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1 rotein shows significant homology to RpfF in Xanthomonas campestris.
2 resistance to a virulent bacterial pathogen, Xanthomonas campestris.
3 omonas syringae, Pseudomonas aeruginosa, and Xanthomonas campestris.
4 a, Ralstonia (Pseudomonas) solanacearum, and Xanthomonas campestris.
5 of Ralstonia (Pseudomonas) solanacearum and Xanthomonas campestris.
6 h foliar bacterial pathogens P. syringae and Xanthomonas campestris.
8 acterized PMMs of Pseudomonas aeruginosa and Xanthomonas campestris, albeit not in the enzymes from P
9 acterial pathogens, Pseudomonas syringae and Xanthomonas campestris, and an oomycete, Peronospora par
11 to few pathovars of Pseudomonas syringae and Xanthomonas campestris, but also enhanced the growth of
12 While phenotypes of a DeltarpfF strain in Xanthomonas campestris could be complemented by its own
14 of Erwinia amylovora, Pseudomonas spp., and Xanthomonas campestris has impeded the control of severa
16 transformed tryptophan 2,3-dioxygenase from Xanthomonas campestris into a monooxygenase for oxidativ
20 , mutagenesis of the active site cysteine in Xanthomonas campestris OleA (Cys(143)) enabled trapping
21 function of two B. subtilis homologs of the Xanthomonas campestris organic hydroperoxide resistance
24 onstrate that XopD, a type III effector from Xanthomonas campestris pathovar vesicatoria (Xcv), suppr
25 itness determinants of the vascular pathogen Xanthomonas campestris pv campestris (Xcc) during infect
26 dependent hypersensitive response (HR) after Xanthomonas campestris pv campestris (Xcc) infection.
29 e adapted vascular phytopathogenic bacterium Xanthomonas campestris pv campestris (Xcc), the causal a
30 aliana accession Landsberg erecta (Ler) with Xanthomonas campestris pv campestris isolate 2D520 resul
31 om cold-treated, heat-treated, and pathogen (Xanthomonas campestris pv campestris)-infected plants, c
32 resistance gene are resistant to strains of Xanthomonas campestris pv vesicatoria (Xcv) expressing t
33 tion of tomato (Lycopersicon esculentum) and Xanthomonas campestris pv vesicatoria (Xcv), to examine
35 e 35S::Pto lines also were more resistant to Xanthomonas campestris pv vesicatoria and Cladosporium f
36 s of both genotypes with virulent bacterial (Xanthomonas campestris pv vesicatoria and Pseudomonas sy
38 YopJ family effector from the plant pathogen Xanthomonas campestris pv vesicatoria, interacts with th
39 eviously identified 25.4-kbp pig region from Xanthomonas campestris pv. campestris (strain B-24).
40 p between the two signals in the Arabidopsis-Xanthomonas campestris pv. campestris (Xcc) compatible i
41 es and extracellular polysaccharide (EPS) in Xanthomonas campestris pv. campestris (Xcc) is regulated
42 ty acid signal DSF controls the virulence of Xanthomonas campestris pv. campestris (Xcc) to plants.
45 structure of apo Zur from the phytopathogen Xanthomonas campestris pv. campestris (XcZur), which rev
46 Alternaria brassicicola Abra43 (Abra43) and Xanthomonas campestris pv. campestris 8004 (Xcc8004), on
49 ) subgroup of the superfamily encoded by the Xanthomonas campestris pv. campestris str. ATCC 33913 ge
52 Banana Xanthomonas wilt disease, caused by Xanthomonas campestris pv. musacearum (Xcm), is a major
55 of the pepper Bs2 gene confers resistance to Xanthomonas campestris pv. vesicatoria (Xcv) pathogenic
58 Xanthomonas outer protein S (XopS), a T3E of Xanthomonas campestris pv. vesicatoria (Xcv), interacts
61 he region was most similar to hrp genes from Xanthomonas campestris pv. vesicatoria and Ralstonia sol
62 ar characterization of the avrBs2 locus from Xanthomonas campestris pv. vesicatoria has revealed that
63 lene-insensitive tomato plants infected with Xanthomonas campestris pv. vesicatoria have greatly redu
64 esponse to virulent and avirulent strains of Xanthomonas campestris pv. vesicatoria in tomato (Lycope
65 , we show that infection of pepper plants by Xanthomonas campestris pv. vesicatoria strains expressin
66 ognizes and confers resistance to strains of Xanthomonas campestris pv. vesicatoria that contain the
67 rulence gene of the bacterial plant pathogen Xanthomonas campestris pv. vesicatoria triggers disease
69 rotein D), a type III secreted effector from Xanthomonas campestris pv. vesicatoria, is a desumoylati
75 ve structural and biochemical studies of the Xanthomonas campestris TDO and a related protein SO4414
76 ize a SAM-I riboswitch (SAM-I(Xcc)) from the Xanthomonas campestris that regulates methionine synthes
77 and Vibrio cholerae, and the plant pathogen Xanthomonas campestris The bioconjugated phanorods could
80 aliana in response to the bacterial pathogen Xanthomonas campestris To tackle this challenge, we firs
82 enic bacteria, like Pseudomonas syringae and Xanthomonas campestris, use the type III secretion syste
84 -acetylglutamate synthase-kinase (NAGS-K) of Xanthomonas campestris, which is inhibited by arginine.
85 HfsA has sequence similarity to GumC from Xanthomonas campestris, which is involved in exopolysacc
86 such as Rhizobium meliloti (succinoglycan), Xanthomonas campestris (xanthan gum), and Salmonella ent
87 cterium carotovorum, Ralstonia solanacearum, Xanthomonas campestris, Xanthomonas oryzae, and Xylella
91 idensis (sIDO) indoleamine 2,3-dioxygenases, Xanthomonas campestris (XcTDO) tryptophan 2,3-dioxygenas
92 , Erwinia chrysanthemi and carotovora (out), Xanthomonas campestris (xps), Pseudomonas aeruginosa (xc
93 nomeric tetratricopeptide repeat domain from Xanthomonas campestris YbgF, which is also able to trime