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1 udin regimen in ACS patients with or without ST-segment elevation.
2 The MIs included 28 (19%) with ST-segment elevation.
3 with out-of-hospital cardiac arrest without ST-segment elevation.
4 iac arrest is uncertain for patients without ST-segment elevation.
5 rdiac arrest, particularly in the absence of ST-segment elevation.
7 tion, and presented more frequently with non-ST segment elevation acute coronary syndrome compared wi
8 syndrome (25%), which consisted of both non-ST-segment elevation acute coronary syndrome (14%) and S
9 ctive invasive strategy in patients with non-ST-segment elevation acute coronary syndrome (NSTE-ACS)
11 ease and stable ischaemic heart disease, non-ST-segment elevation acute coronary syndrome or ST-segme
12 ministration strategies in patients with non-ST-segment elevation acute coronary syndrome undergoing
13 ein IIb/IIIa Inhibition in Patients With Non-ST-Segment Elevation Acute Coronary Syndrome) trial.
14 e key in the management of patients with non-ST-segment elevation acute coronary syndrome, the optima
15 s consisting of patients admitted with a non-ST-segment elevation acute coronary syndrome, we constru
16 A total of 8,404 patients, with or without ST-segment elevation acute coronary syndrome, were rando
17 ein IIb/IIIa Inhibition in Patients With Non-ST-Segment Elevation Acute Coronary Syndrome; NCT0008989
18 compared with prasugrel in patients with non-ST-segment elevation acute coronary syndromes (NSTE-ACS)
19 isk of adverse outcomes versus men after non-ST-segment elevation acute coronary syndromes (NSTEACS)
20 concomitant clopidogrel presenting with non-ST-segment elevation acute coronary syndromes (NSTEACS)
21 myocardial infarction [STEMI], n = 399; non-ST-segment elevation acute coronary syndromes [NSTE-ACS]
23 (RR, 0.84 [95% CI, 0.69-1.04]; P=0.11); non-ST-segment-elevation acute coronary syndrome (RR, 0.84 [
24 esions; (3) patients who have suffered a non-ST-segment-elevation acute coronary syndrome; and (4) pa
25 vation myocardial infarction (84.4%) and non-ST-segment-elevation acute coronary syndromes (71.5%) th
26 t IL-1 receptor antagonist, in patients with ST-segment-elevation acute myocardial infarction or hear
27 Adult (>18 years) comatose survivors without ST-segment elevation after resuscitation from out-of-hos
28 as seen more commonly in admissions with non-ST-segment elevation AMI-CS, nonwhite race, and higher b
29 s with acute myocardial infarction including ST-segment elevation and non-ST-segment elevation were r
31 patency, the quantitative intracoronary ECG ST-segment elevation, and angina pectoris during the sam
33 r access complication, and one had transient ST-segment elevation from air-embolism, without sequelae
35 arction (MI) (18.7% vs. 22.5%; p < 0.001) or ST-segment elevation MI (8.5% vs. 15.7%; p < 0.001).
36 n-hospital reperfusion rates and outcomes of ST-segment elevation MI (STEMI) in renal transplant reci
38 ar risk of incident MI (0.8% annually), with ST-segment elevation MI constituting one-third of all ca
41 20% of MI cases and presented more often as ST-segment elevation MI versus MI not related to a stent
42 ntration was measured in 1,398 patients with ST-segment elevation MI who enrolled in a prospective co
43 h all-cause mortality in patients with acute ST-segment elevation MI who underwent primary percutaneo
46 % had ST-segment elevation MI, 74.3% had non-ST-segment elevation MI, and 8.9% had ventricular tachyc
47 ower EF were more likely to have experienced ST-segment elevation MI, have higher troponin values, an
50 odels were created to explore all MI and non-ST-segment-elevation MI (NSTEMI) versus ST-segment-eleva
52 tion MI hospitalization was smaller than for ST-segment-elevation MI among both women and men (-1.9%
53 rst hospitalization for AMI overall, and for ST-segment-elevation MI and non-ST-segment-elevation MI
54 me course of edema reaction in patients with ST-segment-elevation MI by CMR and assessed its implicat
55 s -8.3% (95% CI, -8.0% to -8.6%).The AAPC in ST-segment-elevation MI changed among women in 2009 (200
60 all, and for ST-segment-elevation MI and non-ST-segment-elevation MI was identified by International
63 lder than 18 years with unstable angina, non-ST segment elevation myocardial infarction (NSTEMI) or S
64 vely enrolled 27 patients with anterior wall ST segment elevation myocardial infarction (STEMI) and 4
65 elevation myocardial infarction (NSTEMI) or ST segment elevation myocardial infarction (STEMI), with
66 ina or urgent PCI for unstable angina or non-ST segment elevation myocardial infarction less than 30
67 raphy (ECG) showed signs of ongoing anterior ST segment elevation myocardial infarction, and emergent
68 raphy (ECG) showed signs of ongoing anterior ST segment elevation myocardial infarction, and emergent
70 SCAD patients more frequently presented with ST-segment elevation myocardial infarction (57% vs. 36%;
72 0; 95% CI: -0.98 to 1.58; p = 0.637) and non-ST-segment elevation myocardial infarction (ATE coeffici
73 without PCI or in those with a diagnosis of ST-segment elevation myocardial infarction (group by PCI
74 ISR PCI were less likely to present with non-ST-segment elevation myocardial infarction (MI) (18.7% v
75 cardiovascular (CV) events in patients with ST-segment elevation myocardial infarction (MI) and mult
76 complete revascularization in patients with ST-segment elevation myocardial infarction (MI) with mul
78 vation myocardial infarction (STEMI) and non-ST-segment elevation myocardial infarction (NSTEMI) pati
79 r the care and outcomes of patients with non-ST-segment elevation myocardial infarction (NSTEMI).
80 ruction commonly affects patients with acute ST-segment elevation myocardial infarction (STEMI) and i
81 cardiovascular (CV) events in patients with ST-segment elevation myocardial infarction (STEMI) and m
82 rt failure and non-fatal AMI at 12-months in ST-segment elevation myocardial infarction (STEMI) and n
86 e carry an increased risk of mortality after ST-segment elevation myocardial infarction (STEMI) compl
87 may have potentially refrained patients with ST-segment elevation myocardial infarction (STEMI) from
88 an alternative to mechanical reperfusion for ST-segment elevation myocardial infarction (STEMI) in se
90 percutaneous coronary intervention (PCI) for ST-segment elevation myocardial infarction (STEMI) may n
91 ical trial, we enrolled patients with either ST-segment elevation myocardial infarction (STEMI) or no
92 Singapore Myocardial Infarction Registry] of ST-segment elevation myocardial infarction (STEMI) patie
94 tated after cardiac arrest in the absence of ST-segment elevation myocardial infarction (STEMI) remai
96 ovement in clinical outcome in patients with ST-segment elevation myocardial infarction (STEMI) treat
101 ping to interrogate the myocardium following ST-segment elevation myocardial infarction (STEMI).
102 e epicardial and microvascular flow in acute ST-segment elevation myocardial infarction (STEMI).
104 imary percutaneous coronary intervention for ST-segment elevation myocardial infarction (STEMI).
105 prove the clinical outcomes in patients with ST-segment elevation myocardial infarction (STEMI).
106 nts standard care for treating patients with ST-segment elevation myocardial infarction (STEMI).
107 antify the edema-based area-at-risk (AAR) in ST-segment elevation myocardial infarction (STEMI).
108 moking is a well-established risk factor for ST-segment elevation myocardial infarction (STEMI); howe
109 CI full bivalirudin was administered in 612 (ST-segment elevation myocardial infarction [STEMI], n =
112 bstruction affects one-half of patients with ST-segment elevation myocardial infarction and confers a
114 revascularization in clinical scenarios with ST-segment elevation myocardial infarction and non-ST-se
115 lirudin Versus Heparin in ST-Segment and Non-ST-Segment Elevation Myocardial Infarction in Patients o
116 ation myocardial infarction (group by PCI or ST-segment elevation myocardial infarction interaction e
117 r CTA first in the diagnostic process in non-ST-segment elevation myocardial infarction is a safe gat
119 her this is true in a real-world prehospital ST-segment elevation myocardial infarction network using
122 ected patients presenting with suspected non-ST-segment elevation myocardial infarction to the emerge
123 orithm for rapid rule-out and rule-in of non-ST-segment elevation myocardial infarction using high-se
124 ment elevation myocardial infarction and non-ST-segment elevation myocardial infarction were consider
125 entation, with <=2 hours of symptoms or with ST-segment elevation myocardial infarction were excluded
126 mber 21, 2017, 440 patients, presenting with ST-segment elevation myocardial infarction within 6 h of
127 oing single-vessel FFR assessment (excluding ST-segment elevation myocardial infarction) from April 1
128 dial infarction and 87,915 patients with non-ST-segment elevation myocardial infarction, 88,542 (96.4
130 segment elevation acute coronary syndrome or ST-segment elevation myocardial infarction, with or with
135 med for stable angina (66%), followed by non-ST-segment-elevation myocardial infarction (45%), and ST
136 was more frequently used among patients with ST-segment-elevation myocardial infarction (84.4%) and n
137 ning the infarct-related artery (IRA) in non-ST-segment-elevation myocardial infarction (MI) can be c
138 cardial infarction (n=5996, 853 deaths), non-ST-segment-elevation myocardial infarction (n=5371, 901
139 ive survival was estimated for patients with ST-segment-elevation myocardial infarction (n=5996, 853
141 1.03); P=0.07; multivessel disease following ST-segment-elevation myocardial infarction (RR, 0.84 [95
144 nary intervention (PCI) for the treatment of ST-segment-elevation myocardial infarction (STEMI) has b
145 ival for overweight and obese patients after ST-segment-elevation myocardial infarction (STEMI) has b
146 imately half of the patients presenting with ST-segment-elevation myocardial infarction (STEMI) have
147 The optimal treatment strategy for treating ST-segment-elevation myocardial infarction (STEMI) in co
148 cardiovascular disease in general and acute ST-segment-elevation myocardial infarction (STEMI) in pa
149 of persistent T2 hyperintensity after acute ST-segment-elevation myocardial infarction (STEMI) is un
150 e ACS clinical presentation consisted of non-ST-segment-elevation myocardial infarction (STEMI) type
151 l patients and for patients with and without ST-segment-elevation myocardial infarction (STEMI) under
152 e direct immediate intervention and therapy, ST-segment-elevation myocardial infarction (STEMI) victi
154 ompared with clopidogrel among patients with ST-segment-elevation myocardial infarction (STEMI), thou
155 vately insured individuals hospitalized with ST-segment-elevation myocardial infarction (STEMI).
156 rse cardiac events (MACE) following an acute ST-segment-elevation myocardial infarction (STEMI).
157 rognostic utility in patients after an acute ST-segment-elevation myocardial infarction (STEMI).
158 ardiac magnetic resonance (MR) imaging after ST-segment-elevation myocardial infarction (STEMI).
159 iated with adverse outcomes in patients with ST-segment-elevation myocardial infarction (STEMI).
160 presentation and management of patients with ST-segment-elevation myocardial infarction (STEMI).
161 Participants were stratified by AMI subtype (ST-segment-elevation myocardial infarction [STEMI] and n
162 o determine if timing of cell delivery after ST-segment-elevation myocardial infarction affects recov
163 1-year cumulative survival for patients with ST-segment-elevation myocardial infarction aged >/=76 ye
164 wn to have prognostic value in patients with ST-segment-elevation myocardial infarction and cardiac a
165 mproves myocardial dynamics in patients with ST-segment-elevation myocardial infarction and is an ind
167 ete revascularization in patients with acute ST-segment-elevation myocardial infarction and multivess
168 complete revascularization in patients with ST-segment-elevation myocardial infarction and multivess
169 tructive nonculprit lesions in patients with ST-segment-elevation myocardial infarction and multivess
170 us coronary intervention in 93 patients with ST-segment-elevation myocardial infarction and multivess
171 nger age categories and in patients with non-ST-segment-elevation myocardial infarction and stable an
172 ) in the United States for patients with non-ST-segment-elevation myocardial infarction and the compa
173 rfusion times and mortality in patients with ST-segment-elevation myocardial infarction are influence
174 ions in time to reperfusion in patients with ST-segment-elevation myocardial infarction as well as in
175 riage of survivors of cardiac arrest without ST-segment-elevation myocardial infarction at the point
176 vely investigated 215 patients admitted with ST-segment-elevation myocardial infarction between April
177 ention among 95 925 patients presenting with ST-segment-elevation myocardial infarction between Janua
178 patients who have undergone primary PCI for ST-segment-elevation myocardial infarction but have resi
179 scoring model was developed and validated on ST-segment-elevation myocardial infarction cohorts from
180 infarction and multivessel disease; and the ST-segment-elevation myocardial infarction culprit vesse
182 Routine thrombus aspiration during PCI for ST-segment-elevation myocardial infarction did not impro
183 During Primary PCI), 440 patients with acute ST-segment-elevation myocardial infarction from 11 UK ho
184 by 9254 operators at 1538 hospitals for non-ST-segment-elevation myocardial infarction from 2009 to
185 cardiovascular magnetic resonance following ST-segment-elevation myocardial infarction have recently
186 ive sample of patients in China admitted for ST-segment-elevation myocardial infarction in 2001, 2006
187 patients with LV dysfunction after extensive ST-segment-elevation myocardial infarction in terms of g
188 ely 4% to 5%, a figure comparable to that of ST-segment-elevation myocardial infarction in the era of
189 igher rates of reperfusion for patients with ST-segment-elevation myocardial infarction in the interv
190 re performed in 1119 patients discharged for ST-segment-elevation myocardial infarction included in a
191 lor maintenance therapy after revascularized ST-segment-elevation myocardial infarction is associated
193 ndings support efforts to implement regional ST-segment-elevation myocardial infarction networks focu
194 , admitted with acute myocardial infarction (ST-segment-elevation myocardial infarction or type I non
195 milar number had acute presentation (49% non-ST-segment-elevation myocardial infarction or unstable a
196 d, single-blind, controlled trial randomized ST-segment-elevation myocardial infarction patients 1:1
198 r obstruction, which occurs in around 50% of ST-segment-elevation myocardial infarction patients post
199 mplete Revascularization), we randomized 627 ST-segment-elevation myocardial infarction patients to f
201 provides better prognostic stratification of ST-segment-elevation myocardial infarction patients trea
202 and led to a relevant decrease in TA use in ST-segment-elevation myocardial infarction patients unde
205 mized, Phase III STEM-AMI OUTCOME trial, 161 ST-segment-elevation myocardial infarction patients were
206 ND In this prospective study, 88 consecutive ST-segment-elevation myocardial infarction patients were
207 tions and the documented survival benefit in ST-segment-elevation myocardial infarction patients who
208 of a prospective trial which randomized 696 ST-segment-elevation myocardial infarction patients with
210 reserve-guided complete revascularization in ST-segment-elevation myocardial infarction patients with
211 selective use of CMR for risk prediction in ST-segment-elevation myocardial infarction patients with
212 METHODS AND Hospitals (n=167 with 23 498 ST-segment-elevation myocardial infarction patients) wer
214 its robust risk stratification of discharged ST-segment-elevation myocardial infarction patients, but
215 eluting stent (SES) at 12-month follow-up in ST-segment-elevation myocardial infarction patients.
216 ognostic value over clinical risk factors in ST-segment-elevation myocardial infarction patients.
217 elop and validate a CMR-based risk score for ST-segment-elevation myocardial infarction patients.
218 pectively collected data on 5665 consecutive ST-segment-elevation myocardial infarction PCI patients
219 aseline differences, clinical outcomes after ST-segment-elevation myocardial infarction PCI were simi
220 oup analysis, we included 1653 patients with ST-segment-elevation myocardial infarction randomized to
223 available process measure for patients with ST-segment-elevation myocardial infarction requiring int
224 2017, treatment times of 2063 patients with ST-segment-elevation myocardial infarction requiring int
225 n 1 h after pPCI in patients presenting with ST-segment-elevation myocardial infarction scheduled for
226 r is recommended in patients presenting with ST-segment-elevation myocardial infarction scheduled to
227 in the Netherlands, enrolling patients with ST-segment-elevation myocardial infarction scheduled to
228 e analysis of bivalirudin versus UFH for non-ST-segment-elevation myocardial infarction to date, biva
229 MVO in a cohort of consecutive patients with ST-segment-elevation myocardial infarction treated with
230 cardiac mortality in patients with high-risk ST-segment-elevation myocardial infarction treated with
231 econdary prevention therapy in patients with ST-segment-elevation myocardial infarction undergoing pe
232 7, we included all consecutive patients with ST-segment-elevation myocardial infarction undergoing pe
233 d with reduced infarct size in patients with ST-segment-elevation myocardial infarction undergoing pe
234 ects of NAC on infarct size in patients with ST-segment-elevation myocardial infarction undergoing pe
235 tor antagonists are delayed in patients with ST-segment-elevation myocardial infarction undergoing pr
238 platelet aggregation (IPA) in patients with ST-segment-elevation myocardial infarction undergoing pr
239 n a head-to-head comparison in patients with ST-segment-elevation myocardial infarction undergoing pr
240 ticagrelor versus prasugrel in patients with ST-segment-elevation myocardial infarction undergoing pr
242 g with an acute coronary syndrome, including ST-segment-elevation myocardial infarction were enrolled
245 A total of 122 P2Y(12)-naive patients with ST-segment-elevation myocardial infarction were randomly
246 gh-risk patients with unstable angina or non-ST-segment-elevation myocardial infarction who did not u
247 cept for those (n=78, 23.6%) presenting with ST-segment-elevation myocardial infarction who underwent
248 ospective cohort of unselected patients with ST-segment-elevation myocardial infarction with paired a
249 levation myocardial infarction or type I non-ST-segment-elevation myocardial infarction), and own a s
252 ccurs in a large proportion of patients with ST-segment-elevation myocardial infarction, adversely af
253 reperfusion strategy for patients with acute ST-segment-elevation myocardial infarction, aiming at re
254 2015 included 120 cases; 75% presented with ST-segment-elevation myocardial infarction, and 80% had
255 O-PCI) in patients with multivessel disease, ST-segment-elevation myocardial infarction, and cardioge
256 d with acute congestive heart failure or non-ST-segment-elevation myocardial infarction, and had mult
257 ikely to include high-risk features, such as ST-segment-elevation myocardial infarction, cardiogenic
260 r ejection fraction <30% within 4 days after ST-segment-elevation myocardial infarction, primary vent
261 henotype were observed, particularly for non-ST-segment-elevation myocardial infarction, reflecting a
262 utaneous coronary intervention for extensive ST-segment-elevation myocardial infarction, the effects
264 patients with cardiogenic shock complicating ST-segment-elevation myocardial infarction, there may be
266 going percutaneous coronary intervention for ST-segment-elevation myocardial infarction, we did not f
290 versus placebo in 120 patients with anterior ST-segment-elevation myocardial infarctions resulting in
291 ing analyses of patients with large anterior ST-segment-elevation myocardial infarctions, bone marrow
292 in patients with unstable angina (UA) or non-ST-segment elevation (NSTE) myocardial infarction (MI).
293 patients resuscitated from VF/pVT OHCA with ST-segment elevation on their postresuscitation ECG, the
294 tients resuscitated from VF/pVT OHCA without ST-segment elevation on their postresuscitation ECG, the
295 rgency coronary angiography in patients with ST-segment elevations on ECG after OHCA, while the role
298 ith acute coronary syndromes with or without ST-segment elevation, the incidence of death, myocardial
300 ction including ST-segment elevation and non-ST-segment elevation were recruited between February 201