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1 thelial cells expressing the Galphas-coupled V2 vasopressin receptor.
2 seven-transmembrane receptor, the Gs-coupled V2 vasopressin receptor.
3 for sequestration and desensitization of the V2 vasopressin receptor.
4 ructure-function relationship studies of the V2 vasopressin receptor, a prototypical G(s)-coupled rec
5 ion mutations in rhodopsin, cone opsins, the V2 vasopressin receptor, ACTH receptor, and calcium-sens
6 ligand-induced endocytosis of two GPCRs: the V2 vasopressin receptor and beta-2 adrenergic receptor,
7 selectivity versus the related V1a, V1b, and V2 vasopressin receptors and short half-life: agonists 3
8 ffect of oral administration of a nonpeptide V2 vasopressin receptor antagonist, OPC 31260, was there
9                                The effect of V2 vasopressin receptor antagonist, OPC-31260, was then
10                         Using the GS-coupled V2 vasopressin receptor as a model system, we systematic
11                                    The human V2 vasopressin receptor belongs to the superfamily of G
12 e permitted an alpha s-coupled receptor (the V2 vasopressin receptor but not the beta 2-adrenergic re
13 is observed with other receptors such as the V2 vasopressin receptor, but did couple with the clathri
14 bition) by vasopressin-induced activation of V2 vasopressin receptors co-expressed at similar levels.
15                  The C terminus of the human V2 vasopressin receptor contains multiple phosphorylatio
16       The extracellular portion of the human V2 vasopressin receptor contains one site susceptible to
17  the two classes of receptors (beta(2)AR and V2 vasopressin receptors), demonstrate reversal of the p
18                                          The V2 vasopressin receptor expressed transiently was glycos
19 hosphatidylinositol hydrolysis), whereas the V2 vasopressin receptor is selectively coupled to Gs (bi
20 ty of this approach, a series of nine mutant V2 vasopressin receptors known to be responsible for X-l
21 )-293 cells, stimulation of the beta(2)AR or V2 vasopressin receptor leads, respectively, to transien
22 urface levels of two folding-defective human V2-vasopressin receptor mutants, which were susceptible
23  inflammatory genes and disappearance of the V2 vasopressin receptor, resulting in loss of AQP2 (conf
24 synthetic phosphopeptide mimicking the human V2 vasopressin receptor tail that binds and functionally
25 vasopressin-regulated water channel) and the V2 vasopressin receptor, that are important to regulated
26                                          The V2 vasopressin receptor undergoes ligand-induced sequest
27                        Palmitoylation of the V2 vasopressin receptor (V2R) and its functional role we
28 -function mutations in the gene encoding the V2 vasopressin receptor (V2R) cause nephrogenic syndrome
29                             The internalized V2 vasopressin receptor (V2R) expressed in HEK 293 cells
30 ease caused by inactivating mutations in the V2 vasopressin receptor (V2R) gene that result in the lo
31                           Stimulation of the V2 vasopressin receptor (V2R) in cultured cells or in vi
32                                          The V2 vasopressin receptor (V2R) plays a key role in the ma
33  identify molecules that might contribute to V2 vasopressin receptor (V2R) trafficking or signaling,
34  roles in the regulation and function of the V2 vasopressin receptor (V2R), a G protein-coupled recep
35 t they had gain-of-function mutations in the V2 vasopressin receptor (V2R).
36 boxy-terminal peptide derived from the human V2 vasopressin receptor (V2Rpp).
37 ing itineraries of wild type (WT) and mutant V2 vasopressin receptors (V2Rs) in polarized Madin-Darby
38                  For class B receptors (e.g. V2 vasopressin receptors), which recycle slowly, beta-ar