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1                                              V. vulnificus infections were reported to the Centers fo
2                                              V. vulnificus inoculated into iron dextran-treated mice
3                                              V. vulnificus is a gram-negative bacterium, considered o
4                                              V. vulnificus is able to use host iron sources such as h
5                                              V. vulnificus mutants with varying effector domain conte
6                                              V. vulnificus strains containing pGTR902 were inoculated
7                                              V. vulnificus was exposed to structural and vegetation a
8                                              V. vulnificus wound infections due to seawater exposure
9                                              V. vulnificus-associated diseases are noted for the rapi
10        In Eastern USA between 1988 and 2018, V. vulnificus wound infections increased eightfold (10-8
11  shifted northwards 48 km p.a. By 2041-2060, V. vulnificus infections may expand their current range
12                                 By 2081-2100 V. vulnificus infections may be present in every Eastern
13                Results showed that ca. 10(3) V. vulnificus bacteria/gram of oyster and higher concent
14  the rtxA1 gene that encodes MARTX(Vv) in 40 V. vulnificus Biotype 1 strains and found four distinct
15   In a survey of the 16S rRNA genotype in 67 V. vulnificus human clinical and nonclinical strains, we
16                               We examined 69 V. vulnificus biotype 1 strains that were genotyped by s
17 thal factor or when naturally delivered as a V. vulnificus MARTX toxin led to loss of mitochondrial m
18 is study, we have cloned and characterized a V. vulnificus type IV pilin (PilA) that shares extensive
19 lated outer membrane protein purified from a V. vulnificus fur mutant had 53% homology with the first
20    Using TnphoA mutagenesis, we identified a V. vulnificus CPS locus, which included an upstream ops
21 sults discussed here confirmed homology of a V. vulnificus CPS locus to the group 1 CPS operon in Esc
22             Phenotypic characterization of a V. vulnificus vvpD mutant, constructed by allelic exchan
23 ilD) mutant of Pseudomonas aeruginosa with a V. vulnificus genomic library.
24 bacteriophages as therapeutic agents against V. vulnificus in an iron-dextran-treated mouse model of
25 hat estrogen is providing protection against V. vulnificus lipopolysaccharide-induced endotoxic shock
26             Exposure to wildfire ash altered V. vulnificus growth and gene expression, depending on t
27 sular genetics and antigenic diversity among V. vulnificus strains.
28                   Both V. vulnificus CPS and V. vulnificus LPS induced inflammation-associated cytoki
29 ered for orthologs of HapR in V. harveyi and V. vulnificus.
30 antibiotic-resistant V. parahaemolyticus and V. vulnificus, including V. parahaemolyticus pandemic st
31 ains), V. parahaemolyticus (30 strains), and V. vulnificus (10 strains) to determine the accuracy of
32  a significant reservoir for species such as V. vulnificus.
33                                         Both V. vulnificus CPS and V. vulnificus LPS induced inflamma
34  localized and systemic infections caused by V. vulnificus in animals.
35 t hospitalizations and deaths were caused by V. vulnificus infection, and most patients were white me
36 inflammation-mediated septic shock caused by V. vulnificus is strongly associated with liver disease,
37 odel for studying systemic disease caused by V. vulnificus.
38 pedes host cell rounding and lysis caused by V. vulnificus.
39 erall levels of these molecules expressed by V. vulnificus isolates.
40 uction of the intrinsic apoptosis pathway by V. vulnificus.
41  septicemia in humans, secretes a PFT called V. vulnificus hemolysin (VVH), which contains a single C
42 hages were effective against three different V. vulnificus strains with various degrees of virulence,
43                        However, two distinct V. vulnificus genotypes or alleles were associated with
44 road exopeptidase activity which may enhance V. vulnificus invasiveness by altering peptides involved
45 tant, containing the transposon and flanking V. vulnificus DNA was cloned, and a probe complementary
46                                          For V. vulnificus, the ability to acquire iron from the host
47 at but were not significantly attenuated for V. vulnificus virulence in mice.
48            This domain is also essential for V. vulnificus pathogenesis during intestinal infection.
49 n of fatty acid metabolism are essential for V. vulnificus to be able to cause disease in mammalian h
50  a niche marker, not always through Fur, for V. vulnificus controlling its entire life cycle.
51 Using the rat, we have developed a model for V. vulnificus endotoxic shock that mimics the sexually d
52  The effector domain region was required for V. vulnificus to inhibit phagocytosis by J774 macrophage
53 ndividuals that develop endotoxic shock from V. vulnificus are males.
54 , TcdA, TcnA, and TcsL; putative toxins from V. vulnificus, Yersinia sp., Photorhabdus sp., and Xenor
55 ransport genes are regulated by iron, and in V. vulnificus, transcriptional regulation by iron depend
56                   The role of the capsule in V. vulnificus biofilms was examined under a variety of c
57 hat a wide variety of capsular carbotypes in V. vulnificus may be readily distinguished by the HPAE f
58 ociated with both CPS and rEPS expression in V. vulnificus, designated the wcr (capsular and rugose p
59  gene is essential for capsule expression in V. vulnificus.
60 that the HlyU protein, a virulence factor in V. vulnificus CMCP6, up-regulates the expression of VV20
61 U-dependent expression of virulence genes in V. vulnificus.
62 n future studies of the role of heme iron in V. vulnificus pathogenesis.
63 xia, has yet to be extensively researched in V. vulnificus.
64  and the extracytoplasmic stress response in V. vulnificus, mutants with defined mutations in rseB an
65 or pilus assembly and cytolysin secretion in V. vulnificus.
66 or the function of the TtpC2-TonB2 system in V. vulnificus.
67  revealing an important role for the T6SS in V. vulnificus ecology.
68 onfirm that phase variation and virulence in V. vulnificus correlate with the amount of CPS expressed
69  mice differed with respect to the infecting V. vulnificus strain.
70                               Interestingly, V. vulnificus infections disproportionately affect males
71 experiments in a murine model of intravenous V. vulnificus infection demonstrated that expression of
72 ences in virulence among naturally occurring V. vulnificus can be explained by diverse abilities to r
73                               The ability of V. vulnificus to cause disease is linked to the producti
74  Inactivation of pilA reduces the ability of V. vulnificus to form biofilms and significantly decreas
75 ed a genome-wide transcriptional analysis of V. vulnificus growing at three different iron concentrat
76 ar typing systems have shown associations of V. vulnificus genotypes and the environmental or clinica
77             We report here the first case of V. vulnificus primary bacteremia due to raw shellfish co
78                              Recent cases of V. vulnificus infections in Los Angeles County suggest t
79 port are present, suggesting that the CPS of V. vulnificus is lipid linked.
80            Here, using a 30-year database of V. vulnificus cases for the Eastern USA, changing diseas
81 cutaneously with 10 times the lethal dose of V. vulnificus and injected intravenously, either simulta
82 2 X 10(9) colony-forming unit (high dose) of V. vulnificus was administered through a mini-laparotomy
83  highly similar to the putative epimerase of V. vulnificus.
84                   The projected expansion of V. vulnificus wound infections stresses the need for inc
85 dicate that an important virulence factor of V. vulnificus is undergoing significant genetic rearrang
86 nce potential between these two genotypes of V. vulnificus.
87 16S rRNA gene, there are two major groups of V. vulnificus designated types A and B.
88 ines the growing international importance of V. vulnificus, particularly in the context of coastal wa
89 sertion mutagenesis in a clinical isolate of V. vulnificus to find genes necessary for virulence, and
90 olved to facilitate the aquatic lifestyle of V. vulnificus but that their emergence also resulted in
91 les are present on the lipopolysaccharide of V. vulnificus, are required for full motility and biofil
92 ge of human-derived peptides by PGI-LysAP of V. vulnificus using three approaches: (i) a quantitative
93 oculated iron dextran-treated mouse model of V. vulnificus disease, was hypersensitive to the fatty a
94 us in an iron-dextran-treated mouse model of V. vulnificus infection.
95 capsulated and nonencapsulated morphotype of V. vulnificus.
96    We previously constructed a fur mutant of V. vulnificus which constitutively expresses at least tw
97                  The hupA deletion mutant of V. vulnificus will be helpful in future studies of the r
98  variants and genetically defined mutants of V. vulnificus M06-24/O was examined by using a CPS-speci
99 k correlates with seasonally high numbers of V. vulnificus bacteria during the summer months.
100 ersus genetic deletions in the CPS operon of V. vulnificus.
101 ntial role of capsule in the pathogenesis of V. vulnificus.
102 hich may contribute both to pathogenicity of V. vulnificus and to its survival under adverse environm
103 ificantly reduced the virulence potential of V. vulnificus, while deletion of duf1 or abh accelerated
104 pulsed-field gel electrophoresis profiles of V. vulnificus strains isolated from blood and oysters as
105              In contrast, the growth rate of V. vulnificus in alkaline peptone water was greater than
106 l gene for VuuA, the vulnibactin receptor of V. vulnificus.
107 ructural gene for HupA, the heme receptor of V. vulnificus.
108           Analysis of the promoter region of V. vulnificus hupA showed a sequence homologous to the c
109          We previously described two sets of V. vulnificus strains with different levels of virulence
110 saccharide of a related pathogenic strain of V. vulnificus (MO6-24) the structure of which was recent
111 accharide purified from a virulent strain of V. vulnificus 6353 did not show cross reactivity with an
112 enome of an encapsulated, clinical strain of V. vulnificus.
113                         Biotype 1 strains of V. vulnificus are most commonly associated with human in
114 pimerase is common to at least 10 strains of V. vulnificus that each express a serologically distinct
115 lar polysaccharides of pathogenic strains of V. vulnificus, there are distinct differences in the det
116 clinical as well as environmental strains of V. vulnificus.
117 e isolates with colony morphology typical of V. vulnificus identified 75% as V. sinaloensis.
118 nked to a previously unrecognized variant of V. vulnificus designated biotype 3.
119 lluscan shellfish are the primary vectors of V. vulnificus disease.
120 asize the importance of CPS for virulence of V. vulnificus and establish a correlation between CPS ex
121 nd septicemia in humans and the virulence of V. vulnificus has been strongly associated with encapsul
122 ng that in addition to capsule, virulence of V. vulnificus requires type IV pili and/or extracellular
123 mes cannot strictly predict the virulence of V. vulnificus strains and further investigation is neede
124 se, exopolysaccharides, and the virulence of V. vulnificus.
125 e pili, suggesting that the pili observed on V. vulnificus are of the type IV class.
126 ulence properties compared to those of other V. vulnificus strains.
127 ted Vibrio species were V. parahaemolyticus, V. vulnificus, and V. alginolyticus; both surveillance s
128 r species (V. cholerae, V. parahaemolyticus, V. vulnificus, and V. mimicus).
129 us Vibrio: V. cholerae, V. parahaemolyticus, V. vulnificus, and V. mimicus.
130        We conclude that V. parahaemolyticus, V. vulnificus, V. cholerae and subpopulations that harbo
131 prevalence of total Vibrio parahaemolyticus, V. vulnificus and V. cholerae and select genes associate
132 inolysis within the zoonotic marine pathogen V. vulnificus.
133 ingly, on-bottom oysters had more pathogenic V. vulnificus than off-bottom oysters.
134 minated raw oysters, the incidence of severe V. vulnificus disease is low.
135 ated with human infections and that a single V. vulnificus strain, evidenced by pulsed-field gel elec
136                                   The single V. vulnificus PecS binds two sites within the pecS-pecM
137                                      All six V. vulnificus strains caused identical skin lesions in s
138 d that V. sinaloensis grew more rapidly than V. vulnificus in seawater at temperatures </= 30 degrees
139          These studies provide evidence that V. vulnificus CPS directly stimulates the expression and
140         Through complementation we show that V. vulnificus is capable of using different TtpC2 protei
141                                          The V. vulnificus pilA gene is part of an operon and is clus
142 sion analysis localized one promoter for the V. vulnificus hupA gene.
143 ence of the 77-kDa protein purified from the V. vulnificus fur mutant had 67% homology with the first
144                  An internal deletion in the V. vulnificus hupA gene, done by using marker exchange,
145                  An internal deletion of the V. vulnificus vuuA gene resulted in the loss of expressi
146 as part of all N-glycans correlates with the V. vulnificus MARTX toxin having broad specificity and t
147 ablished that the MARTXVv toxin is linked to V. vulnificus dependent induction of apoptosis, but the
148 tood; however, its phenotypic resemblance to V. vulnificus and the possibility that it could outcompe
149 ecrosis factor alpha elicited in response to V. vulnificus and measured in cell supernatants were not
150 ess proinflammatory cytokines in response to V. vulnificus.
151 lear cell inflammatory cytokine responses to V. vulnificus.
152 tive stress have increased susceptibility to V. vulnificus septicemia.
153                      The prevalence of total V. vulnificus and the siderophore-related viuB gene also
154 t common rtxA1 gene variant in clinical-type V. vulnificus encodes a toxin with reduced potency and i
155 terface wildfire ashes on Vibrio vulnificus (V. vulnificus) growth and gene expression using transcri
156 lated from blood and oysters associated with V. vulnificus disease.
157 ntly enhances survival of mice infected with V. vulnificus by alleviating hepatic and renal dysfuncti

 
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