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1 al health in both pigs and humans exposed to Trichuris.
2 ical for immunity to the intestinal pathogen Trichuris.
3 ed type 2 cytokine responses and immunity to Trichuris.
4                                              Trichuris and hookworm were rarely detected, resulting i
5 vention, we observed a reduced prevalence of Trichuris and Shigella infection relative to the same ag
6 acts like an "epithelial escalator" to expel Trichuris and that the rate of epithelial cell movement
7  of specimens containing Entamoeba, Ascaris, Trichuris, and hookworm.
8 e gut of mouse strains that are resistant to Trichuris, and IL-25-deficient mice on a genetically res
9                                           In Trichuris, and potentially other Clade I parasites of pl
10 ter infection with the gut-dwelling parasite Trichuris, fail to develop a pathogen-specific CD4+ T he
11 .7% had one or more nematodes, most commonly trichuris (in 3.9%).
12 011 African refugees were giardia (in 5.7%), trichuris (in 5.0%), and schistosoma (in 1.8%); among 55
13 ively expressed in the colon and exposure to Trichuris induced the expression of IL-31 in CD4(+) T ce
14 FN-gamma and TNF-alpha and failed to develop Trichuris-induced intestinal inflammation.
15   Critically, neutralization of IFN-gamma in Trichuris-infected TSLPR(-/-) mice restored Th2 cytokine
16 Blockade of proinflammatory cytokines during Trichuris infection ablates the requirement for IKK-beta
17                               In response to Trichuris infection, IL-31Ralpha(-/-) mice exhibited inc
18  to modulating IL-13 function and control of Trichuris infections.
19 ion of the caecal epithelium by the parasite Trichuris muris allowed us to live image syncytial tunne
20 atory conditions in intestines infected with Trichuris muris and within the tumor microenvironment (B
21  infected with the gastrointestinal helminth Trichuris muris displayed accelerated expulsion of paras
22                                Additionally, Trichuris muris eggs displayed defective hatching in the
23 taneous peristaltic movement and facilitated Trichuris muris expulsion.
24 polygyrus, Nippostronglyus brasiliensis, and Trichuris muris have provided considerable information a
25 on during murine infection with the helminth Trichuris muris However, the mechanisms required for bas
26 c infection by the gastrointestinal nematode Trichuris muris in susceptible AKR mice, which mount a T
27 ngruent infection with the nematode parasite Trichuris muris in the large intestine around the time o
28 the chronically infecting parasitic nematode Trichuris muris in the large intestine of mice is depend
29                           Here, we show that Trichuris muris infection in mice increases coinfection
30        We used the well-established model of Trichuris muris infection to investigate the innate resp
31 ation in MLN and mount Th1 cell responses to Trichuris muris infection.
32 is induced in the cecum of mice resistant to Trichuris muris infection.
33 with the gastrointestinal toxin piroxicam or Trichuris muris infection.
34 nvestigate early infection events using both Trichuris muris infections of mice and murine caecaloids
35  infection with the caecum-dwelling helminth Trichuris muris is dependent on interleukin (IL)-4 and I
36   Expulsion of the gastrointestinal nematode Trichuris muris is mediated by a T helper (Th) 2 type re
37                The p43 (Tm-DLP-1) protein of Trichuris muris of mice comprises 95% of the protein sec
38  Th cells, ICOS(-/-) mice were infected with Trichuris muris or Toxoplasma gondii.
39                                              Trichuris muris resides in intimate contact with its hos
40 IL-12-, IL-4-, and IL-12-deficient mice with Trichuris muris to determine whether IL-10 contributes t
41   In this study, we used the murine whipworm Trichuris muris to investigate the effect of the ferment
42  used the gastrointestinal helminth pathogen Trichuris muris to test the hypothesis that a chronic sy
43                      The intestinal nematode Trichuris muris was recently demonstrated to utilise mic
44  and activity against evolutionarily distant Trichuris muris whipworm adults.
45 e show that intestinal Th2 responses against Trichuris muris worms and Schistosoma mansoni eggs do no
46 minths (Heligmosomoides polygyrus bakeri and Trichuris muris).
47 urveyed the genomes of Trichinella spiralis, Trichuris muris, and Romanomermis culicivorax and identi
48 lminth parasites, Heligmosomoides polygyrus, Trichuris muris, and Schistosoma mansoni.
49             In studies of mice infected with Trichuris muris, blocking B7 ligand interactions inhibit
50 treated mice infected with enteric parasite, Trichuris muris, exhibited attenuated 5-HT production, c
51 infection with the gastrointestinal helminth Trichuris muris, immunity to which is critically depende
52 infection with the gastrointestinal parasite Trichuris muris, memory CD4+ T cells persist in the drai
53 nfection with the gastrointestinal helminth, Trichuris muris, revealing roles for miRNAs in fibrosis
54 okines in immunity to the parasitic helminth Trichuris muris, the local effector mechanism culminatin
55 n infection with the cecum-dwelling nematode Trichuris muris, the majority of inbred strains of mice
56 nic infection with gastrointestinal helminth Trichuris muris, we identify dual functions for RELMbeta
57 le to infection with the intestinal nematode Trichuris muris, whereas their wild-type littermates wer
58 ection with the intestinal helminth parasite Trichuris muris, which yields a chronic infection becaus
59 table and widely-used model of trichuriasis, Trichuris muris.
60 tion with the gut-dwelling helminth parasite Trichuris muris.
61 -scale metabolic model of the mouse whipworm Trichuris muris.
62 oor enclosure and infected with the parasite Trichuris muris.
63 ed in AKR/J mice using a parasite infection, Trichuris muris.
64  and mice infected with the enteric parasite Trichuris muris.
65 ris trichiura and the mouse laboratory model Trichuris muris.
66  with the gastrointestinal nematode parasite Trichuris muris.
67 e large intestine dwelling helminth parasite Trichuris muris.
68 sing the murine model of whipworm infection, Trichuris muris.
69 phiregulin delayed expulsion of the nematode Trichuris muris.
70 sponse against the gastrointestinal helminth Trichuris muris.
71 impaired immunity to the intestinal helminth Trichuris muris.
72 o infection by the gastrointestinal nematode Trichuris muris.
73                                              Trichuris nematodes reproduce within the microbiota-rich
74                               Paradoxically, Trichuris of pigs has shown substantial promise as a tre
75 rugia, which causes lymphatic filariasis and Trichuris, one of the soil-transmitted helminths that in
76  between infection with Schistosoma mansoni, Trichuris, or Strongyloides species and P. falciparum in
77 ry CD4+ T cells develop after infection with Trichuris, persist in the GALT, and mediate protective i
78 0.06), hookworm (prevalence ratio, 0.07), or trichuris (prevalence ratio, 0.27) but were not less lik
79 tory leucocyte proteinase-1 (SLP-1), but the Trichuris protein family differs in being composed of mu
80 nsity, types of worms (ascaris, hookworm, or trichuris), risk of bias, cluster versus individual tria
81 onstrate that p43 is the dominant protein in Trichuris's pseudocoelomic fluid, replacing the major in
82 hogenic for humans and animals: the whipworm Trichuris sp., the roundworm Ascaris sp., the flatworm D
83 E, but not active infections with Ascaris or Trichuris species.
84 oil transmitted parasitic helminths, such as Trichuris spp, are ubiquitous in humans and animals but
85  only the T. muris metabolism but also other Trichuris spp. in understanding host parasite interactio
86         Infection with intestinal whipworms (Trichuris spp.) causes widespread morbidity and may alte
87  administration of ova from the pig whipworm Trichuris suis (T. suis; TSO) has been proposed for the
88 found that soluble products derived from the Trichuris suis (TsSP) significantly affect the different
89 in the porcine proximal colon in response to Trichuris suis (whipworm) infection using 16S rRNA gene-
90 whether infection with the nematode parasite Trichuris suis alters systemic cytokine levels, cellular
91                                              Trichuris suis and Oesophagostomum dentatum larvae were
92 ve shown that administration of the nematode Trichuris suis can be beneficial in treating various imm
93 ined the efficacy and safety of the helminth Trichuris suis in therapy of ulcerative colitis.
94                             The pig whipworm Trichuris suis is important in swine production because
95 rhinitis received three weekly doses of 2500 Trichuris suis ova (n = 45) or placebo (n = 44) over 6 m
96                                              Trichuris suis ova (TSO) have been tested for therapeuti
97 example, to assess the safety or efficacy of Trichuris suis ova in allergies, inflammatory bowel dise
98                       Patients received 2500 Trichuris suis ova or placebo orally at 2-week intervals
99 his study suggests that cytokines induced by Trichuris suis ova treatment do not alter allergic react
100                                              Trichuris suis ova were obtained from the US Department
101                    Eggs of the pig whipworm (Trichuris suis ova) have been shown to be safe in multip
102 tory bowel disease, exposure to the helminth Trichuris suis reduces disease activity.
103                               Treatment with Trichuris suis soluble products during monocyte-to-macro
104                                The whipworm (Trichuris suis) secretes prostaglandin E2 to suppress pr
105 rm (Necator americanus) or porcine whipworm (Trichuris suis) show that they are safe and may be effec
106 novirus (25/176), Giardia spp. (13/176), and Trichuris trichiura (12/176).
107  most prominent species (13.5%), followed by Trichuris trichiura (6.1%), and Cryptosporidium spp. (0.
108 d by the gastrointestinal dwelling nematodes Trichuris trichiura (a whipworm) and Ascaris lumbricoide
109 4.5% for hookworms, and from 0.1 to 5.7% for Trichuris trichiura across the implementation units.
110 hole-genome sequences of the human-infective Trichuris trichiura and the mouse laboratory model Trich
111 3.6% (95% CI 11.5, 15.7), while Hookworm and Trichuris trichiura had overall prevalence of 4.6% (95%
112        The cytokine and antibody response to Trichuris trichiura infection was determined for 96 pers
113    Wheezing status, Ascaris lumbricoides and Trichuris trichiura infection, IL-10 production by perip
114                                    Childhood Trichuris trichiura infections during the first 5 years
115 f currently available anthelminthics against Trichuris trichiura infections is significatively lower
116                                              Trichuris trichiura infestation might be associated with
117                       The parasitic nematode Trichuris trichiura is a significant burden on public he
118 ndary outcomes were Ascaris lumbricoides and Trichuris trichiura prevalence, infection intensity of e
119 iardia lamblia, Enterocytozoon bieneusi, and Trichuris trichiura was found in all animals regardless
120     The percentage of children infected with Trichuris trichiura was highest among children who did n
121 lminths (hookworm, Ascaris lumbricoides, and Trichuris trichiura) are the most widespread NTDs, but b
122 lminths (Ascaris lumbricoides, hookworm, and Trichuris trichiura) are widespread and often occur conc
123                                    Whipworm (Trichuris trichiura) infection is a soil-transmitted hel
124 ections (ie, hookworm, Ascaris lumbricoides, Trichuris trichiura) with the Kato-Katz method.
125 ca), and helminths (Ascaris lumbricoides and Trichuris trichiura), as well as two extrinsic controls
126 sease trichuriasis is caused by the whipworm Trichuris trichiura, a soil-transmitted helminth that ha
127    Ascaris lumbricoides, Necator americanus, Trichuris trichiura, and Strongyloides stercoralis soil-
128 cylostoma duodenale and Necator americanus), Trichuris trichiura, and Strongyloides stercoralis.
129 er, benzimidazoles have low efficacy against Trichuris trichiura, and there are concerns about benzim
130                      Infection with malaria, Trichuris trichiura, Ascaris lumbricoides, and hookworms
131 e orthologue of p43 from the human whipworm, Trichuris trichiura, exhibits similar lipid-binding acti
132 on infection with STH (Ascaris lumbricoides, Trichuris trichiura, hookworm [Ancylostoma duodenale and
133 nfections by measuring Ascaris lumbricoides, Trichuris trichiura, hookworm, and Giardia duodenalis am
134 chlii, Endolimax nana, Ascaris lumbricoides, Trichuris trichiura, Strongyloides stercoralis, and Neca
135                     The major E/S protein of Trichuris trichiura, the human whipworm, is a highly imm
136 fections in humans have low efficacy against Trichuris trichiura.
137 infection with the soil-transmitted helminth Trichuris trichiura.
138  they are not sufficiently effective against Trichuris trichiura.
139 f human infection with the whipworm of dogs, Trichuris vulpis, in a woman with duodenal ulcer disease
140                                              Trichuris (whipworm) infects 1 billion people worldwide
141                                              Trichuris whipworms cause disease and morbidity in human
142               Following exposure to low-dose Trichuris, wild-type C57BL/6 mice exhibit persistent inf
143 -/-) mice exhibited accelerated expulsion of Trichuris with significantly decreased worm burdens comp

 
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