ライフサイエンスコーパス: Trichomonas

1 mperature appears to affect the viability of Trichomonas.

2 In men with NGU, 19.9% were infected with trichomonas.

3 with a decreased risk of a positive test for trichomonas.

4 en were enrolled, and 16% were found to have Trichomonas (46/294).

5 amine the rates of gonorrhea, chlamydia, and trichomonas after US holidays, patient birthdays, and ot

6 nce of RNAs with a distinctive 5' DMG cap in Trichomonas and Giardia lineages that are absent in othe

7 eishmania, Neospora, Plasmodium, Toxoplasma, Trichomonas and Trypanosoma.

8 hol-PP-GlcNAc2Man5 (present in Entamoeba and Trichomonas) and dolichol-PP- and N-linked GlcNAc2 (pres

9 Black race, a new sex partner, a history of trichomonas, and the presence of symptoms were associate

10 rate (62%), followed by Candida with 18% and Trichomonas at 9%.

11 transmitted diseases clinic were tested for trichomonas, chlamydia, and gonorrhea.

12 Clinical isolates and mutant Trichomonas CPI-GC that had reduced affinity to galectin

13 Live Trichomonas depleted the extracellular levels of galecti

14 The prevalence of Trichomonas determined by culture was 5% (15 of 300 spec

15 a indicate that the thioredoxin reductase of Trichomonas differs fundamentally in structure from that

16 enzymes of Plasmodium spp., Trypanosomatida, Trichomonas, Entamoeba and Giardia, with special emphasi

17 n related species such as the avian parasite Trichomonas gallinae and cattle parasite Tritrichomonas

18 s amplified with DNA from Trichomonas tenax, Trichomonas gallinae, Chlamydia trachomatis, Neisseria g

19 nabled the spread of the protozoan parasite, Trichomonas gallinae, from columbids to finches, leading

20 ly early branching eukaryotic lineages, like Trichomonas, Galpha is likely to function independently

21 For each of the three organisms, Trichomonas, Gardnerella, and Candida, positivity at eac

22 l four time points (0, 24, 48, and 72 h) for Trichomonas, Gardnerella, and Candida.

23 However, Trypanosoma and Trichomonas genomes predicted ER degradation-enhancing a

24 Pathogenic parasites of the Trichomonas genus are causative agents of sexually trans

25 by the recently discovered Tom36 receptor of Trichomonas hydrogenosomes, while not allowing for growt

26 guidelines for STIs, advising screening for Trichomonas in women entering correctional facilities.

27 1 acquisition (HR, 1.9; 95% CI, 1.1-3.1) and Trichomonas infection (HR, 1.8; 95% CI, 1.3-2.4).

28 7 days reduced the proportion of women with Trichomonas infection at 1 month test of cure compared w

29 The rates of trichomonas infection in US males are lower than in wome

30 Trichomonas infection was more common in parous non-Hisp

31 ction of galectin-1 and -3 in the context of Trichomonas infection.

32 e gynecologic examination were evaluated for Trichomonas infection.

33 s associated with a 4-fold-increased risk of Trichomonas infection.

34 uire new gonorrhoea, syphilis, chlamydia, or trichomonas infections.

35 Trichomonas is a cause of nongonococcal urethritis (NGU)

36 Trichomonas is an amitochondriate parasitic protozoon sp

37 Thus via CPI-GC binding, Trichomonas is capable of regulating galectin bioavailab

38 lial clones, recombinant galectins, clinical Trichomonas isolates, and mutant protozoan derivatives t

39 ic Aptima Combo 2 (AC2) and Aptima TV (ATV), trichomonas microscopy, and culture.

40 galectin-1 suppressed chemokine responses to Trichomonas or CPI-GC/LPG.

41 east one curable STI (chlamydia, gonorrhoea, trichomonas, or high-titre syphilis) was 51% higher amon

42 positive test for gonorrhea, chlamydia, and trichomonas (p < 0.001 for all).

43 evalence 5.0% (3.8-6.7) and 8.4% (6.8-10.5), trichomonas prevalence 9.4% (7.7-11.5) and 12.2% (10.2-1

44 The percent sensitivity versus control for Trichomonas ranged from 100% at time zero with and witho

45 The OSOM Trichomonas Rapid Test (Genzyme Diagnostics, Cambridge,

46 The OSOM Trichomonas Rapid Test is a simple, objective test that

47 3.2- and 4.2-fold after treating Candida and Trichomonas, respectively.

48 ment of a protocol for differentiating among Trichomonas species that commonly infect humans.

49 or T. vaginalis and its avian sister species Trichomonas stableri, and assemblies of five other speci

50 red the prevalence of chlamydia, gonorrhoea, trichomonas, syphilis, and herpes simplex virus 2 (HSV-2

51 targeted product was amplified with DNA from Trichomonas tenax, Trichomonas gallinae, Chlamydia trach

52 chomonas hominis, Trichomonas vaginalis, and Trichomonas tenax.

53 specimen type routinely used for traditional trichomonas testing and the recommended specimen type fo

54 ction of chlamydia (CT), gonorrhea (GC), and trichomonas (TV).

55 terium Anabaena sp. (44%), and the protozoan Trichomonas vaginalis (46%), which targets its POR to an

56 there was some evidence of association with Trichomonas vaginalis (adjusted OR, 1.56; 95% CI, 1.00-2

57 eisseria gonorrhoeae (males and females) and Trichomonas vaginalis (females only) offered over 12 mon

58 l vaginosis (vaginal pH of 5.0 or above) and Trichomonas vaginalis (immunoassay) regardless of sympto

59 es inoculated with Chlamydia trachomatis and Trichomonas vaginalis (n = 9) or medium (controls; n = 7

60 rhoeae (nucleic acid amplification test) and Trichomonas vaginalis (rapid immunochromatographic assay

61 2 (HSV-2), syphilis, chlamydia, gonorrhoea, Trichomonas vaginalis (together defined as 'any STI') an

62 as been associated with an increased risk of Trichomonas vaginalis (TV) acquisition, it is unknown wh

63 oth prevalent and persistent infections with Trichomonas vaginalis (TV) are common.

64 omatis (CT), Mycoplasma genitalium (MG), and Trichomonas vaginalis (TV) are sexually transmitted infe

65 Trichomonas vaginalis (TV) can be infected with double-s

66 ), Neisseria gonorrhoeae (NG), syphilis, and Trichomonas vaginalis (TV) diagnosed in pregnancy among

67 f human immunodeficiency virus transmission, Trichomonas vaginalis (TV) infection constitutes an impo

68 n is affected by bacterial vaginosis (BV) or Trichomonas vaginalis (TV) infection has not been adequa

69 is (BV), vulvovaginal candidiasis (VVC), and Trichomonas vaginalis (TV) is not standardized.

70 Trichomonas vaginalis (TV) is the most common nonviral s

71 Trichomonas vaginalis (TV) results were compared relativ

72 All clinics used POC assays for Trichomonas vaginalis (TV) testing.

73 lagellar components required for the protist Trichomonas vaginalis (Tv) to swim through the human gen

74 Ureaplasma urealyticum biovar 2 (UU-2), and Trichomonas vaginalis (TV) using nucleic acid amplificat

75 ally transmitted infections (STIs) caused by Trichomonas vaginalis (TV), Chlamydia trachomatis (CT),

76 mammalian cells and protozoan parasites like Trichomonas vaginalis (Tv), the cause of the most common

77 sis (BV), vulvovaginal candidiasis (VVC), or Trichomonas vaginalis (TV), were randomly assigned to re

78 study assessed the performance of the cobas Trichomonas vaginalis (TV)/MG assay (cobas) for the dete

79 xidized form of the [2Fe-2S] ferredoxin from Trichomonas vaginalis (TvFd) are presented.

80 is (BV), vulvovaginal candidiasis (VVC), and Trichomonas vaginalis accounts for a significant proport

81 d Entamoeba histolytica, and the parabasalid Trichomonas vaginalis all belong to Class II of FBAs and

82 7,899 specimens submitted for live clinical Trichomonas vaginalis analyte-specific reagent (ASR) scr

83 o transcription-mediated amplification-based Trichomonas vaginalis analyte-specific-reagent (ASR) tes

84 s specific, since the related human parasite Trichomonas vaginalis and associated purified CPs did no

85 ption-mediated amplification (TMA) assay for Trichomonas vaginalis and BTUB FRET PCR, using self-obta

86 drial carrier family in the hydrogenosome of Trichomonas vaginalis and have shown that this protein,

87 icient in metronidazole-resistant strains of Trichomonas vaginalis and is thought to be necessary for

88 Additional Trichomonas vaginalis and Mycoplasma genitalium screenin

89 sence of a splicing apparatus in the protist Trichomonas vaginalis and show that RNA motifs found in

90 ent in the amitochondriate parasitic protist Trichomonas vaginalis and some but not all other trichom

91 Trichomonas vaginalis and viral pathogens (herpes simple

92 ing Giardia lamblia, Leishmania species, and Trichomonas vaginalis are persistently infected with dsR

93 omonas vaginalis prevalence using the Aptima Trichomonas vaginalis assay (ATV; Gen-Probe) and the pre

94 ts of a commercial NAA test (GenProbe Aptima Trichomonas vaginalis assay; ATV) for T. vaginalis were

95 clinic for a new complaint were screened for Trichomonas vaginalis by culture and by PCR analysis of

96 on as well as to determine the prevalence of Trichomonas vaginalis by culture in a large and diverse

97 amplification tests (NAATs) for detection of Trichomonas vaginalis by vaginal swabs; NAATs for detect

98 clinic for a new complaint were screened for Trichomonas vaginalis by wet-preparation (wet-prep) micr

99 Trichomonas vaginalis causes the most common, nonviral s

100 The human-infective parasite Trichomonas vaginalis causes the most prevalent nonviral

101 The protozoan Trichomonas vaginalis colonizes the human urogenital tra

102 Trichomonas vaginalis colonizes the urogenital tract of

103 The extracellular parasite Trichomonas vaginalis contains a surface glycoconjugate

104 thral swabs were obtained at enrollment, for Trichomonas vaginalis culture; semen specimens were also

105 n of macaques with Chlamydia trachomatis and Trichomonas vaginalis decreases the prophylactic efficac

106 determine if secreted cysteine proteases of Trichomonas vaginalis degrade SLPI and render it nonfunc

107 condary test in improving the sensitivity of Trichomonas vaginalis detection in young women over that

108 This study compared two assays for Trichomonas vaginalis detection, Gen-Probe's transcripti

109 obe was designed and evaluated for detecting Trichomonas vaginalis DNA in a 5' nuclease (TaqMan) assa

110 ydia trachomatis, Neisseria gonorrhoeae, and Trichomonas vaginalis DNA, detected using the BD MAX CT/

111 polymerase chain reaction (PCR) analysis for Trichomonas vaginalis DNA.

112 Infection with Trichomonas vaginalis during pregnancy has been associat

113 The crystal structure of the oxidized Trichomonas vaginalis ferredoxin (Tvfd) showed a unique

114 odified medium to InPouch for the culture of Trichomonas vaginalis from pooled vaginal secretions.

115 Two genes coding for Trichomonas vaginalis glucokinase were isolated and sequ

116 ase (PPi-PFK) of the amitochondriate protist Trichomonas vaginalis has been purified.

117 The utility of TMA for detection of Trichomonas vaginalis has recently been described.

118 tica, Leishmania spp., Trypanosoma cruzi and Trichomonas vaginalis have genes encoding homologues of

119 Nucleic acid amplification tests for Trichomonas vaginalis have improved sensitivity for dete

120 Giardia lamblia, Entamoeba histolytica, and Trichomonas vaginalis have robust oxygen consumption sys

121 plasma genitalium, Chlamydia trachomatis, or Trichomonas vaginalis in 10 patients (8%).

122 ydia trachomatis, Neisseria gonorrhoeae, and Trichomonas vaginalis in liquid-based cytology specimens

123 could also detect Mycoplasma genitalium and Trichomonas vaginalis in men and women reporting a histo

124 The impact of the viability of Trichomonas vaginalis in urine on wet mount, culture, an

125 Trichomonas vaginalis infected with a double-stranded RN

126 l SLPI levels have been correlated with both Trichomonas vaginalis infection and poor reproductive he

127 Trichomonas vaginalis infection in males has been largel

128 Trichomonas vaginalis infection in men is an important c

129 ed to wet mount and PCR for the diagnosis of Trichomonas vaginalis infection in women.

130 Trichomonas vaginalis infection is a sexually transmitte

131 Trichomonas vaginalis infection is estimated to be the m

132 The prevalence of Trichomonas vaginalis infection is highest in women with

133 Trichomonas vaginalis infection is highly prevalent worl

134 Trichomonas vaginalis infection is highly prevalent, may

135 Trichomonas vaginalis infection is the most prevalent no

136 omen in Mombasa, Kenya, to determine whether Trichomonas vaginalis infection was associated with an i

137 = 756 men), we identified 150 men (20%) with Trichomonas vaginalis infection, 358 men (47%) with HIV

138 ns related to the diagnosis and treatment of Trichomonas vaginalis infection, as well as the associat

139 long-acting reversible contraception usage, Trichomonas vaginalis infection, bacterial vaginosis, an

140 esented regarding conditions associated with Trichomonas vaginalis infection, including human immunod

141 k women, being 30 to 40 years of age, recent Trichomonas vaginalis infection, primary or recurrent ge

142 infection, Chlamydia trachomatis infection, Trichomonas vaginalis infection, vulvovaginal candidiasi

143 eria gonorrhoeae, Chlamydia trachomatis, and Trichomonas vaginalis infection.

144 ydia trachomatis, Neisseria gonorrhoeae, and Trichomonas vaginalis infection.

145 Trichomonas vaginalis infections are usually asymptomati

146 eria gonorrhoeae, Mycoplasma genitalium, and Trichomonas vaginalis infections as well as the characte

147 Trichomonas vaginalis infections in men are traditionall

148 tudy was to evaluate potential mechanisms of Trichomonas vaginalis involvement in human immunodeficie

149 Trichomonas vaginalis is a common sexually transmitted i

150 Trichomonas vaginalis is a common sexually transmitted p

151 Trichomonas vaginalis is a common, extracellular, sexual

152 Trichomonas vaginalis is a highly divergent, unicellular

153 Trichomonas vaginalis is a parasite of the urogenital tr

154 Trichomonas vaginalis is a parasitic protozoan and the c

155 Trichomonas vaginalis is a parasitic protozoan purine au

156 Trichomonas vaginalis is a prevalent sexually transmitte

157 Trichomonas vaginalis is a protist that causes the most

158 Trichomonas vaginalis is a protozoan parasite of humans

159 Trichomonas vaginalis is a unicellular microaerophilic e

160 Trichomonas vaginalis is an anaerobic protozoan parasite

161 Trichomonas vaginalis is an extracellular protozoan para

162 Trichomonas vaginalis is an important pathogen in both m

163 Trichomonas vaginalis is an underestimated sexually tran

164 Host parasitism by Trichomonas vaginalis is complex and in part mediated by

165 Host parasitism by Trichomonas vaginalis is complex, and the adhesion to va

166 from the sexually transmitted human parasite Trichomonas vaginalis is described.

167 Trichomonas vaginalis is likely the most prevalent nonvi

168 The protist Trichomonas vaginalis is one of the most common human se

169 Trichomonas vaginalis is one of the most common nonviral

170 ecreted cysteine protease (CP) fraction from Trichomonas vaginalis is shown here to induce apoptosis

171 Trichomonas vaginalis is the agent of a highly prevalent

172 Trichomonas vaginalis is the most prevalent nonviral sex

173 Trichomonas vaginalis is the most prevalent nonviral sex

174 Trichomonas vaginalis is the most prevalent nonviral sex

175 udy were to examine the TVV prevalence in US Trichomonas vaginalis isolates and TVV's associations wi

176 With gap1 from Trichomonas vaginalis obtained earlier, the data include

177 unusual case of extragenital infection with Trichomonas vaginalis of the conjunctiva of a 32-year-ol

178 rican women who used drugs were screened for Trichomonas vaginalis on > or =2 occasions between March

179 230) performed rapid point-of-care tests for Trichomonas vaginalis on self-collected vaginal swabs.

180 ated in patients concomitantly infected with Trichomonas vaginalis or Chlamydia trachomatis in additi

181 isseria gonorrhea, Chlamydia trachomatis, or Trichomonas vaginalis or who had a rapid antigen or posi

182 ins AP65, AP51, AP33 and AP23 synthesized by Trichomonas vaginalis organisms in high iron play a role

183 culture and evaluated their interaction with Trichomonas vaginalis parasites to complement previous s

184 at immune cells perform trogocytosis to kill Trichomonas vaginalis parasites.

185 Trichomonas vaginalis PCR using reagents from a commerci

186 pid antigen or positive urine microscopy for Trichomonas vaginalis performed.

187 Our aim was to determine Trichomonas vaginalis prevalence using the Aptima Tricho

188 Trichomonas vaginalis produces soluble factors that have

189 Trichomonas vaginalis relies on imported purine and pyri

190 Trichomonas vaginalis results were concordant between th

191 binding protein from the primitive eukaryote Trichomonas vaginalis reveals how a single protein can o

192 s the gold standard for clinical culture for Trichomonas vaginalis screening.

193 Our analyses of the Trichomonas vaginalis SCS sequences also confirmed the m

194 Trichomonas vaginalis secretes putrescine that is readil

195 ydia trachomatis, Neisseria gonorrhoeae, and Trichomonas vaginalis Sequencing was used to assess macr

196 the amitochondrial Entamoeba histolytica and Trichomonas vaginalis species were analyzed.

197 The InPouch Trichomonas vaginalis test is the gold standard for clin

198 aginalis were compared with the Affirm VPIII Trichomonas vaginalis test.

199 r point-of-care test (POCT) for the parasite Trichomonas vaginalis that causes the STI trichomoniasis

200 Trichomoniasis results from adhesion of Trichomonas vaginalis to the mucous membrane of the uret

201 iterature has reported increased accuracy of Trichomonas vaginalis transcription-mediated amplificati

202 ydia trachomatis, Neisseria gonorrhoeae, and Trichomonas vaginalis via commercial transcription-media

203 Trichomonas vaginalis virus (TVV) is a non-segmented, 4.

204 LV was found to be more thermoresistant than Trichomonas vaginalis virus 1, but no specific protein m

205 iruses, including Leishmania RNA viruses and Trichomonas vaginalis viruses.

206 GPI of the parabasalid Trichomonas vaginalis was also sequenced.

207 ransport medium to maintain the viability of Trichomonas vaginalis was determined by comparing transp

208 DNA detection, but Chlamydia trachomatis and Trichomonas vaginalis were not.

209 eria gonorrhoeae, Chlamydia trachomatis, and Trichomonas vaginalis were performed.

210 hlamydia trachomatis, Neisseria gonorrhoeae, Trichomonas vaginalis) and the E6/E7 mRNA of human papil

211 ologically assayed chlamydia, gonorrhea, and Trichomonas vaginalis) at the 12-month assessment and th

212 lastida (Leishmania major), the Parabasalia (Trichomonas vaginalis), and the Microsporidia (Vairimorp

213 ) and three partial draft genomes (including Trichomonas vaginalis).

214 (zero for bacterial vaginosis, candida, and Trichomonas vaginalis).

215 Trichomonas vaginalis, a common sexually transmitted par

216 Trichomonas vaginalis, a flagellated protozoan, is the a

217 Trichomonas vaginalis, a human-infective parasite, cause

218 In Trichomonas vaginalis, a Myb1 protein was previously dem

219 In Trichomonas vaginalis, a parabasalian flagellate thought

220 Among them, Trichomonas vaginalis, a parasite adapted to the human g

221 describe the genome sequence of the protist Trichomonas vaginalis, a sexually transmitted human path

222 hlamydia trachomatis, Mycoplasma genitalium, Trichomonas vaginalis, adenovirus, and herpes simplex vi

223 ribed in the divergent unicellular eukaryote Trichomonas vaginalis, although genome analyses reveal t

224 The discovery that Trichomonas vaginalis, an early diverging protist that l

225 hlamydia trachomatis, Neisseria gonorrhoeae, Trichomonas vaginalis, and bacterial vaginosis have been

226 hlamydia trachomatis, Neisseria gonorrhoeae, Trichomonas vaginalis, and bacterial vaginosis testing w

227 ptococcus agalactiae, Chlamydia trachomatis, Trichomonas vaginalis, and Candida spp., as well as thei

228 the pathogenic protozoa Giardia lamblia and Trichomonas vaginalis, and the bacterial pathogens Helic

229 the slime-mold Dictyostelium, the protozoan Trichomonas vaginalis, and the bacterium Burkholderia ps

230 ntamoeba fragilis, Pentatrichomonas hominis, Trichomonas vaginalis, and Trichomonas tenax.

231 he three common organisms causing vaginitis: Trichomonas vaginalis, Candida species, and Gardnerella

232 Deidentified clinical data and tests for Trichomonas vaginalis, Candida, and bacterial vaginosis

233 om Trypanosoma cruzi, Entamoeba histolytica, Trichomonas vaginalis, Cryptococcus neoformans, and Sacc

234 eria gonorrhoeae, Chlamydia trachomatis, and Trichomonas vaginalis, each considered separately) and i

235 hlamydia trachomatis, Neisseria gonorrhoeae, Trichomonas vaginalis, Mycoplasma hominis, Ureaplasma sp

236 Women infected with Neisseria gonorrhoeae, Trichomonas vaginalis, or Chlamydia trachomatis had high

237 en, infections with Neisseria gonorrhoeae or Trichomonas vaginalis, or long-term follow-up.

238 fungal pathogens, as well as protozoa, e.g., Trichomonas vaginalis, Plasmodium berghei, and sporozoit

239 the genome databases for Giardia lamblia and Trichomonas vaginalis, respectively, and represent the l

240 e (rSAHH) cloned from Pseudomonas putida and Trichomonas vaginalis, respectively.

241 A related human trichomonad, Trichomonas vaginalis, showed no cytotoxic effects, indi

242 hydrogenosome-bearing, unicellular eukaryote Trichomonas vaginalis, that contains the active site mot

243 Trichomonas vaginalis, the causative agent for human tri

244 Trichomonas vaginalis, the causative agent of the venere

245 Recent studies have proposed that Trichomonas vaginalis, the causative agent of trichomoni

246 Trichomonas vaginalis, the etiologic agent of the most c

247 e medically important parasites: the protist Trichomonas vaginalis, the hard tick Ixodes ricinus, and

248 s simplex virus 2 infection, genital ulcers, Trichomonas vaginalis, vaginitis or cervicitis, and male

249 CR assay, using primers against pfoB gene of Trichomonas vaginalis, was developed and evaluated using

250 Trichomonas vaginalis, which causes the most common nonv

251 equences in the protists Giardia lamblia and Trichomonas vaginalis, which may represent the deepest k

252 tein-encoding genes in the parasitic protist Trichomonas vaginalis, which represents one of the deepe

253 bation of culture media for the detection of Trichomonas vaginalis.

254 drial protist pathogens: Giardia lamblia and Trichomonas vaginalis.

255 , Entamoeba histolytica, Giardia lamblia and Trichomonas vaginalis.

256 t preparation and a culture method to detect Trichomonas vaginalis.

257 Trypanosoma brucei, Leishmania donovani, and Trichomonas vaginalis.

258 in one of the earliest diverging protozoans, Trichomonas vaginalis.

259 is specifically related to a homologue from Trichomonas vaginalis.

260 a, that represented by the parasitic protist Trichomonas vaginalis.

261 ctably transform the human-infective protist Trichomonas vaginalis.

262 ption of protein-coding genes of the protist Trichomonas vaginalis.

263 as been proposed as a possible treatment for Trichomonas vaginalis.

264 st the urogenital, nonkinetoplastid parasite Trichomonas vaginalis.

265 e causative agent is the parasitic protozoan Trichomonas vaginalis.

266 nomer (rTvCyP1 mono) from the human pathogen Trichomonas vaginalis.

267 aginotropic extracellular protozoan parasite Trichomonas vaginalis.

268 nscription factor in the protozoan parasite, Trichomonas vaginalis.

269 for Candida spp., Gardnerella vaginalis, and Trichomonas vaginalis.

270 ally transmitted infections (STIs) caused by Trichomonas vaginalis.

271 Women were also tested for Trichomonas vaginalis.

272 sekeeping genes, to genetically characterize Trichomonas vaginalis.

273 The parasite he discovered was Trichomonas vaginalis; and, in collaboration with Foucau

274 tested with the Aptima BV and Aptima Candida/Trichomonas vaginitis (CV/TV) assays.

275 For Trichomonas vaginitis (n=55), HIV-1 RNA decreased from 3

276 d after treatment of Candida vulvovaginitis, Trichomonas vaginitis, and bacterial vaginosis.

277 Leukorrhea on microscopy was associated with Trichomonas vaginitis.

278 r gonorrhea, chlamydia, bacterial vaginosis, trichomonas, vulvovaginal candidiasis, pelvic inflammato

279 the major antioxidant defense mechanisms in Trichomonas was confirmed by showing that the parasite r