1 U. maydis deploys many effector proteins to manipulate i
2 In S. cerevisiae
and U. maydis, NPC motility prevented NPCs from clustering.
3 to 5 transformants/micrograms linear DNA
and U. maydis at up to 25 transformants/microgram circular D
4 icity, are very similar in P. flocculosa
and U. maydis, Sporisorium reilianum, and Ustilago hordei.
5 ystatin (CC9) is induced upon penetration
by U. maydis wild type.
6 sulted in a surface-exposed form in
cultured U. maydis cells.
7 nt double-stranded RNA (dsRNA) virus in
each U. maydis subtype.
8 For U. maydis, disruption of ump2 eliminated the filamentous
9 The two genes
from U. maydis and one of the genes from M. violaceum were ex
10 errichrome and ferrichrome A biosynthesis
in U. maydis.
11 e pheromone-responsive MAP kinase cascade
in U. maydis.
12 f such motifs affects peroxisome function
in U. maydis strains challenged with fatty acids.
13 s of filamentous growth and pathogenicity
in U. maydis.
14 ontrol of morphogenesis and pathogenicity
in U. maydis.
15 ole in the recombinational repair pathway
in U. maydis, and imply that it plays a similar key role in
16 DNA repair and recombination proficiency
in U. maydis requires both Rec2 and Rad51.
17 regulators of G2/M cell cycle progression
in U. maydis, interacts and controls the subcellular locali
18 rium formation and cell cycle progression
in U. maydis, which serves as a "toggle switch" to control
19 Recapitulation
in U. maydis of defects in DNA repair and genome stability
20 These results indicate that gap repair
in U. maydis is unlikely to proceed by the mechanism envisi
21 asis for the extreme radiation resistance
in U. maydis.
22 Studies
in U. maydis and Aspergillus nidulans reveal a complex inte
23 efficient homologous recombination system
in U. maydis.
24 Our findings suggest that
in U. maydis, unprotected telomeres arising from Ku depleti
25 re three well-characterized killer toxins
in U. maydis-KP1, KP4, and KP6-which are secreted by the P1
26 emonstrate that KP4 affects (45)Ca uptake
in U. maydis.
27 proteins reported to influence virulence
in U. maydis as the singular divergence that could explain
28 t, NPC motility required F-actin, whereas
in U. maydis, microtubules, kinesin-1, and dynein drove por
29 Mutants
of U. maydis deleted of DSS1 are extremely radiation sensit
30 e phenotypes mirror previous observations
of U. maydis mutants deficient in Brh2 or Rad51.
31 Phenotypic screening
of U. maydis mutants deleted for genes encoding secreted pr
32 olution of a system enabling the survival
of U. maydis under such conditions could be a secondary con
33 nteraction of maize with the fungal
pathogen U. maydis.
34 In telomerase-
positive U. maydis, deletion of rad51 and blm separately caused s
35 confirm and extend earlier observations
that U. maydis hyphae branch extensively on the leaf surface
36 The U. maydis killer toxin KP6 contains two polypeptide chai
37 The U. maydis TER (UmTER) contains a 5'-monophosphate, disti
38 Here, we characterize
the U. maydis effector Sts2 (Small tumor on seedlings 2), wh
39 d in genome defense, that are lacking in
the U. maydis genome due to clean excision events.
40 ranslocation of a number of effectors in
the U. maydis-maize system and show data that suggest that t
41 , lucky moments when major advances made
the U. maydis-maize system what it is now-a well-established
42 Moreover,
the U. maydis ump2 gene, initially detected as an upregulate
43 rformed a functional characterization of
the U. maydis effector Jasmonate/Ethylene signaling inducer
44 The unusual C-terminal extension of
the U. maydis Hac1 homolog, Cib1 (for Clp1 interacting bZIP1
45 We highlight the contribution of
the U. maydis model system but also discuss the differences
46 romoter and iron-regulatory sequences of
the U. maydis sid1 gene were defined by fusing restriction a
47 Here, we show that
the U. maydis class VII chitin synthase and 1,3-beta-glucan
48 We show that
the U. maydis effector ROS burst interfering protein 1 (Rip1
49 e particularly been driven forward using
the U. maydis-maize pathosystem.
50 cc9 is not induced after infection with
the U. maydis effector mutant Deltapep1, which elicits massi
51 in lignin biosynthesis are hypersensitive
to U. maydis infection.
52 t lox3 mutation-based resistance of maize
to U. maydis requires functional Rip1.
53 dependent reduced susceptibility of maize
to U. maydis.
54 mutant plants showed increased resistance
to U. maydis wild-type strains, rip1 deletion strains infec
55 expression and a hypersensitive response
to U. maydis wild-type infection.
56 We found that
upon U. maydis infection of Z. mays, KWL1-b is expressed at s
57 While U. maydis Deltagls1 cells induce strong plant defense re