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1 ouse (Peromyscus maniculatus), infected with Sin Nombre virus.
2 other HPS-associated hantaviruses, including Sin Nombre virus.
3 ntaviruses, including Andes virus (ANDV) and Sin Nombre virus, cause a distinct zoonotic disease, han
4 cessfully passaged a mouse-adapted strain of Sin Nombre virus from deer mice (Peromyscus maniculatus)
5 ombinant PCMV was constructed that contained Sin Nombre virus glycoprotein G1 (SNV-G1) fused in frame
6 d to clarify the involvement of Cdc42 in the Sin Nombre virus internalization and the signaling pathw
7 of virus assembly was recently described for Sin Nombre virus, it is likely that all of the new Ameri
8                 In contrast, the assembly of Sin Nombre virus N protein-HTNV vRNA complexes was inhib
9 d RNA binding by using bacterially expressed Sin Nombre virus N protein.
10                 The K(d) values for HTNV and Sin Nombre virus N proteins were nearly identical for th
11 served nucleotides of the panhandle, whereas Sin Nombre virus N requires the first 23 nucleotides for
12 ection with pathogenic hantaviruses, such as Sin Nombre virus or Andes virus (ANDV).
13 he Andes, Puumala, Prospect Hill, Seoul, and Sin Nombre viruses recognize their individual homologous
14 ibition of host autophagy machinery inhibits Sin Nombre virus replication in cells, suggesting that a
15 e deer mouse with its homologous hantavirus, Sin Nombre virus, results in low levels of immune gene e
16 rdiopulmonary syndrome (HCPS), we quantified Sin Nombre virus S segment viral RNA in plasma samples f
17                  New York 1 virus (NY-1) and Sin Nombre virus (SN) are associated with hantavirus pul
18 f the core domains of NP (NPcore) encoded by Sin Nombre virus (SNV) and Andes virus (ANDV), which are
19                                              Sin Nombre virus (SNV) and Hantaan virus (HTN) infect en
20 used to investigate vertical transmission of Sin Nombre virus (SNV) and SNV-specific antibody.
21                       Andes virus (ANDV) and Sin Nombre virus (SNV) are rodent-borne hantaviruses tha
22                       Andes virus (ANDV) and Sin Nombre virus (SNV) are the etiologic agents of sever
23 sid (N) proteins of hantaviruses such as the Sin Nombre virus (SNV) bind to membranes and viral RNAs,
24                                              Sin Nombre virus (SNV) causes the zoonotic disease hanta
25 a zoonotic disease caused by transmission of Sin Nombre virus (SNV) from chronically infected deer mi
26 , a laboratory animal model for the study of Sin Nombre virus (SNV) infection or associated disease h
27 olved in developing capillary leakage during Sin Nombre virus (SNV) infection.
28 ll isolated from an individual with previous Sin Nombre virus (SNV) infection.
29                                              Sin Nombre virus (SNV) is a highly pathogenic New World
30                                              Sin Nombre virus (SNV) is a rodent-borne hantavirus that
31                                          The Sin Nombre virus (SNV) is the etiologic agent of hantavi
32                                              Sin Nombre virus (SNV) is the primary cause of hantaviru
33                                              Sin Nombre virus (SNV) is thought to establish a persist
34 e sites solubilized in envelope membranes of Sin Nombre virus (SNV) particles.
35 ing of inactivated and fluorescently labeled Sin Nombre virus (SNV) to the integrin PSI domain stimul
36                                              Sin Nombre virus (SNV), a member of the Hantavirus genus
37 n with HPS-associated hantaviruses, NY-1 and Sin Nombre virus (SNV), is inhibited by antibodies to be
38 s maniculatus) are the natural reservoirs of Sin Nombre virus (SNV), the etiologic agent of most HCPS
39 e we show that rhesus macaques infected with Sin Nombre virus (SNV), the primary etiological agent of
40 igate early events in the innate response to Sin Nombre virus (SNV), the principal etiologic agent of
41 cases of HCPS in North America are caused by Sin Nombre virus (SNV), which is carried asymptomaticall
42 ar responses in both Andes virus (ANDV)- and Sin Nombre virus (SNV)-infected A549 and Huh7-TLR3 cells
43 antavirus cardiopulmonary syndrome caused by Sin Nombre virus (SNV).
44  of Peromyscus maniculatus, the reservoir of Sin Nombre virus (SNV).
45 a rare but acute fulminant disease caused by Sin Nombre virus (SNV).
46 er mice are the principal reservoir hosts of Sin Nombre virus, the etiologic agent of most hantavirus
47 eer mice can be experimentally infected with Sin Nombre virus, which now allows provocative examinati