ライフサイエンスコーパス: TCM

1 TCM and TEM cells also required lymphoid tissue to mount

2 TCM exhibit greater plasticity and proliferative capacit

3 TCM is characterized by changes in cardiomyocyte and mit

4 TCM transformation experiments using strain CF revealed

5 TCM(Null) (TLR4(Null), CD14(Null), MD2(Null)), TLR4(Hi),

6 TCM-derived (m)C patterns are associated with reduced ex

7 TCMs enhanced IFN and LAM antiviral activities and impro

8 TCMs had a greater beneficial effect (P = 0.0003) than I

9 TCMs had a similar beneficial effect when compared with

10 The 2-TCM and the Logan analyses are accurate methods to estim

11 HABs than in MABs, and estimates from the 2-TCM and the Logan analyses were highly correlated.

12 The 2-TCM best described the regional kinetics of (18)F-DPA-71

13 tly correlated with that calculated by the 2-TCM.

14 newly methylated F1 Ler segment may act as a TCM source in a process comparable to paramutation in ma

15 ell clone generated in the skin, an abundant TCM cell clone bearing the identical TCR was present in

16 Among the active TCMs, we discovered that baicalein, a specific flavonoid

17 own by siRNA or shRNA impaired TGF-beta1 and TCM induction of alpha-SMA and calponin 1, but not of CO

18 L497Y-S750Q mutations abolished all CTZ and TCM actions without disrupting CX614 activity.

19 CTZ and TCM further slowed desensitization of L/Y mutant recepto

20 Results indicate that CTZ and TCM target deactivation and agonist potency independentl

21 Null), CD14(Null), MD2(Null)), TLR4(Hi), and TCM(Hi) cells and human bronchial epithelial cells with

22 ired for their homeostatic proliferation and TCM-mediated suppression of allograft rejection.

23 s CD62Lhigh central memory T cells (TCM) and TCM cells after L. monocytogenes infection, and both sub

24 ge about all the differences between TEM and TCM cells that may influence tumor treatment outcomes.

25 at distinguish them from circulating TEM and TCM cells.

26 ls upregulate Bcl-6 and co-initiate TFH- and TCM-like gene programs, including expression of the cyto

27 ment and reversal of HIV-1 latency in TN and TCM CD4(+) T cells and suggest that each subset should b

28 TN and TCM cells are distinct cell populations distinguished by

29 on of HIV-1 integration sites between TN and TCM cells that accounted for these observed differences.

30 s direct infection of highly purified TN and TCM cells to address differences in the establishment an

31 CD4+ TN and TCM cells were purified from the blood of 7 HIV-1-infect

32 mpetent virus was recovered from both TN and TCM cells.

33 uantified total HIV-1 DNA in the CD4+ TN and TCM cells.

34 to naive and central memory T cells (TN and TCM), hypoxia enhances the proliferation, viability, and

35 HIF1A mRNA in glycolytically inactive TN and TCM.

36 tivity compared with that observed in TN and TCM.

37 n be distinguished by their localization, as TCM home to secondary lymphoid organs and TEM circulate

38 n also required the presence of TGFbeta1, as TCM cells expressed TGFbeta1 while neutralizing TGFbeta1

39 of bismuth shields; and (f) with organ-based TCM and one bismuth shield.

40 Organ-based TCM provided superior image quality to that with bismuth

41 A combination of organ-based TCM with one bismuth shield reduced the dose by 47.0%.

42 h one bismuth shield, 30.4% with organ-based TCM, and 30.2% with a global reduction in tube current.

43 one bismuth eye shield; (c) with organ-based TCM; (d) with reduced tube current to yield the same dos

44 Because TCM are located within B cell follicles in the spleen wh

45 ciated HIV-1 RNA levels were similar between TCM and TN cells (15 135 vs 18 290 copies/mL, respective

46 sion profiling revealed similarities between TCM-exposed hMSCs and CAFs.

47 Blood TCM concentrations in mid to late pregnancy were associa

48 Blood TCM concentrations in the second trimester were associat

49 We found that maternal blood TCM concentrations were significantly higher for SGA com

50 Third-trimester blood TCM concentrations were also associated with an increase

51 totally 222 parameters integrated from both TCM practice and modern clinical tests.

52 , was determined from a fragment produced by TCM.

53 Both bystander TCM and naive T cells, but fewer TEM cells, migrated to

54 eover, the suppression mediated by bystander TCM cells was largely dependent on IL-15, as IL-15 was r

55 aive counterparts, suggesting that bystander TCM cells have an advantage over their naive counterpart

56 Thus, bystander TCM, but not TEM, CD8+ T cells are potent suppressors ra

57 l-CoA, is used to initiate tetracenomycin C (TCM C) biosynthesis.

58 trate the safety and feasibility of CD19 CAR TCM therapy after HSCT.

59 pathy or tachycardia-induced cardiomyopathy (TCM) has been known for decades as a reversible form of

60 In conclusion, among CD4 TCM cells in PB of aviremic patients on cART, pTfh cells

61 peripheral blood and contain a subset of CD4 TCM cells expressing chemokine receptor CXCR5 similar in

62 CD4(+) TCM cells of sooty mangabeys (SMs), a natural host for S

63 s and RMs and the association between CD4(+) TCM levels and the main virologic and immunologic marker

64 IV DNA increase postdepletion in both CD4(+) TCM and TEM in progressor RMs but decrease in the CD4(+)

65 increased percentages of circulating CD4(+) TCM cells, (ii) increased levels of CD4(+) T cells in th

66 h of time of SIV infection needed for CD4(+) TCM cells to fall to half of their initial levels is <16

67 ir lower susceptibility to infection, CD4(+) TCM cells of SIV-infected SMs are lost with kinetics 20

68 ART despite lower infection levels of CD4(+) TCM and TSCM cells than those seen in pathogenic SIV inf

69 n SIV-infected RMs, and the extent of CD4(+) TCM cell proliferation is associated positively with CD4

70 mechanistic link between the loss of CD4(+) TCM cells and disease progression.

71 tenance of the prohomeostatic role of CD4(+) TCM cells as features distinguishing nonprogressive from

72 lly and longitudinally, the levels of CD4(+) TCM cells in a large cohort of SMs and RMs and the assoc

73 ranslates into increased stability of CD4(+) TCM cells in natural versus nonnatural hosts has not yet

74 mportance of long-term maintenance of CD4(+) TCM homeostasis during HIV/SIV infection.

75 SMs translate into a better-preserved CD4(+) TCM compartment.

76 ermore, the fraction of proliferating CD4(+) TCM cells is significantly lower in SIV-infected SMs tha

77 ess susceptible to SIV infection than CD4(+) TCM cells of RMs.

78 or this cell loss, we also found that CD4(+) TCM cells increase their level of proliferation upon SIV

79 We found that the CD4(+) TCM compartment is significantly more stable in SIV-infe

80 n progressor RMs but decreased in the CD4(+) TCM of 4 out of 5 controllers.

81 in progressor RMs but decrease in the CD4(+) TCM of controllers.

82 istent with a model whereby intrahepatic CD8 TCM cells, being maintained by IL-15-mediated survival a

83 -lived, mainly owing to the inability of CD8 TCM cells to survive in the IL-15-deficient milieu.

84 CD122 (IL-15Rbeta), which suggests that CD8 TCM cells depend on IL-15 for maintenance.

85 and clonotypically diverse CD4(+) and CD8(+) TCM populations might potentially improve adaptive immun

86 s engineered from enriched CD4(+) and CD8(+) TCM subsets and expressing a second-generation CD19 CAR

87 in Lck constitutive activity between CD8(+) TCM and TEM are due to differential regulation by SH2 do

88 The CD4(+)/CD8(+) TCM-derived CD19 CAR T cells (NHL2) exhibited improvemen

89 ell products engineered from enriched CD8(+) TCM subsets, expressing a first-generation CD19 CAR cont

90 In vitro, CD8(+) TRM cells, but not CD8(+) TCM cells, demonstrated increased mitochondrial oxidativ

91 EM cell epitopes, but not with "SYMP" CD8(+) TCM cell epitopes, induced a strong CD8(+) T cell-depend

92 EM cell epitopes, but not with "SYMP" CD8(+) TCM cell epitopes, induced a strong protective HSV-speci

93 such as effector/memory (CD4(+)TEM and CD8(+)TCM) and regulatory (T reg) T cells.

94 ates central and effector memory CD8 T cell (TCM and TEM, respectively) homeostatic proliferation, ma

95 omising effect on the central memory T cell (TCM) population (both CD4(+) and CD8(+)) in adult and ol

96 as central and transitional memory T cells (TCM and TTM, respectively).

97 ifferentiated central memory CD8(+) T cells (TCM cells) (CD45RA(low) CCR7(high) CD44(low) CD62L(high)

98 onofunctional central memory CD8(+) T cells (TCM cells) (CD45RA(low) CCR7(high) CD44(low) CD62L(high)

99 as well as CD62Lhigh central memory T cells (TCM) and TCM cells after L. monocytogenes infection, and

100 apidly expand CD8(+) central memory T cells (TCM) during the acute phase of the primary response that

101 y T cells (T(EM)) to central memory T cells (TCM) following vaccination.

102 er, we show that central memory CD4 T cells (TCM) from HIV-infected individuals have heightened expre

103 t frequency of CD4 + central memory T cells (TCM) in patients who were subsequently Active versus Sta

104 autologous central memory-enriched T cells (TCM) transduced with lentivirus expressing CD19-specific

105 ), but not of central memory CD8(+) T cells (TCM), locally within TG, and improved protection against

106 nsidered to comprise central memory T cells (TCM), which are restricted to the secondary lymphoid tis

107 no-PET imaging human central memory T cells (TCM), which were transgenic for a myeloid peroxidase (MP

108 ompared with <20% in central memory T cells (TCM).

109 A in CD4(+) TSCM and central memory T cells (TCM-) did not significantly change.

110 y of CCR7+ L-selectin+ central memory cells (TCMs).

111 ry CD4(+) T cells, specifically the central (TCM) and transitional memory compartments, harbor the hi

112 chloroethene (TCE) 45 vol %, and chloroform (TCM) 10 vol %).

113 Blood SigmaTHM [sum of chloroform (TCM), bromodichloromethane (BDCM), dibromo-chloromethane

114 rkers of trihalomethanes (THMs): chloroform (TCM), bromodichloromethane, dibromochloromethane, and br

115 g methods on Liuwei Dihuang Wan, a classical TCM preparation in China.

116 belonged to the central-memory compartment (TCM).

117 In contrast, TCM cells, but not TEM cells, mounted a robust response

118 In contrast, TCM diapedesis did not require CXCL12 but was blocked by

119 CTZ, TCM, or L/Y mutation all essentially blocked GluR1 desen

120 sduced primary CD4(+)model, and the cultured TCM(central memory) CD4(+)model.

121 ntly more effective than nCPCs, aCPC-derived TCM, or nCPC-derived exosomes in recovering cardiac func

122 argeted by nCPC-derived TCM and aCPC-derived TCM, respectively.

123 r nCPC-derived TCM but none for aCPC-derived TCM.

124 derived TCM and 513 proteins in aCPC-derived TCM.

125 sion pattern of 804 proteins in nCPC-derived TCM and 513 proteins in aCPC-derived TCM.

126 were significantly targeted by nCPC-derived TCM and aCPC-derived TCM, respectively.

127 cting 8 identified pathways for nCPC-derived TCM but none for aCPC-derived TCM.

128 esent in S. glaucescens fermentations during TCM C production, suggesting that it could contribute to

129 This enzyme was shown to be present during TCM C production and could play a role in generating mal

130 RCTs comparing either TCM formulations alone or in combination with interferon

131 Engineered TCM-derived CD19 CAR T cells were infused 2 days after H

132 IV-dependent sRNAs are required to establish TCM events.

133 After extravasation, TCMs displayed agile movement within BM cavities, remain

134 e components from Indigo naturalis, a famous TCM herb that has been widely used for the treatment of

135 Quality evaluation for TCM preparations could be conducted based on chemical in

136 As negative training samples for TCM learning we used coding and intron sequences of plan

137 archical classification model was tested for TCM syndromes prediction based on totally 222 parameters

138 (BDCM, 0.62; DBCM, 0.53; TBM, 0.54) than for TCM (0.37).

139 A is not used directly as a starter unit for TCM C biosynthesis in vivo and argue against an involvem

140 s, the BM functions as a major reservoir for TCMs by providing specific recruitment signals that act

141 l ester, which is the biomarker derived from TCM of anthrose.

142 Notably, a novel PARP1 inhibitor from TCM has been identified from the natural products enrich

143 nstrate that specific plant metabolites from TCMs can directly interfere with key bacterial virulence

144 monocytes or TLR4 expressing cell lines (HEK-TCM) abrogates the respective inflammatory signal.

145 he use of a PA projection resulted in higher TCM values for chest CT (P < .001) owing to the higher a

146 ection localizer radiography owing to higher TCM values, whereas the organ doses from PA localizer ra

147 In TCM/TCdM the methylation state of one allele is altered

148 ts in increased constitutive Lck activity in TCM to levels similar to TEM, as well as increased cytot

149 fficiently characterize active components in TCM and their targets, which may bring a new light for a

150 l growth as efficiently as hMSCs cultured in TCM nor do they show increased SDF-1 expression.

151 as increased cytotoxic effector function in TCM Collectively, this work demonstrates a role for cons

152 HIV-1 DNA was significantly higher in TCM compared to TN cells (2179 vs 684 copies/106 cells,

153 However, essential ingredients in TCM herbs have not been fully identified, and their prec

154 of mitochondria was predominantly present in TCM.

155 and macrophages was significantly reduced in TCM.

156 Increased expression of redox regulators in TCM cells inversely correlated with the generation of re

157 o define the physical meaning of yin-yang in TCM by correlating it with biochemical processes.

158 roduced per infected TN cell as per infected TCM cell.

159 s to the lungs and lymph nodes by inhibiting TCM-induced lymphangiogenesis and angiogenesis in the pr

160 we investigated Sini Decoction, a well-known TCM consisting of three herbs, as a model.

161 Thus, antigen-reactive skin TRM and LN TCM cell clones were derived from a common naive T cell

162 kin simultaneously generates skin TRM and LN TCM cells in similar numbers from the same naive T cells

163 in both CD4(+) central memory T lymphocytes (TCM) and CD4(+) effector memory T lymphocytes (TEM) in p

164 y developed transductive confidence machine (TCM) techniques, we developed a new program TSSP-TCM for

165 model system [tumor-conditioned macrophages (TCM)].

166 Compared with normal macrophages, TCMs exhibited higher p53 levels, enhanced p53 binding t

167 es: (1) transparency change mechanochromism (TCM), (2) luminescent mechanochromism (LM), (3) colour a

168 ested the effect of tumor conditioned media (TCM) on gene expression in human mesenchymal stem cells

169 ence of either tumor cell conditioned media (TCM) or tumor cells.

170 negative MDA-MB-231 tumor-conditioned media (TCM) to determine the factors that may be secreted by va

171 MDA-MB-231 tumor conditioned media (TCM) was employed to accelerate spontaneous metastasis i

172 essful cases, Traditional Chinese Medicinal (TCM) formulae can achieve synergistic effects in therape

173 onal medicine, Traditional Chinese Medicine (TCM ), Ayurveda, naturopathy, chiropractic, osteopathy,

174 omarker panel, traditional Chinese medicine (TCM) drug intervention for validating the close relation

175 Traditional Chinese Medicine (TCM) has been developed for thousands of years and has f

176 Ancient traditional Chinese medicine (TCM) has effectively relied on the theory of yin-yang ba

177 Traditional Chinese medicine (TCM) practices have put forth Shenks as a promising trea

178 Traditional Chinese Medicine (TCM) preparations have been used in China for thousands

179 Traditional Chinese Medicine (TCM) treatment has been commonly used to treat Chronic H

180 ed efficacy of traditional Chinese medicine (TCM) treatment in Dutch children with asthma in areas wi

181 acteristics of traditional Chinese medicine (TCM) used to treat pediatric asthma, we conducted a nati

182 o characterize traditional Chinese medicine (TCM), as part of the "herbalome" project, with the rever

183 d treatment in traditional Chinese medicine (TCM), is a major indicator of the occurrence, developmen

184 sing method of Traditional Chinese Medicine (TCM), results in great changes in pharmacology and pharm

185 development of traditional Chinese medicine (TCM), we conducted a systematic review and meta-analysis

186 rein, we used traditional Chinese medicines (TCMs) as examples in a late-stage modification toolbox a

187 Traditional Chinese Medicines (TCMs) have been historically used to treat bacterial inf

188 enturies with traditional Chinese medicines (TCMs).

189 TGF-beta1 or tumour cell conditioned medium (TCM) elevated alpha-SMA, calponin 1 and collagen 1 A1 (C

190 hibitory effect of tumor-conditioned medium (TCM) on LPS-induced CCL5 expression.

191 (hMSCs) exposed to tumor-conditioned medium (TCM) over a prolonged period of time assume a CAF-like m

192 : effector memory (TEM ) and central memory (TCM ).

193 t overall generation of both central memory (TCM) and effector memory (TEM) CD8+ T cells was severely

194 mory (TEM), and longer-lived central memory (TCM) and stem cell memory (TSCM) CD8 T cells identified

195 evels of infection of CD4(+) central memory (TCM) and stem cell memory (TSCM) T cells.

196 Central memory (TCM) CD4 T cells are the major cellular reservoir for HI

197 ed the peripheral blood (PB) central memory (TCM) CD4(+) T cell subsets designated peripheral T folli

198 Central memory (TCM) CD4(+) T cells are the principal reservoir of laten

199 virus production from TN and central memory (TCM) CD4+ T cells isolated from HIV-1-infected individua

200 y cells, particularly in the central memory (TCM) cell subset.

201 affecting CCR7(+) naive and central memory (TCM) cells has the potential of treating TEM-mediated di

202 irst evidence that bystander central memory (TCM), but not effector memory (TEM), CD8+ T cells suppre

203 bited a resting phenotype of central memory (TCM), while peptide-specific CD8(+) T cells showed a mor

204 +)CD45RO(+)IL-4(+) producing central memory (TCM, CD45RO(+)CCR7(+)CD27(+); Fo = 1.1% versus 0.5%; p =

205 nctions in the presence of either metastatic TCM or metastatic tumor cells.

206 methylome are Trans Chromosomal Methylation (TCM) and Trans Chromosomal deMethylation (TCdM) in which

207 the processes trans-chromosomal methylation (TCM) and trans-chromosomal demethylation (TCdM).

208 roach for CLCA, the Technology Choice Model (TCM).

209 ors adapted from the temporal context model (TCM).

210 sing the 1- and 2-tissue-compartment models (TCMs) as well as the Logan analysis to estimate total vo

211 omography (CT) with tube current modulation (TCM).

212 tions at the CO2 Technology Center Mongstad (TCM), Norway.

213 conducted to evaluate differences in noise, TCM curves, and organ doses, respectively.

214 sers was significantly more than that of non-TCM users in school-age children.

215 Importantly, the survival of TEM, but not TCM, CD8+ cells was reduced without MCP-1, whereas the h

216 Due to the complexity of TCM, changes in metabolites should be investigated metab

217 retrospectively fulfilled common criteria of TCM, 79 patients had a diagnosis of DCM, and 91 had a di

218 L5 inhibition, and the inhibitory effects of TCM, PGE(2), and cAMP analog on LPS-induced CCL5 express

219 t a unique mechanism to secure engagement of TCM during an ongoing effector response.

220 tablished a method for quality evaluation of TCM preparations by combination of chemical ingredients

221 ells differentiated to higher frequencies of TCM at low doses of MP Rapa MPs.

222 improve the cytotoxic effector functions of TCM for adoptive cell therapy applications.

223 reover, the function to eliminate a graft of TCM, but not TEM, CD8+ cells was impaired without MCP-1.

224 ng that it could contribute to initiation of TCM C biosynthesis in vivo.

225 ssification based on a proper integration of TCM and modern clinical indexes was significantly higher

226 Nondirected intermolecular interactions of TCM with cellular material were ruled out as reason for

227 re the effectiveness, safety and legality of TCM preparations.

228 Interestingly, the migration of TCM, but not TEM, CD8+ cells to inflammatory sites was s

229 The number of TCM users was significantly more than that of non-TCM us

230 hat this results from a lower probability of TCM reaching threshold signaling owing to the decreased

231 Conversely, cytokine-driven proliferation of TCM and TSCM memory cells resulted in phenotypic convers

232 -1, whereas the homeostatic proliferation of TCM, but not TEM, CD8+ cells was weakened in MCP-1-/- mi

233 lishing the divergent effector properties of TCM and TEM.

234 ts of randomized controlled trials (RCTs) of TCM formulations reported in China in 1998-2008 for trea

235 ike EBV, includes a substantial reservoir of TCM CD4+ TH1 precursors, which continuously fuels TH1-po

236 ese pTfh cells, which constitute a subset of TCM CD4 T cells, can be readily monitored in peripheral

237 of deuterium label into the starter unit of TCM C.

238 tics and prescription patterns of the use of TCM in children with asthma.

239 e to mediate the constitutive recruitment of TCMs from the blood.

240 s with CHB, suggesting that further study of TCMs in the treatment of CHB is warranted, both in precl

241 epatitis B (CHB) in Asian countries based on TCM syndrome diagnosis, also called "ZHENG".

242 cturally related flavonoids present in other TCMs, such as quercetin, also inactivated the SPI-1 T3SS

243 cells with a central memory-like phenotype (TCM cells).

244 ze T cells toward central memory phenotypes (TCM), or to suppress immune function, depending on the c

245 The most commonly prescribed TCM formula is Ding-chuan-tang, or Xing-ren (Semen Armen

246 complement chemical ingredient in separating TCM preparation from different manufacturers and batches

247 ovar Typhimurium, we discovered that several TCMs can attenuate this key virulence pathway without af

248 Conclusion: Some TCMs seem effective as alternative remedies for patients

249 P doses, vaccines increased antigen-specific TCM, resulting in enhanced T cell expansion measured dur

250 mokine receptor analysis, all EBNA1-specific TCM CD4+ T cells were TH1 committed.

251 evelopmentally related central memory CD8 T (TCM) cells express elevated levels of CD122 (IL-15Rbeta)

252 Depletion of CD4(+) central memory T (TCM) cells dictates the tempo of progression to AIDS in

253 Central memory T (TCM) cells in lymph nodes (LNs) and resident memory T (T

254 effect on the survival of central memory T (TCM) cells in lymph nodes.

255 ing feature is that CD4(+) central memory T (TCM) cells in SIV-infected SMs are less infected than th

256 ollicular helper (TFH) and central memory T (TCM) cells.

257 IV-1 infected TN cells less efficiently than TCM cells.

258 EC-mediated tumor growth, we discovered that TCM-treated LEC ('tumor-educated LEC') secrete high amou

259 Given that TCM cells survive relatively longer in oxidative tumor m

260 onments, we investigated the hypothesis that TCM cells possess relatively greater antioxidative capac

261 The study results indicate that TCM treatment of children living in more polluted urban

262 Here, we report that TCM cells exhibit a relative increase compared with TEM

263 Intravital microscopy (IVM) showed that TCMs roll efficiently in BM microvessels via L-, P-, and

264 The TCM device can reversibly switch between transparent and

265 At At1g64790 the TCM- and TCdM-derived (m)C patterns are maintained in th

266 al, they produced as many virions as did the TCM cells (if not more virions).

267 Examining the TCM, we quantified changes in the expression pattern of

268 genes including Bcl6 and Cxcr5, but not the TCM-related genes Klf2 and Sell.

269 routinely believed to be unrelated with the TCM syndrome diagnosis.

270 he importance of latent infection within the TCM compartment and again focus attention on these cells

271 were generated by one-step thermochemolysis (TCM) at 140 degrees C in 5 min to provide specific bioma

272 Thus, TCM technique has produced a plant-oriented promoter pre

273 es in modern medicine could be beneficial to TCM syndrome diagnostics in an integrative way.

274 f HIV-1 infection is lower in TN compared to TCM cells, as much virus is produced from the TN populat

275 e, polarizing vaccine-induced T cells toward TCM is an intriguing strategy to enhance T cell expansio

276 larization of differentiating T cells toward TCM.

277 Adoptively transferred TCMs accumulated more efficiently in the BM than naive a

278 alobacter strains (UNSWDHB and CF) transform TCM to dichloromethane, with inconsistent carbon isotope

279 of cyclothiazide (CTZ), trichlormethiazide (TCM), and CX614 were compared at wild-type GluR1 and "no

280 Trichloromethane (TCM) is a frequently detected and persistent groundwater

281 techniques, we developed a new program TSSP-TCM for the prediction of plant promoters that also prov

282 Using TSSP-TCM program we annotated promoters in the whole Arabidop

283 7-fold for CD4(+) transitional memory [TTM], TCM, effector memory [TEM], and TSCM cells, respectively

284 We examined two loci that undergo TCM/TCdM in the Arabidopsis C24/Landsberg erecta (Ler) F

285 ed, and 57.95% (N = 26 585) of them had used TCM.

286 igated with Monte Carlo simulations by using TCM curves with fixed start angles (0 degrees , 90 degre

287 consistencies and differences among various TCM samples, which is helpful to ensure the effectivenes

288 ut fewer TEM cells, migrated to DLN, whereas TCM cells exhibited faster turnover than their naive cou

289 contact hypersensitivity responses, whereas TCM cells mediated delayed and attenuated responses.

290 Treatment of stromal cells with TCM from PC-3 cells transfected with GPC-1 shRNA increas

291 in TEM following TCR ligation compared with TCM Furthermore, we observed superior cytotoxic effector

292 toxic effector function in TEM compared with TCM, and we provide evidence that this results from a lo

293 Chest CT with TCM was performed after one localizer radiographic exami

294 strain CF were identical to experiments with TCM and Vitamin B(12) (epsilon(13)C(Vitamin B12) = -26.0

295 myocardial biopsy samples from patients with TCM and compared them with samples from patients with di

296 are warranted to characterize patients with TCM by endomyocardial biopsy more clearly.

297 significantly lower degree in patients with TCM compared with patients with DCM and ICM.

298 Patients with TCM, on the basis of clinical criteria, had stronger myo

299 d severe structural changes in patients with TCM.

300 ees ; for chest CT, a spiral trajectory with TCM was used.