undergoes transient
141                                       In the Rous sarcoma virus (RSV) Gag protein, the 25 amino-acid
142                                       In the Rous sarcoma virus (RSV) Gag protein, the M domain is co
143 een these two hypotheses, we made use of the Rous sarcoma virus (RSV) Gag protein, the PR of RSV IS i
144 erminal 100 residues of the non-myristylated Rous sarcoma virus (RSV) Gag protein.
145                                      We used Rous sarcoma virus (RSV) Gag together with membrane sens
146 eption was the 11-amino-acid p2b sequence of Rous sarcoma virus (RSV) Gag, which could fully restore
147 essential steps in understanding the chicken Rous sarcoma virus (RSV) genome association with a nonpe
148 elatively short, 82 nucleotide region of the Rous sarcoma virus (RSV) genome, called muPsi, was shown
149              Uniquely, the simple retrovirus Rous sarcoma virus (RSV) has a Gag structural protein th
150                     The major late domain of Rous sarcoma virus (RSV) has been mapped to a PPPY motif
151                              The L domain of Rous sarcoma virus (RSV) has been shown to interact with
152 t, the L domains of oncoretroviruses such as Rous sarcoma virus (RSV) have a more N-terminal location
153           We produced kinetically stabilized Rous sarcoma virus (RSV) intasomes with human immunodefi
154     Site-directed mutagenesis of recombinant Rous sarcoma virus (RSV) integrase (IN) allowed us to ga
155 nhanced when the serine at amino acid 124 of Rous sarcoma virus (RSV) integrase is replaced by alanin
156                                          The Rous sarcoma virus (RSV) is an exception, in that its ge
157         About one-third of the MA protein in Rous sarcoma virus (RSV) is phosphorylated.
158 we have examined whether the alpharetrovirus Rous sarcoma virus (RSV) is susceptible to inhibition by
159 proteins that specifically interact with the Rous sarcoma virus (RSV) L domain in vitro and in vivo.
160                             The discovery of Rous sarcoma virus (RSV) led to the identification of ce
161 taining the ts gene under the control of the Rous sarcoma virus (RSV) long terminal repeat (LTR) and
162 dogenous AAV promoters, p5 and p40, with the Rous sarcoma virus (RSV) long terminal repeat (LTR) and
163 eracts with the Schmidt-Ruppin strain of the Rous sarcoma virus (RSV) long terminal repeat (LTR) betw
164                                          The Rous sarcoma virus (RSV) long terminal repeat (LTR) cont
165                                          The Rous sarcoma virus (RSV) long terminal repeat (LTR) cont
166 CC-3' present on the noncoding strand of the Rous sarcoma virus (RSV) long terminal repeat (LTR) in a
167 eceptor (ecoR) cDNA under the control of the Rous sarcoma virus (RSV) long terminal repeat (LTR) prom
168 alovirus (CMV) immediate-early promoter, the Rous sarcoma virus (RSV) long terminal repeat, and the a
169 ts within the avian leukosis virus (ALV) and Rous sarcoma virus (RSV) LTR enhancers in a pattern iden
170 ssociation of HIV-1 Gag, as well as purified Rous sarcoma virus (RSV) MA and Gag, depends strongly on
171 in vitro flotation assay to directly measure Rous sarcoma virus (RSV) MA-membrane interaction in the
172 rted repeat (IR) within the U5 region of the Rous sarcoma virus (RSV) mRNA forms a structure composed
173                            In this report, a Rous sarcoma virus (RSV) mutant having two acidic-to-bas
174                                          The Rous sarcoma virus (RSV) negative regulator of splicing
175 135-nucleotide (nt) direct repeats flank the Rous sarcoma virus (RSV) oncogene src and are composed o
176 virus-based vector LNCX, contain an internal Rous sarcoma virus (RSV) or cytomegalovirus (CMV) promot
177 ctivity compared with strong but nonspecific rous sarcoma virus (RSV) or cytomegalovirus promoters.
178       The late assembly domain (L-domain) of Rous sarcoma virus (RSV) or human immunodeficiency virus
179 virus (EIAV) is functionally homologous with Rous sarcoma virus (RSV) p2b and human immunodeficiency
180 ent of Gag in in vivo and in vitro assembled Rous sarcoma virus (RSV) particles and to compare these
181 However, in the presence of budding HIV-1 or Rous sarcoma virus (RSV) particles, some glycoproteins,
182                                          The Rous sarcoma virus (RSV) polyadenylation site (PAS) is v
183 mmunodeficiency virus type 1 (HIV-1) PTAP or Rous sarcoma virus (RSV) PPPY L domain in the p9 protein
184 t the enzymatic and structural properties of Rous sarcoma virus (RSV) PR are exquisitely sensitive to
185  pHyde gene under the control of a truncated Rous sarcoma virus (RSV) promoter (AdRSVpHyde) was gener
186 he cDNA for human iNOS was cloned behind the Rous sarcoma virus (RSV) promoter to create adenovirus (
187                                              Rous sarcoma virus (RSV) promoter-driven expression of t
188 novirus containing the HERG gene driven by a Rous sarcoma virus (RSV) promoter.
189 ontrol of the human cytomegalovirus (CMV) or Rous sarcoma virus (RSV) promoters.
190  enzyme, resulting in a highly active mutant Rous sarcoma virus (RSV) protease that displays many cha
191 ld be initiated by electroporation of cloned Rous sarcoma virus (RSV) proviral DNA into the developin
192     Expression of the Gag-Pol polyprotein of Rous sarcoma virus (RSV) requires a -1 ribosomal framesh
193                                              Rous sarcoma virus (RSV) requires large amounts of unspl
194                    We asked whether a single Rous sarcoma virus (RSV) RNA can be translated and subse
195                The 5' untranslated region of Rous sarcoma virus (RSV) RNA is a highly ordered structu
196 inding site in the 5' untranslated region of Rous sarcoma virus (RSV) RNA play an integral role in mu
197 ing element in the 3' untranslated region of Rous sarcoma virus (RSV) RNA was found to promote Rev-in
198                 We previously identified the Rous sarcoma virus (RSV) stability element (RSE), an RNA
199 the single-particle cryo-EM structure of the Rous sarcoma virus (RSV) strand transfer complex (STC) i
200 a mouse monoclonal antibody directed against Rous sarcoma virus (RSV) subgroup A Env that will be use
201  produce milligram quantities of the soluble Rous sarcoma virus (RSV) synaptic complex that is kineti
202 describes new mutations in the CA protein of Rous sarcoma virus (RSV) that were designed to test whet
203   In chick embryo fibroblasts transformed by Rous sarcoma virus (RSV) the tyrosine phosphorylation of
204 V), feline immunodeficiency virus (FIV), and Rous sarcoma virus (RSV) to critically address the role
205                                          The Rous sarcoma virus (RSV) transmembrane (TM) glycoprotein
206 ne transport, the multidomain Gag protein of Rous sarcoma virus (RSV) undergoes importin-mediated nuc
207 RNA(Lys3) as the replication primer, whereas Rous sarcoma virus (RSV) uses tRNA(Trp).
208  discovery from Ray Erikson's group that the Rous sarcoma virus (RSV) v-Src-transforming protein had
209 ol vector containing a constitutively active Rous sarcoma virus (RSV) viral promoter driving the luci
210         A PPPPY motif within the L domain of Rous sarcoma virus (RSV) was previously characterized as
211 y reactions with purified CA proteins of the Rous sarcoma virus (RSV) were used to define factors tha
212         We previously reported that a mutant Rous sarcoma virus (RSV) with an alternate polypurine tr
213 t here that IP6 is also used for assembly of Rous sarcoma virus (RSV), a retrovirus from a different
214                                              Rous sarcoma virus (RSV), a simple retrovirus, needs to
215 resembles the PPPPY motif in the L domain of Rous sarcoma virus (RSV), an avian retrovirus.
216  HIV-1, Moloney murine leukemia virus (MLV), Rous sarcoma virus (RSV), and human T-cell lymphotropic
217 human immunodeficiency virus type 1 (HIV-1), Rous sarcoma virus (RSV), and Mason-Pfizer monkey virus
218 articles of the prototypic avian retrovirus, Rous sarcoma virus (RSV), by using scanning transmission
219         In some orthoretroviruses, including Rous Sarcoma Virus (RSV), CA carries a short and hydroph
220 ton Rous recovered a virus, now known as the Rous sarcoma virus (RSV), from a chicken sarcoma, which
221                                          For Rous sarcoma virus (RSV), Gag proteins are synthesized i
222 osed based on the existence of MA mutants in Rous sarcoma virus (RSV), murine leukemia virus, human i
223         During assembly and morphogenesis of Rous sarcoma virus (RSV), proteolytic processing of the
224                                          For Rous sarcoma virus (RSV), the size determinant maps to t
225                               In the case of Rous sarcoma virus (RSV), the size determinant maps to t
226           In prior studies of the retrovirus Rous sarcoma virus (RSV), we observed that the Gag prote
227 , a Gammaretrovirus, and the Alpharetrovirus Rous sarcoma virus (RSV), were susceptible to inhibition
228 eplication-competent shuttle vector based on Rous sarcoma virus (RSV), with alternate retroviral PPTs
229  the utility of the system, we developed new Rous sarcoma virus (RSV)-based replication-incompetent v
230 tween the PBS and the CA dinucleotide of the Rous sarcoma virus (RSV)-derived vector RSVP(A)Z to matc
231 the endogenous polypurine tract (PPT) of the Rous sarcoma virus (RSV)-derived vector RSVP(A)Z was rep
232 ions in the 5' end of the U3 sequence of the Rous sarcoma virus (RSV)-derived vector RSVP(A)Z.
233 stablishment of the transformed phenotype in Rous sarcoma virus (RSV)-infected cells.
234 eplication-defective adenoviral vectors [Adv.Rous sarcoma virus (RSV)-nf] representing three families
235  vector encoding a peptide inhibitor of PKA [Rous sarcoma virus (RSV)-protein kinase A inhibitor (PKI
236                                              Rous sarcoma virus (RSV)-tk bearing the HSV1-tk gene wer
237 the robustly induced enzymatic activities in Rous sarcoma virus (RSV)-transformed chicken cells.
238 tures found in osteoclasts, macrophages, and Rous sarcoma virus (RSV)-transformed fibroblasts.
239 ng a well-established retroviral model-avian Rous sarcoma virus (RSV)-we analyzed changes in an RSV v
240 icken embryo fibroblasts (CEF) infected with Rous sarcoma virus (RSV).
241 odel retroviral envelope protein (EnvA) from Rous sarcoma virus (RSV).
242 +)-ATPase (SERCA2a) under the control of the Rous sarcoma virus (RSV).
243 ric interactions between the Gag proteins of Rous sarcoma virus (RSV).
244 ansforming viral oncogene product encoded by Rous sarcoma virus (RSV).
245 d CRM1-dependent nuclear cycling was that of Rous sarcoma virus (RSV).
246 -membrane interaction of the alpharetrovirus Rous sarcoma virus (RSV).
247 ecades of study on an avian RNA tumor virus, Rous sarcoma virus (RSV).
248                                           In Rous sarcoma virus, splicing control is achieved in part
249 downstream from the 3'ss of the heterologous Rous sarcoma virus src gene.
250 e 3' splice site in the BPV-1 late pre-mRNA, Rous sarcoma virus src pre-mRNA, human immunodeficiency
251    As also found for other retroviruses, the Rous sarcoma virus structural protein Gag is necessary a
252 he negative regulator of splicing (NRS) from Rous sarcoma virus suppresses viral RNA splicing and is
253 ng (NRS) is a long cis-acting RNA element in Rous sarcoma virus that contributes to unspliced RNA acc
254 a mutant of the viral matrix (MA) protein of Rous sarcoma virus that disrupts viral RNA dimerization.
255 gate Gag protein structure and processing in Rous sarcoma virus, the prototype of the avian sarcoma a
256 e used cryo-electron tomography to visualize Rous sarcoma virus, the prototypic alpharetrovirus.
257 e-bound nonreceptor tyrosine kinase Src from Rous sarcoma virus, these interactions are mediated by a
258                                 In HIV-1 and Rous sarcoma virus, this region forms a rod-like structu
259  to as antisense, can inhibit replication of Rous sarcoma virus through hybridization to viral RNA.
260  initially identified as a phosphoprotein in Rous sarcoma virus-transformed cells.
261 iruses containing the cellular receptors for Rous sarcoma virus (Tva) or ecotropic murine leukemia vi
262                                 We find that Rous sarcoma virus virions, like the human immunodeficie
263 by his discovery of reverse transcriptase in Rous sarcoma virus virions.
264                  By cryoelectron tomography, Rous sarcoma virus VLPs show an organized hexameric latt
265  natural or mutated 5' leader sequences from Rous sarcoma virus were expressed in avian cells in the
266 ses, including the prototypic oncoretrovirus Rous sarcoma virus, were synthesized on cytosolic riboso
267                             The discovery of Rous sarcoma virus, which was reported by Peyton Rous in
268 HT29, and CaCo2) compared with a nonspecific Rous sarcoma virus-yCD virus.

 
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