戻る
「早戻しボタン」を押すと検索画面に戻ります。 [閉じる]

コーパス検索結果 (left1)

通し番号をクリックするとPubMedの該当ページを表示します
1                                              SHFV encodes three more ORFs on its genome than the othe
2                                              SHFV is a member of a new virus family which includes th
3                                              SHFV is unique among arteriviruses in having three N-ter
4                                              SHFV PLP1gamma is able to cleave at both downstream and
5                                              SHFV replicated in >90% of macaque MPhis but in only app
6 acilitated by next-generation sequencing, 96 SHFV body TRSs were identified that were functional in b
7               However, little is known about SHFV's ecology and molecular biology and the mechanism b
8 han macaque cultures both prior to and after SHFV infection.
9 ultures with human IL-10 before and/or after SHFV infection decreased production of IL-6, IL-1beta, a
10                                     Although SHFV infected approximately 50% of macaque and baboon mD
11 ggest that SG formation is not induced by an SHFV infection due to recruitment of G3BP to sites of vi
12  similar positions in the LDV-C 3'(-)NCR and SHFV 3'(-)209 RNAs.
13 g patterns were obtained with uninfected and SHFV-infected extracts, indicating that the four protein
14 only in cis at a single downstream site, but SHFV PLP1gamma can cleave at both the downstream nsp1gam
15 and in Alamogordo in 1989 were caused not by SHFV but by two novel divergent arteriviruses.
16     In baboon but not macaque cell cultures, SHFV infection upregulated IL-10R1, a subunit of the IL-
17 e and respiratory syndrome virus, diminished SHFV replication, identifying CD163 as an important SHFV
18                                       During SHFV infection, we detected signatures of selection on O
19 y mechanism and expanded coding capacity for SHFV, which may also be characteristic of other nidoviru
20 munoprecipitating with endogenous G3BP1 from SHFV-infected cell lysates detected multiple viral repli
21                                     Further, SHFV enters and replicates in human monocytes, indicatin
22  The results of this study shed light on how SHFV enters its target cells.
23 plication, identifying CD163 as an important SHFV entry component.
24 enuated virus growth ability was observed in SHFV mutants with impaired expression of nsp2TF and nsp2
25 y sequence, and C-rich motif in -2/-1 PRF in SHFV-infected cells.
26                                    All known SHFV variants are monophyletic and share three open read
27 tion reactions done with wild-type or mutant SHFV nsp1 constructs.
28                          We describe two new SHFV variants subclinically infecting wild African red-t
29  the in vitro reactions, Western blotting of SHFV-infected, MA104 cell lysates with SHFV nsp1 protein
30 Cys63 was identified as the catalytic Cys of SHFV PLP1alpha and is adjacent to an Ala instead of the
31      Even though intracellular production of SHFV RNA activates the stress sensor, PKR, SGs are not i
32 between G3BP1 and the nsp2 and N proteins of SHFV was observed in reciprocal co-immunoprecipitation a
33 fferential infection outcome, the targets of SHFV infection, macrophages (MPhis) and myeloid dendriti
34     Phospholipases A2 and D had no effect on SHFV entry.
35 n contrast, we detected selection focused on SHFV ORFs 5a and 5, which encode putative membrane prote
36 ine, and concanamycin A dramatically reduced SHFV entry efficiency, whereas the macropinocytosis inhi
37 , suggesting a role for IL-10 in suppressing SHFV-induced proinflammatory cytokine production in maca
38  of in vitro autoprocessing of an N-terminal SHFV nonstructural 1a polypeptide fragment showed that e
39 rious cellular pathways, we demonstrate that SHFV invades cells by low-pH-dependent, actin-independen
40 chalasin B, and cytochalasin D indicate that SHFV does not hijack the actin polymerization pathway.
41                Our experiments indicate that SHFV enters target cells by low-pH-dependent endocytosis
42 nd electron microscopy results indicate that SHFV entry occurs by a dynamin-dependent clathrin-mediat
43 Comparative sequence analysis suggested that SHFV ORFs 2a, 2b, and 3 are related to ORFs 2 through 4
44                 Evidence which suggests that SHFV ORFs 4 through 6 are related to ORFs 2a through 3 a
45                                          The SHFV genome encodes nine ORFs that are presumed to be ex
46                                          The SHFV PLP1alpha catalytic Cys63 is unique among arterivir
47                                 To date, the SHFV life cycle is almost completely uncharacterized on
48 lecule identified as an entry factor for the SHFV-related porcine reproductive and respiratory syndro
49  also detected G3BP cleavage products in the SHFV-infected cell lysates and hypothesize that cleavage
50 TRSs were identified for the majority of the SHFV 3' ORFs, and four previously identified TRSs were f
51 nd sequenced 6,314 nt from the 3' end of the SHFV genome.
52     Here, we describe the first steps of the SHFV life cycle.
53  184 (68-nt sequence) from the 3' end of the SHFV negative-strand RNA.
54 n to be proximal by homology modeling of the SHFV nsp1s on porcine respiratory and reproductive syndr
55          The coding capacity for each of the SHFV ORFs as well as the potential mass, pI and number o
56 HFV PLP1s were predicted by alignment of the SHFV PLP1 region sequences with each other as well as wi
57 lternative products generated by each of the SHFV PLP1s cleaving at sites within the N-terminal regio
58               Several unique features of the SHFV PLP1s were discovered.
59  residues and cleavage sites for each of the SHFV PLP1s were predicted by alignment of the SHFV PLP1
60    This is the first functional study of the SHFV PLP1s.
61 rypsin) abrogated entry, indicating that the SHFV cell surface receptor is a protein.
62 e molecular masses as those that bind to the SHFV 3'(-)209 RNA also bind to the LDV-C 3'(-)NCR RNA an
63 elevating virus C (LDV-C), competed with the SHFV 3'(-)209 RNA in competition gel mobility shift assa
64 asmic extracts formed two complexes with the SHFV 3'(-)209 RNA, and results from competition gel mobi
65 ptide fragment showed that each of the three SHFV PLP1s is active, and the predicted catalytic Cys re
66       The data showed that each of the three SHFV PLP1s is an active protease.
67 ly divergent simian arteriviruses related to SHFV, Mikumi yellow baboon virus 1 (MYBV-1) and Southwes
68  protein 1alpha (MIP-1alpha), in response to SHFV infection were observed in macaque but not baboon c
69                                        Using SHFV reverse genetics, we confirmed critical roles of ns
70              Simian hemorrhagic fever virus (SHFV) causes a fatal hemorrhagic fever in macaques but a
71              Simian hemorrhagic fever virus (SHFV) causes a severe and almost uniformly fatal viral h
72              Simian hemorrhagic fever virus (SHFV) causes highly lethal disease in Asian macaques res
73 aques, while simian hemorrhagic fever virus (SHFV) causes lethal viral hemorrhagic fever.
74  arterivirus simian hemorrhagic fever virus (SHFV) is 209 nucleotides (nt) in length.
75              Simian hemorrhagic fever virus (SHFV) is an arterivirus that causes severe disease in ca
76 al region of simian hemorrhagic fever virus (SHFV) nonstructural polyprotein 1a is predicted to encod
77  with either simian hemorrhagic fever virus (SHFV) or Kibale red colobus virus 1 (KRCV-1) and assesse
78  nsp1beta of simian hemorrhagic fever virus (SHFV) was identified as a key factor that transactivates
79              Simian hemorrhagic fever virus (SHFV) was recently reclassified and assigned to the new
80 fection with simian hemorrhagic fever virus (SHFV), a member of the family Arteriviridae, does not in
81              Simian hemorrhagic fever virus (SHFV), a simian arterivirus, causes asymptomatic infecti
82  arterivirus Simian hemorrhagic fever virus (SHFV).
83 receptor for simian hemorrhagic fever virus (SHFV; a simian arterivirus), a rare mode of virus entry
84  same virus, simian hemorrhagic fever virus (SHFV; Arteriviridae).
85 e the 1960s, simian hemorrhagic fever virus (SHFV; Nidovirales, Arteriviridae) has caused highly fata
86 ng of SHFV-infected, MA104 cell lysates with SHFV nsp1 protein-specific antibodies detected only the
87                  Furthermore, -2/-1 PRF with SHFV PRF signal RNA can be stimulated by heterotypic nsp