ライフサイエンスコーパス: S. enterica

1 S. enterica cleanly converted all of the acid to acetyl-

2 S. enterica serotype Agona was isolated concurrently fro

3 S. enterica serovar Typhi carrying pTETlpp induced Frag

4 S. enterica serovar Typhimurium and S. flexneri cell ent

5 S. enterica serovar Typhimurium entry requires a functio

6 S. enterica serovar Typhimurium oligonucleotide microarr

7 S. enterica serovar Typhimurium strain 14028 produces tw

8 S. enterica subsp. salamae encodes the Salmonella pathog

9 (9 genovars) in 12 multiplex PCR mixes on 11 S. enterica strains.

10 oquinolone resistance in a collection of 136 S. enterica isolates, including 111 with intermediate or

11 sylvania and nearby states during July 2004: S. enterica serotypes Javiana, Anatum, Thompson, Typhimu

12 lly identify 12 serovars (16 genovars) in 24 S. enterica strains.

13 ta on 291 Salmonella isolates, including 250 S. enterica subspecies I strains from 32 serovars (52 ge

14 he cecum and large intestine with 10x LD(50) S. enterica serovar Typhimurium challenge at 7 days post

15 A total of 52 S. enterica serotype Enteritidis isolates representing 1

16 owth on 10 mM acetate, acs(+) induction in a S. enterica strain that cannot acetylate (i.e. inactivat

17 Our data suggest that the 12-2 antigen is a S. enterica subspecies I-specific LPS modification that

18 mented the flagella-deficient phenotype of a S. enterica acnB mutant, and the isolated AcnB5-4 polype

19 ve in IRG1 rescued the virulence defect of a S. enterica serovar Typhimurium mutant specifically defe

20 e corrected the pantothenate auxotrophy of a S. enterica yhhK strain, supporting in vitro evidence ob

21 We show that STM0557, a S. enterica subspecies I-specific gene encoding an inner

22 We also show that a S. enterica cobT strain that synthesizes GkCblS ectopica

23 f phenotypic similarity and diversity across S. enterica subspecies I and shown how the core genome o

24 differentiate the two invasive avian-adapted S. enterica serovar Gallinarum biotypes Gallinarum and P

25 concept that the emergence of human-adapted S. enterica is linked to human cultural transformations.

26 or the disease promoted by the human-adapted S. enterica serovar Typhi.

27 be associated with antibody production after S. enterica serovar Enteritidis vaccination.

28 uced colonization resistance of mice against S. enterica serotype Typhimurium.

29 CD8 T cells contribute to protection against S. enterica serovar Typhimurium in mice, but little is k

30 tory proteins and is highly conserved in all S. enterica serovars.

31 fim, bcf, and stb) were conserved within all S. enterica strains included in this study.

32 Although S. enterica serovars Enteritidis and Typhimurium are res

33 Thus, although S. enterica serovar Typhimurium and S. flexneri utilize

34 I, BlnI, and SpeI were most concordant among S. enterica serovar Typhimurium strains.

35 -population transmission appear to shape AMR S. enterica population structure in different hosts and

36 cs to study 90 antimicrobial resistant (AMR) S. enterica isolates from bovine and human hosts in New

37 with the serovar Typhi plasmid pHCM1 and an S. enterica serovar Typhimurium plasmid pR27.

38 utant retained intact LPS, we constructed an S. enterica serovar Typhimurium triple-knockout (TKO) mu

39 rect the tricarballylate growth defect of an S. enterica apbC mutant.

40 MSX and MSO inhibited the growth of an S. enterica DeltamddA strain unless glutamine or methion

41 The EutD protein restored growth of an S. enterica pta strain on acetate as the source of carbo

42 the diminished pathogenicity phenotype of an S. enterica sptP mutant.

43 romoter and was used to support growth of an S. enterica strain under conditions that demanded CobC-l

44 A comparison of the E. coli and S. enterica AraC regulons, coupled with a bioinformatic

45 We conclude that E. coli and S. enterica derive these unique genes from a common sour

46 The E. coli and S. enterica proteins were the most similar, although the

47 preciably more strongly than the E. coli and S. enterica proteins.

48 on the concentrations, inhibits E. coli and S. enterica serovar Typhimurium in an additive or antago

49 dentified consensus sequence for E. coli and S. enterica.

50 ion of rpoS is conserved between E. coli and S. enterica.

51 ee bacteria strains (B. cereus, E. coli, and S. enterica) and a yeast cell (S. cerevisiae), ranging i

52 ties for S. enterica serovar Enteritidis and S. enterica serovar Typhimurium strains when a minimum o

53 PFGE for S. enterica serovar Enteritidis and S. enterica serovar Typhimurium strains, respectively.

54 each of S. enterica serovar Enteritidis and S. enterica serovar Typhimurium.

55 ch to decrease colonization of C. jejuni and S. enterica in poultry gut along with other beneficial a

56 amination of drinking water by C. jejuni and S. enterica was also observed, suggesting a potential fu

57 on the roles of their aligned Y. pestis and S. enterica partners and showed that up to 73% of the pr

58 S. bongori, S. enterica subsp. salamae, and S. enterica subsp. arizonae share features of the infect

59 Salmonella enterica serotype Typhi (ST) and S. enterica serotype Paratyphi A (SPA) isolates were inc

60 rica in the liver, subsequently confirmed as S. enterica serotype I 4,5,12:-:1,2.

61 form from E. amylovora restored growth of as S. enterica cobB mutant strain on low acetate.

62 h nontyphoidal Salmonella serotypes, such as S. enterica serotype Typhimurium, are characterized by a

63 vealed multiple independent lineages such as S. enterica serovars S.

64 In contrast, a highly modified attenuated S. enterica serovar Typhimurium strain was not present i

65 e characteristics and genomes of 10 atypical S. enterica serovars linked to multistate foodborne outb

66 udy used transcriptional differences between S. enterica wild-type and ridA strains to explore the br

67 s that could be used to discriminate between S. enterica serovar Typhimurium isolates from the same g

68 Matches between S. enterica clustered regularly interspaced short palind

69 These results show that S. bongori, S. enterica subsp. salamae, and S. enterica subsp. arizo

70 we identified EspJ homologues in S. bongori, S. enterica subsp. salamae, and Salmonella enterica subs

71 Microscopy studies indicated that both S. enterica serovar Typhimurium and S. flexneri were loc

72 ntly shed by wildlife including turtles, but S. enterica subsp. enterica serovar Typhimurium or lesio

73 xt of the selective pressures encountered by S. enterica in vivo.

74 , there was significant biofilm formation by S. enterica serovar Typhimurium.

75 dent PRA formation in vitro was inhibited by S. enterica YjgF and the human homolog UK114.

76 almonella enterica infections represented by S. enterica serovar Newport has increased markedly among

77 In summary, MddA is the mechanism used by S. enterica to respond to oxidized forms of methionine,

78 A colanic acid/cellulose S. enterica serovar Typhimurium double mutant formed a m

79 that the MLST scheme employed here clustered S. enterica serovar Newport isolates in distinct molecul

80 gy searches to determine how far the E. coli/S. enterica paradigm can be generalised to other flagell

81 Concurrently, S. enterica subsp. salamae infection of J774.A1 macropha

82 Under aerobic conditions, S. enterica performs the corrinoid-dependent degradation

83 viously uncharacterized branch that contains S. enterica adapted to multiple mammalian species.

84 of SpvD is highly conserved across different S. enterica serovars, but residue 161, located close to

85 multiple methods is needed to differentiate S. enterica serovar Typhimurium isolates that geneticall

86 ings indicate that c-Abl is activated during S. enterica serovar Typhimurium infection and that its p

87 One hundred twenty-eight S. enterica serovar Typhimurium strains isolated from ca

88 ndings indicate that the sensor PhoQ enables S. enterica to respond to both host- and bacterial-deriv

89 population structure of commonly encountered S. enterica serotype Enteritidis outbreak isolates in th

90 rne illnesses caused by Salmonella enterica (S. enterica) every year.

91 om Vibrio cholerae, Yersinia enterocolitica, S. enterica serovar Typhimurium, and Klebsiella pneumoni

92 for immune responses and survival following S. enterica infection.

93 uorescein, biotin and digoxigenin coding for S. enterica, L. monocytogenes and E. coli, respectively.

94 BlnI, SfiI, and PacI as most concordant for S. enterica serovar Enteritidis, while XbaI, BlnI, and S

95 able sequence and microarray information for S. enterica subspecies I strains.

96 9% and 96% for five-enzyme combined PFGE for S. enterica serovar Enteritidis and S. enterica serovar

97 Here we show that apbC is required for S. enterica to use tricarballylate as a carbon and energ

98 scriminatory PFGE-based subtyping scheme for S. enterica serovar Enteritidis that relies on a single

99 100% correlation among Dice similarities for S. enterica serovar Enteritidis and S. enterica serovar

100 port here that CD8 T-cell lines derived from S. enterica serovar Typhimurium-infected BALB/c mice lys

101 cterization of the lytic domain of FlgJ from S. enterica as the model enzyme.

102 parameters of K(m) and k(cat) for FlgJ from S. enterica were determined to be 0.64 +/- 0.18 mg ml(-1

103 . enterica serovar Typhimurium and four from S. enterica serovar Typhi were used to create an assay c

104 Six genetic loci from S. enterica serovar Typhimurium and four from S. enteric

105 var Typhimurium or pulsed with proteins from S. enterica serovar Typhimurium culture supernatants.

106 he His6-tagged PduS cobalamin reductase from S. enterica was produced at high levels in Escherichia c

107 d in the prediction and prevention of future S. enterica outbreaks.

108 to Escherichia coli phages lambda and HK97, S. enterica phage ST64T, or a Shigella flexneri prophage

109 However, S. enterica serotype Typhimurium strains carrying deleti

110 In S. enterica serovar Typhimurium strains that had the abi

111 In S. enterica serovar Typhimurium, the PmrA/PmrB two-compo

112 In S. enterica serovar Typhimurium, the reduction in activi

113 In S. enterica, 2AA inactivates a number of pyridoxal 5'-ph

114 In S. enterica, the Ser-derived enamine/imine inactivates a

115 In S. enterica, this carboxysome-like structure (hereafter

116 ere, we demonstrate that Dap accumulation in S. enterica elicits a proline requirement for growth and

117 ling the genetic basis of host adaptation in S. enterica.

118 A model for tricarballylate catabolism in S. enterica is proposed.

119 esponse to osmotic challenge is conserved in S. enterica, dependence on these two sRNA regulators is

120 H, the predominant cysteine desulfhydrase in S. enterica.

121 , and have studied this phenotype further in S. enterica.

122 e and 28 HEG out of 130 Phage and 36 HEGs in S. enterica Typhi CT18, which shows that it is more effi

123 ppears to have been acquired horizontally in S. enterica serovar Typhimurium.

124 tative OmpR binding sites were identified in S. enterica serovar Typhi, 22 of which were associated w

125 (28)/FlgM interactions were also isolated in S. enterica serovar Typhimurium.

126 We further demonstrate that, like in S. enterica, the structural genes required for the flage

127 on, we screened a transposon library made in S. enterica serovar Typhimurium for the ability to persi

128 ding of the effects Dap has on metabolism in S. enterica, and likely other organisms, and highlight t

129 pproximately half the pseudogenes present in S. enterica serovar Typhi were conserved.

130 bial drug resistance is a growing problem in S. enterica that threatens to further compromise patient

131 uencing (RNA-seq) to map the AraC regulon in S. enterica.

132 The same residues in S. enterica ArnT are also needed for function.

133 CblS proteins restore alpha-RP synthesis in S. enterica lacking the CobT enzyme.

134 Here, we show that in S. enterica serotype Typhi, the causative agent of typho

135 We show that in S. enterica, BcsE is not essential for cellulose synthes

136 is of the determinants of thermotolerance in S. enterica serovar Typhimurium, we isolated the chr-1 m

137 onia-lyase (DpaL) alleviated Dap toxicity in S. enterica by catalyzing the degradation of Dap to pyru

138 und that SP control of rpoS transcription in S. enterica involves repression of the major rpoS promot

139 een in the absence of glucose and, unlike in S. enterica, causes a substantial growth defect.

140 ce factor known to be upregulated in vivo in S. enterica serovar Typhimurium infection of mice.

141 ause infection in different hosts, including S. enterica serovar Enteritidis (multiple hosts), S. Gal

142 ineages belonging to 28 serovars, including, S. enterica serovars S.

143 H8 infected S. enterica serotypes Enteritidis and Typhimurium and Es

144 biosynthetic gene, wcaM, was introduced into S. enterica serovar Typhimurium strain BJ2710 and was fo

145 Introduction of tviA into S. enterica serotype Typhimurium rendered flhDC transcri

146 ductase) reduces the growth of intracellular S. enterica serovar Typhimurium and has no effect on ext

147 ane permeability) upon infection by invasive S. enterica serovar Typhimurium than do infected control

148 context suggest that the evolution of known S. enterica sublineages is mediated mostly by two mechan

149 e types and other S. enterica serovars, like S. enterica serovar Infantis, possessing SGI1, while DT1

150 We identified matching S. enterica serotype Typhimurium isolates from 28 patien

151 It appears that MDR S. enterica serotype Typhi has emerged as a predominant

152 NO during coincubation with N. meningitidis, S. enterica, or E. coli.

153 Among the methods, S. enterica subsp. enterica serovars 4,5,12:i:-, Typhimu

154 conduct phenotypic comparison with the model S. enterica serovar Typhimurium.

155 was demonstrated using different Vi-negative S. enterica derivatives.

156 nd phenotypically characterized nontyphoidal S. enterica strains to 11 previously sequenced S. enteri

157 G, or sthABCDE did not reduce the ability of S. enterica serotype Typhimurium to colonize the spleen

158 The half-saturation affinity of S. enterica serovar Typhimurium for H2 is only 2.1 micro

159 c phenotype caused by the rpoD1181 allele of S. enterica allows past in vitro results to be incorpora

160 in the formation of an extensive biofilm of S. enterica serovar Typhimurium.

161 ch treatment against established biofilms of S. enterica and P. aeruginosa, respectively.

162 ave been isolated from the culture broths of S. enterica and uropathogenic E. coli, but MGE and TGE h

163 ntribute to long-term intestinal carriage of S. enterica serotype Typhimurium in genetically resistan

164 e genes important for intestinal carriage of S. enterica serotype Typhimurium in vertebrate animals.

165 e addition of propionaldehyde to cultures of S. enterica caused growth arrest from 8 to 20 mM, but no

166 Despite the high genetic diversity of S. enterica, all ancient bacterial genomes clustered in

167 ntitative, spatial, and temporal dynamics of S. enterica interactions are key to understanding how im

168 SfiI, PacI, and NotI) for 74 strains each of S. enterica serovar Enteritidis and S. enterica serovar

169 exchange, and loss play in the evolution of S. enterica sublineages, which to a certain extent are r

170 of an rpoE null mutant and the psp genes of S. enterica and Shigella flexneri are highly induced dur

171 alamae strain 3588/07 against the genomes of S. enterica subsp. enterica serovar Typhimurium strain L

172 hat EutQ is required during anoxic growth of S. enterica on ethanolamine and tetrathionate.

173 gene is rarely found outside subspecies I of S. enterica and often present in nonfunctional allelic f

174 terica, and the dose-dependent inhibition of S. enterica by a soluble carbohydrate antiadhesive.

175 Inoculations of S. enterica serovar Typhimurium and E. coli resulted in

176 ay complementary roles in the interaction of S. enterica serovar Typhimurium with the host intestinal

177 ci were useful in distinguishing isolates of S. enterica serovars Typhimurium and Newport that had di

178 Isolates of S. enterica serovars Typhimurium and Newport that were r

179 d used to amplify PCR targets in isolates of S. enterica serovars Typhimurium and Newport.

180 present the most common clinical isolates of S. enterica subsp. enterica.

181 nonoxidative early intracellular killing of S. enterica serovar Typhimurium by human macrophages and

182 A 50,000-CFU transposon library of S. enterica serovar Typhimurium strain SL1344 was serial

183 Toward this goal, a random library of S. enterica typhimurium 14028 genomic DNA was cloned ups

184 and persistence of international lineages of S. enterica serovars in food production chain is support

185 the previously uncharacterized aer locus of S. enterica serovar Typhimurium revealed them to be cont

186 A fis mutant of S. enterica serovar Typhimurium showed a ninefold increa

187 A library of 960 signature-tagged mutants of S. enterica serovar Choleraesuis was constructed and scr

188 Random transposon mutants of S. enterica serovar Typhimurium were screened for impair

189 re that transcription of the vapBC operon of S. enterica is controlled by a recently discovered regul

190 ace major lineages and ecological origins of S. enterica serotype Enteritidis.

191 The pathogenesis of S. enterica depends on flagella, which are appendages th

192 wn or potential roles in the pathogenesis of S. enterica.

193 was used to understand the pathogenicity of S. enterica serovar Choleraesuis in its natural host and

194 xamined the distribution of PFGE patterns of S. enterica serotype Typhi isolates from patients with a

195 aecal shedding and intestinal persistence of S. enterica serotype Typhimurium ATCC14028 in Salmonella

196 l in assessing the evolutionary potential of S. enterica sublineages and aid in the prediction and pr

197 he GTPase and ribosome-binding properties of S. enterica BipA.

198 wo organisms showed this to be a property of S. enterica rather than of the FadR proteins per se.

199 Here we show that the PduQ protein of S. enterica is an iron-dependent alcohol dehydrogenase u

200 echanisms that determine net growth rates of S. enterica within the host.

201 r infection, suggesting that the recovery of S. enterica serotype Typhimurium from fecal samples clos

202 ACDEFG did not result in reduced recovery of S. enterica serotype Typhimurium from fecal samples coll

203 terogeneity, and increased NAL resistance of S. enterica serovar Typhi.

204 To improve sensitivity of S. enterica serovar Typhimurium detection, multiwalled c

205 ost common clinical and nonhuman serovars of S. enterica in the United States.

206 d microbiological techniques and serovars of S. enterica were determined by PCR and/or agglutination

207 h five strains each of the target species of S. enterica and L. monocytogenes, along with five strain

208 complicate efforts to control the spread of S. enterica serovar Heidelberg in food animal and human

209 ype protein, supported growth of a strain of S. enterica devoid of Acs (acetyl-CoA synthetase; AMP-fo

210 The outbreak strain of S. enterica serotype Typhimurium was cultured from a pat

211 We report that wild-type strains of S. enterica grow on decanoic acid, whereas wild-type E.

212 of the growth behavior of mutant strains of S. enterica lacking specific functions encoded by the 17

213 wild-type but not for attenuated strains of S. enterica serovar Typhimurium.

214 (by single and double mutations) strains of S. enterica serovars Typhimurium and Typhi were recovere

215 Phenotypically, strains of S. enterica that lack RidA accumulated significantly mor

216 cherichia coli is similar overall to that of S. enterica but is seen in the absence of glucose and, u

217 ubtilis also enhanced the thermotolerance of S. enterica.

218 imple method for identifying new variants of S. enterica serovar Typhimurium in the field.

219 B signaling pathway and promote virulence of S. enterica serovar Typhimurium in mice.

220 ibitor, Co(III) hexaammine, had no effect on S. enterica serovar Typhimurium invasion of Caco-2 epith

221 pecies in all samples, with detection of one S. enterica and two Listeria TRFs in all cases, and dete

222 e of two different haplotypes following oral S. enterica serovar Typhimurium infection.

223 eement with results obtained in the original S. enterica serovar Typhimurium STM screen, illustrating

224 ca serovar Typhimurium phage types and other S. enterica serovars, like S. enterica serovar Infantis,

225 ved in other Salmonella strains, i.e., other S. enterica serovar Typhimurium phage types and other S.

226 biquitous serovars of the bacterial pathogen S. enterica and recently has been emerging in many count

227 y following eradication of the fowl pathogen S. enterica serovar Gallinarum in the mid-20th century.

228 for bacterial colonization after pathogenic S. enterica serovar Enteritidis inoculation and for circ

229 ated by low Mg(2+) and C18G as in pathogenic S. enterica.

230 e serotypes impact the ecology of pathogenic S. enterica on-farm.

231 Closely related S. enterica serotype Typhimurium isolates were cultured

232 fications were absent in the closely related S. enterica serovar Typhimurium LT2 and from a mutant of

233 ions occurring in this locus in FQ-resistant S. enterica serovar Typhimurium epidemic clones resulted

234 smid-associated genes in multidrug resistant S. enterica serovar Heidelberg, antimicrobial resistance

235 e transfer; different isolates from the same S. enterica serovar can exhibit significant variation in

236 TRFs in all cases, and detection of a second S. enterica TRF in 91% of cases.

237 enterica strains to 11 previously sequenced S. enterica genomes to carry out the most comprehensive

238 branch that also includes the human-specific S. enterica Paratyphi C, illustrating the evolution of a

239 es are missing (e.g., avrA, sopB, and sseL), S. enterica subsp. salamae invades HeLa cells and contai

240 ar cathepsin inhibitor (stefin B) suppressed S. enterica Typhimurium-induced cell death.

241 These results demonstrate that systemic S. enterica infection and diarrhea/colitis are distinct

242 These gene panels distinguish all tested S. enterica subspecies I serovars and their known genova

243 nterica ridA causes Ser sensitivity and that S. enterica RidA and its homologs from other organisms h

244 ing 12/15-lipoxygenase (12/15-LOX), and that S. enterica serovar Typhimurium and S. flexneri share ce

245 We further demonstrate that S. enterica LT2 retained the ability to grow on 1,2-prop

246 urther into this pathway, we also found that S. enterica serovar Typhimurium and S. flexneri activate

247 The results of this study indicate that S. enterica serovar Typhimurium can outgrow E. coli in h

248 Our results indicate that S. enterica synthesizes alpha-R, a metabolite that had n

249 oreover, the data raise the possibility that S. enterica DeltafrdABCD DeltasdhCDA double mutants and

250 We also show that S. enterica yhhK strains accumulate pro-PanD, and that n

251 Overall this work shows that S. enterica serovar Enteritidis strains circulating in U

252 The S. enterica fadR strains grow more rapidly than the wild

253 The S. enterica serovar Typhimurium hybrid strains showed si

254 As the S. enterica species comprises sublineages that differ gr

255 xpressed in C. elegans intestinal cells, the S. enterica TTSS-exported effector protein SptP inhibite

256 to the sequence reported in GenBank for the S. enterica serovar Typhimurium LT2 strain.

257 In the S. enterica ACS structure, the propyl group of adenosine

258 The flagella-release phenotype of the S. enterica fliL mutant has a bearing on FliL-dependent

259 olog complemented the Ser sensitivity of the S. enterica ridA mutant.

260 The chromosomal copy of the S. enterica serovar Typhimurium sigma(28) structural gen

261 Most lineages of the S. enterica subspecies Typhimurium cause gastroenteritis

262 es between the two organisms showed that the S. enterica FadE and FadBA enzymes were responsible for

263 ons that exhibit significant homology to the S. enterica transducing phage ES18.

264 y was not enhanced by Mg(2+) and, unlike the S. enterica CobA enzyme, it was >50% inhibited by Mn(2+)

265 Unlike the S. enterica enzyme, CobA(Mm) used cobalamin (Cbl) as a s

266 tial DNA and glucuronide binding affinity to S. enterica GusR.

267 This difference in growth was attributed to S. enterica having higher cytosolic levels of the induci

268 glycerol dehydratase family, in contrast to S. enterica, which relies on a B12-dependent enzyme.

269 A, SopA, SopB, SopD, and SopE2 contribute to S. enterica serotype Typhimurium invasion of epithelial

270 sceptibility of caveolin-1-deficient mice to S. enterica serovar Typhimurium.

271 NPs as molecular markers for the response to S. enterica serovar Enteritidis may result in the enhanc

272 inacin genes with the phenotypic response to S. enterica serovar Enteritidis, an F1 population of chi

273 ons of sire alleles with progeny response to S. enterica serovar Enteritidis.

274 onal and if it was evolutionarily similar to S. enterica serovar Typhimurium CorA.

275 the median C(T) values for M. tuberculosis, S. enterica, and EBV cfDNA were significantly lower in b

276 et al. that oral inoculation with wild-type S. enterica serovar Typhimurium strains lead to bacteria

277 egulator (prpR)) were evaluated in wild-type S. enterica serovar Typhimurium TR6583 and prpB(-) or pr

278 encoding PagL, PagP, and LpxR into wild-type S. enterica serovar Typhimurium.

279 surveillance for nontyphoidal and typhoidal S. enterica infections among inpatients and outpatients

280 and invasive infections due to non-typhoidal S. enterica infections resulted in the highest burden, c

281 Some hazards, such as non-typhoidal S. enterica, were important causes of FBD in all regions

282 Unlike S. enterica serotype Typhimurium, serotype Typhi or a se

283 We show that the previously unsequenced S. enterica serovar 9,12:l,v:- belongs to the B clade of

284 In this study, we used S. enterica serovar Typhimurium as an in vivo heterologo

285 In vitro methylation analyses utilizing S. enterica and Thermotoga maritima CheR proteins and MC

286 conditions, E. coli failed to grow, whereas S. enterica grew well.

287 While S. enterica serovar Typhimurium has been shown to kill i

288 was first formed followed by challenge with S. enterica serovar Typhimurium, there was significant b

289 lysed bone marrow macrophages infected with S. enterica serovar Typhimurium or pulsed with proteins

290 H2-M3-transfected fibroblasts infected with S. enterica serovar Typhimurium SL3261 or treated with S

291 oducing EcN to mice previously infected with S. enterica substantially reduced intestinal colonizatio

292 l vaccine, rabbits were orally infected with S. enterica Typhimurium strain chi3987 harboring phagemi

293 the first 1 or 2 weeks after infection with S. enterica serotype Typhi.

294 To identify cases of human infection with S. enterica serotype Typhimurium potentially related to

295 tion model, mice were orally inoculated with S. enterica 24 h post-initiation of abrupt withdrawal fr

296 ating antibody levels after inoculation with S. enterica serovar Enteritidis bacterin vaccine.

297 um chloride (DPI), but infection of MDM with S. enterica serovar Typhimurium did not cause an increas

298 These operons are widely distributed within S. enterica but absent from the closely related Escheric

299 reaching the level in cells infected with WT S. enterica serovar Typhimurium, than the level in host

300 ty did not reach the level of wild-type (WT) S. enterica serovar Typhimurium.