ライフサイエンスコーパス: SN

1 SN biopsy identified significantly smaller LN metastases

2 SN levels were elevated in patients with CPVT and follow

3 SN mapping was performed using lymphoscintigraphy and SP

4 SN production is upregulated in conditions with cardiomy

5 SN treatment also lowered the incidence of early afterde

6 SN-AI(95) was an independent contributor of poor motor o

7 SNs were identified and removed from 2859 patients who h

8 rols, with AUC = 0.992 (95% CI 0.983-1.000), SN = 0.963 (95% CI 0.913-1.000), and SP = 0.967 (95% CI

9 pically very difficult to produce sub-100nm, SN-38-encapsulated nanoparticles without modification of

10 Therefore, these sub-100nm, SN-38-encapsulated photonic micelles show great promise

11 on of the excised specimens revealed that 14 SNs removed under ePLND or after unblinding contained ra

12 on of the excised specimens revealed that 14 SNs removed under ePLND or after unmasking contained rad

13 Data on 279 patients (139 ND and 140 SN) could be analyzed.

14 ith L-DOPA compared to baseline (200 of 1807 SN-VTA voxels; P(corrected) = 0.046), although the small

15 4,402 survivors, 495 (11.2%) developed 1,269 SNs.

16 assay involves the extraction of free SN-38, SN-38G by protein precipitation, and subsequent acid hyd

17 Among the 2,748 SN-negative patients, SN biopsy would not have been offe

18 At a pre-selected cutoff of >80% SN and SP for the affiliated PancRISK score, we demonstr

19 Accordingly, SN attenuated isoproterenol induced autophosphorylation

20 Adding SN biopsy in patients with negative PSMA PET/CT results

21 During ePLND 22 additional SNs were resected (7 in the hybrid tracer group and 15 i

22 During ePLND, 22 additional SNs were resected (7 in the hybrid-tracer group and 15 i

23 c/F bottles as well as resin-free BacT/Alert SN and BD Bactec standard anaerobic bottles.

24 Median cross-sectional area of all SN metastases was 0.13 mm; in US true-positive nodes, it

25 lloid to create a road map that depicted all SNs.

26 e only allowed intraoperative removal of all SNs in 40% of patients in the hybrid tracer group and in

27 idance allowed intraoperative removal of all SNs in only 40% of patients in the hybrid-tracer group a

28 Among SN-positive patients, 39 (7.1%) had an abnormal US.

29 tively, and no significant differences among SN (68.5%), CN (26.4%), and SASF (5.2%).

30 and 6 patients with penile cancer during an SN biopsy procedure using indocyanine green-(99m)Tc-nano

31 r bottle types, SA and FA Plus (aerobic) and SN and FN Plus (anaerobic), was performed in a clinical

32 tely 17,000 nuclei from matched cortical and SN samples.

33 patients, such as the use of PSMA PET/CT and SN biopsy rather than ePLND as the preferred approach fo

34 overexpressing cells toward daunorubicin and SN-38, respectively, in concentration ranges that qualif

35 rns between the three networks, with FPN and SN activity predominant in early windows and DN stimulat

36 a the SNc-related nigrostriatal pathway) and SN (hypothetically via the VTA-related mesocorticolimbic

37 (di-, tri-, and tetra-) between striatum and SN N-glycans with the detection of phosphorylated N-glyc

38 abundances of N-glycans in the striatum and SN of rodent brains, serving as a foundation for identif

39 The total N-glycomes from the striatum and SN were characterized using database mining (GlycoStore)

40 ation of N-glycans from the rat striatum and SN; reproducibility, resolution, and relative quantitati

41 807 substantia nigra-ventral tegmental area (SN-VTA) voxels, P(corrected) = 0.038), particularly in t

42 substantia nigra and ventral tegmental area (SN/VTA), medial temporal lobe, or subsequent memory perf

43 ur aetiological understanding by associating SN cell type expression profiles with specific disease r

44 Because SN inhibits CaMKIIdelta (Ca(2+)/calmodulin-dependent pro

45 ticorrelation between SN and DMN and between SN and ECN compared with patients with unfavorable outco

46 ectivity and greater anticorrelation between SN and DMN and between SN and ECN compared with patients

47 ession models tested the association between SN-AI(95) and clinical scores.

48 Beyond PD, we find associations between SN DaNs and GABAergic neuron gene expression and multipl

49 cking and hydrophobicity interaction between SN-38, and a unique class of photonic nanoporphyrin mice

50 Interactions between SN and CaMKIIdelta were mapped using pull-down experimen

51 ontrol subjects; and 3) the relation between SN-centered network dynamics and clinical symptoms.

52 the protein layer to release antibody-bound SN-38.

53 higher (P < 0.001) with BBB disruption, but SN-38 was only detected in <50% of the samples and only

54 owed that the extent of tumor penetration by SN-38 was significantly higher in mice receiving the tar

55 ac communication, CMs, directly contacted by SNs in co-cultures, were larger than the non-targeted on

56 Based on the evidence that cardiac SNs are unevenly distributed throughout the myocardial w

57 Circulating SN (secretoneurin) concentrations are increased in patie

58 T (high-sensitivity troponin T), circulating SN levels declined after resuscitation, as the risk of a

59 ecommend SNB in most cN1 patients with clear SNs after primary chemotherapy because the high SNB fals

60 which has only been reported in collismycin SN.

61 ally-located substantia nigra pars compacta (SN) and medially-located ventral tegmental area (VTA), b

62 By conjugating SN-38 to the dendrimer via different linker technologies

63 the ipsilateral compared with contralateral SN.

64 Detaching SN unmasked ectopic pacemaking in AVRs and pacemaker act

65 rly in the more medial, anterior, and dorsal SN-VTA.

66 at 11 nM (EC(50)), time-dependently doubled SN-38 toxicity in a period of 7 days at 10 nM, and half-

67 lower sensitivity to Cav1.3 variants during SN DA-like pacemaking compared with Cav1.2 during aSM-li

68 e classification analysis identified dynamic SN-centered cross-network interaction patterns as factor

69 In both cohorts, we found that dynamic SN-centered cross-network interactions were significantl

70 st 6 months after surgery, thus establishing SN as the standard of care in OC.

71 Excised SNs were histopathologically assessed and considered pos

72 n PSMA PET/CT were considered candidates for SN biopsy with indocyanine green-(99m)Tc-nanocolloid or

73 sing spectral synthesis models developed for SN 2017iuk, we show that these features are characterize

74 cycle length in AVRs, indicating a role for SN-like pacemaker mechanisms.

75 Patient selection for SN biopsy can be assisted by National Comprehensive Canc

76 ients, US is not an effective substitute for SN biopsy.

77 5-year survival have increased the risk for SNs and debilitating health conditions for survivors yet

78 e presence of trace level of SN-38G and free SN-38 in plasma, which suggests an improved therapeutic

79 The assay involves the extraction of free SN-38, SN-38G by protein precipitation, and subsequent a

80 A standard Nissen fundoplication (SN) was performed in cases with an abdominal length >1.5

81 late an R2* asymmetry index (SN-AI); greater SN-AI indicated greater relative R2* in the ipsilateral

82 Among those patients, 548 had SN metastases.

83 The 95th percentile of R2* (hereafter, SN-AI(95)) was compared according to infarct location wi

84 vidual infarcted voxels associated with high SN-AI(95) through voxel-based lesion-symptom mapping.

85 and average maps consistently showed higher SN R2* at 1 year if ipsilateral striatum was infarcted t

86 on of the infarct was associated with higher SN-AI(95) at 1 year independently from infarct volume, S

87 external capsule were associated with higher SN-AI(95) at 1 year.

88 f the 20 patients with HIV, 14 (70%) had HIV-SN.

89 in micelles (NPM), the extremely hydrophobic SN-38 was successfully encapsulated into NPM with signif

90 nctions, reduction of TH-expressing cells in SN, and TH mRNA/protein levels in midbrain/striatum.

91 Purpose To assess longitudinal changes in SN R2* by using MRI in the setting of ipsilesional supra

92 the detection of phosphorylated N-glycans in SN which were not detected in the striatum.

93 Slow migration is the default mode in SN-mDA neurons, while fast, laterally-directed migration

94 T cells across the endothelial monolayer in SN of hemiparkinsonian monkeys.

95 ective connectivity between brain regions in SN and FPN distinguished the ADHD and TD groups, with rD

96 DA neurons we demonstrate its direct role in SN-mDA tangential migration.

97 ns, hyperchromic and bathochromic effects in SNs containing all co-pigment sources, except for PRE.

98 ound 19, concentration-dependently increased SN-38-mediated cancer cell death at 11 nM (EC(50)), time

99 terally to calculate an R2* asymmetry index (SN-AI); greater SN-AI indicated greater relative R2* in

100 In line with CaMKIIdelta and RyR inhibition, SN treatment decreased Ca(2+) spark frequency and dimens

101 lantation ipsilateral to the vector-injected SN.

102 Visual inspection, SN-AI(95), and average maps consistently showed higher S

103 owed by triethylamine induced intramolecular SN(Ar) displacement reaction and subsequent Michael addi

104 escence guidance was used for intraoperative SN identification.

105 escence-guidance was used for intraoperative SN identification.

106 ugated with active metabolite of irinotecan (SN-38), on Trop-2 positive cervical cancer cell lines an

107 index (0.725), and absolute difference of IT-SN GCIPL thickness (0.715).

108 For macular asymmetry parameters, log IT/SN index had the largest AUROC (0.734), followed by log

109 best measures of asymmetry analysis (log IT/SN index) and those of cpRNFL, GCIPL, and ONH parameters

110 The IT/SN asymmetry index not only had the largest pAUROC based

111 lar ganglion cell asymmetry parameter was IT/SN asymmetry index, which could be a new parameter to de

112 Mechanistic studies mapped SN binding to the substrate binding site in the catalyti

113 of distinguishing CPT-11 from its metabolite SN-38.

114 al SN-38 and its glucuronidation metabolite (SN-38G) using stable isotope dilution (SID) ultrahigh-pe

115 ar whether Reelin acts directly on migrating SN-mDA neurons and how it affects their cell morphology

116 cell morphologies of tangentially migrating SN-mDA neurons in 3D and identify two distinct migration

117 of the SPECT/CT images, the number of missed SN's were recorded and their resection was pursued when

118 of the SPECT/CT images, the number of missed SNs was recorded and their resection was pursued when th

119 thocyanins and colour in strawberry nectars (SNs), sweetened with sucrose (SNS), maltose syrup (SNM)

120 regarding the risk of subsequent neoplasms (SNs) in children with pathogenic neurofibromatosis type

121 years from diagnosis), subsequent neoplasms (SNs), and chronic health conditions were evaluated in th

122 The sciatic nerve (SN) showed increased nonuniform, internodal segments sug

123 LAI were correlated to the salience network (SN) and language network (LN) and anti-correlated to the

124 The salience network (SN) is dysregulated in many neuropsychiatric disorders,

125 ensorimotor network (SMN), salience network (SN), and default-mode network (DMN)-and in neurotransmit

126 ork interactions among the salience network (SN), central executive network, and default mode network

127 tention network (DAN), the salience network (SN), the somatosensory network (SMN) and the between-net

128 lt mode network (DMN), and salience network (SN).

129 s: (i) cingulo-opercular "salience" network (SN) anchored in the right anterior insula, dorsal anteri

130 hese evident functions, sympathetic neurons (SNs) also provide trophic input to cardiomyocytes (CMs),

131 opaminergic neurons in the substantia nigra (SN) and affected the integrity of the nigrostriatal syst

132 rafficking, in vivo in the substantia nigra (SN) and the serum of 1-methyl-4-phenyl-1,2,3,6-tetrahydr

133 (DA) neuronal loss in the substantia nigra (SN) and ventral tegmental area (VTA), supporting an impo

134 ated from the striatum and substantia nigra (SN) can give an insight into the establishment and patho

135 cell type patterns in the substantia nigra (SN) from 53 donors with and without PD, as well as in in

136 expression profile of the substantia nigra (SN) from PD patients, analyzed post mortem.

137 Background The substantia nigra (SN) is suspected to be affected after remote infarction,

138 n of neutral lipids in the substantia nigra (SN) of Parkinson's disease (PD) patients and its relatio

139 Dopamine neurons in the substantia nigra (SN) pars compacta are selectively lost during the progre

140 d DA neurons in the medial substantia nigra (SN) projecting either to dorsal or ventral striatum.

141 ynuclein (A53T) in the rat substantia nigra (SN) to produce degeneration of SN dopamine neurons, loss

142 anscriptomic atlas for the substantia nigra (SN), generated by sequencing approximately 17,000 nuclei

143 ns make projections to the substantia nigra (SN), ventral tegmental area (VTA) and ventrolateral-vent

144 in dopamine neurons of the substantia nigra (SN).

145 unilaterally into the rat substantia nigra (SN).

146 GLT-1 expression in murine substantia nigra (SN).

147 g secondary sulfate (SS), secondary nitrate (SN), biomass burning (BB), pyrolyzed organic carbon (OP)

148 AN), sodium sulfate (SS), or sodium nitrate (SN) solutions with 1 M phosphate buffer (PB) at differen

149 ane antigen (PSMA) PET/CT and sentinel node (SN) biopsy in PSMA PET/CT-negative patients for primary

150 Sentinel node (SN) biopsy is accurate in operable oral and oropharyngea

151 e US could avoid the need for sentinel node (SN) biopsy, but in most single-institution reports, the

152 taneous melanoma, the risk of sentinel node (SN) metastasis varies according to several clinicopathol

153 d to identify determinants of sentinel node (SN) status for incorporation into an externally validate

154 Knowing that the hybrid sentinel node (SN) tracer indocyanine green (ICG)-(99m)Tc-nanocolloid g

155 In right atrial preparations, sinus node (SN) was dominant and AVRs displayed 1:1 impulse conducti

156 phatic drainage patterns and sentinel nodes (SNs) of individual prostate cancer patients.

157 a roadmap that depicted all sentinel nodes (SNs).

158 FB coupling between the DAN and DMN (but not SN and DMN) is associated with greater sustained attenti

159 l striatum was infarcted than if it was not (SN-AI(95), 4.25 vs -0.88; P < .001), but this was not ob

160 lop a new relationship between smoke number (SN) and BC mass concentration.

161 In vitro analyses of SN/CM co-cultures, ex vivo confocal and multiphoton imag

162 y enables delivery of high concentrations of SN-38 to tumors.

163 We use a data set consisting of SN-BC mass concentration pairs, collected using certific

164 K-1) selectively potentiated cytotoxicity of SN-38, 5-fluorouracil (5-FU) and mitoxantrone, but not t

165 K-1) selectively potentiated cytotoxicity of SN-38, 5-fluorouracil and mitoxantrone, but not that of

166 tantia nigra (SN) to produce degeneration of SN dopamine neurons, loss of striatal dopamine levels, a

167 led administration of an efficacious dose of SN-38, achieving significant regression of the SW620 tum

168 are a likely target mediating the effects of SN DA activation.

169 In vivo effects of SN were investigated in CPVT mice (RyR2 [ryanodine recep

170 differ from those observed in the ejecta of SN 2017iuk at later times.

171 our knowledge, the oncologic equivalence of SN biopsy and neck lymph node dissection (ND; standard t

172 Whether secondary involvement of SN worsens overall clinical outcome after a supratentori

173 ults revealed the presence of trace level of SN-38G and free SN-38 in plasma, which suggests an impro

174 Circulating levels of SN and other biomarkers were assessed in patients with c

175 GM1 administration protected against loss of SN dopamine neurons and striatal dopamine levels, reduce

176 th cohorts, dynamic time-varying measures of SN-centered cross-network interactions were correlated w

177 Tangential migration of SN-mDA neurons is altered in absence of Reelin signaling

178 ght aberrancies in task-evoked modulation of SN and FPN connectivity in children with ADHD.

179 associated virus-9-induced overexpression of SN attenuated arrhythmic induction during stress testing

180 ripartite connectivity-based parcellation of SN with a limbic, cognitive, motor arrangement.

181 ow significant contribution to protection of SN anthocyanins and colour.

182 pratentorial infarct and the relationship of SN signal change to clinical outcome.

183 = .003), yielding a 2.4-fold higher risk of SN (95% CI, 1.3 to 4.3; P = .005) in the NF1-affected in

184 d that more accurately estimates the risk of SN positivity in patients with melanoma than currently a

185 lop a primary tumor are at increased risk of SN when compared with non-NF1 childhood cancer survivors

186 due to the ultra-flat aromatic structure of SN-38, it is typically very difficult to produce sub-100

187 e unmasked, causing transient suppression of SN pacemaking.

188 tivity, we hypothesized that upregulation of SN in patients protects against cardiomyocyte mechanisms

189 The combined use of SN biopsy and PSMA PET/CT identified all pN1 patients (1

190 Relative rates (RRs) and 95% CIs of SNs by mutation status were estimated using multivariabl

191 atment-related risks, for the development of SNs.

192 In line with the trophic effect of SNs, and with local neuro-cardiac communication, CMs, di

193 cohort, the 20-year cumulative incidence of SNs in NF1 childhood cancer survivors was 7.3%, compared

194 survey, the 15-year cumulative incidence of SNs was higher among survivors with multimodal therapy (

195 that both the acute and chronic influence of SNs on cardiac function and structure is enacted as a re

196 c nodal surgery underestimated the number of SNs in 60%-80% of patients.

197 c nodal surgery underestimated the number of SNs in 60-80% of patients.

198 ce a significant difference in the number of SNs visualized (P = 0.836).

199 aging lymphadenectomy including resection of SNs related to the ovary, SPECT/CT was performed within

200 ndividuals with a primary tumor, the risk of SNs was 2.8-fold higher in patients with irradiated NF1

201 In contrast, the risk of SNs was not significantly elevated after exposure to alk

202 kylating agents, confer an increased risk of SNs.

203 decrease was not observed in sodium (SS- or SN-PB) droplets, and no pH differences were observed bet

204 ables related to palatal contour (PP to PAS, SN to PAS, palatal surface angle) and occlusal planes (U

205 ntially expressed genes from both PD patient SN and SNCA-A53T hNs were associated with regulatory mot

206 Among the 2,748 SN-negative patients, SN biopsy would not have been offered to 22.1%, 13.4%, a

207 antibody conjugated to the cytotoxic payload SN-38.

208 This study aimed to determine human PD SN neutral lipid content and distribution in dopaminergi

209 noma protein B (GPNMB) signaling in human PD SN.

210 antly accumulated intracellular lipids in PD SN, while adjacent astrocytes had a reduced lipid load o

211 levated levels of NEAT1 expression in the PD SN compared with control brains, an elevation that was r

212 In the MSLT-II screening phase population, SN tumor volume was usually too small to be reliably det

213 ologic parameters to more accurately predict SN positivity.

214 Myocardial pro-SN expression was also increased in CPVT mice, and furth

215 SPECT/CT enhanced the detection of prostate SNs in more ectopic locations in 17 of the 25 patients a

216 SPECT/CT enhanced the detection of prostate SNs in more ectopic locations in 17 of the 25 patients,

217 The neurobiological identification of a rat SN, together with a demonstration of its functional rele

218 the anatomical profile of the identified rat SN is similar to that of nonhuman primates and humans.

219 subdivisions and to identify a potential rat SN based on functional connectivity patterns from the in

220 We identified a putative rat SN, which consists of primarily the ventral anterior ins

221 ctional connectivity architecture of the rat SN is supported by the mouse neuronal tracer data.

222 Finally, we demonstrated that the rat SN responds to conditioned cues and increases functional

223 Finally, we assessed the rat SN's response to conditioned cues in rats (n = 21) with

224 at 100 degrees C) was established to release SN-38, conjugated to the antibody by carbonate linkage.

225 After unblinding 18 remaining SNs were identified (6 in the hybrid group and 12 in the

226 After unmasking, 18 remaining SNs were identified (6 in the hybrid group and 12 in the

227 rall, 75.9% of the intraoperatively resected SNs in the hybrid tracer group and 51.8% of the SNs in t

228 rall, 75.9% of the intraoperatively resected SNs in the hybrid-tracer group and 51.8% of the SNs in t

229 sufficient anatomical sensitivity to resolve SN subregions.

230 cribed in humans, identification of a rodent SN would provide the ability to mechanistically interrog

231 ty with dorsal attention (DAN) and salience (SN) networks.

232 Diagnostic tests such as sensitivity (SN), specificity (SP), positive and negative predictive

233 This improved panel showed both sensitivity (SN) and specificity (SP) to be >85%.

234 (HIV) participants compared to seronegative (SN) controls.

235 In computer simulations of a sick SN, ectopic foci in AVR were unmasked, causing transient

236 Due to the relatively small size, SN-NPM possessed superior long tumor retention time (>5d

237 DA signaling favors the predominance of SMN-SN activity, whereas 5-HT signaling favors the predomina

238 laser-capture micro-dissected striatonigral (SN) and striatopallidal (SP) medium spiny neurons (MSNs)

239 A PET/CT results and 31 underwent subsequent SN biopsy after negative PSMA PET/CT results.

240 PSMA PET/CT results and underwent subsequent SN biopsy.

241 the thrombin-specific, fluorogenic substrate SN-59 (100 muM).

242 rmality and the time to peak supernormality (SN@).

243 spectroscopic observations of the supernova SN 2017iuk, which is associated with the gamma-ray burst

244 and superotemporal (ST), IT and superonasal (SN), IT and inferonasal (IN), ST and IN as well as tempo

245 release half-life of 21h achieved sustained SN-38 exposure in blood, above the target concentration.

246 tensive alpha-synucleinopathies earlier than SN dopamine neurons while exhibiting milder cell loss in

247 r cognitive performance and start index than SN at baseline, both groups improved on improvement inde

248 To determine the possibility that SNs are missed, this feasibility study compared their de

249 The SN was segmented bilaterally to calculate an R2* asymmet

250 ndomly assigned to (1) the ND arm or (2) the SN arm (experimental arm: biopsy alone if negative, or f

251 varying cross-network interactions among the SN, central executive network, and default mode network

252 formation of NEAT1-based paraspeckles at the SN and demonstrated coincreases of NEAT1 and paraspeckle

253 We addressed these gaps by determining the SN risk in 167 NF1-affected versus 1,541 non-NF1-affecte

254 Remarkably, CM size followed the SN distribution pattern in other mammals, including huma

255 and progressive loss of DA neurons from the SN and terminals in the striatum, accompanied by extensi

256 h was 8 days in the ND arm and 7 days in the SN arm (P < .01).

257 d ND approaches, with lower morbidity in the SN arm during the first 6 months after surgery, thus est

258 rm and 90.7% (95% CI, 0.84% to 0.95%) in the SN arm, confirming the equivalence with P < .01.

259 tive SPECT/CT revealed no differences in the SN identification rate between ICG-(99m)Tc-nanocolloid a

260 se results suggest that NM-MRI signal in the SN is a proxy for function of dopamine neurons in the ni

261 mplitude of compound action potential in the SN suggested that the functional capacity of the heavily

262 est in the DAN, at intermediate speed in the SN, and slowest in the DMN.

263 , and decreased alpha-synucleinopathy in the SN.

264 in varying levels of NM accumulation in the SN; however, the ability of NM-MRI to measure dopamine f

265 GFAP-Cre-GFP particles were infused into the SN of 8-week-old YY1 (flox/flox) mice to generate a regi

266 fusion of AAV5-GFAP-Cre-GFP vectors into the SN resulted in region-specific astrocytic YY1 deletion a

267 ared functional connectivity profiles of the SN across rats, marmosets (n = 10), and humans (n = 30).

268 emonstrated the oncologic equivalence of the SN and ND approaches, with lower morbidity in the SN arm

269 berrations in time-varying engagement of the SN with the central executive network and default mode n

270 rticolimbic pathway with core regions of the SN.

271 Though the SN was initially described in humans, identification of

272 al seed had a stronger connection within the SN in slow 6-slow 3 bands.

273 l striatum and resting blood flow within the SN.

274 B (with no further axillary treatment if the SNs are negative), irrespective of axillary status befor

275 If the SNs were pN0, patients generally received no further axi

276 urther axillary treatment (SNB only); if the SNs were pN1, completion AD (SNB + AD) was usually perfo

277 in the hybrid tracer group and 51.8% of the SNs in the "free" ICG group were removed solely under fl

278 in the hybrid-tracer group and 51.8% of the SNs in the free-ICG group were removed solely under fluo

279 ton coupled electron transfer (PCET) at the {SN} ligated site, pointing at a potential functional rol

280 tive for HCN4, and SERCA2a was comparable to SN.

281 beta(2)-receptor protein were comparable to SN.

282 L decreased rifampicin-induced resistance to SN-38, the active metabolite of irinotecan, in LS174T ce

283 selective approach to measure free and total SN-38 and its glucuronidation metabolite (SN-38G) using

284 harmacological interference with the trophic SN/CM signalling axis.

285 ded in human cells by the endonuclease Tudor-SN (TSN).

286 m 3,477 patients with melanoma who underwent SN biopsy at Melanoma Institute Australia (MIA) were ana

287 he number of patients undergoing unnecessary SN biopsy would be significantly reduced compared with u

288 at 1 year independently from infarct volume, SN-AI(95) at baseline, microbleeds, age, and sex (beta =

289 ,635 underwent SNB; 108 patients (6.6%) were SN positive.

290 in AVRs and pacemaker action potentials were SN-like.

291 With respect to sites where SNs were resected, remaining hotspots may have been caus

292 to age-matched controls, the levels of whole SN triglycerides were correlated with inflammation-atten

293 cT2 cN0/1 patients whose SNs are disease-free (pN0) after primary chemotherapy ca

294 oblast cell-surface antigen 2 (Trop-2), with SN-38, which is conjugated to the antibody by a cleavabl

295 et to select characteristics associated with SN status for inclusion in a multiple binary logistic mo

296 invasion were significantly associated with SN status.

297 r for tumor margin demarcation combined with SN biopsy.

298 that tumor margin demarcation combined with SN identification has potential but that some practical

299 ximally accelerated cell death combined with SN-38 at 17 nM.

300 of mainly tannins which could interact with SNs proteins and honey proline.