戻る
「早戻しボタン」を押すと検索画面に戻ります。 [閉じる]

コーパス検索結果 (１語後でソート)

通し番号をクリックするとPubMedの該当ページを表示します
1 ctor phase of a secondary immune response to Trichinella spiralis.
2 zing hepatitis following oral infection with Trichinella spiralis.
3 racterize cellular responses to muscle-stage Trichinella spiralis.
4 yperplasia in the mouse after infection with Trichinella spiralis.
5 the course (0 to 289 h) of an infection with Trichinella spiralis.
6         We report a draft genome sequence of Trichinella spiralis, a food-borne zoonotic parasite, wh
7              The mouse is a natural host for Trichinella spiralis, a worm that establishes chronic in
8 polygyrus, Nippostrongylus brasiliensis, and Trichinella spiralis) alters intestinal epithelial cell
9 ated with significantly delayed expulsion of Trichinella spiralis and increased deposition of muscle
10 pulsion of two other gut-dwelling nematodes (Trichinella spiralis and Nippostrongylus brasiliensis).
11 due were detected in the parasitic nematodes Trichinella spiralis and Trichinella pseudospiralis.
12 p necrotizing hepatitis after infection with Trichinella spiralis, and inflammation is dependent on t
13            We used a natural mouse parasite, Trichinella spiralis, and multipoint intravital time-lap
14 tant strains) and the muscle-stage larvae of Trichinella spiralis as a representative parasitic nemat
15 MMC) recruitment coincides with expulsion of Trichinella spiralis, at a time when the majority of the
16 is provided here that the parasitic nematode Trichinella spiralis can catalyze the conversion and thu
17  Although the forthcoming genome sequence of Trichinella spiralis can provide invaluable comparative
18 ection of mammalian skeletal muscle cells by Trichinella spiralis causes host nuclei to become polypl
19                                              Trichinella spiralis creates a unique intracellular habi
20       The L1 stage of the parasitic nematode Trichinella spiralis displays on its surface glycoprotei
21 pond vigorously to intestinal infection with Trichinella spiralis, eliminating a role for T-bet in MC
22 rimary infection with the parasitic nematode Trichinella spiralis, eosinophils play an important immu
23                   Because mice infected with Trichinella spiralis experience a pronounced, but transi
24 hown to have a critical role in clearance of Trichinella spiralis from the intestinal tract.
25 le for the expulsion of the related nematode Trichinella spiralis from these animals.
26 hanced expulsion of the intestinal nematode, Trichinella spiralis, from the small intestine.
27 at wild boar meat products contaminated with Trichinella spiralis had entered the food chain in Germa
28 te mitochondrial DNA (mtDNA) of the nematode Trichinella spiralis has been amplified in four overlapp
29     The antibody response to the L1 stage of Trichinella spiralis has been described as biphasic.
30           Studies with rodents infected with Trichinella spiralis, Heligmosomoides polygyrus, Nippost
31 ed in the survival of the parasitic nematode Trichinella spiralis in an intracellular environment are
32 on triggered by the intraepithelial nematode Trichinella spiralis in jejunal epithelium from BALB/c m
33  of acute inflammation during development of Trichinella spiralis in the muscle.
34 Infection of mice with the nematode parasite Trichinella spiralis induces changes in the proteome of
35 tinal infection with the parasitic nematode, Trichinella spiralis, induces a pronounced eosinophilia
36 anthelmintic properties were evaluated using Trichinella spiralis infected mice.
37 in were observed in the jejunal submucosa of Trichinella spiralis-infected BALB/c mice.
38               Jejunal segments isolated from Trichinella spiralis-infected mice were used to assess 5
39  mice and in mesenteric lymph nodes (mLN) of Trichinella spiralis-infected mice.
40                 Previous work has shown that Trichinella spiralis-infected rats transport IgE from pl
41                                              Trichinella spiralis infection elicits a vigorous IgE re
42 hat robust induction of IL4 responses during Trichinella spiralis infection enhance the presence of n
43                                              Trichinella spiralis infection induces a strong type 2 c
44              We previously demonstrated that Trichinella spiralis infection inhibits host inducible N
45                                        After Trichinella spiralis infection of mice, we show that bas
46 e mast cell responses and inflammation after Trichinella spiralis infection or the induction of food
47                                              Trichinella spiralis is a highly destructive parasitic n
48                                              Trichinella spiralis is an obligate parasite of animals
49   Expulsion of the gastrointestinal nematode Trichinella spiralis is associated with pronounced masto
50        Infection with the parasitic nematode Trichinella spiralis is initiated when the L1 larva inva
51  parasites, Nippostrongylus brasiliensis and Trichinella spiralis, is similar in that both require IL
52 inophils protect intracellular, muscle-stage Trichinella spiralis larvae against NO-mediated killing.
53                                              Trichinella spiralis larvae were identified in wild boar
54 ccompanying the expulsion of the GI parasite Trichinella spiralis may be dependent on IL-4 and mediat
55                               Infection with Trichinella spiralis rarely leads to significant morbidi
56 olate-induced peritonitis and infection with Trichinella spiralis, stimulated similar responses in Ga
57 metabolizing enzymes in secreted products of Trichinella spiralis suggests that endoparasites use sim
58                  In parasitic infection with Trichinella spiralis, the immune response incorporates b
59           We have used the parasite helminth Trichinella spiralis to study the generation and differe
60 y response caused by an intestinal parasite, Trichinella spiralis, to study the relationship between
61              We have surveyed the genomes of Trichinella spiralis, Trichuris muris, and Romanomermis
62          Infection of mice with the nematode Trichinella spiralis triggers recruitment and differenti
63 ulsion of another gastrointestinal nematode, Trichinella spiralis, unlike N. brasiliensis expulsion,
64 binant human chymase efficiently degrade the Trichinella spiralis virulence factor heat shock protein
65 n the larval stage of the parasitic nematode Trichinella spiralis was shown to invade epithelial cell
66  MC in the jejunum of BALB/c mice exposed to Trichinella spiralis were assessed during the course of
67 t it does not contribute to immunity against Trichinella spiralis, which lives within IEC.