ライフサイエンスコーパス: TR

1 TR dropped to 0.2 mmh(-1) within eight days after applic

2 TR expressing TNgO(GT4) was resistant to neutralization

3 TR RR spectroscopy offers the advantage of simultaneousl

4 TR-FRET resolved an equilibrium between two distinct str

5 TR-Mphi were enriched in TAC3 and associated with mitoch

6 able analysis, combining 90% to 99% and 100% TR was independently associated with improved OS, compar

7 Survival curves for 90% to 99% TR and 100% TR were similar, and a change in probability of improved

8 ned in more than 50% of their areas, with 12 TRs (48%) being overall better than their matched Wildli

9 claration as TRs, vegetation condition in 13 TRs (52%) declined in more than 50% of their areas, with

10 ion condition (greenness and moisture) in 25 TRs.

11 y of gene expression (noncanonical or type 3 TR signaling).

12 ed over more than 25% of their areas, with 3 TRs being overall better than their matched WLSs.

13 after declaration, vegetation condition in 5 TRs was harmed over more than 25% of their areas, with 3

14 and yeast 40Ss distribute similarly across 5'TRs, indicating considerable evolutionary conservation.

15 proved OS, compared with 50% to 89% and <50% TR.

16 evis POM, and Lactobacillus plantarum (TR-7, TR-71, TR-14) were used to promote the fermentation of t

17 M, and Lactobacillus plantarum (TR-7, TR-71, TR-14) were used to promote the fermentation of the beve

18 h of L. brevis POM, and L. plantarum (TR-71, TR-14), observing higher antioxidant properties in the f

19 obability of improved OS was observed at 90% TR.

20 c response to be expanded, from pCR to >=90% TR and ypN0.

21 to trimodality therapy, in addition to >=90% TR in the primary tumor bed.

22 Survival curves for 90% to 99% TR and 100% TR were similar, and a change in probability

23 High-quality (grade A) TR Doppler was present in 68% at rest and 34% at peak ex

24 in (amino acids [aa] 1100 to 1150) abolished TR replication.

25 methods to successfully image low-abundance TR in living cells.

26 performed at baseline, during radial access, TR band application, and at discharge.

27 ith right heart remodeling differ between AF-TR and left-sided heart disease-TR.

28 right ventricular end-systolic volume in AF-TR (P<0.001).

29 Optical pathway activation failed to alter TR to innately bitter quinine in either sex.

30 duced in both hands during radial access and TR band application but is not associated with future lo

31 fusion was observed during radial access and TR band application in the homolateral thumb (-32%, -32%

32 The development of AF and TR was unrelated to recurrent MR.

33 simultaneously binds mitotic chromosomes and TR DNA to segregate virus genomes to daughter cell nucle

34 also assist in the investigation of Ig- and TR-related genetic predispositions to diseases.

35 or tandem repeat arrays (CentC, knob180, and TR-1) are discontinuous and frequently interspersed with

36 1123 reduced MCM6 association with LANA and TR replication.

37 cient at cell-to-cell spread, whereas TB and TR cell-to-cell spread is poor.

38 ions in fibroblasts, whereas TB40/e (TB) and TR are low in UL128-131, are RL13 intact, and produce vi

39 Mutations in the catalytic TERT and TR subunits of telomerase compromise activity, while oth

40 e relationship between TR volume (TRVol) and TR fraction (TRF) with all-cause mortality.

41 microbiome profiles in both humans and APOE-TR mice.

42 rmed by comparing the gut microbiota of APOE-TR mice.

43 Relative to injured APOE3 TR mice, injured APOE4 TR mice had more inflammation, ne

44 tive to injured APOE3 TR mice, injured APOE4 TR mice had more inflammation, neurodegeneration, apopto

45 iet (HFD) accelerates these effects in apoE4-TR mice at middle age.

46 tly, our results reveal how state-of-the-art TR-XANES provides deeper insights of ultrafast nonadiaba

47 After declaration as TRs, vegetation condition in 13 TRs (52%) declined in mo

48 ing that suppression of conditioned aversive TR can be dissociable from the effects of unconditioned

49 usion of devalued sucrose inhibited aversive TR in male but not female rats.

50 ients were divided into 2 groups by baseline TR severity: none/trace/mild TR (<=Mild TR) (n = 501 [83

51 compared with GDMT, irrespective of baseline TR grade (p(interaction) = 0.16, 0.29, and 0.21 respecti

52 Degree of baseline TR may be an important marker of long-term outcomes in t

53 y sought to examine the relationship between TR volume (TRVol) and TR fraction (TRF) with all-cause m

54 associate with mitotic chromosomes and bind TR DNA.

55 for a cofactor, Dot1L, in gene activation by TR during vertebrate development.

56 ence, we propose that the T(1) determined by TR-vSFG in combination with DFT-MD simulations can be wi

57 s, one to assess lung tissue-resident cells (TR-Mphi) and two for their polarization (classically and

58 In conclusion, TRs are required for normal proliferation and T(3) signa

59 We now report that the same conformational TR-FRET based immunoassay detects polyglutamine- and tem

60 preferentially and coordinately corecruited TRs, assemble into hierarchical combinations at hepatic

61 he HCR amplification analysis of cytoplasmic TR.

62 GFP) expression from a replication-deficient TR plasmid.

63 sisting of 7 simultaneous assays that detect TR(34) (a 34-bp tandem repeat in the promoter region), T

64 l patients listed on the TraumaRegister DGU (TR-DGU) in Germany between 2002 and 2015 aged 16 years o

65 signals using STAIR-UTE imaging at different TRs.

66 r between AF-TR and left-sided heart disease-TR.

67 s effective donor materials and made DPPEZnP-TRs a family of best low-bandgap donor materials in the

68 Two genes, THRA and THRB, encode TRs; THRB2 has been associated with cone viability.

69 e-wide genotyping of both short and expanded TRs.

70 he transcriptomic changes involved in feline TR by sequencing RNA extracted from 14 teeth (7 with and

71 be a potential therapeutic target in feline TR.

72 e the fluorescence molecular beacon H(1) for TR recognition, H(2) for hybrid chain reaction (HCR) and

73 roughput, cell-based reporter gene assay for TR agonist or antagonist activity.

74 previous high-throughput time-resolved FRET (TR-FRET) screen, we identified a class of compounds that

75 Furthermore, we include data from TR-FRET experiments that validate this simulation approa

76 endent prognostic implications of functional TR.

77 e treatment of moderate to severe functional TR in 30 patients enrolled in the TRI-REPAIR (TrIcuspid

78 ymptomatic and moderate to severe functional TR.

79 We examined 547 patients with functional TR using CMR to quantify TRVol and TRF.

80 In patients with functional TR, this study sought to examine the relationship betwee

81 Furthermore, TRs have recently been implicated in a range of complex

82 f detection, its inherent sensitivity (e.g., TR-FRET versus radiometric detection), and the ability t

83 Here we describe a second kinesin-14 gene, TR-1 kinesin (Trkin), that is required to mobilize neoce

84 solved vibrational sum frequency generation (TR-vSFG) spectroscopy with ab initio DFT-based molecular

85 A Glycine-TR adduct was used as a control sample to identify the p

86 tes, similar to human mutation carriers, had TR insufficiency but an otherwise preserved transcriptom

87 rticular, a paired-end tetraloop (no hanging TR) is stable in (CAG)n = even, but such situation canno

88 lved in regulatory activities of the hepatic TR, NR1H4 (FXR; farnesoid X receptor), as our model syst

89 s provided a framework for understanding how TR, TERT, and other proteins from ciliate as well as ver

90 nts, Lisa boosted the performance of imputed TR cistromes and outperformed alternative methods in ide

91 Using TR ChIP-seq peaks or imputed TR binding sites, Lisa probes the chromatin models using

92 munocytochemistry of odontoclasts located in TR lesions.

93 ted with an additional 2.3-fold reduction in TR grade (odds ratio [OR]: 0.44; 95% confidence interval

94 Reducing strand slipping in TR hairpins by sequence interruptions at the loop sugges

95 RLFS in TRs are positionally conserved between herpesviruses.

96 To gain insight into TR pathogenesis, we characterised the transcriptomic cha

97 h fluorescent probes and used intramolecular TR-FRET to assess interlobe distances when CaM is bound

98 to assess the impact of surgery for isolated TR, comparing survival for isolated severe TR patients w

99 RNA-seq analysis of isolated TR-APhi and BM-APhi from 4- and 32-weeks exposed mice re

100 sorders, 4th Edition, Text Revision (DSM -IV-TR), along with assessment of severity of illness using

101 h an acute manic or mixed episode per DSM-IV-TR criteria were enrolled in this randomized, placebo-co

102 lations were based on considering the DSM-IV-TR criterion as the reference standard.

103 ects organized by Biotype and then by DSM-IV-TR diagnosis (n = 1409) using voxel-based morphometry wi

104 ealthy control subjects, organized by DSM-IV-TR diagnosis.

105 ature on cancer-related PTSD has used DSM-IV-TR diagnostic criteria; the revised DSM-5 PTSD criteria

106 unified model to estimate maximum likelihood TR lengths.

107 at healthy individuals harbor dozens of long TR alleles not captured by current genome-wide methods.

108 7 (27%) of 63 lesions were categorized as LR-TR Equivocal, and 12 of these lesions were incompletely

109 gory based on LI-RADS treatment response (LR-TR) category (viable or nonviable) were calculated by us

110 alues for the LI-RADS Treatment Response (LR-TR) Viable and Nonviable categories were calculated for

111 Interreader association for the LR-TR category was moderate (kappa = 0.55; 95% confidence i

112 logic tumor necrosis, the accuracy of the LR-TR Nonviable category was 67%-71%, and the negative pred

113 logic tumor necrosis, the accuracy of the LR-TR Viable category for the three readers was 60%-65%, an

114 Six (11%) of 53 treated lesions were LR-TR equivocal by consensus, with most (five of six) incom

115 d specificity ranged from 85% to 97% when LR-TR equivocal assessments were treated as nonviable.

116 When LR-TR equivocal assessments were treated as viable, sensiti

117 NA (TR) accumulated at the expense of mature TR, consistent with a role for ZCCHC8 in mediating TR 3'

118 nsistent with a role for ZCCHC8 in mediating TR 3' end targeting to the nuclear RNA exosome.

119 roximately 19% shorter with the DEPC method (TR, 5.7 msec) than with the SEPC method (TR, 2.8 msec +/

120 od (TR, 5.7 msec) than with the SEPC method (TR, 2.8 msec +/- 4.2) (P < .05).

121 rculation status was associated with </=mild TR (odds ratio, 18.6; 95% CI, 5.3-65.2; P<0.001) and low

122 patients with >=Mod TR compared with <=Mild TR treated with GDMT alone (83.0% vs. 64.3%; hazard rati

123 line TR severity: none/trace/mild TR (<=Mild TR) (n = 501 [83.6%]) and moderate/severe TR (>=Mod TR)

124 nical outcomes compared to those with <=Mild TR.

125 ic peptide (p = 0.02) than those with <=Mild TR.

126 ups by baseline TR severity: none/trace/mild TR (<=Mild TR) (n = 501 [83.6%]) and moderate/severe TR

127 ath or HFH was higher in patients with >=Mod TR compared with <=Mild TR treated with GDMT alone (83.0

128 Patients with >=Mod TR had more severe MR (p = 0.0005) despite smaller left

129 with severe secondary MR who also had >=Mod TR had worse clinical and echocardiographic characterist

130 outcomes in patients with and without >=Mod TR severity compared with GDMT alone.

131 Patients with >=Mod TR were more likely to be New York Heart Association fun

132 = 501 [83.6%]) and moderate/severe TR (>=Mod TR) (n = 98 [16.4%]).

133 lly relevant chemicals capable of modulating TR activity via the collaborative interagency Tox21 proj

134 As compared with pure AR, AR + MR + TR exhibit the largest mortality risk.

135 Compared with pure AR, AR + MR + TR exhibited the highest adjusted risk of death (2.4-fol

136 was an increasing incidence of recurrent MR, TR, and new AF over time.

137 um showed near complete specificity for MUC4 TR glycopeptides and peptides, relative to all component

138 erted transcriptional regulation by multiple TRs at CRMs.

139 lipping dynamics and conformations of (CAG)n TR hairpins.

140 Nevertheless, TRs also rapidly activate intracellular second-messenger

141 cant confounding cytotoxicity and other, non-TR-specific mechanisms common to the transactivation ass

142 the physiological relevance of noncanonical TR signaling, we generated knockin mice with a mutation

143 s provide in vivo evidence that noncanonical TR signaling exerts physiologically important cardiometa

144 to identify IL-36 antagonists using a novel TR-FRET binding assay.

145 on peptide component in the same NF, nuclear TR can also be sensitively detected.

146 this account, we review the achievements of TR RR spectroscopy of nearly 50 years of research in thi

147 Indirect activation of TR through activation of its heterodimer partner, the re

148 rs hundreds of thousands of TRs, analysis of TR expansions has been mainly limited to known pathogeni

149 ce, that together with enhanced apoptosis of TR-APhi and reorganization of transcriptional responses,

150 d Thrb2 deletion, we examined the effects of TR inhibition.

151 This study sought to determine the impact of TR on outcomes in patients with heart failure and severe

152 HgO(GT2a) increased cell-free infectivity of TR in both cell types, but spread in fibroblasts was imp

153 findings support the view that inhibition of TR locally in the retina is a therapeutic strategy for r

154 y sequenced trio to profile the landscape of TR expansions in a healthy family and validate novel exp

155 fully used to assay the expression levels of TR in HeLa, HepG2 and HL-7702 cells, demonstrating that

156 mL(-1) ) or a combination in the presence of TR antagonist NH3 (100 nm) or following short interferin

157 P9 expression is involved in the progress of TR pathogenesis and that MMP9 may be a potential therape

158 ced apoptosis and decreased proliferation of TR-APhi.

159 1 or more MitraClip devices and reduction of TR by at least 1 grade, were evaluated before discharge

160 Significant reduction of TR through decrease of annular dimensions, improvements

161 acy analysis showed encouraging reduction of TR, which may potentially result in improved clinical ou

162 ield, which also illustrates how the role of TR RR spectroscopy in molecular life science has changed

163 within the region compromised segregation of TR DNA to daughter cells, as assessed by retention of gr

164 nce within the region reduced segregation of TR DNA to daughter cells, but not episome maintenance.

165 Optimal thresholds of TR for differentiating patients in terms of OS were inve

166 ents on five manually curated collections of TRs have shown that Dot2dot is more accurate than other

167 to be suitable for whole-genome discovery of TRs.

168 The need of comprehensive lists of TRs for association studies and the absence of a computa

169 uman genome harbors hundreds of thousands of TRs, analysis of TR expansions has been mainly limited t

170 tural diversity for direct ligand effects on TR and imply that other potential target sites in the th

171 slips back and forth between states with one TR hanging at the 5' or 3' end.

172 d valve intervention (TVI) on post-operative TR, length of hospital stay, and on a composite endpoint

173 leads independently predicted post-operative TR.

174 Pre-operative TR severity and presence of transvalvular leads independ

175 ikingly, ME spread far better than did TB or TR in the presence of neutralizing antibodies on both ce

176 was still more efficient than that of TB or TR.

177 he lungs with no contribution to the overall TR-APhi pool.

178 all survival (OS) was examined by percentage TR in the primary tumor bed and pathologic nodal stage (

179 rnative methods in identifying the perturbed TRs.

180 t for beverages inoculated with L. plantarum TR-7.

181 e growth of L. brevis POM, and L. plantarum (TR-71, TR-14), observing higher antioxidant properties i

182 lus brevis POM, and Lactobacillus plantarum (TR-7, TR-71, TR-14) were used to promote the fermentatio

183 cal classes have been identified as possible TR modulators, and the receptors appear highly selective

184 ong the subset of patients with high-quality TR Doppler signal.

185 uding the left and right thalamic radiation (TR), superior longitudinal fasciculus (SLF), corpus call

186 Reductions in transpiration rate (TR) and leaf area were greatest with foramsulfuron appli

187 in appetitive or aversive taste reactivity (TR) to sucrose was observed indicating that initial sucr

188 Traut's Reagent (TR) was used to thiolate Bovine serum albumin (BSA) in s

189 the immunoglobulin (Ig) and T cell receptor (TR) gene loci.

190 ent actions of the thyroid hormone receptor (TR) during development and in hypothyroidism.

191 Loss of thyroid hormone receptor (TR)alpha1 abolishes T(3) signalling via extracellular si

192 nd Dot1L in turn functions as a T3 receptor (TR) coactivator to promote vertebrate development.

193 s-inducing ligand (TRAIL; TNFSF10) receptor (TR) is a pro-apoptotic receptor whose contribution to ch

194 r thyroid-hormones (TH) and their receptors (TR) coordinate the larval recruitment of the coral-reef-

195 Thyroid hormone receptors (TRs) are critical endocrine receptors that regulate a mu

196 are mediated via thyroid hormone receptors (TRs) or by non-genomic mechanisms.

197 get gene transcription through T3 receptors (TRs).

198 materials, such as natural taste receptors (TRs) regarding receptor/ligand affinity.

199 Thyroid hormone (TH) and TH receptors (TRs) alpha and beta act by binding to TH response elemen

200 Single Texas red (TR) dye was specifically attached in the junction of the

201 34-bp tandem repeat in the promoter region), TR(46), G54W (a change of G to W at position 54), G54R,

202 d specificity of transcriptional regulators (TRs) often prohibits the development of such ultra-high-

203 tion by multiple transcriptional regulators (TRs).

204 Quantitation of tricuspid regurgitant (TR) severity can be challenging with conventional echoca

205 index, >= moderate tricuspid regurgitation (TR) (7%, 35%, and 53%, respectively), and right ventricu

206 moderate or severe tricuspid regurgitation (TR) 20.8%, and new atrial fibrillation (AF) 32.4%.

207 options for severe tricuspid regurgitation (TR) are limited, and additional interventional approache

208 e exercise testing, tricuspid regurgitation (TR) Doppler estimates and invasive measurement of pulmon

209 ients with isolated tricuspid regurgitation (TR) in the absence of left-sided valvular dysfunction ar

210 Severe tricuspid regurgitation (TR) is associated with high morbidity and mortality rate

211 Tricuspid regurgitation (TR) is common among adults with corrected tetralogy of F

212 The presence of tricuspid regurgitation (TR) may affect prognosis in patients with mitral regurgi

213 Functional tricuspid regurgitation (TR) with a structurally normal tricuspid valve (TV) may

214 oderate and severe) tricuspid regurgitation (TR), the decision to intervene is influenced by right ve

215 in silico deletion to find the most relevant TRs.

216 ntromeres made up of the minor tandem repeat TR-1.

217 Tandem repeat (TR) expansions have been implicated in dozens of genetic

218 aining glycopeptides from the tandem repeat (TR) sequence of MUC4, an aberrantly expressed mucin in p

219 n of replication within the terminal repeat (TR) region.

220 LANA binds the KSHV terminal-repeat (TR) sequence through its carboxy-terminal domain to medi

221 LANA binds the KSHV terminal-repeat (TR) sequence to mediate DNA replication and tethers KSHV

222 In particular, tandem repeats (TRs) (i.e. short, almost identical sequences that lie ad

223 repeat regions such as the terminal repeats (TRs).

224 DNA trinucleotide repeats (TRs) can exhibit dynamic expansions by integer numbers o

225 engineered human APOE targeted replacement (TR) mice following repeated mild TBI (rmTBI) using a lat

226 tion in human and APOE-targeted replacement (TR) transgenic mice.

227 nd mitotic chromosomes or bind and replicate TR DNA.

228 Tiger Reserves (TRs) are India's PAs with highest protection and managem

229 Tissue-resident (TR) alveolar macrophages (APhi) are long-lived, self-ren

230 small, possibly due to existence of residual TR and the large reduction in TV opening area.

231 Treatment-resistant (TR) ER+ cell lines (MCF-7:5C and MCF-7:TAM1) were used f

232 Among them, time-resolved (TR) resonance Raman (RR) is a particularly versatile too

233 Tooth resorption (TR) in domestic cats is a common and painful disease cha

234 owth efficacy as 'high therapeutic response (TR (+))' and 'low therapeutic response (TR (-))'.

235 nse (TR (+))' and 'low therapeutic response (TR (-))'.

236 at the commercial harvest (CH) or tree-ripe (TR) stages were immediately exposed to cold treatment (4

237 rriers, genomically extended telomerase RNA (TR) accumulated at the expense of mature TR, consistent

238 The sensitive imaging of telomerase RNA (TR) in living cells is crucial for improved guidance in

239 in 1292 patients with significant secondary TR (median age 71 [62-78]; 50% male).

240 eling in patients with significant secondary TR and to investigate its prognostic implications.

241 RV remodeling in significant secondary TR has been underexplored.

242 In patients with significant secondary TR, patients with RV systolic dysfunction have worse cli

243 ained the ability to replicate and segregate TR DNA, suggesting a unique role for the residues.

244 ained the ability to replicate and segregate TR DNA, the two principal components of episome persiste

245 Thresholds for mild, moderate, and severe TR were derived based on natural history outcome data.

246 of 3,276 adult patients with isolated severe TR from November 2001 to March 2016.

247 d TR, comparing survival for isolated severe TR patients who underwent surgery with those who did not

248 Of 3,276 patients with isolated severe TR, 171 (5%) underwent tricuspid valve surgery, includin

249 In patients with isolated severe TR, surgery is not associated with improved long-term su

250 ld TR) (n = 501 [83.6%]) and moderate/severe TR (>=Mod TR) (n = 98 [16.4%]).

251 2 patients with TOF or PS and mild to severe TR who underwent isolated PVR (66.8%) or PVR+TVI (33.2%)

252 In patients with TOF or PS and significant TR, concomitant TVI is safe and results in better early

253 tive to two velocity regimes within a single TR, resulting in a shorter imaging time compared with th

254 The SERS enhancement factors of single TR dye molecules located in the conjunction region in di

255 d angle-resolved photoemission spectroscopy (TR-ARPES).

256 were analyzed in the backgrounds of strains TR and Merlin (ME).

257 m, the Time-Resolved Hydrodynamic Stretcher (TR-HS), which allows the investigation of the dynamic me

258 Strong TR peptide ligands from platelet GP1balpha and G-protein

259 solved X-ray Absorption Near Edge Structure (TR-XANES).

260 owning and drying in about half of the study TRs, with these trends often being similar or worse than

261 agnosed with moderate to severe, symptomatic TR in the absence of untreated left-heart disease and de

262 The fact that TR-1 repeats often co-occur with knob180 repeats suggest

263 dine-producing strains by FACS, showing that TR engineering towards a more focused ligand spectrum ca

264 al methods, we formalized the structure that TRs induce in dot-plot matrices where a sequence is comp

265 These results suggest that TRs' elevated protection alone may be insufficient to pr

266 The TR secondary structure is believed to play a role in CAG

267 The TR-HS integrates high-speed imaging and computer vision

268 ow concentration were used for attaching the TR adducts to the SERS substrate.

269 many of the genomic changes mediated by the TR in hypothyroidism are independent of NCoR1, suggestin

270 ormation was experimentally confirmed in the TR and viral Bcl-2 promoter of Kaposi sarcoma-associated

271 enerated knockin mice with a mutation in the TR DNA-binding domain that abrogates binding to DNA and

272 nce chemicals were readily identified in the TR transactivation assay, but only a single novel, direc

273 Further, FD values in the TR were negatively correlated with the severity of posit

274 sence of enzymatic activity, increase in the TR-FRET signal between the biotin-bound Eu(III)-labeled

275 Here, we solve the structure of the TR LysG of Corynebacterium glutamicum, which detects all

276 reverse the ligand-independent action of the TR on many gene targets and did not fully rescue the hig

277 Systemic and ocular treatment with the TR antagonists NH-3 and 1-850 increased cone density by

278 CoR1DeltaID), which cannot interact with the TR, in Pax8-KO mice, which make no thyroid hormone.

279 ting the complex, 3' overhang regions of the TRs were labeled with three distinct monolayer double-st

280 In 8 of these TRs analysed for change from before to after declaration

281 ose To validate a new short repetition time (TR) adiabatic inversion recovery (STAIR) prepared UTE (S

282 epair and/or recombination may contribute to TR expansion.

283 vine cardiomyocytes, with the sensitivity to TR blockade being age-dependent.

284 -resolved Forster resonance energy transfer (TR-FRET) detection method for PTMs of cysteine residues

285 lved fluorescence resonance energy transfer (TR-FRET) to study structural changes in CaM that may pla

286 -resolved Forster resonance energy transfer (TR-FRET).

287 lved luminescence resonance energy transfer (TR-LRET) was developed for the detection of beta-amyloid

288 suggests that the current role of the TRKIN/TR-1 system is to facilitate the meiotic drive of the KI

289 Regardless of surgery type, TR decreased by at least 1 echocardiographic grade in 35

290 Using TR antagonists and Thrb2 deletion, we examined the effec

291 Using TR ChIP-seq peaks or imputed TR binding sites, Lisa prob

292 yl-1,10-phenanthroline)(2)](+) complex using TR-XANES.

293 The various TR RR spectroscopic methods can cover a wide dynamic ran

294 3' end maturation mechanism that vertebrate TR shares with replication-dependent histones.

295 Thus, the question of whether TRs are an important screening target for protection of

296 ZCCHC8 associated with TR and was required for telomerase function.

297 d kinesin that specifically colocalizes with TR-1 in meiosis, forming long drawn out neocentromeres.

298 Our results suggest that in patients with TR secondary to AF, TV annuloplasty should be effective

299 3) have been changed from 'RSLR(crit) = (m x TR(e)) x Sed + i' and 'Sed(crit) = (RSLR - i)/(m x TR(e)

300 x Sed + i' and 'Sed(crit) = (RSLR - i)/(m x TR(e))', respectively. The definition of the variables i