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2 n test responses to Candida albicans, mumps, Trichophyton, and a bacterial antigen made from lysed St
3 conclude that different immune responses to Trichophyton are mediated by distinct T cell repertoires
7 aysians (Orang Asli [OA]), tinea imbricata-a Trichophyton concentricum fungal skin infection-emerged
8 nvestigated an understudied fungal pathogen, Trichophyton concentricum, by analyzing the skin microbi
10 o demonstrate that the hedgehog dermatophyte Trichophyton erinacei produces two beta-lactam antibioti
12 te hypersensitivity skin test reactions to a Trichophyton extract exhibited IgE antibody binding to a
15 of Eurotiomycetes including Aspergillus and Trichophyton from within a lichen-forming ancestral grou
16 ytes was 37.6%; of isolates belonging to the Trichophyton genus, 88.3% were the T. rubrum complex, an
17 on antifungal drugs, the recently identified Trichophyton indotineae isolates have exhibited signific
18 use in agricultural and other settings; and Trichophyton indotineae, driven by the under-regulated u
21 tes, including Trichophyton rubrum (n = 15), Trichophyton mentagrophytes (n = 7), Trichophyton tonsur
22 ogenic dermatophytes Trichophyton rubrum and Trichophyton mentagrophytes as well as against the breas
24 d individual responses to tetanus toxoid and Trichophyton mentagrophytes were significantly higher th
25 linical strains each of Trichophyton rubrum, Trichophyton mentagrophytes, and Epidermophyton floccosu
26 nfections in humans are Trichophyton rubrum, Trichophyton mentagrophytes, and Trichophyton interdigit
27 human pathogenic fungi, Trichophyton rubrum, Trichophyton mentagrophytes, Aspergillus fumigatus, and
28 ncountered in a clinical mycology laboratory-Trichophyton mentagrophytes, Trichophyton rubrum, Tricho
29 ophytes tested included Trichophyton rubrum, Trichophyton mentagrophytes, Trichophyton tonsurans, Epi
30 r forms of severe disease caused by Candida, Trichophyton, Phialophora, and Exophiala species, includ
31 Forty-eight dermatophyte isolates, including Trichophyton rubrum (n = 15), Trichophyton mentagrophyte
32 Distinct immune responses in humans to the Trichophyton rubrum Ag, Tri r 2, are associated with dif
34 recombinant DC-HIL to bind the dermatophytes Trichophyton rubrum and Microsporum audouinii, but not s
35 tested against the pathogenic dermatophytes Trichophyton rubrum and Trichophyton mentagrophytes as w
36 mologous protein, Tri r 4, was cloned from a Trichophyton rubrum cDNA library, and the recombinant pr
40 ri r 2, derived from the dermatophyte fungus Trichophyton rubrum, exhibits unique immunologic charact
41 parators against 25 clinical strains each of Trichophyton rubrum, T. mentagrophytes, and Epidermophyt
42 ty study, 5 blinded clinical strains each of Trichophyton rubrum, Trichophyton mentagrophytes, and Ep
43 mon species causing infections in humans are Trichophyton rubrum, Trichophyton mentagrophytes, and Tr
44 od using four common human pathogenic fungi, Trichophyton rubrum, Trichophyton mentagrophytes, Asperg
46 logy laboratory-Trichophyton mentagrophytes, Trichophyton rubrum, Trichophyton tonsurans, and M. cani
53 tinct antigens derived from the dermatophyte Trichophyton that serve as targets for diverse immune re
54 = 15), Trichophyton mentagrophytes (n = 7), Trichophyton tonsurans (n = 11), and Epidermophyton floc
56 with an extract from the dermatophyte fungus Trichophyton tonsurans can result in either immediate (I
61 Neosartorya fischeri, Microsporum canis, and Trichophyton tonsurans were shown to be able to catalyze
63 ophyton rubrum, Trichophyton mentagrophytes, Trichophyton tonsurans, Epidermophyton floccosum, and Mi
64 ition over time was examined in responses to Trichophyton using primary PBMC cultures established fro