ライフサイエンスコーパス: VD

1 VD class GABAergic neurons are generated in the late L1

2 VD could therefore be a strong candidate for liver cance

3 VD deficiency is associated with liver fibrosis progress

4 VD in the 6 x 6 mm(2) SCP scans, the DCP (all scans), an

5 VD motor neurons develop after the L1 moult; they take o

6 VD of the radial peripapillary capillaries was evaluated

7 VD showed a direct relationship with geographic altitude

8 VD(3) incorporation in MM was not influenced by the FA u

9 1,25(OH)(2)VD(3) binds the nuclear VD receptor (VDR) that binds tar

10 These findings suggest that 1,25(OH)(2)VD(3) can affect human immune responses by regulating FO

11 Although 1,25(OH)(2)VD(3) can promote FOXP3 expression in CD4(+) T cells wit

12 1,25(OH)(2)VD(3) induced CXCL12 expression in vivo and in vitro in

13 is unknown whether this effect of 1,25(OH)(2)VD(3) is mediated through direct binding of VDR to the F

14 nstrated 1,25-dihydroxyvitamin D (1,25(OH)(2)VD(3)) increased the number of BCR-ABL ALL cells only wh

15 vity by such VDREs in response to 1,25(OH)(2)VD(3).

16 1,25-Dihyroxyvitamin D(3) [1,25(OH)(2)VD(3)] affects the functions of immune cells including T

17 ity was the main mechanism for ajoenes and 2-VD.

18 ,3-dithiin (3-VD), 2-vinyl-4H-1,2-dithiin (2-VD) and (E)- and (Z)-ajoene, which are the most importan

19 )- and (Z)-ajoene, 2-vinyl-4H-1,2-dithiin (2-VD), diallyl sulphide (DAS) and diallyl disulphide (DADS

20 ockdown of miR-98 led to a reduction of 1,25-VD anti-growth effect and overexpression of miR-98 suppr

21 te cancer and a potential biomarker for 1,25-VD anti-tumor action.

22 mical carcinogenesis to investigate how 1,25-VD prevents malignant transformation.

23 ing of the molecular mechanism by which 1,25-VD prevents tumorigenesis remains incomplete.

24 In this model, 1,25-VD promoted expression of the DNA repair genes RAD50 and

25 Correspondingly, 1,25-VD protected cells from genotoxic stress and growth inhi

26 growth inhibitive miR-98 is induced by 1,25-VD provides a potential therapeutic target for prostate

27 arker potential of miR-98 in predicting 1,25-VD response.

28 R-98 levels in blood are increased upon 1,25-VD treatment in mice suggesting the biomarker potential

29 ced by 1alpha,25-dihydroxyvitamin D(3) (1,25-VD) in LNCaP.

30 cts of 1alpha,25-dihydroxyvitamin D(3) (1,25-VD) in tumorigenesis.

31 ormation that could not be prevented by 1,25-VD, defining an essential role for VDR in mediating the

32 Mechanistic dissection revealed that 1,25-VD-induced miR-98 is mediated through both a direct mech

33 in mediating the anticancer effects of 1,25-VD.

34 rials for the encapsulation of vitamin D(3) (VD(3)).

35 e (DATS), allicin, 3-vinyl-4H-1,3-dithiin (3-VD), 2-vinyl-4H-1,2-dithiin (2-VD) and (E)- and (Z)-ajoe

36 Overall response rates were 73% (VD), 80% (VTD), and 70% (VMP).

37 for 2 months; D2 (n=22), animals were fed a VD-deficient AIN-93G diet and were kept under incandesce

38 eficiency were randomized into two groups, a VD group (n = 29) and a placebo group (n = 29).

39 rmal anterior neurite, which caused aberrant VD commissure patterning along the A/P axis.

40 uclear hormone receptor, to prevent aberrant VD synaptic wiring in later larval and adult stages.

41 In addition, VD anterior neurites had underextension defects in the V

42 Additionally, VD-iTreg cells suppressed the proliferation of target CD

43 cycline-dependent transactivator mice (Adipo-VD) to stimulate adipose tissue-specific lymphangiogenes

44 is in murine adipose tissue, and obese Adipo-VD mice exhibited enhanced glucose clearance, lower insu

45 0(+) macrophages were reduced in obese Adipo-VD s.c. adipose tissue with evidence of increased immune

46 On beta-3 adrenergic stimulation, Adipo-VD mice exhibited more rapid and increased glycerol flux

47 e follow up of puerperae undergoing ED after VD.

48 n practice patterns in determining LoS after VD.

49 and thickness measurements (ONH GBC), 3) all VD measurements from the macula and ONH (vessel density

50 ces GSH deficiency and epigenetically alters VD-biosynthesis pathway genes.

51 In eyes with AMD, VD decreases with age in the foveal (beta = -0.211, P <

52 onfirmed the presence of the biopolymers and VD(3) in the capsules.

53 after dehydration with FD compared to CD and VD methods.

54 gher sensory acceptability score than CD and VD murta.

55 Reflectance-compensated CD and VD parameters for both NFLP and SVC could be useful in t

56 ling of the postsynaptic apparatus in DD and VD neurons using targeted expression of the acetylcholin

57 which are divided into two subgroups: DD and VD.

58 beneficially altered epigenetic enzymes, and VD-metabolism genes in hepatocytes.

59 erstanding stability and formation of MM and VD(3) loading is an essential first step towards manipul

60 In addition, SD and VD were significantly lower in the DRL of subjects with

61 ship between signal strength index (SSI) and VD.

62 These data indicate that pathways of VC and VD are not independent but affect each other, and this e

63 pressions of key genes of interest in VC and VD functions.

64 d Rgn play important roles connecting VC and VD pathways in mice.

65 ferent between mice and humans during VC and VD synthesis and transportation.

66 with circulating WBC samples for both VC and VD variants (r = 0.78 and 0.75, respectively).

67 m 25(OH)VD levels < 30 ng/mL were defined as VD insufficiency/deficiency.

68 s unassisted vaginal delivery (VD), assisted VD, elective CS, and emergency CS (defined by before or

69 we observed that some postembryonically born VD neurons had a posterior neurite instead of a normal a

70 Sixty-two (52.5%) were born by VD, 22 by L-CS (18.6%), and 34 by E-CS (28.8%).

71 ndomized, double-blinded, placebo-controlled VD supplementation trial in pregnant women at high risk

72 re compared for SCN5A variants C (VC) and D (VD).

73 N) classes, the dorsal D (DD) and ventral D (VD) neurons, extend axons along both the dorsal and vent

74 imed to evaluate the influence of vitamin D (VD) deficiency on cardiac metabolism, morphology, and fu

75 tor for childhood asthma, whereas vitamin D (VD) has emerged as a modifiable prenatal exposure.

76 Vitamin D (VD) has immunomodulatory properties, but whether immune

77 Vitamin C (VC) and vitamin D (VD) have been widely used as the dietary supplements and

78 Vitamin D (VD) is a fat-soluble vitamin with high deficiency levels

79 Vitamin D (VD) is important in hepatic fibrogenesis in animal model

80 s, conditions associated with low Vitamin D (VD) levels.

81 pigenetic alterations that impair Vitamin D (VD) metabolism genes in the livers of HFD-fed mice.

82 Vitamin D (VD) supplementation has been shown to reverse these proc

83 ch individual voxel, the VD absorbed dose, D(VD), calculated assuming uniform density, was corrected

84 The D(VD) calculations showed a good agreement with D(3DRD).

85 d dose values, D(3DRD), were compared with D(VD) and D(VDd), using the relative difference Delta(VD/3

86 ues, D(3DRD), were compared with D(VD) and D(VDd), using the relative difference Delta(VD/3DRD).

87 density, was corrected for density, giving D(VDd).

88 Vitamin D3 (VD), a hydrophobic micronutrient, was successfully incor

89 The ICP and DCP VD were not significantly lower in the glaucoma group.

90 CFT, IRT, ORT, foveal SCP-VD, and foveal DCP-VD and a significant positive correlation with subfoveal

91 CFT, IRT, ORT, foveal SCP-VD, and foveal DCP-VD were significantly greater than those in the other gr

92 al (SCP-VD) and deep capillary plexuses (DCP-VD) of the foveal and parafoveal areas were examined in

93 ipapillary microvasculature showed decreased VD and flow in POAG with paracentral loss, supporting it

94 and term newborns after vaginal deliveries (VD) is still inconclusive and little is known on the cha

95 very defined as unassisted vaginal delivery (VD), assisted VD, elective CS, and emergency CS (defined

96 compared by delivery type: vaginal delivery (VD), cesarean section (CS) after labor (L-CS), or electi

97 reastfeeding compared with vaginal delivery (VD).

98 Delta(VD/3DRD) was less than 3.5%, except for the tumor of cas

99 elationship between voxel bin-averaged Delta(VD/3DRD) and density, rho: case 1 (Delta = -0.56rho + 0.

100 At the voxel level, density-binned Delta(VD/3DRD) and Delta(VDd/3DRD) were plotted against rho an

101 D(VDd), using the relative difference Delta(VD/3DRD).

102 At the voxel level, the Delta(VD/3DRD) range was 0%-14% for cases 1 and 2, and -3% to

103 vel, density-binned Delta(VD/3DRD) and Delta(VDd/3DRD) were plotted against rho and fitted with a lin

104 method with the Monte Carlo approach (Delta(VDd/3DRD) < 1.1%), but with a lesser extent for the tumo

105 At the voxel level, the Delta(VDd/3DRD) range decreased for the 3 clinical cases (case

106 Parkinson's disease (PD), vascular dementia (VD), senile dementia (SD), mild cognitive impairment (MC

107 ory evoked PSPs invade the ventral dendrite (VD), as well as the opposite where visual PSPs invade th

108 Perfusion density and vessel length density (VD) were examined from both the superficial capillary pl

109 e metrics, parafoveal vessel length density (VD), and perfusion density (PD) were corrected for magni

110 Vessel density (VD) and perfusion density (PD) in the SCP within the Ear

111 Both vessel density (VD) and the integrated OCTA by ratio analysis signal (IO

112 5.8%) AMD to measure retinal vessel density (VD) from the superficial capillary plexus in the foveal,

113 l capillary density (CD) and vessel density (VD) were calculated using a reflectance-compensated algo

114 y plexuses (DCP): parafoveal vessel density (VD), adjusted flow index (AFI), and percent area of nonp

115 deep capillary plexus (DCP) vessel density (VD), and foveal avascular zone (FAZ) size were measured

116 erimeter, nonperfusion area, vessel density (VD), and presence of intraretinal microvascular abnormal

117 The vessel density (VD), defined as the percentage area occupied by flow pix

118 (OCT-A) devices by comparing vessel density (VD), fractal dimension (FD), and foveal avascular zone (

119 ulate skeleton density (SD), vessel density (VD), fractal dimension (FD), and vessel diameter index (

120 c features on OCTA by way of vessel density (VD), skeletal density (SD), fractal dimension (FD), vess

121 Vessel density (VD), vessel length density (VLD), vessel diameter index

122 vessel tortuosity (VT), and vessel density (VD), were analyzed comprehensively.

123 talline lens thickness (LT), vitreous depth (VD), and axial length (AL), were measured and compared w

124 amethasone (RD) or bortezomib-dexamethasone (VD) but it is changing rapidly for 2 reasons.

125 of induction with bortezomib-dexamethasone (VD; n = 168; intravenous bortezomib 1.3 mg/m(2), days 1,

126 gmatism correlated with globally diminishing VD and PD, was more symmetrical for vertical than horizo

127 ented with an outbreak of vesicular disease (VD) that was associated with an increase in neonatal mor

128 navirus that causes acute vesicular disease (VD), that is clinically indistinguishable from foot-and-

129 -AMT PET parameters (volume of distribution [VD], characterizing tracer transport, and unidirectional

130 t removal of the regulatory variable domain (VD) in Drp1 enhances formation of a functional Drp1-Mff

131 In both SRL and DRL, VD (P = 0.0010 and P = 0.0003, respectively), VLD (P < 0

132 effect of freeze-drying (FD), vacuum drying (VD), convective drying (CD), microwave-vacuum drying (MV

133 In HbSS eyes, VD was lower in the DCP (47.7%, P = .008) but not in the

134 er than our proposed ED benchmarks following VD: 2 days after spontaneous vaginal deliveries (SVD) an

135 D: adjusted OR, 1.00; 95% CI, 0.89-1.13; for VD: adjusted OR, 1.11; 95% CI, 0.99-1.25; for SD: adjust

136 r ring superior quadrant was 12% greater for VD and 16% greater for PD versus that in the inferior qu

137 nner ring nasal quadrant was 40% greater for VD and 48% greater for PD versus that in the temporal qu

138 ve correlation between somatic mutations for VD-related genes and the TGF-beta pathway.

139 n of the liver, as an intermediate organ for VD metabolism, contributes partly to this deficiency.

140 This study implies a protective role for VD sufficiency throughout pregnancy, particularly in att

141 Furthermore, VD deficiency was related to increased cytokines release

142 t VD/F of 0.41 L and the monkey, the highest VD/F of 392.95 L.

143 The simple allometric human VD/F and MLP-corrected Cl/F were 2311.51 L and 51.35 L/h

144 The serum levels of 25-hydroxy VD, TGF-beta1, TIMP-1, MMP2 and MMP9 were measured at ba

145 iffness and inflammation but did not improve VD levels.

146 In VD class neurons, which normally do not remodel, the tra

147 and higher lactate dehydrogenase activity in VD-deficient animals.

148 rease in oxidative stress and alterations in VD regulatory genes.

149 with a statistically significant decrease in VD and PD within all Early Treatment Diabetic Retinopath

150 actional shortening and ejection fraction in VD-deficient animals.

151 such as pde-4 to keep the cAMP levels low in VD.

152 The reflectance compensation step in VD calculation significantly improved repeatability, nor

153 t effect on the interindividual variation in VD.

154 expressed in DD neurons but is repressed in VDs by UNC-55/COUP-TF.

155 In addition, we observe that increasing VD increases the amount of surfactant delivered to the a

156 rs should exercise caution when interpreting VD data from OCTA scans.

157 e were no significant differences in isotime VD/VT or transcutaneous Pco(2) among treatments.

158 nimals were fed an AIN-93G diet with 1000 IU VD/kg of chow and were kept under fluorescent light for

159 0.56; 95% CI = 0.31-1.00) or early and late VD sufficiency (adjusted odds ratio = 0.36; 95% CI = 0.1

160 l VD insufficiency, those with early or late VD sufficiency (adjusted odds ratio = 0.56; 95% CI = 0.3

161 larger AVC, VT, FAZ-A, and FAZ-CI and lower VD than those in the control group (P < 0.001 for all).

162 Eyes with exudative AMD demonstrated lower VD, especially in the parafoveal (29.8% +/- 6.3% vs 33.0

163 The mouse showed the lowest VD/F of 0.41 L and the monkey, the highest VD/F of 392.9

164 s were evaluated that combined 1) all macula VD and thickness measurements (Macula GBC), 2) all ONH V

165 Decreasing macular VD on OCTA correlated with increasing peripheral capilla

166 Among the overall macular VD parameters, the SVC VD had the best diagnostic accura

167 cipants showed significantly reduced macular VD, PD, and GC-IPL thickness compared with MCI and contr

168 g (T(loc) = 39 degrees C) and during maximal VD elicited by heating (T(loc) = 43 degrees C) and 28 mM

169 Repeated RIPC improves maximal VD but does not affect NO-mediated VD in the cutaneous m

170 However, the maximal VD was augmented (Pre: 2.5 +/- 0.2, Post: 3.8 +/- 0.5 fl

171 caused by AD (MCI-A), MCI caused by VaD (MCI-VD), and MCI caused by ODs (MCI-O).

172 Mean VD, FD, and FAZ values between the instruments were comp

173 The mean VD in the SCP and DCP was 46.94% (+/-3.11%) and 52.48% (

174 s maximal VD but does not affect NO-mediated VD in the cutaneous microvasculature.

175 ctions in both skeletal muscle mass and Mito(VD) have been reported following mountaineering expediti

176 al muscle mitochondrial volume density (Mito(VD)) over equivalent normoxic training.

177 lysed for mitochondrial volume density (Mito(VD)), capillarity, fibre types and respiratory capacity

178 determined that intermyofibrillar (IMF) Mito(VD) was augmented (P = 0.028) by 11.5 +/- 9.2% from Pre

179 study demonstrates that skeletal muscle Mito(VD) may increase with 28 days acclimation to 3454 m.

180 drial characteristics, with emphasis on Mito(VD), while minimizing changes in energy balance.

181 re was no change in subsarcolemmal (SS) Mito(VD).

182 As a result, total Mito(VD) (IMF + SS) was increased (P = 0.031) from 6.20 +/- 1

183 ons, capillary-to-fibre ratio and total Mito(VD) increased (P < 0.05) following ET (18 +/- 16 and 43

184 mum (ACD), 14 mum (AD), 13 mum (LT), 14 mum (VD), and 16 mum (AL).

185 drants; however, especially more so nasally (VD: 0.63; P < .001; PD: 0.0089; P = .001).

186 Performances of NFLP-VD and SVC-VD were similar to the corresponding CD param

187 when fmi-1 was expressed exclusively in non-VD neighboring neurons, suggesting a cell nonautonomous

188 DD, but not VD, MNs reverse their cellular polarity in a development

189 1,25(OH)(2)VD(3) binds the nuclear VD receptor (VDR) that binds target DNA sequences known

190 re validated clinically, where an absence of VD supplementation was associated with low TGF-beta path

191 Our analyses of VD deprivation (VDD) in in vivo models of liver tumor fo

192 Bioaccessibility of VD is influenced by formation of mixed micelles (MM) dur

193 However, potential contributions of VD to liver tumor progression in the context of TGF-beta

194 Upon correction of VD levels, TGF-beta1 and TIMP-1 levels were decreased, a

195 The axon and synaptic defects of VD neurons could be rescued when fmi-1 was expressed exc

196 ng food structures for improving delivery of VD.

197 This study sought to determine the effect of VD supplementation on serum fibrotic markers in chronic

198 r when performing retinal OCTA evaluation of VD values.

199 hich is essential for inhibitory function of VD-iTreg cells.

200 and left ventricular mass after 4 months of VD deficiency.

201 The proportions of VD insufficiency/deficiency were 72.5%, 91.3%, and 86.5%

202 reduced (42.3% compared to 86.1%) release of VD in simulated gastric fluid and an increased (36.0% co

203 The slope of VD versus SSI was greatest when signal strength was adju

204 psulation increased the thermal stability of VD(3), and FTIR confirmed the presence of the biopolymer

205 Blood samples for 25(OH)VD and the procollagen type III N-terminal peptide (P3NP

206 Serum 25(OH)VD levels < 30 ng/mL were defined as VD insufficiency/de

207 The 25(OH)VD levels decreased from 26.3 +/- 10.7 ng/mL at baseline

208 ntly increased, which catabolizes both 25(OH)VD(3) and 1alpha,25-hydroxyvitaminD(3).

209 he liver is the principal site for the 25(OH)VD(3) biosynthesis.

210 thione (GSH) and 25-hydroxyvitamin D3 (25(OH)VD(3)).

211 benefits of GSH treatment in reducing 25(OH)VD(3)-deficiency.

212 als (DAA) would increase 25-hydroxyVD [25(OH)VD] levels.

213 the effects of signal strength reduction on VD measurements on the Optovue/AngioVue (Optovue, Inc, F

214 ckness measurements (Macula GBC), 2) all ONH VD and thickness measurements (ONH GBC), 3) all VD measu

215 d a combination of macular thickness and ONH VD measurements as the greatest contributors.

216 postpartum and compared outcomes after CD or VD, including foreign language publications, were identi

217 No correlation exists between OAG and PD or VD.

218 ew agents (except pomalidomide) to the RD or VD regimens were superior to the double combinations in

219 DH (3D-RD), and a DK approach (VoxelDose, or VD).

220 MP did not appear to offer an advantage over VD in transplantation-ineligible patients with myeloma t

221 For eyes with DME, parafoveal VD in the superficial layer at baseline was an independe

222 mong all biomarkers, higher inner parafoveal VD in the superficial layer at baseline correlated most

223 predict visual improvement, outer parafoveal VD in the superficial layer at the baseline showed the l

224 Among all POAG eyes, peripapillary VD and IOS of the affected hemisphere correlated signifi

225 racentral group showed reduced peripapillary VD (38.0 +/- 2.0%, 35.0 +/- 2.2%, respectively; P = 0.00

226 presence of IRMA, and reduced peripapillary VD in the superior temporal and inferior temporal region

227 FAZ area and temporal peripapillary VD are predictors of DR progression.

228 Any prolonged LoS post VD (LoS > ED) should be reviewed and audited if need be.

229 s among mothers with early and late prenatal VD insufficiency, those with early or late VD sufficienc

230 e combined effect of early and late prenatal VD status in during pregnancies in women with and withou

231 ariable representing early and late prenatal VD sufficiency (25(OH)D level >= 30 ng/mL) status during

232 We also examined the effect of prenatal VD level on early life asthma or recurrent wheeze progre

233 specific inhibitors of caspase activation (Q-VD) and NLRP3 activation (MCC950).

234 M-54 or a combination of necrostatin-1 and q-VD-OPh.

235 However, Q-VD-OPh, a potent inhibitor of caspase activity provided

236 ed by the broad-spectrum caspase inhibitor Q-VD-OPh and was associated with both caspase-3 and caspas

237 rtyl-[-2,6-difluorophenoxy]-methyl ketone (Q-VD-OPh).

238 Of note, the administration of Q-VD-OPh showed no protective effect on oligomycin A-induc

239 and peripheral VF loss groups showed reduced VD (P < 0.001 and P = 0.009, respectively) and IOS (P <

240 icipants) demonstrated significantly reduced VD, SD, and FD and greater VDI in patients with FEVR com

241 nstruments are not interchangeable regarding VD, FD, and FAZ for both the superficial and deep capill

242 Regional VD and IOS were measured from the affected hemisphere co

243 Over 80% of patients remained VD insufficient/deficient.

244 Retinal VD is decreased in eyes with exudative AMD compared with

245 presence of central GA also impacted retinal VD and FAZ morphology.

246 Macular retinal VD, ganglion cell complex (GCC) thickness, and visual fi

247 aucoma group, the SVC and all-plexus retinal VD (mean +/- standard deviation: 47.2%+/-7.1% and 73.5%+

248 be considered when assessing changes in RPC VD in glaucoma and other ocular diseases.

249 Custom software was used to quantify RPC VD.

250 re needed to investigate whether reduced RPC VD and the factors that affect it are associated with an

251 ltivariate model associated with reduced RPC VD were older age (beta = -0.0123 per decade; SRC = -0.2

252 dentify systemic factors associated with RPC VD with a significance level set at 0.05.

253 ticipants showed significantly decreased SCP VD and PD in the 3-mm ring (P = 0.001 and P = 0.002, res

254 red with MCI and significantly decreased SCP VD and PD in the 3-mm ring (P = 0.008 and P = 0.004, res

255 d P = 0.004, respectively) and decreased SCP VD in the 6-mm circle (P = 0.047) compared with MCI and

256 There was no difference in SCP VD or PD between MCI and controls (P > 0.05).

257 Mean SCP VD values in 3 x 3-mm(2) scans were significantly higher

258 e correlation with CFT, IRT, ORT, foveal SCP-VD, and foveal DCP-VD and a significant positive correla

259 In group I, CFT, IRT, ORT, foveal SCP-VD, and foveal DCP-VD were significantly greater than th

260 er, and vessel densities of superficial (SCP-VD) and deep capillary plexuses (DCP-VD) of the foveal a

261 The SD, VD, and FD of the parafoveal capillaries were lower in u

262 Subsequent supplementing VD led to restoration of TGF-beta member expression with

263 Reflectance-compensated SVC VD measurement by PR-OCTA detected glaucoma with high ac

264 e glaucoma participants, the hemispheric SVC VD values were highly correlated with the corresponding

265 g the overall macular VD parameters, the SVC VD had the best diagnostic accuracy as measured by the a

266 Performances of NFLP-VD and SVC-VD were similar to the corresponding CD parameters.

267 dverse events were more common with VTD than VD or VMP.

268 this study provides additional evidence that VD plays an important role in the reversal of hepatic fi

269 Our data indicate that VD deficiency is associated with energetic metabolic cha

270 that binds target DNA sequences known as the VD response element (VDRE).

271 ns, an anterior neurite that will become the VD axon extends along the anteroposterior (A/P) axis in

272 Phosphorylation of a PKA site bordering the VD disassembled the filamentous DeltaVD mutant and accel

273 The cdh-4 is expressed by the VD neurons and seems to function in the same genetic pat

274 his provides a biochemical mechanism for the VD-deficiency and potential benefits of GSH treatment in

275 P2 and MMP9 were significantly higher in the VD group than in the placebo group.

276 9 levels were significantly increased in the VD group.

277 in the VNC, we did not observe FMI-1 in the VD neurons themselves, suggesting that fmi-1 might be wo

278 th auditory PSPs being more prominent in the VD than visual PSPs in the LD.

279 and C-terminal alpha-helical segments in the VD-replacing linker converted Drp1 from constitutively a

280 sttranslational modifications in or near the VD alter the conformation of a membrane-proximal oligome

281 The expression of the VD metabolism genes CYP2R1 and CYP27A1 (25-hydroxylase),

282 During the early development of the VD neurons, an anterior neurite that will become the VD

283 nown details of the early development of the VD neurons, indicating that the neurite defects arose du

284 s, but whether immune cell expression of the VD receptor (VDR) impacts costimulatory blockade induced

285 onsistent with an autoinhibitory role of the VD, we identified Arg-376 in the Drp1 stalk domain as ne

286 es expressed in the proximal segments of the VD.

287 er the nitric oxide (NO) contribution to the VD response to local heating (P > 0.05).

288 For each individual voxel, the VD absorbed dose, D(VD), calculated assuming uniform den

289 howed lower relative gamma power compared to VD and E-CS (P = 0.005).

290 ymmetry index was higher in L-CS compared to VD and E-CS (P = 0.03).

291 Total VD (3-mm ETDRS circle) was lower in HbS variant eyes tha

292 , it is not expressed in the ventral D-type (VD) GABAergic motorneurons, which are defective in fmi-1

293 iated with ASD when compared with unassisted VD.

294 freeze- (FD), convective- (CD) and vacuum- (VD) drying.

295 we hypothesized that cutaneous vasodilatory (VD) function elicited by localized heating would be incr

296 The roles of dose volume (VD), flow rate, and multiple aliquot delivery are invest

297 defects in the VNC in fmi-1 animals, whereas VD commissure growth along the dorsoventral (D/V) axis o

298 Whether VD replacement during and after DAA therapy can improve

299 Fifty-four CHC patients with VD deficiency were randomized into two groups, a VD grou

300 ow-up, median progression-free survival with VD, VTD, and VMP was 14.7, 15.4, and 17.3 months, respec