ライフサイエンスコーパス: SAS

1 SAS are, nevertheless, important because they prompt dos

2 SAS arises when an allele is beneficial to one sex but h

3 SAS code (SAS Institute, Inc., Cary, North Carolina) for

4 SAS should be considered as an adjuvant treatment to ame

5 SAS(C) version 9.3 software was used to calculate descri

6 SAS-4 deletion led to a progressive loss of centrioles,

7 SAS-6 constitutes the cartwheel, and SAS-6 levels remain

8 SAS-6 from Chlamydomonas reinhardtii and Danio rerio was

9 SAS-6 mutants with alanine substitutions in a previously

10 SAS-6 oligomerisation is driven by two independent inter

11 SAS-6 proteins are thought to impart the ninefold symmet

12 esions using PROC CALIS in SAS, version 9.1 (SAS Institute, Inc., Cary, North Carolina) and Mplus, ve

13 red for centriole duplication: SPD-2, ZYG-1, SAS-5, SAS-6, and SAS-4.

14 Sas-6, and DSas-4/CPAP-orthologues of ZYG-1, SAS-6, and SAS-4, respectively-are required for centriol

15 sponse time on control trials (TMS(CT)), (2) SAS 200 ms prior to the go cue (SAS), (3) suprathreshold

16 centriole duplication: SPD-2, ZYG-1, SAS-5, SAS-6, and SAS-4.

17 sential protein spindle assembly abnormal 6 (SAS-6), which forms a structural scaffold templating the

18 by the spindle assembly abnormal protein 6 (SAS-6) family.

19 of the spindle assembly abnormal protein 6 (SAS-6) is an essential step in the centriole assembly pr

20 w conserved proteins (ZYG-1/Sak/Plk4, SAS-6, SAS-5/Ana2, and SAS-4), and is often initiated by the fo

21 to the SAS-6 coiled coil recruits the SAS-6-SAS-5 complex to the mother centriole, where a ZYG-1 kin

22 erver ICC 0.95-0.97, interobserver ICC 0.91; SAS intraobserver ICC 0.95-0.99, interobserver ICC 0.93;

23 A SAS macro (SAS Institute Inc., Cary, North Carolina) is

24 d that the release of planned movements by a SAS is mediated by subcortical, possibly brainstem, path

25 The early release of movement by a SAS was significantly delayed (P < 0.001, average delay

26 on, could delay the release of movement by a SAS.

27 Administration of a SAS accelerated ankle dorsiflexion in both groups, but m

28 The presentation of a SAS alone at -200 ms resulted in the release of the plan

29 In 25% of trials, a SAS was delivered simultaneously with the imperative sti

30 e we define the volume of correlation, Vc, a SAS invariant derived from the scattered intensities tha

31 the imperative stimulus was combined with a SAS, presumably through release of a subcortically store

32 P patients in trials both with and without a SAS would argue in favor of a cortically stored response

33 In some cases, alternative SAS would give rise to mRNAs encoding proteins with diff

34 genes have between one and three alternative SAS, but PAS are more dispersed.

35 to 54 degrees S) in southern South America (SAS), to quantify the coupling of SAM and regional wildf

36 -6-depleted embryos, the levels of ZYG-1 and SAS-5 are reduced and the ZYG-1- and SAS-5-dependent rec

37 s centriole assembly by protecting ZYG-1 and SAS-5 from degradation.

38 G-1 and SAS-5 are reduced and the ZYG-1- and SAS-5-dependent recruitment of SAS-6 to the nascent cent

39 ent among standard ABC/2, modified ABC/2 and SAS: (mean) 12.8 (SD 16.3), 8.9 (9.2), 12.8 (13.1) cm(3)

40 tion requires notably the proteins SAS-5 and SAS-6, which have functional equivalents across eukaryot

41 Plk4) and its downstream effectors SAS-5 and SAS-6.

42 ner centriole cartwheel components SAS-6 and SAS-5/Ana2/STIL, which then recruit SAS-4/CPAP, which in

43 DSas-4/CPAP-orthologues of ZYG-1, SAS-6, and SAS-4, respectively-are required for centriole duplicati

44 duplication: SPD-2, ZYG-1, SAS-5, SAS-6, and SAS-4.

45 eins (ZYG-1/Sak/Plk4, SAS-6, SAS-5/Ana2, and SAS-4), and is often initiated by the formation of an in

46 SAS-6 constitutes the cartwheel, and SAS-6 levels remain low until centriole assembly is init

47 djacent segment of the SAS-6 coiled coil and SAS-5, prevented SAS-6 recruitment and cartwheel assembl

48 (available as a worksheet, Excel macro, and SAS macro) is a standardized scoring system for making D

49 R, Matlab and SAS/IML code for implementing lossless data reduction is

50 sed on marginal structural models (MSMs) and SAS (SAS Institute, Inc., Cary, North Carolina).

51 Statistical software code in both R and SAS is provided.

52 esponse time (TMS(SAS)), or (4) TMS(SAS) and SAS presented concurrently (TMS+SAS).

53 delay = 35.0 +/- 12.9 ms) when TMS(SAS) and SAS were presented concurrently.

54 available user-friendly programs (Stata and SAS) to implement meta-analysis for dose-response data.

55 Here we show that supercritical antisolvent (SAS) precipitation using carbon dioxide, a process that

56 e to program using standard software such as SAS PROC SURVEYLOGISTIC (SAS Institute Inc., Cary, North

57 xpressed animal centriole components such as SAS-6 are recruited to the SPB.

58 ined with high-throughput functional assays, SAS mutational libraries can expedite the functional ass

59 ures, comprises a flexible loop that assists SAS-6 oligomerisation.

60 tch used by the widely distributed bacterial SAS enzymes.

61 pendent step, whose target is unlikely to be SAS-6, triggers cartwheel assembly.

62 A dimerization interaction between SAS-6 N-terminal "head" domains was previously shown to

63 the presence of hyperelasticity in high-BMI SAS patients was also statistically significant (P < 0.0

64 nificant (P < 0.05) when compared to low-BMI SAS patients.

65 hereby simultaneously fitting models to both SAS data as well as orthogonal information.

66 The dimensions determined by SAS agree well with those obtained by (dried-state) atom

67 ase of targeted ballistic wrist movements by SAS is mediated, in part, by a fast conducting transcort

68 ontractility was unaffected but that cMyBP-C(SAS(t/t)) hearts showed decreased diastolic function at

69 ng wild-type cMyBP-C, the transgenic cMyBP-C(SAS(t/t)), cMyBP-C(ADA(t/t)), and cMyBP-C(DAD(t/t)) mice

70 ning either Ser-273-Ala-282-Ser-302 (cMyBP-C(SAS)), Ala-273-Asp-282-Ala-302 (cMyBP-C(ADA)), or Asp-27

71 A protein called SAS-7 is required for daughter centrioles to become moth

72 (human head and neck squamous cell carcinoma SAS/mouse breast carcinoma 4T1) wound mouse model.

73 apoptosis in human tongue squamous carcinoma SAS cells through mitochondrial pathway.

74 try and electron microscopy, we characterize SAS-6 and show that it self-assembles into stable tetram

75 We provide SAS code (SAS Institute, Inc., Cary, North Carolina) and a simulat

76 SAS code (SAS Institute, Inc., Cary, North Carolina) for implement

77 its the inner centriole cartwheel components SAS-6 and SAS-5/Ana2/STIL, which then recruit SAS-4/CPAP

78 in acute exacerbations of COPD, SGRQ-C, CRQ-SAS, and FEV1 were greater in benralizumab-treated patie

79 e self-administered standardised format (CRQ-SAS), pre-bronchodilator forced expiratory volume in 1 s

80 MS(CT)), (2) SAS 200 ms prior to the go cue (SAS), (3) suprathreshold TMS 70 ms prior to the mean SAS

81 f overfitting the inherently low-dimensional SAS data.

82 efold symmetry, suggesting that two distinct SAS-6 oligomerization architectures can direct the same

83 ZYG-1/Sak/Plk4) and its downstream effectors SAS-5 and SAS-6.

84 , we show that in the Caenorhabditis elegans SAS-6, a segment of the N-terminal globular domain, unre

85 showing that, instead, Caenorhabotis elegans SAS-6 self-assembles into a spiral arrangement.

86 In theory, experimental SAS curves can be reconstituted from a linear combinatio

87 nsembles of model structures to experimental SAS data that rigorously avoids overfitting.

88 Before these tests, subjects received five SAS while standing to verify normal function of the reti

89 the possibilities presented by such flexible SAS-6 segments for the control of centriole formation.

90 g region of Pcp1/pericentrin is critical for SAS-6 interaction.

91 les than males, it represents a hot spot for SAS, offering a refuge for recessive male-beneficial but

92 SAMS is the most frequent SAS, and mild myalgia may affect 5% to 10% of statin use

93 A new user-friendly SAS macro (SAS Institute, Inc., Cary, North Carolina) is

94 resent the Bayesian Ensemble Estimation from SAS (BEES) program.

95 molecular dynamics simulations starting from SAS-6 head domain crystallographic structures, including

96 For 488 genes, SAS were identified downstream of the originally assigne

97 SAS compared with normal flow high gradient SAS (36+/-5% versus 22+/-2% and 38+/-5% versus 21+/-2% f

98 w up compared with normal flow high gradient SAS (adjusted HR 2.17 [1.51-3.13]; P<0.0001 for stroke v

99 tomatic patients with low flow high gradient SAS and preserved left ventricular ejection fraction hav

100 rtality was higher in low flow high gradient SAS compared with normal flow high gradient SAS (36+/-5%

101 -dependent covariate, low flow high gradient SAS displayed considerable mortality risk during follow

102 cal low-gradient (PLG) or high-gradient (HG) SAS.

103 matic HG (mean pressure gradient >=40 mm Hg) SAS with preserved left ventricular ejection fraction.

104 onsistent high-gradient (MG >/=40 mm Hg) (HG-SAS group) and with that of patients with a moderate AS

105 showed better survival in PLG-SAS than in HG-SAS, both in the overall population (48% versus 31%; P<0

106 Patients were categorized into HG-SAS (n=144) and PLG-SAS (n=205) according to mean transv

107 because the majority of them evolve into HG-SAS over time.

108 G-SAS are en route toward the more severe HG-SAS form, because the majority of them evolve into HG-SA

109 associated with improved survival in the HG-SAS group (hazard ratio: 0.18; p = 0.001) and in the PLG

110 r: 64 +/- 4%) compared with patients with HG-SAS (1-year: 96 +/- 1%; 5-year: 82 +/- 3%) or MAS (1-yea

111 a less malignant form of AS compared with HG-SAS, because their spontaneous outcome is better.

112 ording to the AVA, with 187 patients with HG-SAS.

113 ment or the removal of luminal SAS-6 hinders SAS-6 (or centriole) assembly at the outside wall of mot

114 However, how SAS-6 oligomerisation is controlled remains unclear.

115 sults provide mechanistic insights in to how SAS responses are rapidly established by light condition

116 However, SAS data is typically low dimensional and difficult to i

117 erization site of algae, nematode, and human SAS-6 variants, but also that another ligand specificall

118 another ligand specifically recognizes human SAS-6.

119 ons conducive to widespread fire activity in SAS will continue throughout the 21st century.

120 We used Cox proportional hazards analysis in SAS 9.2 survey procedures to estimate associations after

121 gram the previous risk-averaging approach in SAS.

122 the odds of skin lesions using PROC CALIS in SAS, version 9.1 (SAS Institute, Inc., Cary, North Carol

123 Web site, contain implementation examples in SAS software (SAS Institute, Inc., Cary, North Carolina)

124 Hyperelastic floppy eyelid in SAS patients was statistically significant (P < 0.05) wh

125 rovide helpful hints for creating figures in SAS/GRAPH that meet the requirements of the Journal.

126 Floppy eyelid syndrome was more frequent in SAS patients than in normal subjects (P < 0.05), but no

127 its kinase activity-independent function in SAS-6 recruitment.

128 The MCMC procedure in SAS (called PROC MCMC) is particularly designed for Baye

129 tions have shown how the GENMOD procedure in SAS (SAS Institute Inc., Cary, North Carolina) can be us

130 wise elimination (Proc Logistic procedure in SAS).

131 re estimated using PROC GENMOD procedures in SAS 9.4.

132 ASF1 and CAF-1-dependent pathways, including SAS-I- and Rtt109p-dependent acetylation events at H4-K1

133 ilure in worm embryos, indicating that large SAS-5 assemblies are necessary for function in vivo.

134 monstrate that oligomerization of Leishmania SAS-6 can be inhibited by a small molecule in vitro and

135 esent in a human tongue carcinoma cell line, SAS.

136 er the recruitment or the removal of luminal SAS-6 hinders SAS-6 (or centriole) assembly at the outsi

137 The removal or release of luminal SAS-6 requires Plk4 and the cartwheel protein STIL.

138 A SAS macro (SAS Institute Inc., Cary, North Carolina) is given in an

139 A new user-friendly SAS macro (SAS Institute, Inc., Cary, North Carolina) is provided t

140 Here we show that Leishmania major SAS-6 crystallizes as a 9-fold symmetric cartwheel and p

141 ) suprathreshold TMS 70 ms prior to the mean SAS-evoked response time (TMS(SAS)), or (4) TMS(SAS) and

142 gnosis codes in the Veterans Affairs Medical SAS data sets.

143 lyzed data from the Veterans Affairs Medical SAS Datasets and Decision Support System for entire coho

144 ated-measures regression models (PROC MIXED; SAS Institute), adjusted for all other dietary water sou

145 Indeed, multiple SAS subfamilies are encoded in widespread conserved bici

146 on and ciliation can arise in the absence of SAS-6 self-oligomerization.

147 We found that acute administration of SAS at concentrations equivalent to those used to treat

148 l analysis demonstrates the applicability of SAS to monitor complex solution-based self-assembly.

149 ural analysis to uncover the architecture of SAS-5 and examine its functional implications in vivo.

150 Whereas the molecular architecture of SAS-6 and its role in initiating centriole formation are

151 Assembly of SAS-6 dimers to form the centriolar cartwheel requires t

152 934 tweets was performed by a combination of SAS 9.4 for descriptive and inferential statistics inclu

153 ay structure of the amino-terminal domain of SAS-6 from zebrafish, and we show that recombinant SAS-6

154 vide a proof-of-principle that inhibition of SAS-6 oligomerization by small molecules is feasible.

155 d by the observation that elevated levels of SAS-6 in Drosophila cells resulted in higher order struc

156 Management of SAS requires making the possible diagnosis, altering or

157 across hour of sampling using PROC MIXED of SAS with orthogonal contrasts to determine linear and qu

158 necessary to form higher-order oligomers of SAS-5: a trimeric coiled coil and a novel globular dimer

159 Ana2 is the likely functional orthologue of SAS-5 and that it is also related to the vertebrate STIL

160 o structural errors, even in the presence of SAS-6 self-oligomerization.

161 that PpIX-SDT suppress the proliferation of SAS cells via arresting cell cycle at G2/M phase and act

162 he ZYG-1- and SAS-5-dependent recruitment of SAS-6 to the nascent centriole fails.

163 urogenesis in mice by conditional removal of SAS-4, a protein that is required for centriole biogenes

164 al centriole duplication factors upstream of SAS-6 recruitment and procentriole formation.

165 d whether analysis of the genomic context of SASs can indicate possible functional roles.

166 MAP identified a number of SASs that suppressed pS6 (Ser235/236), a marker for acti

167 riole biogenesis does not strictly depend on SAS-6 self-assembly, and may require preexisting centrio

168 Correlation analyses were run on SAS 9.2.

169 exo- Protoporphyrin based SDT (PpIX-SDT) on SAS cells in vitro and in vivo.

170 show that overexpression of either Cad99C or SAS causes a dramatic increase in apical membrane at the

171 Overexpression of Cad99C or SAS results in similar, but distinct effects, suggesting

172 to DM, but evidence linking statins to other SAS is largely anecdotal.

173 pression and apoptosis rate in wild-type p53 SAS cells were found in the SDT group, while p53-mutated

174 However, when the SAS software package (SAS Institute Inc., Cary, North Carolina) is used for an

175 During S phase, SAS-6 molecules are first recruited to the proximal lume

176 were categorized into HG-SAS (n=144) and PLG-SAS (n=205) according to mean transvalvular gradient (me

177 AVA] </=1.0 cm(2)) aortic stenosis (AS) (PLG-SAS group) with that of patients with a severe AS (AVA <

178 urvival curves showed better survival in PLG-SAS than in HG-SAS, both in the overall population (48%

179 Our study indicates that PLG-SAS is a less malignant form of AS compared with HG-SAS,

180 azard ratio: 0.18; p = 0.001) and in the PLG-SAS group (hazard ratio: 0.50; p = 0.04) but not in the

181 /=50%, we identified 187 patients in the PLG-SAS group.

182 ly, at last echocardiographic follow-up, PLG-SAS demonstrated significant increases in mean gradient

183 further demonstrated that patients with PLG-SAS are en route toward the more severe HG-SAS form, bec

184 nt for other risk factors, patients with PLG-SAS had a 1.71-fold increase in overall mortality and a

185 Patients with PLG-SAS had reduced overall survival (1-year: 89 +/- 2%; 5-y

186 on a few conserved proteins (ZYG-1/Sak/Plk4, SAS-6, SAS-5/Ana2, and SAS-4), and is often initiated by

187 invasive ant Nylanderia fulva, a postzygotic SAS leads daughters to preferentially carry alleles from

188 st understory and are shade tolerant prevent SAS when exposed to shade.

189 f the SAS-6 coiled coil and SAS-5, prevented SAS-6 recruitment and cartwheel assembly.

190 arily conserved centriole/basal body protein SAS-4 regulates centriole duplication in metazoa and bas

191 lf-oligomerization of the centriolar protein SAS-6, but how the 9-fold symmetry is invariantly establ

192 mo-oligomerization of the centriolar protein SAS-6, but whether SAS-6 self-assembly can dictate cartw

193 lf-oligomerization of the centriolar protein SAS-6.

194 centriole component, the coiled-coil protein SAS-7, as a regulator of centriole duplication, assembly

195 The highly conserved protein SAS-6 constitutes the center of the cartwheel assembly t

196 The conserved centriole protein, SAS-6, is a cartwheel component that functions early in

197 iole formation requires notably the proteins SAS-5 and SAS-6, which have functional equivalents acros

198 calization of CEP135(full) binding proteins (SAS-6 and CPAP) and the pericentriolar localization of g

199 We provide SAS code (SAS Institute, Inc., Cary, North Carolina) and

200 from zebrafish, and we show that recombinant SAS-6 self-associates in vitro into assemblies that rese

201 AS-6 and SAS-5/Ana2/STIL, which then recruit SAS-4/CPAP, which in turn helps assemble the outer centr

202 Here, we show that ZYG-1 recruits SAS-6 to the mother centriole independently of its kinas

203 cp1/pericentrin, interacts with and recruits SAS-6.

204 that the protein phosphatase PP2A regulates SAS-5 to control centriole duplication.

205 are presented to explain how PP2A regulates SAS-5.

206 o and that inhibiting proteolysis can rescue SAS-5 levels and the centriole duplication defect of PP2

207 n marginal structural models (MSMs) and SAS (SAS Institute, Inc., Cary, North Carolina).

208 have shown how the GENMOD procedure in SAS (SAS Institute Inc., Cary, North Carolina) can be used to

209 p of synthetic acylphloroglucinol scaffolds (SASs).

210 (CES-D), the Zung Self-Rating Anxiety Scale (SAS), and the Child Attitude Toward Illness Scale (CATIS

211 rity Index (ISI), Self-Rating Anxiety Scale (SAS), Self-Rating Depression Scale (SDS), sleep paramete

212 Modern small-angle scattering (SAS) experiments with X-rays or neutrons provide a compr

213 Small-angle scattering (SAS) has a proven ability to detect and characterize sol

214 Small-angle scattering (SAS) measurements are a popular method for characterizin

215 agments called substrate activity screening (SAS) has been applied to the development of low molecula

216 adherin 99C (Cad99C) and Stranded at Second (SAS) - in conferring apical character in Drosophila tubu

217 modified ABC/2, semiautomated segmentation (SAS), fully automatic measurement methods; shape, densit

218 However, sexually antagonistic selection (SAS) may promote diversity by selecting different allele

219 construction of a systematic allelic series (SAS) using massively parallel single-nucleotide mutagene

220 ies; but such "sluggish attention shifting" (SAS) appeared only when dyslexics shifted their attentio

221 An SIS/SAS model of gonorrhea transmission in a population of h

222 ain implementation examples in SAS software (SAS Institute, Inc., Cary, North Carolina) and R languag

223 icles, and Penta-BDE-spiked artificial soil (SAS).

224 ampled into a Statistical Association Space (SAS) to evaluate their dynamic correlation.

225 nd circulates within the subarachnoid space (SAS) of the brain and spinal cord, where it exchanges wi

226 portant molecular events underlying specific SAS responses have been identified.

227 w flow high gradient severe aortic stenosis (SAS) with no or minimal symptoms.

228 30% of patients with severe aortic stenosis (SAS; indexed aortic valve area <0.6 cm(2)/m(2)) present

229 Startling acoustic stimuli (SAS) can accelerate reaction times ("StartReact" effect)

230 released when a startling acoustic stimulus (SAS) is presented immediately prior to, or coincident wi

231 nic material, the surface active substances (SAS), reduced sulphur species (RS) and catalytically act

232 ensuing hyperexcitability by sulfasalazine (SAS), a US Food and Drug Administration-approved drug th

233 rd software such as SAS PROC SURVEYLOGISTIC (SAS Institute Inc., Cary, North Carolina).

234 ted with various statin-associated symptoms (SAS), including statin-associated muscle symptoms (SAMS)

235 dy mass index (BMI) in sleep apnea syndrome (SAS) patients compared to normal subjects.

236 ponse known as the shade avoidance syndrome (SAS).

237 ocess known as the shade avoidance syndrome (SAS).

238 ons known as the "shade-avoidance syndrome" (SAS).

239 at the tetrameric small alarmone synthetase (SAS) RelQ from the Gram-positive pathogen Enterococcus f

240 Single-domain small alarmone synthetases (SASs) are RSH family members that contain the (p)ppGpp s

241 We conclude that SAS-7 functions at the earliest step in centriole duplic

242 Our results firmly establish that SAS-6 can impose cartwheel symmetry on its own and indic

243 Here, we found that SAS-6 assembly can be shaped by preexisting (or mother)

244 We also show that SAS-7 binds SPD-2 and regulates SPD-2 centriolar recruit

245 These results suggest that SAS-6 self-assembly may be an initial step in the format

246 Intriguingly, our genetic data suggest that SAS-7 is required for daughter centrioles to become comp

247 Sequence analysis suggests that SAS-6 spirals are restricted to specific nematodes.

248 The SAS Macro for our empirical Bayes test for periodicity i

249 y a direct interaction between ZYG-1 and the SAS-6 coiled coil that explains its kinase activity-inde

250 lecular-weight ligands predicted to bind the SAS-6 head domain and inhibit protein oligomerization.

251 found that the probability densities in the SAS increase as the peaks in two cues are approached.

252 f the high probability density region in the SAS suggests a nonlinear correlation between two cues.

253 This study introduces the SAS/MCMC procedure and demonstrates the application of t

254 Our approach prevents over-fitting of the SAS data and can be used with a newly defined metric, RS

255 teraction between an adjacent segment of the SAS-6 coiled coil and SAS-5, prevented SAS-6 recruitment

256 ic activity are abolished by deletion of the SAS-specific C-terminal helix 5alpha.

257 inding to the SAS-6 coiled coil recruits the SAS-6-SAS-5 complex to the mother centriole, where a ZYG

258 erminal dimerisation interface stabilise the SAS-6 oligomer.

259 Here, we report that the SAS-4 homolog in the flagellated protozoan Trypanosoma b

260 capable of distributing rapidly through the SAS along the entire neuraxis with reproducible, anatomi

261 cles were also capable of moving through the SAS but did not achieve as widespread distribution.

262 We propose that ZYG-1 binding to the SAS-6 coiled coil recruits the SAS-6-SAS-5 complex to th

263 the Yale Open Data Access Project using the SAS Clinical Trials Data Transparency platform.

264 dependent, unblinded, statistician using the SAS procedure Proc Plan.

265 tion of N-glycosylation efficiency using the SAS software, employing the 120 sequences studied as a t

266 However, when the SAS software package (SAS Institute Inc., Cary, North Ca

267 We have validated five of these SASs as being toxic (toxSASs), with neutralization by th

268 iation of volumetric in-vitro tumour tissue (SAS spheroids) to demonstrate concurrent operation of la

269 -evoked response time (TMS(SAS)), or (4) TMS(SAS) and SAS presented concurrently (TMS+SAS).

270 or to the mean SAS-evoked response time (TMS(SAS)), or (4) TMS(SAS) and SAS presented concurrently (T

271 , average delay = 35.0 +/- 12.9 ms) when TMS(SAS) and SAS were presented concurrently.

272 TMS(SAS) and SAS presented concurrently (TMS+SAS).

273 ar association of hyperelasticity and FES to SAS patients but no association between obesity and FES.

274 ed mother centrioles becomes inaccessible to SAS-6, correlating with a block for reduplication.

275 owth-promoting molecular pathways leading to SAS However, it is unknown how plants that complete thei

276 CEP135 physically links to SAS-6 near the site of microtubule nucleation and binds

277 presence of FES was estimated in relation to SAS and BMI.

278 les, which theoretically predisposes them to SAS in large parts of their genome.

279 can be difficult to incorporate into typical SAS modeling workflows, especially for users that are no

280 We used SAS survey procedures to plot distributions of TBI and p

281 Data were analysed using SAS 9.1.3.

282 was performed as the primary analysis using SAS v9.

283 Data were analyzed using SAS statistical software.

284 om 1 310 727 examinations (analyzed by using SAS 9.3) provided median values, as well as means and 25

285 out by fitting linear mixed models by using SAS PROC MIXED.

286 home zip code to the transplant center using SAS URL access to GoogleMaps.

287 Armitage trend analyses were conducted using SAS 9.2.

288 Analyses were conducted using SAS-callable SUDAAN to correct for complex sampling desi

289 Our analysis was performed using SAS 9.3 and Stata 12.

290 All analyses were performed using SAS software.

291 , methods for evaluating mass and validating SAS-based models and resolution have been inadequate.

292 of the centriolar protein SAS-6, but whether SAS-6 self-assembly can dictate cartwheel and thereby ce

293 are well understood, the mechanisms by which SAS-5 and its relatives function is unclear.

294 egulates SPD-2 centriolar recruitment, while SAS-7 centriolar localization is SPD-2-independent.

295 Participants were randomly assigned with SAS (version 9.2, block size 2-9) in a 1:1 ratio, strati

296 We show that PP2A physically associates with SAS-5 in vivo and that inhibiting proteolysis can rescue

297 total of 135 participants (81 patients with SAS and 54 normal subjects) had a full ophthalmologic ex

298 We prospectively studied 349 patients with SAS and preserved left ventricular ejection fraction.

299 Analyses were performed with SAS PROC MIXED.

300 ng to ordinary least squares regression with SAS procedures for multiple imputation and analysis of c