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1 ous RNA viruses (e.g., influenza A virus and West Nile virus).
2 ther emerging flaviviral infections, such as West Nile virus.
3 est Nile virus and an sfRNA-deficient mutant West Nile virus.
4 bility to infection by influenza A virus and West Nile virus.
5 HIV-1, Hepatitis C virus, Dengue virus, and West Nile virus.
6 ans, lymphocytic choriomeningitis virus, and West Nile virus.
7 rovide new insights into the pathogenesis of West Nile virus.
8 ed enhanced susceptibility to infection with West Nile virus.
9 acute respiratory syndrome coronavirus, and West Nile virus.
10 acute respiratory syndrome-coronavirus, and West Nile virus.
11 ontribute to weak, cross-reactive binding to West Nile virus.
12 rtality in mice infected with chikungunya or West Nile virus.
13 vine viral diarrhea virus, dengue virus, and West Nile virus.
14 es jamaicensis), and the recent emergence of West Nile virus.
15 h as dengue fever, malaria, chikungunya, and West Nile virus.
16 ated chimeric vaccine for protection against West Nile virus.
17 l as diagnostic testing for Dengue fever and West Nile virus.
18 st other flaviviruses, such as Zika virus or West Nile virus.
19 esions reported in cases of Dengue fever and West Nile virus.
20 iviral therapeutics against either Kunjin or West Nile viruses.
21 mosquito, the vector of filaria parasites or West Nile viruses.
22 viviruses, such as Japanese encephalitis and West Nile viruses.
23 avivirus related to yellow fever, dengue and West Nile viruses.
24 nd is similar to the one found in dengue and West Nile viruses.
25 other known flaviviruses such as dengue and West Nile viruses.
26 ublications to probe fusion of influenza and West Nile viruses.
27 lavivirus like the dengue, yellow fever, and West Nile viruses.
28 primary DENV3, 67 secondary DENV, 36 primary West Nile virus, 38 primary ZIKV, and 35 ZIKV with previ
31 nzootic (wildlife) cycles, as in the case of West Nile virus accompanying geographic expansion into t
32 We apply this framework to the spread of the West Nile virus across North America, an important recen
34 , we infected mosquitoes with the flavivirus West Nile virus and an sfRNA-deficient mutant West Nile
35 mmunologic exposure to arboviruses including West Nile virus and Eastern equine encephalitis virus ha
37 es evaluated in humans in settings including West Nile virus and HIV infection and in pre-exposure pr
38 to reveal that, for at least two infections, West Nile virus and Lyme disease, large hosts should be
40 ons against divergent RNA viruses, including West Nile virus and lymphocytic choriomeningitis virus.
41 Culex pipiens is the mosquito that vectors West Nile Virus and other human-pathogenic flavivruses i
43 r three (11%): two with potential pathogens (West Nile virus and Toxoplasma gondii) and one with Stre
44 able to reduce the replication of infectious West Nile virus and yellow fever virus in cell culture w
45 A viruses (Sindbis virus, hepatitis C virus, West Nile virus, and dengue virus), DNA viruses (vaccini
48 illustrated by bluetongue, Lyme disease, and West Nile virus, and it is also emerging, as illustrated
49 velope protein (E) of dengue viruses (DENV), West Nile virus, and Japanese encephalitis virus (JEV) a
51 ars, including epidemics of dengue virus and West Nile virus, and the most recent explosive epidemic
52 'nyong-nyong virus, Rift Valley fever virus, West Nile virus, and yellow fever virus), 8 bacteria (Ba
54 ow fever, dengue, Japanese encephalitis, and West Nile viruses, and vaccination with an inactivated v
55 ow fever, dengue, Japanese encephalitis, and West Nile viruses, and vaccination with an inactivated w
57 including tick-borne encephalitis virus and West Nile virus, antagonize IFN-I signaling by inhibitin
60 s like dengue virus, yellow fever virus, and West Nile virus are enveloped particles spread by mosqui
62 at all serotypes of dengue virus, as well as West Nile virus, are highly sensitive to both methotrexa
63 Virus, Chikungunya virus, dengue virus, and West Nile virus, as well the human polyomaviruses BK/JC/
66 ciency of Kunjin virus.IMPORTANCE Kunjin and West Nile viruses belong to the arthropod-borne flavivir
68 uding hepatitis C virus, yellow fever virus, West Nile virus, chikungunya virus, Venezuelan equine en
70 was little evidence that scaly-leg mites or West Nile virus contributed to recent declines in adult
72 iruses of the family Flaviviridae, including West Nile virus, dengue virus, and hepatitis C virus, as
73 culture by pathogenic RNA viruses, including West Nile virus, dengue virus, hepatitis C virus, influe
74 gue; and mosquito-borne dengue, malaria, and West Nile virus disease, include (a) selection of spatia
75 e utility of PrimalSeq by measuring Zika and West Nile virus diversity from varied sample types and s
76 y different viruses, including dengue virus, West Nile virus, Ebola virus, Marburg virus, and Zika vi
82 gressive declines over recent years, in 2012 West Nile virus epidemics resurged nationwide, with the
85 e its introduction in North America in 1999, West Nile virus has produced the 3 largest arboviral neu
87 e, including dengue, Zika, yellow fever, and West Nile virus, identifies conserved regions modified b
92 ral property of neurotropic flaviviruses, as West Nile virus indiscriminately killed both tumor and n
93 lly, capsid protein of Dengue virus, but not West Nile virus, induced ribosomal stress and apoptosis.
96 rulent Semliki Forest virus (SFV) as well as West Nile virus infection and demonstrate rapid and robu
97 included numbers of residents diagnosed with West Nile virus infection between May 30, 2012, and Dece
99 infections were strikingly inhibited, while West Nile virus infection was unchanged, in cells that l
103 sproportionately affecting the elderly, like West Nile virus, influenza virus, norovirus, or other em
107 inst dengue and related flaviviruses such as West Nile virus is the viral serine protease NS2B-NS3.
108 y relevant members of the flavivirus family: West Nile virus, Japanese encephalitis virus, and dengue
109 ncluding diffusive spread from an epicentre (West Nile virus), jump dispersal on a network (foot-and-
110 e role of motif V in viral replication using West Nile virus (Kunjin subtype) T407A and S411A mutants
112 he three N-linked glycosylation sites in the West Nile virus NS1 protein completely attenuates mouse
113 pes for protective antibody 22NS1, targeting West Nile Virus NS1, could potentially be valuable in un
116 exposed to any of the four dengue viruses or West Nile virus, or vaccinated against yellow fever viru
118 73 cases of WNND, 225 of West Nile fever, 17 West Nile virus-positive blood donors, and 19 deaths in
119 gh resolution X-ray cocrystal structure with West Nile virus protease provide a basis for the design
121 a complex formed during the interaction of a West Nile virus RNA stem loop structure with the human T
123 vated vaccinia virus or H(2)O(2)-inactivated West Nile virus showed high virus-specific neutralizing
125 ivo properties of previously uncharacterized West Nile virus strains and West Nile-like viruses.
127 Culex pipiens is a major carrier of the West Nile Virus, the leading cause of mosquito-borne dis
128 ielded a significant reduction of dengue and West Nile virus titers in cell-based assays of virus rep
129 the presence of morbillivirus, herpesvirus, West Nile virus, Toxoplasma gondii, and Brucella spp.
130 uch as eastern equine encephalitis virus and West Nile virus, underscore the need for research aimed
134 nt identifies current and future hotspots of West Nile virus where mitigation efforts should be focus
135 demonstrate single-tube duplex detection of West Nile virus (WNV) and chikungunya virus (CHIKV) RNA.
139 sid antigens of the viral zoonotic pathogens West Nile virus (WNV) and Rift Valley fever virus (RVFV)
143 ven the rapid spread of flaviviruses such as West Nile virus (WNV) and Zika virus, it is critical tha
145 decreased the replication of the flavivirus West Nile virus (WNV) as well as that of other types of
147 e transfer of immune plasma against DENV and West Nile virus (WNV) can enhance Zika virus (ZIKV) infe
149 V infection in ex vivo CNS tissue.IMPORTANCE West Nile virus (WNV) causes substantial morbidity and m
150 ells by the RNA virus dengue virus (DENV) or West Nile virus (WNV) does not result in the production
151 cleavage by furin of prM on partially mature West Nile virus (WNV) during virus entry contributes to
154 Il22(-/-) mice were more resistant to lethal West Nile virus (WNV) encephalitis, but had similar vira
159 e United States experienced one of its worst West Nile virus (WNV) epidemics, reporting 5,387 human c
161 nts who survive neuroinvasive infection with West Nile virus (WNV) exhibit chronic cognitive sequelae
165 e its introduction to North America in 1999, West Nile virus (WNV) has had devastating impacts on nat
166 e first introduced to North America in 1999, West Nile virus (WNV) has spread rapidly across the cont
167 introduction in New York City, NY, in 1999, West Nile virus (WNV) has spread to all 48 contiguous st
168 introduction in New York City, NY, in 1999, West Nile virus (WNV) has spread to all 48 contiguous st
170 ibution of the ISG viperin to the control of West Nile virus (WNV) in genetically deficient cells and
173 gan transplant recipients with donor-derived West Nile virus (WNV) infection (encephalitis 3, asympto
174 ntibody isolated from a patient, neutralizes West Nile virus (WNV) infection at a postattachment stag
175 I3K signaling is critical for the control of West Nile virus (WNV) infection by regulating type I IFN
177 tive immune responses is required to control West Nile virus (WNV) infection in peripheral and centra
179 ents with a clinical picture consistent with West Nile virus (WNV) infection, which was defined as no
186 acute Japanese encephalitis virus (JEV) and West Nile virus (WNV) infections is the premembrane/enve
187 s, chimeric viruses were generated using the West Nile virus (WNV) infectious clone, into which EIIIs
191 dentify novel therapeutic targets.IMPORTANCE West Nile virus (WNV) is a clinically relevant pathogen
210 g of WNV-specific T cell immunity.IMPORTANCE West Nile virus (WNV) is an encephalitic flavivirus that
223 previously produced a replication-defective West Nile virus (WNV) lacking NS1 (WNV-DeltaNS1) that co
227 ions 10 and 11 from dengue virus (DENV) into West Nile virus (WNV) NS1 (RQ10NK) changed its relative
229 ical determinants of distinct pathologies of West Nile virus (WNV) NY99 (pathogenic) and WNV Eg101 (n
230 mination, and lethality after infection with West Nile virus (WNV) or several other pathogenic viruse
231 t persist after recovery from infection with West Nile virus (WNV) or Zika virus (ZIKV) impact hippoc
232 alleles, we investigated how IRF5 modulates West Nile virus (WNV) pathogenesis and host immune respo
235 election on RNA virus genomes, we quantified West Nile virus (WNV) quasispecies diversity after passa
239 ted that type I interferon (IFN-I) restricts West Nile virus (WNV) replication and pathogenesis in pe
240 strict the vertebrate host's IFN-I response, West Nile virus (WNV) replication is sensitive to pretre
244 e illustrate our approach by focusing on the West Nile virus (WNV) spread in North America that has s
246 of human leukocyte antigen (HLA)-restricted West Nile virus (WNV) T-cell ligands and characterizatio
248 ChimeriVax-WN02 is a novel live-attenuated West Nile virus (WNV) vaccine containing modified WNV pr
249 ng host immunity against the live attenuated West Nile virus (WNV) vaccine strain, the nonstructural
251 will affect the abundance and seasonality of West Nile virus (WNV) vectors, altering the risk of viru
252 this epitope fail to recognize fully mature West Nile virus (WNV) virions and accordingly neutralize
253 14 subjects with a history of infection with West Nile virus (WNV), (ii) 34 healthy subjects of diffe
257 dently generated CMV-Cre Irf5fl/fl mice with West Nile virus (WNV), a pathogenic neurotropic flavivir
259 optimal priming of adaptive immunity against West Nile virus (WNV), an emerging vector-borne virus, d
260 ous system (CNS) that restricts infection by West Nile virus (WNV), an encephalitic flavivirus of glo
261 ts significance as an antiviral gene against West Nile virus (WNV), an encephalitic flavivirus, in ce
262 zed the antiviral activity of Ifitm3 against West Nile virus (WNV), an encephalitic flavivirus, using
263 NV, as well as against a related flavivirus, West Nile virus (WNV), and an alphavirus, Sindbis virus
264 n by dengue virus (DENV), Zika virus (ZIKV), West Nile virus (WNV), and hepatitis C virus (HCV).
265 including dengue viruses (DENV-1 to DENV-4), West Nile virus (WNV), and Japanese encephalitis virus (
266 flaviviruses, including dengue virus (DENV), West Nile virus (WNV), and Zika virus (ZIKV), highlight
267 virus, yellow fever virus, dengue virus, and West Nile virus (WNV), are a serious concern for human h
270 t the closely related dengue virus (DENV) or West Nile virus (WNV), can efficiently infect key placen
273 pairs replication of flaviviruses, including West Nile virus (WNV), dengue virus (DENV), and Zika vir
274 viruses from the Flavivirus genus, including West Nile virus (WNV), dengue virus (DENV), and Zika vir
275 E33 is sensitive to the maturation state of West Nile virus (WNV), despite its recognition of an acc
276 xtensive screen of CC-F1 lines infected with West Nile virus (WNV), including comprehensive immunophe
278 gence of mosquito-borne RNA viruses, such as West Nile virus (WNV), is facilitated by genetically com
279 ivirus (SCFV) vaccine candidate derived from West Nile virus (WNV), is intrinsically adjuvanted with
280 erpesvirus 68 (MHV-68) with influenza virus, West Nile virus (WNV), or vesicular stomatitis virus (VS
281 epresentative members of lineage one and two West Nile virus (WNV), previously was isolated from Cule
282 ow fever virus (YFV), Zika virus (ZIKV), and West Nile virus (WNV), profoundly affect human health.
283 naturally occurring nonpathogenic strain of West Nile virus (WNV), the Kunjin strain (WNV(KUN)), rem
286 tural dynamics of the frameshift signal from West Nile virus (WNV), which stimulates -1 PRF at very h
287 s with mortality among rodents infected with West Nile virus (WNV), which suggests that this is a pri
289 Examination of cellular miRNA profiles in West Nile virus (WNV)-infected HEK293 and SK-N-MC cells
290 onors provides opportunities for identifying West Nile virus (WNV)-infected persons before symptoms d
297 ropic RNA viruses (e.g., measles virus [MV], West Nile virus [WNV], Sindbis virus [SV], rabies virus
298 ne viruses) such as chikungunya, dengue, and West Nile viruses, yet for reasons that are largely unkn
299 nese encephalitis virus (YF/JE), or chimeric West Nile virus (YF/WN) vaccines, followed by a secondar
300 option for detection of newer TTIs including West Nile virus, Zika virus (ZIKV), and Babesia microti