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1 em, which is restricted to EI Tor biotype of Vibrio cholerae O1.
2 ed two novel non-toxigenic (ctxA/B-negative) Vibrio cholerae O1.
3 spected cases who tested positive to PCR for Vibrio cholerae O1.
4 ubunit of cholera toxin from classical Inaba Vibrio cholerae O1 569B; the strain also contains a merc
7 all cases of laboratory-confirmed toxigenic Vibrio cholerae O1 and O139 infection reported to the Ce
8 lus that is expressed by epidemic strains of Vibrio cholerae O1 and O139, is required for colonizatio
10 ulted in lower vibriocidal responses against Vibrio cholerae O1, and there was a positive relationshi
11 oled, adsorbed against in vitro-grown El Tor Vibrio cholerae O1, and used to probe a genomic expressi
17 r advance.PXVX0200, based on live attenuated Vibrio cholerae O1 classical Inaba vaccine strain CVD 10
18 annose-sensitive hemagglutinin (MSHA) of the Vibrio cholerae O1 El Tor biotype is a member of the fam
19 utinin (MSHA) to the colonization ability of Vibrio cholerae O1 El Tor biotype strains and O139 Benga
21 live attenuated oral vaccine derived from a Vibrio cholerae O1 El Tor Inaba strain by a series of de
24 investigated the transcriptional profile of Vibrio cholerae O1 El Tor strain C6706 under virulence g
28 e production of several virulence factors in Vibrio cholerae O1, including cholera toxin and the pilu
29 ing in an area in which at least one case of Vibrio cholerae O1 infection had been confirmed by cultu
30 symptomatic cholera, we estimated an annual Vibrio cholerae O1 infection incidence rate of 535 per 1
35 s, 73 (30%) Campylobacter isolates, 45 (18%) Vibrio cholerae O1 isolates, and 33 (14%) Salmonella iso
36 antibodies to the surface polysaccharide of Vibrio cholerae O1 (lipopolysaccharide) and of Vibrio ch
42 ss, and ecological interactions of toxigenic Vibrio cholerae O1 populations in two distinctive habita
43 From these isolates, we generated 44 whole Vibrio cholerae O1 sequences and analyzed them in the co
44 red between 1817 and 1923 and were caused by Vibrio cholerae O1 serogroup strains of the classical bi
45 ine-treated lipopolysaccharide (DeALPS) from Vibrio cholerae O1, serotype Inaba, to cholera toxin (CT
47 epitope of the O-specific polysaccharide of Vibrio cholerae O1, serotype Ogawa, were conjugated to b
50 igens of some Gram-negative bacteria such as Vibrio cholerae O1 (the causative agent of cholera) or E
51 a toxin and the toxin coregulated pilus, the Vibrio cholerae O1 virulence determinants having the lar
52 ntibodies and vibriocidal antibodies against Vibrio cholerae O1 were assessed using microneutralizati
53 between the classical and El Tor biotypes of Vibrio cholerae O1 were determined under conditions that
54 rapid, sensitive and selective detection of Vibrio cholerae O1 which converts the antibody-antigen b
55 g frame with significant homology to rfbE of Vibrio cholerae O1, which is postulated to encode perosa
56 dministration of the cholera vaccine (killed Vibrio cholerae O1 whole cells and recombinant cholera t