ライフサイエンスコーパス: Streptococcus

1 r isolation of the large-zone beta-hemolytic streptococcus.

2 re studies and applications within the genus Streptococcus.

3 influenza; 2 human herpesvirus 6; 2 group B Streptococcus; 2 Streptococcus pneumoniae; 1 HSV; 1 pare

4 Prevotella (8.8%), Propionibacterium (7.2%), Streptococcus (3.9%), Selenomonas (3.7%), Corynebacteriu

5 Staphylococcus (403/950, 42.4%) and streptococcus (337/950, 35.5%) species were most common.

6 Streptococcus abundance showed an inverse association ov

7 Streptococcus agalactiae (group B Streptococcus [GBS]) i

8 Streptococcus agalactiae (group B streptococcus, or GBS)

9 Here we describe Streptococcus agalactiae DPC7040, a human faecal isolate

10 Listeria monocytogenes, Streptococcus spp., Streptococcus agalactiae, Streptococcus anginosus group,

11 relationship with percent of Firmicutes and Streptococcus and a negative association with percent Pr

12 Streptococcus and Actinomyces are key potential microbia

13 type strain of the type species of the genus Streptococcus and an important human pathogen that cause

14 fied genes, found in Listeria, Enterococcus, Streptococcus and Staphylococcus genomes, can inhibit ty

15 ent in infant humans (e.g., Bifidobacterium, Streptococcus, and Bacteroides), and chimpanzee gut micr

16 Cytomegalovirus, Escherichia coli, group B Streptococcus, and other infections contributed to 30 (1

17 treptococcus spp., Streptococcus agalactiae, Streptococcus anginosus group, Streptococcus pneumoniae,

18 We also show that AcrIIA21 inhibits SpyCas9, Streptococcus aureus Cas9 (SauCas9) and SinCas9 with low

19 everal orthologs to engineer two variants of Streptococcus canis Cas9-Sc(++) and a higher-fidelity mu

20 d pig-specific species within Lactobacillus, Streptococcus, Clostridium, Desulfovibrio, Enterococcus,

21 ovirus (HMPV) illness complicated by group A Streptococcus coinfection, progressing to acute respirat

22 Haemophilus-low cluster (n = 121), in which Streptococcus demonstrated the highest relative abundanc

23 we quantified the secondary invasive group A Streptococcus disease risk among household contacts.

24 determined X-ray crystal structures of both Streptococcus dysgalactiae Rgg2 and S. thermophilus Rgg3

25 eonatal mice had high relative abundances of Streptococcus, Escherichia, and Enterococcus and increas

26 The group A Streptococcus (GAS) causes diseases that range from mild

27 The concept that a minority of group A streptococcus (GAS) emm types are more "rheumatogenic" t

28 The human pathogen group A streptococcus (GAS) encounters the host defense factor c

29 ostics Inc., Murrieta, CA) to detect group A Streptococcus (GAS) from throat specimens.

30 ctions, we studied i.p. infection by group A Streptococcus (GAS) in wild-type (WT) and Il17d (-/-) mi

31 Group A Streptococcus (GAS) infection causes a range of diseases

32 Group A Streptococcus (GAS) infection causes a range of life-thr

33 is triggered in children by repeated group A Streptococcus (GAS) infections that lead to neuropsychia

34 Group A Streptococcus (GAS) is a human-specific pathogen and maj

35 Group A streptococcus (GAS) is a major pathogen that impacts hea

36 Group A Streptococcus (GAS) is one of the leading causes of bact

37 Group A Streptococcus (GAS) is the etiologic agent of numerous h

38 induced in the skin of patients with group A Streptococcus (GAS) NF.

39 Pharyngitis due to group A Streptococcus (GAS) represents a major cause of outpatie

40 Group A streptococcus (GAS) species cause bacterial pharyngitis

41 ens causing puerperal sepsis include group A Streptococcus (GAS), and epidemiological analyses have i

42 binding in the major human pathogen group A Streptococcus (GAS).

43 Group A Streptococcus (GAS; Streptococcus pyogenes) is a bacteri

44 treatment of Streptococcus pyogenes (group A Streptococcus [GAS]) infections.

45 Streptococcus pyogenes (group A Streptococcus [GAS]) is a human pathogen responsible for

46 Streptococcus pyogenes (group A Streptococcus [GAS]), a major human-specific pathogen, r

47 M12 (emm12) Streptococcus pyogenes (group A Streptococcus, GAS) to cause scarlet fever has been link

48 against the frequent human pathogen group B Streptococcus (GBS) and other extracellular bacteria.

49 bit growth and biofilm production in Group B Streptococcus (GBS) and Staphylococcus aureus.

50 Group B Streptococcus (GBS) can be found to colonize about 25% o

51 Maternal immunization against group B streptococcus (GBS) could protect infants from invasive

52 The rates of early-onset Group B Streptococcus (GBS) disease (EOGBS) have declined since

53 der to minimize the impact of infant group B streptococcus (GBS) disease in the United Kingdom.

54 as utilised to covalently ligate the Group B Streptococcus (GBS) GBS67 protein antigen with the CpGOD

55 The burden of non-invasive Group B Streptococcus (GBS) infections in adults is unknown.

56 Group B streptococcus (GBS) is a leading cause of young infant m

57 trointestinal (GI) colonization with group B Streptococcus (GBS) is an important precursor to late-on

58 Group B streptococcus (GBS) is the leading cause of neonatal inv

59 choriodecidual inoculation of either group B Streptococcus (GBS) or saline (n = 5/group).

60 Licensure of a Group B streptococcus (GBS) polysaccharide-protein conjugate vac

61 reus, we found the neonatal pathogen group B Streptococcus (GBS) to be remarkably resistant to platel

62 Group B Streptococcus (GBS), is a leading cause of neonatal deat

63 aused by Escherichia coli (E. coli), Group B Streptococcus (GBS), Listeria monocytogenes, Haemophilus

64 Streptococcus agalactiae (group B Streptococcus [GBS]) is an important cause of invasive i

65 rate biosynthesis pathways in species of the Streptococcus genus of high clinical and veterinary impo

66 rview of the genomic diversity of the entire Streptococcus genus.

67 Firmicutes (phylum level) and Haemophilus or Streptococcus (genus level) dominant.

68 Streptococcus gordonii (mitis group) has been shown to b

69 In addition, we found that Streptococcus gordonii (S. gordonii) and Enterococcus fa

70 Streptococcus gordonii is a primary colonizer of the hum

71 ococcus mutans, Streptococcus sanguinis, and Streptococcus gordonii, they exhibited antibacterial eff

72 pxB abundance in Streptococcus sanguinis and Streptococcus gordonii.

73 omprised of Corynebacterium, Staphylococcus, Streptococcus, Haemophilus and Moraxella species in both

74 Reported outbreaks of invasive group A Streptococcus (iGAS) infections among people who inject

75 emm types commonly found in invasive group A Streptococcus (iGAS) strains recovered through the Cente

76 th increased abundance of Staphylococcus and Streptococcus in the lungs, yet their roles in disease p

77 es during the acute and resolution phases of Streptococcus-induced sepsis.

78 ortance, but despite the ubiquity of group A Streptococcus infections, the relationship between infec

79 We show that AcrIIA20 strongly inhibits Streptococcus iniae Cas9 (SinCas9) and weakly inhibits S

80 dozens of species became fatally infected by Streptococcus iniae Piscivores and benthivores were disp

81 Group A Streptococcus is a pathogen of global importance, but de

82 y urine specimen with >=10(4) CFU/ml group B Streptococcus is significant for asymptomatic bacteriuri

83 extent that species belonging to the genera Streptococcus, Listeria, Staphylococcus, Lactobacillus,

84 -NAAN-3[Formula: see text] PAM preference of Streptococcus macacae Cas9 (SmacCas9).

85 titative polymerase chain reaction to detect Streptococcus mitis, Streptococcus sobrinus, Streptococc

86 l interaction, while the cariogenic pathogen Streptococcus mutans (mutans group) interacts with the f

87 to measure the local glucose consumption of Streptococcus mutans (S. mutans) biofilms.

88 Introducing the same mutation in Streptococcus mutans also provided a phage resistance ph

89 In early childhood caries, Streptococcus mutans and Candida albicans are often co-i

90 cterial species, including caries-associated Streptococcus mutans as well as several periodontal path

91 Using the Streptococcus mutans HdrRM LRS as a model, we demonstrat

92 ically the acidophilic and caries-associated Streptococcus mutans in 17-year old Swedish adolescents

93 Streptococcus mutans is an etiologic agent of human dent

94 When Streptococcus mutans is transferred from a preferred car

95 though some studies focused on understanding Streptococcus mutans' response to fluoride, the mechanis

96 Using an oral bacterium (Streptococcus mutans), we find that microbial cells sett

97 ctivities against 3 single-species biofilms: Streptococcus mutans, Actinomyces naeslundii, and Entero

98 good dental health, whereas Prevotella spp., Streptococcus mutans, and Human herpesvirus 4 (Epstein-B

99 tivity against periodontal pathogens such as Streptococcus mutans, Porphyromonas gingivalis, Fusobact

100 Streptococcus mitis, Streptococcus sobrinus, Streptococcus mutans, Streptococcus oralis, and Candida

101 imum bactericidal concentration test against Streptococcus mutans, Streptococcus sanguinis, and Strep

102 In Streptococcus mutans, two Spx homologues, SpxA1 and SpxA

103 factor is cariogenic microorganisms such as Streptococcus mutans.

104 bohydrate metabolized by the dental pathogen Streptococcus mutans.

105 noglobulin (IVIG) from patients with group A Streptococcus necrotizing soft tissue infections demonst

106 1.5 times higher risk of CLD than those with Streptococcus or Prevotella (SP)-dominated microbiota (R

107 Streptococcus agalactiae (group B streptococcus, or GBS) is a common cause of bacteremia a

108 yromonas pasteri, Prevotella nanceiensis and Streptococcus oralis decreased, while Veillonella specie

109 nas gingivalis, Fusobacterium nucleatum, and Streptococcus oralis were formed on titanium specimens f

110 treptococcus sobrinus, Streptococcus mutans, Streptococcus oralis, and Candida albicans in the saliva

111 calibacterium (p-for-interaction = 0.03) and Streptococcus (p-for-interaction = 0.004).

112 Streptococcus parasanguinis, a hydrogen peroxide (H(2)O(

113 ively associated with Acinetobacter species, Streptococcus parasanguinis, Streptococcus salivarius, a

114 r clinical application, further detection of Streptococcus pneumonia from 50 to 5x10(4) CFU/mL were s

115 sphate lactonase (UlaG), a protein marker of Streptococcus pneumonia.

116 were Staphylococcus aureus (34/37 [91.9%]), Streptococcus pneumoniae (10/11 [90.9%]), and Enterobact

117 ), whereas blood cultures most commonly grew Streptococcus pneumoniae (33%), followed by S. aureus (2

118 ogens were Klebsiella pneumoniae (86 [31%]), Streptococcus pneumoniae (54 [20%]), HIV (40 [15%]), and

119 Streptococcus pneumoniae (9/44 [20%]) and Staphylococcus

120 The most common pathogens were Streptococcus pneumoniae (93 of 143, 65%) and Haemophilu

121 ens, Streptococcus sanguinis (ComGC(SS)) and Streptococcus pneumoniae (ComGC(SP)), revealing that thi

122 ), human herpesvirus 6 (HHV-6) (n = 30), and Streptococcus pneumoniae (n = 14).

123 n = 70, 21%), Plasmodium (n = 35, 10%), and Streptococcus pneumoniae (n = 31, 9%).

124 Streptococcus pneumoniae (Pnc) serotypes differ in invas

125 Streptococcus pneumoniae (pneumococcus) is a leading cau

126 Streptococcus pneumoniae (pneumococcus) is a principal c

127 ant polysaccharide conjugate vaccine against Streptococcus pneumoniae (pneumococcus).

128 The lowest IE prevalence was found with Streptococcus pneumoniae (S pneumoniae) 1.2% (0.8-1.6) a

129 tion of Staphylococcus aureus (S. aureus) or Streptococcus pneumoniae (S. pneumoniae), respectively;

130 cations for urinary antigen tests (UATs) for Streptococcus pneumoniae (SP) and Legionella pneumophila

131 Streptococcus pneumoniae (Spn) colonizes the nasopharynx

132 Streptococcus pneumoniae (Spn) is an important Gram-posi

133 Streptococcus pneumoniae (Spn) must acquire iron from th

134 Colonization of the nasopharynx with Streptococcus pneumoniae (Spn), although a prerequisite

135 In ovoid-shaped Streptococcus pneumoniae (Spn), septal and peripheral (e

136 Streptococcus pneumoniae (the pneumococcus) is a common

137 Streptococcus pneumoniae (the pneumococcus) is a major c

138 that is also conserved in pathogens such as Streptococcus pneumoniae and Mycobacterium tuberculosis

139 iotic susceptibility and resistance for both Streptococcus pneumoniae and Neisseria gonorrhoeae.

140 were diagnosed with influenza or bacterial (Streptococcus pneumoniae and Staphylococcus aureus) etio

141 Common causative organisms are Streptococcus pneumoniae and Staphylococcus aureus.

142 nce and vaccine escape in the human pathogen Streptococcus pneumoniae can be largely attributed to co

143 Streptococcus pneumoniae can cause disease in various hu

144 ral infections, but the impact of viruses on Streptococcus pneumoniae carriage prevalence and load re

145 Streptococcus pneumoniae choline kinase (sChoK) has prev

146 with positive UAT more often had a positive Streptococcus pneumoniae culture (25.4% vs 1.9%, P < .00

147 Listeria innocua, Pseudomonas aeruginosa and Streptococcus pneumoniae did not interfere the detection

148 ve anaerobe and opportunistic human pathogen Streptococcus pneumoniae generates large amounts of hydr

149 Previous studies in Streptococcus pneumoniae have shown that hsdS inversions

150 ies and provides a general strategy to block Streptococcus pneumoniae IgA1 protease activity to poten

151 copy single particle reconstructions how the Streptococcus pneumoniae IgA1 protease facilitates IgA1

152 Specifically, the Streptococcus pneumoniae IgA1 protease subscribes to an

153 e vaccine (Prevnar-13) against the bacterium Streptococcus pneumoniae induced immune responses that w

154 Streptococcus pneumoniae infection can result in bactere

155 th influenza, mice are better protected from Streptococcus pneumoniae infection due to a population o

156 cortical impact model followed by secondary Streptococcus pneumoniae infection in mice.

157 Secondary Streptococcus pneumoniae infection is a significant caus

158 re, we used a murine model of intrapulmonary Streptococcus pneumoniae infection to investigate the ro

159 cture or bacteremia with Escherichia coli or Streptococcus pneumoniae infection).

160 esponses of AMs and exaggerated responses to Streptococcus pneumoniae infection.

161 Streptococcus pneumoniae is a devastating global pathoge

162 SP_0782 from Streptococcus pneumoniae is a dimeric protein that poten

163 Streptococcus pneumoniae is a leading cause of pneumonia

164 Streptococcus pneumoniae is a major cause of community-a

165 Streptococcus pneumoniae is a major cause of pneumonia,

166 Streptococcus pneumoniae is a major human pathogen that

167 Streptococcus pneumoniae is a major respiratory pathogen

168 Streptococcus pneumoniae is a natural colonizer of the h

169 Streptococcus pneumoniae is a significant cause of otiti

170 The exopolysaccharide capsule of Streptococcus pneumoniae is an important virulence facto

171 Streptococcus pneumoniae is an opportunistic human patho

172 Streptococcus pneumoniae is an opportunistic human patho

173 Streptococcus pneumoniae is one of the world's leading b

174 Streptococcus pneumoniae is responsible for severe infec

175 Streptococcus pneumoniae is responsible for the majority

176 etermine a structural envelope of SpNOX, the Streptococcus pneumoniae NADPH oxidase (NOX), a prokaryo

177 a three-dimensional structure of the related Streptococcus pneumoniae PBP2X suggests that some substi

178 Opportunistic pathogens such as Streptococcus pneumoniae secrete a giant metalloprotease

179 Streptococcus pneumoniae serotype 1 is the predominant c

180 verified here for Shigella sonnei O-antigen, Streptococcus pneumoniae serotype 12F, and Staphylococcu

181 nvasive pneumococcal disease (IPD) caused by Streptococcus pneumoniae serotype 2 (Sp2) is infrequent.

182 Identifying Streptococcus pneumoniae serotypes by urinary antigen de

183 n of asymptomatic nasopharyngeal carriage of Streptococcus pneumoniae to invasive pneumococcal diseas

184 Etiology was defined by blood culture, Streptococcus pneumoniae urinary antigen detection, sput

185 er vaccination with the 13-valent-conjugated Streptococcus pneumoniae vaccine were assessed in a MAIT

186 n sub-Saharan Africa sub-optimally interrupt Streptococcus pneumoniae vaccine-serotype (VT) carriage

187 Streptococcus pneumoniae was cultured in 33 episodes (51

188 lmonella enterica, Staphylococcus aureus and Streptococcus pneumoniae were also isolated.

189 Haemophilus influenzae and Streptococcus pneumoniae were the commonest bacterial pa

190 puncture or infection by Escherichia coli or Streptococcus pneumoniae) and endotoxaemia.

191 rata, 2 coagulase-negative Staphylococcus, 1 Streptococcus pneumoniae).

192 Such has been the case with Streptococcus pneumoniae, an important human pathogen, a

193 evolution, transmission and pathogenesis of Streptococcus pneumoniae, an opportunistic human-adapted

194 lin (Ig) levels, specific antibodies against Streptococcus pneumoniae, and allergen-specific IgE, as

195 kin 17A (IL-17A) response against colonizing Streptococcus pneumoniae, and its transition to a pathog

196 s agalactiae, Streptococcus anginosus group, Streptococcus pneumoniae, and Streptococcus pyogenes), p

197 histoplasmosis; pneumonia (viral, bacterial, Streptococcus pneumoniae, and unspecified pneumonia); in

198 e closely related Streptococcus pyogenes and Streptococcus pneumoniae, and while research on GBS TCSs

199 hepatitis B, Haemophilus influenzae type b, Streptococcus pneumoniae, rotavirus, measles, meningitis

200 In oval-shaped Streptococcus pneumoniae, septal and longitudinal peptid

201 t transcription waves defining competence in Streptococcus pneumoniae.

202 icobacter pylori, Moraxella catarrhalis, and Streptococcus pneumoniae.

203 logous stimulation with Escherichia coli and Streptococcus pneumoniae.

204 man respiratory mucus and the human pathogen Streptococcus pneumoniae.

205 requently identified organism on culture was Streptococcus pneumoniae.

206 an herpesvirus 6; 2 group B Streptococcus; 2 Streptococcus pneumoniae; 1 HSV; 1 parechovirus; 1 enter

207 ective response against the type III group B Streptococcus polysaccharide was comprised within 2 of t

208 x of oral taxa, such as Neisseria lactamica, Streptococcus, Prevotella nanceiensis, Fusobacterium, an

209 al taxonomic units (OTUs) falling within the Streptococcus, Prevotella, Acinetobacter, Treponema, and

210 Virulent strains of Streptococcus pyogenes (gram-positive group A Streptococ

211 tics is a global concern in the treatment of Streptococcus pyogenes (group A Streptococcus [GAS]) inf

212 Streptococcus pyogenes (group A Streptococcus [GAS]) is

213 Streptococcus pyogenes (group A Streptococcus [GAS]), a

214 ity of North-East Asian serotype M12 (emm12) Streptococcus pyogenes (group A Streptococcus, GAS) to c

215 arget and off-target activities of Cas9 from Streptococcus pyogenes (SpCas9) and the SpCas9 variants

216 he exogenous protein Cas9 from the bacterium Streptococcus pyogenes (SpCas9) in plasma samples by mea

217 off-target cleavage compared with wild-type Streptococcus pyogenes (SpCas9) in vivo.

218 acCas9 to its well-established ortholog from Streptococcus pyogenes (SpyCas9), and further engineer a

219 treptococcus pyogenes (gram-positive group A Streptococcus pyogenes [GAS]) recruit host single-chain

220 Here, we report that Streptococcus pyogenes also hijack lymphatic vessels to

221 mber of TCSs compared to the closely related Streptococcus pyogenes and Streptococcus pneumoniae, and

222 Herein, we report the development of the Streptococcus pyogenes anti-CRISPR/Cas9 protein, AcrIIA4

223 The targeting scope of Streptococcus pyogenes Cas9 (SpCas9) and its engineered

224 e this constraint, we engineered variants of Streptococcus pyogenes Cas9 (SpCas9) to eliminate the NG

225 udy by Walton et al. structurally engineered Streptococcus pyogenes Cas9 (SpCas9) to near-PAMless SpR

226 he most commonly used genome editing protein Streptococcus pyogenes Cas9 (SpyCas9), we used both self

227 cus iniae Cas9 (SinCas9) and weakly inhibits Streptococcus pyogenes Cas9 (SpyCas9).

228 Using the Streptococcus pyogenes CRISPR-Cas adaptation machinery,

229 occus aureus, Staphylococcus epidermidis and Streptococcus pyogenes in a soft-tissue wound biofilm mo

230 , and laboratory testing for confirmation of Streptococcus pyogenes infection is required to prevent

231 ded with an unexpected elevation in invasive Streptococcus pyogenes infections.

232 Streptococcus pyogenes is a human-restricted pathogen mo

233 CRISPR-Cas9 from Streptococcus pyogenes is an RNA-guided DNA endonuclease

234 Type II-A SpCas9 protein from Streptococcus pyogenes is the most investigated and high

235 Two near-identical clinical Streptococcus pyogenes isolates of emm subtype emm43.4 w

236 e first complete, closed genome sequences of Streptococcus pyogenes strains NCTC 8198(T) and CCUG 420

237 Recently, two related Streptococcus pyogenes strains with reduced susceptibili

238 ontaining Lancefield group A carbohydrate of Streptococcus pyogenes to study the effects of bacterial

239 To overcome these challenges, the pathogen Streptococcus pyogenes utilizes the protein Cpa, a pilus

240 Type II-A SpCas9 from Streptococcus pyogenes was the first Cas9 nuclease used

241 Invasive strains of Streptococcus pyogenes with significantly reduced suscep

242 9 are derived from Staphylococcus aureus and Streptococcus pyogenes(5,7).

243 Group A Streptococcus (GAS; Streptococcus pyogenes) is a bacterial pathogen for whic

244 ginosus group, Streptococcus pneumoniae, and Streptococcus pyogenes), positive percent agreement (PPA

245 nella enterica, Clostridioides difficile, or Streptococcus pyogenes, multiple highly conserved DNA MT

246 In the human pathogen Streptococcus pyogenes, production of secreted virulence

247 e measured affinities of Cas9 nucleases from Streptococcus pyogenes, Staphylococcus aureus, and Franc

248 Herein we report that in the human pathogen Streptococcus pyogenes, the adaptive response to Mn limi

249 cribes such a situation after infection with Streptococcus pyogenes.

250 reover, the baseline abundance of the genera Streptococcus, Ruminococcus_torques_group, Eubacterium_h

251 s spp, and 1 or more Bifidobacterium spp and Streptococcus salivarius subsp thermophilus, might produ

252 bacter species, Streptococcus parasanguinis, Streptococcus salivarius, and Veillonella species and in

253 nine residues produced by certain strains of Streptococcus salivarius, which almost exclusively resid

254 rom two naturally competent human pathogens, Streptococcus sanguinis (ComGC(SS)) and Streptococcus pn

255 re significantly improved, and the growth of Streptococcus sanguinis and Porphyromonas gingivalis was

256 cts spxB transcription and SpxB abundance in Streptococcus sanguinis and Streptococcus gordonii.

257 Streptococcus sanguinis is a primary colonizer of teeth

258 The SRR adhesin from Streptococcus sanguinis strain SK1 has tandem sialoglyca

259 Streptococcus sanguinis, an abundant and benign inhabita

260 entration test against Streptococcus mutans, Streptococcus sanguinis, and Streptococcus gordonii, the

261 hain reaction to detect Streptococcus mitis, Streptococcus sobrinus, Streptococcus mutans, Streptococ

262 Rothia mucilaginosa, Streptococcus sp., and Veillonella parvula were selected

263 hogens were Staphylococcus species (60%) and Streptococcus species (30%).

264 crease in relative abundance of Moraxella or Streptococcus species 3 weeks after day 1 of hospitaliza

265 Increased Streptococcus species abundance the summer after hospita

266 Enrichment of Moraxella or Streptococcus species after bronchiolitis hospitalizatio

267 ere we analyse >1300 genomes of 70 different Streptococcus species and identify nearly 800 prophages

268 ere more frequent in the CPVC group, whereas Streptococcus species were less frequent (14% vs 42%, P

269 le virus and Toxoplasma gondii) and one with Streptococcus species with other bacteria below the repo

270 previously identified in the genomes of two Streptococcus species, albeit with no confirmed evidence

271 m-sensing regulated transcription factors in Streptococcus species, controlling virulence, antimicrob

272 that a 10-fold increase in the abundance in Streptococcus spp. and Prevotella salivae was associated

273 that a 10-fold increase in the abundance in Streptococcus spp. and Prevotella salivae was associated

274 re, we show that certain bacteria, including Streptococcus spp. and Veillonella dispar, co-occur in m

275 s on bacteria such as Staphylococcus aureus, Streptococcus spp., Bacillus subtilis, and Mycobacterium

276 to form polymicrobial biofilms with various Streptococcus spp., including mitis and mutans group str

277 nsis, Listeria spp., Listeria monocytogenes, Streptococcus spp., Streptococcus agalactiae, Streptococ

278 in is the first sactipeptide identified from Streptococcus spp.; it contains two sequential four amin

279 obesity increased the risk of EOS by group B Streptococcus, Staphylococcus aureus, and Escherichia co

280 In the current study, we showed that oral Streptococcus strain SK95 and pneumococcal strain D39 bo

281 Thus, cps from oral Streptococcus strains can make acapsular pneumococcus vi

282 Group A streptococcus (Strep A) surface M protein, an alpha-heli

283 Streptococcus suis is part of the pig commensal microbio

284 Streptococcus suis serotype 2 is an important bacterial

285 NAD(+) homeostasis in the zoonotic pathogen Streptococcus suis.

286 T2(low); endotype C, virus(RSV/RV)microbiome(Streptococcus)T2(low); and endotype D, virus(RV-C)microb

287 tive genomes spanning at least 80 species of Streptococcus The resulting gene and allelic trees provi

288 discrimination between species of the genus Streptococcus, the most common genus in the mouth and im

289 ty of the closely related ComRS systems from Streptococcus thermophilus and Streptococcus vestibulari

290 Streptococcus thermophilus ASCC 1275 has two chain lengt

291 yoelectron microscopy (cryo-EM) structure of Streptococcus thermophilus Rgg3 bound to its quorum-sens

292 may be a long-suspected fratricidal agent of Streptococcus thermophilus.

293 ity, tests against Staphylococcus aureus and Streptococcus uberis samples are performed showing negli

294 ial taxa at the genus or species level, with Streptococcus, Veillonella, and Rothia species being mos

295 ter treatment with HA, whereas in stratum 2, Streptococcus, Veillonella, Rothia, and Granulicatella d

296 tion, a 10-fold increase in the abundance in Streptococcus vestibularis and Prevotella spp. was assoc

297 systems from Streptococcus thermophilus and Streptococcus vestibularis Despite high similarity, we s

298 ation of a schizophrenia-enriched bacterium, Streptococcus vestibularis, appear to induces deficits i

299 ing intensification 1, the patient developed Streptococcus viridans sepsis, which required 4 days in

300 A single zOTU (Streptococcus) was present in all samples (n = 925) of t

301 robic genera (Escherichia, Enterococcus, and Streptococcus), with multiple ciprofloxacin-resistance m