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1                                              UCS and AMT, as an adjuvant to standard medical therapy
2                                              UCS has the advantage of faster improvement in corneal c
3                                              UCS predictability was varied on a trial-by-trial basis
4                                              UCS-evoked depolarization of amygdala neurons may instru
5                                              UCS-evoked responses in both amygdala and PAG were inhib
6                                              UCSs shared proteomic features with gynecologic carcinom
7 edator feces may also be less effective as a UCS because they are poorly predictive of the actual pre
8                        Proteins containing a UCS (UNC-45/CRO1/She4p) domain are necessary for the inc
9 try and a parameterised volume geometry of a UCS IV.3-C type periprosthetic femur fracture were simul
10 Sf9 cell expression system, which required a UCS (UNC-45/CRO1/She4p) family myosin chaperone from Pla
11                            We propose that a UCS dimer links two myosins at their motor domains and t
12                                Shortly after UCS, women were included if they gave birth to a live ba
13 re 7,281 (6.7%), OCS were 26,467 (24.2%) and UCS were 14,106 (12.9% of all births and 53.3% out of al
14 10.2, and 22.9 +/- 10.1 days in MT, AMT, and UCS groups, respectively, with a significant difference
15 on every trial, whereas in others the CS and UCS are paired intermittently.
16                   In many studies the CS and UCS are paired on every trial, whereas in others the CS
17 r or absent node condensation in myo2-E1 and UCS rng3-65 mutants.
18 how a dissociation between CR expression and UCS expectancy following differential delay conditioning
19  learning process mediates CR expression and UCS expectancy.
20 een MT and AMT (P = .001) and between MT and UCS (P = .001), but not between UCS and AMT (P = .9).
21 S and LoS > ED was observed for both PCS and UCS.
22 ed the identities of conditioned stimuli and UCS.
23 interval) elapses between CS termination and UCS onset.
24                                     The anti-UCS serum reacted with a P. carinii protein that migrate
25 kDa protein was immunoprecipitated with anti-UCS serum and shown to react with a monoclonal antibody
26 cological inactivation of the PAG attenuated UCS-evoked responses in the amygdala and impaired acquis
27 tween MT and UCS (P = .001), but not between UCS and AMT (P = .9).
28  de novo in the absence of UCS proteins, but UCS proteins promote myosin stability and interactions w
29                       Only one locus, called UCS, supports the production of MSG mRNA.
30                               This so-called UCS domain can adopt discrete conformations to efficient
31                      Uterine carcinosarcoma (UCS) is a rare and highly aggressive gynecological malig
32  carcinoma (USC) and uterine carcinosarcoma (UCS) tumors are uniquely aggressive, suggesting that the
33 de (PI) for treating uterine carcinosarcoma (UCS).
34                     Uterine carcinosarcomas (UCSs) are highly aggressive malignancies associated with
35 haracterizations of uterine carcinosarcomas (UCSs).
36 % identical to the P. carinii f. sp. carinii UCS.
37  cells with proteins containing a conserved 'UCS' domain.
38 isoenergetic amounts of uncooked cornstarch (UCS; 1.76 +/- 0.41 g/kg) given in random order on consec
39 ), overall CS (OCS) and urgent/emergency CS (UCS).
40  UCS presentations paired with the CS+ (CS + UCS) and CS- (CS - UCS), as well as presentations of the
41 paired with the CS+ (CS + UCS) and CS- (CS - UCS), as well as presentations of the UCS alone were inc
42 , OCS; planned CS, PCS; urgent/emergency CS, UCS), in Friuli Venezia Giulia (a region of North-Easter
43  the uncertainty inherent in intermittent CS-UCS pairing procedures.
44 To better understand the influence of the CS-UCS pairing rate on brain activity, the experimenters pr
45 nd fusiform gyrus activity paralleled the CS-UCS pairing rate, whereas the insula and dorsolateral pr
46 yrus activity increased linearly with the CS-UCS pairing rate.
47 ing working memory processes maintain the CS-UCS representation during the trace interval.
48                                 Differential UCS expectancy (awareness) was observed only on perceive
49 as also observed independent of differential UCS expectancies.
50          The most important risk factors for UCS were (in descending order of statistical significanc
51 romosomes, suggesting that the mechanism for UCS-MSG recombination is reciprocal exchange.
52 abilisation is challenging, particularly for UCS IV.3-C fractures.
53 imen PI and should be standard treatment for UCS.
54                           These VP units for UCS remained responsive to sucrose reward after learning
55  more emphasis on the role of EMT drivers in UCS progression and scope of targeting these molecules.
56  have highlighted the pivotal role of EMT in UCS progression and mortality.
57 relevant, we show that miR-200 expression in UCS cells induces a robust mesenchymal-epithelial transi
58 duced deformation and failure of fixation in UCS IV.3-C periprosthetic femur fractures.
59 OMC state and it induces 325% improvement in UCS.
60 omeres, indicating a crucial role of R805 in UCS-client interactions.
61 ty, specimens exhibited 26-37% reductions in UCS, primarily due to membrane tearing from repeated exp
62                   The range of EMT scores in UCS was the largest among all tumor types studied via Th
63 ngent cocaine or methylphenidate injections (UCS retrieval) 1 h before the extinction sessions decrea
64 Theory and evidence suggest that instructive UCS inputs to the amygdala will be inhibited when the UC
65                    To gain more insight into UCS function, we determined the atomic structure of the
66 cally, attitude changes induced by the liked UCS are based on a transfer of positive affective value
67                    Moreover, by manipulating UCS aversiveness via reinforcer inflation, we showed tha
68     To distinguish between these two models, UCS/MSG junctions in the genome were compared with UCS/M
69               Adults with chemotherapy-naive UCS or ovarian carcinosarcoma (OCS) were randomly assign
70 one (CS+) was paired with a loud white-noise UCS and a second tone (CS-) was presented alone.
71 st myosin can fold de novo in the absence of UCS proteins, but UCS proteins promote myosin stability
72  In addition, probe trials that consisted of UCS presentations paired with the CS+ (CS + UCS) and CS-
73       This review covers the epidemiology of UCS, theories regarding its origin, and the current stat
74  by differences in the explicit estimates of UCS probability or explicit knowledge that the UCS inten
75  by differences in the explicit estimates of UCS probability or explicit knowledge that the UCS inten
76        SCR and participants' expectations of UCS presentation were monitored during conditioning.
77 ons are proposed for the conserved family of UCS proteins: helping to fold myosin motor proteins and
78                   The maximum improvement of UCS occurs at 20% addition of VA in saturated condition.
79        A single dose (1.76 +/- 0.41 g/kg) of UCS at bedtime maintains plasma glucose concentrations >
80 t without generating the very high levels of UCS diversity previously seen.
81 ough both groups exhibited similar levels of UCS expectancy, the devaluation group had significantly
82  skin conductance and continuous measures of UCS expectancy were recorded concurrently with whole-bra
83                     The aggressive nature of UCS is attributed to the metaplastic transformation of c
84 oned stimulus association, but the origin of UCS inputs to the amygdala is unknown.
85 lationship with the participants' ratings of UCS expectancy.
86 r, suggesting the need for a reevaluation of UCS functions in aversive conditioning.
87 t a CS+ retains access to representations of UCS value in distinct regions of ventral prefrontal cort
88                     Finally, by treatment of UCS-xenografted mice with miR-200c incorporated in DOPC
89 icant observation was recorded in the VdD of UCS models.
90            The T. gondii genome contains one UCS family myosin co-chaperone (TgUNC).
91 2.3% alginate concentration, with an optimal UCS achieved at 4.6%.
92 other soil strengthening systems, an overall UCS peak of 5033 kPa is achieved.
93 and aggressiveness of miR-200-overexpressing UCS cell lines.
94                          LoS (post OCS, PCS, UCS) was the outcome measure.
95 redominantly associated with longer LoS post UCS, for PCS LoS was significantly longer with birthweig
96 ned response (UCR) produced by a predictable UCS can also develop.
97 nitude related to the accuracy of predicting UCS presentations.
98 ry, adjuvant chemotherapy, and radiotherapy, UCS has a high recurrence rate and poor overall prognosi
99                           Due to its rarity, UCS remains relatively unexplored, and specific treatmen
100 the DNA encoding the P. carinii f. sp. ratti UCS was not unique in the genome.
101                  Groups 1, 2, and 3 received UCS with medical therapy (MT), AMT with MT, and MT alone
102                 Unc45-/Cro1p-/She4p-related (UCS) protein Rng3p restored the full motility and actin-
103 the hyperparameters of the ANN-predicted P(s)UCS-ES that can be replicated in practical scenarios of
104 compression strength of expansive soils (P(s)UCS-ES).
105 ne most influential parameters affecting P(s)UCS-ES, collected from a broader range of 145 published
106 ' algorithm) were deployed to assess the P(s)UCS-ES.
107 egy in Thailand's Universal Coverage Scheme (UCS) benefit package.
108 ms following an unplanned caesarean section (UCS).
109 ite, termed the upstream conserved sequence (UCS), through two related but distinct mechanisms.
110 nce, called the upstream conserved sequence (UCS), which is encoded by a single locus.
111 nce, called the Upstream Conserved Sequence (UCS), which is in frame with the contiguous MSG sequence
112                           UNC-45/CRO1/She4p (UCS) proteins have variously been proposed to affect the
113                  In budding yeast the single UCS protein (She4p) acts on both myosin-I isoforms (Myo3
114 ward cues in reward learning and uses stable UCS population and firing codes to encode sucrose reward
115 unpleasant odors [the unconditioned stimuli (UCS)] in a partial reinforcement schedule.
116 rsus disliked people (unconditioned stimuli, UCS) at initially neutral places (conditioned stimuli, C
117 fect EDA responses to the aversive stimulus (UCS) or alter participants' mood.
118 g revaluation of the unconditional stimulus (UCS) in human fear conditioning.
119 tioning, an aversive unconditional stimulus (UCS) is repeatedly paired with a neutral conditional sti
120 ed stimulus (CS) and unconditioned stimulus (UCS) coterminate.
121 e response (SCR) and unconditioned stimulus (UCS) expectancy were measured concurrently as participan
122 stimulus (CS) and an unconditioned stimulus (UCS) has been learned.
123 e conditioning as an unconditioned stimulus (UCS) when presented in large amounts.
124 red with an aversive unconditioned stimulus (UCS).
125 red with an aversive unconditioned stimulus (UCS).
126 reward was received [unconditioned stimulus (UCS)].
127 cyclosporine A (CsA, unconditioned stimulus, UCS), animals were sensitized with the hapten.
128  salient reinforcer (unconditioned stimulus, UCS).
129 ulus, and the uniaxial compression strength (UCS) are investigated.
130 6 MPa, and an ultimate compressive strength (UCS) of 85.3 MPa.
131 vior of the unconfined compressive strength (UCS) of cohesive soil reconstituted with cement and lime
132 rosity, and unconfined compressive strength (UCS) of the pure OPC (0%, mixture A) and the OPC-CSA pas
133             Unconfined compressive strength (UCS) tests demonstrated a linear increase in strength up
134 es, including uniaxial compressive strength (UCS), significantly affected fines: the greater the UCS,
135 ering their unconfined compressive strength (UCS).
136 light on the molecular mechanisms supporting UCS, particularly emphasizing the importance of EMT, we
137                      Upper crossed syndrome (UCS) refers to the altered muscle activations and moveme
138   Previous work identified a sequence termed UCS, which is present at the beginning of nearly all MSG
139 domain of unknown function, and a C-terminal UCS domain known to interact with the myosin head.
140                            The COOH-terminal UCS domain of Rng3p alone restored motility to pure Myo2
141 oth the N-terminal (central) and C-terminal (UCS) domains of Rng3p can concentrate independently in c
142 trix and sensitivity analysis indicated that UCS is greatly affected by MDD, then the consistency lim
143               Test performance revealed that UCS inflation did not alter expectancy ratings, but the
144 MSG genes linked to the UCS, suggesting that UCS-MSG junctions are formed by recombination during pop
145                        Our data suggest that UCSs do not develop from EACs via EMT.
146                                          The UCS contains several potential start sites for translati
147                                          The UCS memory retrieval-extinction procedure has superior r
148                                          The UCS models witnessed an overfitting problem because the
149                                          The UCS/MSG junctions in the mRNA matched those in the genom
150 al pathways that relay information about the UCS to the amygdala by recording neurons in the amygdala
151 ression site model is that mRNAs acquire the UCS by RNA splicing.
152 tive client-binding groove did not alter the UCS domain secondary structure or structural stability b
153 n which a single MSG sequence resided at the UCS locus in 80 to 90% of the organisms, showing that P.
154 rom a population highly heterogeneous at the UCS locus reproduced this heterogeneity.
155 organisms that were already different at the UCS locus.
156 lack the UCS, the correspondence between the UCS/MSG junctions in transcripts and those in the genome
157                                     Both the UCS and MSG genes were shown to be located at the ends o
158              We conclude that while both the UCS and the Central domains bind the myosin head with hi
159                       We found that both the UCS and the Central domains bind to the myosin motor dom
160 st the 123-amino-acid peptide encoded by the UCS.
161 cialis carinii has a unique locus called the UCS, which controls surface antigen variation by acting
162 of MSG, the nucleotide sequence encoding the UCS peptide was ligated to the 5' end of an MSG gene and
163 . carinii, it was of interest to examine the UCS locus in the related P. carinii f. sp. ratti.
164   To investigate this issue, we examined the UCS locus in P. carinii from rats that had been exposed
165  begins with 380 nucleotides copied from the UCS locus.
166  between the representations (i.e., from the UCS to the CS).
167                                    Given the UCS's importance in P. carinii f. sp. carinii, it was of
168 ignificantly affected fines: the greater the UCS, the lower the fines due to microcrack extension.
169            Corneal clarity was better in the UCS group at 21 (P = .008) and 30 days (P = .002), but n
170 hat in P. carinii, translation begins in the UCS to produce a pre-MSG protein, which is subsequently
171 ne if translation of MSG mRNAs begins in the UCS, polyclonal antiserum was raised against the 123-ami
172 s show that the addition of VA increases the UCS continuously in saturated conditions.
173     Given that few if any MSG mRNAs lack the UCS, the correspondence between the UCS/MSG junctions in
174 baculovirus carrying an MSG gene lacking the UCS expressed a nonglycosylated 130-kDa protein.
175  (CS - UCS), as well as presentations of the UCS alone were included to assess UCR diminution.
176  techniques to study the interactions of the UCS and Central domains with the myosin motor domain.
177   To determine the chaperone activity of the UCS and Central domains, we used two different methods:
178  loop altered the secondary structure of the UCS domain (by decreasing the alpha-helix content), lead
179 r understand the chaperoning function of the UCS domain of the UNC-45B chaperone, we engineered mutat
180        To explore the functional role of the UCS in the trafficking of MSG, the nucleotide sequence e
181                           The results of the UCS model's performance revealed that R exceeded 0.9 in
182                 The inhibitory effect of the UCS retrieval manipulation on cocaine-priming-induced re
183  did not contribute to the attachment of the UCS to the 5' ends of MSG mRNAs.
184        In Experiment 1, the intensity of the UCS was decreased after acquisition for one group (deval
185            In Experiment 2, the value of the UCS was increased after acquisition for one group (infla
186 is modified by conscious expectations of the UCS, conditioned diminution of the UCR can be expressed
187 the myosin head with high affinity, only the UCS domain displays chaperone activity.
188 d memory retrieval-extinction procedure, the UCS memory retrieval manipulation decreases renewal and
189                    In the current study, the UCS of the MSG from human-derived P. carinii was obtaine
190 ication of the second method showed that the UCS domain but not the Central domain prevents the therm
191 It has previously been demonstrated that the UCS domain is responsible for the chaperone-like propert
192    Using the first method, we found that the UCS domain is sufficient to prevent misfolding of a titi
193 S probability or explicit knowledge that the UCS intensity had changed.
194 S probability or explicit knowledge that the UCS intensity had changed.
195 Southern blot analysis demonstrated that the UCS was present in a single copy per genome, whereas mul
196                        Here we show that the UCS was present in one copy per haploid genome, but that
197  the genome indicates that attachment to the UCS is both necessary and sufficient for transcription o
198    One possibility is that attachment to the UCS locus is required for the transcription of an MSG ge
199 ith respect to the MSG genes attached to the UCS locus.
200 er diminution of the UCR was observed to the UCS when it followed the CS+ compared to when it followe
201 d a different set of MSG genes linked to the UCS, suggesting that UCS-MSG junctions are formed by rec
202 s to the amygdala will be inhibited when the UCS is expected, but this has not been found during fear
203                   The mechanism by which the UCS becomes part of different MSG mRNAs is not obvious b
204               Insect cells infected with the UCS-MSG hybrid gene expressed a 160-kDa protein which wa
205 vation, and movement pattern in men with the UCS.
206 e surface methodology (RSM) to estimate the (UCS, MPa) of cohesive soil stabilized with cement and li
207                                         This UCS leader sequence is necessary for initiation of trans
208 le common mechanism for the function of this UCS (UNC-45/CRO1/She4p) protein family.
209 side the cleft binding epitope accessible to UCS binding.
210 ng, whereas the population that responded to UCS did not change.
211 ifferent MSG genes were linked to the unique UCS locus in different members of a P. c. carinii popula
212 owed that expression of these mutants in USC/UCS cell lines enhanced tumor-initiating capacity, drove
213    Improvement in pain score was better with UCS than AMT (P value: .012, .002, and .012 on days 7, 1
214 G junctions in the genome were compared with UCS/MSG junctions in mRNA.
215 onstrates that the CCEP for individuals with UCS is feasible and effective, improving muscle activati
216         The study enrolled 536 patients with UCS and 101 patients with OCS, with 449 and 90 eligible,
217        Primary analysis was on patients with UCS, distributed as follows: 40% stage I, 6% stage II, 3
218                 Among eligible patients with UCS, PC was assigned to 228 and PI to 221.
219             With 264 events in patients with UCS, the power for an overall survival (OS) hybrid nonin
220 tential treatment approach for patients with UCS.
221                            The fission yeast UCS protein (Rng3p) concentrates in nodes containing myo
222 determined the atomic structure of the yeast UCS protein, She4p, at 2.9 A resolution.

 
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