ライフサイエンスコーパス: Torpedo

1 c acetylcholine receptor-rich membranes from Torpedo.

2 plex and provides an entry site for the XRN2 torpedo.

3 as performed too, after X-ray irradiation of torpedoes.

4 with torpedoes versus Purkinje cells without torpedoes.

5 ted shows 94% homology (86% identity) to the Torpedo 87 kDa protein and 50% homology to the cysteine-

6 In addition, antibodies against either the Torpedo 87 kDa protein or human dystrobrevin demonstrate

7 utrophin/dystrophin related protein, and the Torpedo 87K protein.

8 ibody 383C binds to the alpha subunit of the Torpedo acetylcholine (ACh) receptor as well as to its V

9 t a refined model of the membrane-associated Torpedo acetylcholine (ACh) receptor at 4A resolution.

10 olesterol on the ion-channel function of the Torpedo acetylcholine receptor (nAChR) and the novel lip

11 Immunization with Torpedo acetylcholine receptor (TAChR) induces experimen

12 induced in C57Bl/6 mice by immunization with Torpedo acetylcholine receptor (TAChR), to demonstrate t

13 Mice expressing the Torpedo acetylcholine receptor alpha-chain as a neo-self

14 el gating by using a structural model of the Torpedo acetylcholine receptor at 4-A resolution, record

15 Based on the Torpedo acetylcholine receptor structure, Unwin and coll

16 lity of residues alpha187 to alpha199 of the Torpedo acetylcholine receptor with monoclonal antibody

17 The mice were immunized with Torpedo acetylcholine receptor, and EAMG was assessed by

18 ns with identical peptide/MHC specificity in Torpedo acetylcholine receptor-alpha-transgenic animals

19 modified to either F (KI = 0.6 microM, as in Torpedo acetylcholinesterase) or Y (KI = 0.032 microM, a

20 For Torpedo AChE, a K(S) of 0.5+/- 0.2 mM obtained from subs

21 the carboxyl terminus in comparison with the Torpedo AChE, and three of the 14 aromatic residues that

22 l by measuring the inhibition constants with Torpedo AChE, fetal bovine serum AChE, human butyrylchol

23 ds differently to human AChE than it does to Torpedo AChE.

24 el was generated in which the immunodominant Torpedo AChR (T-AChR) alpha subunit is expressed in appr

25 d in C57BL6 (B6) mice by three injections of Torpedo AChR (TAChR) in adjuvant.

26 ockout mutants (IFN-gamma-/-, IL-12-/-) with Torpedo AChR (TAChR).

27 a previously unsuspected interaction between Torpedo AChR and the adaptor protein Grb2.

28 gene knockout (KO) mice were immunized with Torpedo AChR in CFA.

29 ha-bungarotoxin-bound AChR, (e) binds to the Torpedo AChR in either state mainly by an entropy-driven

30 es at the ACh binding sites of the mouse and Torpedo AChR shows mouse residue Ile-gamma116 as likely

31 this change does not affect affinity for the Torpedo AChR sites.

32 that TG mice had reduced activation of anti-Torpedo AChR Th1 cells, but increased anti-AChR Ab synth

33 ne to the desensitized/carbamylcholine-bound Torpedo AChR with higher affinity compared to the restin

34 that (i) CrV interacts with the desensitized Torpedo AChR with higher affinity than with the resting

35 e equilibrium binding affinity of [3H]ACh to Torpedo AChR-rich membranes.

36 ng affinity to the mouse muscle AChR and the Torpedo AChR.

37 eferred the desensitized conformation of the Torpedo AChR.

38 reversibly inhibited ACh-induced currents of Torpedo AChRs expressed in Xenopus oocytes.

39 d maintenance of Delta expression by veinlet/torpedo activity.

40 muscle and a neuronal nAChR, we photolabeled Torpedo alpha(2)betagammadelta and expressed human alpha

41 to characterize steroid-binding sites in the Torpedo alpha(2)betagammadelta nAChR in its native membr

42 ow-to-high affinity transition (L->H) at the Torpedo alpha-delta nicotinic acetylcholine receptor neu

43 4-191 with the corresponding region from the Torpedo alpha1 subunit, we introduced a cluster of five

44 and Phe227 at the lipid-exposed face of the Torpedo alpha1M1 segment.

45 ABAAR potentiator, has been shown to inhibit Torpedo alpha2betagammadelta nAChRs, binding in the ion

46 its human muscle (alphabetaepsilondelta) and Torpedo (alphabetagammadelta) nAChR expressed in Xenopus

47 of mouse AChR, whereas the affinity for the Torpedo alphagamma-site was slightly increased.

48 e photolabeled alpha4Tyr(195) (equivalent to Torpedo alphaTyr(190)) in Segment C as well as beta2Val(

49 potential energy profiles are seen when the Torpedo and alpha 7 models are compared.

50 o, cell membranes from the electric organ of Torpedo and from the rat brain were transplanted to frog

51 non-hair identity in the transition between torpedo and mature stage.

52 alphaA-conotoxins EIVA and EIVB, block both Torpedo and mouse alpha1-containing muscle subtype nAChR

53 Both psi-conotoxins block Torpedo and mouse nicotinic acetylcholine receptors (nAC

54 hose mediated by the transmembrane receptors Torpedo and Notch.

55 frog oocytes, which thus acquired functional Torpedo and rat neurotransmitter receptors.

56 oned in humans; COLQ cDNA has been cloned in Torpedo and rodents but not in humans.

57 d dropout of Purkinje cells, Purkinje axonal torpedoes and Bergmann gliosis.

58 levels of IFs and abnormal organelles in the torpedoes and soma of Purkinje cells, as well as in the

59 egraded by 5' --> 3' exonuclease activities (torpedoes) and so induces dissociation of Pol II from th

60 etion of Purkinje cells, with empty baskets, torpedoes, and astrogliosis characterized by a disorgani

61 These torpedoes are abnormal swellings of Purkinje cell axons

62 tem studies, Purkinje cell axonal swellings (torpedoes) are present to a greater degree in essential

63 exin resulted in the formation of cerebellar torpedoes as early as 1 month of age.

64 ell epitope within residues alpha 146-162 of Torpedo californica (t), tAChR, plays an important role

65 Cocrystallization of the enzyme from Torpedo californica (TcCK) with ADP-Mg(2+), nitrate, and

66 n the C57BL/6 background were immunized with Torpedo californica acetylcholine receptor (AChR) and ev

67 ipid-exposed transmembrane segment M4 of the Torpedo californica acetylcholine receptor (AChR) focuse

68 n mice, EAMG is induced by immunization with Torpedo californica acetylcholine receptor (AChR) in com

69 e assessed in transgenic mice expressing the Torpedo californica acetylcholine receptor (TAChR) alpha

70 h the aged and nonaged crystal structures of Torpedo californica acetylcholinesterase inhibited by th

71 For Torpedo californica acetylcholinesterase, monomeric and

72 Although BSF inhibits both mouse and Torpedo californica AChE, PMSF does not react measurably

73 y structure of a transition-state complex of Torpedo californica AChE-m-(N,N,N-trimethylammonio)-2,2,

74 long-chain alkanol sites on the desensitized Torpedo californica AChR and to investigate if these sit

75 fragments comprising residues 143-210 of the Torpedo californica alpha-subunit were expressed in E. c

76 o acid substitutions at the M4 domain of the Torpedo californica and mouse acetylcholine receptor sug

77 of the homologous acetylcholinesterase from Torpedo californica complexed with TMTFA (2.66 +/- 0.28

78 ent upon binding to AChR-rich membranes from Torpedo californica electric organ.

79 cotinic acetylcholine receptors (nAChR) from Torpedo californica electric tissue in their membrane-bo

80 ntial enrichment methodology and the AChR in Torpedo californica electroplax membranes were used to f

81 In this regard, we used the Torpedo californica nAChR and a series of barbiturate an

82 technique their interaction with the AChBP, Torpedo californica nAChR and chimeric receptor composed

83 s contributing to their binding sites in the Torpedo californica nAChR.

84 e and kinetics of azietomidate inhibition of Torpedo californica nAChRs and time-resolved photolabeli

85 as used to examine structural changes in the Torpedo californica nicotinic acetylcholine receptor (AC

86 r more transmembrane segments (M1-M4) of the Torpedo californica nicotinic acetylcholine receptor (AC

87 To further define the surface of the Torpedo californica nicotinic acetylcholine receptor (nA

88 oclonal antibodies (mAbs) on the function of Torpedo californica nicotinic acetylcholine receptor (nA

89 The lipid requirements of the Torpedo californica nicotinic acetylcholine receptor (nA

90 esterol) was used to identify domains in the Torpedo californica nicotinic acetylcholine receptor (nA

91 s EST is also highly homologous (90%) to the Torpedo californica post-synaptic 87 kDa phosphoprotein.

92 The AChR from Torpedo californica was labeled with a fluorescent probe

93 nicotinic acetylcholine receptor (AChR) from Torpedo californica were measured using sequential-mixin

94 nicotinic acetylcholine receptor (AChR) from Torpedo californica were used to determine binding chara

95 vesicles isolated from the electric organ of Torpedo californica, a model cholinergic synapse, contai

96 ith heterologous AChR from the electric fish Torpedo californica, has been used extensively.

97 nicotinic acetylcholine receptor (AChR) from Torpedo californica.

98 les were isolated from the electric organ of Torpedo californica.

99 an the nicotinic acetylcholine receptor from Torpedo californica.

100 teins (nicotinic acetylcholine receptors) of Torpedo cholinergic membrane.

101 hloride channel belonging to the CIC family (Torpedo CIC-0) has functional features that suggest that

102 napologetically subjective essay recalls the Torpedo Cl(-) channel in the years when it had neither a

103 The Drosophila gene torpedo/Egfr (top/Egfr) encodes a homolog of the vertebr

104 ng oocyte and is thought to locally activate torpedo/Egfr (top/Egfr), the Drosophila homolog of the E

105 nicotinic acetylcholine receptor (AChR) from Torpedo electric organ and mammalian muscle contains hig

106 In addition, we isolated MuSK from Torpedo electric organ and used nanoelectrospray tandem

107 odot assays, synaptic vesicles purified from Torpedo electric organ are also immunoreactive for PMCA

108 he nicotinic acetylcholine receptor from the Torpedo electric organ has long been recognized, and one

109 nicotinic acetylcholine receptor (nAChR) in Torpedo electric organ membranes.

110 ]APFBzcholine with nAChR-rich membranes from Torpedo electric organ revealed equal affinities (K(eq)

111 ed on dystroglycan and other constituents of Torpedo electric organ synaptic membranes.

112 choline receptor (nAChR)-rich membranes from Torpedo electric organ with [(14)C]halothane and determi

113 choline receptor (nAChR)-rich membranes from Torpedo electric organ with a photoactivatable analog, [

114 rs as a single 2.4 kb transcript abundant in Torpedo electric organ, moderately expressed in spinal c

115 f ClC-0, a chloride (Cl(-)) channel from the Torpedo electric organ.

116 ding partners in postsynaptic membranes from Torpedo electric organ.

117 e of electrocytes in isolated columns of the Torpedo electric organ.

118 EM image of the transmembrane domain of the torpedo electric ray nicotinic channel, we were provided

119 The native receptor was purified from Torpedo electric tissue and functionally reconstituted i

120 The immobilization of Torpedo electrocyte membranes on the surface of micropla

121 method development was the immobilization of Torpedo electrocyte membranes rich in nicotinic acetylch

122 ized with the AChR on the innervated face of Torpedo electrocytes.

123 ously identified noncompetitive inhibitor of Torpedo electroplax nAChR, also isolated from C. purpura

124 Studies on Torpedo electroplax nicotinic acetylcholine (ACh) recept

125 otinic acetylcholine receptors (nAChRs) from Torpedo electroplax, using (19)F nuclear magnetic resona

126 n AChE) increased the K(S) to 4-10 mM in the Torpedo enzyme and to about 33 mM in the human enzyme.

127 d Bcl-X(L) was abrogated in X-ray irradiated torpedo erythrocytes.

128 nscription termination and regulation of the torpedo exonuclease Rat1p.

129 ends of genes, and regulates the activity of torpedo exonuclease Rat1p.

130 ors phosphorylated by Cdk9 was the 5'-to-3' "torpedo" exoribonuclease Xrn2, required in transcription

131 nd beta2Ser(113) in Segment E (equivalent to Torpedo gammaLeu(109) and gammaTyr(111), respectively).

132 rmal looking seeds that were arrested in the torpedo growth stage.

133 e and the Drosophila EGF receptor homologue, Torpedo, in the surrounding somatic follicle cells.

134 etylcholine receptor from the electric fish, Torpedo, is the prototypic ligand-gated ion channel, yet

135 o mechanism whereby a 5'-3' RNA exonuclease (torpedo) latches itself onto the 5' end of RNA protrudin

136 ration of the neurosensory retina within the torpedo lesion.

137 56]), nummular pigmentation (85.7% [48/56]), torpedo-like lesions (10.7% [6/56]), and circumferential

138 sis of the macula or peripapillary area and "torpedo-like" lesions along the vascular arcades may als

139 ew clinical observations in ESCS include (1) torpedo-like, deep atrophic lesions with a small hyperpi

140 Torpedo maculopathy (TM) is a rare, congenital condition

141 l hypoplasia is a phenocopy of grade 1 NCMD, torpedo maculopathy is a phenocopy of grade 2 NCMD, and

142 ckled hyper-AF lesions surrounding right eye torpedo maculopathy site and hyper-AF lesions in the lef

143 Two patients with torpedo maculopathy were examined at baseline and then a

144 with congenital ZIKV infection: 1 child had torpedo maculopathy, 1 child had a chorioretinal scar wi

145 atrophy that now can be expanded to include torpedo maculopathy, vascular changes, and hemorrhagic r

146 ray irradiation of nucleated erythrocytes of Torpedo marmorata and Caretta caretta and the effect of

147 nts can be suppressed by the introduction of Torpedo marmorata CLC-0 or Arabidopsis thaliana CLC-c an

148 was developed based on the recently refined Torpedo marmorata nACh receptor.

149 BP) and the transmembrane (TM) domain of the Torpedo marmorata nAChR.

150 ves were considered as binding proteins, the Torpedo marmorata nicotinic acetylcholine receptor (nACh

151 ys-loop receptors based on homology with the Torpedo marmorata nicotinic acetylcholine receptor infer

152 eems to play a role in the erythropoiesis of Torpedo marmorata Risso and in Caretta caretta.

153 of the nicotinic acetylcholine receptor from Torpedo marmorata, reveals an asymmetric ion channel wit

154 receptor isolated from the electric organ of Torpedo marmorata.

155 ion on nucleated circulating erythrocytes of Torpedo marmorata.

156 stone and U snRNA genes, suggesting that the torpedo mechanism is not limited to poly(A) site-depende

157 Then, we explore in detail the torpedo mechanism whereby a 5'-3' RNA exonuclease (torpe

158 Our results suggest a combined allosteric/torpedo mechanism, in which PP1-dependent slowing down o

159 under normal conditions by an Xrn2-mediated torpedo mechanism.

160 ranscripts and premature termination by the "torpedo" mechanism is a widespread mechanism that limits

161 o a model for termination, the "sitting duck torpedo" mechanism, where poly(A) site-dependent deceler

162 However recent in vivo studies revealed a 'torpedo' mechanism for Pol I termination: co-transcripti

163 subunits and for other protein components in Torpedo membrane preparations, such as RAPsyn and Na(+)-

164 roperties measured both in flux studies with Torpedo membrane vesicles and by single-channel analysis

165 ne photoaffinity labeling of both the native Torpedo membranes and the isolated nAChR was saturable,

166 Native Torpedo membranes contain approximately 35 mol % CH but

167 To seek such sites, Torpedo membranes enriched in nicotinic acetylcholine re

168 , anionic lipids that are abundant in native Torpedo membranes, also stabilize the receptor in the re

169 of ice-embedded "giant" tubular crystals of Torpedo membranes, which had been partially flattened to

170 inhibition of agonist-induced cation flux in Torpedo membranes.

171 nic acetylcholine receptor (nAChR) in native Torpedo membranes.

172 Our results suggest a unified allosteric/torpedo model in which Rat1 is not a dedicated terminati

173 A central untested feature of the torpedo model is that there is kinetic competition betwe

174 The torpedo model of transcription termination asserts that

175 The torpedo model of transcription termination by RNA polyme

176 scriptional termination, as envisaged in the torpedo model.

177 rase II via the nascent transcript-i.e., the torpedo model.

178 ion of the original cryo-electron microscopy Torpedo model; the only pentameric ligand-gated ion chan

179 is model complements the current "allosteric-torpedo" model of transcription termination, and could e

180 An integrated "allosteric-GENEi-torpedo" model that could explain this paradox predicts

181 degrading nascent RNA revising the current 'torpedo' model of termination, which posits that RNA deg

182 An alternative 'torpedo' model postulated that poly(A) site cleavage pro

183 The allosteric and torpedo models have been used for 30 yr to explain how t

184 characterize mTFD-MPAB interactions with the Torpedo (muscle-type) nAChR.

185 To identify regions of the Torpedo Na,K-ATPase alpha-subunit that interact with mem

186 2)]-n-pentyldiazirine photoincorporated into Torpedo nAcChoR-rich membranes mainly in the alpha subun

187 acts as a positive allosteric potentiator of Torpedo nACh receptor (nAChR) and binds to a novel site

188 nd beta1-Cys447 in the lipid-exposed face of Torpedo nAChR alpha1M4 and beta1M4, respectively.

189 ld decrease in the apparent affinity for the Torpedo nAChR and a corresponding 150-fold increase in t

190 are the state-dependent photolabeling of the Torpedo nAChR before and after purification and reincorp

191 The Torpedo nAChR delta-subunit residue corresponding to gam

192 es of the neuronal alpha4beta2 nAChR and the Torpedo nAChR display a high degree of structural homolo

193 olabeling studies using [3H]-3-azioctanol in Torpedo nAChR identified alphaE262 as a site of desensit

194 receptor (nAChR), have been localized in the Torpedo nAChR in the desensitized state by use of a phot

195 oligand binding assays, AziPm stabilized the Torpedo nAChR in the resting state, whereas propofol sta

196 ed the binding of [(3)H]phencyclidine to the Torpedo nAChR ion channel in the resting and desensitize

197 hotoincorporated into amino acids within the Torpedo nAChR ion channel with the efficiency of photoin

198 The rat anti-Torpedo nAChR mAbs examined here are known to inhibit li

199 lpha subunit interface is a binding site for Torpedo nAChR negative allosteric modulators (TFD-etomid

200 h that interactions with gamma Trp-55 of the Torpedo nAChR play a crucial role in agonist binding and

201 of the 5-HT3A and alpha4beta2 nAChRs against Torpedo nAChR revealed MA -4', 0', and 4' residues withi

202 tein) and of the electron-microscopy-derived Torpedo nAChR structure.

203 Our data indicate that the Torpedo nAChR transmembrane domain structure is a better

204 itization in the transmembrane domain of the Torpedo nAChR using time-resolved photolabeling with the

205 With the recent 4 A resolution of the Torpedo nAChR, and the crystal structure of the AChBP, m

206 With the recent 4 A resolution of the Torpedo nAChR, and the crystal structure of the related

207 For Torpedo nAChR, photoaffinity-labeling studies with the c

208 after purification and reconstitution of the Torpedo nAChR, the difference in structure between the r

209 interaction of a long chain alcohol with the Torpedo nAChR, we have used the photoactivatible alcohol

210 [(3)H]tetracaine and [(3)H]phencyclidine to Torpedo nAChR-rich membranes (IC(50) values of 0.8 mm).

211 annel and the lipid-protein interface of the Torpedo nAChR.

212 8-11, and 13-14 are essential for binding to Torpedo nAChR.

213 alent to the surface exposed to lipid in the Torpedo nAChR.

214 packed than the corresponding region of the Torpedo nAChR.

215 e receptor (GlyR), based on the structure of Torpedo nAChR.

216 hat of acetylcholine for embryonic mouse and Torpedo nAChRs expressed in Xenopus oocytes, respectivel

217 ontribute to the ACh binding sites in mutant Torpedo nAChRs expressed in Xenopus oocytes.

218 he interactions of agonists and antagonists, Torpedo nAChRs were expressed in Xenopus oocytes, and eq

219 uilibrium binding of ion channel blockers to Torpedo nAChRs with higher affinity in the nAChR desensi

220 scale of a rapid phase of TID inhibition in Torpedo nAChRs, suggesting the formation of a transient

221 were tested for inhibitory activity against Torpedo nAChRs.

222 e and frequency dependencies of MEPPs at the Torpedo nerve-electrocyte junction are best described by

223 GABA receptors were constructed based on the torpedo neuromuscular-like nicotinic receptor structure.

224 e of the alphaM3 transmembrane domain of the Torpedo nicotinic acetylcholine receptor (AChR) was char

225 peted with alpha-bungarotoxin for binding to Torpedo nicotinic acetylcholine receptor (nAChR) (IC(50)

226 ue Thr(422) at the lipid-exposed face of the Torpedo nicotinic acetylcholine receptor (nAChR) alpha1M

227 The activity of the muscle-type Torpedo nicotinic acetylcholine receptor (nAChR) is high

228 The Torpedo nicotinic acetylcholine receptor (nAChR) is the

229 Although the Torpedo nicotinic acetylcholine receptor (nAChR) reconst

230 acaine is a noncompetitive antagonist of the Torpedo nicotinic acetylcholine receptor (nAChR) that bi

231 C) is a potent competitive antagonist of the Torpedo nicotinic acetylcholine receptor (nAChR) that bi

232 Interactions of benzophenone (BP) with the Torpedo nicotinic acetylcholine receptor (nAChR) were ch

233 Photoaffinity labeling of the Torpedo nicotinic acetylcholine receptor (nAChR) with [3

234 rientation compatible with activation of the Torpedo nicotinic acetylcholine receptor (nAChR), we use

235 ster within the agonist binding sites of the Torpedo nicotinic acetylcholine receptor (nAChR).

236 synthesized as a photoaffinity probe for the Torpedo nicotinic acetylcholine receptor (nAChR).

237 atic amine noncompetitive antagonists in the Torpedo nicotinic acetylcholine receptor (nAChR).

238 er peptide, spanning residues 181-198 of the Torpedo nicotinic acetylcholine receptor alpha1 subunit,

239 id agonist [(3)H]arecolone methiodide to the Torpedo nicotinic acetylcholine receptor has been correl

240 ignificantly, ultrastructural studies of the Torpedo nicotinic acetylcholine receptor indicate that t

241 ls of the open state of the heteropentameric Torpedo nicotinic acetylcholine receptor pore domain are

242 ting into some, but not all, subunits of the Torpedo nicotinic acetylcholine receptor to a degree tha

243 ical pentameric ligand-gated ion channel the Torpedo nicotinic acetylcholine receptor(10,11), the lar

244 (+)-epibatidine, and (+/-)-epibatidine, with Torpedo nicotinic acetylcholine receptor-enriched membra

245 [(3)H]Azietomidate photoincorporated into Torpedo nicotinic acetylcholine receptor-rich membranes.

246 nferred from the structure of the homologous Torpedo nicotinic acetylcholine receptor.

247 olve apo and agonist-bound structures of the Torpedo nicotinic receptor embedded in a lipid nanodisc.

248 mine neurotoxins tightly bound to the coated Torpedo nicotinic receptor were eluted with methanol, an

249 t biotinylated-alpha-bungarotoxin binding to Torpedo-nicotinic acetylcholine receptors in a concentra

250 tps1 embryos do not develop past late torpedo or early cotyledon stage.

251 d shown by peptide microsequencing to be the Torpedo ortholog of the small leucine-rich repeat chondr

252 estored by the introduction of rat MuSK or a Torpedo orthologue.

253 hape [thickened axonal profiles (P = 0.006), torpedoes (P = 0.038)] and changes in axonal connectivit

254 ere were approximately 7x more Purkinje cell torpedoes per section (12.6 +/- 7.9 versus 1.7 +/- 1.4,

255 tron tomography and subtomogram averaging of Torpedo postsynaptic membrane that receptors are connect

256 y electron microscopy of tubular crystals of Torpedo postsynaptic membranes embedded in amorphous ice

257 The "torpedo" Rat1-Rai1 exonuclease (XRN2 in humans) greatly

258 Mutations of this glutamate residue in Torpedo ray ClC channels alter gating in electrophysiolo

259 ic receptors using a structural model of the Torpedo receptor at 4 A resolution, recordings of curren

260 ctron microscopic study of the non-activated Torpedo receptor had suggested that these sites might be

261 ine the cryo-EM structure of the muscle-type Torpedo receptor in complex with ScNtx, a recombinant sh

262 enesis, spatially restricted activity of the TORPEDO receptor tyrosine kinase first recruits follicle

263 tudied further by single-channel analysis of Torpedo receptors reconstituted in giant liposomes.

264 F, corresponding to positions 290 and 331 in Torpedo) rendered the enzyme 10-fold less sensitive to e

265 Previously, cryo-EM of intact membrane from Torpedo revealed that the lipid bilayer surrounding the

266 Catch and consumption of torpedo scad (Megalaspis cordyla) over the western India

267 g information on the population structure of torpedo scad stocks it is crucial to provide population

268 2 seeds were nonviable and developed only to torpedo-shaped embryos, indicative of arrested seed embr

269 One set belongs to the gurken-torpedo signaling pathway and affects the development of

270 are noncompetitive antagonists (NCAs) of the Torpedo species nicotinic acetylcholine receptor (nAChR)

271 was observed in the embryo from seeds at the torpedo stage and later, in seedling, leaf, stem, and ro

272 detected in the endosperm from seeds at the torpedo stage and later.

273 d throughout the root epidermal layer in the torpedo stage embryo when the cell-specific pattern of G

274 tion undergoes a marked reduction at the mid-torpedo stage of Arabidopsis embryogenesis.

275 embryos that were arrested at or before the torpedo stage of development.

276 traffic 10-kD fluorescent dextran in the mid-torpedo stage of development.

277 In the torpedo stage of embryo development, ATML1 mRNA disappea

278 ze exclusion limit was found to occur at the torpedo stage of embryogenesis in Arabidopsis; at this t

279 pproximately 1/4 of which aborted before the torpedo stage, suggesting that fab1-2 represents a compl

280 embryo within them was arrested at the heart-torpedo stage.

281 ed with the activities of WER and CPC during torpedo stage.

282 , that maintain dilated plasmodesmata at the torpedo stage.

283 gy and carbon metabolism and abortion at the torpedo stage.

284 gene expression to a profile resembling late-torpedo-stage embryogenesis.

285 trastructure of PD in early-, mid-, and late-torpedo-stage embryos and in young leaves.

286 half that of the wild type at the heart and torpedo stages.

287 embryo development arrested from globular to torpedo stages.

288 chick cerebellum, chick ciliary ganglia, and Torpedo synaptic vesicles.

289 Following immunization with Torpedo (t) AChR, the IL-4(-/-) mice readily developed s

290 to track in a 5'-3' direction like a guided torpedo that ultimately helps dissociate the RNA polymer

291 te with Xrn2, the nuclear 5'-3' exonuclease "torpedo" that facilitates transcription termination at t

292 nt in cesa9 embryos, visually inspected from torpedo to bent cotyledon, consistent with no reduction

293 gurken (grk), a TGFalpha-like protein, with torpedo (top), the Drosophila EGF receptor (Egfr).

294 tly seen on the axons of Purkinje cells with torpedoes versus Purkinje cells without torpedoes.

295 On the basis of Torpedo vesicle studies, TID is thought to selectively i

296 Torpedo vesicles are negative for the sarcoplasmic/ endo

297 vesicles isolated from the electric organ of Torpedo were determined using a pH-jump protocol.

298 ar TBP-positive immunoreactivity, and axonal torpedos were readily found by light and electron micros

299 nhibition of the macroscopic response of the Torpedo wild type of about 52%, whereas the alpha C418W

300 The exonuclease torpedo Xrn2 loads onto nascent RNA 5'-PO(4) ends and ch