ライフサイエンスコーパス: SUI

1 SUI and PD are common conditions in women postpartum tha

2 Ten years after SUI mesh surgery, 1 of every 30 women may require a seco

3 (POP sling group), and sling for SUI alone (SUI sling group).

4 ith LLD responded differently to LLD-CBT and SUI, depending on their baseline PPH score.

5 The combined use of POP mesh and SUI mesh sling was associated with the highest erosion a

6 in the pelvic floor for treatment of POP and SUI.

7 y exposures of interest, multiple mesh-based SUI procedures increased the risk for complications (HR,

8 h possible directions of association between SUI and PD in population-based sample of Czech mothers.

9 ic regressions examined relationship between SUI a PD accounting for range of other risk factors.

10 ltimately predisposing some women to develop SUI.

11 s after birth, 650 mothers (17.6%) developed SUI and 641 (17.3%) displayed signs of PD.

12 95% CI, -0.096 to 0.322), 0.62 and 0.77 for SUI (treatment difference of -0.153; 95% CI, -0.268 to 0

13 t women undergoing an incident procedure for SUI with synthetic mesh in Ontario, Canada, from April 1

14 men who underwent a mesh-based procedure for SUI, the median age was 52 (interquartile range [IQR], 4

15 Yearly volume of mesh-based procedures for SUI performed by the treating surgeons and their surgica

16 use for SUI (POP sling group), and sling for SUI alone (SUI sling group).

17 women who are undergoing primary surgery for SUI.

18 n-ablative transurethral laser treatment for SUI as a minimally invasive and non-implantable procedur

19 n-ablative transurethral laser treatment for SUI without thermal damage to the urethral mucosa.

20 les might serve as a potential treatment for SUI.

21 ir without mesh but concurrent sling use for SUI (POP sling group), and sling for SUI alone (SUI slin

22 ve unmasking of SUI in women who do not have SUI preoperatively (with or without prolapse reduction).

23 ther candidate genes that may be involved in SUI and to study the influence of estrogen and progester

24 ne on ECM proteins thought to be involved in SUI.

25 nificantly larger reduction in GDS scores in SUI compared with LLD-CBT at the end of treatment (EMMD,

26 canine model of stress urinary incontinence (SUI) and no measurable functional agonism at the key sel

27 he treatment of stress urinary incontinence (SUI) and pelvic organ prolapse (POP) have produced highl

28 tors related to stress urinary incontinence (SUI) and postnatal depression (PD) after birth, and (2)

29 eatment of male stress urinary incontinence (SUI) has increased over the last decade.

30 Stress urinary incontinence (SUI) is a common and bothersome condition.

31 Stress urinary incontinence (SUI) is a prominent incontinence caused by increased abd

32 POP) repair and stress urinary incontinence (SUI) to augment and strengthen weakened tissue.

33 Stress urinary incontinence (SUI) with more than 1 symptom or interval treatment; or

34 he incidence of stress urinary incontinence (SUI), a condition that affects 30-60% of the female popu

35 prolapse (POP), stress urinary incontinence (SUI), urge urinary incontinence (UUI), and hernias, are

36 ment for female stress urinary incontinence (SUI).

37 enetic basis of stress urinary incontinence (SUI).

38 ho present with stress urinary incontinence (SUI).

39 Initially, SUI at 6 weeks was slightly, but significantly associate

40 -CBT) or supportive unspecific intervention (SUI).

41 Surgery for POP may unmask occult SUI in many women.

42 ilable is not sufficient for POP with occult SUI, there is some information available to guide clinic

43 as not significantly related to new cases of SUI at 6 months (OR 1.48, 95% CI 0.91-2.39).

44 The intended goal of surgical correction of SUI and POP is durable restoration of normal anatomy and

45 tion of genes involved in the development of SUI could lead to new therapies for the treatment of SUI

46 pport a genetic basis for the development of SUI, but some of the evidence is contradictory.

47 pse may also be linked to the development of SUI.

48 ch are thought to lead to the development of SUI.

49 enetic predisposition for the development of SUI.

50 us studies on the preoperative prediction of SUI following repair of POP have been conducted in an ef

51 to the use of vaginal mesh for treatment of SUI.

52 d lead to new therapies for the treatment of SUI.

53 ir to prevent the postoperative unmasking of SUI in women who do not have SUI preoperatively (with or

54 ents were randomized to LLD-CBT (n = 126) or SUI (n = 125), of whom 229 (mean [SD] age, 70.2 [7.1] ye

55 who underwent transvaginal repair for POP or SUI with mesh between January 1, 2008, and December 31,

56 placement of vaginal mesh for POP repair or SUI surgery.

57 erative identification of occult (and overt) SUI will facilitate the use of an appropriate prophylact

58 ted probabilities of treatment failure (POP, SUI, UI) from parametric survival modeling for the ureth

59 Urethropexy prevented SUI longer than no urethropexy.

60 action and low urethral pressure, resembling SUI.

61 rgery are at risk for developing symptomatic SUI postoperatively.

62 ; 95% CI, 1.76%-2.56%) and the lowest in the SUI sling group (1.16%; 95% CI, 1.03%-1.31%).

63 ; 95% CI, 2.31%-3.21%) and the lowest in the SUI sling group (1.57%; 95% CI, 1.41%-1.74%).

64 Pelvic organ prolapse is closely related to SUI, and the genes thought to be involved in the develop

65 tments are surgical procedures used to treat SUI by inserting an artificial mesh into the urethra.

66 , parity and higher BMI were associated with SUI.

67 ction in GDS scores in LLD-CBT compared with SUI at all time points (week 5: EMMD, -4.08; 95% CI, -6.

68 ll tolerated treatment modality for men with SUI.

69 intervene for POP in women who present with SUI are based on the available literature although conte