ライフサイエンスコーパス: ST

1 ST and LRST increased proanthocyanidin polymerization an

2 ST mostly affected wine characteristics by increasing al

3 ST recruitment of STRIPAK facilitates PP2A-mediated deph

4 ST-13578 is a single-locus variant of ST-1504, previousl

5 STing determines the sequence type of traditional 7-gene

6 STing shows superior accuracy and performance for standa

7 allergen sensitization at 4 years: NS 19.1%, ST 28.6%, FA 44.4%.

8 e specifically expressed in the stem, and 10 STs responded to the diurnal rhythm.

9 oral expression patterns in leaf tissues, 10 STs were specifically expressed in the stem, and 10 STs

10 Here, 105 ST genes were identified and clustered into eight subfam

11 ; eczema during the first 12 months: NS 19%, ST 32%, FA 64%; and aeroallergen sensitization at 4 year

12 This event, FRB 200428 (ST 200428A), was detected on 28 April 2020 by the STARE2

13 pe of these strains to be E:P1.21-7,16:F5-36:ST-1157 (cc1157); when analyzed phylogenetically, compar

14 65.6%; pet ownership at 12 months: NS 71.5%, ST 81.5%, FA 45.8%; eczema during the first 12 months: N

15 The results clearly demonstrated 50 ST genes had different spatiotemporal expression pattern

16 FA group: family history of eczema NS 44.6%, ST.

17 tes of interest varied between NS (n = 698), ST (n = 27), and FA (n = 61) groups as follows, suggesti

18 l coronary angiography, and complete (>=70%) ST-segment resolution 1 hour after pPCI.

19 .909 (91 sequence types [STs]) and 0.842 (77 STs), respectively.

20 ng the six remaining PubMLST loci (0.897, 79 STs) fails to reach the benchmark recommended for a refe

21 We performed DNA and RNA sequencing on 80 STs, 26 SMs, and 22 melanomas with Spitzoid features (MS

22 thod, has a DI of 0.914 and distinguishes 88 STs from the 448 isolates evaluated.

23 ys, 584 (69.0%) were procedural, 126 (14.9%) ST-related, and 136 (16.1%) spontaneous.

24 ients underwent tracheostomy, 58 (41%) via a ST, and 85 (59%) via a PT.

25 Escherichia coli heat-stable enterotoxin A (ST) and evaluated under conditions of static fluid, apic

26 bles were not statistically different across STs.

27 cardiovascular disease in general and acute ST-segment-elevation myocardial infarction (STEMI) in pa

28 In acute ST-segment-elevationl infarction, IMR and RRR, but not C

29 Myocardial damage due to acute ST-segment elevation myocardial infarction (STEMI) remai

30 h all-cause mortality in patients with acute ST-segment elevation MI who underwent primary percutaneo

31 During Primary PCI), 440 patients with acute ST-segment-elevation myocardial infarction from 11 UK ho

32 ardiography for a selective use of CMR after ST-segment-elevation myocardial infarction.

33 r ejection fraction <30% within 4 days after ST-segment-elevation myocardial infarction, primary vent

34 , infarct size, and clinical outcomes, after ST-segment-elevation myocardial infarction.

35 We developed the algorithm STing to provide turn-key solutions for molecular typing

36 0 (12.6) years, 67.0% were men, 81.3% had an ST-elevation myocardial infarction, and 43.3% had cardia

37 esenting with AMI, myocarditis simulating an ST-elevation myocardial infarction (STEMI) presentation,

38 interval [CI]: 0.73 to 0.89; p < 0.001), and ST and restenosis (hazard ratio: 0.74; 95% CI: 0.57 to 0

39 t-elevation myocardial infarction (45%), and ST-segment-elevation myocardial infarction (33%).

40 ical differences observed between the PT and ST groups with regard to bleeding complications (3.5% vs

41 the ratio of instantaneous values of SL and ST trends, is tightly controlled about the treadmill spe

42 ophoblast hallmarks were defined in TSCs and ST including expression of C19MC miRNAs and the macaque

43 GTs and STs both showed responses to the initial lever presentat

44 tory B subunits by the SV40 Small T antigen (ST) or mutation/deletion of PP2A subunits alters the abu

45 Admissions were classified as ST-elevation myocardial infarction (STEMI), non-STEMI (N

46 ately two- to threefold higher at HT than at ST, and HT-growth causes an ~19- to 23-fold increase in

47 Survival analysis between ST/CC groups and risk factors for fatal outcome (logisti

48 nhibit the inactivated state of the channel, ST-2262 is equipotent in a protocol that favors the rest

49 We compared STing to six of the most widely used programs for genome

50 Complete ST-segment resolution 1 hour after pPCI was present in 5

51 farct-related artery before pPCI or complete ST-segment resolution 1 h after pPCI in patients present

52 ain of C. jejuni belonging to clonal complex ST-45, with evidence of adaptation and selection in the

53 mated by the new-generation tonometer Corvis ST.

54 eline corneal SPs were measured using Corvis ST (Oculus Optikgerate GmbH).

55 results were obtained by VN-ST, improving CS-ST method by nearly 7.5%.

56 The methods CS-ST, VN-S, and VN-ST performed well for accelerating mono

57 oelectronic behavior in terms of both DeltaE(ST) and the optical energy gap of two constitutional iso

58 , owing to the small exchange energy (DeltaE(ST) < 1600 cm(-1)) in these emitters.

59 onor results in a prohibitively large DeltaE(ST) (0.68-0.77 eV).

60 (TADF) in spite of a relatively large DeltaE(ST) measured through phosphorimetry (0.33-0.37 eV).

61 ed through the accurate prediction of DeltaE(ST) using correlated wave-function-based calculations.

62 e quantum yields along with the small DeltaE(ST) suggest their potential as thermally activated delay

63 ion of chorionic gonadotropin by TSC-derived ST reflects a reprogramming of macaque TSCs to an earlie

64 its robust risk stratification of discharged ST-segment-elevation myocardial infarction patients, but

65 troduction worldwide, caused by the emerging ST-13578 clone.

66 oing single-vessel FFR assessment (excluding ST-segment elevation myocardial infarction) from April 1

67 ower EF were more likely to have experienced ST-segment elevation MI, have higher troponin values, an

68 ic richness (Ar = 4.298) were similar, and F(ST) was low (0.031 overall).

69 between Mozambique and Western Australia (F(ST) = 0.377), identifying the Indian Ocean basin as a ba

70 lso carry signatures of selection based on F(ST) scans and PCAdapt, which represents a significant en

71 e of population structure (non-significant F(ST) < 0.001) for M. alfredi along this coast.

72 Using a Q(ST)-F(ST) analysis, we found that this difference in crossover

73 older novel genes more often overlap with F(ST) outlier regions.

74 1.03); P=0.07; multivessel disease following ST-segment-elevation myocardial infarction (RR, 0.84 [95

75 cardiovascular magnetic resonance following ST-segment-elevation myocardial infarction have recently

76 The AAPC for ST-segment-elevation MI hospitalization overall was -8.3

77 rst hospitalization for AMI overall, and for ST-segment-elevation MI and non-ST-segment-elevation MI

78 incidence of MI or cardiovascular death for ST-related (1.0% vs. 2.1%; p < 0.001) and spontaneous ev

79 re performed in 1119 patients discharged for ST-segment-elevation myocardial infarction included in a

80 moking is a well-established risk factor for ST-segment elevation myocardial infarction (STEMI); howe

81 going percutaneous coronary intervention for ST-segment-elevation myocardial infarction, we did not f

82 imary percutaneous coronary intervention for ST-segment-elevation myocardial infarction.

83 patients who have undergone primary PCI for ST-segment-elevation myocardial infarction but have resi

84 K, is associated with ST and is required for ST-PP2A-induced cell transformation.

85 tion MI hospitalization was smaller than for ST-segment-elevation MI among both women and men (-1.9%

86 hat the ST-13578 outbreak clone evolved from ST-1504 by recombination.All tested strains were penicil

87 Compared to GTs, STs attributed more incentive salience to social-related

88 on signal" with consensus sequence: M(1)GXXX[ST].

89 ars, 72 (76.6%) were men, and 65 (69.1%) had ST-segment elevation myocardial infarction.

90 lonization with Blastocystis, with identical ST sequences as their respective donors.

91 s -8.3% (95% CI, -8.0% to -8.6%).The AAPC in ST-segment-elevation MI changed among women in 2009 (200

92 Wine antioxidant capacity decreased in ST wines likely by decreases in catechin and quercetin c

93 Circulating EPA in ST-segment elevation myocardial infarction (STEMI) relat

94 DEHEART trial (Bivalirudin Versus Heparin in ST-Segment and Non-ST-Segment Elevation Myocardial Infar

95 dotropin secretion was > 4000-fold higher in ST culture media compared to TSC media.

96 utic approach deserves to be investigated in ST-segment elevation MI patients.

97 rt failure and non-fatal AMI at 12-months in ST-segment elevation myocardial infarction (STEMI) and n

98 s coronary intervention improves outcomes in ST-segment-elevation myocardial infarction.

99 selective use of CMR for risk prediction in ST-segment-elevation myocardial infarction patients with

100 , levels of plasma oxytocin were measured in STs and GTs seven days after the last PCA training sessi

101 during the lever interaction period only in STs.

102 that clonal complex (CC) 446 (which includes STs 298 and 446) isolates were repeatedly cultured at 1

103 s with acute myocardial infarction including ST-segment elevation and non-ST-segment elevation were r

104 distinct (P wave, PR segment, QRS interval, ST segment, T wave, and TP segment) and 2 composite, con

105 AD, leaflet redundancy, and T-wave inversion/ST-segment depression (all p < 0.0001) but not with mitr

106 end-systolic diameter, and T-wave inversion/ST-segment depression (all p <= 0.001).

107 We hypothesized that LR, ST or their combination (LRST) would affect flavonoid co

108 ic) and IgA (mucosal) responses against LTB, ST, and LptD epitopes.

109 belonging to MLST sequence type 11 the major ST among serovar Enteritidis.

110 The MLST-STs were more widely distributed among core genome group

111 ed with a lower risk of long-term mortality, ST, and restenosis in patients undergoing PCI for stable

112 th much higher biomarker thresholds, (2) new ST-segment depression of >=1 mm for the primary and >=0.

113 (RR, 0.84 [95% CI, 0.69-1.04]; P=0.11); non-ST-segment-elevation acute coronary syndrome (RR, 0.84 [

114 let function, in two cohorts following a non-ST elevation acute coronary syndrome (NSTE-ACS) event.

115 esions; (3) patients who have suffered a non-ST-segment-elevation acute coronary syndrome; and (4) pa

116 vation myocardial infarction (STEMI) and non-ST-segment elevation myocardial infarction (NSTEMI) pati

117 lirudin Versus Heparin in ST-Segment and Non-ST-Segment Elevation Myocardial Infarction in Patients o

118 ction including ST-segment elevation and non-ST-segment elevation were recruited between February 201

119 odels were created to explore all MI and non-ST-segment-elevation MI (NSTEMI) versus ST-segment-eleva

120 all, and for ST-segment-elevation MI and non-ST-segment-elevation MI was identified by International

121 ngestive heart failure, unstable angina, non-ST-elevation myocardial infarction, syncope).

122 med for stable angina (66%), followed by non-ST-segment-elevation myocardial infarction (45%), and ST

123 ease and stable ischaemic heart disease, non-ST-segment elevation acute coronary syndrome or ST-segme

124 The AAPC for non-ST-segment-elevation MI hospitalization was smaller than

125 henotype were observed, particularly for non-ST-segment-elevation myocardial infarction, reflecting a

126 stration of P2Y(12) Receptor Blockers In Non-ST Elevated Acute Coronary Syndromes With Initial Invasi

127 erial endothelium to a greater extent in non-ST elevation MI compared with stable CAD patients.

128 ts treated for symptomatic CAD including non-ST elevation MI, along with healthy age-matched subjects

129 e ACS clinical presentation consisted of non-ST-segment-elevation myocardial infarction (STEMI) type

130 ina or urgent PCI for unstable angina or non-ST segment elevation myocardial infarction less than 30

131 in patients with unstable angina (UA) or non-ST-segment elevation (NSTE) myocardial infarction (MI).

132 gh-risk patients with unstable angina or non-ST-segment-elevation myocardial infarction who did not u

133 d with acute congestive heart failure or non-ST-segment-elevation myocardial infarction, and had mult

134 tent thrombosis (ST); or 3) spontaneous (non-ST or non-procedure-related).

135 non-invasive management of patients with non-ST elevation myocardial infarction (NSTEMI), but the tri

136 in patients aged 70 years or older with non-ST-elevation acute coronary syndrome (POPular AGE): the

137 In patients with non-ST-segment elevation acute coronary syndrome (NSTEACS),

138 ministration strategies in patients with non-ST-segment elevation acute coronary syndrome undergoing

139 ein IIb/IIIa Inhibition in Patients With Non-ST-Segment Elevation Acute Coronary Syndrome) trial.

140 e key in the management of patients with non-ST-segment elevation acute coronary syndrome, the optima

141 ein IIb/IIIa Inhibition in Patients With Non-ST-Segment Elevation Acute Coronary Syndrome; NCT0008989

142 compared with prasugrel in patients with non-ST-segment elevation acute coronary syndromes (NSTE-ACS)

143 concomitant clopidogrel presenting with non-ST-segment elevation acute coronary syndromes (NSTEACS)

144 ISR PCI were less likely to present with non-ST-segment elevation myocardial infarction (MI) (18.7% v

145 rdiac arrest, particularly in the absence of ST-segment elevation.

146 Addition of ST under flow conditions increased apical and basolatera

147 scribe the discovery and characterization of ST-2262, a Na(V)1.7 inhibitor that blocks the extracellu

148 The durations of ST and SL trends are determined to be independent of tre

149 The averages of scaling exponents of ST and SL MARS residuals are slightly smaller than 0.5.

150 Among patients that do not use any form of ST product, SRP is an effective treatment modality for t

151 n is essential for in-hospital management of ST-segment-elevation myocardial infarction.

152 he literature, the statistical properties of ST and SL time series originate from the superposition o

153 Higher rates of ST-elevation AMI-CS were noted in the Midwest and West.

154 Women had lower rates of ST-segment elevation presentation (73.0% versus 78.7%),

155 To date there are few reports of ST-segment elevation myocardial infarction (STEMI) cause

156 At all speeds considered, the trends of ST and SL are strongly correlated and are statistically

157 ST-13578 is a single-locus variant of ST-1504, previously reported globally, including in Isra

158 We hope that the adoption of STing will help to democratize microbial genomics and th

159 Our implementation of STing uses an innovative k-mer search strategy that elim

160 In total, 81% of STs harbored kinase fusions and/or truncations.

161 In Experiment 2, social behavior of STs and GTs was compared using social interaction and ch

162 Further, dopamine cells of STs showed a significantly higher proportion of cells re

163 The majority of STs and SMs have kinase fusions as primary initiating ge

164 PPV), and negative predictive value (NPV) of STs for metronidazole/ornidazole were 33.3%/16.6%, 94.2%

165 ed social 'peers' to compare the tendency of STs and GTs to attribute incentive salience to social re

166 arction (MI) (18.7% vs. 22.5%; p < 0.001) or ST-segment elevation MI (8.5% vs. 15.7%; p < 0.001).

167 segment elevation acute coronary syndrome or ST-segment elevation myocardial infarction, with or with

168 (SV) below 1 kHz, but had little effect on P(ST).

169 he cochlear input drive (P(DIFF) = P(SV) - P(ST)) before and after creating an SCD.

170 scala vestibuli (P(SV)) and scala tympani (P(ST)) at the basal cochlea in cadaveric human ears, and e

171 Four factors (chest pain, ST-elevation, absence of coronary artery disease history

172 malized concentrations of sum total PCB (PCB(ST)), seven indicator PCB congeners, and their sum (PCB(

173 egion, and class most strongly predicted PCB(ST), while similarly, region, class, and feeding locatio

174 We also discovered that PCB(ST), PCB(E7), and the seven indicator congeners all occu

175 out of 47 patients, and only 7 had positive STs.

176 Using a Q(ST)-F(ST) analysis, we found that this difference in cro

177 that in some low-risk nonimmediate reactions ST are not mandatory, especially in children.

178 d concentrations of skin test (ST) reagents, ST and drug provocation test (DPT) protocols, and diagno

179 his issue, we studied a carbapenem-resistant ST-15 K. pneumoniae isolate (Kp3380) that displayed a re

180 Angina, initial shockable rhythm, ST-segment elevation, and absence of known coronary arte

181 cardiac mortality in patients with high-risk ST-segment-elevation myocardial infarction treated with

182 Liposomes composed of RP+ST(LC)(()(low concentration)()) showed spherical and irr

183 ongissimus thoracis (LT) and Semitendinosus (ST) were evaluated after storage under air, or 70% O(2)/

184 d as nonsensitized (NS), sensitizedtolerant (ST), or food allergic (FA) based on skin prick testing a

185 m a residue of oil processing, stigmasterol (ST) and/or hydrogenated phosphatidylcholine (HPC) for th

186 re (HF), myocardial infarction (MI), stroke (ST), cardiovascular disease (CVD) and chronic kidney dis

187 ples, pre- and post-FMT, by PCR and subtype (ST) analyses.

188 implant (AADI) placed in the superotemporal (ST) versus the inferonasal (IN) quadrant in pediatric ey

189 3 groups: eyes with prior glaucoma surgery (ST), eyes with medically treated glaucoma (MT), and eyes

190 ereas upon induction of syncytiotrophoblast (ST) differentiation multinuclear structures appeared, in

191 owth of A. thaliana at standard temperature (ST; 23 degrees C) is associated with a mutation rate of

192 mpared with spatial (S) and spatio-temporal (ST) filters.

193 vailability and concentrations of skin test (ST) reagents, ST and drug provocation test (DPT) protoco

194 Skin tests (STs) and single-blind placebo-controlled drug provocatio

195 Here, we show that ST not only displaces common PP2A B subunits but also pr

196 The ST-13578 clone was identified almost exclusively (99%) i

197 umulation, but limited information about the ST family in the important sugar-yielding crop Saccharum

198 WGS analysis confirmed clonality among the ST-13578 population.

199 infarction and multivessel disease; and the ST-segment-elevation myocardial infarction culprit vesse

200 The strong coupling between the ST and SL trends ensures that the concomitant changes of

201 e-free survival was worse in MSF than in the ST and SM groups (P = 0.0073); and (iv) classification i

202 (95% confidence interval 56.5%-75.7%) in the ST group (P = .15).

203 The rate of ECC loss was the highest in the ST group compared to that in the MT and NG groups (63.8%

204 Placement of the AADI in the ST quadrant has better IOP-related outcomes and is a saf

205 yes (33%) in the IN and 96 eyes (67%) in the ST quadrants.

206 , suggesting that across these measures, the ST group was more similar to the NS than the FA group: f

207 y expansion and functional divergence of the ST gene family and will enable the further investigation

208 esence of Kpi may explain the success of the ST-15 clone.

209 -dense regions support a hypothesis that the ST-13578 outbreak clone evolved from ST-1504 by recombin

210 5% and 68% on real datasets, compared to the ST procedure.

211 DI in the IN quadrant in children unless the ST location is not a viable option.

212 d that the Kpi system is associated with the ST-15 clone.

213 icated STP13, pGlcT2, VGT3, and TMT4 are the STs with most affinity for glucose/fructose and SUT1_T1

214 Leaf removal (LR), shoot thinning (ST) and their combination (LRST) are known to increase b

215 e secondary endpoints were stent thrombosis (ST) or restenosis and peri-procedural complications.

216 ); 2) definite or probable stent thrombosis (ST); or 3) spontaneous (non-ST or non-procedure-related)

217 f the fall-back transition to Standard Time (ST) on MVA risk, further supporting the hypothesis that

218 calculate scaling exponents of stride time (ST), stride length (SL), and stride speed (SS) of human

219 Compared to ST and TG groups, optimal neural vessel density and bran

220 hat tandem duplication events contributed to ST gene expansions of two subfamilies, PLT and STP, in S

221 oth baseline conditions and with exposure to ST enterotoxin and suggests that further investigations

222 ing population capable of differentiating to STs and EVTs in vitro thereby establishing an experiment

223 heostomy (PT) or open surgical tracheostomy (ST) performed by one of three surgical services.

224 d were motivated toward it ("sign-trackers," STs).

225 identify rats that are more (sign-trackers; STs) or less (goal-trackers; GTs) prone to attribute inc

226 iatic nerve transection: Simple Transection (ST), Simple Transection & Glue (TG), Stepwise Transectio

227 r access complication, and one had transient ST-segment elevation from air-embolism, without sequelae

228 The sugar transporter (ST) family is considered to be the most important gene f

229 The optimal treatment strategy for treating ST-segment-elevation myocardial infarction (STEMI) in co

230 tations from the categories of Spitz tumors (ST) and Spitz melanoma (SM).

231 settling rate (ISR), supernatant turbidity (ST), sediment solids content (SSC), and water recovery (

232 (related to extracellular matrix turnover), ST-2, and N-terminal pro-B-type natriuretic peptide.

233 d by a previously unreported molecular type (ST-13578), initially observed in Israel in 2014.

234 ducing K. pneumoniae (KPC-KP) sequence type (ST) 16 clone in a clonal complex (CC) 258-endemic settin

235 We found that 22 of 26 sequence type (ST) 69 isolates from this collection contained an intact

236 Clonal sequence type (ST)-306 and ST615 are representative of the two major se

237 ase is Salmonella Typhimurium sequence type (ST)313.

238 re-associated lineages: MRSA (sequence type [ST] 5); VREfc (ST6); CipREc (ST131), and VREfm (clade A)

239 nces between EAEC strains of sequence types (STs) epidemiologically associated with asymptomatic carr

240 Sequence types (STs) showed a random spatial and temporal distribution.

241 ed detection of phylogroups, sequence types (STs), H30, H30Rx, and 53 virulence genes (VGs).

242 ly distributed P. aeruginosa sequence types (STs), termed "high-risk clones." We noted that clonal co

243 rnative scheme are 0.909 (91 sequence types [STs]) and 0.842 (77 STs), respectively.

244 ella Heidelberg ST15, Salmonella Typhimurium ST 19, and Salmonella II 42:r:- ST1208 that included bot

245 ates classified as a previously undocumented ST.

246 non-ST-segment-elevation MI (NSTEMI) versus ST-segment-elevation MI (STEMI) over time.

247 The methods CS-ST, VN-S, and VN-ST performed well for accelerating monoexponential T(1rh

248 The best results were obtained by VN-ST, improving CS-ST method by nearly 7.5%.

249 For biexponential mapping, the VN-ST was the best method starting with MNAD of 7.4% for AF

250 which part of the ECG signal (e.g., T-wave, ST-interval) is significantly associated with the hypogl

251 , 22 (4.7%) were procedural, 63 (13.5%) were ST-related, and 383 (81.8%) spontaneous.

252 s serve as the control manifolds about which ST and SL fluctuate.

253 vely investigated 215 patients admitted with ST-segment-elevation myocardial infarction between April

254 vated regulatory T cells was associated with ST (OR = 2.89, 95% CI 1.03-8.16, P = .045).

255 RN4, a member of STRIPAK, is associated with ST and is required for ST-PP2A-induced cell transformati

256 er growth in SU and STG groups compared with ST and TG groups.

257 and EV-associated cytokines in patients with ST-elevation myocardial infarction (STEMI).

258 Randomized Open-Label Trial in Patients with ST-Elevation Myocardial Infarction Referred for Primary

259 Among patients with ST-segment elevation MI and multivessel coronary artery

260 ntration was measured in 1,398 patients with ST-segment elevation MI who enrolled in a prospective co

261 cardiovascular (CV) events in patients with ST-segment elevation myocardial infarction (MI) and mult

262 may have potentially refrained patients with ST-segment elevation myocardial infarction (STEMI) from

263 bstruction affects one-half of patients with ST-segment elevation myocardial infarction and confers a

264 t IL-1 receptor antagonist, in patients with ST-segment-elevation acute myocardial infarction or hear

265 ompared with clopidogrel among patients with ST-segment-elevation myocardial infarction (STEMI), thou

266 presentation and management of patients with ST-segment-elevation myocardial infarction (STEMI).

267 mproves myocardial dynamics in patients with ST-segment-elevation myocardial infarction and is an ind

268 tructive nonculprit lesions in patients with ST-segment-elevation myocardial infarction and multivess

269 us coronary intervention in 93 patients with ST-segment-elevation myocardial infarction and multivess

270 Patients with ST-segment-elevation myocardial infarction developing ad

271 oup analysis, we included 1653 patients with ST-segment-elevation myocardial infarction randomized to

272 2017, treatment times of 2063 patients with ST-segment-elevation myocardial infarction requiring int

273 available process measure for patients with ST-segment-elevation myocardial infarction requiring int

274 in the Netherlands, enrolling patients with ST-segment-elevation myocardial infarction scheduled to

275 Adult patients with ST-segment-elevation myocardial infarction undergoing pr

276 In a cohort of patients with ST-segment-elevation myocardial infarction undergoing pr

277 platelet aggregation (IPA) in patients with ST-segment-elevation myocardial infarction undergoing pr

278 ticagrelor versus prasugrel in patients with ST-segment-elevation myocardial infarction undergoing pr

279 n a head-to-head comparison in patients with ST-segment-elevation myocardial infarction undergoing pr

280 In patients with ST-segment-elevation myocardial infarction undergoing pr

281 One hundred patients with ST-segment-elevation myocardial infarction were randomiz

282 A total of 122 P2Y(12)-naive patients with ST-segment-elevation myocardial infarction were randomly

283 ospective cohort of unselected patients with ST-segment-elevation myocardial infarction with paired a

284 ic ability of admission LUS in patients with ST-segment-elevation myocardial infarction.

285 raphic recanalization rates in patients with ST-segment-elevation myocardial infarction.

286 a cohort of stable reperfused patients with ST-segment-elevation myocardial infarction.

287 are largely unknown in SCAD presenting with ST-segment elevation myocardial infarction (STEMI).

288 In patients presenting with ST-segment elevation myocardial infarction and an ischem

289 mber 21, 2017, 440 patients, presenting with ST-segment elevation myocardial infarction within 6 h of

290 n 1 h after pPCI in patients presenting with ST-segment-elevation myocardial infarction scheduled for

291 r is recommended in patients presenting with ST-segment-elevation myocardial infarction scheduled to

292 For patients presenting with ST-segment-elevation myocardial infarction, national qua

293 In patients presenting with ST-segment-elevation myocardial infarction, percutaneous

294 ry intervention for patients presenting with ST-segment-elevation myocardial infarction.

295 on-human primate study, animals treated with ST-2262 exhibited reduced sensitivity to noxious heat.

296 ive genomic analysis, we found higher within-ST sequence diversity in ST615 compared with ST306 and d

297 comatose survivors of cardiac arrest without ST elevation.

298 with out-of-hospital cardiac arrest without ST-segment elevation.

299 iac arrest is uncertain for patients without ST-segment elevation.

300 Adult (>18 years) comatose survivors without ST-segment elevation after resuscitation from out-of-hos