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1  contribute to the thrombogenic potential of S. sanguis.
2  we have also investigated the F-ATPase from S. sanguis.
3 gesting additional mechanisms of response to S. sanguis.
4 und to hydroxyapatite to promote adhesion of S. sanguis.
5 model of platelet aggregation in response to S. sanguis.
6 s with strains of Streptococcus gordonii and S. sanguis.
7 nctional adhesion epitopes on the surface of S. sanguis.
8 uced to the same extent in the nonpathogenic S. sanguis.
9 ggregating doses of 4 to 40 x 10(9) cells of S. sanguis 133-79.
10 ms by Western blot indicated that S. oralis, S. sanguis, A. viscosus, A. odontolyticus, and A. israel
11 eration murine hybridomas that produced anti-S. sanguis adhesin monoclonal antibodies (mAb1).
12 th experimental salivary films and inhibited S. sanguis adhesion in a dose-dependent fashion.
13             These data strongly suggest that S. sanguis adhesion to sHA is maximized when several adh
14  hypothesis that aggregation occurs in vivo, S. sanguis (Agg+ or Agg- suspension) was infused intrave
15  in salivary films to form binding sites for S. sanguis, an in vitro model of saliva-coated teeth was
16 utans is more tolerant of low pH values than S. sanguis and hence pathogenic.
17 y is induced under acidic conditions in both S. sanguis and S. mutans; however, it is not induced to
18 harides occurred primarily on the strains of S. sanguis and S. oralis while G-containing polysacchari
19                                Next, PAAP(+) S. sanguis and type II collagen were tested for T cell c
20                    gingivalis, F. nucleatum, S. sanguis, and A. naeslundii were grown on smooth, acid
21                                  The PAAP(+) S. sanguis appear to activate adult memory, rather than
22 mutans and Streptococcus sanguinis (formerly S. sanguis), as a model to probe the possible mechanisms
23       We found that S. mitis, S. oralis, and S. sanguis, as well as oral actinomycetes, including A.
24                                      Feeding S. sanguis at 6 days postpartum delayed the onset of art
25 c activity expressed by S. mutans VA-29R and S. sanguis ATCC 10556 against X-Pro4-nitroanilide and X-
26 s provide evidence that S. mutans VA-29R and S. sanguis ATCC 10556 possess a pathway for the complete
27 on and expression in Escherichia coli of the S. sanguis ATPase operon.
28 y immunoglobulin A and alpha-amylase forms a S. sanguis-binding site.
29 mmune arthritis, DBA/1J pups were given live S. sanguis, CB11, or type II collagen intragastrically.
30                                              S. sanguis CbpA- mutants were constructed and tested for
31  atpD deletion strain of E. coli showed that S. sanguis-E. coli hybrid enzymes were able to degrade A
32                        The similarity to the S. sanguis enzyme and the presence of a putative zinc-bi
33  specific antibodies or peptides block PAAP, S. sanguis fails to induce platelet aggregation in vitro
34  are consistent with a thrombogenic role for S. sanguis in human disease, contributing to the develop
35                              Therefore, Agg+ S. sanguis induced platelet aggregation in vitro.
36                                              S. sanguis IUOM-11M alpha-glucosidase (EC 3.2.1.20) demo
37 Analysis of starch degradation products from S. sanguis IUOM-11M and A. viscosus IUOM-62 demonstrated
38                                              S. sanguis IUOM-11M and JC804, S. mutans 6715, S. saliva
39 ose inhibited sucrose-dependent synthesis of S. sanguis IUOM-11M insoluble polysaccharide and both pr
40   We therefore hypothesized that circulating S. sanguis might cause coronary thrombosis and signs of
41                             To learn whether S. sanguis modulates transmucosally the Ag-specific deve
42                                   S. mutans, S. sanguis, P. gingivalis, and F. nucleatum were incubat
43                          Pneumonia caused by S. sanguis resolves over the course of 8 d, whereas pneu
44 xhibited by selected strains of S. gordonii, S. sanguis, S. mutans, S. mitis, and S. oralis but only
45 trains); and one strain each of S. gordonii, S. sanguis, S. sobrinus, and S. vestibularis.
46 um, P. gingivalis, P. intermedia, S. mutans, S. sanguis, Selenomonas sputigena, T. denticola, and T.
47  ATCC 15987 degraded starch and maltose, and S. sanguis SS34 degraded only maltose.
48                     Western blot analyses of S. sanguis surface macromolecules showed antigens at 36
49 roliferated more when incubated with PAAP(+) S. sanguis than with PAAP(-) Streptococcus gordonii or t
50 reparations from S. aureus, B. subtilis, and S. sanguis were not able to induce NO from lipopolysacch
51 region with 10 tandem repeats of a 20-mer in S. sanguis, which is replaced by a similar but less repe
52                         The pre-treatment of S. sanguis with the Neem stick extract or the gallotanni