ライフサイエンスコーパス: SBP

1 SBP goals of <140 and < 160mmHg following SR with EVT ap

2 SBP was also associated positively with all-cause, diabe

3 SBP was lowered and UtA endothelial function was enhance

4 SBP was significantly reduced by exercise over 8 months

5 SBP-7455 inhibited starvation-induced autophagic flux in

6 nths who met treatment targets (HbA1c <7.0%, SBP <130 mm Hg, LDL cholesterol <100 mg/dL [<70 mg/dL if

7 MI (-0.30, -0.39 to -0.20 Kg/m2, P < 0.001), SBP (-1.43, -1.70 to -1.16 mm Hg, P < 0.001), and smokin

8 iable adjustment, baseline and mean achieved SBP of 120 to 129 mm Hg demonstrated the lowest risk for

9 Baseline and mean achieved SBP of 120 to 129 mm Hg identified the lowest risk patie

10 ated baseline and time-updated mean achieved SBP quartiles (<120, 120 to 129, 130 to 139, >=140 mm Hg

11 udy sought to determine the optimal achieved SBP and whether the treatment effects of sacubitril/vals

12 Similarly, young adult SBP >=130 mm Hg (compared with <120 mm Hg) was associate

13 organic nitrate does not decrease ambulatory SBP in subjects with elevated BP.

14 the difference in change in 24 h ambulatory SBP between the groups.

15 The insignificant change in ambulatory SBP (mean +/- standard deviation) was -0.6 +/- 6.2 mm Hg

16 y patients with pre-EVT SBP of >=140mmHg, an SBP of <140mmHg was associated with a higher likelihood

17 ary, our results confirm the existence of an SBP pathway for pentose assimilation in cellulolytic clo

18 Among hypertensive adults, targeting an SBP of less than 120 mm Hg, compared with less than 140

19 Differences in plasma LDL-C and SBP compared with participants with both genetic scores

20 followed up through 2018, genetic LDL-C and SBP scores were used as instruments to divide participan

21 MIA trial with baseline and 1-year LDL-C and SBP values by January 28, 2019.

22 oncentrations in patients with cirrhosis and SBP indicate reduced odds of surviving for 90 days.

23 ients with decompensated liver cirrhosis and SBP.

24 ing data from 71 patients with cirrhosis and SBP.

25 In non-MetS, strategies to control HbA1c and SBP should be prioritised as these have the largest impa

26 netic risk scores and lower LDL-C levels and SBP was associated with dose-dependent lower risks of ma

27 onal multivariable analyses of mortality and SBP are not substantially confounded by reverse causatio

28 relationships governing both SBP-surface and SBP-SBP interactions and how they give rise to different

29 of aorta (COA), we hypothesized that for any SBP, patients with mild COA (COA peak velocity <2 m/s) w

30 During follow-up, average SBP was 135 mm Hg (125-145).

31 d the effectiveness of fluoroquinolone-based SBP prophylaxis in an era and area of frequent antibioti

32 Baseline SBP did not modify the treatment effect of sacubitril/va

33 lic BP adjusted for medications and baseline SBP, (ii) medication burden, and (iii) safety.

34 Average baseline SBP was 159.4 mm Hg.

35 primary outcome was not modified by baseline SBP (interaction p = 0.50) and was similar when adjustin

36 4 weeks, which was not modified by baseline SBP.

37 attainment were greater with lower baseline SBP (OR, 1.27 [95% CI, 1.22-1.33] per 10 mm Hg) and with

38 DL-C goal attainment, whereas lower baseline SBP and North American location predicted 1-year SBP goa

39 e independently associated with death before SBP (subdistribution hazard risk, 6.47; P = .034).

40 the study assessed the relationship between SBP change and Kansas City Cardiomyopathy Questionnaire

41 rely on the sedoheptulose 1,7-bisphosphate (SBP) pathway, using pyrophosphate-dependent phosphofruct

42 the underlying relationships governing both SBP-surface and SBP-SBP interactions and how they give r

43 e the magnitude of the shift in systolic BP (SBP) among Blacks and Whites from the Southeast between

44 er 2 sequential visits for both systolic BP (SBP) and diastolic BP (DBP), and further assessed the di

45 sal relationship exists between systolic BP (SBP) and/or diastolic BP (DBP) and risk of Alzheimer's d

46 mary outcome was mean change in systolic BP (SBP) at 12 months.

47 rtension: >=90th percentile for systolic BP (SBP) or diastolic BP (DBP).

48 sive individuals with different systolic BP (SBP) values.

49 nkage to care; and 2) change in systolic BP (SBP).

50 ivided into groups according to systolic BP (SBP): G1 (n = 16), resting SBP <110 mmHg and G2 (n = 14)

51 = 0.11, P = 3.56 x 10(-06)) and systolic BP (SBP, r(g) = 0.06, P = 0.01), but not pulse pressure (PP,

52 a greater reduction in 24-hour systolic BP (SBP; from 138 to 124 mm Hg) compared with sodium restric

53 f the observed effect of WHR was mediated by SBP for ischemic stroke (proportion mediated: 12%, 95% C

54 OppA is a member of the Cluster C SBP family, and unlike other SBP families, some members

55 Differences in plasma LDL-C, SBP, and cardiovascular event rates between the groups w

56 is Importantly, we found that one chlamydial SBP, OppA3 (Ct) , possessed dual substrate recognition p

57 RCTs reported improved TC, HDL cholesterol, SBP, and HOMA-IR.

58 tudies, improvements in TC, HDL cholesterol, SBP, DBP, HOMA-IR, and acute/chronic FMD remained signif

59 which fructose-bisphosphate aldolase cleaves SBP into dihydroxyacetone phosphate and erythrose 4-phos

60 We show that a unique closed SBP conformation does not exist for transported substrat

61 o 4 groups and were offered diets containing SBP at the level of 0%, 3%, 5%, and 7%.

62 A decrease in PWV(CF) , PWV(CR) , SBP and DBP (-25%, -17%, -4% and -8%, respectively; P <

63 Furthermore, we demonstrated that, in Dab2 SBP, R42 significantly contributes to the inhibition of

64 The Dab2 SBP residues that interact with sulfatides resemble thos

65 nal family history of AD UK Biobank dataset (SBP [beta(GSMR) = -0.12, p = .02], DBP [beta(GSMR) = -0.

66 nal family history of AD UK Biobank dataset (SBP [beta(GSMR) = -0.16, p = .02], DBP [beta(GSMR) = -0.

67 DBP/SBP of reference charts for all women and for each ethni

68 low-salt diets also significantly decreased SBP and DBP levels.

69 tional and safety outcomes between different SBP goals after EVT with SR.

70 s was used to assess the effect of different SBP goals on clinical outcomes.

71 variation), and 3,805 (68%) had a discharge SBP <130 mm Hg.

72 During SBP, peritoneal MAIT cell frequencies increased most amo

73 In contrast, adding either SBP, non-HDL-C, diabetes mellitus, or smoking to a model

74 nalysis including only patients with pre-EVT SBP of >=140mmHg, an SBP of <140mmHg was associated with

75 s more prevalent in patients who experienced SBP excursions at least 20% above the individual referen

76 lic blood pressure, upper-to-lower-extremity SBP gradient, aortic isthmus ratio, presence of collater

77 Laying hens fed SBP had lower (P < 0.01) serum total lipids, cholesterol

78 For SBP-dated primate-specific protein-coding genes (PSGs),

79 population attributable fractions of 28% for SBP>=130 mm Hg and 17% for non-HDL-C>=130 mg/dL.

80 goal increased to 52% for LDL-C and 75% for SBP.

81 obesity duration was largely attenuated for SBP, DBP, and HDL-C.

82 at goal for LDL-C, and 65% were at goal for SBP at baseline.

83 erences ranging from -3.20 to -7.62 mmHg for SBP and from -2.50 to -4.22 mmHg for DBP.

84 (increase in mean WMHV per 10 mm Hg greater SBP 7%, 95% CI 1-14, p=0.024; increase in mean WMHV per

85 ); percentage of patients who met all HbA1c, SBP, and LDL cholesterol targets; and mean reductions in

86 In non-MetS, higher HbA1c, SBP, and number of MetS components were linked to poorer

87 Patient Health Questionnaire-9 score, HbA1c, SBP, and LDL cholesterol.

88 mong hospitalized older patients with HFrEF, SBP <130 mm Hg is associated with poor outcomes.

89 mbination of PP 60 mm Hg or greater and high SBP of 140 mm Hg or greater showed the strongest associa

90 in patients with out-of-target low and high SBP.

91 ored graft survival, the combination of high SBP and high PP showed the best correlation across all a

92 ataset revealed a significant effect of high SBP lowering the risk of AD (beta(GSMR) = -0.19, p = .04

93 ounders and child height, we observed higher SBP in children exposed to gestational diabetes mellitus

94 ; increase in mean WMHV per one SD change in SBP 15%, 3-29, p=0.012; increase in mean WMHV per 1 SD c

95 Change in SBP was directly associated with change in NT-proBNP (p

96 DC values correlated modestly with change in SBP, but not in renal hypoxia, TNF-alpha levels, or rena

97 of fruit/vegetable leads to 3-6% decrease in SBP; or, a 10% increase in cereal intake lowers SBP by 3

98 ) was assessed as the absolute difference in SBP divided by the mean over two sequential visits every

99 nths, there was no significant difference in SBP reduction from baseline in the THRIVES versus contro

100 nsated cirrhosis and are further enriched in SBP.

101 CI 2.11-5.18], P < 0.001) and large falls in SBP (HR for the lowest quintile, 2.20 [95% CI 1.33-3.63]

102 risk was only associated with large falls in SBP (HR, 1.21 [95% CI 1.00-1.48], P = 0.017).

103 observed with both large rises and falls in SBP.

104 t 0.5 percentage points in HbA1c, 5 mm Hg in SBP, or 10 mg/dL in LDL cholesterol.

105 not statistically significant improvement in SBP reduction.

106 ment provides new evidence of improvement in SBP, suggesting that strategies and programs implemented

107 Every 10 mmHg increase in SBP>125 mmHg was associated with an increased risk of MA

108 BP) at age 53 years and greater increases in SBP and DBP between 43 and 53 years were positively asso

109 re was reduced in all 3 groups: -5.1 mmHg in SBP (95%CI -10.1, 0.0; p = 0.003) in AT; -4.0 mmHg in SB

110 I -10.1, 0.0; p = 0.003) in AT; -4.0 mmHg in SBP (95%CI -7.8, -0.5; p = 0.027) in RT; and -3.2 mmHg i

111 ce showed benefit of a 10-mm Hg reduction in SBP for cardiovascular outcomes among patients with hype

112 ntensive lowering to a 10-mm Hg reduction in SBP for cardiovascular outcomes in patients with a histo

113 experienced a modestly greater reduction in SBP versus usual care (-13.1 mm Hg vs. -9.7 mm Hg), but

114 t reduction in HbA1c, >=5-mm Hg reduction in SBP, >=10-mg/dL reduction in LDL cholesterol); percentag

115 l an unrecognized diversity of plasticity in SBPs.

116 , whereas absolute risk reductions increase (SBP: 1.1%, 2.3%, 5.4%, 10.3%, respectively; non-HDL-C: 1

117 One inhibitor, SBP-7455 (compound 26), displayed improved binding affin

118 Instead, SBPs sample a range of conformations that activate trans

119 x interplay between ligand-SBP interactions, SBP conformational dynamics and substrate transport.

120 A large SBP variation (top vs. bottom tertiles), measured on ave

121 A large SBP variation was also associated with the progression o

122 A large SBP variation was associated with an increased dementia

123 reveal the complex interplay between ligand-SBP interactions, SBP conformational dynamics and substr

124 High but not low SBP was associated with an increased risk of ischemic li

125 diabetes mellitus (high strength) to a lower SBP target was mixed.

126 with lifetime exposure to lower LDL-C, lower SBP, or both.

127 oximately linear, supporting calls for lower SBP targets.

128 Compared to WB, P women had lower SBP/DBP at 12, 20 and 37 weeks gestation.

129 s higher than the median had 2.9-mm Hg lower SBP and an OR of 0.82 for major coronary events (95% CI,

130 had 13.9-mg/dL lower LDL-C, 3.1-mm Hg lower SBP, and an OR of 0.61 for major coronary events (95% CI

131 ; or, a 10% increase in cereal intake lowers SBP by 3%; a simultaneous increase of 10% in fruit-veget

132 At GD17.5, maternal SBP was measured; at GD18.5, maternal nitrate/nitrite co

133 -11.7 years; 93.5% had hypertension and mean SBP was 138.33 (23.64) mm Hg.

134 Patients with higher pre-procedural mean SBP had a greater chance of a good outcome (p = 0.03).

135 hile control group showed a significant mean SBP (diastolic BP) decrease of 11.2 (7.9) mm Hg at 12 mo

136 g (n=168), THRIVES showed a significant mean SBP (diastolic BP) decrease of 11.7 (7.0) mm Hg while co

137 ntinuous intra-procedural increase of median SBP (+11%) and mean arterial pressure (MAP, +10%, both p

138 ong Whites 45 to 54 years of age, the median SBP was 18 mm Hg (95% CI, 16-21 mm Hg) lower in 2005 tha

139 Every decrease in 10 mmHg SBP <=125 mmHg was associated with an increased risk of

140 ntracellular pathogens often encode multiple SBPs, while only one, OppA, is encoded in the E. coli op

141 r OppABCDF (OppABCDF (Ct) ) encodes multiple SBPs (OppA1 (Ct) , OppA2 (Ct) , and OppA3 (Ct) ).

142 cooperative interactions between neighboring SBPs and higher order structure formation.

143 mm Hg (95% CI: 0.48, 2.92)), higher neonatal SBP (per 10-mm Hg increase; age 3 years: beta = 1.26 mm

144 blood pressure [DBP] >=90 mm Hg) and normal (SBP <140 mm Hg and DBP <90 mm Hg) blood pressure.

145 Elastance index (but not SBP) was predictive of longitudinal increases in LVMI (r

146 h longer intervals between the assessment of SBP variation and the diagnosis of dementia.

147 is study sought to determine associations of SBP <130 mm Hg with outcomes in patients with HFrEF.

148 s of these results for the de novo design of SBP-surface binding systems.

149 -vegetable can further offset the effects of SBP by 6%.

150 se causation and confirm positive effects of SBP on all-cause, CVD and diabetes mortality.

151 Heritability of SBP and DBP ranged from 16.8% to 40.4% for daytime, slee

152 After the immobilization of SBP-tagged PAS1 to the sensing layers, PAS1-based SIS se

153 Dietary inclusion of SBP could improve hen performance, health, egg quality,

154 Dietary inclusion of SBP linearly (P < 0.01) decreased egg yolk malondialdehy

155 The dietary inclusion of SBP linearly (P < 0.01) increased feed intake, egg produ

156 tudy meta-analysis of 299,024 individuals of SBP or DBP as exposure variables against three different

157 r at least 15 years since the measurement of SBP variation.

158 nd REGARDS (n=6294) with the main outcome of SBP distribution.

159 The 95th percentile of SBP decreased 60 mm Hg for Whites and 70 mm Hg for Black

160 revealed loss of LPMs in the early phase of SBP, but levels increased after treatment.

161 Antibiotic prophylaxis of SBP appears to be less efficient in patients with known

162 ion for fluoroquinolone-based prophylaxis of SBP.

163 Pooled reductions of SBP were -4.38 mm Hg (95% CI, -7.27 to -2.16) for angiot

164 G2 displayed slower return of SBP, rMSSD and SD1 HRV indices during recovery from exer

165 Significantly higher risks of SBP development during antibiotic prophylaxis were obser

166 -1.34, P = 0.337) for risk within 5 years of SBP variation measurement to 3.13 (95% CI 2.05-4.77; P <

167 lots of parental mortality against offspring SBP were approximately linear, supporting calls for lowe

168 provide evidence of programming of offspring SBP trajectories by gestational diabetes, hypertensive d

169 Patients were divided into 3 groups based on SBP goal in the first 24 hours after EVT.

170 tihypertensive therapy is currently based on SBP.

171 inversely correlated with genetic effects on SBP, mood instability and neuroticism.

172 omized controlled trials to evaluate optimal SBP reduction goals in patients with HFrEF.

173 However, the optimal SBP goal following EVT is still unknown.

174 the overall significant effect for the other SBP analyses (beta(GSMR) = -0.14, p = .03).

175 f the Cluster C SBP family, and unlike other SBP families, some members recognize two distinctly diff

176 en) as an instrumental variable for parental SBP and examined associations with parents' cause-specif

177 protein isolate (PPI) and sugar beet pectin (SBP) at concentrated solutions (~2.0 wt%).

178 n fused to the streptavidin-binding peptide (SBP) and (ii) motor, neck, and coiled-coil domains from

179 ng approach based on silica binding peptide (SBP) for direct immobilization of PAS1 on the SiO(2) sur

180 e refer to as the sulfatide-binding peptide (SBP), contains two potential sulfatide-binding motifs re

181 Solid-binding peptides (SBPs) recognizing inorganic and synthetic interfaces hav

182 me (SIRS), spontaneous bacteria peritonitis (SBP), and pneumonia; and O: the CLIF consortium organ fa

183 o prevent spontaneous bacterial peritonitis (SBP) in patients colonized with multidrug-resistant orga

184 ents with spontaneous bacterial peritonitis (SBP).

185 r without spontaneous bacterial peritonitis (SBP).

186 ds revealed that the synteny-based pipeline (SBP) is most suited for recently duplicated genes, where

187 l biomicroscopy of corneal sub-basal plexus (SBP).

188 heast confirm the improvements in population SBP levels since 1960.

189 ationship between such a spiking-band power (SBP) and neural activity remains unclear, as does the ca

190 The effects of pH (7-2) and PPI-SBP mixing ratios (1:1-20:1) on coacervates formation we

191 ndent of pH(phi1) increased to pH 5.5 as PPI-SBP mixing ratio increased to 20:1.

192 recommend targeting systolic blood pressure (SBP) <130 mm Hg in heart failure with preserved ejection

193 .3, SD 5.6, Kg/m2), systolic blood pressure (SBP) (cases 129.0, SD 14.3; controls 129.3, SD 15.0, mm

194 Elevated systolic blood pressure (SBP) after successful revascularization (SR) via endovas

195 ase was mediated by systolic blood pressure (SBP) and blood glucose levels, respectively.

196 Systolic blood pressure (SBP) and diastolic blood pressure (DBP) are important pr

197 Higher systolic blood pressure (SBP) and diastolic blood pressure (DBP) at age 53 years

198 s all participants, systolic blood pressure (SBP) and fasting blood glucose were also contributors, a

199 ere randomized to a systolic blood pressure (SBP) goal of either less than 120 mm Hg (intensive treat

200 ines recommend that systolic blood pressure (SBP) in patients with heart failure with reduced ejectio

201 1c) of at least 8%, systolic blood pressure (SBP) of at least 140 mm Hg, or low-density lipoprotein (

202 drop >= 20 mm Hg in systolic blood pressure (SBP) or >= 10 in diastolic blood pressure (DBP) upon sta

203 An individual systolic blood pressure (SBP) reference value was defined as the median of the fi

204 solute variation in systolic blood pressure (SBP) was assessed as the absolute difference in SBP divi

205 rtension and office systolic blood pressure (SBP) with major adverse cardiovascular events (MACEs) an

206 l (LDL-C) and lower systolic blood pressure (SBP) with the risk of cardiovascular disease has not bee

207 pecifically lipids, systolic blood pressure (SBP), diabetes mellitus, and smoking-with incident CHD e

208 The systolic blood pressure (SBP), diastolic blood pressure (DBP) and mean arterial p

209 ilability to reduce systolic blood pressure (SBP), improve vascular function and increase fetal growt

210 m creatinine (SCr), systolic blood pressure (SBP), renal hypoxia, and renal vein levels of pro-inflam

211 ested produced a decrease in blood pressure (SBP).

212 tal-cholesterol and systolic blood pressure (SBP).

213 ut of proportion to systolic blood pressure (SBP).

214 goal <70 mg/dL) or systolic blood pressure (SBP, goal <140 mm Hg) at 1 year post-randomization.

215 a of baseline high (systolic blood pressure [SBP] >=140 mm Hg or diastolic blood pressure [DBP] >=90

216 oglycan-recycling substrate binding protein (SBP) MppA, which is responsible for recycling peptidogly

217 pA) serves as the substrate-binding protein (SBP) of the oligopeptide transport system responsible fo

218 is a periplasmic substrate binding protein (SBP).

219 Substrate-binding proteins (SBPs) are associated with ATP-binding cassette importers

220 The effect of including sugar beet pulp (SBP) in laying hen diets on performance, egg quality, bl

221 We coexpressed the putative SBP genes (oppA1(Ct) , oppA2(Ct) , oppA3(Ct) ) along wit

222 Eggs laid by hens receiving SBP had linearly (P < 0.01) greater protein and lower et

223 s, and 13 cases (16.9%) of de novo/recurrent SBP.

224 ES intervention did not significantly reduce SBP compared with controls.

225 Candesartan reduced SBP by -6.56 mm Hg (P < .001; n = 240).

226 Sacubitril/valsartan reduced SBP by 5.2 mm Hg (95% confidence interval: 4.4 to 6.0) c

227 However, sacubitril/valsartan reduced SBP more in women (6.3 mm Hg) than men (4.0 mm Hg) (inte

228 Evidence on reducing SBP for cardiovascular outcomes in patients with a histo

229 more BP excursion of 20% below the reference SBP and required more frequent use of sympathomimetic dr

230 sting SBP <110 mmHg and G2 (n = 14), resting SBP between 120-110 mmHg.

231 g to systolic BP (SBP): G1 (n = 16), resting SBP <110 mmHg and G2 (n = 14), resting SBP between 120-1

232 n, normotensive subjects with higher resting SBP (110 to 120 mmHg) offered delayed autonomic recovery

233 Forty-two participants (54 +/- 11 y, resting SBP/DBP 137 +/- 9/86 +/- 6 mmHg) were randomly allocated

234 Here we used a son's SBP (>1 million Swedish men) as an instrumental variable

235 C. thermosuccinogenes contains a significant SBP pool, an unusual metabolite that is elevated during

236 Intriguingly, in some cases, similar SBP conformations are formed by both transported and non

237 Despite similar SBP, the mild COA group displayed higher arterial afterl

238 fterload compared with controls with similar SBP.

239 Similarly, SBP goal of <160mmHg was associated with lower odds of m

240 trates on the conformational dynamics of six SBPs and the impact on transport.

241 targeted SQUAMOSA PROMOTER BINDING-LIKE (SPL/SBP) transcription factors by activating SINGLE FLOWER T

242 Of these, 5,615 had stable SBP (<=20 mm Hg admission to discharge variation), and 3

243 ionally associated with age, smoking status, SBP and refractive error; and ISOS-RPE was additionally

244 Importantly, competition of the streptavidin-SBP interaction by the addition of biotin to the culture

245 actors collected in mid-adulthood: systolic (SBP) and diastolic blood pressure (DBP), high-density-li

246 and CT on endothelial function and systolic (SBP)/diastolic blood pressure (DBP) in individuals with

247 Heart rate (HR), systolic (SBP) and diastolic blood pressure (DBP), rate-pressure p

248 ta-analysis assessed reductions in systolic (SBP) and diastolic blood pressure from pharmacological t

249 the effect of dietary patterns on systolic (SBP) and/or diastolic blood pressure (DBP) levels.

250 wave velocity (PWV), together with systolic (SBP) and diastolic (DBP) blood pressure.

251 0 mmHg; P = 5.57 x 10(-25)) on migraine than SBP (1.05 [1.03-1.07]/10 mmHg; P = 2.60 x 10(-07)) and a

252 ssociated with better clinical outcomes than SBP of <180mmHg.

253 itself stabilises the closed state and that SBP closure is triggered by physically bridging the gap

254 based on published PSG lists and showed that SBP generated a conservative data set of PSGs by masking

255 These data suggest that SBP may underestimate LV afterload in this population.

256 The SBP may enhance the decoding performance of neural inter

257 In the order "A", the SBP and DBP increased at the midpoint of the session.

258 On IPTW analysis, the SBP goal of <140mmHg was associated with a higher likeli

259 In the order "B", the SBP and DBP increased only immediately after the end of

260 showed the most significant reduction in the SBP (42 +/- 2 mmHg and 35 +/- 2 mmHg, respectively).

261 Similarly, lowering the SBP of all individuals to <130 mm Hg or lowering low-den

262 nding induces a conformational change of the SBP and it is thought that this closed state is recognis

263 eptides play a role in the regulation of the SBP by acting on plasma ACE, plasma renin and the vascul

264 or transport arises from slow opening of the SBP or the selectivity provided by the translocator.

265 ridging the gap between the two lobes of the SBP.

266 Without substrate, the SBP has been proposed to rest in an open-state, which is

267 ine two-dimensional cursor-control task, the SBP performed equally well or better than the TCR.

268 or better than that of the TCR, and that the SBP correlates better with the firing rates of lower sig

269 gs of neural activity, here we show that the SBP is dominated by local single-unit spikes with spatia

270 unclear, as does the capability of using the SBP to decode complicated behaviour.

271 n- to closed-state transition of VcSiaP, the SBP of the sialic acid TRAP transporter from V. cholerae

272 ents were divided into 4 groups according to SBP (high >=120 mm Hg, low <120 mm Hg) and DBP (high >70

273 ted with lower odds of mortality compared to SBP goal of <180mmHg.

274 requirement for hemicraniectomy compared to SBP goal of <180mmHg.

275 nd lower odds of hemicraniectomy compared to SBP goal of <180mmHg.

276 In comparison to SBP, Doppler-derived arterial load indices correlate mor

277 revealed that linkage to care contributed to SBP change.

278 stimated the risk of dementia in relation to SBP variation measured at different time windows (i.e.,

279 tolic blood pressure gave similar results to SBP.

280 idium thermocellum indeed can convert S7P to SBP, and have similar affinities for S7P and the canonic

281 converts sedoheptulose 7-phosphate (S7P) to SBP, after which fructose-bisphosphate aldolase cleaves

282 Average overall follow-up SBP was 149.9 mm Hg.

283 was similar when adjusting for time-updated SBP, regardless of sex.

284 tients with decompensated cirrhosis (19 with SBP) and analyzed them by flow cytometry, quantitative r

285 tients with decompensated cirrhosis (67 with SBP) and quantified the soluble form of the mannose rece

286 nd HF readmission at 1 year, associated with SBP <130 mm Hg, were 1.32 (1.15 to 1.53; p < 0.001), 1.1

287 d 1-year all-cause mortality associated with SBP 110 to 129 mm Hg (vs. >=130 mm Hg) were 1.50 (1.03 t

288 Compared with SBP (beta=0.24 [95% CI, 0.02-0.45]), elastance index (be

289 be a better predictor of LVMI compared with SBP alone.

290 GSH levels correlated negatively with SBP, DBP and MBP values in all participants (p = 0.0010;

291 Participants with SBP genetic scores higher than the median had 2.9-mm Hg

292 were repeated after excluding patients with SBP <110 mm Hg.

293 curred in 7% and 4% of matched patients with SBP <130 mm Hg versus >=130 mm Hg, respectively (hazard

294 e test and validation cohorts, patients with SBP and higher concentrations of soluble CD206 in ascite

295 s of serial ascites fluid from patients with SBP revealed loss of LPMs in the early phase of SBP, but

296 he levels of GSSG correlated positively with SBP, DBP and MBP values in all participants (p = 0.0410;

297 38) and a longer duration of time spent with SBP above 180 mmHg (p = 0.029).

298 identifies the critical Dab2 residues within SBP that are responsible for sulfatide binding.

299 Adjusted odds of 1-year SBP goal attainment were greater with lower baseline SBP

300 and North American location predicted 1-year SBP goal attainment.