ライフサイエンスコーパス: SPD

1 SPD had better prediction; (AUC: 0.713; 95%CI: 0.646-0.7

2 SPD has stereoselective PK and PD.

3 SPD is inhibitory at concentrations of >=10 muM and is f

4 SPD peak amplitudes between EO-EC were smaller in opioid

5 SPD plays vital roles in cell function and survival, inc

6 SPD stereoisomers (141 or 283 mg/kg) did not cause NTD.

7 SPD stereoisomers' anticonvulsant activity was comparati

8 SPD stereoisomers' PK-PD relationship was evaluated in r

9 SPD was defined as a Kessler Psychological Distress Scal

10 SPD-1 differs from other midzone components in that it i

11 SPD-1 is a conserved microtubule-bundling protein that l

12 SPD-2 and SPD-5 are codependent for localization to the

13 SPD-304 may employ a similar mechanism of inhibition.

14 SPD-5 exists mostly as a monomer but also forms complexe

15 SPD-5 is required for the centrosomal localization of ga

16 SPD-MAA decreased pIgR in HBECs.

17 SPD-MAA stimulated TGF-B in alveolar macrophage cells.

18 SPD-NGS is rapid, reproducible, simple to perform and an

19 SPD-Pilot/Project-Award-2015; AIRC-MFAG-11831.

20 carboranylbutyl) SPD/SPM derivatives (SPD-1, SPD-5, SPM-1, SPM-5); N1,N10-diethyl-N5-(4-carboranylbut

21 (K6 scores 5-12 = MPD, and K6 scores >=13 = SPD).

22 suggest that the interactions between SPD-2, SPD-5, and PLK-1 do not result in formation of cytoplasm

23 (2S,3S)-SPD, (2R,3R)-SPD, and racemic-SPD have similar anticonvulsant activit

24 (2R,3S)-SPD and (2S,3R)-SPD higher clearance led to a 50% lower plasma exposure

25 better than those of (2R,3S)-SPD and (2S,3R)-SPD, making them good candidates for development as new,

26 (2R,3S)-SPD and (2S,3R)-SPD higher clearance led to a 50% lower

27 rofile that are better than those of (2R,3S)-SPD and (2S,3R)-SPD, making them good candidates for dev

28 (2S,3S)-SPD, (2R,3R)-SPD, and racemic-SPD have similar anticonvu

29 tunable LED-based light source that achieves SPDs similar to various conditions of daylight and incan

30 In parallel experiments, adult SPD rats achieved more reversals in a probabilistic reve

31 r factor V Quebec, alpha delta-SPD, or alpha-SPD (gray platelet syndrome).

32 In anaphase, centrosomes lose PLK-1 and SPD-2 and transition to a weak, brittle state that enabl

33 In vitro, PLK-1 and SPD-2 directly protected centrosome scaffolds from force

34 lts suggest that, before anaphase, PLK-1 and SPD-2 respectively confer strength and ductility to the

35 the centriole duplication factors ZYG-1 and SPD-2.

36 PCM assembly in vivo, Polo-like kinase-1 and SPD-2/Cep192.

37 SPD-2 and SPD-5 are codependent for localization to the PCM, but S

38 r in schizophrenic (1.22 +/- 0.24 cm(3)) and SPD (1.20 +/- 0.23 cm(3)) patients than controls (1.37 +

39 s between the coiled-coil proteins SPD-5 and SPD-2 and the kinase PLK-1 are critical for PCM assembly

40 fficiently active or physically active), and SPD (determined using the 6-item Kessler Psychological D

41 g immigration status, physical activity, and SPD (Wald F4 = 14.12; P < .001).

42 f immigration status, physical activity, and SPD with all-cause mortality.

43 We measured CC16 and SPD in sputum supernatants of 84 asthmatic patients and

44 CC16 and SPD levels are upregulated in SRA and correlate with rem

45 sthmatics, we additionally measured CC16 and SPD levels in BAL and assessed smooth muscle area (SMA),

46 BAL CC16 and SPD levels were also higher in SRA compared to mild-mode

47 Induced sputum CC16 and SPD were significantly higher in patients with severe as

48 acts genetically with dynein heavy chain and SPD-5, another coiled-coil protein required for centroso

49 PUT), are the major polyamines detected, and SPD is required for growth.

50 associations between sleep disturbances and SPD were generally stronger among NH-Black and Hispanic/

51 with inactivation of PLK-1 (Polo kinase) and SPD-2 (Cep192).

52 ith 2- to 4-fold higher odds of both MPD and SPD during pregnancy among African American women.

53 ogistic regression-estimated odds of MPD and SPD during pregnancy that were associated with eviction

54 ing sites abolish PCMD-1 phosphorylation and SPD-5 binding in vitro and destabilize the PCM scaffold

55 levels of BrdU incorporation in the PUT and SPD microcosms, but not in the CTRs.

56 of FACS-sorted cells indicated that PUT- and SPD-transforming bacteria were composed similarly of a d

57 doxovir, elvucitabine, Racivir, Reverset and SPD 754 are nucleoside reverse transcriptase inhibitors

58 MPD was reported in 60% of the sample and SPD in 8%.

59 Schizophrenic and SPD patients have volume reduction in the pulvinar, but

60 investigated associations between sleep and SPD among a large, nationally representative, and racial

61 Our observations suggest that the SPV and SPD and their associated dopamine-containing socket cell

62 We show that the SPV and SPD spicule-associated sensory neurons and the spicule s

63 , and HD were also apparent, whereas TTM and SPD were largely influenced by the same latent genetic f

64 orders, and another was exclusive to TTM and SPD.

65 second factor loaded exclusively on TTM and SPD.

66 ministration of the AMPA receptor antagonist SPD 502 significantly reduced the amount of AMPA-induced

67 We applied SPD-NGS to two classes of proteases, the intracellular c

68 as a member of a novel family of ASH (ASPM, SPD-2, Hydin) domains.

69 hich interact with the ASH-RhoGAP-like (ASPM-SPD-2-Hydin homology and Rho-GTPase Activating Domain-li

70 Only the assembled SPD-5 networks, and not unassembled SPD-5 protein, funct

71 esults suggest that the interactions between SPD-2, SPD-5, and PLK-1 do not result in formation of cy

72 henomenological and comorbid overlap between SPD and trichotillomania-these disorders likely share ov

73 We also show that SAS-7 binds SPD-2 and regulates SPD-2 centriolar recruitment, while

74 215, and Aurora A kinase family members, but SPD-5 accumulates at centrosomes in mutants lacking thes

75 codependent for localization to the PCM, but SPD-2 localizes to centrioles independently of SPD-5.

76 cy for the 5' HOXD genes causes not SHFM but SPD and point to the presence of a novel locus for SHFM

77 usly reported for N5-[4-(o-carboranyl)butyl] SPD/SPM derivatives (SPD-5 and SPM-5) but similar to tho

78 the most relevant amine was PUT, followed by SPD, HIS, TYR, CAD, SPM, PHE, and SER, with the total BA

79 fee brews, the order was PUT, SPM, TYR, CAD, SPD, PHE, HIS, and SER, but at a very low level in compa

80 synthesized: N1- and N5-(4-carboranylbutyl) SPD/SPM derivatives (SPD-1, SPD-5, SPM-1, SPM-5); N1,N10

81 iated in some cases with features of classic SPD and in all cases with a novel foot phenotype.

82 Conversely, SPD-MAA stimulated TGF-B release in both HBECs and mouse

83 n each of these parameters (CPT-AC, MTH-CVc, SPD-CVc) was used to identify NCD independently.

84 Surfactant protein D (SPD) plays an important role in surfactant homeostasis a

85 protein was made from surfactant protein D (SPD), HIV-1 Gag as a test antigen, and CD40L, where SPD

86 lled with MAA-adducted surfactant protein D (SPD-MAA) were studied herein.

87 by reduced oxidative stress and DNA damage, SPD may have a protective action against chemically indu

88 instrument--the Survey of the Previous Day (SPD)--to quantify MMM as well as media-nonmedia and nonm

89 is 1 of 13 platelet storage pool deficiency (SPD) mouse mutants.

90 es on patients with storage pool deficiency (SPD) who are specifically deficient in platelet dense gr

91 ha delta, and delta storage pool deficiency (SPD), thrombasthenia, and in two new patients with isola

92 ing due to platelet storage pool deficiency (SPD).

93 ta as points on symmetric positive definite (SPD) manifold.

94 trict subset of symmetric positive definite (SPD) matrices, which form a unique mathematical structur

95 ic structure of symmetric positive definite (SPD) matrices.

96 e development of severe plastic deformation (SPD) for improving the physical and mechanical propertie

97 via changing the severe plastic deformation (SPD) processing pathway.

98 t described for factor V Quebec, alpha delta-SPD, or alpha-SPD (gray platelet syndrome).

99 hesion defect by RBCs was specific for delta-SPD; it was not observed in either von Willebrand's dise

100 deficient in platelet dense granules (delta-SPD) have suggested a role for dense granule substances,

101 sponse to all agonists was impaired in delta-SPD and was associated with an inability of these platel

102 effect of RBCs on platelet adhesion in delta-SPD appears to be chemical rather than physical in natur

103 impaired prothrombinase activation in delta-SPD platelets were corrected by the addition of adenosin

104 ysis of the type of adhesion defect in delta-SPD suggest that ADP may be involved in the process requ

105 e more complicated Previous studies in delta-SPD suggested an adhesion defect that was strongly influ

106 in platelet adhesion in patients with delta-SPD, most significantly at Hct values of 30% to 40%.

107 n normal subjects and in patients with delta-SPD.

108 latory molecule produced by B. dendrobatidis SPD and its precursor, putrescine (PUT), are the major p

109 e study of selective peripheral denervation (SPD) in cervical dystonia (CD) patients with primary or

110 Spectral power density (SPD) indexed by electroencephalogram (EEG) recordings ha

111 N5-(4-carboranylbutyl) SPD/SPM derivatives (SPD-1, SPD-5, SPM-1, SPM-5); N1,N10-diethyl-N5-(4-carbor

112 [4-(o-carboranyl)butyl] SPD/SPM derivatives (SPD-5 and SPM-5) but similar to those of native SPD and

113 Substance P derivatives (SPDs) induced apoptosis and inhibited growth of lung can

114 Split Plot Design (SPD) employing three maize varieties (Shalimar Maize Hyb

115 Scotopic pupil diameter (SPD), pupil contraction amplitude, and velocity of pupil

116 istory within the solar protoplanetary disk (SPD).

117 s released by Shockley partial dislocations (SPDs) accompanied by the formation of stacking faults.

118 n men with schizotypal personality disorder (SPD) compared with healthy control (HC) subjects.

119 Schizotypal personality disorder (SPD) is a schizophrenia-spectrum disorder that is biolog

120 Schizotypal personality disorder (SPD) shares social deficits and cognitive impairment wit

121 s, 12 with schizotypal personality disorder (SPD), and 12 normal control subjects.

122 as having schizotypal personality disorder (SPD).

123 ients with schizotypal personality disorder (SPD).

124 Skin picking disorder (SPD) is characterized by the repetitive and compulsive p

125 Excoriation (skin-picking) disorder (SPD) is a disabling, underrecognized condition in which

126 platelet storage pool deficiency disorders (SPD).

127 s conducted through short path distillation (SPD) of an acylglycerol mixture (containing 67% MAGs) pr

128 en sleep and serious psychological distress (SPD) have lacked racial/ethnic diversity and generalizab

129 ight's evolving spectral power distribution (SPD).

130 lower mantle to a single-perovskite domain (SPD; calcium-rich bridgmanite) at the deeper lower mantl

131 cture of the FAM111A serine protease domain (SPD) is unknown.

132 eins are required for centriole duplication: SPD-2, ZYG-1, SAS-5, SAS-6, and SAS-4.

133 observed that one daily hour of play during SPD partially rescued the behavioral performance in the

134 Providing one daily hour of play during SPD partially rescued the cognitive skills in these rats

135 However, the molecular basis for SPD-2 centriolar localization remains unknown.

136 pharmacologic or psychological treatment for SPD.

137 Furthermore, SPD-5 interacts genetically with a dynein heavy chain.

138 s of sleep duration were more likely to have SPD, and the magnitude of the association was strongest

139 in schizophrenia, the similar patterns of HC-SPD age-related GMV reduction suggest that SPD showed no

140 lly and molecularly accurate model for human SPD.

141 ; the same type of expansion occurs in human SPD mutations.

142 Thus, we have identified SPD-2 as a factor critical for the two basic functions o

143 In SPD, no clear directionality emerged between ACC and PC,

144 in these responses in thrombasthenia, as in SPD, further show the close association between the gene

145 of both NRF2 and p62-dependent autophagy in SPD-mediated liver protection was confirmed using a carb

146 etry showed that the neocortical deficits in SPD were especially prominent in the left superior and m

147 e decarboxylase, the rate-limiting enzyme in SPD synthesis, did not affect cancer cell growth in vitr

148 Neurocognitive findings in SPD implicate difficulty with response inhibition (suppr

149 nalyses of a phosphorylated region (PReM) in SPD-5 revealed a helical hairpin that dimerizes to form

150 e, pulvinar and MDN together were reduced in SPD (0.14%) and schizophrenic (0.15%) patients vs contro

151 rable outcome had a significant reduction in SPD compared with patients with a poor prognosis.

152 reviews are not statistically significant in SPD.

153 An adenovirus 5 (Ad5) vaccine incorporating SPD-Gag-CD40L was much stronger than Ad5 expressing Gag

154 eramide than the 4NQO group, which indicated SPD's ability to prevent the 4NQO-induced carcinogenesis

155 Scale, with a total score of >=13 indicating SPD).

156 macokinetic (PK) analysis of four individual SPD stereoisomers.

157 Interestingly, SPD-2 interacts genetically with dynein heavy chain and

158 By embedding motion data into SPD manifolds, we integrate unsupervised K-Nearest Neigh

159 ment, while SAS-7 centriolar localization is SPD-2-independent.

160 To our knowledge, SPD-3 is the first example of a link between mitochondri

161 l)-o-carboranyl]butyl inverted question mark SPD/SPM derivatives (ASPD-5, ASPM-5, DHSPD-5, and DHSPM-

162 During maturation, SPD-2 gradually accumulates at the centrosome in a manne

163 -5 and SPM-5) but similar to those of native SPD and SPM.

164 We show that SPD-2 is monomeric, and neither SPD-2 nor SPD-5 exists in complex with PLK-1.

165 No SPD has been localized to these regions of either the mo

166 nm), the stacking faults still exist, but no SPDs are found.

167 at SPD-2 is monomeric, and neither SPD-2 nor SPD-5 exists in complex with PLK-1.

168 Exogenous administration of SPD drove tumor aggressiveness in an immune-dependent ma

169 es have reported that oral administration of SPD protects against liver fibrosis and hepatocarcinogen

170 gh a systematic screening of 39 analogues of SPD-304, a dual inhibitor of tumor necrosis factor (TNF)

171 e of NRF2 in mediating the health benefit of SPD, particularly in the context of liver pathologies.

172 (PRs) and 95% confidence intervals (CIs) of SPD for each sleep characteristic, overall and by race/e

173 the determinants and health consequences of SPD attributable to objectively measured sleep across ra

174 ;p62(-/-) mice, as the protective effect of SPD was significantly reduced in NRF2 or p62 single knoc

175 orspermidine (NSPD), a chemical homologue of SPD, on the structure of DNA and gene expression.

176 n, competition study showed the impotence of SPD to replace with histone H1 in histone H1-DNA complex

177 D-2 localizes to centrioles independently of SPD-5.

178 midine synthesis, enhances the inhibition of SPD at 1 and 10 muM.

179 ng mitosis in mutants with reduced levels of SPD-5, microtubules assemble but radiate from condensed

180 The midzone localization of SPD-1 is perturbed in embryos depleted of other midzone

181 t difference in the biological mechanisms of SPD and schizophrenia.

182 Multimerization of SPD-5 is driven by interactions between multiple dispers

183 analyze PLK-1-potentiated multimerization of SPD-5, the main PCM scaffold protein in C. elegans.

184 on and suppression in the pathophysiology of SPD, findings remarkably similar to those previously rep

185 The prevalence of SPD was 3.4% for NH-Whites, 4.1% for NH-Blacks, and 4.5%

186 The chemical structures of SPD and NSPD are different only with the number of methy

187 symptoms, we carried out the first study of SPD to assess the caudate and putamen for comparison wit

188 which are affected in a specific subgroup of SPD.

189 Teratogenicity of SPD stereoisomers was evaluated in SWV mice strain, susc

190 wn using a plasmid lacking the N terminus of SPD that produced a single trimer fusion protein.

191 ) boron compounds, are greater than those of SPD/SPM.

192 hanisms for known ADRs are proposed based on SPD off-target activities.

193 efects in Ap-3, our findings suggest that pa SPD mutants are defective in a more downstream event of

194 Whereas the mocha and pearl SPD mutants have defects in Ap-3, our findings suggest t

195 ession of the AP-3 complex leads to platelet SPD.

196 Mice exposed to 4NQO plus SPD had lower levels of C16 ceramide than the 4NQO group

197 Meanwhile, the polyamines SPD and SPM were found in all soya milks, being stable t

198 t (CPT-AC), and CVc with spatial processing (SPD-CVc).

199 solved using semidefinite graph programming (SPD) to optimally partition the graph into two structura

200 13-subjects meeting proposed SPD criteria for DSM-5 free from other current psychiatr

201 e show that the microtubule-bundling protein SPD-1/MAP-65 and BMK-1/kinesin-5 act as a brake opposing

202 te involves the conserved centrosome protein SPD-2/CEP192 and CDK activity from the mitotic cell.

203 CM assembly requires the coiled-coil protein SPD-5.

204 s, interactions between the scaffold protein SPD-5 and kinase PLK-1 are essential for PCM formation.

205 nteractions between the coiled-coil proteins SPD-5 and SPD-2 and the kinase PLK-1 are critical for PC

206 Among five known core centriole proteins, SPD-2/Cep192 is the first recruited to the site of daugh

207 s of three pericentriolar material proteins: SPD-5, the kinase aurora-A, and gamma-tubulin.

208 f two polyamines, Spermidine and Putrescine (SPD-PUT), was tested in multiple combinations (10 muM pl

209 (2S,3S)-SPD, (2R,3R)-SPD, and racemic-SPD have similar anticonvulsant activity and a PK profil

210 sec-Butylpropylacetamide (racemic-SPD) is a chiral CNS-active amide derivative of valproic

211 We found that recombinant SPD-5 could polymerize to form micrometer-sized porous n

212 at increasing physical activity and reducing SPD to mitigate disparities in mortality risk.

213 so show that SAS-7 binds SPD-2 and regulates SPD-2 centriolar recruitment, while SAS-7 centriolar loc

214 CRD)) or SP-D(n/CRD) with eight GXY repeats (SPD(GXY)(8)(n/CRD)).

215 A second SPD-NGS library (Lib hP), which displayed virtually the

216 ies coupled with next generation sequencing (SPD-NGS) that allows up to 10,000-fold deeper sequence c

217 Spermidine (SPD), a naturally occurring polyamine, has been recogniz

218 The induction of autophagy by spermidine (SPD) treatment reduced the severity of lesions and the i

219 te that the polyamine metabolite spermidine (SPD) was elevated in the GBM tumor microenvironment.

220 mpound, i.e. putrescine (PUT) or spermidine (SPD), or with no addition as controls (CTRs).

221 identification of the polyamine spermidine (SPD) as another significant immunomodulatory molecule pr

222 effects of trivalent polyamines, spermidine (SPD) and norspermidine (NSPD), a chemical homologue of S

223 enic polyamines; spermine (SPM), spermidine (SPD) and putrescine (PUT), or electrochemical investigat

224 of new boron-containing spermidine/spermine (SPD/SPM) analogues have been synthesized: N1- and N5-(4-

225 New boron-containing spermidine/spermine (SPD/SPM) analogues have been synthesized: N5-[4-(2-amino

226 4.17 x 10(5) and 1.46 x 10(5)M(-1) for SPM, SPD and PUT, respectively, which correlated well with th

227 The fabricated biosensor responded to SPM, SPD and PUT over an extended dynamic range of 0.04-100 m

228 In the unassembled state, SPD-5 exhibits numerous intramolecular crosslinks that a

229 A stepwise SPD process in a UIC KDL 5 system (vacuum 10(-3)mbar, fe

230 specially those with terminal N-substituted (SPD-1, SPM-1) boron compounds, are greater than those of

231 Synpolydactyly (SPD) is a dominantly inherited congenital limb malformat

232 Synpolydactyly (SPD) is a limb malformation that shows a characteristic

233 Human synpolydactyly (SPD) is an inherited congenital limb malformation caused

234 inherited limb malformation synpolydactyly (SPD).

235 shows 4-5 times higher affinity to DNA than SPD.

236 e potent with a longer plasma half-life than SPDs.

237 uorescence microscopy, it becomes clear that SPD tends to align DNA orientation, whereas NSPD induces

238 We conclude that SPD is an effective treatment for patients with secondar

239 We further demonstrate that SPD confers liver protection by enhancing NRF2 signaling

240 Our results demonstrate that SPD functions as a cancer cell-derived metabolite that d

241 In this study, we demonstrate that SPD is a noncanonical NRF2 inducer, and that MAP1S is a

242 This work also demonstrated that SPD is able to concentrate n-3 PUFAs in MAG form by dist

243 Surprisingly, we also find that SPD-2 is required for centrosome duplication and genetic

244 Surprisingly, we found that SPD-3GFP localized to mitochondria.

245 It was hypothesized that SPD would be associated with reduced fractional anisotro

246 The comet assay indicated that SPD reduced 4NQO-induced DNA damage, likely related to t

247 We propose that SPD-5, along with dynein, is required for centrosome mat

248 We show that SPD-2 is monomeric, and neither SPD-2 nor SPD-5 exists i

249 C-SPD age-related GMV reduction suggest that SPD showed no progressive GMV loss, pointing to an impor

250 one components are depleted, suggesting that SPD-1 may play an early role in the pathway of midzone a

251 Furthermore, the SPD should have been thicker in the past when the mantle

252 In the SPD group, larger lateral ventricle volumes correlated w

253 Lung-specific overexpression of D/A in the SPD(-/-) mouse resulted in hetero-oligomer formation wit

254 In the SPD, participants first recalled (typed) what they did d

255 ming of formation of their precursors in the SPD.

256 Euclidean-based approaches by leveraging the SPD manifold's ability to preserve motion variations and

257 In later parts of the SPD, participants analysed activities and their timing a

258 rms the SPD sheath cell into its sister, the SPD neuron.

259 ratus in 68 avian species, and show that the SPD was present in virtually all of the soaring taxa inv

260 ed by the Wnt receptor LIN-17 transforms the SPD sheath cell into its sister, the SPD neuron.

261 erations of the correlation matrix under the SPD geometry may not necessarily be coherent (i.e., the

262 ms into the Pd shell layers to eliminate the SPDs.

263 c NPs were annealed at 523 K for 10 min, the SPDs at the surface of the NPs coated with a single atom

264 However, these SPDs had low potency and short half-lives.

265 This SPD-Gag-CD40L protein activated CD40-bearing cells and b

266 f active (AC50 < 1 uM) results are unique to SPD, with bias towards higher activity in public resourc

267 em may prove a beneficial target in treating SPD and other compulsive behaviors.

268 tment combinations, the combination of 20 uM SPD and 20 uM PUT was found to be the optimum, based on

269 ssembled SPD-5 networks, and not unassembled SPD-5 protein, functioned as a scaffold for other PCM pr

270 In two unrelated SPD families, this duplication was directly transmitted

271 ized neural network, designed for vectorized SPD data, outperforms state-of-the-art methods on three

272 apturing hierarchical patterns in vectorized SPD data.

273 ingressing furrow in wild-type embryos; when SPD-1 is depleted, there is no visible midzone, and only

274 IV-1 Gag as a test antigen, and CD40L, where SPD serves as a scaffold for the multitrimer protein com

275 nhibition on in vitro gene expression, where SPD shows definitely higher potency in enhancement but N

276 CC16 BAL levels correlated with ED, while SPD BAL levels correlated with SMA and RBM.

277 o June 26, 2015) and included 66 adults with SPD.

278 sleep health was positively associated with SPD, and the magnitude of the association was generally

279 Here we report a father and daughter with SPD who carry a 117-kb microdeletion at the 5' end of th

280 t in the combined cohort of individuals with SPD and schizophrenia.

281 study involved 54 neuroleptic-naive men with SPD and 54 male HC subjects aged 18 to 55 years recruite

282 Men with SPD showed global and widespread smaller regional GMV.

283 lated GMV reductions are greater in men with SPD than HC subjects, providing presumptive evidence on

284 voxelwise analysis showed participants with SPD had significantly smaller GMV in the left superior t

285 ophrenic patients (n = 10) and patients with SPD (n = 10) both of whom never received medication were

286 Patients with SPD (n = 16), schizophrenic patients (n = 42), and age-

287 ver 30 min) in medication-free patients with SPD (n=16) who met the criteria for cognitive impairment

288 was significantly elevated in patients with SPD and reduced in schizophrenic patients.

289 s significantly larger than in patients with SPD and significantly smaller than in schizophrenic pati

290 All of the patients with SPD and subsamples of the schizophrenic patients (n = 27

291 for the putamen being found in patients with SPD for the ventral slice and the lowest value for the r

292 y be related to the sparing of patients with SPD from frank psychosis.

293 Patients with SPD showed reduced volume and elevated relative glucose

294 sotropy did not correlate significantly with SPD disease severity, or depressive or anxiety scores.

295 Subjects with SPD exhibited significantly reduced fractional anisotrop

296 ve relationships with age in both those with SPD and HC subjects, without any group by age interactio

297 tracheal epithelial cells were treated with SPD-MAA and sIgA and TGF-B was measured.

298 were attenuated by pre- or co-treatment with SPD (100 mg/ml).

299 SF relative volumes were found in women with SPD compared with controls.

300 emographic reconstructions and modeling with SPDs, however, are stymied by a lack of accepted methods