ライフサイエンスコーパス: TF

1 TF and ectonucleotidase activities were measured using c

2 TF is also shown to induce signalling through activation

3 TFs that autoregulate were highly conserved between tiss

4 ssembled genome-wide binding profiles of 104 TFs in the K562 and GM12878 cell lines, which loosely mo

5 creased to 2428 structural profiles for 1900 TFs from 39 different species.

6 ork comprising 1380 interactions between 265 TFs and 108 cytokine gene promoters.

7 brary in three hPSC lines, we discovered 290 TFs, including 241 that were previously unreported, that

8 First, 33 TF genes up-regulated during poplar wood formation were

9 ssed transcription factor (TF) genes from 54 TF families, notably bHLH, MYB, ERF, MYB-related, NAC, a

10 me, a comprehensive library containing 1,564 TF genes and 1,732 TF splice isoforms.

11 library containing 1,564 TF genes and 1,732 TF splice isoforms.

12 mmon set of direct, functional targets for a TF.

13 f the Joubert syndrome protein TALPID3, is a TF-associated component.

14 nscription factor (TF) binding by mutating a TF binding motif, which in turn may affect the activity

15 oise (random fluctuations) in the level of a TF that is synthesized in stochastic bursts.

16 rnatively, TF-tagging techniques, in which a TF is fused to a DNA-modifying enzyme that marks TF-bind

17 7p, Gcr1p and Gcr2p, that act together as a 'TF collective', an important but poorly characterized mo

18 Chromatin accessibility, TF binding, and gene expression patterns in resting and

19 d identify hundreds of differentially active TFs, yet find very little transcriptomic changes in stea

20 ional cis-eQTL SNVs are more likely to alter TF binding sites than rare SNVs in the human population.

21 ession filtering identified commonly altered TFs and coregulator genes, including well-established (e

22 Alternatively, TF-tagging techniques, in which a TF is fused to a DNA-m

23 on factors (TFs) at baited enhancers amplify TF binding at target enhancers, a phenomenon we term cro

24 Membrane-anchored TF directly interacts with substrates and induces allost

25 ICAM (intercellular adhesion molecule)-1 and TF expression following TNF (tumor necrosis factor)-alph

26 evious therapeutics directly linking Dox and TF, cytotoxicity of CDT resulted from nuclear entry by D

27 and miRNA regulation pairs, their FFLs, and TF-miRNA mediated regulatory networks in two major types

28 on network features such as network size and TF-binding affinities.

29 ated the genomic tracks, inference tool, and TF-binding profile similarity clusters.

30 conserved between tissues, but less so, and TFs regulate different combinations of microRNAs in a ti

31 allele did not show any interaction with any TFs.

32 Two significant sources of evidence are TF binding locations and the transcriptional responses t

33 These approaches assign TFs and cofactors to genes via a binary designation of '

34 of scRNA-Seq data with the ability to assign TFs to specific activation points in the model.

35 ization operates as an antenna that attracts TF molecules in a promiscuous exchange among myriads of

36 naive TF target genes are more likely to be TFs themselves than those of formative TFs, suggesting d

37 ffect the co-regulating associations between TFs (i.e. "rewiring the co-regulator network").

38 /helix-loop-helix transcription factor (bHLH TF), MdbHLH3.

39 vent E2A from forming heterodimers with bHLH TFs or from forming homodimers.

40 nal regulatory module (TRM) (e.g. co-binding TFs), which helps decipher the combinatorial regulatory

41 al assays to identify and isolate biosensing TFs, and a quantum-dot Forster Resonance Energy Transfer

42 binding affinity and stability of the bound TF.

43 Prior studies have assumed that decoy-bound TFs are protected from degradation, and in this case dec

44 Interestingly, while protection of bound TFs from degradation slows the time-scale of fluctuation

45 in the unbound TF levels, the decay of bound TFs leads to faster fluctuations and smaller noise propa

46 etable as the "linking rate," by which bound TFs interact with Mediator components, enables our model

47 ing ligule development, the E class MADS-box TF GRCD5 activates GhCYC3 expression.

48 plore the mechanism of activation of PTEN by TF, the association of "membrane-associated guanylate ki

49 These two regions-the central TF-binding footprint and the eRNA initiation cores-defin

50 e-learning algorithm able to jointly combine TFs ChIP-Sequencing data and gene expression compendia t

51 nference greatly expands the high-confidence TF network map in both yeast and human.

52 The interacted and cooperated TFs can potentially form a transcriptional regulatory mo

53 ene-expression program; the ensemble of core TFs and their regulatory loops constitutes core transcri

54 These core TFs form interconnected feed-forward transcriptional loo

55 Next, we determined significantly correlated TF-gene/miRNA and miRNA-gene/TF pairs with regulation di

56 n-based GRNs to accurately predict covarying TF-target relationships in natural variation data sets,

57 Downstream, CRC TFs coordinately regulate gene expression networks in Ew

58 Furthermore, we identify Sox17 as a critical TF upregulated during the sprouting and remodeling of co

59 We reveal >1300 CRM TF-binding variants associated with target gene expressi

60 ratively estimates model parameters of CRMs, TF activities, and corresponding regulation on gene tran

61 tinuous-State Hidden Markov Models TF (CSHMM-TF) method which integrates probabilistic modeling of sc

62 t effects on TF binding sites from different TF families on a cancer-type basis.

63 method to directly construct a differential TF co-regulation network from paired disease-to-normal C

64 ased mixture model for monomeric and dimeric TF-binding motifs and an expectation maximization algori

65 and the transcriptional responses to direct TF perturbations.

66 its autoregulation and a robust, downstream TF cascade involving PPARG.

67 ntly, we determined 288 and 664 dysregulated TF-miRNA-gene FFLs in SE and NSE, respectively.

68 These models allow us to characterize each TF's specificity, activity and interactions with chromat

69 It has been proposed that each TF interacts with a dedicated MED subunit to induce spec

70 We find that at least eight TFs bind the IME1 promoter when nutrients are ample.

71 Unexpectedly, nuclear expression of the EMT-TF ZEB2 in human primary melanoma has been shown to corr

72 p.Ser117HisfsTer10) in F3, the gene encoding TF, causing premature termination of TF (TFshort) in a w

73 nregulate or silence the fibroblast-enriched TFs.

74 to upregulate or activate the iPSC-enriched TFs while downregulate or silence the fibroblast-enriche

75 from mRNA, and SRTs deposited by exogenous, TF-transposase fusions can be used to map TFBS.

76 reased plasma levels of NETs, tissue factor (TF) activity, and sC5b-9 were detected in patients.

77 emonstrate that inhibition of tissue factor (TF) and the downstream coagulation proteases factor Xa a

78 Tissue factor (TF) is the primary initiator of blood coagulation in viv

79 Tissue factor (TF) signalling has been associated with alterations in A

80 rough direct interaction with tissue factor (TF).

81 f membrane proteins, CorA and tissue factor (TF).

82 rd binding the MB cell transcription factor (TF) BACH2 at the expense of the germinal center (GC) TF

83 nce variants affecting transcription factor (TF) binding at CRMs have a strong potential to influence

84 ory elements can alter transcription factor (TF) binding by mutating a TF binding motif, which in tur

85 c distances over which transcription factor (TF) binding influences gene expression is important for

86 ichment at the central transcription factor (TF) binding regions and at the flanking eRNA initiation

87 Genome-wide transcription factor (TF) binding signal analyses reveal co-localization of TF

88 constructed miRNA and transcription factor (TF) co-regulation networks for both humans and mice.

89 ing protein, mRNA, and transcription factor (TF) DNA-binding activity for mouse liver tissues collect

90 the lineage-specifying transcription factor (TF) Foxp3.

91 fferentially expressed transcription factor (TF) genes from 54 TF families, notably bHLH, MYB, ERF, M

92 hrough a separation of transcription factor (TF) inputs at the individual enhancers.

93 matin accessibility at transcription factor (TF) motifs in the context of Sox9(EGFP) populations, we

94 ferentially accessible transcription factor (TF) motifs were identified.

95 th the prohypertrophic transcription factor (TF) myocyte enhancer factor-2 (MEF2).

96 ER membrane-associated transcription factor (TF) released by self-cleavage.

97 tional targets of each transcription factor (TF) requires convergent evidence from independent source

98 ile the reported short transcription factor (TF) residence times on the DNA with the high specificity

99 T cells 5 (NFAT5) is a transcription factor (TF) that mediates protection from adverse effects of hyp

100 elix-loop-helix (bHLH) transcription factor (TF), regulates root Fe acquisition genes.

101 r-binding protein beta transcription factor (TF), while the T allele did not show any interaction wit

102 ulatory sequences and transcription factors (TF) motifs.

103 se types of elements, transcription factors (TF) were identified whose binding sites were enriched or

104 g data on the role of transcription factors (TFs) - particularly the ETS and IRF families - in regula

105 , 1554 genes encoding transcription factors (TFs) and a total of 514 single nucleotide polymorphisms

106 rately predicting the transcription factors (TFs) and cofactors that drive transcriptomic differences

107 on highly researched transcription factors (TFs) and cytokines, resulting in an incomplete portrait

108 w aim to identify key transcription factors (TFs) and functional enhancers that regulate processes as

109 interactions between transcription factors (TFs) and genome-wide targets typically elude biochemical

110 wo regulators such as transcription factors (TFs) and microRNAs (miRNAs).

111 ace between DNA-bound transcription factors (TFs) and the RNA Pol II machinery.

112 eractions between the transcription factors (TFs) and the target genes.

113 mplex crosstalk among transcription factors (TFs) and their target genes, with a method able to handl

114 ize information about transcription factors (TFs) and their targets or only study these as a post-pro

115 subfamily of homeobox transcription factors (TFs) are involved in abiotic stress responses and plant

116 ied the VOZ1 and VOZ2 transcription factors (TFs) as IBI1-interacting partners, which are transcripti

117 Transcription factors (TFs) at baited enhancers amplify TF binding at target en

118 OSINTE BRANCHED1-like transcription factors (TFs) belonging to the TEOSINTE BRANCHED1/CYCLOIDEA/PROLI

119 anscription, numerous transcription factors (TFs) bind to targets in a highly coordinated manner to c

120 Transcription factors (TFs) define identities of both the starting fibroblasts

121 recent collections of transcription factors (TFs) from the JASPAR and UniPROBE databases, methylated

122 or a diverse group of transcription factors (TFs) in many different types of human cells.

123 1 and cDC2, depend on transcription factors (TFs) IRF8 and IRF4, respectively.

124 gnaling effectors and transcription factors (TFs) on enhancers.

125 d Stp2 are paralogous transcription factors (TFs) regulated by the Ssy1-Ptr3-Ssy5 (SPS) amino acid se

126 y, we reveal that the transcription factors (TFs) regulating cell identity contain prion-like domains

127 ple sequence-specific transcription factors (TFs) that mediate association of Tup1-Cyc8 co-repressor

128 SP activates a set of transcription factors (TFs) that orchestrate nodule organogenesis and infection

129 networks (GRNs) link transcription factors (TFs) to their target genes and represent maps of potenti

130 sed genes that encode transcription factors (TFs) were analysed.

131 d various families of transcription factors (TFs) were differentially expressed in root system during

132 landscape mediated by transcription factors (TFs), we present the Human TFome, a comprehensive librar

133 ue family of bacteria transcription factors (TFs), which activate transcription in a manner distinct

134 regulatory regions to transcription factors (TFs).

135 in 1 (AP-1) family of transcription factors (TFs).

136 in the abundances of transcription factors (TFs).

137 constituted by three transcription factors (TFs): KLF15, TCF4 and NKX2-2, in Ewing sarcoma cells.

138 Here, we explored transcriptional factors (TFs) that reduce wood recalcitrance and improve sacchari

139 The primary outcome was treatment failure (TF), defined as 30-day mortality or persistent bacteremi

140 Transition fibers (TFs) regulate cilia gating and make the primary cilium a

141 e S. salsa (S, habitat III), and tidal flat (TF, habitat IV) across a salinity gradient.

142 phenols (TSP), tannins (TT) and flavonoids (TF) were determined.

143 ave been proposed, ranging from the flexible TF billboard model to the stringent enhanceosome model.

144 ll levels (p < 0.001), except DM and TAD for TF (p = 0.622).

145 for TSF, but was lower than MDM and TAD for TF.

146 We also discovered that formative TF target genes are marked by permissive epigenomic sign

147 to be TFs themselves than those of formative TFs, suggesting denser hierarchies among naive TFs.

148 Vs that bear primary or transmitted/founder (TF) Envs with modifications at Env residue 375 that enab

149 ulate key properties of transmitted/founder (TF) or primary HIV-1 isolates, such as CCR5 tropism, tie

150 that the combination of low tumor fraction (TF) and limited number of DNA fragments restricts low-di

151 silico study, we simulate single cells from TF/pathway perturbation bulk RNA-seq experiments.

152 is important for inferring target genes from TF chromatin immunoprecipitation followed by sequencing

153 It is thus not clear if functional TF and pathway analysis tools established for bulk seque

154 e relevant signaling pathways and functional TFs (transcription factors) regulating these processes i

155 ings identify Med19/Med1 as a composite GATA TF interface and suggest that binary MED subunit-TF part

156 hysically interacts with the Drosophila GATA TFs, Pnr and Serpent (Srp), in vivo and in vitro through

157 ediator subunit serves as a cofactor of GATA TFs in Drosophila, as shown in mammals.

158 2 at the expense of the germinal center (GC) TF BCL6, leading to pre-memory transcriptional reprogram

159 ntly correlated TF-gene/miRNA and miRNA-gene/TF pairs with regulation direction.

160 with COVID-19 platelet-rich plasma generated TF-bearing NETs that induced thrombotic activity of HAEC

161 Here, we test two recently generated TF SHIVs, SHIV.D.191859 and SHIV.C.CH848, which were des

162 Despite a common understanding that Gli TFs are utilized to convey a Hh morphogen gradient, gene

163 ABC-treated cells and EMP had greater TF activity, while tenofovir disoproxil fumarate- and te

164 entify the components of a known heterotypic TF cluster.

165 iding quantitative evidence that heterotypic TF interactions distinguish cell type-specific CRE activ

166 s resident in communities identified as high-TF clusters during previous population-based mapping.

167 mpositions and configurations of homeodomain TF complexes.

168 he genomic binding profiles of different Hox TFs, even among the posterior group paralogs that share

169 the differential abilities of posterior Hox TFs to bind to previously inaccessible chromatin drive p

170 We aimed to address how similar Hox TFs diverge to induce different positional identities.

171 imilar DNA-binding motifs of the various HOX TFs contrast with the wide-range of HOX-dependent geneti

172 changes rather than quantitative changes in TF gene expression are more likely associated with chang

173 eration of activated protein C and increases TF, augmented FX activation.

174 This report explores the initial TF responses to OSKM as the molecular underpinnings for

175 ic TF profiles and recording and integrating TF-binding events across time.

176 In this study, we extensively investigated TF and miRNA regulation pairs, their FFLs, and TF-miRNA

177 To address how interactions between four key TFs contribute to cis-regulation in mouse ESCs, we assay

178 aring the gains and losses of targets of key TFs across cell states is often not informative.

179 the subnetworks regulated by the B-lymphoid TF Ebf1 and T-lymphoid TF Gata3 revealed a surprising ro

180 ted by the B-lymphoid TF Ebf1 and T-lymphoid TF Gata3 revealed a surprising role in common activation

181 s fused to a DNA-modifying enzyme that marks TF-binding events across the genome as they occur, do no

182 se binary partnerships sufficient to mediate TF functions?

183 We analyse ChIP-seq data from MEIS, TFs which are broadly expressed across mouse branchial a

184 ), target genes and regulatory motifs (miRNA-TF-gene) related to CP.

185 ed the Continuous-State Hidden Markov Models TF (CSHMM-TF) method which integrates probabilistic mode

186 Platelet activation and monocyte TF expression were associated with markers of coagulatio

187 t activation and platelet-dependent monocyte TF expression, which were associated with COVID-19 sever

188 Moreover, TFs associated with murine EGA are not enriched in cattl

189 ng are enhanced in nucleosomes with multiple TF binding sites.

190 the combinatorial binding events of multiple TFs.

191 Moreover, Myt(MYT)-TF levels were upregulated in mouse and human beta cells

192 Our analyses revealed that naive TF target genes are more likely to be TFs themselves tha

193 s, suggesting denser hierarchies among naive TFs.

194 TFs, including closure of neurodevelopmental TF binding sites and increased expression and binding ca

195 Without NFI TFs, SCs lose their hair-regenerating capability, and pr

196 515 experienced the best global outcomes (no TF and no AKI).

197 Specifically, we used TFs, miRNAs and non-TF genes to form feed-forward loops (FFLs) using genomic

198 ions through increasing the accessibility of TF motifs NR1H4 and OLIG (OLIGI and OLIG2), respectively

199 e study identified unique characteristics of TF viruses thus prompting further investigation into vir

200 lationship between the gain or disruption of TF (transcription factor)-binding motifs, inferred from

201 o recapitulated by the soluble ectodomain of TF (sTF).

202 Alterations in groups of TF-binding profiles (i.e. "co-binding changes") can affe

203 We established a temporal hierarchy of TF binding to gene promoters within the same family as w

204 Thus, identification of TF binding sites (TFBSs) is key to understanding gene ex

205 udy is the first systematic investigation of TF and miRNA regulation and their co-regulation in two m

206 ng signal analyses reveal co-localization of TF binding sites based on inferred cis-regulatory module

207 important but poorly characterized model of TF cooperativity.

208 mic techniques have uncovered a multitude of TF binding sites in ESCs, yet a major challenge lies in

209 or unique applications, such as recording of TF occupancy over time and cell type specificity through

210 chastic model demonstrated the separation of TF inputs is sufficient to explain these findings.

211 atile approach will enable unique studies of TF-mediated gene regulation in live animal models.

212 ncoding TF, causing premature termination of TF (TFshort) in a woman with unexplained bleeding.

213 ynamics emerging from nonspecific binding of TFs and highlights the dual role of decoys as attenuator

214 to two cell lines with a rich collection of TFs and identify combinatorial binding patterns that sho

215 y Fisher Exact Tests to assess enrichment of TFs and cofactors.

216 mily as well as across different families of TFs.

217 ize the binding and expression of a group of TFs, Tye7p, Gcr1p and Gcr2p, that act together as a 'TF

218 Homodimerization or heterodimerization of TFs are required for DNA binding and the association int

219 provide a resource for the identification of TFs which regulate the dynamical properties of human gen

220 We generated unique lists of TFs (n = 2662), coactivators (COA; n = 766); corepressor

221 r knowledge about the protein copy number of TFs in the nucleus is limited.

222 iew is focused on the ever-growing number of TFs whose effects on ageing are evolutionarily conserved

223 that in the genome the overall occupancy of TFs is the primary determinant of activity.

224 data to identify target genes of a panel of TFs.

225 tial legitimate transcriptional responses of TFs to OSKM reprogramming were also observed in the repr

226 n we term cross-interaction stabilization of TFs.

227 el and infers context-specific CRMs based on TF-gene binding events and gene expression data for a pa

228 somatic mutations have different effects on TF binding sites from different TF families on a cancer-

229 es the effect of MYC, a well-known oncogenic TF.

230 Multiple white-opaque TFs can co-assemble into complex condensates as observed

231 the presence of DNA binding motifs for other TFs, including TCF1.

232 isease-associated repeat expansions in other TFs (HOXA13, RUNX2, and TBP) were similarly found to alt

233 ing site accessibility change for many other TFs, including closure of neurodevelopmental TF binding

234 man cells, including interactions with other TFs, and propose that Bhlhe40 is a central mediator of b

235 o the BHJ film top surface, changes the PBDB-TF/Y6 vertical phase separation and intermixing, and red

236 trate how a single sequence-specific pioneer TF can synergize with remodelers to activate sets of gen

237 Specialized pioneer TFs bind nucleosome-wrapped DNA to perform this chromati

238 e developed a footprinting method to predict TF footprints in active chromatin elements (TRACE) to im

239 h-dependent SBF/MBF clustering and predicted TF dynamics that were confirmed in live cell PALM experi

240 ll lines were exposed to recombinant-TF (rec-TF), or activated using a PAR2-agonist peptide and the p

241 In contrast, continuous exposure to rec-TF up to 14 days, resulted in lower PTEN antigen levels,

242 Seven cell lines were exposed to recombinant-TF (rec-TF), or activated using a PAR2-agonist peptide a

243 n and binding capacity of activity-regulated TFs.

244 tterns that show well-known TRMs and related TF co-binding events.

245 activity of other major chromatin remodeling TFs such as TCF1.

246 d to the live mouse brain and used to report TF occupancy.

247 pression signature based on over-represented TFs correlated with poor survival and unfavorable progno

248 oss the genome as they occur, do not require TF-specific antibodies and offer the potential for uniqu

249 Colocalization of NRL and the retinal TF CRX correlated with rod-specific ncRNA expression, wh

250 RFX TFs play key functions in the control of ciliogenesis in

251 We demonstrated that Sox17 TF regulates the transcriptional activation of Nestin's

252 ng direct association between these specific TF and local histone acetylation in human hearts.

253 show utility in defining cell type-specific TF profiles and recording and integrating TF-binding eve

254 quired to maintain expression of MN-specific TFs.

255 for the endodermal and pulmonary specifying TF GATA6 in lung adenocarcinoma (LUAD) progression.

256 ognizes partial binding sites display stable TF-DNA interactions similar to those observed in complex

257 nterface and suggest that binary MED subunit-TF partnerships are probably oversimplified models.

258 We also extensively survey TF-RBP crosstalk, highlighting how SUB1, a previously un

259 Applied to gene sets derived from targeted TF perturbation experiments, Lisa boosted the performanc

260 stablished the proof of concept of targeting TF as a new target in CAR-NK immunotherapy for effective

261 argets captured in root cells using temporal TF perturbation account for 50% of NLP7-regulated genes

262 ightly better than PPM models for 314 tested TFs (or their DNA-binding domains) from four families (b

263 T viruses produced more viral particles than TF viruses.

264 We highlight the importance of the TF HIV-1 phenotype and the role of different DC subsets

265 uch difference in replicative fitness of the TF viruses in cultures treated with and without IFN-alph

266 erve as bright fluorescent indicators of the TF-DNA bound (on bead) and unbound states.

267 ficity through conditional expression of the TF-enzyme fusion.

268 and experimental evidence for a role of the TFs NURR1 and ERR1 in modulating the expression pattern

269 For these TFs, a gene expression signature was built to assess the

270 ally, we analyzed expression levels of these TFs in dorsolateral prefrontal cortex (DLPFC) of SCZ pat

271 ne-sensing bacterial aTF and to develop this TF into an optical sensor for progesterone.

272 anges in terms of the topic changes for this TF.

273 We show that these three TFs have both redundant and specific functions in ependy

274 ishes super-enhancers of each of these three TFs to activate their transcription.

275 ments suggest that the nucleosome barrier to TF binding is modulated by dynamic nucleosome unwrapping

276 However, prolonged exposure to TF results in the reduction in PTEN antigen with concurr

277 Nucleosomes are refractory to TF binding, and often must be removed from regulatory re

278 permissive and dynamic chromatin relative to TF usage.

279 fically impairs the recruitment of DYF-19 to TFs.

280 e in coordinating the recruitment of FBF1 to TFs.

281 compare three methods for determining total (TF) and total soluble fluorides (TSF) in 5 child formula

282 The transient TF-target interactions captured uncover the early mode-o

283 ide evidence supporting the existence of two TF-mediated allosteric activation pathways.

284 ary degradation rates for both bound/unbound TF states, we find rich noise behaviors.

285 finity decoys, noise in the level of unbound TF always monotonically decreases to the Poisson limit w

286 he time-scale of fluctuations in the unbound TF levels, the decay of bound TFs leads to faster fluctu

287 gs overexpressing previously uncharacterized TF genes increased total glucan hydrolysis on average co

288 Specifically, we used TFs, miRNAs and non-TF genes to form feed-forward loops

289 Furthermore, we show that VOZ TFs require nuclear localization for their contribution

290 ded catalogs of putative open regions, where TFs can recognize their motifs and regulate gene express

291 Yet, how the DNA sequence determines which TF complex forms at any given site is poorly understood.

292 rrent study was undertaken to identify which TFs and coregulators are commonly altered in PCa.

293 ficient mixing and spatial separation, while TF-labeled semiconductor quantum dots serve as bright fl

294 AP1-IRF composite elements (AICEs) and with TF PU.1 at Ets-IRF composite elements (EICEs), it is unc

295 and lineage-specific motifs associated with TF binding therein.

296 , proliferation and invasion associated with TF during chronic diseases.

297 Since IRF8 and IRF4 can each interact with TF BATF3 at AP1-IRF composite elements (AICEs) and with

298 tations observed in solid malignancies, with TF detection sensitivity as low as 10(-5).

299 resolved 'trans-omic' datasets together with TF binding profiles and chromatin conformation data to i

300 haliana zinc finger transcription factor (ZF-TF), S-nitrosothiol (SNO) Regulated 1 (SRG1), is a centr