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1 pression, cognitive disorders, arthritis and Sjogren's syndrome).
2 (diabetes, thyroiditis); and rheumatologic (Sjogren's syndrome).
3 e diseases (lupus, rheumatoid arthritis, and Sjogren's syndrome).
4 ab in patients with rheumatoid arthritis and Sjogren's syndrome.
5 desiccating stress (DS) model that resembles Sjogren's syndrome.
6 ant CD4(+) infiltration in the LG, mimicking Sjogren's syndrome.
7 iduals with systemic lupus erythematosus and Sjogren's Syndrome.
8 ormal T-cell development and activation, and Sjogren's Syndrome.
9 antibodies in the serum resemble autoimmune Sjogren's syndrome.
10 epletion in systemic lupus erythematosus and Sjogren's syndrome.
11 and aberrant histopathology associated with Sjogren's syndrome.
12 ent from inflammation in the pathogenesis of Sjogren's syndrome.
13 chizophrenia greatly reduce risk for SLE and Sjogren's syndrome.
14 mandibular gland (SMG), a critical target of Sjogren's syndrome.
15 no such studies published or in progress for Sjogren's syndrome.
16 immunologic and nonimmunologic mechanisms in Sjogren's syndrome.
17 ry process in salivary glands that resembles Sjogren's syndrome.
18 systemic sclerosis, rheumatoid arthritis, or Sjogren's syndrome.
19 esembles the disease manifestations of human Sjogren's syndrome.
20 tients with systemic lupus erythematosus and Sjogren's syndrome.
21 imilar to that found in humans with lupus or Sjogren's syndrome.
22 en's syndrome, develop an illness similar to Sjogren's syndrome.
23 function that is highly reminiscent of human Sjogren's syndrome.
24 mation and are models for the human disorder Sjogren's syndrome.
25 rimal gland damage in these murine models of Sjogren's syndrome.
26 om infection or autoimmune disorders such as Sjogren's syndrome.
27 lands, similar to that in the human disorder Sjogren's syndrome.
28 e antibodies was less in patients with RA or Sjogren's syndrome.
29 ematosus and 125 white patients with primary Sjogren's syndrome.
30 hocytes in the initiation and propagation of Sjogren's syndrome.
31 ation and are a model for the human disorder Sjogren's syndrome.
32 d neck cancer or autoimmune diseases such as Sjogren's syndrome.
33 a reactive infiltrate, often associated with Sjogren's syndrome.
34 n spontaneously develops that is a model for Sjogren's syndrome.
35 oimmune response such as that encountered in Sjogren's syndrome.
36 nction of lacrimal glands in mouse models of Sjogren's syndrome.
37 inflammation, and serve as a model for human Sjogren's syndrome.
38 ding insulin-dependent diabetes mellitus and Sjogren's syndrome.
39 hoid infiltrate that occurs in patients with Sjogren's syndrome.
40 or autoimmune insulin-dependent diabetes and Sjogren's syndrome.
41 nection with the autoimmune exocrinopathy of Sjogren's syndrome.
42 provide insight into the immunopathology of Sjogren's syndrome.
43 not satisfy the criteria for a diagnosis of Sjogren's syndrome.
44 milar to those seen in the pseudolymphoma of Sjogren's syndrome.
45 The other was from a patient with secondary Sjogren's syndrome.
46 europathy and the neuropathy associated with Sjogren's syndrome.
47 ell neoplasms that are often associated with Sjogren's syndrome.
48 /lpr mice are a model for the human disorder Sjogren's syndrome.
49 esions and are a model for the human disease Sjogren's syndrome.
50 and IgG-positive cells, resemble lesions of Sjogren's syndrome.
51 ip biopsy findings that met the criteria for Sjogren's syndrome.
52 ce of complete heart block in a patient with Sjogren's syndrome.
53 upus erythematosus, rheumatoid arthritis and Sjogren's syndrome.
54 BCCAO may be present in patients with Sjogren's syndrome.
55 acute visual loss is found in a patient with Sjogren's syndrome.
56 he systemic symptoms associated with primary Sjogren's syndrome.
57 associated with the autoimmune exocrinopathy Sjogren's syndrome.
58 60 in systemic lupus erythematosus (SLE) and Sjogren's syndrome.
59 targeted in systemic lupus erythematosus and Sjogren's syndrome.
60 nd diseases such as head and neck cancer and Sjogren's syndrome.
61 s, polymyositis/dermatomyositis, and primary Sjogren's syndrome.
62 tients with systemic lupus erythematosus and Sjogren's syndrome.
63 linked to development of type 1 diabetes and Sjogren's syndrome.
64 ses such as systemic lupus erythematosus and Sjogren's syndrome.
65 ce of an infectious environmental trigger in Sjogren's syndrome.
66 lved in both innate and adaptive immunity in Sjogren's syndrome.
67 esults of a large-scale association study of Sjogren's syndrome.
68 in turn may predispose to the development of Sjogren's syndrome.
69 y for systemic lupus erythematosus (SLE) and Sjogren's syndrome.
70 cent confidence interval, 0.59 to 1.73]; and Sjogren's syndrome, 1.42 [95 percent confidence interval
72 ported most frequently by PBC cases included Sjogren's syndrome (17.4%) and Raynaud's syndrome (12.5%
73 ot the clinical profile; 7 had lupus, 14 had Sjogren's syndrome, 7 had both, and 23 were asymptomatic
75 athology and aberrant function mimic that of Sjogren's syndrome, a human autoimmune disease character
77 eases systemic lupus erythematosus (SLE) and Sjogren's syndrome affect nine times more women than men
78 sclerosis affects 49,000 adults, and primary Sjogren's syndrome affects from 0.4 million to 3.1 milli
80 sk for SLE and 16-fold variation in risk for Sjogren's syndrome among individuals with common C4 geno
81 ntal factors that control the development of Sjogren's syndrome, an autoimmune disease mainly involvi
84 nt discoveries from studies in patients with Sjogren's syndrome and animal models suggest a complex i
87 en deficiency may promote the progression of Sjogren's syndrome and its associated lacrimal gland inf
88 eles in men, women's greater risk of SLE and Sjogren's syndrome and men's greater vulnerability to sc
90 e hormone, may be useful in the treatment of Sjogren's syndrome and other human autoimmune diseases.
94 d is an autoantigen present in patients with Sjogren's syndrome and systemic lupus erythematosus.
95 f the statistical model appear to operate in Sjogren's syndrome and systemic sclerosis whereas only t
96 oral dryness is experienced by sufferers of Sjogren's syndrome and those treated with irradiation fo
97 risk for SLE, 31-fold variation in risk for Sjogren's syndrome, and 1.7-fold variation in schizophre
98 e diseases, including lupus, scleroderma and Sjogren's syndrome, and had a prominent transcriptomic o
99 develop autoantibodies, sialadenitis, as in Sjogren's syndrome, and immune complex-mediated nephriti
100 caused by medications, chronic diseases like Sjogren's syndrome, and medical treatments, such as radi
103 mechanistic insight into the development of Sjogren's Syndrome, and suggest the potential of NF-kapp
104 c inflammatory demyelinating polyneuropathy, Sjogren's syndrome, and systemic lupus erythematosus tha
105 Currently, no effective treatments exist for Sjogren's syndrome, and there is a limited understanding
106 athomechanisms and involvement of viruses in Sjogren's syndrome, and to highlight the areas for futur
107 cell arteritis, ankylosing spondylitis, and Sjogren's syndrome, and to provide an overall estimate o
109 the lacrimal gland, and the presence of anti-Sjogren's syndrome antigen A and anti-Sjogren's syndrome
111 the human Ago2 minimal RISC system to purify Sjogren's syndrome antigen B (SSB)/autoantigen La as an
113 f anti-Sjogren's syndrome antigen A and anti-Sjogren's syndrome antigen B antibodies in the serum res
116 tients with systemic lupus erythematosus and Sjogren's syndrome are influenced by the HLA class II ge
117 Autoimmune myositis, vasculitis and primary Sjogren's syndrome are rare diseases in which these ther
118 tricted autoantigens (e.g., M3R) targeted in Sjogren's syndrome are specifically cleaved by granzyme
119 eratitis or KCS, a condition that is seen in Sjogren's syndrome, are dependent on NF-kappaB, but are
120 ics of systemic lupus erythematosus (SLE) or Sjogren's syndrome as the era of genome-wide association
121 ate of saliva and tear flow in patients with Sjogren's syndrome, as well as improving subjective symp
122 ong similarity to the 52-kD Ro/SSA lupus and Sjogren's syndrome auto-antigen and the RET finger prote
124 age and the generation of novel fragments of Sjogren's syndrome autoantigens in this form of apoptosi
126 seen in the salivary glands of patients with Sjogren's syndrome, but it is not known whether the micr
127 to implicate viral infection in the cause of Sjogren's syndrome, but there are no definitive studies
128 us to develop a therapy in the treatment of Sjogren's syndrome by increasing the water permeability
129 ry glands from pathologic conditions such as Sjogren's syndrome can result in glandular destruction a
131 (95% CI: 0.8, 7.8) for RA, scleroderma, and Sjogren's syndrome combined and 1.9 (95% CI: 0.6, 6.2) f
135 Hashimoto's thyroiditis and Graves' disease, Sjogren's syndrome, dermatitis herpetiformis, recurrent
136 017), the findings for rheumatoid arthritis, Sjogren's syndrome, dermatomyositis or polymyositis, or
137 ch is a common target of the autoimmunity of Sjogren's syndrome, develop an illness similar to Sjogre
138 or autoimmune disorders, and in the case of Sjogren's syndrome directly in saliva, requiring about a
139 dose-ranging cohort 1, patients with a EULAR Sjogren's Syndrome Disease Activity Index (ESSDAI) score
140 hemolytic anemia, interstitial cystitis, and Sjogren's syndrome had higher prevalence rates among pat
141 mothers with systemic lupus erythematosus or Sjogren's syndrome, had a second child with cardiac NL (
142 (MHC) locus, an association that in SLE and Sjogren's syndrome has long been thought to arise from a
143 ogenesis of systemic lupus erythematosus and Sjogren's syndrome has provided a strong rationale to sp
145 ing excellent oral hygiene, individuals with Sjogren's syndrome have elevated levels of dental caries
146 Recent studies on each of these steps in Sjogren's syndrome have suggested their role in pathogen
147 es), including systemic lupus erythematosus, Sjogren's syndrome, hemolytic anemia, thrombocytopenia,
148 syndrome in 4, systemic lupus erythematosus/Sjogren's syndrome in 1, and undifferentiated autoimmune
150 s had manifestations of autoimmune diseases (Sjogren's syndrome in 4, systemic lupus erythematosus/Sj
151 immunosuppressive medications and secondary Sjogren's syndrome in the development of lymphoma in SLE
152 nitive studies are still lacking for primary Sjogren's syndrome, incidental reports indicating potent
153 ncluding systemic lupus erythematosus (SLE), Sjogren's syndrome, inflammatory bowel disease and multi
163 oes not cause lacrimal gland inflammation, a Sjogren's syndrome-like pathology in lacrimal tissue, or
164 lacrimal tissue, leading to inflammation, a Sjogren's syndrome-like pathology, and aqueous tear defi
165 on also increases incidence of a spontaneous Sjogren's syndrome-like pathology, characterized by lymp
167 crimal gland lesions in this murine model of Sjogren's syndrome may contribute to the predominantly T
168 nts with rheumatoid arthritis (RA) (n = 80), Sjogren's syndrome (n = 30), polymyositis and dermatomyo
169 nkylosing spondylitis (AS) (n = 10), primary Sjogren's syndrome (n = 6), juvenile chronic arthritis (
170 NA-1, the Caenorhabditis elegans ortholog of Sjogren's Syndrome Nuclear Antigen 1, is a constituent o
173 other autoimmune diseases, including primary Sjogren's syndrome (OR = 2.45 in Chinese, OR = 2.35 in E
174 crinopathy can be encountered alone (primary Sjogren's syndrome) or in the presence of another autoim
176 h autoimmune disease (P = 0.005), especially Sjogren's syndrome (P = 0.001) and thyroid disease (P =
177 ex (ESSDAI) score of 5 or higher and a EULAR Sjogren's Syndrome Patient Reported Index (ESSPRI) score
178 Kingdom, HERV-K113 was found in 15.6% of 96 Sjogren's syndrome patients (p < 0.01) and 11.9% of 109
179 or salivary gland biopsies from controls and Sjogren's syndrome patients identified increased express
180 Accordingly, labial biopsies from primary Sjogren's syndrome patients showed reduced Gal1 expressi
182 rom Sjogren's syndrome patients, 37 from non-Sjogren's syndrome patients), 2 of which were positive.
183 Ninety-two samples were examined (55 from Sjogren's syndrome patients, 37 from non-Sjogren's syndr
184 ents from parental NOD mice or human primary Sjogren's syndrome patients, but not serum IgG from heal
188 on injury, Salmonella infection, uveitis and Sjogren's syndrome, PEPITEM reduced T cell recruitment i
189 2 diabetes, B-cell lymphoma, lichen planus, Sjogren's syndrome, porphyria cutanea tarda, rheumatoid-
190 ncluding systemic lupus erythematosus (SLE), Sjogren's syndrome, primary biliary cirrhosis, and activ
191 stemic lupus erythematosus (SLE) and primary Sjogren's syndrome (pSS) are typically characterized by
201 repertoire in the parotid glands of primary Sjogren's syndrome (pSS) patients before and after ritux
204 veral autoimmune diseases, including primary Sjogren's syndrome (pSS), are associated with an increas
206 n which a total of 497 subjects with primary Sjogren's syndrome received 150 international units of h
207 nce interval (CI): 1.35 - 1.51, p < 0.0001], Sjogren's syndrome-related (aHR: 1.67, 95% CI:1.46 - 1.9
208 .67, 95% CI:1.46 - 1.90, p < 0.0001) and non-Sjogren's syndrome-related subtypes (aHR: 1.38, 95% CI:
209 cleroderma, systemic lupus erythematosus, or Sjogren's syndrome (relative risk = 2.0, 95% confidence
210 c NHL subtypes were strongly associated with Sjogren's syndrome: salivary gland (OR = 290, 95% CI: 33
211 received an alternative diagnosis, including Sjogren's syndrome, sensory chronic inflammatory demyeli
212 e and safety of minor salivary gland biopsy, Sjogren's syndrome serves as a prototype model to study
217 detection of patient serum autoantibodies in Sjogren's Syndrome (SjS), a chronic autoimmune disease a
218 The role of complement in the etiology of Sjogren's syndrome (SjS), a human autoimmune disease man
219 s that plays a direct role in development of Sjogren's syndrome (SjS), a systemic autoimmune disease
220 vely, many features of primary and secondary Sjogren's syndrome (SjS), an autoimmune disease affectin
221 s reacting against salivary gland tissues in Sjogren's syndrome (SjS), an autoimmune disease targetin
222 M3R is often the target of autoantibodies in Sjogren's syndrome (SjS), chemical agonists for M3R are
223 al glands (LGs) of male NOD mice, a model of Sjogren's syndrome (SjS), exhibit immune cell infiltrati
225 vary gland epithelial cells in patients with Sjogren's syndrome (SS) and in NOD and NODscid mice expr
226 salivary glands of control subjects without Sjogren's syndrome (SS) and patients with SS who had low
227 E) and heart block, as well as patients with Sjogren's syndrome (SS) and systemic lupus erythematosus
228 -associated meibomian gland disease (MGD) or Sjogren's syndrome (SS) aqueous tear deficiency (ATD).
229 ubgroups with meibomian gland disease (MGD), Sjogren's syndrome (SS) aqueous tear deficiency, or neur
230 cular surface in autoimmune diseases such as Sjogren's syndrome (SS) causes an aqueous-deficient dry
232 44-year-old woman with a 12-year history of Sjogren's syndrome (SS) developed a low-grade mucosa-ass
234 inflammation accelerates the development of Sjogren's syndrome (SS) in genetically susceptible mice.
244 hronic inflammation of the ocular surface in Sjogren's syndrome (SS) is associated with a vision-thre
247 ogenesis can occur in the salivary glands of Sjogren's syndrome (SS) patients and is associated with
248 Cathepsin S (CTSS) is highly increased in Sjogren's syndrome (SS) patients tears and in tears and
250 uropean Cooperative Group (EEC) criteria for Sjogren's Syndrome (SS) should lead to less confusion in
252 Salivary gland inflammation is a hallmark of Sjogren's syndrome (SS), a common autoimmune disease cha
254 (RA), type 1 diabetes mellitus (DM), primary Sjogren's syndrome (SS), and systemic lupus erythematosu
255 6 normal subjects, six subjects with primary Sjogren's syndrome (SS), and three subjects with SS/SLE
258 have been reported in patients with primary Sjogren's syndrome (SS), the role of IL-7 in this diseas
259 acrimal and salivary glands, readily induced Sjogren's syndrome (SS)-like KCS in the recipients.
278 medical comorbidities (i.e., hypothyroidism, Sjogren's syndrome, systemic lupus erythematosus), parti
279 requently found in the sera of patients with Sjogren's syndrome, systemic lupus erythematosus, and co
280 of autoantibodies in patients suffering from Sjogren's syndrome, systemic lupus erythematosus, and ne
281 targeted by autoantibodies in patients with Sjogren's syndrome, systemic lupus erythematosus, and ne
282 nly associated with each other, particularly Sjogren's syndrome, systemic lupus erythematosus, and sy
283 hildren appear to be more closely related to Sjogren's syndrome than systemic lupus erythematosus.
284 ated with the most common symptom of primary Sjogren's syndrome, the loss of salivary gland function.
286 nt part in the autoimmune disease process of Sjogren's syndrome; therefore, several therapies aiming
287 alpha-fodrin protein, common autoantigens in Sjogren's syndrome, via extracellular vesicles in an apo
289 ave been observed in APS1 patients including Sjogren's syndrome, vitiligo, alopecia, uveitis, and oth
296 and spatial analyses of tissues affected by Sjogren's syndrome were undertaken, including the quanti
297 tibodies have been observed in patients with Sjogren's syndrome who are characterised by dryness of b
298 In this report, we describe a female with Sjogren's syndrome who had acute visual loss due to ocul
299 in our understanding of the pathogenesis of Sjogren's syndrome with a focus on the relationship betw
300 y increase UWS flow in patients with primary Sjogren's syndrome, without causing significant adverse