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1 tor; Hopscotch, a JAK kinase; and Stat92E, a STAT transcription factor.
2 ng sites of the specific lineage-determining STAT transcription factor.
3 tyrosine kinases and inhibits activation of STAT transcription factors.
4 tokine-induced activation of Jak kinases and STAT transcription factors.
5 nstitutively activated and regulates several STAT transcription factors.
6 154 and cytokine signaling via NF-kappaB and STAT transcription factors.
7 of IL-6, which signals through activation of STAT transcription factors.
8 arious genes via tyrosine phosphorylation of STAT transcription factors.
9 s the tyrosine and serine phosphorylation of STAT transcription factors.
10 os induction in conjunction with the SRF and STAT transcription factors.
11 taining receptors, JAK tyrosine kinases, and STAT transcription factors.
12 tyrosine phosphorylate select members of the Stat transcription factors.
13 of its receptors, two JAK kinases and three STAT transcription factors.
14 including the JAK1 and JAK2 kinases and the STAT transcription factors.
15 the interaction between JAK kinases and the STAT transcription factors.
16 protein kinase C, intracellular calcium, and Stat transcription factors.
17 s alongside one JAK signal mediator and four STAT transcription factors.
18 nd specifically the translocation of IRF and STAT transcription factors.
19 differentiate was associated with decreased STAT transcription factor activation and failure to upre
20 ediate augmented and sustained activation of Stat transcription factors and are accordingly hyperacti
21 Ns) requires tyrosine phosphorylation of the Stat transcription factors and is mediated by the Jak fa
22 ant target for inhibitory cross-talk between STAT transcription factors and nuclear receptors in a va
23 K family are required for activation of both STAT transcription factors and the ERK/MAP kinase pathwa
24 scribes the role of Jak tyrosine kinases and Stat transcription factors and their putative function i
25 s of dominant-negative forms of JAK kinases, STAT transcription factors, and Raf-1 in transient trans
26 lated through direct arginine methylation of STAT transcription factors, and STAT3 signaling is known
30 transducers and activators of transcription (STAT) transcription factors become phosphorylated on tyr
31 ion of Polycomb response elements, chromatin states, transcription factor binding sites, RNA polymera
32 wo newly reported structures of homodimeric 'STAT' transcription factors bound to DNA reveal at atomi
33 ases (JAKs) classically signal by activating STAT transcription factors but can also regulate gene ex
34 s by which a cytokine specifically induces a Stat transcription factor can depend on the activation s
35 h relationships between inherited epigenetic states, transcription factor-DNA binding affinity thresh
36 us, IL-6 plays essential roles in activating STAT transcription factors, enhancing neutrophil recruit
38 tective immunity are guided by activation of STAT transcription factor family members in response to
41 transducers and activator of transcription (STAT) transcription factor family, a process that requir
43 the effect that inhibition of JAK2 and these STAT transcription factors has on the HG-induced increas
46 ociated Jak tyrosine kinases and cytoplasmic Stat transcription factors, including critical physiolog
47 nine phosphorylation of multiple cytoplasmic STAT transcription factors, including one, STAT5b, that
48 l transducer and activator of transcription (STAT) transcription factors mediate intracellular signal
49 New features include analysis of chromatin states, transcription factor motifs and DNase I footprin
50 egrative analysis of liver lincRNA chromatin states, transcription factor occupancy, and growth hormo
51 integrating genome-wide surveys of chromatin states, transcription factor occupancy, and tissue expre
52 subset of these genes is regulated by Rel or STAT transcription factors of the Imd and Jak-STAT pathw
53 ransduction and activators of transcription (STAT) transcription factors, or AKT and transforming gro
54 t can bind to the same DNA binding motifs as STAT transcription factors, seems to regulate TH2 respon
57 transducers and activators of transcription (STAT) transcription factors (STAT1, STAT3, and STAT5).
61 late THi differentiation through a selective STAT transcription factor that functions to regulate lin
62 n genes, and by inhibiting the activation of STAT transcription factors that are necessary for glioge
65 e receptors, induce JAK kinases and activate STAT transcription factors to stimulate the transcriptio
66 n the constitutive activity of NF-kappaB and STAT transcription factors, which drive expression of mu
67 nd on the constitutive activity of NF-kB and STAT transcription factors, which drive expression of mu
68 Jak protein tyrosine kinases and cytoplasmic STAT transcription factors, which then translocate to th