ライフサイエンスコーパス: Wernicke

1 Wernicke's aphasia is characterized by severe word and s

2 Wernicke's aphasia occurs after a stroke to classical la

3 Wernicke's area has been assumed since the 1800s to be t

4 Wernicke-Korsakoff syndrome in patients with cancer is u

5 internal capsule (RR, 2.2; 95% CI, 1.1-4.4), Wernicke's area (RR, 2.0; 95% CI, 1.1-3.8), or basal gan

6 tutor song but not for unfamiliar song in a Wernicke-like brain region (the caudomedial nidopallium)

7 Both semantic aphasia and Wernicke's aphasia cases showed multimodal semantic impa

8 herefore, patients with semantic aphasia and Wernicke's aphasia have partially distinct impairment of

9 ng activations at the anterolateral belt and Wernicke's area, where the responses were correlated wit

10 an thiamin metabolism result in beriberi and Wernicke encephalopathy.

11 area versus its right-hemisphere homolog and Wernicke's area versus its right-hemisphere homolog.

12 lization (e.g. Broca's area in the left, and Wernicke's area in the right hemisphere) have been repor

13 humans, Broca's area in the frontal lobe and Wernicke's area in the temporal lobe are crucially invol

14 cation of temporal lobe epilepsy, mania, and Wernicke's aphasia-compared to the sparse speech and cog

15 Language areas of the brain (Broca's and Wernicke's area) responded as the premises and the concl

16 sical arcuate pathway connecting Broca's and Wernicke's areas directly, we show a previously undescri

17 guage processing regions such as Broca's and Wernicke's areas for the word problems and the horizonta

18 while they were remote from both Broca's and Wernicke's areas in the remainder.

19 bidirectional connection between Broca's and Wernicke's areas probably through the arcuate fasciculus

20 nd not only in language regions (Broca's and Wernicke's areas), but also specific regions in bilatera

21 four regions of interest (ROIs): Broca's and Wernicke's areas, and their anatomic homologues in the r

22 These regions include Broca's and Wernicke's areas, primary auditory and visual cortex, an

23 arcuate fasciculus, which links Broca's and Wernicke's areas, the core neocortical language regions

24 essive right-sided activation of Broca's and Wernicke's areas.

25 other motor regions, and between Broca's and Wernicke's areas.

26 influenced cortical structure in Broca's and Wernicke's language areas, as well as frontal brain regi

27 zed in perisylvian cortex (i.e., Broca's and Wernicke's regions), while beta ERS was predominantly ri

28 f the direct connections between Broca's and Wernicke's territories in the left hemisphere.

29 es in direct connections between Broca's and Wernicke's territories, with extreme leftward lateraliza

30 thy subjects and seed regions in Broca's and Wernicke's, we recapitulate this extended network that i

31 hwind's region (i.e., anterior segment), and Wernicke's to Geschwind's region (i.e., posterior segmen

32 arcuate tract connecting Broca's speech and Wernicke's comprehension centers; a lesion of the tract

33 protocols to prevent refeeding syndrome and Wernicke encephalopathy.

34 he strength of the RSFC between the VWFA and Wernicke's area predicts performance on a semantic class

35 tivity was left lateralized (except anterior Wernicke's area that exhibited rightward lateralization)

36 ic error production to lesions in and around Wernicke's area.

37 tion of the underlying lesion site, known as Wernicke's area, remains controversial.

38 ogy in cases of diencephalic amnesia such as Wernicke Korsakoff Syndrome (WKS), there is also functio

39 cal network, but independently of awareness, Wernicke's area processes predictive events in time and

40 uage-specific effective connectivity between Wernicke's and Broca's areas in the PPA patient group.

41 Both Wernicke's area and its right homolog displayed a negati

42 imple classifications according to the Broca-Wernicke-Lichtheim model inadequately describe the diver

43 ing with its original discovery in monkey by Wernicke (1881) and in human by Obersteiner (1888), to i

44 n the superior temporal gyrus, the so-called Wernicke's area.

45 Twelve participants with chronic Wernicke's aphasia and 12 control participants performed

46 erior and middle temporal gyri, in classical Wernicke's area.

47 overlaps with some versions of the classical Wernicke area, the present results demonstrate its invol

48 Different from the classical Wernicke-Geschwind model, the present study revealed a b

49 demonstrated foci not only within classical Wernicke's area but also within the middle and inferior

50 and superior longitudinal fasciculi connect Wernicke's and Broca's areas.

51 egments of the arcuate fasciculus connecting Wernicke's to Broca's region (i.e., long segment), Broca

52 athway composed of three segments connecting Wernicke's to Broca's region (i.e. long segment), Wernic

53 risk allele, p=7.64x10(-5)), which contains Wernicke's area.

54 the left posterior superior temporal cortex (Wernicke's area).

55 Transmodal areas in the midtemporal cortex, Wernicke's area, the hippocampal-entorhinal complex and

56 nitive impairment, early Alzheimer dementia, Wernicke-Korsakoff syndrome, and "alcoholic dementia," a

57 The difficulty of diagnosing Wernicke's encephalopathy in the critical care setting i

58 egenerative disorders (Huntington's disease, Wernicke's encephalopathy and Parkinson's disease).

59 the development of the neurological disorder Wernicke-Korsakoff Syndrome as well as the potentially f

60 -correlated activity with auditory (only for Wernicke's area) and visual cortices that suggests integ

61 ith a different functional specificity; e.g. Wernicke's area responded specifically to speech sounds

62 l links with posterior language areas (e.g., Wernicke's area), because it is presumed to be involved

63 the left posterior superior temporal gyrus (Wernicke's area) and motor production processes occurred

64 ined activation only in the right hemisphere Wernicke's area in 4/5 of the cases.

65 s verbal and non-verbal modalities; and (ii) Wernicke's aphasia, associated with poor auditory-verbal

66 ions (AVHs) are accompanied by activation in Wernicke's and right homologous regions.

67 Activation in Wernicke's area in both hemispheres was obtained irrespe

68 ole brain and region of interest analysis in Wernicke's aphasia and control participants found that s

69 in time-to-peak concentration of contrast in Wernicke's area, relative to the homologous region on th

70 he hypothesis that comprehension deficits in Wernicke's aphasia result from double disconnection.

71 r, a memory-specific left-sided dominance in Wernicke's area for speech perception has been demonstra

72 ields; the language network on epicentres in Wernicke's and Broca's areas; the explicit memory/emotio

73 ly more activation than the control group in Wernicke's (left laterosuperior temporal) area and relia

74 l comprehension is significantly impaired in Wernicke's aphasia but the capacity to comprehend visual

75 rd and sentence comprehension impairments in Wernicke's aphasia come almost exclusively from patients

76 For 50 patients without infarct in Wernicke's area, we found a strong Pearson correlation b

77 the presence of hypoperfusion or infarct in Wernicke's area, we tested the hypothesis that the sever

78 tivation of concepts from sensory inputs (in Wernicke's aphasia).

79 tten word and picture semantic processing in Wernicke's aphasia, with the wider aim of examining how

80 Semantic processing of written words in Wernicke's aphasia was additionally supported by recruit

81 n various Brodmann's areas--BA 22 (including Wernicke's area), BA 44 (part of Broca's area), BA 45 (p

82 rior temporal regions bilaterally (including Wernicke's area).

83 Alcohol-induced Wernicke-Korsakoff syndrome (WKS) culminates in bilatera

84 demonstrated SSC's superiority at localizing Wernicke's area.

85 SSC is a robust paradigm for localizing Wernicke's area, making it an important clinical tool fo

86 the ICU with symptoms that may mimic or mask Wernicke's encephalopathy, we suggest abandoning the ban

87 proximately 180 msec), to cortex in and near Wernicke's ( approximately 210 msec) and then Broca's (

88 HGG tumors localized near Wernicke's area lead to transfer its function to the hea

89 ns remote from the classic language areas of Wernicke and Broca.

90 ons of the anatomically ill-defined areas of Wernicke and Broca.

91 Suspected cases of Wernicke's encephalopathy in other fedratinib trials led

92 a, ataxia and memory loss, characteristic of Wernicke encephalopathy.

93 t the planum temporale is a key component of Wernicke's receptive language area, which is also implic

94 y encompassing temporoparietal components of Wernicke's area, Broca's area, and dorsal premotor corte

95 A diagnosis of Wernicke encephalopathy was supported by magnetic resona

96 te matter damage, rather than dysfunction of Wernicke's area itself.

97 ited the entire triad of classic features of Wernicke-Korsakoff syndrome: confusion, ataxia, and opht

98 duced thiamine deficiency (PTD) rat model of Wernicke's encephalopathy (WE).

99 (PTD) was used to produce a rodent model of Wernicke-Korsakoff syndrome that results in acute neurol

100 deficiency and describe how rodent models of Wernicke-Korsakoff Syndrome aid in developing a more det

101 with the magnitude of delay in perfusion of Wernicke's area on magnetic resonance perfusion-weighted

102 add further evidence for the crucial role of Wernicke's area (Brodmann's area 22) in word comprehensi

103 Our results highlight the important role of Wernicke's area in forming vivid musical imagery through

104 The syndrome of Wernicke's aphasia is thus likely to reflect damage not

105 ces neurological symptoms similar to that of Wernicke-Korsakoff Syndrome in animals.

106 the functional and anatomical boundaries of 'Wernicke's area' have become so broad as to be meaningle

107 r temporal cortex [Brodmann area (BA) 37, or Wernicke's Wortschatz], left cerebellum, left thalamus a

108 emporal/fusiform gyrus, Broca's area, and/or Wernicke's area accounted for most acute improvement aft

109 (the left laterosuperior temporal cortex, or Wernicke's area, and the left inferior frontal gyrus, or

110 with tumors located close to the Broca's or Wernicke's areas of the left hemisphere.

111 the lesion affects the classical Broca's or Wernicke's language areas.

112 s area) and bilateral posterior perisylvian (Wernicke's area on the left) regions.

113 ge functional connectivity (except posterior Wernicke's area that exhibited predominant long-range co

114 ion or lateralization of language-receptive (Wernicke) (28% of patients) and language-expressive (Bro

115 vealed a posterior superior temporal region (Wernicke's area, traditionally considered a language-spe

116 lative underactivation in posterior regions (Wernicke's area, the angular gyrus, and striate cortex)

117 he temporal-lobe language-sensitive regions (Wernicke's area) also show effectively little or no sepa

118 ohol misuse disorders or non-alcohol-related Wernicke-Korsakoff syndrome in patients.

119 cy and associated features of cancer-related Wernicke-Korsakoff syndrome in the published literature.

120 aware of the risk factors for cancer-related Wernicke-Korsakoff syndrome, and that they are vigilant

121 cke's to Broca's region (i.e. long segment), Wernicke's to Geschwind's region (i.e. posterior segment

122 his pattern is not predicted by the standard Wernicke-Geschwind model, and may become apparent when l

123 oca's area) and posterior superior temporal (Wernicke's area) regions, in addition to other language

124 has been associated with superior temporal (Wernicke's) and inferior frontal (Broca's) cortical area

125 largely agree with the classical notion that Wernicke's area, defined here as middle superior tempora

126 K-core analysis showed that Wernicke's area was engaged by the "core" with weaker co

127 The Wernicke's aphasia group displayed an 'over-activation'

128 mantic impairment, although as expected, the Wernicke's aphasia group showed greater deficits on audi

129 and verbal fluency (FAS), in localizing the Wernicke's area and studied the association between geno

130 n clinical aphasiology today is based on the Wernicke-Lichtheim model of aphasia formulated in the la

131 and colleagues raised what they termed the 'Wernicke conundrum', noting widespread variability in th

132 e temporal and angular gyri corresponding to Wernicke's area.

133 n the thalamus and Broca's area extending to Wernicke's area, supporting the hypothesized (negative)

134 ent connecting the inferior parietal lobe to Wernicke's territory.

135 On one hand, fronto-temporal projections to Wernicke's area may not be unique to the arcuate fascicu

136 Articles relevant to Wernicke's encephalopathy, vitamin and electrolyte defic

137 etal language areas outside the traditional "Wernicke area," namely, in the middle temporal, inferior

138 tes were immediately adjacent to sites where Wernicke's aphasia was elicited in the same patients.

139 Whereas Wernicke's area interacted within the intrinsic auditory

140 ociative bundle connecting Broca's area with Wernicke's area, and other language regions of the tempo

141 ' effects of stimulus repetition: cases with Wernicke's aphasia showed initial improvement with repet

142 because of its functional connectivity with Wernicke's area.SIGNIFICANCE STATEMENT The visual word f

143 Patients with Wernicke's aphasia and semantic aphasia were distinguish

144 case series design to compare patients with Wernicke's aphasia and those with semantic aphasia on Wa

145 ere observed in the brain from patients with Wernicke-Korsakoff syndrome, the clinical manifestation

146 ds); (2) the VWFA has preferential RSFC with Wernicke's area and other core regions of the language s

147 arietal areas, traditionally included within Wernicke's area, leave single word comprehension intact

148 le severe chronic alcoholic subjects without Wernicke-Korsakoff disease compared with 7 male normal c