ライフサイエンスコーパス: Salmonella

1 balamin contrasts with enteric bacteria like Salmonella.

2 ions for the establishment of infection with Salmonella.

3 (0.4%) had a positive culture for typhoidal Salmonella.

4 many Enterobacteriaceae such as E. coli and Salmonella.

5 ly influence antibody-mediated protection to Salmonella.

6 ts into the evolution of host adaptations in Salmonella AB toxins, their cell and tissue tropisms, an

7 Salmonella adapts to the challenges oxidative stress imp

8 tigens that are genetically conserved across Salmonella and confer broad, non-serotype-specific prote

9 wn to act as a global RNA-binding protein in Salmonella and Escherichia coli, binding to dozens of sm

10 During infection with Salmonella and many other pathogens, flagellin is a majo

11 Nontyphoidal Salmonella and Salmonella Paratyphi are responsible for

12 easing antibacterial resistance in typhoidal Salmonella and the dearth of novel antimicrobials on the

13 from three different bacteria (Haemophilus, Salmonella, and Mycobacterium) as our model systems and

14 rmined that seasonal peaks of Campylobacter, Salmonella, and Shiga toxin-producing Escherichia Coli (

15 We previously demonstrated that the Salmonella anti-inflammatory response activator SarA (St

16 loodstream infections caused by nontyphoidal Salmonella are a major public health concern in Africa,

17 idates against S. Paratyphi and nontyphoidal Salmonella are presented, with a focus on the research f

18 E. coli and Salmonella are two of the most common bacterial pathogen

19 n, mannan, and LPS from Escherichia coli and Salmonella as well as to the monosaccharides l-fucose, d

20 mbined MIL-RPA approach enabled detection of Salmonella at levels as low as 10(3) CFU mL(-1).

21 anthrax letter attacks, and 1984 Rajneeshee Salmonella attacks, the threat of future epidemics/pande

22 S. aureus, Klebsiella spp. and non-typhoidal Salmonella bacteria.

23 r appeared to be the only site that harbored Salmonella bacteria.

24 sensor for online and sensitive detection of Salmonella based on immunomagnetic separation, fluoresce

25 immunized mice but did not bind live intact Salmonella because of surface inaccessibility of this de

26 Here we show that EPS of Salmonella biofilms is a cooperative trait whose benefit

27 We sought to describe Salmonella bloodstream infections, antimicrobial resista

28 ake SPI-1 an attractive target in preventing Salmonella carriage and colonization in chickens to redu

29 nt in understanding the effect of intestinal Salmonella carriage on the microbiome and metabolome of

30 Salmonella causes grave systemic infections in humans an

31 the proposed biosensor to detect selectively Salmonella cells with a low limit of detection of 1.5 *

32 ithin the intestinal environment dictated by Salmonella-commensal interaction.

33 inked with the probe during the formation of Salmonella-containing vacuole (SCV).

34 hoid toxin is secreted into the lumen of the Salmonella-containing vacuole by a secretion mechanism s

35 la virulence gene expression, rupture of the Salmonella-containing vacuole, and host cell death.

36 facilitates the delivery of itaconate to the Salmonella-containing vacuole.

37 poptosis, or necroptosis had minor impact on Salmonella control.

38 ics of antimicrobial resistance in typhoidal Salmonella, covering data of at least 24 different count

39 and environmental exposures to nontyphoidal Salmonella disease in East Africa.

40 c infection typhoid to invasive nontyphoidal Salmonella disease in humans.

41 ariant associated with invasive nontyphoidal Salmonella disease is Salmonella Typhimurium sequence ty

42 A single copy of Salmonella DNA was quantified in just 10 min and the per

43 ldwide develop foodborne illnesses caused by Salmonella enterica (S. enterica) every year.

44 t Listeria innocua, Pseudomonas fluorescens, Salmonella enterica and Bacillus cereus has not been pre

45 d the PT reaction using purified DndCDE from Salmonella enterica and IscS from Escherichia coli.

46 st Escherichia coli, Listeria monocytogenes, Salmonella enterica and Staphylococcus aureus.

47 virulence of the important enteric pathogens Salmonella enterica and Vibrio cholerae by repressing Ar

48 Salmonella enterica bloodstream infections are an import

49 ed vitamin B-12, [13C]-cyanocobalamin, using Salmonella enterica by providing [13C2]-ethanolamine as

50 ericia coli, phosphorothioate epigenetics in Salmonella enterica Cerro 87, and oxidation-induced abas

51 The Salmonella enterica effector SteD depletes mature MHC cl

52 Neisseria meningitidis, Vibrio cholerae, and Salmonella enterica harbor these prophages.

53 Multidrug-resistant (MDR) Salmonella enterica has been deemed a high-priority path

54 mance of PRAP by analyzing the genomes of 26 Salmonella enterica isolates from Shanghai, China.

55 Two hundred and sixty-four Salmonella enterica isolates recovered over a 16-year pe

56 The most common Salmonella enterica pathovariant associated with invasiv

57 man bacterial pathogens Escherichia coli and Salmonella enterica produce a biofilm matrix composed pr

58 s)RidA-2 complemented the growth defect of a Salmonella enterica ridA mutant, an in vivo model of 2AA

59 We reviewed Salmonella enterica serotype Typhi infections reported t

60 Salmonella enterica serotype Typhimurium (S. Typhimurium

61 teric fever, a bacterial infection caused by Salmonella enterica serotypes Typhi and Paratyphi A, fre

62 Analysis of the distribution of Salmonella enterica serovar Derby (S.

63 Salmonella enterica serovar Enteritidis is a common caus

64 Salmonella enterica serovar Enteritidis is a major cause

65 Here, we present a focused minireview on Salmonella enterica serovar Panama, a serovar responsibl

66 Salmonella enterica serovar Typhi (S. Typhi) causes subs

67 man populations, with the causative pathogen Salmonella enterica serovar Typhi implicated in many out

68 Salmonella enterica serovar Typhi isolates from the 2 ho

69 n, whereas the secretion of Typhoid toxin in Salmonella enterica serovar Typhi relies on a muramidase

70 Typhoid toxin is a virulence factor of Salmonella enterica serovar Typhi, the causative agent o

71 Salmonella enterica serovar Typhimurium (S Typhimurium)

72 The human pathogen Salmonella enterica serovar Typhimurium (S Typhimurium)

73 the ancestral regulatory system PhoP/PhoQ of Salmonella enterica serovar Typhimurium (S. Typhimurium)

74 Infection of human macrophages with Salmonella enterica serovar Typhimurium (S. Typhimurium)

75 udy, we used a mouse model of infection with Salmonella enterica serovar Typhimurium (STM) to identif

76 host defense against the bacterial pathogen Salmonella enterica serovar Typhimurium (STm).

77 uick and accurate detection of low levels of Salmonella enterica serovar typhimurium and enteritidis

78 rolling infection by two bacterial pathogens-Salmonella enterica serovar Typhimurium and Shigella fle

79 Using two species, Escherichia coli and Salmonella enterica serovar Typhimurium as model microbe

80 that the facultative intracellular pathogen Salmonella enterica serovar Typhimurium decreases H-NS a

81 g different susceptibilities to infection by Salmonella enterica serovar Typhimurium has just been pu

82 Slc11a1 is proposed to restrict growth of Salmonella enterica serovar Typhimurium in host tissues

83 ity (mAb 3H3) can disrupt biofilms formed by Salmonella enterica serovar Typhimurium in vitro and in

84 In addition, the intracellular pathogen Salmonella enterica serovar Typhimurium initiates an ant

85 utilization bacterial microcompartment from Salmonella enterica serovar Typhimurium LT2, one of the

86 C3, D1, E1, G, I, K, N, O, and Q); however, Salmonella enterica serovar Typhimurium was the most pre

87 In Salmonella enterica serovar Typhimurium, DSFs repress th

88 We show that both flagellins of Salmonella enterica serovar Typhimurium, FliC and FljB,

89 In Salmonella enterica serovar Typhimurium, siroheme is pro

90 d a striking defect in their ability to kill Salmonella enterica serovar Typhimurium, which was rescu

91 ) responses to bacterial infections, such as Salmonella enterica serovar Typhimurium.

92 ls and cell lines, typically challenged with Salmonella enterica serovar Typhimurium.

93 Exposure to the dominant NTS serovars, Salmonella enterica serovars Typhimurium and Enteritidis

94 sess the ability of PCR for the detection of Salmonella enterica shedding and to compare that ability

95 Studies in Escherichia coli and Salmonella enterica showed that such sRNAs are natural p

96 ter rodentium NleB effectors, as well as the Salmonella enterica SseK effectors are glycosyltransfera

97 We report that typhoidal and nontyphoidal Salmonella enterica strains activate MAIT cells.

98 Here, we recovered eight Salmonella enterica subsp. enterica genomes from human s

99 equence the entire chromosome and plasmid of Salmonella enterica subsp. enterica serovar Bareilly and

100 nosus R0011 secretome (LrS) on TNF-alpha and Salmonella enterica subsp. enterica serovar Typhimurium

101 system may contribute to the persistence of Salmonella enterica subsp. enterica serovar Typhimurium

102 Salmonella enterica subspecies enterica serovar Typhi (S

103 Typhoid fever is caused by Salmonella enterica subspecies enterica serovar Typhi (S

104 We present the genome sequences of Salmonella enterica tailed phages Sasha, Sergei, and Sol

105 on with the intracellular bacterial pathogen Salmonella enterica Typhimurium.

106 Unlike ScThi5, LpThi5 functioned in vivo in Salmonella enterica under multiple growth conditions.

107 Salmonella enterica variants exhibit diverse host adapta

108 ella pneumoniae, Mycobacterium tuberculosis, Salmonella enterica, and Staphylococcus aureus, we repor

109 luding the causative agent of salmonellosis, Salmonella enterica, can occur as a result of eco-evolut

110 DNA MTases, like those from Vibrio cholerae, Salmonella enterica, Clostridioides difficile, or Strept

111 without POTRA domains from Escherichia coli, Salmonella enterica, Haemophilus ducreyi and Neisseria g

112 the total allelic diversity (panallelome) of Salmonella enterica, Mycobacterium tuberculosis, Pseudom

113 Enterococcus spp., Escherichia coli, Salmonella enterica, Staphylococcus aureus and Streptoco

114 Here, we report that, in Salmonella enterica, the sirtuin deacylase CobB long iso

115 a on two key organisms, Escherichia coli and Salmonella enterica, we show that copper resistance requ

116 ISPR based redox conduit in both E. coli and Salmonella enterica.

117 natural and challenged C. jejuni and natural Salmonella enterica.

118 Intestinal carriage of Salmonella Enteritidis (SE) in the chicken host serves a

119 eously exposed to an inoculum of inactivated Salmonella Enteritidis and a chronic heat stress (CHS).

120 Salmonella Enteritidis and Salmonella Typhimurium are ma

121 SPS phage can both prevent the spread of AMR Salmonella Enteritidis infection in chickens and shift t

122 l and 82 (47.7%) from blood, 53 (30.8%) were Salmonella Enteritidis ST11 and 62 (36.0%) Salmonella Ty

123 source of human infection by some clades of Salmonella Enteritidis ST11 in East Africa, but not of h

124 We identified cgMLST clusters within Salmonella Enteritidis ST11, Salmonella Heidelberg ST15,

125 against challenge with nontyphoidal serovar Salmonella Enteritidis than with another nontyphoidal se

126 No interference/cross-reactivity from Salmonella enteritidis, Enterobacter agglomerans, Pseudo

127 te cyclases from the Gram-negative bacterium Salmonella Enteritidis, identifying AdrA as the most pot

128 Escherichia coli, Salmonella enteritidis, Listeria innocua, Pseudomonas ae

129 to inject effector proteins that facilitate Salmonella entry, establishment of an intracellular nich

130 CD4 T cell depletion in mice where virulent Salmonella establish chronic infection, similar to chron

131 ns included astrovirus, norovirus, Shigella, Salmonella, ETEC, sapovirus, and typical EPEC.

132 4 in memory immunity, primary challenge with Salmonella expressing flagellin modified to largely evad

133 When such mutations were introduced into Salmonella flagellin, motility was abolished.

134 ost metabolites, resident gut microbiota and Salmonella following inoculation of SE in two-week-old l

135 hes shed light on the catalytic mechanism of Salmonella FraB and of phosphosugar deglycases in genera

136 s declared the Danish broiler industry to be Salmonella free.

137 ture for sampling and detection of typhoidal Salmonella from environmental samples including drinking

138 Some enteric bacteria including Salmonella have evolved the propanediol-utilising microc

139 clusters within Salmonella Enteritidis ST11, Salmonella Heidelberg ST15, Salmonella Typhimurium ST 19

140 plication of intravacuolar pathogens such as Salmonella Here, we show that this mechanism requires ac

141 Our previous work in Escherichia coli and Salmonella identified a mechanism of translational repre

142 berg ST15, Salmonella Typhimurium ST 19, and Salmonella II 42:r:- ST1208 that included both human and

143 r and memory CD4(+) T-cell responses against Salmonella in mice.

144 , the developed biosensor was used to detect Salmonella in milk.

145 chastic dynamic modelling of transmission of Salmonella in parent flocks and combined that with the r

146 nces in the molecular detection of typhoidal Salmonella in the environment, and outline challenges an

147 cantly repressed invasion gene expression by Salmonella in the murine colitis model, indicating that

148 taomicron genes that were upregulated during Salmonella-induced gut inflammation and were predicted t

149 ch is associated with increased mortality in Salmonella-infected humans, was exacerbated by CD4 deple

150 Salmonella-infected, CD4-depleted 129X1/SvJ mice remaine

151 We find that Rab32 interacts with IRG1 on Salmonella infection and facilitates the delivery of ita

152 Salmonella infection can cause gastroenteritis in health

153 Here we use a model of auxotrophic Salmonella infection in germ-free mice to show that live

154 t flocks and eggs at the hatchery in case of Salmonella infection in parent flocks in the Danish poul

155 tion, the role of cell death during systemic Salmonella infection remained elusive.

156 This work describes a mouse model of Salmonella infection that recapitulates several aspects

157 showed that this miRNA specifically inhibits Salmonella infection via modulation of endolysosomal tra

158 yroptosis, necroptosis, and apoptosis during Salmonella infection, we infected mice and macrophages d

159 le for cell death associated caspases during Salmonella infection.

160 es M cells and SFB levels to protect against Salmonella infection.

161 hepatobiliary system as the site of chronic Salmonella infection.

162 4 T cells, which are crucial for immunity to Salmonella infection.

163 inflammation, anemia, and susceptibility to Salmonella infection.

164 d Th17 responses in ex-germ-free mice during Salmonella infection.

165 te host defense in homeostasis and following Salmonella infection.

166 isease tolerance pathways in endotoxemia and Salmonella infection.

167 ntestinal microbial community in response to Salmonella infection.

168 Of 52 821geocodable Salmonella infections (>96%), 48 111 (91.1%) were domest

169 re essential for resistance against systemic Salmonella infections and can express the highest protec

170 g human-relevant differences in nontyphoidal Salmonella infections, whereas differentiated human THP-

171 had significantly higher inflammation after Salmonella infections.

172 hogenesis in the intestinal epithelium where Salmonella initiates infection, indicating that IFN-I si

173 control strategies for invasive nontyphoidal Salmonella (iNTS) disease, which is increasingly becomin

174 Salmonella is a major causative agent of foodborne illne

175 The intracellular bacterial pathogen Salmonella is able to evade the immune system and persis

176 ampled from two comprehensive collections of Salmonella isolates from African patients with bloodstre

177 rototypical intracellular bacterial pathogen Salmonella led us to discover that type I IFN (IFN-I) re

178 certain key metabolites beyond the realm of Salmonella life.

179 Thus, CRTAM enhances susceptibility to Salmonella, likely by promoting the inflammatory respons

180 Depolarization in respiring Salmonella mediates intense bactericidal activity of rea

181 thought all PA emulsions evalauted inhibited Salmonella, morphological changes to this antimicrobial

182 Here, we present structures of the Salmonella MS-ring, revealing a high level of variation

183 Overall, Salmonella Newport was the most (p < 0.05) susceptible s

184 Although live auxotrophic Salmonella no longer causes inflammation, its mucosal vi

185 Nontyphoidal Salmonella (NTS) are among the most common etiological a

186 e more susceptible to recurrent nontyphoidal Salmonella (NTS) bacteremia.

187 r was 22%, which was mainly due to non-Typhi Salmonella (NTS) diagnoses being misclassified as malari

188 way isolates were compared with nontyphoidal Salmonella (NTS) isolated from persons with bloodstream

189 Nontyphoidal Salmonella (NTS) organisms are a major cause of gastroen

190 Nontyphoidal Salmonella (NTS) was identified in 671 enrolled inpatien

191 re shedding light on how humoral immunity to Salmonella operates.

192 nto how we can detect signatures of invasive Salmonella organisms interacting with the host during in

193 transmission routes for Salmonella Typhi and Salmonella Paratyphi A.

194 Nontyphoidal Salmonella and Salmonella Paratyphi are responsible for significant mor

195 caused by the pathogens Salmonella Typhi and Salmonella Paratyphi.

196 host to understand the temporal dynamics of Salmonella pathogenesis and to identify its lifestyle fo

197 Moreover, IFN-I signaling promoted in vivo Salmonella pathogenesis in the intestinal epithelium whe

198 ces were associated with lower expression of Salmonella pathogenicity island (SPI) 1 genes in S.

199 lD, that encodes the master-regulator of the Salmonella Pathogenicity Island 1 (SPI-1), was present i

200 at H-NS is both a repressor and activator of Salmonella Pathogenicity Island 1 gene expression, and b

201 Regulatory cross-talk between two major Salmonella pathogenicity islands, SPI-1 and SPI-2, was r

202 However, the host cues that Salmonella perceives to undergo this switch remain uncle

203 This finding was in contrast to short-tailed Salmonella podoviruses, illustrating that tailed phages

204 ponin resulted in >6 log CFU/ml reduction in Salmonella population.

205 However, there is continuous Salmonella pressure from the environment, and a number o

206 s also hampered colonization of the pathogen Salmonella, prolonging host survival.

207 ing let-7i-3p miRNA as a strong inhibitor of Salmonella replication and performing in-depth analysis

208 Transcriptomic and proteomic analysis in Salmonella revealed regulatory crosstalk and hierarchica

209 get genes in pathogenic Escherichia coli and Salmonella revealed using chromatin immunoprecipitation

210 Three articles reveal how Salmonella rewires macrophage polarization (Panagi et al

211 Cryptosporidium and Shigella, Listeria, and Salmonella (rho = 0.51, 0.51, 0.46; p < 0.04).

212 The Salmonella samples were spiked with Salmonella type B, i

213 estically acquired salmonellosis and leading Salmonella serotypes are poorly understood.

214 ) and Quillaja Saponin was evaluated against Salmonella serotypes Newport, Oranienburg and Typhimuriu

215 antimicrobial varied substantially among the Salmonella serotypes tested.

216 Additionally, Salmonella serotypes Typhimurium and Newport also formed

217 sequently provide protection against another Salmonella serovar is determined by the accessibility of

218 ability of protective Abs elicited with one Salmonella serovar to engage with and consequently provi

219 Pathogenic Salmonella serovars produce clinical manifestations rang

220 antibody could efficiently concentrate both Salmonella serovars with a capturing efficiency >95%.

221 ilage pathogens, such as E. coli, S. aureus, Salmonella sp., Listeria sp., yeast and moulds, making i

222 cile (55.0%), Campylobacter species (20.9%), Salmonella species (12.4%), and Shigella/EIEC species (1

223 Nontyphoidal Salmonella species are globally disseminated pathogens a

224 demonstrated a broad host range covering all Salmonella species with one reporter detecting 99.3% of

225 lass II tetramers to interrogate endogenous, Salmonella-specific CD4(+) helper T cells, we show that

226 Analysis of Salmonella-spiked blood samples with the SP-PCR resulted

227 The counts of cecal Salmonella spp. increased in the CTL21 group compared to

228 Peptides identified for two or fewer Salmonella strains were evaluated as potential serovar m

229 inst PhIP, MelQ, and MelQx in TA98 and TA100 Salmonella strains, and this activity was not affected b

230 exposure to pathogenic and non - pathogenic Salmonella strains, highlighted the possibility of the p

231 Salmonella switches to a flagellin-low phenotype as infe

232 ison with the homologous interactions in the Salmonella T3SS sorting platform revealed clear differen

233 h in turn enhance expression of flagellin by Salmonella thereby amplifying its ability to elicit cell

234 n down-regulation of flagellin expression by Salmonella These findings reveal a previously unrecogniz

235 ole for type 1 IFN signaling in switching of Salmonella to a flagellin-low phenotype.

236 st serves as a reservoir for transmission of Salmonella to humans through the consumption of poultry

237 and enhances flagella-dependent adhesion of Salmonella to phosphatidylcholine vesicles and epithelia

238 The Salmonella samples were spiked with Salmonella type B, introduced into the biosensor via the

239 these, 304 (5.3%) were culture positive for Salmonella Typhi (249 [81.9%]) or Paratyphi A (55 [18.1%

240 Salmonella Typhi activates the host DNA damage response

241 Of these, 7,591 (87%) were Salmonella Typhi and 1114 (13%) were S. Paratyphi.

242 94% (2093/2230) of isolates were Salmonella Typhi and 6% (137/2230) were S. Paratyphi.

243 agriculture are key transmission routes for Salmonella Typhi and Salmonella Paratyphi A.

244 s an enteric disease caused by the pathogens Salmonella Typhi and Salmonella Paratyphi.

245 Salmonella Typhi contributed most to the enteric fever h

246 Salmonella Typhi is a major cause of fever in children i

247 Typhoid fever caused by Salmonella Typhi is a major public health concern in low

248 Two MDR Salmonella Typhi isolates from India were found by whole

249 s and increasing antimicrobial resistance in Salmonella Typhi that have served to increase interest i

250 enterica subspecies enterica serovar Typhi (Salmonella Typhi) is the cause of typhoid fever and a hu

251 s tissue polymerase chain reaction [PCR] for Salmonella Typhi).

252 ization of mice with live typhoidal serovar, Salmonella Typhi, generates cross-reactive immune respon

253 virulence factor for the bacterial pathogen Salmonella Typhi, which causes typhoid fever in humans.

254 ties were also promising, especially against Salmonella typhimurium (MBC = 0.44 mg/mL).

255 cells, rendered mice susceptible to invasive Salmonella typhimurium (S.t.) infection.

256 race the emergence and evolutionary paths-of Salmonella Typhimurium (STM) from nine years of Australi

257 on and long-term pathogen persistence during Salmonella Typhimurium (STm) infection.

258 arance of intracellular infections caused by Salmonella Typhimurium (STm) requires IFN-gamma and the

259 munization with the trimeric porin OmpD from Salmonella Typhimurium (STmOmpD) protects against infect

260 ing with the treatment of mice infected with Salmonella typhimurium and affording preliminary promisi

261 Salmonella Typhimurium and its monophasic variant S.

262 ass II effector TMD-chaperone complexes from Salmonella Typhimurium and Pseudomonas aeruginosa, respe

263 Salmonella Enteritidis and Salmonella Typhimurium are major causes of bloodstream i

264 ntal work with Serratia marcescens in water, Salmonella Typhimurium ATCC 14028 was inoculated in wate

265 Salmonella Typhimurium can invade and survive within mac

266 Salmonella Typhimurium diarrhea serves as a paradigm, an

267 rential fluorescence induction to screen the Salmonella Typhimurium genome for loci that respond, at

268 ibe how aspartate/malate can trigger initial Salmonella Typhimurium gut-lumen colonization in mice, p

269 Salmonella Typhimurium is metabolically adaptable and ca

270 sex toward a systemic immune stimulation by Salmonella typhimurium lipopolysaccharide (LPS).

271 A Salmonella Typhimurium mutant deficient in flagellin met

272 he pathogen killing was evaluated by plating Salmonella typhimurium on agar plates and showed that th

273 n of Lacc1 (Lacc1(Deltamye)) were given oral Salmonella Typhimurium or dextran sodium sulfate.

274 ative pathogens such as Shigella flexneri or Salmonella Typhimurium remains incompletely understood [

275 invasive nontyphoidal Salmonella disease is Salmonella Typhimurium sequence type (ST)313.

276 nteritidis ST11, Salmonella Heidelberg ST15, Salmonella Typhimurium ST 19, and Salmonella II 42:r:- S

277 itidis ST11 in East Africa, but not of human Salmonella Typhimurium ST313 infection.

278 Salmonella Typhimurium ST313 was isolated exclusively fr

279 e Salmonella Enteritidis ST11 and 62 (36.0%) Salmonella Typhimurium ST313.

280 Specifically, we screen Salmonella typhimurium strains expressing and delivering

281 ylation of flagellin facilitates adhesion of Salmonella Typhimurium to hydrophobic host cell surfaces

282 erococcus faecalis, Pseudomonas fluorescens, Salmonella typhimurium, Staphylococcus aureus); and fung

283 inactivation of a pathogenic species such as Salmonella typhimurium, using a luminometer assay.

284 contain the Na(+)/H(+) antiporter NhaA from Salmonella Typhimurium.

285 idis than with another nontyphoidal serovar, Salmonella Typhimurium.

286 NOX2 collapses the DeltapH of intracellular Salmonella Typhimurium.

287 Salmonella typhoid toxin contributes to typhoid disease

288 The depolarization experienced by Salmonella undergoing oxidative stress impairs folding o

289 In addition, IgM-mediated Salmonella uptake was decreased, and MIICs were less clu

290 As a paradigmatic example, Salmonella uses two type-3 secretion systems to inject e

291 this need, a method was developed to detect Salmonella using luciferase reporter bacteriophages.

292 through August 2017 in Tanzania and isolated Salmonella using standard methods.

293 ty at these locations included expression of Salmonella virulence factors, genes involved in pertussi

294 n was associated with elevated intracellular Salmonella virulence gene expression, rupture of the Sal

295 d in 11 outpatients (0.07%), while typhoidal Salmonella was found in 49 outpatients (0.3%).

296 italized children in Bamako, while typhoidal Salmonella was uncommon.

297 played by lysine methylation of flagellin in Salmonella, which requires the methylase FliB.

298 competition synergistically protect against Salmonella wild-type infection.

299 Klebsiella, Enterobacter, Vibrio, Shigella, Salmonella, Yersinia, Mycobacterium and Bacillus-yet are

300 cteriophages that infect Escherichia coli or Salmonella, yet, less is known about the packaging motor