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1                                              TNF beta expression correlated significantly with the oc
2                                              TNF-beta polymorphisms within the donor liver did not co
3                                              TNF-beta that is encoded by lymphotoxin-alpha gene (LTA)
4 6, IL-12p70, IL-10, GM-CSF, VEGF, MIP-1beta, TNF-beta, IFN-alpha2 and IL-7 in response to an LPS chal
5 tile of bFGF, fibronectin, thrombospondin-2, TNF-beta, or VEGF had an odds ratio (OR) of 11.8 for hav
6 ha, tumor necrosis factor alpha (TNF-alpha), TNF-beta, and IL-6 expression.
7 nes tumor necrosis factor alpha (TNF-alpha), TNF-beta, and interleukin-1 alpha and chemokines macroph
8                   Lymphotoxin (LT, LT alpha, TNF beta) is a member of the immediate TNF family that a
9 thway that is independent of Fas, TNF alpha, TNF beta, and ATP, but is dependent upon extracellular c
10  characterized by the presence of TNF alpha, TNF beta, and cells expressing TNFR.
11 re analyzed for the expression of TNF alpha, TNF beta, and their receptors (p55 and p75 TNFR), utiliz
12 we suggest that lymphotoxin's (LT, LT-alpha, TNF-beta) crucial role in these processes is pivotal and
13 IL-1beta, tumor necrosis factor (TNF) alpha, TNF-beta, and IL-6 and in the IL-1 receptor antagonist (
14 ective in tumor necrosis factor (TNF)-alpha, TNF-beta, lymphotoxin-beta, or TNFR1, TNFR2, Fas, or dea
15 XCL9), proinflammatory cytokines (TNF-alpha, TNF-beta, IL-6), chemokines related to host-commensal ju
16 dd, fas, fas ligand (fas L), ice, TNF-alpha, TNF-beta, TNFR1, TNFR2, TRAF1, TRAF2, TRAF3, cIAP2, and
17 decreased activity of this Fas-, TNF alpha-, TNF beta-, ATP-independent pathway associated with very
18           The presence of both TNF alpha and TNF beta expression was demonstrated in most JRA and JSp
19  the regulation of IL-17, IL-22, IL-28A, and TNF-beta cytokines in GBS patients.
20 mphokines: interferon-gamma, IL-2, IL-4, and TNF-beta) in tissues obtained at autopsy from subjects w
21 rease of proinflammatory cytokines, IL-6 and TNF-beta, indicating that exosomes containing TAR RNA co
22 n was also achieved when IL-1beta, IL-6, and TNF-beta were added to macrophages with cross-linked cos
23  yet it elicited production of TNF-alpha and TNF-beta, and induced TCR Vbeta-specific proliferation s
24 , which contains the genes for TNF-alpha and TNF-beta, is located on chromosome 6 within the major hi
25 ceptor, they came to be called TNF-alpha and TNF-beta.
26  Tumor necrosis factor alpha (TNF-alpha) and TNF-beta mRNAs were upregulated in advanced stages of di
27  tumor necrosis factor alpha (TNF-alpha) and TNF-beta, which is consistent with functional stimulatio
28  tumor necrosis factor alpha (TNF-alpha) and TNF-beta.
29 ses the release of IFN-gamma, TNF-alpha, and TNF-beta through direct binding to multiple conserved re
30 CL10, IL-1beta, IL-10, sCD14, TNF-alpha, and TNF-beta) achieved a sensitivity of 0.90 (95% CI: 0.87-0
31 CD14, tumor necrosis factor [TNF]-alpha, and TNF-beta) achieved a sensitivity of 0.90 (95% confidence
32 te-macrophage-CSF, IFN-gamma, TNF-alpha, and TNF-beta) release by both TALL-104 and LAK cells, ligati
33 plasma levels of tumor necrosis factor beta (TNF-beta).
34 eta (IL-1 beta), tumor necrosis factor-beta (TNF-beta), or interleukin-6 (IL-6), messenger RNA was el
35 1-like cells, but the cytokine modulation by TNF-beta and the clinical significance of this cytokine
36                Compared with medium control, TNF-beta significantly decreased IL-5 (p = 0.0004) and I
37 h diseases, including Th1 (IFN-gamma, CXCL9, TNF-beta) and Th17 (CCL20) markers.
38 ker (MSM) upstream of the TNFB gene encoding TNF-beta and at a restriction fragment length polymorphi
39  These findings suggest a potential role for TNF-beta in the pathogenesis of AD.
40  cytokines examined include IL-2, IFN-gamma, TNF-beta/LT, IL-4, IL-5, IL-10, and TNF-alpha.
41                               Interestingly, TNF-beta significantly decreased IgE (p = 0.02), but not
42 We have examined the ability of lymphotoxin (TNF-beta), to stimulate rolling, adhesion, and transmigr
43                            The prominence of TNF beta in the synovium in particular subgroups of JRA
44 nal antibodies in the presence or absence of TNF-beta.
45                                The effect of TNF-beta on cytokine production was investigated by cult
46                                The effect of TNF-beta on IgE production was determined by culturing p
47 FA1/2), and the other in the first intron of TNF-beta (TNFB1/2).
48 showed significant increase in the levels of TNF-beta, IL-5, PTX3 in T1DM, T2DM and obesity groups in
49 ntibodies and investigated the production of TNF-beta by ELISA.
50 ce proliferation of human PBLs or release of TNF-beta, but did induce TNF-alpha release.
51 rried out to determine the potential role of TNF-beta in AD.
52                          The mean +/- SEM of TNF-beta production in AD was significantly lower than n
53 n rates and the presence of any TNF-alpha or TNF-beta alleles.
54                  Our study demonstrates that TNF-beta production is downregulated in AD.
55  the crystal structures of TNF alpha and the TNF beta/TNF-receptor(I) complex and a model of an anti-
56 extends from HLA class II to upstream of the TNF-beta gene.
57 nalysis for four polymorphic loci within the TNF-beta gene (NcoI, TNFc, aa13, and aa26).
58 pha gene construct, which contains the whole TNF-beta gene with 1.2 kb of 5' flanking sequence, 1.1 k