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1 al skin flora (e.g., Cutibacterium acnes and Staphylococcus epidermidis).
2 li DH5alpha) and halotolerant cells (such as Staphylococcus epidermidis).
3 y to the phenol-soluble modulins (PSMs) from Staphylococcus epidermidis.
4 ould confer protection against S. aureus and Staphylococcus epidermidis.
5 r crRNA primary processing and maturation in Staphylococcus epidermidis.
6 nsing (QS) receptor in the emerging pathogen Staphylococcus epidermidis.
7 n binding protein present in most strains of Staphylococcus epidermidis.
8 the Gram-positive device-associated pathogen Staphylococcus epidermidis.
9 ostridium subterminale, Escherichia coli, or Staphylococcus epidermidis.
10 d b and strains of Staphylococcus aureus and Staphylococcus epidermidis.
11 timulate growth and formation of biofilms by Staphylococcus epidermidis.
12 ntration on killing of a clinical isolate of Staphylococcus epidermidis.
13 usceptibility to vancomycin in an isolate of Staphylococcus epidermidis.
14 well as Gram-positive Bacillus subtilis and Staphylococcus epidermidis.
15 phenol-soluble modulin, a factor secreted by Staphylococcus epidermidis.
16 also in whole blood cultures stimulated with Staphylococcus epidermidis.
17 is of prosthetic-device infections caused by Staphylococcus epidermidis.
18 gens were Staphylococcus aureus, followed by Staphylococcus epidermidis.
19 S) to type 35 well-characterized isolates of Staphylococcus epidermidis.
20 heads incubated with and without exposure to Staphylococcus epidermidis.
21 included, with 250 (77%) episodes involving Staphylococcus epidermidis.
22 otic resistance in Staphylococcus aureus and Staphylococcus epidermidis.
23 ee of four mAbs showed cross-reactivity with Staphylococcus epidermidis.
24 culture in the ATB-P group was positive for Staphylococcus epidermidis.
25 y sepsis caused by Staphylococcus aureus and Staphylococcus epidermidis.
26 adulthood, with an increase of the commensal Staphylococcus epidermidis.
27 /- 0.9-fold) on exposure to S aureus but not Staphylococcus epidermidis.
28 une responses with Staphylococcus aureus and Staphylococcus epidermidis.
29 a GT-B folded teichoic acid polymerase from Staphylococcus epidermidis.
30 formation is the primary virulence factor of Staphylococcus epidermidis.
31 njected with viable Staphylococcus aureus or Staphylococcus epidermidis (1,000 or 500,000 colony-form
32 jects with PJI (40 Staphylococcus aureus, 40 Staphylococcus epidermidis, 10 Staphylococcus lugdunensi
34 ant staphylococci, with S aureus (42.0%) and Staphylococcus epidermidis (20.0%) as the predominant sp
35 en biofilm formation and hemagglutination in Staphylococcus epidermidis, 20 skin isolates and 19 pros
36 am-positive bacteria were isolated in 78.5%; Staphylococcus epidermidis (28.6%) was the most common m
42 ity islands have been found in the genome of Staphylococcus epidermidis, a generally less virulent re
45 ry trajectory and functional distribution of Staphylococcus epidermidis-a keystone skin microbe and o
46 f all Gram-negative/fungal episodes) whereas Staphylococcus epidermidis accounted for only 1 death (0
47 izing filter (2.1%) became contaminated with Staphylococcus epidermidis after 1 month of treatment, w
48 .01 muM, while the truncated analogue of the Staphylococcus epidermidis AIP-1 (3) elicited an IC50 va
49 f bacterial commensals (Cutibacterium acnes, Staphylococcus epidermidis) among OA versus Malaysian an
51 tion of two related opportunistic pathogens, Staphylococcus epidermidis and Enterococcus faecalis, wa
52 rthopedic device-related infection caused by Staphylococcus epidermidis and four different Cutibacter
53 haride produced by Staphylococcus aureus and Staphylococcus epidermidis and is an effective target fo
54 r medical applications, virulence factors in Staphylococcus epidermidis and Klebsiella pneumoniae are
55 esulted in a 1- to 2-log enhanced killing of Staphylococcus epidermidis and other microbes in vitro c
56 acteria, mostly normal skin bacteria such as Staphylococcus epidermidis and Propionibacterium acnes,
57 OH radicals, Cu, Fe, Mn, Zn, and ABA against Staphylococcus epidermidis and Pseudomonas aeruginosa we
59 hic alpha-helix is inducible within hours by Staphylococcus epidermidis and slowly by another mechani
66 erized by a low bacterial load (dominated by Staphylococcus epidermidis and streptococci), high preva
67 phomonas maltophilia, Staphylococcus aureus, Staphylococcus epidermidis and Streptococcus pyogenes in
68 It also demonstrates potent activity against Staphylococcus epidermidis and Streptococcus spp. (MIC90
69 tive behavior of living materials comprising Staphylococcus epidermidis and telechelic block copolyme
70 nce genes (mecA in Staphylococcus aureus and Staphylococcus epidermidis and vanA or vanB in Enterococ
72 nst gram-positive (Staphylococcus aureus and Staphylococcus epidermidis) and gram-negative (Escherich
73 in vitro >/= 90% reduction of Gram-positive (Staphylococcus epidermidis) and Gram-negative (Pseudomon
74 the upper airway (Staphylococcus aureus and Staphylococcus epidermidis) and intestinal microbiota (L
75 inst selected bacteria (Escherichia coli and Staphylococcus epidermidis) and yeast (Candida albicans)
76 (Escherichia coli), Gram-positive bacteria (Staphylococcus epidermidis), and T7 bacteriophage virus
77 ates of Staphylococcus aureus, 2 isolates of Staphylococcus epidermidis, and 7 isolates of Staphyloco
80 nst Escherichia coli, Staphylococcus aureus, Staphylococcus epidermidis, and group B Streptococcus.
82 ong skin microbes, Staphylococcus aureus and Staphylococcus epidermidis are associated with AD, but h
84 ections with the leading nosocomial pathogen Staphylococcus epidermidis are characterized by biofilm
89 coagulase-negative staphylococci, including Staphylococcus epidermidis, are often considered a conta
90 tive staphylococci, with the leading species Staphylococcus epidermidis, are the predominant cause of
91 and to pathogens Bacillus thuringiensis and Staphylococcus epidermidis, as by the survival rate of t
92 le cells of another Gram-positive bacterium, Staphylococcus epidermidis, as well as of the Gram-negat
93 nation ability, by artificially loading with Staphylococcus epidermidis at different stages of proces
94 t in in vitro antibacterial activity against Staphylococcus epidermidis at loadings of silver that ar
95 eudomonas aeruginosa, Bacillus subtilis, and Staphylococcus epidermidis at the single cell and popula
96 a KT2440, Salmonella Typhimurium ATCC 14028, Staphylococcus epidermidis ATCC 12228, Enterococcus faec
97 ut against Escherichia coli (ATCC 11229) and Staphylococcus epidermidis (ATCC 12228) using Kirby Baue
99 rt of the failure of linezolid treatment for Staphylococcus epidermidis bacteremia associated with a
100 at in normal skin and that colonization with Staphylococcus epidermidis bacteria amplified this effec
102 s been implicated as an Escherichia coli and Staphylococcus epidermidis biofilm adhesin, the formatio
103 ucosamine (PNAG) is a major component of the Staphylococcus epidermidis biofilm extracellular matrix.
106 electrical current reduced the viability of Staphylococcus epidermidis biofilms in conjunction with
110 mediate-frequency alleles in an arcC gene of Staphylococcus epidermidis, but not in a homologous gene
111 ere also seen with PS/A-producing strains of Staphylococcus epidermidis, but not with transposon muta
112 lling of 10(3)-10(8) colony forming units/ml Staphylococcus epidermidis by 10(5)-10(8) human neutroph
114 trains of Escherichia coli and one strain of Staphylococcus epidermidis by hierarchy cluster analysis
115 d that N-deacetylation of beta-1,6-GlcNAc in Staphylococcus epidermidis by the PgaB homolog, IcaB, an
117 examined against Pseudomonas aeruginosa and Staphylococcus epidermidis by varying the dose, the time
118 lebsiella pneumoniae, Staphylococcus aureus, Staphylococcus epidermidis, Candida albicans, and K. kin
120 re, we show that the abundant skin commensal Staphylococcus epidermidis contributes to skin barrier i
123 endophthalmitis, the odds of having positive Staphylococcus epidermidis cultures were significantly l
124 tant Staphylococcus aureus cultures, 9.2% of Staphylococcus epidermidis cultures, and 2.5% of P. aeru
125 artments were observed for viruses, archaea, Staphylococcus epidermidis, Cutibacterium acnes, and Mal
126 ellular adhesin (PIA) in the pathogenesis of Staphylococcus epidermidis CVC-associated infection.
128 atment of experimental methicillin-resistant Staphylococcus epidermidis endophthalmitis was evaluated
130 s maltophilia ) and Gram-positive bacteria ( Staphylococcus epidermidis , Enterococcus faecalis , and
131 llin-resistant Staphylococcus aureus (MRSA), Staphylococcus epidermidis, Enterococcus faecalis, Acine
132 strains that have been tested, B. subtilis, Staphylococcus epidermidis, Enterococcus faecalis, and E
133 d on surfaces, and belong to species such as Staphylococcus epidermidis, Enterococcus faecalis, Pseud
137 ort on an analysis of sequential isolates of Staphylococcus epidermidis from cultures of blood obtain
140 Here, we report the crystal structure of the Staphylococcus epidermidis glucose/H(+) symporter in an
142 rR, a DtxR homologue initially identified in Staphylococcus epidermidis, governs the expression of th
143 Typhimurium (all gram-negative bacteria) and Staphylococcus epidermidis (gram-positive) showed marked
144 hylococcus lugdunensis (groups I and II) and Staphylococcus epidermidis (groups I-III) were sequentia
151 ding clinical isolates of MRSA and MSSA) and Staphylococcus epidermidis identified one candidate that
152 oli, nontypeable Haemophilus influenzae, and Staphylococcus epidermidis Importantly, while replacemen
153 The most common organisms identified were Staphylococcus epidermidis in 30.1% (135/448), Streptoco
154 The relationship of initial concentration of Staphylococcus epidermidis in blood cultures and time to
155 isolation of a strictly anaerobic strain of Staphylococcus epidermidis in pure culture from the site
156 es the selective and capacitive detection of Staphylococcus epidermidis in synthetic urine also conta
158 endophthalmitis and revealed the presence of Staphylococcus epidermidis in the vitreous of a culture-
159 ocess in the type III-A CRISPR-Cas system of Staphylococcus epidermidis, in the absence of phage sele
160 factor (STF) and phenol-soluble modulin from Staphylococcus epidermidis induced HIV-LTR trans-activat
162 be effective at reducing both S. aureus and Staphylococcus epidermidis infections in a murine animal
163 nd show that the common blood-borne pathogen Staphylococcus epidermidis influences this in vitro mode
164 e accumulation-associated protein (Aap) from Staphylococcus epidermidis is a biofilm-related protein
166 The skin-colonizing commensal bacterium Staphylococcus epidermidis is a leading cause of hospita
179 oteins whose secretion from stationary-phase Staphylococcus epidermidis is dependent on SPase activit
180 Methicillin (beta-lactam) resistance in Staphylococcus epidermidis is mediated by the mecA gene,
186 edical devices in hospitalized patients, and Staphylococcus epidermidis is the leading cause of infec
190 , we demonstrated that the saePQRS region in Staphylococcus epidermidis is transcriptionally regulate
192 s level from the OW (126) and ER (117), with Staphylococcus epidermidis isolates being the most commo
193 Similar proteolytic capacities were found in Staphylococcus epidermidis isolates but not in Staphyloc
194 ofiling 14,884 pairwise interactions between Staphylococcus epidermidis isolates cultured from 18 peo
195 the species level as containing 203 (73.3%) Staphylococcus epidermidis isolates, 10 (3.6%) Staphyloc
197 aoperative vitreous culture grew 1 colony of Staphylococcus epidermidis, judged a likely contaminant
198 tic pathogens such as Staphylococcus aureus, Staphylococcus epidermidis, Klebsiella pneumoniae, Klebs
199 t between individuals in single centers, and Staphylococcus epidermidis lineages persist within and,
200 ead of multidrug-resistant, hospital-adapted Staphylococcus epidermidis lineages underscores the need
202 valuated whether the commensal microorganism Staphylococcus epidermidis may enhance production of ant
203 sibility, we report the crystal structure of Staphylococcus epidermidis MDD (1.85 A resolution) and,
207 y a normal microbial resident of human skin, Staphylococcus epidermidis, might also act as an antimic
208 us aureus (MSSA) (14), methicillin-resistant Staphylococcus epidermidis (MRSE) (17), methicillin-susc
209 endocarditis caused by methicillin-resistant Staphylococcus epidermidis (MRSE) and a rare linezolid-r
211 illin-resistant Stapylococcus aureus (MRSA), Staphylococcus epidermidis (MRSE), and Mycobacterium spp
212 ermidis (MRSE) (17), methicillin-susceptible Staphylococcus epidermidis (MSSE) (9), other coagulase-n
213 010, we recovered 38 methicillin-susceptible Staphylococcus epidermidis (MSSE) isolates from endophth
214 were 1 x 108 colony forming units of either Staphylococcus epidermidis (n = 100) or Enterobacter clo
216 = 99); 176 received tail vein injections of Staphylococcus epidermidis on postoperative day (POD) 10
219 mentally inoculated with Enterobacteriaceae, Staphylococcus epidermidis or Staphylococcus hominis yie
220 mentally inoculated with Enterobacteriaceae, Staphylococcus epidermidis or Staphylococcus hominis yie
221 romonas gingivalis, Fusobacterium nucleatum, Staphylococcus epidermidis, or Cutibacterium acnes, and
222 cillus actinomycetemcomitans, skin commensal Staphylococcus epidermidis, or skin pathogen Streptococc
226 Therefore, we investigated the impact of the Staphylococcus epidermidis phenotype on colonization of
228 monstrates that quorum-sensing regulation in Staphylococcus epidermidis protects it from key mechanis
229 carditis due to Rhodotorula mucilaginosa and Staphylococcus epidermidis, proven by culture and histop
230 show here that d-lactate dehydrogenase from Staphylococcus epidermidis reduces a broad spectrum of 2
233 demonstrate that a model type III system in Staphylococcus epidermidis relies upon the activities of
234 e structural and biochemical features of two Staphylococcus epidermidis RNase J paralogs, RNase J1 an
237 ensing gate for the early stage detection of Staphylococcus epidermidis (S. epidermidis) biofilm form
238 positive Enterococcus durans (E. durans) and Staphylococcus epidermidis (S. epidermidis) by PAA combi
239 emical experiments show that both TthCsm and Staphylococcus epidermidis (S. epidermidis) Csm (SepCsm)
240 cnes, Staphylococcus aureus (S. aureus), and Staphylococcus epidermidis (S. epidermidis) with lauric
244 a significant reduction in viable counts of Staphylococcus epidermidis, Saccharomyces cerevisiae, an
245 ria and viruses, including Escherichia coli, Staphylococcus epidermidis, Salmonella enterica, MS2, P2
246 DNA fragment was identified and cloned from Staphylococcus epidermidis (Se) using femA from S. aureu
247 ginosa [PA], Staphylococcus aureus [SA], and Staphylococcus epidermidis [SE]) was tested by using Kir
248 in commensal and leading nosocomial pathogen Staphylococcus epidermidis senses and efficiently inacti
250 ecreted polypeptides from the skin commensal Staphylococcus epidermidis, soluble tuberculosis factor
252 ng, and wound; the most common isolates were Staphylococcus epidermidis, Staphylococcus aureus, and C
253 gel surface is active against Gram-positive (Staphylococcus epidermidis, Staphylococcus aureus, and S
254 identification of 20 blood-borne pathogens (Staphylococcus epidermidis, Staphylococcus aureus, Bacil
256 ebsiella pneumoniae, Pseudomonas aeruginosa, Staphylococcus epidermidis, Staphylococcus aureus, Enter
257 s detected by radial diffusion assay against Staphylococcus epidermidis, Staphylococcus aureus, Esche
260 Staphylococcus spp., Staphylococcus aureus, Staphylococcus epidermidis, Staphylococcus lugdunensis,
262 documented isolation of a strictly anaerobic Staphylococcus epidermidis strain, confirmed by rpoB gen
265 ve bacterial species (Staphylococcus aureus, Staphylococcus epidermidis, Streptococcus pneumoniae, En
266 lanB) which have substantial homology to the Staphylococcus epidermidis structural gene (epiA) and a
267 y expressed repeats of the AtlE amidase from Staphylococcus epidermidis suggest that the repeating se
268 y related accumulation-associated protein of Staphylococcus epidermidis, suggesting the possibility o
269 mmation initiated by a single application of Staphylococcus epidermidis supernatant, correlating with
271 SdrG is a cell wall-anchored adhesin from Staphylococcus epidermidis that binds to the Bbeta chain
272 the growth of both Staphylococcus aureus and Staphylococcus epidermidis, the latter were unable to el
273 of PSMs in in vitro and in vivo biofilms of Staphylococcus epidermidis, the leading cause of indwell
274 ntified as Staphylococcus lugdunensis (one), Staphylococcus epidermidis (three), Staphylococcus haemo
275 ght to be a crucial factor in the ability of Staphylococcus epidermidis to produce a biomaterial-base
279 that transcription across the targets of the Staphylococcus epidermidis type III-A CRISPR-Cas system
280 inetobacter and Moraxella species (type II), Staphylococcus epidermidis (type III), Porphyromonas and
282 ms from the primary broth culture other than Staphylococcus epidermidis, viridans group streptococci,
283 oll-like receptors with Candida albicans and Staphylococcus epidermidis was 2.5- and 2.9-fold higher
284 hermic preservation by investigating whether Staphylococcus epidermidis was capable of growing in a s
285 miting the spread of conjugative plasmids in Staphylococcus epidermidis was first described in 2008.
288 ated catheters from studied patients against Staphylococcus epidermidis was used to determine the ant
289 ysiology of the leading nosocomial pathogen, Staphylococcus epidermidis, we analyzed the genome of bi
290 n beta-defensin 3 in the nosocomial pathogen Staphylococcus epidermidis, we discovered an antimicrobi
292 e ligands for E. coli, Citrobacter freundii, Staphylococcus epidermidis were 100%, 2.6% and 8.6% resp
293 Fibrin gels (1500 microm thick) containing Staphylococcus epidermidis were formed in Boyden-type ch
295 ein fractions from Staphylococcus aureus and Staphylococcus epidermidis, were able to distinguish bet
296 did not occur after fermentation, similar to Staphylococcus epidermidis, while S. aureus and S. sapro
298 h of the pathogens Staphylococcus aureus and Staphylococcus epidermidis with comparable IC(50) to van
299 ynebacterium glutamicum, Microcuccus luteus, Staphylococcus epidermidis, Yersinia ruckeri, Escherichi
300 level, dominated by Cutibacterium acnes and Staphylococcus epidermidis, yet each person harbors a un