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1                                              VGAT and calcium binding protein-28K immunoreactivities
2                                              VGAT appeared more evenly distributed in the striatal pa
3                                              VGAT immunofluorescent puncta were first seen sparsely i
4                                              VGAT immunoreactivity in the OPL was predominantly local
5                                              VGAT immunostaining was present at P0 and older ages in
6 ridization revealed coexpression of SNAP-25, VGAT (vesicular GABA transporter), and GAD65/67 (glutami
7    In contrast, DAMGO inhibited EPSCs in 46% VGAT neurons but did not elicit LTP in any VGAT neurons
8  biosynthesis genes (e.g. unc-25/GAD, unc-47/VGAT).
9                     Real-time imaging with a VGAT-pHluorin fusion provides a useful approach to explo
10 MAT2 (vesicular monoamine transporter 2) and VGAT (vesicular GABA transporter), consistent with vesic
11 T2- (vesicular glutamate transporter 2), and VGAT- (vesicular GABA transporter) mRNA in specific subr
12 utely dissociated retinas showed GAD(65) and VGAT immunoreactivity in both A-type and B-type horizont
13                  Specific GABA, GAD(65), and VGAT immunostaining was localized to horizontal cell bod
14 monstrate the presence of GABA, GAD(65), and VGAT in horizontal cells of the guinea pig retina, and s
15 econd, the activity of SV-associated GAD and VGAT seems to be coupled because inhibition of GAD also
16                          By P15, the GAT and VGAT immunostaining patterns appear similar to the adult
17  acid decarboxylase) 65 and 67 isoforms, and VGAT (vesicular GABA transporter) in interneurons from t
18 e that 380-nm light stimulation via OPN5 and VGAT (the vesicular GABA/glycine transporter) in retinal
19 and the relative densities of SST-, PV-, and VGAT/SOX6-positive neurons were quantified.
20 ar transporters so far identified (VAChT and VGAT) were first described and cloned in C. elegans; in
21 o quantify the density of labeled VGLUT2 and VGAT immunoreactivity onto GnRH neurons.
22 ve behavioral effects, such as LH VGLUT2 and VGAT neurons [4-7] and orexin- (ORX) and melanin-concent
23 howed vesicular colocalization of VGLUT3 and VGAT.
24 % VGAT neurons but did not elicit LTP in any VGAT neurons even in morphine-treated mice.
25                      Some double-labeled BDA/VGAT varicosities were seen apposed to small somata labe
26 of receptors within genetically defined BNST VGAT neurons that may serve as therapeutic targets for r
27  activation of Gq-mediated signaling in BNST VGAT neurons and saw increased activity within ventral m
28 highlight that Gq-mediated signaling in BNST VGAT neurons can drive downstream network activity that
29 d that activation of hM3Dq receptors in BNST VGAT neurons can induce a long-term depression-like stat
30 ch to profile the receptorome of single BNST VGAT neurons.
31  GAD(65), taken up into synaptic vesicles by VGAT, and likely released by a vesicular mechanism from
32      Intriguingly, compared to CAMK2A cells, VGAT cells showed decreased remapping induced by environ
33 ctron microscopy by using well-characterized VGAT antibodies.
34                                          CRF/VGAT-only neurons were highest in the BSTLcn, lateral ce
35        In contrast, lower percentages of CRF/VGAT only neurons populated the sublenticular extended a
36 tively), which had higher complements of CRF/VGAT/VGluT2-labeled neurons (33%, 29%, 67%, respectively
37 led because inhibition of GAD also decreases VGAT activity.
38     Optogenetically increasing or decreasing VGAT(+) dPAG activity changes the probability of escape
39   Taken together, these findings demonstrate VGAT immunoreactivity in both amacrine and horizontal ce
40                             Developmentally, VGAT expression precedes VGLUT1.
41 vesicular GABA transporter (VGAT) (MPOA(ESR1 VGAT) neurons) robustly promoted USV(+) mounting, and co
42 al characterization of the newly established VGAT-ChR2(H134R)-EYFP, ChAT-ChR2(H134R)-EYFP, Tph2-ChR2(
43 medial ganglionic eminence and few expressed VGAT, found in GABAergic interneurons.
44 re occupied by mostly VGluT2 neurons and few VGAT neurons.
45 nclude that the activity of tonically firing VGAT(+) dPAG neurons sets a threshold for escape initiat
46 us and the neocortex, VGLUT3 was absent from VGAT-positive terminals in the striatum.
47                                 Furthermore, VGAT immunoreactivity overlapped or was immediately adja
48 (VGLUT1) and gamma-aminobutyric acid (GABA) (VGAT) transporter biosynthesis.
49 amatergic (VGlut1 and VGlut2) and GABAergic (VGAT) synapses demonstrated that overall synaptic patter
50  contrast, we found activation of GABAergic (VGAT+) SuM neurons (SuM(VGAT+)) neurons did not alter dr
51 rst-order neurons are inhibitory (GABAergic, VGAT(+)) or excitatory (glutamatergic, VGLUT2(+)).
52 ubunit mRNAs and diminished levels of GAD65, VGAT, GluR1, and GABAAR alpha1 and alpha2 were observed
53 in GABAergic neuronal markers (GAD65, GAD67, VGAT) and increased pro-inflammatory cytokines (TNF-alph
54 urons are inhibitory (GABAergic/glycinergic, VGAT-positive) in nature.
55 the channelrhodopsin-2 (ChR2) variant H134R [VGAT-ChR2(H134R)-EYFP] in a reduced synaptic preparation
56                                  Activity in VGAT(+) dPAG cells transiently decreases at escape onset
57  had any effect on mEPSCs or evoked EPSCs in VGAT neurons.
58 ed frequency of mEPSCs in VGluT2, but not in VGAT, dorsal horn neurons.
59 ng GABAergic SC neurons using optotagging in VGAT-ChR2 mice.
60 ons of a kainic acid-induced model of TLE in VGAT-ChR2 transgenic mice.
61 IPSCs and mIPSCs, correlating with increased VGAT (vesicular GABA transporter) puncta.
62 sing cells and a reduction in the inhibitory VGAT/gephyrin-positive synaptic sites on CA1 neurons in
63 ripts in excitatory (VGluT1) and inhibitory (VGAT) neurons across A1 layers; (2) heteromeric nAChR bi
64 , while ATR associates only with inhibitory (VGAT(+)) vesicles.
65 GAT+), and putative excitatory interneurons (VGAT-) in spinal cord slices from adult mice of both sex
66 rabrachial neurons, inhibitory interneurons (VGAT+), and putative excitatory interneurons (VGAT-) in
67 MAT2 expression in GABAergic neurons lacking VGAT is sufficient to sustain GABA release.
68 tive and acute chemogenetic activation of LH VGAT(+) neurons was profoundly wake promoting, whereas a
69                              In contrast, LH(VGAT) neuron inhibition or LH(VGLUT2) neuron activation
70 sed mice to report being food-restricted, LH(VGAT) neuron activation or LH(VGLUT2) neuron inhibition
71 ons drove calorie-specific feeding, while LH(VGAT) neurons drove calorie-indiscriminate food intake.
72 neuronal activity changes in the narcoleptic VGAT-Cre mice by expressing the calcium sensor GCaMP6 in
73 t, and puff NMDA currents in VGluT2, but not VGAT, neurons.
74 BS-induced LTP in VGluT2-expressing, but not VGAT-expressing, lamina II neurons.
75                                     Numerous VGAT and VGLUT2 labeled varicosities were observed appos
76 porter (VGAT) and NPY was found in 13-15% of VGAT-immunoreactive boutons in laminae I-II, and 5% of t
77                                   Only 6% of VGAT boutons presynaptic to large lamina I projection ne
78 emale mice to compare the neural activity of VGAT and CAMK2A cells during exploration of unaltered en
79                               The density of VGAT puncta increased with development, first within the
80 not control medium, increased the density of VGAT puncta.
81                                Expression of VGAT, GAD67, and GAT-1 was not associated with working m
82                          The localization of VGAT immunoreactivity to mouse and rat retina was evalua
83                                The number of VGAT-ir terminals onto GnRH dendrites was reduced in the
84                           Photoinhibition of VGAT neurons in the MeApd decreased LQ, and decreased c-
85 measured following either photoinhibition of VGAT or photoexcitation of VGluT2 neurons, and brains we
86         This led to a long-term reduction of VGAT density in the dorsal horn and reduced microglial p
87 in SPN local collaterals, down-regulation of VGAT expression in local processes of SPNs, and impaired
88 A was also coexpressed in a subpopulation of VGAT/VGluT2 mRNA ("multiplexed") cells, which were most
89  the relative densities of all SST-, PV-, or VGAT/SOX6-positive neurons were unaltered in schizophren
90 GluT2+) and GABA/glycine (known as VIAAT+ or VGAT+) vesicular transporters in the Mo5, Mo7, Amb, and
91 tained a single transporter, either VMAT2 or VGAT, we conclude that the secretory organelles of DAerg
92 nd that vesicular GABA transporter-positive (VGAT(+)) dPAG neurons fire action potentials tonically i
93 eased microglial engulfment of predominantly VGAT synapses but did not alter the engulfment of A-fibr
94 psin 2-enhanced yellow fluorescence protein (VGAT-ChR2-YFP)-expressing mice and a novel fibreoptic 'l
95 of this atypical dileucine-like motif in rat VGAT indicates that the transporter recycles by interact
96 ransduction that transcriptionally regulates VGAT by NO.
97                     In rat and mouse retina, VGAT occurred in the inner retina by postnatal day 1 (P1
98                               In rat retina, VGAT-immunoreactive cell bodies also contained GABA, gly
99 r antagonist or genetic deletion of Slc32a1 (VGAT) in Nts neurons unmasked also an excitatory effect
100                                     Specific VGAT immunoreactivity was localized to numerous varicose
101 vation of GABAergic (VGAT+) SuM neurons (SuM(VGAT+)) neurons did not alter drive aversion or active c
102  GnRH neurons, and at the time of the surge, VGAT-containing vesicles decrease and VGLUT2-containing
103  cells, and demonstrate optogenetically that VGAT neurons in the amygdala and bed nucleus of stria te
104 roscopy of mouse and rat retinae showed that VGAT immunoreactivity was localized to horizontal cell p
105 na II, we found that around one-third of the VGAT boutons that contacted them were NPY-immunoreactive
106                                       Third, VGAT and SV-associated Ca(2+)calmodulin-dependent kinase
107                                         Thus VGAT is the first of a new family of neurotransmitter tr
108 se varied with season in a manner similar to VGAT inputs.
109 se expressing the vesicular GABA transporter VGAT and primary afferent A-fibre terminals in neonates.
110 ocalized with the vesicular GABA transporter VGAT in the CA1 region of the hippocampus.
111 expression of the vesicular GABA transporter VGAT was unchanged; however, there was a significant inc
112 AT (also known as vesicular GABA transporter VGAT) transports GABA or glycine into synaptic vesicles.
113 expression of the vesicular GABA transporter VGAT, drastically suppresses drinking, even in water-cra
114 ndependent of the vesicular GABA transporter VGAT.
115 sicular gamma-aminobutyric acid transporter (VGAT)-expressing inhibitory neurons exhibited comparable
116 express ESR1 and vesicular GABA transporter (VGAT) (MPOA(ESR1 VGAT) neurons) robustly promoted USV(+)
117  presence of the vesicular GABA transporter (VGAT) and NPY was found in 13-15% of VGAT-immunoreactive
118      We used the vesicular GABA transporter (VGAT) as a marker for inhibitory presynaptic terminals t
119 er-2 (VGluT2) or vesicular GABA transporter (VGAT) neurons in the spinal dorsal horn of male and fema
120 ynaptophysin and vesicular GABA transporter (VGAT) revealed a group of small-sized ( approximately 0.
121 cid motif in the vesicular GABA transporter (VGAT) that controls its synaptic localization and activi
122  GAT-3), and the vesicular GABA transporter (VGAT) was evaluated by using immunohistochemistry with w
123 ression level of vesicular GABA transporter (VGAT) was upregulated, and no change in the synaptic ves
124 mparison of this vesicular GABA transporter (VGAT) with a vesicular transporter for monoamines shows
125 xpression of the vesicular GABA transporter (VGAT) within recurrent collaterals of SPNs.
126 nits, GAD65, the vesicular GABA transporter (VGAT), and the neuronal glutamate transporter (EAAC1) cD
127 ecarboxylase and vesicular GABA transporter (VGAT), markers of GABAergic neurons.
128 es, adult female vesicular GABA transporter (VGAT)-Cre and vesicular glutamate transporter 2 (VGluT2)
129 e depolarized in vesicular GABA transporter (VGAT)-expressing inhibitory neurons than those in vesicu
130 tory neurons and vesicular GABA transporter (VGAT)-expressing inhibitory neurons, which normally have
131 LTP in 12-16% of vesicular GABA transporter (VGAT)-expressing inhibitory neurons.
132 n-(ChR2)-EYFP in vesicular GABA transporter (VGAT)-expressing neurons.
133 ties followed by vesicular GABA transporter (VGAT).
134 h VGLUT3 and the vesicular GABA transporter (VGAT).
135 VGLUT-2), or the vesicular GABA transporter (VGAT).
136 esicles (SVs) by vesicular GABA transporter (VGAT).
137 imately 27%) and vesicular GABA transporter (VGAT)/p38 IR (approximately 41%) was found, but not in v
138 MAT2)] and GABA [vesicular GABA transporter (VGAT)].
139 that express the vesicular GABA transporter (VGAT; a marker for GABA-releasing neurons).
140 n schizophrenia: vesicular GABA transporter (VGAT; a marker of all GABA neurons) and SOX6 (a marker o
141  gamma-aminobutyric acid (GABA) transporter (VGAT) in patch and matrix throughout the striatum.
142  gamma-aminobutyric acid (GABA) transporter (VGAT) to horizontal cell processes in primate and rodent
143  gamma-aminobutyric acid (GABA) transporter (VGAT), and vesicular acetylcholine transporter (VAChT) t
144  gamma-aminobutyric acid (GABA) transporter (VGAT), which transports the inhibitory amino acid transm
145  gamma-aminobutyric acid (GABA) transporter (VGAT)-expressing BNST neurons using hM3Dq, but neither h
146  and the vesicular GABA/glycine transporter (VGAT) were evaluated in the developing mouse retina by u
147 inergic [vesicular GABA/glycine transporter (VGAT)] vesicular transporters in postnatal retina.
148 yme (GAD(65)) and its vesicular transporter (VGAT).
149 sin and the vesicular GABAergic transporter, VGAT).
150 cular GABA and glutamate (Glu) transporters (VGAT and VGLUT1, respectively), and preCGG hippocampal a
151                               A few varicose VGAT-immunoreactive processes entered the OPL from the I
152 lso found to receive dual-phenotype (VGLUT + VGAT) inputs; these varied with season in a manner simil
153 5-HT, and DbetaH expression, whereas VGLUT1, VGAT, and VAChT showed no change.
154   Amacrine cell somata characterized by weak VGAT immunoreactivity in the cytoplasm were located in t
155                                  At P5, weak VGAT immunostaining was also observed in horizontal cell
156          CRF mRNA(+) neurons colocalize with VGAT (GABA) and VGluT2 (glutamate) mRNAs in different pr
157 ed LQ, and decreased c-Fos expression within VGAT neurons, within the MeApd as a whole, and within th

 
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