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1 din by IFN-alpha was seen, controlled by the STAT3 transcription factor.
2  NOX2 subunits in MDSC was controlled by the STAT3 transcription factor.
3 A cells through activation of Src kinase and STAT3 transcription factor.
4 CR signal transduction and the activation of STAT3 transcription factor.
5 R signal transduction, and the activation of STAT3 transcription factor.
6 phosphorylation and nuclear translocation of STAT3 transcription factor.
7 outgrowth and constitutive activation of the Stat3 transcription factor.
8 leukin 6-dependent pathway that involves the STAT3 transcription factor.
9 T3 phosphorylation and the activation of the STAT3 transcription factor.
10 ion factor, whereas IL-6 signals through the STAT3 transcription factor.
11  a number of cytokines through activation of Stat3 transcription factor.
12 y, acting at least in part through c-Jun and STAT3 transcription factors.
13 ial protein substrates such as the STAT1 and STAT3 transcription factors.
14 and Activator of Transcription 1 (STAT1) and STAT3 transcription factors.
15 bioimaging to the assessment of NFkappaB and STAT3 transcription factor activated reporters during th
16 P < .001) that was mediated by p65 NF-kB and STAT3 transcription factor activation.
17 e of inhibiting TCR signaling and activating STAT3 transcription factor activity as efficiently as wi
18 transducer and activator of transcription 3 (Stat3) transcription factors and re-enter the cell cycle
19 T was also associated with activation of the STAT3 transcription factor, and the combination of STAT3
20 the levels of several mitogenic kinases, the STAT3 transcription factor, and the stress-activated pro
21                                     CREB and STAT3 transcription factors are required for synergy and
22 decrease constitutive phosphorylation of the STAT3 transcription factor at Tyr705 in the pancreatic c
23 omatin state in ERG(+) IM cells enriched for STAT3 transcription factor binding sites and elevated ex
24 osine kinase inhibitor in combination with a STAT3 transcription factor decoy, we found enhanced anti
25 ther Bcl-2 antisense or NF-kappaB, STAT1 and STAT3 transcription factor decoys oligodeoxyribonucleoti
26 ses, related to defective MyD88 adaptor- and Stat3 transcription factor-dependent T follicular regula
27             We found that interleukin-6- and STAT3 transcription factor-dependent upregulation of Not
28 -)) showed a loss of NF-kappaB and decreased STAT3 transcription factor DNA binding activities.
29                            Activation of the Stat3 transcription factor has been reported in tubular
30                            Expression of the STAT3 transcription factor in the heart is cardioprotect
31 ed the DNA-binding activity of the STAT1 and STAT3 transcription factors in nuclear extracts and also
32                                              STAT3 transcription factor induces differentiation of na
33          Cytokines that signal via STAT1 and STAT3 transcription factors instruct decisions affecting
34                                          The STAT3 transcription factor is an important initiator of
35                                          The STAT3 transcription factor is an important regulator of
36                                          The STAT3 transcription factor is critical for cytokine sign
37               IL6-mediated activation of the STAT3 transcription factor is necessary for transformati
38 ha mRNA expression through activation of the STAT3 transcription factor pathway and subsequent expres
39 ncreases in the binding of the NF-kappaB and STAT3 transcription factors shortly after PH failed to o
40                                              STAT3 transcription factor signaling in specific T helpe
41         Furthermore, we found that FOXO1 and STAT3, transcription factors that are indispensable for
42 es the tyrosine phosphorylation of STAT1 and STAT3, transcription factors that can mediate the induct
43  implicated in ROS generation via binding of Stat3 transcription factor to a central component of the
44                 Although the tumor-promoting Stat3 transcription factor was unaltered in these tumor
45 ere activated predominantly by the STAT5 and STAT3 transcription factors, whereas transcription of Eg
46 to the cytokine IL-6 activates the oncogenic STAT3 transcription factor, which directly represses the
47 ecific interaction between PAX3-FKHR and the STAT3 transcription factor, which results in a dramatic