ライフサイエンスコーパス: TGN

1 TGN exit was unaffected by Rab6 depletion.

2 The median 6-TGN and TPMT levels were 953 pmol/8 x 10(5) RBC (IQR 145

3 mbined presence of an ER export signal and a TGN retention motif.

4 confirmed the role of (199)KxGxYR(204) as a TGN retention signal by using chimeras between MERS-CoV

5 Overall, our study reveals a role for a TGN golgin and ITSN-1 in linking to the actin cytoskelet

6 GN and post-TGN cargo without the need for a TGN marker that universally cosegregates with all cargo.

7 TO OLIGOGALACTURONIDE2 (AtTRAPPC11/ROG2), a TGN/EE-associated, evolutionarily conserved TRAPP protei

8 Most (96%) patients underwent a TGN.

9 lates TGN integrity, as evidenced by altered TGN/EE association of several residents, including SYNTA

10 are detected in the trans-most cisternae and TGN compartments.

11 GBF1 modulates both ER and TGN exit, the latter dramatically affecting the size of

12 id transfer proteins possessing dual (ER and TGN) targeting motifs that have been hypothesized to con

13 argeting mechanism of PI4KB to the Golgi and TGN membranes is unknown.

14 -Golgi network (TGN) to endosome traffic and TGN homotypic fusion.

15 plex mediate the transport of MIG-14/Wls and TGN-38/TGN38 cargo proteins from the recycling endosome

16 ing into AP1-coated vesicles for anterograde TGN-plasma membrane transport.

17 d composition to functional polar sorting at TGN subdomains remain unknown.

18 Thus, pH biosensing by TGN PI4P allows for direct metabolic regulation of prote

19 pH biosensing by TGN PI4P in response to nutrient availability governs pr

20 Furthermore, we find that retrograde cargo TGN-38 is trapped in early endosomes after depletion of

21 s, there was no association between red cell TGN levels and taking 6-MP with food versus without (206

22 LVs produced from TGN compartments (TGN-LVs) stained lighter than LVs and contained the cell

23 In addition, AtPep1 and different TGN/EE markers colocalized only rarely, implying that th

24 Are lipids also sorted into distinct TGN-derived carriers?

25 s to early endosomes/trans-Golgi network (EE/TGN) and is constitutively endocytosed through a monoubi

26 dynamic trans-Golgi network/early endosome (TGN/EE) facilitates cargo sorting and trafficking and pl

27 ther the trans-Golgi network/early endosome (TGN/EE)-localized vacuolar H(+)-ATPase activity nor the

28 GLUT4 from a perinuclear recycling endosome/TGN compartment.

29 at the trans-Golgi network/early endosomes (TGN/EE) for vacuolar targeting.

30 ER-TGN contact sites (ERTGoCS) have been visualized by elec

31 Monthly erythrocyte TGN levels (pmol/8 x 108 erythrocytes) were measured ove

32 dosome/prevacuolar compartment (PVC) and for TGN homotypic fusion.

33 LVs produced from TGN compartments (TGN-LVs) stained lighter than LVs and

34 tion, silencing retromer or disrupting Golgi/TGN organization all impair efficient TSH-dependent cAMP

35 inhibiting PKA II/interfering with its Golgi/TGN localization, silencing retromer or disrupting Golgi

36 F1 [GARG]) targets like GBF1, supports Golgi/TGN architecture, and facilitates secretion.

37 vates all ARFs (including ARF6) at the Golgi/TGN and recruits additional ARF effectors to the Golgi/T

38 protein kinase A (PKA) response at the Golgi/TGN.

39 cruits additional ARF effectors to the Golgi/TGN.

40 c reticulum and, after passage through Golgi/TGN to the cell division plane, transformed into fusogen

41 findings support the notion that HCV hijacks TGN-endosome trafficking to facilitate particle assembly

42 HID-1 KO cells also exhibit defects in TGN acidification together with mislocalization of the G

43 s a link to a more general role of exomer in TGN organization.

44 urrently known molecular players involved in TGN transport, three different adaptor protein (AP) comp

45 minant negative (DN) Rab GTPases involved in TGN-endosome trafficking steps.

46 herers, high intra-individual variability in TGN levels contributed to increased relapse risk (hazard

47 in clearance using the novel AQP4 inhibitor, TGN-020.

48 g of phosphorylation translocates Cab45 into TGN-derived vesicles, which goes along with an increased

49 ns at the donor endosome membrane to mediate TGN trafficking.

50 etermine their specific localization, namely TGN and ERGIC/cis-Golgi for MERS-M and IBV-M, respective

51 role of surgical transgastric necrosectomy (TGN) for walled-off pancreatic necrosis (WON) in selecte

52 fic retrogradely to the trans-Golgi network (TGN) and activate endogenous Gs-proteins in the retromer

53 ompartmentalized to the Trans-Golgi Network (TGN) and also to milk.

54 in pathways between the trans-Golgi network (TGN) and endosomes.

55 inent enrichment in the trans-Golgi network (TGN) and post-Golgi vesicles.

56 FR/RTK anchoring on the trans-Golgi network (TGN) and recycling back to the plasma membrane, leading

57 ves sequentially to the trans-Golgi network (TGN) and recycling endosomes before nuclear translocatio

58 ograde transport to the trans-Golgi network (TGN) and recycling to the plasma membrane.

59 trafficking toward the trans-Golgi network (TGN) and the Golgi apparatus correlates with transductio

60 KOR1 cycles between the trans-Golgi network (TGN) and the plasma membrane (PM).

61 7B was localized to the trans-Golgi network (TGN) and the plasma membrane of the soma and dendrites b

62 somal hydrolases at the trans-Golgi network (TGN) are well understood.

63 ATP7B targeting to the trans-Golgi network (TGN) but reduce its Cu-transport activity.

64 rade transport from the trans-Golgi network (TGN) by facilitating localized actin assembly at the TGN

65 sicles that emerge from trans-Golgi network (TGN) compartments and regulates polarized membrane traff

66 the trans cisternae and trans-Golgi network (TGN) compartments.

67 of ARF1 and BIG4 at the trans-Golgi network (TGN) depends on ECHIDNA (ECH), a plant homolog of yeast

68 sport carriers with the trans-Golgi network (TGN) depends on the concerted action of two types of tet

69 ightly regulated at the trans-Golgi network (TGN) during constitutive secretion.

70 a complex to reach the trans-Golgi network (TGN) for subsequent ciliary targeting.

71 , suggesting a role for trans-Golgi network (TGN) functions and retrograde transport in HSV entry.

72 brane proteins from the trans-Golgi network (TGN) has been shown to occur through tubular carriers th

73 ses at the level of the trans-Golgi network (TGN) has remained elusive.

74 V16 pseudogenome in the trans-Golgi network (TGN) in Pyk2-depleted cells, suggesting that the kinase

75 early endosomes and the trans-Golgi network (TGN) in unstimulated human colonic epithelial cells.

76 post-Golgi compartment trans-Golgi Network (TGN) is a central hub divided into multiple subdomains h

77 The plant trans-Golgi network (TGN) is a central trafficking hub where secretory, vacuo

78 The trans-Golgi network (TGN) is a major site for sorting of cargo to either the

79 c vesicles at the yeast trans-Golgi network (TGN) is believed to be mediated by their coalescence wit

80 ate (PI4P) in the yeast trans-Golgi network (TGN) is dependent on intracellular pH, indicating PI4P i

81 egrity of the Golgi and trans-Golgi network (TGN) is disrupted by brefeldin A (BFA), which inhibits t

82 ncipal functions of the trans Golgi network (TGN) is the sorting of proteins into distinct vesicular

83 at colocalizes with the trans-Golgi network (TGN) marker TGN46 in KSHV-infected PEL cells.

84 sly, we showed that the trans-Golgi network (TGN) membrane tether/golgin, GCC88, modulates the Golgi

85 sport carriers from the trans-Golgi network (TGN) must coordinate specification of lipid composition

86 alpha-secretase at the trans-Golgi network (TGN) of both transfected HeLa cells and mouse primary ne

87 enance of the Golgi and trans Golgi network (TGN) PI4P pools, however, the actual targeting mechanism

88 differentially affected trans-Golgi network (TGN) pools of PI(4)P and post-TGN traffic.

89 UT4 mobilization from a trans-Golgi network (TGN) storage compartment, establishing that insulin, in

90 lated checkpoint at the trans-Golgi network (TGN) that controls the surface delivery of the delta opi

91 nding centrin Cdc31, in trans-Golgi network (TGN) to endosome traffic and TGN homotypic fusion.

92 s from endosomes to the trans-Golgi network (TGN) to prevent proteolytic processing or by directing n

93 beta1 integrin from the trans-Golgi network (TGN) to the EC surface, thus allowing FN fibrillogenesis

94 s cargo proteins at the trans-Golgi network (TGN) to the endosome/lysosome pathway.

95 for transport from the trans-Golgi network (TGN) to the late endosome/prevacuolar compartment (PVC)

96 ber of cargoes from the trans-Golgi network (TGN) to the plasma membrane in Saccharomyces cerevisiae

97 mbrane protein from the trans-Golgi network (TGN) to the plasma membrane in the root epidermis of Ara

98 sports enzymes from the trans-Golgi network (TGN) to the vacuole.

99 exit of GPP130 from the trans-Golgi network (TGN) toward lysosomes is mediated by the sorting recepto

100 e-dependent endosome-to-trans-Golgi network (TGN) transport of the cation-independent mannose 6-phosp

101 4-phosphate [PI(4)P] on trans-Golgi network (TGN) vesicles were recruited to mitochondria-ER contact

102 or several decades, the trans-Golgi network (TGN) was considered the most distal stop and hence the u

103 on of SGs occurs at the trans-Golgi network (TGN) where their soluble cargo aggregates to form a dens

104 s from endosomes to the trans-Golgi network (TGN), and its mutation leads to severe protein mislocali

105 intracellularly in the trans-Golgi network (TGN), and we identified two motifs in the distal part of

106 at ANK localizes to the trans-Golgi network (TGN), clathrin-coated vesicles and the plasma membrane.

107 localized in the Golgi/trans-Golgi network (TGN), in the early endosomes, and on the plasma membrane

108 ograde transport to the trans-Golgi network (TGN), is involved in the presentation of ligands from My

109 ) from endosomes to the trans-Golgi network (TGN), is thought to consist of a cargo-selective VPS26-V

110 esicle formation at the trans-Golgi network (TGN), likely aiding the transport of cargo proteins from

111 At the trans-Golgi network (TGN), phosphatidylinositol 4-phosphate (PtdIns4P) plays

112 ein is localized to the trans-Golgi network (TGN), prevacuolar compartment (PVC), and plasma membrane

113 and associated with the trans-Golgi network (TGN), suggesting that FgVps35 functions at the donor end

114 of the receptor in the trans-Golgi network (TGN), to the effect that overexpressed PIST reduces acti

115 They form at the trans-Golgi network (TGN), where their soluble content aggregates to form a d

116 id is trafficked to the trans-Golgi network (TGN), whereupon it enters the nucleus during mitosis.

117 ining vesicles from the trans-Golgi network (TGN), which is regulated by a domain of protein RS1 (RSC

118 show a critical role of trans-Golgi network (TGN)-endosome trafficking during the assembly, but princ

119 e lectin, LMAN2, limits trans-Golgi Network (TGN)-to-endosomes traffic of GPRC5B, an exosome cargo pr

120 questration within the trans- Golgi network (TGN).

121 ingolipid levels at the trans-Golgi network (TGN).

122 esicle fission from the trans-Golgi network (TGN).

123 complex traffics to the trans-Golgi network (TGN).

124 ling from the PM to the trans-Golgi network (TGN).

125 sicles destined for the trans-Golgi network (TGN).

126 protein sorting at the trans-Golgi network (TGN).

127 o cargo fusion with the trans-Golgi network (TGN).

128 branes derived from the trans-Golgi network (TGN).

129 cretory proteins at the trans-Golgi network (TGN).

130 hat intersects with the trans-Golgi network (TGN).

131 oat associated with the trans-Golgi network (TGN).

132 sternal margins and the Trans-Golgi network (TGN).

133 LB1 associated with the trans-Golgi network (TGN)/early endosome (EE) and tracked along filamentous a

134 hinery operating at the trans-Golgi network (TGN)/endosome interface.

135 adherence, red cell thioguanine nucleotide (TGN) levels, and risk of relapse in children with TMPT w

136 sured as erythrocyte thioguanine nucleotide [TGN] levels) in children with acute lymphoblastic leukem

137 supports TGN integrity and recruits numerous TGN-localized ARF effectors.

138 acyl-chain length links lipid composition of TGN subdomains with polar secretory trafficking of PIN2

139 l microsomal spread and 2) identification of TGN and post-TGN cargo without the need for a TGN marker

140 ences the morphology and interconnections of TGN-associated secretory vesicles.

141 ing clathrin-coated vesicles, caused loss of TGN localization and somatodendritic polarity of ATP7B.

142 ant cell division in which a large number of TGN-derived membrane vesicles fuse with one another to f

143 Thus, recruitment of TGN-PI(4)P-containing vesicles at mitochondria-ER contac

144 argo selectivity for betagamma regulation of TGN to PM transport and a novel role for betagamma in me

145 crucial role for SLC9A7 in the regulation of TGN/post-Golgi pH homeostasis and glycosylation of expor

146 nterfering RNA (siRNA)-mediated silencing of TGN-associated adaptor proteins and a panel of dominant

147 found that Eqt-SM is enriched in a subset of TGN-derived secretory vesicles that are also enriched in

148 TRANSPORT TO THE VACUOLE1 (MTV1) as its only TGN-associated members.

149 Finally, we show that HCV NS2 perturbs TGN architecture, redistributing TGN membranes to closel

150 d spatially distinct subdomains of the plant TGN and suggest that functional redundancy can be achiev

151 tudies showed that cleavage occurs in a post-TGN compartment and that lysosomotropic agents enhance s

152 spread and 2) identification of TGN and post-TGN cargo without the need for a TGN marker that univers

153 Golgi network (TGN) pools of PI(4)P and post-TGN traffic.

154 eir arrival at the cell surface and the post-TGN breakup of large pleomorphic membrane fragments that

155 in (neuro-)endocrine cells, we now quantify TGN budding kinetics of constitutive and regulated secre

156 S2 perturbs TGN architecture, redistributing TGN membranes to closely associate with HCV core protein

157 AtTRAPPC11/ROG2 regulates TGN integrity, as evidenced by altered TGN/EE associatio

158 a reaction that requires Ca(2+), the soluble TGN-resident protein Cab45 is necessary for the sorting

159 and the TGN Ca(2+) pump (SPCA1) in specific TGN microdomains.

160 TGN SNAREs, and underwent Ca(2+)-stimulated TGN recruitment.

161 AC5 and RISAP is detectable at the subapical TGN compartment.

162 Unexpectedly, GBF1 also supports TGN integrity and recruits numerous TGN-localized ARF ef

163 Surgical TGN is an excellent 1-stage surgical option for symptoma

164 Here we show that TGN-localized E3/49K comprises both newly synthesized an

165 The TGN is a key compartment for the sorting and secretion o

166 The TGN is disrupted and vesicle budding from Golgi cisterna

167 The TGN-localized coiled-coil protein TNO1 is a putative tet

168 colocalizes with secretory proteins and the TGN Ca(2+) pump (SPCA1) in specific TGN microdomains.

169 ion of proteins related to endosomes and the TGN trafficking pathway.

170 nsion for both the cisternal margins and the TGN, whereas Golgi stack proliferation was unaffected.

171 irs by their degree of colocalization at the TGN and by the evolution of colocalization during their

172 s to defective MPR carrier biogenesis at the TGN and endosomes.

173 of a novel functional protein complex at the TGN and its key members: cytosolic PKD2 binds ARF-like G

174 demonstrate the sorting of native SM at the TGN and its transport to the plasma membrane by specific

175 The primary role of PS flip at the TGN appears to be to control the oxysterol-binding prote

176 responsible for synthesis of PtdIns4P at the TGN are not well characterized.

177 Blocking vesicle formation at the TGN revealed cis-SNARE complexes.

178 that generation of putrescine by ODC1 at the TGN stimulates release of SGLT1-containing vesicles.

179 ormation of this multiprotein complex at the TGN that controls constitutive secretion of matrix metal

180 facilitating localized actin assembly at the TGN through K33-linked ubiquitination of coronin 7.

181 mes and lysosomes is thought to occur at the TGN through recognition of sorting signals in the cytoso

182 y of the plasma membrane also operate at the TGN to confer sorting of an integral membrane protein an

183 we show that sphingomyelin production at the TGN triggers a signalling pathway leading to PtdIns(4)P

184 complexes promote vesicle generation at the TGN with different cargo specificity and destination.

185 es sorting of O-glycosylated proteins at the TGN, as well as Rab8-dependent post-Golgi trafficking of

186 PSIN1/AP-1 and MTV1/AP-4 pairs reside at the TGN, high-resolution microscopy reveals them as spatiall

187 availability governs protein sorting at the TGN, likely by regulating sterol transfer to the TGN by

188 eractions of p14 with activated Rab11 at the TGN, resulting in p14 sorting into AP1-coated vesicles f

189 At the TGN, soluble proteins are sorted based on the instructio

190 to capture endosome-derived vesicles at the TGN, were individually relocalised to mitochondria and t

191 At the TGN, where PKD2 interacts with active ARF1, PKD2, and AR

192 binds to different receptor proteins at the TGN, which trigger release of vesicles with different tr

193 or mediating efficient vesicle fusion at the TGN.

194 n by controlling dense core formation at the TGN.

195 hosphatidylinositol 4-kinase function at the TGN.

196 for the sorting of secretory cargoes at the TGN.

197 b11 and AP-1 both colocalize with p14 at the TGN.

198 e sorting of specific cargo molecules at the TGN.

199 regation of protein and lipid domains at the TGN.

200 f the ART/Rsp5 ubiquitylation complex at the TGN.

201 R1 negatively regulates ATL1 activity at the TGN/EE and thus controls stress responses initiated by A

202 ll surface regulates membrane traffic at the TGN/endosomal interface.

203 red for AP-1-dependent transport between the TGN and endosomes in yeast.

204 thrin/AP-mediated cycling of ANK between the TGN, endosomes, and the cell surface regulates membrane

205 onto SGs during budding, but rather exit the TGN into nonregulated vesicles to get incorporated to SG

206 ergo progressive sorting after they exit the TGN toward the cell surface.

207 ld be required for the viral DNA to exit the TGN.

208 ar sites and reveals a critical role for the TGN in GPCR signaling.Recent investigations suggest that

209 e incoming viral genome dissociates from the TGN and associates with microtubules after the onset of

210 een implicated in polar trafficking from the TGN but the underlying mechanisms linking lipid composit

211 flux carrier to the plasma membrane from the TGN during hook development and defects in BIG or ARF1 r

212 ing the transport of cargo proteins from the TGN for proper location; but EPS1's impact on physiologi

213 that timely exit of these proteins from the TGN is critical for effective pre-invasive immune respon

214 rough tubular carriers that emanate from the TGN or through vesicular carriers that recruit GGA (Golg

215 G cargoes immediately after budding from the TGN revealed that, surprisingly, the bulk of two studied

216 key regulators of protein transport from the TGN so far is elusive.

217 pathway lead to relocation of PIST from the TGN to an endosome-like compartment.

218 newly synthesized membrane proteins from the TGN to apical membrane in live zebrafish.

219 ither protein causes ANK dispersion from the TGN to cytoplasmic endosome-like puncta.

220 ansmembrane autophagy protein ATG9A from the TGN to preautophagosomal structures.

221 that CTL1 regulates protein sorting from the TGN to the PM through its function in lipid homeostasis.

222 RK2ct-KERE inhibits cargo transport from the TGN to the PM.

223 roteins Pma1 and Can1 are missorted from the TGN to the vacuole in drs2 cells.

224 of SORLA in endosomes and depletion from the TGN, and in an overall enhanced APP processing.

225 PV-harboring transport vesicles bud from the TGN, followed by association with mitotic chromosomes.

226 mation of tubular membrane carriers from the TGN, perinuclear accumulation of early endosomes and imp

227 generic ACT7-dependent trafficking from the TGN, the EXOCYST84b (EXO84b) tethering factor mediates P

228 From the TGN, the protein is targeted to growing cell plates duri

229 oprotein lipase (LPL) during export from the TGN.

230 o inhibits Cu-dependent trafficking from the TGN.

231 equired for receptor sorting to and from the TGN.

232 These findings identify the TGN as a major site for alpha-secretase processing in He

233 APP Y687 residue that accumulates APP in the TGN (Trans-Golgi Network) and diminishes its amyloidogen

234 in the accumulation of endogenous APP in the TGN and enhanced alpha-secretase processing.

235 eptors can be released from retention in the TGN by coexpression of the plasma membrane-associated sc

236 -secretase following APP accumulation in the TGN increases the levels of the membrane-bound C-termina

237 In alp3, PEN3 accumulates in the TGN, causing delays in recruitment to the host-pathogen

238 BACE1 processing is the minor pathway in the TGN.

239 sponsible for retaining the M protein in the TGN.

240 ell wall polysaccharide epitopes seen in the TGN.

241 an mediate lipoprotein lipase sorting in the TGN.

242 ssumes a transmembranous conformation in the TGN.

243 The receptors were arrested in the TGN.

244 tion and thus impacts Cab45 retention in the TGN.

245 ng site also affected Ca(2+) import into the TGN and secretory cargo sorting.

246 n plants and provides novel insight into the TGN-associated clathrin-coated vesicle trafficking machi

247 iched in secretory vesicle subdomains of the TGN and are critical for de novo polar secretory sorting

248 accompanied by apparent fragmentation of the TGN and redistribution of K15P to a dispersed peripheral

249 retory granule formation at the level of the TGN are still elusive.

250 ts define the GLUT4-containing region of the TGN as a sorting and storage site from which different c

251 , in part, through utilizing a region of the TGN devoted to specialized cargo transport in general ra

252 LUT4 is retained in an element/domain of the TGN from which newly synthesized lysosomal proteins are

253 Disruption of the TGN significantly reduced nuclear translocation of viral

254 x with actin to modulate the function of the TGN/EE at the intersection of the exocytic and endocytic

255 or-ligand pair is largely independent of the TGN/EE.

256 This correlated with alkalinization of the TGN/post-Golgi compartments, suggestive of a gain-of-fun

257 LISA and coimmunoprecipitation show that the TGN/endosomal small GTPase Rab14 and PKCiota interact di

258 ense proteins continuously cycle through the TGN and that timely exit of these proteins from the TGN

259 1 facilitates the recruitment of GARP to the TGN and compensates for the endosome-to-TGN trafficking

260 The syntaxin TlgB(Tlg2) localizing to the TGN appears to mediate retrograde traffic connecting pos

261 ate that MERS-CoV M protein localizes to the TGN because of the combined presence of an ER export sig

262 likely by regulating sterol transfer to the TGN by Osh1, a member of the conserved oxysterol-binding

263 Transport from recycling endosomes to the TGN has also been reported, but much less is understood

264 proteins from the recycling endosome to the TGN in Caenorhabditis elegans.

265 ted retrograde-protein (GARP) complex to the TGN in the absence of Ypt6.

266 de transport of CI-MPR from endosomes to the TGN independently of the core retromer trimer.

267 s suggest that retrograde trafficking to the TGN induces local Gs-protein activation and cAMP/PKA sig

268 possibility that compartments distal to the TGN mediate or contribute to biosynthetic sorting.

269 of internalized Shiga toxin B subunit to the TGN, as well as recycling of internalized transferrin to

270 adation in lysosomes and retrieves it to the TGN, where insulin--responsive vesicles are formed.

271 uitment of the PI3K C2A kinase domain to the TGN-induced deltaR export downstream of NGF.

272 n of PtdIns4P and recruitment of AP-1 to the TGN.

273 r-coated compartment that brings them to the TGN.

274 ARL1, in turn, localizes Arfaptin2 to the TGN.

275 gh endosomal trafficking compartments to the TGN.

276 red for efficient recruitment of GARP to the TGN.

277 -like GTPase (ARL1) and shuttles ARL1 to the TGN.

278 h AP-4, and for recruitment of tepsin to the TGN.

279 cruit golgins and GARP, respectively, to the TGN.

280 lator of retrograde-carrier tethering to the TGN.

281 for the delivery of retrograde cargos to the TGN.

282 bility to transfer phosphatidylserine to the TGN.

283 mutant protein was correctly targeted to the TGN/post-Golgi vesicles, but its N-linked oligosaccharid

284 fusion of endosome-derived carriers with the TGN and recycling endosomes, respectively.

285 ive tethering factor that interacts with the TGN t-SNARE SYP41 and is required for correct localizati

286 ting post-Golgi (sorting) endosomes with the TGN.

287 APP processing pathways can occur within the TGN compartment along the secretory pathway.

288 the evolution of colocalization during their TGN-to-surface transport.

289 In this TGN compartment, apical secretion and endocytic membrane

290 port was nearly normal as was trans Golgi to TGN transport.

291 UB13 fusion protein (YFP-PUB13) localizes to TGN and Golgi compartments and that PUB13, PI4Kbeta1, an

292 gh recognition of a specific WLM endosome-to-TGN sorting motif.

293 the TGN and compensates for the endosome-to-TGN trafficking defects in dysfunctional Ypt6 conditions

294 t only Arl1 and Imh1 can restore endosome-to-TGN trafficking in ypt6-deleted cells.

295 restoring GARP localization and endosome-to-TGN transport in ypt6-deleted cells.

296 to a pronounced defect in CI-MPR endosome-to-TGN transport.

297 of tubular profiles required for endosome-to-TGN transport.

298 Furthermore, adherers with varying TGN levels had varying 6MP dose intensity (odds ratio [O

299 lasma membrane only in border cells, whereas TGN-LVs containing the XG and PGA/RG-I epitopes fuse wit

300 xhibited Ca(2+)-stimulated interactions with TGN SNAREs, and underwent Ca(2+)-stimulated TGN recruitm