コーパス検索結果 (left1)
通し番号をクリックするとPubMedの該当ページを表示します
1 TNF beta expression correlated significantly with the oc
2 TNF-beta polymorphisms within the donor liver did not co
3 TNF-beta that is encoded by lymphotoxin-alpha gene (LTA)
4 6, IL-12p70, IL-10, GM-CSF, VEGF, MIP-1beta, TNF-beta, IFN-alpha2 and IL-7 in response to an LPS chal
5 tile of bFGF, fibronectin, thrombospondin-2, TNF-beta, or VEGF had an odds ratio (OR) of 11.8 for hav
7 nes tumor necrosis factor alpha (TNF-alpha), TNF-beta, and interleukin-1 alpha and chemokines macroph
9 thway that is independent of Fas, TNF alpha, TNF beta, and ATP, but is dependent upon extracellular c
11 re analyzed for the expression of TNF alpha, TNF beta, and their receptors (p55 and p75 TNFR), utiliz
12 we suggest that lymphotoxin's (LT, LT-alpha, TNF-beta) crucial role in these processes is pivotal and
13 IL-1beta, tumor necrosis factor (TNF) alpha, TNF-beta, and IL-6 and in the IL-1 receptor antagonist (
14 ective in tumor necrosis factor (TNF)-alpha, TNF-beta, lymphotoxin-beta, or TNFR1, TNFR2, Fas, or dea
15 XCL9), proinflammatory cytokines (TNF-alpha, TNF-beta, IL-6), chemokines related to host-commensal ju
16 dd, fas, fas ligand (fas L), ice, TNF-alpha, TNF-beta, TNFR1, TNFR2, TRAF1, TRAF2, TRAF3, cIAP2, and
17 decreased activity of this Fas-, TNF alpha-, TNF beta-, ATP-independent pathway associated with very
20 mphokines: interferon-gamma, IL-2, IL-4, and TNF-beta) in tissues obtained at autopsy from subjects w
21 rease of proinflammatory cytokines, IL-6 and TNF-beta, indicating that exosomes containing TAR RNA co
22 n was also achieved when IL-1beta, IL-6, and TNF-beta were added to macrophages with cross-linked cos
23 yet it elicited production of TNF-alpha and TNF-beta, and induced TCR Vbeta-specific proliferation s
24 , which contains the genes for TNF-alpha and TNF-beta, is located on chromosome 6 within the major hi
26 Tumor necrosis factor alpha (TNF-alpha) and TNF-beta mRNAs were upregulated in advanced stages of di
27 tumor necrosis factor alpha (TNF-alpha) and TNF-beta, which is consistent with functional stimulatio
29 ses the release of IFN-gamma, TNF-alpha, and TNF-beta through direct binding to multiple conserved re
30 CL10, IL-1beta, IL-10, sCD14, TNF-alpha, and TNF-beta) achieved a sensitivity of 0.90 (95% CI: 0.87-0
31 CD14, tumor necrosis factor [TNF]-alpha, and TNF-beta) achieved a sensitivity of 0.90 (95% confidence
32 te-macrophage-CSF, IFN-gamma, TNF-alpha, and TNF-beta) release by both TALL-104 and LAK cells, ligati
34 eta (IL-1 beta), tumor necrosis factor-beta (TNF-beta), or interleukin-6 (IL-6), messenger RNA was el
35 1-like cells, but the cytokine modulation by TNF-beta and the clinical significance of this cytokine
38 ker (MSM) upstream of the TNFB gene encoding TNF-beta and at a restriction fragment length polymorphi
42 We have examined the ability of lymphotoxin (TNF-beta), to stimulate rolling, adhesion, and transmigr
48 showed significant increase in the levels of TNF-beta, IL-5, PTX3 in T1DM, T2DM and obesity groups in
55 the crystal structures of TNF alpha and the TNF beta/TNF-receptor(I) complex and a model of an anti-
58 pha gene construct, which contains the whole TNF-beta gene with 1.2 kb of 5' flanking sequence, 1.1 k