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1                                              SLC also induced calcium mobilization specifically in ma
2                                              SLC and Epstein-Barr virus-induced molecule 1 ligand che
3                                              SLC elicited a substantial infiltration of DCs and T cel
4                                              SLC injection led to significant increases in CD4 and CD
5                                              SLC is a highly efficacious chemoattractant for lymphocy
6                                              SLC is likely to play an important role in regulating th
7                                              SLC was a potent in vitro chemoattractant for cultured,
8                                              SLC-mediated antitumor responses were lymphocyte depende
9                                              SLCs interact with several important drugs, and a quarte
10  CC chemokine CCL21, also known as Exodus-2, SLC, 6Ckine, and TCA4 induces both the adhesion and migr
11 emokines, Exodus-1/LARC/MIP-3alpha, Exodus-2/SLC/6Ckine/TCA4, and Exodus-3/CKbeta11/MIP-3beta, were f
12  Its ligands, CCL-19 (MIP-3beta) and CCL-21 (SLC), play an important role in the migration of these c
13 nt drugs, and a quarter of the more than 400 SLC genes are associated with human diseases.
14                             We show that 6CK/SLC is an agonist for the lymphocyte chemoattractant rec
15 ty of circulating lymphocytes respond to 6CK/SLC and MIP-3beta in large part through their common rec
16            The CC chemokine known as 6Ckine (SLC, Exodus-2, or TCA4) has been identified as a ligand
17 isolated by us and others as Exodus-2/6Ckine/SLC/TCA4) is highly potent and highly specific for stimu
18 olar concentrations of MCH strongly activate SLC-1-related pathways through G(alpha)i and/or G(alpha)
19 hese cells are likely to be the sought-after SLCs.
20 sity increased the probability of evoking an SLC and decreased mean SLC latencies while increasing th
21  single functional ELC gene, leaving only an SLC gene that is expressed in lymphatic endothelium and
22 es confirmed increased IL-7, SDF-1alpha, and SLC gene expression in MHC class II+ CD45- epithelial ce
23  polymorphisms in genes encoding for ABC and SLC transporters may have a significant impact on the ph
24 ivity, as excretion of BR depends on ABC and SLC transporters.
25                    A large number of ABC and SLC transpoters exist; however, only a small number have
26                This study implicates BLC and SLC in chronic inflammation and presents further evidenc
27 a was not necessary for induction of BLC and SLC in inflamed tissues, whereas, in contrast, tumor nec
28 ross-kissing interactions (SL-C to SL-D' and SLC' to SL-D) and stack in an end-to-end manner (SL-C to
29                     We propose that ELC- and SLC-expressing T zone stromal cells play a central role
30                               Hepatocyte and SLC trypan blue uptake were minimal and similar in both
31 art from their latencies, SLC kinematics and SLC field potential parameters were intensity independen
32                      The incidence of PN and SLC in patients with LC is high.
33                         Importantly, SL1 and SLC are functionally interchangeable, and separate excha
34                           Therefore, SL1 and SLC are likely essential comoviral RNA structures that p
35                   While the stems of SL1 and SLC share little sequence similarity, their end loops ar
36    The levels of IL-7, SDF-1alpha, TECK, and SLC mRNA inversely correlated with the kinetics of regen
37 at expression of IL-7, SDF-1alpha, TECK, and SLC mRNA is induced in the thymic stroma during T cell d
38                                         Anti-SLC Abs, but not control or anti-eotaxin Abs, blocked th
39                            In addition, anti-SLC Ab did not inhibit CHS responses when given at the t
40 se to hapten challenge was inhibited by anti-SLC Ab treatment in a dose-dependent manner.
41                   A single injection of anti-SLC Ab given at the time of sensitization with FITC inhi
42 tudies, we examined the consequences of anti-SLC Ab treatment during Ag priming on T cell function in
43                    Animals treated with anti-SLC Ab during hapten sensitization were not tolerant to
44 aining lymph nodes of mice treated with anti-SLC Ab during hapten sensitization.
45                                           As SLC transporters have high similarity in their membrane
46 he suppressor of long chain base auxotrophy (SLC, strain 7R4) showed a heat-sensitive phenotype that
47  the potential to sequester latent TGF-beta (SLC) to the cell surface where TGF-beta activation could
48  Fny-CMV plays a role similar to that of BMV SLC in interacting with the CMV replicase.
49 s indicates that the requirements in the BMV SLC are highly specific.
50 n of the Cucumber mosaic virus (CMV) and BMV SLCs indicates that the requirements in the BMV SLC are
51 at the plt defect is due to the loss of both SLC and EBI-1 ligand chemokine (ELC) expression in secon
52    Neutrophils roll, but are not arrested by SLC, whereas they respond to immobilized IL-8 with rapid
53 itro requires a structure named stem-loop C (SLC) that contains a clamped adenine motif.
54  virus (BMV), a stem-loop structure named C (SLC) is present within the tRNA-like region and is requi
55 n this region, a stem-loop structure, called SLC, is necessary and sufficient for the binding of the
56 es (PN) detection and secondary lung cancer (SLC) diagnosis.
57 nd are encoded by two solute-linked carrier (SLC) gene families: ZnT (SLC30) and Zip (SLC39).
58 porter Bor1, a member of the solute carrier (SLC) 4 transporter family with homology to the human bic
59 ansport studies suggest that solute carrier (SLC) and ATP binding cassette (ABC) multispecific "drug"
60 ncluding those controlled by solute carrier (SLC) and ATP-binding cassette (ABC) transporters and dru
61 s a member of a gene family, solute carrier (SLC) family 26, that encodes anion transporters and rela
62 variants in the proximity of solute carrier (SLC) genes.
63 ma membrane transporter, the solute carrier (SLC) human multidrug and toxin extrusion protein 1 (hMAT
64 cids and their transporters, solute carrier (SLC) members.
65 h more than 400 members, the solute carrier (SLC) membrane transport proteins are the largest family
66                              Solute carrier (SLC) membrane transport proteins control essential physi
67                  A number of solute carrier (SLC) proteins are subject to changes in expression and a
68 ed the genes that encode the solute carrier (SLC) proteins SLC12A2 and SLC12A4.
69 2 (SLC22A7), a member of the solute carrier (SLC) superfamily, was a facilitative transporter for cGM
70  proteins that belong to the solute carrier (SLC) superfamily.
71     OATP2B1, a member of the solute carrier (SLC) transporter family, is an important mechanism of su
72 drug molecules, which hijack solute carrier (SLC) transporters for active transport into the body.
73     There are over 420 human solute carrier (SLC) transporters from 65 families that are expressed ub
74                             Solute carriers (SLC) and ABC transporters represented an important subse
75 ette (ABC) transporters and solute carriers (SLCs), have been identified in platelets.
76 lls is suppressed in CCL19 (ELC)- and CCL21 (SLC)-deficient paucity of lymph node T cells mice, but n
77 /MCP-1, CXCL12/SDF-1, CCL5/RANTES, and CCL21/SLC.
78                            Stem Leydig cell (SLC) transplantation shows promise in this regard howeve
79 ultures of steroidogenic small luteal cells (SLCs), LH, and forskolin stimulated phosphorylation of D
80 ignaling complex with the surrogate L chain (SLC) components lambda5 and Vpre-B.
81 chain paired with two surrogate light chain (SLC) components.
82 posed of Ig heavy and surrogate light chain (SLC), signals pre-BII-cell proliferative expansion.
83 ed with a two-subunit surrogate light chain (SLC).
84                  We previously characterized SLC, a stem-loop structure in the 5' untranslated region
85 s secondary lymphoid tissue chemoattractant (SLC) and macrophage inflammatory protein (MIP)-3beta, im
86 in response to the lymphoid homing chemokine SLC/CCL21: CD4(-) Valpha14i-negative NKT cells that were
87 al venules, stromal cells, and the chemokine SLC.
88 amma (Mig) and secondary lymphoid chemokine (SLC) gene expression within allografts and spleens respe
89 votal role for secondary lymphoid chemokine (SLC) in directing dendritic cell trafficking from periph
90 and 21 (CCL21)/secondary lymphoid chemokine (SLC), a ligand for CC chemokine receptor 7 (CCR7), has b
91  loss of secondary lymphoid-organ chemokine (SLC) expression in lymphoid tissues.
92          Secondary lymphoid organ chemokine (SLC) is expressed in high endothelial venules and in T c
93 iency of secondary lymphoid organ chemokine (SLC), a CC chemokine that chemoattracts both dendritic c
94 acks the secondary lymphoid organ chemokine (SLC)-ser gene and has disrupted trafficking of T cells a
95 luminal secondary lymphoid tissue chemokine (SLC) (6Ckine, Exodus-2, thymus-derived chemotactic agent
96 rt that secondary lymphoid-tissue chemokine (SLC) (also known as 6Ckine, Exodus-2, and thymus-derived
97         Secondary lymphoid tissue chemokine (SLC) and B lymphocyte chemoattractant (BLC) are homing c
98 okines (secondary lymphoid tissue chemokine (SLC) and EBV-induced molecule 1 ligand chemokine (ELC))
99 K), and secondary lymphoid tissue chemokine (SLC) but not of other chemokines.
100 express secondary lymphoid tissue chemokine (SLC) into growing B16 melanoma could result in a substan
101         Secondary lymphoid tissue chemokine (SLC) is a CC chemokine that is selective in its recruitm
102         Secondary lymphoid-tissue chemokine (SLC), a CC chemokine, is expressed in secondary lymphoid
103         Secondary lymphoid tissue chemokine (SLC), a recently discovered chemokine for naive T cells,
104 actant, secondary lymphoid tissue chemokine (SLC), by T zone stromal cells is found to be markedly de
105 ngkine, secondary lymphoid-tissue chemokine (SLC), EBI1-ligand chemokine (ELC), fractalkine, macropha
106 tabase, secondary lymphoid-tissue chemokine (SLC), is expressed in the high endothelial venules of ly
107 ncludes secondary lymphoid tissue chemokine (SLC), which promotes the colocalization of naive, nonpol
108 LC) and secondary lymphoid tissue chemokine (SLC).
109  DCs to secondary lymphoid tissue chemokine (SLC)/CC chemokine ligand 21 (CCL21) were significantly l
110 s CCR7 (secondary lymphoid-tissue chemokine (SLC)/CC ligand (CCL)21), CXCR4 (stromal cell-derived fac
111         Secondary lymphoid tissue chemokine (SLC, also referred to as Exodus 2 or 6Ckine) is a recent
112 vement, secondary lymphoid tissue chemokine (SLC, CCL21) and Epstein-Barr virus-induced molecule 1 li
113 such as secondary lymphoid tissue chemokine (SLC; CCL21) to its counterreceptor, CCR7.
114                               The chemokines SLC (CCL 21) and BLC (CXCL13) were present, as were B220
115 - that are known as sex-limited chromosomes (SLCs).
116   The second case is for the Salt Lake City (SLC) region for August 2012.
117                              Salt Lake City (SLC), Utah, and the surrounding intermountain region exp
118     We also introduce Self-Label Clustering (SLC), an unsupervised clustering method relying on featu
119 xt of the entire tRNA-like element, both CMV SLC-like motifs are recognized by the BMV replicase.
120 h their SLCs replaced with two different CMV SLCs were defective for replication.
121 Ps) composed of sequences of low complexity (SLC) have been shown to serve a variety of important cel
122 eplicated associations of the amine compound SLC (solute-carrier) transporters gene set with the lear
123 iseases using a "sneaking ligand construct" (SLC) selectively inhibiting NF-kappaB in the activated e
124 is paper, a sampling-based learning control (SLC) method is used to guide the design of control field
125 e Guyot (SG), single spur pruned low cordon (SLC) and single spur pruned high wire cordon (HSLC) as t
126 n sequencing (NGS) techniques to deciphering SLC sequences.
127 that increasing stimulus intensity decreased SLC latencies while increasing their precision, which wa
128             Surrogate light-chain-deficient (SLC-/-) mice harbored elevated levels of antinuclear ant
129 ne-linked cautery and ultrasonic dissection (SLC+UD) from December 2002 to January 2004.
130 ts and associated challenges toward drugging SLCs, as well as highlight opportunities for future drug
131 ILC when compared with historic rates during SLC.
132                                         ELC, SLC, and TECK comprised high affinity ligands (IC50 <15
133 as abolished in the presence of soluble ELC, SLC (CCR7 ligands), and TECK (a CCR9 ligand).
134         Administration of amplicons encoding SLC (HSV-SLC) into s.c. tumors established previously re
135 tal structures of prokaryotic and eukaryotic SLC transporters indicating the location of both (or one
136 c. injection of lysate-pulsed DCs expressing SLC promoted the migration of T cells to the immunizatio
137 rters and the organic solute carrier family (SLC) proteins.
138 diverse heavy chains are paired with a fixed SLC were expressed in mammalian, Escherichia coli, and p
139 A CP was found to have higher affinities for SLC and the B box compared with those of wild-type CP an
140 hemokine receptor 7 (CCR7), the receptor for SLC and for macrophage inflammatory protein (MIP)-3beta
141  there are several specific requirements for SLC recognition.
142 These results indicate an important role for SLC during sensitization for CHS and suggest a strategy
143 the hypothalamus, consistent with a role for SLC-1 in mediating the effects of MCH on feeding.
144  for both CD4 and CD8 lymphocyte subsets for SLC-mediated tumor regression.
145  four of eight BALB/c mice and three of four SLC:ddY mice expressed one or more opacity (Opa) protein
146 r, splenic and lymph node-derived cells from SLC-treated tumor-bearing mice secreted significantly mo
147 ubtraction of field potential latencies from SLC latencies revealed a fixed time delay between the tw
148 s tumor, lymph node-derived lymphocytes from SLC-treated tumor-bearing mice demonstrated enhanced cyt
149 rradiated autologous tumor, splenocytes from SLC-treated mice secreted significantly more IFN-gamma a
150  natural ligand of a particular orphan GPCR (SLC-1) that is sequentially homologous to the somatostat
151      While the majority of CMV isolates have SLC-like elements similar to that of Fny-CMV, a second g
152 dministration of amplicons encoding SLC (HSV-SLC) into s.c. tumors established previously resulted in
153 mbined transduction of either tumor with HSV-SLC and HSV-CD40L resulted in a more enhanced antitumor
154 ing one of the largest families in the human SLC superfamily.
155 sensing and signalling hypothesis identifies SLC and ABC transporter-mediated communication between o
156                                  Immobilized SLC increased the adhesion of HUT-78 T cells and human P
157                 We now show that immobilized SLC strongly induces beta2 integrin-mediated binding of
158                                           In SLC, the incidence of ARF coresurged with the occurrence
159 P-3beta signaling, and occurs efficiently in SLC(low/-) HEV segments in wild-type mice, and in the SL
160                  BMV RNA3s with mutations in SLC were transfected into barley protoplasts, and the re
161 response to albuterol in Latinos, notably in SLC genes that include membrane transport proteins invol
162 or loss of cyclin A activity was observed in SLC-1 cells.
163 d nonmucoid pharyngeal isolates recovered in SLC from 1984 to 1999 were studied by sequencing the emm
164 that an ADME gene-centered network-including SLC and ABC "drug" transporters, "drug" metabolizing enz
165 nvolved in lymphoid organogenesis, including SLC, BLC, ELC, SDF1, and BAFF.
166 the fact that a sulfonamide CA IX inhibitor (SLC-0111) is presently in phase I clinical trials.
167        In immunocompetent mice, intratumoral SLC injection led to a significant increase in CD4 and C
168                  Apart from their latencies, SLC kinematics and SLC field potential parameters were i
169 ately derive from undifferentiated stem LCs (SLCs), which are postulated to be present in testes befo
170 ee stem loops (SLB-SLD), and that stem loops SLC and SLD play prominent roles in packaging.
171 essed by T zone stromal cells that also make SLC.
172 ability of evoking an SLC and decreased mean SLC latencies while increasing their precision; subtract
173 une strategy, we show that amplicon-mediated SLC and CD40L delivery may mimic lymph node conditions n
174 re we describe recent approaches to modulate SLC transporter function, with an emphasis on the use of
175                                    Moreover, SLC induces firm adhesion of naive T lymphocytes via bet
176  preferentially bind to an RNA element named SLC that contains the core promoter for genomic minus-st
177                           In the lymph node, SLC is believed to play an important role in the initiat
178                               The ability of SLC to chemoattract both Th1 lymphocytes and dendritic c
179       To this end, the antitumor activity of SLC and CD40L expressed singly or in combination using t
180 rom human PDAC cells, oral administration of SLC-0111 and injection of gemcitabine increased intratum
181 ically modified mice, oral administration of SLC-0111 and injection of gemcitabine reduced numbers of
182 nal biology allow better characterization of SLC pharmacology.
183 highly connected tissue-specific clusters of SLC transporters, ABC transporters, and DMEs.
184 sensing and signaling network" consisting of SLC and ABC transporters, as well as DMEs and regulatory
185 uture perspectives in the rational design of SLC drugs.
186  cells, suggesting that the dysregulation of SLC/ELC- expression alone in Ltbr-/- thymi can be suffic
187                               This effect of SLC was seen in both static and flow chamber adhesion as
188                  We show that the effects of SLC perturbation are mimicked by manipulation of either
189 trong rationale for additional evaluation of SLC in regulation of tumor immunity and its use in lung
190 a strong rationale for further evaluation of SLC in tumor immunity and its use in cancer immunotherap
191 phoid organs, is important for expression of SLC and BLC in secondary lymphoid organs during developm
192 duces inflammation and ectopic expression of SLC and BLC in the adult animal.
193 due to a genetic defect in the expression of SLC and that SLC mediates the entry of naive T cells and
194               In addition, the expression of SLC in lymphatic endothelium suggests that the migration
195       Here we demonstrate that expression of SLC is undetectable in plt mice.
196                                Expression of SLC was also observed in the pancreas of prediabetic NOD
197                   Intratumoral injections of SLC inhibited tumor growth in a CD8+, T cell-dependent m
198                   Intratumoral injections of SLC-expressing DCs resulted in tumor growth inhibition t
199 mise in this regard however, practicality of SLC isolation/transplantation impede clinical translatio
200                            Quantification of SLC kinematics and field potential parameters revealed t
201  behavior of mRNA levels for a wide range of SLC and ABC transporters in the rodent kidney throughout
202 s to the SLC locus; however, the sequence of SLC introns and exons in plt mice is normal.
203 , bile) via "matching" or homologous sets of SLC (e.g., SLC21, SLC22, SLC47) and ABC transporters.
204 ermined the high-resolution NMR structure of SLC, which demonstrated that a 5'-AUA-3' triloop region
205                                   Studies of SLC transporter uptake to-date relied on radioisotope- o
206 r) determine the onset time and precision of SLCs.
207 st in elucidating the physiological roles of SLCs as well as growing recognition of their therapeutic
208 OSS and dispersed South Louisiana crude oil (SLC) in laboratory microcosms.
209 The time is right for a systematic attack on SLC structure, specificity, and function, taking into ac
210                         Each was enriched on SLC, the former at 25 degrees C, the latter at 5 degrees
211     Furthermore, RIP-mOVA transgenic mice on SLC/ELC deficient background (plt) demonstrated signific
212 lymphatic markers such as VEGFR-3, LYVE-1 or SLC.
213 nificantly better than either control DCs or SLC alone.
214                                        Other SLC transporters have emerged as pharmacology targets.
215                       Along with OCT2, other SLC-family drug transporters are potentially part of an
216         The approach can be applied to other SLC and ATP-binding cassette drug transporters to define
217 5 was also evident with Dmu, but the overall SLC- and L chain-dependent requirements for Dmu maturati
218        In addition, chemokines, particularly SLC (CCL21), were also required for IS-CD8(+) cells' adh
219  We present design rules for IDPs possessing SLCs that phase separate into diverse assemblies within
220 d allowed for discrimination between primary SLCs and less frequent, long-latency startle responses (
221 teresting family of solute carrier proteins (SLCs), some of which have been suggested as being involv
222 growth factor I, and LH, 40% of the putative SLCs came to express 3betaHSD and to synthesize testoste
223  which LCs had been eliminated, the putative SLCs colonized the interstitium and subsequently express
224                                 The putative SLCs expanded over the course of 6 months while remainin
225 acid human orphan G-protein-coupled receptor SLC-1 expressed in HEK293 cells binds MCH with sub-nanom
226 cently the orphan G protein-coupled receptor SLC-1 was identified as the MCH receptor (MCHR).
227 dition, we demonstrate that the MCH receptor SLC-1 is expressed in adipocytes, suggesting that fat ce
228        An orphan G protein-coupled receptor (SLC-1/GPR24) has recently been identified as a receptor
229                 Recently an orphan receptor, SLC-1, has been identified as an MCH receptor (MCH-R1).
230        Intratumoral injection of recombinant SLC evidenced potent antitumor responses and led to comp
231                     Injection of recombinant SLC in the axillary lymph node region led to a marked re
232          Exploring the basis for the reduced SLC expression led to identification of further disrupti
233 n-sensitive B cell sticking does not require SLC or MIP-3beta signaling, and occurs efficiently in SL
234 we examined short-latency startle responses (SLCs) in larval zebrafish and tested the hypothesis that
235 een short-latency and long-latency C-starts (SLCs vs. LLCs) in larval zebrafish.
236 een short-latency and long-latency C-starts (SLCs vs. LLCs) in larval zebrafish.
237 d to other gene families of similar stature, SLCs are relatively understudied.
238                     An outbred mouse strain (SLC:ddY) previously reported to be naturally susceptible
239                                           TB SLC with higher grades of lesion opacity at baseline may
240 raphs of consecutive patients with active TB SLC seen at a single tertiary referral center with 6 mon
241 uded 203 eyes of 183 patients with active TB SLC.
242 risk factors for paradoxical worsening of TB SLC (OR 7.555, 95% CI: 1.78-32.02; P = 0.006; OR 7.434,
243 hat the chemokine CCL21 (also known as TCA4, SLC, 6Ckine), a ligand for the chemokine receptor CCR7,
244  with islet expression of the chemokine TCA4/SLC.
245               Our results indicate that TCA4/SLC can induce the development and organization of lymph
246   Technically, SILC is more challenging than SLC.
247 appear to be more susceptible to injury than SLCs.
248 tic defect in the expression of SLC and that SLC mediates the entry of naive T cells and antigen-stim
249                This report demonstrates that SLC can both induce antitumor responses and enhance the
250        Thus, we provide direct evidence that SLC and CCR7 participate in the emigration of DCs from p
251                             The finding that SLC is a potent chemokine for DC as well as naive T cell
252                            We show here that SLC is a potent chemokine for mature DC but does not act
253 he Malvasia as the more effective one), that SLC led to the lowest level of TAC and that 8 buds/plant
254                           Here, we show that SLC binds to alpha(v)beta8, an integrin expressed by nor
255                            We also show that SLC-1 messenger RNA and protein is expressed in the vent
256                                          The SLC contains nonimmunoglobulin-like peptide extensions o
257 t is determined by its Ig components and the SLC, we investigated the regulation of pre-BCR surface e
258 and activation stage-dependent manner by the SLC approach.
259                       As in pre-B cells, the SLC in nonlymphoid cells supported only a limited degree
260 tify peptides in the capsid that contact the SLC, the B-box RNA, and the encapsidated RNA.
261 re of GAS strains associated with ARF in the SLC region, 964 mucoid and nonmucoid pharyngeal isolates
262 ) HEV segments in wild-type mice, and in the SLC-negative HEVs of DDD/1 mice.
263 tion represents a deletion that includes the SLC gene expressed in secondary lymphoid organs and the
264  of multispecific "drug" transporters of the SLC and ABC transporter families.
265 presents a well-characterized example of the SLC family.
266 ency and the precision in the latency of the SLC field potentials were linearly correlated to the lat
267 e pre-BCR also signals downregulation of the SLC genes (VpreB and lambda5), thereby limiting this exp
268 cause of the heat-sensitive phenotype of the SLC strain 7R4.
269 This Perspective provides an overview of the SLC superfamily, including their biochemical and functio
270                            Expression of the SLC-related chemokine, Epstein Barr virus-induced molecu
271 this was due to an intrinsic property of the SLC.
272 sing this replicase, we demonstrate that the SLC-like structure in Fny-CMV plays a role similar to th
273                 The plt mutation maps to the SLC locus; however, the sequence of SLC introns and exon
274 ow that the "smart" fields learned using the SLC method can achieve robust manipulation of supercondu
275                                          The SLCs mediate the movement of ions, drugs, and metabolite
276 toplasts, we found that BMV RNA3s with their SLCs replaced with two different CMV SLCs were defective
277 s T cell-attracting chemokine (CTACK), three SLC, and four ELC genes or pseudogenes are present in so
278                                        Thus, SLCs are emerging as important targets for therapeutic i
279  chemotaxis of murine T cells and B cells to SLC but not to other homeostatic chemokines.
280 t L-selectin-mediated enhanced chemotaxis to SLC required intact intracellular kinase function.
281 lectin-mediated enhancement of chemotaxis to SLC was observed for all naive lymphocytes and effector/
282 1023-base pair cDNA and is 35% homologous to SLC-1.
283 pha14i-positive NKT cells did not migrate to SLC/CCL21.
284 lable methodology and results with regard to SLC sequencing and assembly.
285 wth factor beta was decreased in response to SLC treatment.
286 tin-sensitive Na(+)-Li(+) counter-transport (SLC) activity, an established marker for hypertension.
287  of both major classes of drug transporters, SLC and ABC, in resting human blood neutrophils.
288 ing in monitoring the activity in tubercular SLC.
289 FAF) images of eyes affected with tubercular SLC from the acute stage until resolution of lesions usi
290 o both primary and MK cells, the tumorigenic SLC-1 cell line did not accumulate in a specific cell cy
291 otypes share a duplication that includes two SLC genes, which demonstrate different expression patter
292 n-like peptide extensions on each of the two SLC components.
293                                       The UD+SLC group had a decreased duration of inflow occlusion (
294                                 We next used SLC gene-modified DCs as a treatment of established tumo
295                                Here, we used SLC as a treatment for tumors established from the poorl
296 ique for liver parenchymal transection using SLC and UD in noncirrhotic livers is safe and may provid
297  of CA9 in PDAC cells, or inhibited CA9 with SLC-0111, incubated them with gemcitabine, and assessed
298 6% of analysed patients) were diagnosed with SLC.
299 site immunization of tumor-bearing mice with SLC gene-modified DCs pulsed with tumor lysate elicited
300 we showed MHC class II-positive cells within SLC-staining lymphatic channels in the mouse dermis.

 
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