ライフサイエンスコーパス: SPM

1 SPM is applicable for analyses of longitudinal data in m

2 SPM single subject and group analysis compared to the co

3 SPM single-subject analysis, and a group analysis in com

4 SPM's alternative pre-whitening method, FAST, performed

5 SPMs actively promote resolution of inflammation by cont

6 SPMs are also decreased in advanced plaques of fat-fed L

7 SPMs have potential to serve as targeted therapeutic age

8 SPMs, which include lipoxins, demonstrate comprehensive

9 Me(2)SPM administration to mice revealed that Me(2)SPM signif

10 Me(2)SPM can functionally substitute for the native polyamine

11 ate that the (R,R)-diastereomer of 1,12-Me(2)SPM represents a promising lead compound in developing a

12 PM administration to mice revealed that Me(2)SPM significantly decreases spermidine levels in multipl

13 Importantly, Me(2)SPM was detectable in brain tissue, the organ most affec

14 e mimetic, (R,R)-1,12-dimethylspermine (Me(2)SPM), to reduce the intracellular spermidine pools of SR

15 blasts from SRS patients can accumulate Me(2)SPM, resulting in significantly decreased spermidine lev

16 tent of dissemination of MBLs such as VIM-2, SPM-1 and NDM among Gram-negative pathogens cannot be ex

17 mib) were associated with a lower risk for a SPM in comparison with CA.

18 In this paper, the result of integrating a SPM module into a ToF-SIMS system is presented illustrat

19 Resolvin E1 (RvE1), a SPM derived from omega-3 fatty acids, has been reported

20 All SPM and GES isolates, targets not present in commerciall

21 Altered SPMs and age-associated decrease in brain DHA in APOE4 f

22 few studies have compared survival after an SPM to survival of the same cancer occurring as first pr

23 Overall, 5-year survival after an SPM was 33.1% lower for children, 20.2% lower for AYAs,

24 Administration of resolvin D4 (RvD4), an SPM that was enriched at the natural onset of thrombus r

25 ing humoral immunity, potentially through an SPM-mediated mechanism.

26 ess cost explains the exclusion of VIM-2 and SPM-1 from Escherichia coli and Acinetobacter baumannii,

27 n, leading to decreased acetyl-S565 COX2 and SPM production.

28 tributes to 15-lipoxygenase-1 expression and SPM formation in M2-like MDM.

29 of residual autocorrelated noise in FSL and SPM leads to heavily confounded first level results, par

30 ging (fMRI) analysis packages: AFNI, FSL and SPM, with regard to temporal autocorrelation modeling.

31 than the autocorrelation modeling of FSL and SPM.

32 ar required to differentiate GES-, IMP-, and SPM-producing isolates.

33 ed from n-3 PUFAs, including epoxy-PUFAs and SPM-precursors, increase linearly with elevated intake o

34 er than those of Sigmaparabens in sludge and SPM.

35 ) (LTB(4)), prostaglandin E(2) (PGE(2)), and SPMs are expressed on lymphocytes.

36 and time-period, the risk of developing any SPM or any haematological malignancy was significantly r

37 aspirin to n-3 fatty acids did not alter any SPMs in either group.

38 We used archived SPM samples of the German Environmental Specimen Bank (E

39 trospective information (when using archived SPM) for fish monitoring, complementing the currently us

40 gered specialized proresolving mediators (AT-SPMs), such as aspirin-triggered resolvins (AT-RvDs) and

41 These AT-SPMs are active at considerably lower concentrations tha

42 Augmented SPMs were continued.

43 bridge for the inorganic adsorption between SPM-MWCNTs and sediment.

44 (ToF) SIMS) with means to alternate between SPM and SIMS measurements, is one approach to meet that

45 natural ingredients for enhancing bioactive SPM concentrations in bread.

46 dose-dependent increase in peripheral blood SPM concentration.

47 oil leads to an increase in peripheral blood SPM concentrations and reprograms peripheral blood cells

48 Survival by SPM status was significantly worse in younger vs older p

49 ine was PUT, followed by SPD, HIS, TYR, CAD, SPM, PHE, and SER, with the total BAs content decreasing

50 We conclude that highly phagocytic, CD138(+) SPM-like cells with an anti-inflammatory phenotype may p

51 First, characteristic SPM images of nanoparticles are collected in aqueous sus

52 Assessment of circulating SPM concentrations 6-months after treatment initiation e

53 usion (I/R) injury, to increased circulating SPMs and decreased remote organ inflammation.

54 For the most common SPMs in AYAs, the absolute difference in 5-year survival

55 15-lipoxygenase-1 expression and consequent SPM biosynthesis involving ERK-1/2 during M2 polarizatio

56 s administration of MaR1, a highly conserved SPM, propagated inflammatory resolution after SCI, as re

57 Together, these findings identify a CSF SPM signature that is associated with disease severity a

58 murine peritonitis accompanied by decreased SPM levels without affecting proinflammatory leukotriene

59 ing death as a competing risk for developing SPM.

60 1); surface sediment: 90.2-70.3 ng g(-1) dw; SPMs: 187.7-157.6 ng g(-1) dw).

61 omes observed in longitudinal studies, i.e., SPM relates the stochastic dynamics of variables (e.g.,

62 t not overweight was associated with earlier SPM emergence.

63 a platform for developing more elaborate EC-SPM setups.

64 ray scan patterns, only found on high-end EC-SPM setups such as hopping mode scan, were easily implem

65 lectrochemical-scanning probe microscope (EC-SPM) is presented.

66 f" parts to deliver a simple yet powerful EC-SPM equipment capable of performing simple space-resolve

67 we found a temporal regulation of endogenous SPMs during self-resolving inflammatory arthritis.

68 both ECs and VSMCs, potentially facilitating SPM biosynthesis.

69 sciences to the study of natural flocculated SPM filling both this spatial and dimensional gap.

70 0(5), 4.17 x 10(5) and 1.46 x 10(5)M(-1) for SPM, SPD and PUT, respectively, which correlated well wi

71 laques and offer a mechanistic rationale for SPM therapy to promote plaque stability.

72 eripheral blood cells, indicating a role for SPM in mediating the immune-directed actions of this sup

73 APOE genotype interactions were observed for SPMs expressed as a ratio of their parent compound, high

74 between pediatric overweight/obesity and FPM/SPM emergence, adjusting for age, sex, and race/ethnicit

75 t US-representative data and affirm that FPM/SPM emergence varies by race/ethnicity and sex and is po

76 ssociation of body mass index (BMI) with FPM/SPM emergence in a representative sample of US children

77 ard fMRI analysis packages (e.g., AFNI, FSL, SPM) focus on preprocessing and univariate analyses, lea

78 or paracrine signaling via locally generated SPMs in the vasculature may represent a novel homeostati

79 we recorded significantly (p < 0.05) higher SPM in the Ajkwa Estuary compared to neighboring estuari

80 dings contribute to our understanding of how SPM synthesis is regulated during the inflammatory respo

81 How SPMs regulate early LPS signaling, including activation

82 However, how SPMs are regulated during the inflammatory response, and

83 ased metabololipidomics was used to identify SPMs from n-3 polyunsaturated fatty acids in human IBD c

84 ipidomic profiling was performed to identify SPMs.

85 ls, we tested whether n-3 fatty acids impact SPM profiles in patients with CAD and promote clot remod

86 s from obese individuals exhibit an impaired SPM signature.

87 iated with heightened inflammation, impaired SPM:LT ratio, defective efferocytosis, and a decrease in

88 Importantly, SPMs lack the immunosuppressive properties of classical

89 This improved SPM and labeling protocol is an important step toward a

90 In SPM single subject analysis the hypermetabolic areas wer

91 In SPM single-subject analysis, the hypermetabolic areas we

92 esponders are maintained, with a decrease in SPM concentrations in patients resistant to DMARD therap

93 The development of hidden image in SPM is achieved by referencing of images obtained in the

94 over, gene expression of enzymes involved in SPM biosynthesis were altered in submandibular glands (S

95 We report the archaeal lipidome in SPM from diverse oceanic regimes.

96 ion toward the M2 phenotype, but its role in SPM production and lipid mediator biosynthesis in genera

97 data were processed by published routines in SPM and AFNI.

98 trum of the current Carba-R assay to include SPM, GES, and expanded IMP variants, increasing the glob

99 synthesis, whereas FLAP inhibition increases SPM levels, and 4) that the 15-LOX-1 inhibitor 3887 supp

100 es, 3) zileuton blocks resolution-initiating SPM biosynthesis, whereas FLAP inhibition increases SPM

101 he groundwork for further investigation into SPMs as alternatives to immunosuppressive therapies like

102 se in the cytoplasmic:nuclear ratio of a key SPM biosynthetic enzyme, 5-lipoxygenase.

103 sion and subcellular localization of the key SPM biosynthetic enzyme 5-lipoxygenase (LOX) in vascular

104 = 22) and Sao Paulo metallo-beta-lactamase (SPM)-producing (n = 14) isolates.

105 s, MerTK cleavage during inflammation limits SPM biosynthesis and the resolution response.

106 soft tissues may result from dysregulated LM-SPM production, and that inhibition of 15-PGDH activity

107 ork where also specialized proresolving LMs (SPMs) are formed.

108 increased AT-RvD3, RvD6, AT-PD1, and AT-LXB4 SPMs identified in Lovaza-treated patients with CAD enha

109 Second primary malignancies (SPM) were observed in 8 patients (16%).

110 s of developing second primary malignancies (SPM).

111 We used statistical parametric mapping (SPM V.12) software to compare groups and investigate vox

112 Statistical parametric mapping (SPM) is a technique with which one can delineate brain a

113 We used Statistical Parametric Mapping (SPM) to analyze time-series contributions of body segmen

114 ex, by using statistical parametric mapping (SPM).

115 group using statistical parametric mapping (SPM).

116 diment, and suspended particulate materials, SPMs) from Miankaleh Wildlife/Gorgan Bay, (Caspian Sea,

117 aeal lipids in suspended particulate matter (SPM) and sediments mainly focused on a small class of fu

118 d primarily to suspended particulate matter (SPM) at sites in areas with a high land-cover fraction o

119 s in estuarial suspended particulate matter (SPM) concentration.

120 from archived suspended particulate matter (SPM) for identifying fish species using metabarcoding, w

121 Suspended particulate matter (SPM) is present in the natural aquatic environment as lo

122 TMPP sorbed to suspended particulate matter (SPM) was 56.4% of the total mass in wastewater, which wa

123 inal effluent, suspended particulate matter (SPM), and sludge collected from two WWTPs (denoted as WW

124 to study the dynamics of the Sao Paulo MBL (SPM-1) from beta-lactam-resistant Pseudomonas aeruginosa

125 We measured SPMs and proinflammatory eicosanoid levels using liquid

126 o induced specialized proresolving mediator (SPM) biosynthetic enzymes arachidonate lipoxygenase (ALO

127 conserved specialized proresolving mediator (SPM) hosting potent anti-inflammatory and proresolving p

128 al of the specialized proresolving mediator (SPM) resolvin D1 (RvD1) to actively promote the resoluti

129 al blood specialized pro-resolving mediator (SPM) concentrations are linked with both DMARD responsiv

130 that the specialized pro-resolving mediator (SPM) Resolvin D1 (RvD1) promotes resolution of inflammat

131 y specialized pro-resolving lipid mediators (SPM) in histologically-defined stable and vulnerable reg

132 by specialized proresolving lipid mediators (SPM) that limit the host response within the affected ti

133 rolac or specialized proresolving mediators (SPM) called resolvins increases disease-free survival ra

134 ecently, specialized proresolving mediators (SPM) have been found to exert profound beneficial effect

135 produce specialized proresolving mediators (SPM; i.e., resolvins, protectins, and maresins) that pla

136 Specialized pro-resolving mediators (SPM-lipoxins, resolvins, protectins, and maresins) are p

137 am specialized proresolving lipid mediators (SPMs) 14-hydroxydocosahexaenoic acid, 17-hydroxydocosahe

138 of specialized proresolving lipid mediators (SPMs) derived from eicosapentaenoic acid (EPA) and docos

139 at specialized proresolving lipid mediators (SPMs) generated from docosahexaenoic acid (DHA) can modu

140 d specialized pro-resolving lipid mediators (SPMs), e.g., 17-hydroxy-DHA and 18-hydroxy-EPA.

141 of specialized proresolving lipid mediators (SPMs), generation of specific growth factors contributin

142 Specialized pro-resolving lipid mediators (SPMs)-resolvins (Rv), protectins (PD) and maresins (MaR)

143 se specialized proresolving lipid mediators (SPMs; lipoxins, resolvins, and protectins) stimulate res

144 thesized specialized proresolving mediators (SPMs) and proinflammatory and prothrombotic lipid mediat

145 Specialized proresolving mediators (SPMs) biosynthesized from docosahexaenoic acids (DHAs) i

146 -derived specialized proresolving mediators (SPMs) in 2-, 9-, and 18-mo-old APOE3 and APOE4 male and

147 -derived specialized proresolving mediators (SPMs) play a critical role in the active resolution of i

148 ed to as specialized proresolving mediators (SPMs) play an important role in the restorative response

149 oids and specialized proresolving mediators (SPMs) regulate leukocyte function and inflammation.

150 dogenous specialized proresolving mediators (SPMs) that actively stimulate resolution of inflammation

151 zed that specialized proresolving mediators (SPMs), a class of immune regulators promoting resolution

152 Specialized proresolving mediators (SPMs), identified in recent years, are endogenous mediat

153 , termed specialized proresolving mediators (SPMs), in the cerebrospinal fluid (CSF) of adults with T

154 Specialized proresolving mediators (SPMs), including resolvin D2 (RvD2), promote the active

155 Specialized proresolving mediators (SPMs), like resolvins (Rvs), regulate secretion from CGC

156 ther two specialized proresolving mediators (SPMs), lipoxin B4 (LXB4) and resolvin E1 (RvE1), modulat

157 ecialized endogenous proresolving mediators (SPMs).

158 s called specialized proresolving mediators (SPMs).

159 between specialized pro-resolving mediators (SPMs) like resolvin D1 (RvD1) and pro-inflammatory facto

160 Specialized pro-resolving mediators (SPMs) play an important role in the resolution of inflam

161 tion of specialized pro-resolving mediators (SPMs).

162 ed to patients with single primary melanoma (SPM).

163 ecifically in the synaptic plasma membranes (SPMs) of the ipsilateral dorsal horn.

164 f the following secondary plant metabolites (SPMs): glucosinolate breakdown products, carotenoids, ch

165 Incorporating a Scanning Probe Microscope (SPM) unit which provides topography measurements with hi

166 ecular layers in surface plasmon microscopy (SPM) is suggested.

167 n the wide-field surface plasmon microscopy (SPM).

168 (HAADF-STEM) and scanning probe microscopy (SPM) suggests that piezoelectric BaTiO(3) NPs display a

169 g high resolution scanning probe microscopy (SPM), and specifically using the subset of SPM known as

170 overview of using Scanning Probe Microscopy (SPM), in particular Scanning Tunneling Microscopy (STM),

171 y high-resolution scanning probe microscopy (SPM).

172 The Stochastic Process Model (SPM) represents a general framework for modeling the joi

173 t molars (FPMs) and second permanent molars (SPMs) is an important developmental milestone influencin

174 cytic MPhi resembling small peritoneal MPhi (SPM) that expressed CD138(+) and the scavenger receptor

175 microfluidic sample preparation multiplexer (SPM) and assay procedure is developed to improve amplifi

176 microfluidic sample preparation multiplexer (SPM) has been developed and evaluated for Ebola virus de

177 rognostic ability of somatic point mutation (SPM) and copy number variation (CNV) data, gene-level an

178 erparamagnetic multiwalled carbon nanotubes (SPM-MWCNTs) in an aqueous system containing Lake Tai sed

179 nemase classes tested include VIM, IMP, NDM, SPM, KPC, and GES.

180 penemases such as KPC, NMC-A, IMI, SME, NDM, SPM, IMP, VIM, and OXA-23, 40, 48, 58, 72, 181, and 232

181 dence of second primary malignant neoplasms (SPMs) is a well-known late effect after cancer, few stud

182 The new SPM can run up to 80 assays in parallel using a pneumati

183 Environmental DNA was extracted from nine SPM samples differing in location, organic content, and

184 Therefore, the adsorption of SPM-MWCNT on the sediment should proceed through a multi

185 However, when normalized to the amount of SPM, concentrations were on average >20 times higher at

186 Application of SPM's voxel-based morphometry on the modulated images of

187 nduced inflammation, providing a new area of SPM activity to investigate in this major area of therap

188 on and prevented the related biosynthesis of SPM in response to pathogenic Escherichia coli, assessed

189 dence that the inappropriate biosynthesis of SPM in the lesioned spinal cord hampers the resolution o

190 ry in adult female mice, the biosynthesis of SPM is not induced in the lesion site up to 2 weeks afte

191 cids are precursors in the biosynthesises of SPM, the aim of the present study was to establish the r

192 Using the concept of SPM, we developed a novel technique for quantification o

193 e-cessation further enhanced the efficacy of SPM-adjuvanted P6 vaccination.

194 suggesting that the greater driving force of SPM allowed achievement of steady state.

195 n we investigate expression and functions of SPM in intestinal inflammation.

196 The impact of SPM status on cancer-specific death was examined using m

197 rapy alone (CA), the cumulative incidence of SPM overall was 1.33% at year 3.

198 se activity is required for the induction of SPM-biosynthetic pathways in human M2-like monocyte-deri

199 WT, suggesting a more stable interaction of SPM in the deep pore cavity.

200 administration of intermediate precursors of SPM biosynthesis (e.g., 17-HDHA) could be more efficient

201 Accurate prediction of SPM hydrodynamics requires the quantification of 3D floc

202 The ratio of SPM images allows one to visualize this redistribution a

203 rospective cohort study assessed the risk of SPM in MM using the Taiwan National Cancer Registry and

204 Few data describe the risk of SPM in patients with MM in Asia.

205 We compared risks of SPM using a cause-specific Cox regression model consider

206 (SPM), and specifically using the subset of SPM known as noncontact atomic force microscopy (ncAFM).

207 The package estimates several versions of SPM currently available in the literature including disc

208 stimulating local endogenous biosynthesis of SPMs and the generation of specific growth factors.

209 LXB4 or RvE1 up-regulated concentrations of SPMs.

210 Breadmaking caused a decrease of SPMs.

211 These results suggest that delivery of SPMs, specifically RvD4, modulates the severity of throm

212 The elevation of SPMs noted here appeared to be sex mediated, meaning tha

213 droxy-DHA (HDHA) and unbalanced formation of SPMs (in particular D-series Rvs) accompanied by enhance

214 To assess the survival impact of SPMs in adolescents and young adults (AYAs) (15-39 years

215 clusion and Relevance: The adverse impact of SPMs on survival is substantial for AYAs and may partial

216 ce also have increased circulating levels of SPMs and less lung injury after I/R.

217 The levels of SPMs, particularly resolvin D1 (RvD1), and the ratio of

218 ese, the morbidly obese had higher levels of SPMs-RvD3, RvD4 and PD1-and white blood cells (WBC) and

219 infections and an unexpected high number of SPMs, which may limit its use.

220 with decreased cortical DHA and a number of SPMs, which together may contribute to the development o

221 a decrease in the production of a number of SPMs.

222 may be responsible for the overabundance of SPMs that we observed.

223 At the same time, a distinct pattern of SPMs was profiled, with an increase for D-series resolvi

224 ysis, we identified unbalanced production of SPMs (i.e., D- and E-series resolvins, protectin D1, mar

225 tical factor for the deficient production of SPMs by obese leukocytes because the formation of D-seri

226 stinal inflammation and to study profiles of SPMs, we generated a novel animal model of lymphatic obs

227 cularly resolvin D1 (RvD1), and the ratio of SPMs to pro-inflammatory leukotriene B4 (LTB4), are sign

228 s research highlights the beneficial role of SPMs in pulmonary bacterial infections and provides the

229 ssolved organic matter (DOM) and sediment on SPM-MWCNTs under various conditions and the interaction

230 The impact of breadmaking on SPMs in microgreens (7 days) and leaves (14 days) in pak

231 all and for each cancer occurring as a PM or SPM by age at diagnosis.

232 ed as having 14 cancers occurring as a PM or SPM were included.

233 dant prostaglandins and leukotrienes (M1) or SPMs (M2).

234 Overall, SPMs show promise as novel vaccine adjuvants with the ab

235 e error rates for the fMRI software packages SPM, FSL, and AFNI, as well as a nonparametric permutati

236 The impact of a particular SPM diagnosis on survival may inform age-specific preven

237 nship between supplementation and peripheral SPM concentrations.

238 d the relationship between changes in plasma SPM concentrations and peripheral blood platelet and leu

239 gs elucidate the potential utility of plasma SPM concentrations as biomarkers of DMARD responsiveness

240 Assessment of plasma SPM concentrations using lipid mediator profiling demons

241 fy subsets of large PMs (LPM) and small PMs (SPM), which differed in granularity and maturation marke

242 lated vascular cells and tissues can produce SPMs and assessed expression and subcellular localizatio

243 and in sterile inflammation in vivo promotes SPM biosynthesis by a mechanism involving an increase in

244 ors that are active in AT and AR and propose SPMs, including 15-epi-LXA(4) or MaR1, as a potential st

245 ed by an increase and decrease of respective SPMs.

246 Results SPM exhibited excellent ICCs (0.97, 0.85, and 0.83 for T

247 r adult (>/=40 years) patients with the same SPMs.

248 have lower levels and/or absence of specific SPMs that were restored with Lovaza; these SPMs promote

249 thways, producing epimeric forms of specific SPMs, whereas other drugs can disrupt timely resolution.

250 olyamines, norspermidine (NSP) and spermine (SPM), also inhibit amphibian lymphocyte proliferation, b

251 Finally, we determined spermine (SPM) sensitivity of these uncharacterized SNPs and found

252 ost important biogenic polyamines; spermine (SPM), spermidine (SPD) and putrescine (PUT), or electroc

253 The results showed that DOM can stabilize SPM-MWCNTs by providing sterically and electrostatically

254 that the 15-LOX-1 inhibitor 3887 suppresses SPM formation in M2 macrophages.

255 Post-surgery, SPM and platelet levels decreased in morbidly obese nond

256 ral, CNV data predicts prognosis better than SPM data with the exception of the LGG cohort.

257 hitening method, FAST, performed better than SPM's default.

258 and released more tumor necrosis factor than SPMs.

259 Our results demonstrate that SPM levels were broadly elevated in plasma collected fro

260 We show here that SPM biosynthesis is increased and inflammation resolutio

261 These results indicate that SPM may provide novel therapeutic tools for the treatmen

262 Here we demonstrated for the first time that SPM is a promising source of eDNA for metabarcoding anal

263 animal studies is reviewed, indicating that SPMs are physiologic mediators and pharmacologic agonist

264 The SPM incidence per 100 person-years (95% confidence inter

265 2 representing 20 million children), and the SPM analysis included ages 9 to 13 y ( n = 2,774 represe

266 We developed an R package stpm for the SPM-methodology.

267 ing an on-chip concentration protocol in the SPM and the single molecule detection capability of the

268 There was an increase in the SPM precursors 18-HEPE, 17-HDHA, and 14-HDHA after n-3 f

269 DPA supplementation increased the SPM resolvin D5n-3DPA (RvD5n-3DPA) and maresin (MaR)-1,

270 LXB4 or RvE1 also induced expression of the SPM biosynthetic enzymes 12-lipoxygenase and 15-lipoxyge

271 te this dysregulation, the abundances of the SPM products of these enzymes (ie, RvD1 and RvD2) were u

272 ent the first software implementation of the SPM-methodology by providing an R package stpm, which wa

273 The SPMs present in self-resolving arthritic joints include

274 ulting in increased CaV2.2 expression in the SPMs of the dorsal horn.

275 ica oleracea var. sabellica) differ in their SPM concentration.

276 These SPM-like cells are not restricted to the peritoneum and

277 c SPMs that were restored with Lovaza; these SPMs promote macrophage phagocytosis of blood clots.

278 potential therapeutic applications for this SPM to promote mucosal healing in the intestine.

279 g actions of MaR1 in SCI, we found that this SPM facilitated several hallmarks of resolution of infla

280 All three SPMs were found to be effective in elevating murine Ab l

281 itors and hydrolyzed beta-lactam products to SPM-1.

282 The fabricated biosensor responded to SPM, SPD and PUT over an extended dynamic range of 0.04-

283 th muscle cells (VSMCs) converted 17-HDHA to SPMs, including resolvin D1 (RvD1) and other D-series re

284 increases acetylated COX2 and N-AS-triggered SPMs in microglia of AD mice, leading to resolution of n

285 er, these studies suggest that an underlying SPM dysregulation could be contributing to SS progressio

286 n metrics such as TBV, GMV, and WMV by using SPM.

287 alized Cox proportional hazards models using SPM and CNV data for 29 different TCGA cohorts.

288 ity together with promoting resolution using SPM represents a novel therapeutic strategy to resolve c

289 Together, they suggest that low vascular SPMs may enable progression of chronic vascular inflamma

290 In vitro, SPM treatment inhibited mouse aortic smooth muscle cell

291 LPMs vs SPMs from patients with cirrhosis had different transcri

292 hat were differentially regulated in LPMs vs SPMs, including those that regulate the cycle, metabolis

293 8% of 8-y-olds had all FPMs emerged, whereas SPM emergence varied more.

294 Therefore, we investigated whether SPM production is altered in NOD/ShiLtJ using metabololi

295 rognosis, and a unique cohort LGG, for which SPM data was more predictive than CNV data and the predi

296 Given that 3-8% of patients with SPM develop additional primary melanomas, new markers pr

297 te the risk of MPM compared to patients with SPM or healthy controls.

298 ; p = 0.0007) when compared to patients with SPM.

299 association of pathogens and pollutants with SPM and their impact on aquatic life.

300 respectively, but they were lower than with SPM.